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Baker, Brent A. "Characterization of skeletal muscle performance and morphology following acute and chronic mechanical loading paradigms". Morgantown, W. Va. : [West Virginia University Libraries], 2007. https://eidr.wvu.edu/etd/documentdata.eTD?documentid=5325.
Pełny tekst źródłaTitle from document title page. Document formatted into pages; contains xii, 270 p. : ill. (some col.). Includes abstract. Includes bibliographical references.
Short, Kevin R. "Histochemical and biochemical changes in human muscle following 17 days of unilateral lower limb suspension". Virtual Press, 1997. http://liblink.bsu.edu/uhtbin/catkey/1063203.
Pełny tekst źródłaHuman Performance Laboratory
Tomc, Lyn Kathryn. "Role of MEF2 proteins in the activation of the c-jun and MCK genes in skeletal muscle /". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0018/MQ56210.pdf.
Pełny tekst źródłaPathare, Neeti C. "Metabolic adaptations following disuse and their impact on skeletal muscle function". [Gainesville, Fla.] : University of Florida, 2005. http://purl.fcla.edu/fcla/etd/UFE0010024.
Pełny tekst źródłaTypescript. Title from title page of source document. Document formatted into pages; contains 171 pages. Includes Vita. Includes bibliographical references.
Yost, John. "Influence of selection for breast muscle mass on pH and metabolism of supracoracoideus muscle from male and female turkey". Morgantown, W. Va. : [West Virginia University Libraries], 1999. http://etd.wvu.edu/templates/showETD.cfm?recnum=892.
Pełny tekst źródłaTitle from document title page. Document formatted into pages; contains viii, 81 p. : ill. Vita. Includes abstract. Includes bibliographical references.
Monteiro, André Antonio. "Blood flow change in human masseter muscle elicited by voluntary isometric contraction". Stockholm : Kongl. Carolinska Medico Chirurgiska Institutet, 1990. http://catalog.hathitrust.org/api/volumes/oclc/21700760.html.
Pełny tekst źródłaPasquier, Bernard. "Traitement chirurgical des paralysies du muscle grand oblique de l'œil par affaiblissement du muscle petit oblique". Bordeaux 2, 1995. http://www.theses.fr/1995BOR23087.
Pełny tekst źródłaKim, Jeong-Su. "The relationship of growth factor and muscle soreness to muscle hypertrophy". Virtual Press, 1998. http://liblink.bsu.edu/uhtbin/catkey/1101585.
Pełny tekst źródłaSchool of Physical Education
Hourdé, Christophe Keller Angélica Ferry Arnaud. "Étude de la coordination des phénotypes contractile et métabolique du muscle strié squelettique à travers des modèles animaux". Créteil : Université de Paris-Val-de-Marne, 2007. http://doxa.scd.univ-paris12.fr:8080/theses-npd/th0393949.pdf.
Pełny tekst źródłaVersion électronique uniquement consultable au sein de l'Université Paris 12 (Intranet). Titre provenant de l'écran-titre. Bibliogr. : 410 réf.
Fernandes, Santos Amanda Aparecida. "The effect of joint angle on surface electromyography amplitude of hamstring muscles". Thesis, Queensland University of Technology, 2022. https://eprints.qut.edu.au/231385/1/Amanda%20Aparecida_Fernandes%20Santos_Thesis.pdf.
Pełny tekst źródłaByrne, Christopher. "Muscle function after exercise-induced muscle damage". Thesis, Bangor University, 2001. https://research.bangor.ac.uk/portal/en/theses/muscle-function-after-exerciseinduced-muscle-damage(2bbf5fe1-f35b-4b7b-9790-ff3a04b86875).html.
Pełny tekst źródłaKjellgren, Daniel. "Human extraocular muscles : molecular diversity of a unique muscle allotype". Doctoral thesis, Umeå : Univ, 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-260.
Pełny tekst źródłaArc-Chagnaud, Coralie. "Regulation of antioxidant defenses in the prevention of skeletal muscle deconditioning". Thesis, Montpellier, 2019. http://www.theses.fr/2019MONT4005.
