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Artykuły w czasopismach na temat "Myosine1g"
CASSAR-MALEK, I., A. LISTRAT i B. PICARD. "Contrôle hormonal des caractéristiques des fibres musculaires après la naissance". INRAE Productions Animales 11, nr 5 (6.12.1998): 365–77. http://dx.doi.org/10.20870/productions-animales.1998.11.5.3965.
Pełny tekst źródłaPICARD, B., L. LEFAUCHEUR, B. FAUCONNEAU, H. REMIGNON, Y. CHEREL, E. BARREY i J. NEDELEC. "Dossier : Caractérisation des différents types de fibres musculaires dans plusieurs espèces : production et utilisation d’anticorps monoclonaux dirigés contre les chaînes lourdes de myosine rapide IIa et IIb". INRAE Productions Animales 11, nr 2 (2.04.1998): 145–63. http://dx.doi.org/10.20870/productions-animales.1998.11.2.3926.
Pełny tekst źródłaMaxeiner, Sebastian, Nian Shi, Carmen Schalla, Guelcan Aydin, Mareike Hoss, Simon Vogel, Martin Zenke i Antonio S. Sechi. "Crucial role for the LSP1–myosin1e bimolecular complex in the regulation of Fcγ receptor–driven phagocytosis". Molecular Biology of the Cell 26, nr 9 (maj 2015): 1652–64. http://dx.doi.org/10.1091/mbc.e14-05-1005.
Pełny tekst źródłaEldin, P., B. Cornillon, D. Mornet i JJ Léger. "Une nouvelle jeunesse pour les myosines". médecine/sciences 11, nr 7 (1995): 1005. http://dx.doi.org/10.4267/10608/2400.
Pełny tekst źródłaLecarpentier, Y., JC Lambry, D. Chemla i C. Coirault. "La myosine, moteur moléculaire musculaire." médecine/sciences 14, nr 10 (1998): 1077. http://dx.doi.org/10.4267/10608/913.
Pełny tekst źródłaMénétrey, Julie, Amel Bahloul i Anne Houdusse. "Une myosine à contre-sens". médecine/sciences 22, nr 2 (luty 2006): 120–22. http://dx.doi.org/10.1051/medsci/2006222120.
Pełny tekst źródłaJURIE, C., J. NÉDELEC i B. PICARD. "Production des anticorps monoclonaux spécifiques des chaînes lourdes de myosine". INRAE Productions Animales 11, nr 2 (2.04.1998): 146–49. http://dx.doi.org/10.20870/productions-animales.1998.11.2.3927.
Pełny tekst źródłaPICARD, B., i M. DURIS. "Caractérisation des chaînes lourdes de myosine dans le muscle de bovin". INRAE Productions Animales 11, nr 2 (2.04.1998): 150–52. http://dx.doi.org/10.20870/productions-animales.1998.11.2.3928.
Pełny tekst źródłaLEFAUCHEUR, L., i P. ECOLAN. "Composition en chaînes lourdes de myosine des fibres musculaires de type II chez le porc". INRAE Productions Animales 11, nr 2 (2.04.1998): 152–54. http://dx.doi.org/10.20870/productions-animales.1998.11.2.3929.
Pełny tekst źródłaRÉMIGNON, H., i V. DESROSIERS. "Recherche d’anticorps dirigés contre les différents types de fibres chez le poulet". INRAE Productions Animales 11, nr 2 (2.04.1998): 157–59. http://dx.doi.org/10.20870/productions-animales.1998.11.2.3931.
Pełny tekst źródłaRozprawy doktorskie na temat "Myosine1g"
Janardhana, Kurup Akshai. "Étude de la myosine nonconventionelle myosine1g dans l'asymétrie droite-gauche du poisson zèbre". Electronic Thesis or Diss., Université Côte d'Azur, 2022. http://www.theses.fr/2022COAZ6028.