Pełny tekst źródłaMusculoskeletal system plays a key role in organism’s well-functioning and is responsible for a large variety of functions such as posture, locomotion, balance, and activities of daily life. The quality of the skeletal muscle is therefore capital to maintain quality of life and, in the long term, survival. Hypoactivity and aging are two situations that cause skeletal muscle deconditioning, therefore sharing common characteristics: loss of muscle strength, muscular atrophy and MyHC redistribution, as well as IMAT accumulation. To date, there is plenty of evidence supporting a causative link between oxidative stress phenomenon and muscle deconditioning.The general aim of this PhD thesis was to evaluate the impact of the modulation of the antioxidant defenses on the prevention of muscle deconditioning. It has been studied from two perspectives, the first one in the context of aging and the second in the context of hypoactivity.The first study aimed to evaluate frailty in old female animals, using WT and G6PD-overexpressing mice. We evaluated muscle quality parameters and oxidative stress markers. Finally, we performed a transcriptomic analysis of muscle samples and highlighted differentially expressed genes in both groups of mice.The second study was conducted to evaluate the effects of a cocktail enriched in antioxidant/anti-inflammatory molecules in a 2-month hypoactivity experiment (Bedrest model). Our results clearly demonstrate the ineffectiveness of this type of supplementation in the prevention of muscle mass and strength loss. Moreover, data regarding muscle molecular mechanisms highlight an alteration of recovery processes in the supplemented subjects.Finally, the conclusions of our two studies gave clues on the suitable antioxidant modulation strategy for the prevention of skeletal muscle deconditioning. It seems preferable to focus on the stimulation of endogenous defense system whether than towards exogenous supply of nutritional antioxidants. Nevertheless, the complexity of redox signaling requires better understanding to optimize countermeasures in muscle wasting situations
Wood, Stephanie Ann Cardinal Trevor R. "A morphological and hemodynamic analysis of skeletal muscle vasculature : a thesis /". [San Luis Obispo, Calif. : California Polytechnic State University], 2008. http://digitalcommons.calpoly.edu/theses/16/.
Pełny tekst źródła"July 2008." "In partial fulfillment of the requirements for the degree [of] Master of Science in Engineering with a specialization in Biomedical Engineering." "Presented to the faculty of California Polytechnic State University, San Luis Obispo." Major professor: Trevor Cardinal, Ph.D. Includes bibliographical references (leaves 96-101). Also available on microfiche and online.
McMillan, Anne Sinclair. "Human masseter motor unit behaviour". Thesis, University of British Columbia, 1989. http://hdl.handle.net/2429/30673.
Pełny tekst źródłaDentistry, Faculty of
Graduate
Gordon, Debra Anne. "Analysis of force parameters used to assess the fatigability of mammalian motor units". Diss., The University of Arizona, 1988. http://hdl.handle.net/10150/184368.
Pełny tekst źródłaArcher, Akibi A. A. "Two dimensional spatial coherence of skeletal muscle's natural vibrations during voluntary contractions". Thesis, Georgia Institute of Technology, 2010. http://hdl.handle.net/1853/42803.
Pełny tekst źródłaDiaz, Zagal Sergio. "Conception et développement d’un mini-actionneur à muscle artificiel : application à la robotique médicale". Toulouse, INSA, 2007. http://eprint.insa-toulouse.fr/archive/00000205/.
Pełny tekst źródłaThe experience of the laboratory in the matter of pneumatic artificial muscle for the robotics is applied to the development of mini-actuators with artificial muscles intended for miniature robot-like systems. The field of medical robotics is particularly aimed. The goal of this work is to develop actuators whose characteristics and performances correspond to those presented by the natural muscles. These actuators will be used in the field of robotics and the medical field. Thus, initially we were interested in the development and the study of the pneumatic artificial muscle of McKibben. It is a type of very powerful actuator, presenting satisfactory properties, approaching those of the natural muscles enormously. So their use in the field of robotics is very advantageous. However, the energy source that they use - compressed air - presents a serious restraint with respect to a flexible or medical use. So in the second time, we adapted these muscles of McKibben to the biomedical field by replacing the compressed air by exchanging resins of ions or hydrogels. We thus developed and studied muscles mecanochimic based on McKibben’s muscles
Aydin, Jan. "Skeletal muscle calcium homeostasis during fatigue : modulation by kinases and mitochondria /". Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-247-7/.
Pełny tekst źródłaFoxton, Ruth. "Dysferlin in skeletal muscle and skeletal muscle disease". Thesis, University of Newcastle Upon Tyne, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268429.
Pełny tekst źródłaVlahovich, Nicole. "The role of cytoskeletal tropomyosins in skeletal muscle and muscle disease". Thesis, View thesis, 2007. http://handle.uws.edu.au:8081/1959.7/32176.
Pełny tekst źródła潘明施 i Ming-see Angela Poon. "Modulation of cutaneous reflexes in a finger muscle during voluntary contractions". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1990. http://hub.hku.hk/bib/B31209956.
Pełny tekst źródłaSong, Yafeng, Sture Forsgren, Jiguo Yu, Ronny Lorentzon i Per Stål. "Effects on contralateral muscles after unilateral electrical muscle stimulation and exercise". Umeå universitet, Anatomi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-64452.
Pełny tekst źródłaGerber, Aimee. "Determination of strength imbalance of the lower extremities". Virtual Press, 2002. http://liblink.bsu.edu/uhtbin/catkey/1231347.
Pełny tekst źródłaSchool of Physical Education
Witt, Jonathan Derek. "Inspiratory muscle training attenuates the human respiratory muscle metaboreflex". Thesis, University of British Columbia, 2007. http://hdl.handle.net/2429/31606.
Pełny tekst źródłaEducation, Faculty of
Kinesiology, School of
Graduate
Pillitteri, Paul J. "Regeneration of Rat Skeletal Muscle Following a Muscle Biopsy". Ohio University / OhioLINK, 2002. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1118087917.
Pełny tekst źródłaWoodhouse, Samuel. "The role of Ezh2 in adult muscle stem cell fate". Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610201.
Pełny tekst źródłaGazzola, Morgan, i Morgan Gazzola. "Le gain de force du muscle lisse des voies aériennes dans l'asthme : une étude translationnelle". Doctoral thesis, Université Laval, 2019. http://hdl.handle.net/20.500.11794/37226.
Pełny tekst źródłaL’asthme est un désordre respiratoire obstructif qui affecte plus de 330 millions de personnes à travers le monde. Les symptômes de cette pathologie comprennent de l’essoufflement, de l’oppression thoracique, de la sibilance et de la toux, et surviennent suivant l’inhalation de facteurs déclencheurs (virus, allergène, pollution…). La pathologie de l’asthme est caractérisée par une inflammation chronique et variable au sein du système respiratoire, un remodelage des voies aériennes ainsi qu’une hyperréactivité bronchique. L’équipe de recherche du Dr Ynuk Bossé travaille sur la physiologie du muscle lisse des voies aériennes, et plus spécifiquement sur l’augmentation des capacités contractiles du muscle lisse en réponse à un tonus (i.e. contraction soutenue). Ce phénomène nommé le gain de force du muscle lisse a été observé avant mon arrivée dans ce laboratoire sur des trachées de moutons et de souris montées en bain d’organe, ainsi qu’in vivo chez la souris. Des travaux antérieurs ont démontré que la présence d’un tonus augmentait la réactivité bronchique de souris en réponse à l’inhalation d’une forte dose de métacholine. Le but de cette thèse était d’explorer dans une dynamique translationnelle, les rouages moléculaires de ce phénomène et d’en définir les impacts sur la fonction respiratoire in vivo. Dans une première étude réalisée au début de mon doctorat, nous avons étudié les conséquences du tonus sur la réactivité bronchique in vivo chez l’humain. Nous avons ainsi observé que l’augmentation du tonus, provoquée par l’inhalation répétée de faibles doses de métacholine durant une période de 30 minutes, augmentait la réactivité bronchique en réponse à l’inhalation d’une forte dose de métacholine. De plus, nous avons également observé grâce à l’utilisation de la technique des oscillations forcées que cette augmentation de la réactivité bronchique était liée à une augmentation de la résistance des voies périphériques. Nous avons donc confirmé dans cette étude que la présence d’un tonus augmente la réactivité bronchique chez de jeunes humains en santé. Dans une seconde étude, conduite tout au long de mon doctorat, nous avons étudié les mécanismes moléculaires responsables du gain de force du muscle lisse en réponse à un tonus. Nous avons ainsi observé que ce phénomène n’était pas lié à une potentialisation de la phosphorylation de la chaîne légère de myosine, mais plutôt provoqué par une augmentation de la filamentogénèse d’actine. Nous avons également déterminé que cette augmentation de la filamentogénèse d’actine était en partie provoquée par une inhibition de la dépolymérisation des filaments d’actine suite à l’inhibition de la protéine cofiline. Nous avons donc démontré que le tonus augmente la filamentogénèse d’actine au sein des cellules musculaires lisses, ce qui pourrait contribuer à une augmentation des capacités contractiles. Finalement, dans une troisième étude entreprise durant la dernière partie de mon doctorat, nous tentons de comprendre les liens entre le phénomène du gain de force du muscle lisse et l’inflammation présente dans l’asthme. Il semblerait que la présence d’une inflammation provoquée par de la poudre d’acariens chez la souris augmente la réactivité bronchique, mais empêche le développement du gain de force du muscle lisse. En revanche, les résultats obtenus sont encore préliminaires. Il est actuellement impossible de tirer des conclusions fermes. Ainsi, nous nous interrogeons toujours à l’égard du rôle de l’inflammation sur le gain de force du muscle lisse des voies aériennes. Dans sa globalité, cette thèse démontre que le gain de force du muscle lisse des voies aériennes est provoqué par un remodelage du cytosquelette d’actine, et que ce phénomène augmente la réactivité bronchique in vivo chez l’humain. De plus, cette thèse ouvre des voies de recherche afin de déterminer si ce phénomène pourrait être impliqué dans l’hyperréactivité bronchique dans l’asthme.