Pełny tekst źródłaLeft-Right (LR) asymmetry refers to the asymmetric placement of organs across the midline. Dysregulation of LR asymmetry establishment during animal development can lead to misplaced organs - a situation that can be lethal.I use zebrafish as a model to study the mechanisms that establish LR asymmetry. In zebrafish, a ciliated organ - Kupffer's Vesicle (KV) acts as a central LR organizer (LRO). The Nodal ligand Southpaw (Spaw) and its antagonist Dand5 are initially expressed in a symmetric fashion around the LRO. The beating of cilia in the LRO establishes a directional fluid flow that represses Dand5 on the left, allowing Spaw to spread on the left side through auto-induction and thereby direct the laterality of heart, brain and viscera.In contrast to many vertebrates, Drosophila establishes LR asymmetry independently of cilia using chiral cell remodeling controlled by the unconventional Myosin Myo1D. Our group showed previously that zebrafish Myo1D regulates LR asymmetry by orienting KV cilia and promoting the formation of a symmetry-breaking fluid flow. In addition to myo1d, the zebrafish genome encodes the closely related gene myo1g. The objective of my PhD work has been to study the contribution of Myo1G to the establishment of zebrafish LR asymmetry.KV acts as a central LRO that controls the laterality of the different organs of the animal. Accordingly, myo1d mutants that have an altered LRO flow present LR defects at the level of the heart, brain and viscera. In contrast, myo1g mutants present LR defects in heart and brain but not viscera, suggesting that myo1g may exert a flow-independent function in LR asymmetry.In order to directly test this hypothesis, I inactivated myo1g in dnaaf1 mutants which lack ciliary motility and present therefore no LRO flow. While the asymmetric movement of cardiac precursors is randomized to either the left or right side in dnaaf1 single mutants, asymmetric precursor cell movement is altogether lost in myo1g ; dnaaf1 double mutants, demonstrating thereby that myo1g functions indeed independently of the LRO flow.My work reveals that in contrast to Myo1D which regulates LRO cilia orientation, Myo1G is required for the Nodal-mediated transfer of laterality information from the central LRO to different target tissues. In myo1g mutants, spaw expression remains limited to the posterior of the embryo, properly guiding the laterality establishment of the posterior viscera, whereas the anterior heart and brain fail to establish laterality.In the context of the establishment of Zebrafish LR asymmetry, the Nodal ligand Spaw binds to a receptor complex formed by Acvr2, Alk4 and the co-receptor Oep. Productive ligand/receptor interactions trigger the phosphorylation of the downstream transcription factors Smad2/3, which then translocate into the nucleus to activate downstream genes. Nodal signal transduction induces spaw itself (which therefore propagates through auto-induction) as well the Nodal antagonist lefty1.When I injected equal amounts of spaw mRNA in WT and myo1g mutants and assessed the response by monitoring lefty1 activation, myo1g mutant embryos displayed a weaker response to spaw overexpression than their WT siblings, indicating thereby an impairment in Nodal signal transduction. In contrast, misexpression of constitutively activated smad2 produced equivalent responses in WT and myo1g mutant animals.Of particular interest, previous studies have implicated Myosin1 proteins in the endocytic trafficking of TGFβ receptor molecules. Different endocytic pathways have been shown to either promote TGFβ receptor signal transduction or conversely trigger receptor degradation. My work shows that myo1g mutants present a decrease in the number of Nodal-receptor positives endosomes, suggesting thereby that Myo1G may regulate LR asymmetry by controlling TGFβ receptor trafficking
El, Bahloul Amel. "Caractérisation fonctionnelle et structurale de trois myosines non conventionnelles : la myosine VI, la myosine VIIa et la myosine IB d'Acanthamoeba". Paris 11, 2004. http://www.theses.fr/2004PA112138.
Pełny tekst źródłaHarricane-Vors, Marie-Cécile. "Etudes in vitro de l'interaction entre l'actine et des protéines associées : gelsoline, caldesmone, myosine". Montpellier 1, 1990. http://www.theses.fr/1990MON11303.
Pełny tekst źródłaLheureux, Karine. "Transduction mécano-chimique dans le muscle squelettique : étude comparative des complexes acto-myosine à l'état monomérique et filamenteux". Montpellier 1, 1995. http://www.theses.fr/1995MON1T015.
Pełny tekst źródłaBonafé, Nathalie. "Structure et régulation du complexe actine-myosine dans le muscle squelettique". Montpellier 1, 1994. http://www.theses.fr/1994MON1T009.
Pełny tekst źródłaStordeur, Jean-Marc. "Evaluation de la taille de l'infarctus du myocarde thrombolysé par le dosage de la myosine plasmatique". Montpellier 1, 1991. http://www.theses.fr/1991MON11213.
Pełny tekst źródłaSeyer, Pascal. "Etude de l'implication directe de l'activité mitochondriale dans la régulation de la différenciation des myoblastes". École nationale supérieure agronomique (Montpellier), 2005. http://www.theses.fr/2005ENSA0027.
Pełny tekst źródłaGuimard, Laurent. "Modélisation et synthèse de peptides interagissant avec une protéine cible : application au complexe calmoduline-RS20". Montpellier 1, 1995. http://www.theses.fr/1995MON1T037.
Pełny tekst źródłaBarjot, Catherine. "Propriétés in vitro des cellules satellites des muscles lents et rapides de lapin, et modalités de la régénération musculaire in vivo". Montpellier 1, 1995. http://www.theses.fr/1995MON1T036.
Pełny tekst źródłaRauzi, Matteo. "Mapping Subcellular Forces Controlling Morphogenesis". Thesis, Aix-Marseille 2, 2010. http://www.theses.fr/2010AIX22025.
Pełny tekst źródłaDuring embryonic development tissue remodeling leads to shape changes: for example, tissues can elongate, invaginate, and stretch. Distinct signaling pathways regulate in space and time these behaviors through the control ofthe actin cytoskeleton and of myosin-based tension required for cell shapechange. What is the spatiotemporal pattern of subcellular forcesthat orient tissue morphogenesis? What is the nature of the generated forces and finally, what lies at the origin of such forces? These are the main questions that my thesis tries to illuminate. I use the germbandelongation of the Drosophila embryo as a model system to investigate themechanics of tissue morphogenesis
Streszczenia konferencji na temat "Myosine1g"
SIMON, M. F., H. CHAP i L. DOUSTE-BLAZY. "EFFECTS OF SIN 1 ON PLATELET ACTIVATION INDUCED BY THROMBIN IN HUMAN PLATELETS". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643423.
Pełny tekst źródłaLefort, Claire, Mathieu Chalvidal, Alexis Parenté, Véronique BLANQUET, Henri Massias, Laetitia MAGNOL i Emilie Chouzenoux. "Imagerie 3D par microscopie multiphotonique appliquée aux sciences du vivant : la chaine instrumentale et computationnelle FAMOUS". W Les journées de l'interdisciplinarité 2022. Limoges: Université de Limoges, 2022. http://dx.doi.org/10.25965/lji.221.
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