Asthma is an obstructive respiratory disorder affecting more than 330 million people worldwide. The symptoms include breathlessness, chest oppression, wheezing and cough. The symptoms are variable in nature and severity and generally coincide with the inhalation of environmental factors (viruses, allergens, pollution…). The pathology of asthma is characterized by several typical features, such as airway inflammation, airway remodeling and airway hyperresponsiveness. The research team of Dr Ynuk Bossé is specialized in the study of lung physiology and airway smooth muscle mechanics. Of particular interest is a phenomenon called ‘force adaptation’. Force adaptation is a time-dependent gain in the contractile capacity of airway smooth muscle in response to tone (i.e., a sustained contraction). This phenomenon was observed in vitro in isolated ovine and murine tissues, as well as in vivo in mice. Previous work has demonstrated that the presence of tone, provoked by repeated exposures to low doses of methacholine during 20 min, increases airway responsiveness to the inhalation of a high dose of methacholine. The aim of this thesis was to decipher the molecular mechanisms of force adaptation in vitro and to explore the impact of this phenomenon on respiratory function in vivo. In a first study, which was conducted at the beginning of my PhD, we assessed the effect of tone on airway responsiveness in young healthy adults. We demonstrated that tone, which was generated by repeated inhalations of low doses of methacholine during 30 min, enhances airway responsiveness to a high dose of methacholine. Moreover, with the use of the force oscillation technique, we demonstrated that this effect was predominant in the peripheral airways. Therefore, this study confirmed that airway smooth muscle tone increases airway responsiveness in young healthy adults. In a second study, conducted over the entire course of my PhD, we investigated the molecular mechanisms responsible for the gain in contractile capacity induced by tone. We observed that force adaptation does not rely on molecular mechanisms enhancing the phosphorylation of the myosin light chain but rather occurs in conjunction with an increase in actin filamentogenesis. We further demonstrated that this increase in actin filamentogenesis may stem not only from actin polymerization but also from the inhibition of actin filament depolymerization via the inhibition of the protein cofilin. Therefore, the results of this study suggested that tone increase the contractile capacity of airway smooth muscle by fostering actin filamentogenesis. Finally, in a third study started at the end of my PhD, we are trying to understand the links between the gain in contractile capacity induced by tone and airway inflammation in asthma. We are using a mouse model of allergic airway inflammation induced by repeated exposures to house dust mite. While allergic inflammation increases airway responsiveness, it seems to diminish the phenomenon of force adaptation. However, the results obtained so far will require further investigations. It is currently impossible to reach authoritative conclusions. We are still left wondering whether airway inflammation alters the gain in contractile capacity induced by tone. Overall, this thesis is demonstrated that force adaptation increases airway responsiveness in vivo in human and, at the molecular level, the phenomenon seems to rely on an active remodeling of the actin cytoskeleton. Moreover, this thesis opens new research areas, which will need to be further explored in order to determine whether the gain in contractile capacity induced by tone is implicated in airway hyperresponsiveness in asthma
Asthma is an obstructive respiratory disorder affecting more than 330 million people worldwide. The symptoms include breathlessness, chest oppression, wheezing and cough. The symptoms are variable in nature and severity and generally coincide with the inhalation of environmental factors (viruses, allergens, pollution…). The pathology of asthma is characterized by several typical features, such as airway inflammation, airway remodeling and airway hyperresponsiveness. The research team of Dr Ynuk Bossé is specialized in the study of lung physiology and airway smooth muscle mechanics. Of particular interest is a phenomenon called ‘force adaptation’. Force adaptation is a time-dependent gain in the contractile capacity of airway smooth muscle in response to tone (i.e., a sustained contraction). This phenomenon was observed in vitro in isolated ovine and murine tissues, as well as in vivo in mice. Previous work has demonstrated that the presence of tone, provoked by repeated exposures to low doses of methacholine during 20 min, increases airway responsiveness to the inhalation of a high dose of methacholine. The aim of this thesis was to decipher the molecular mechanisms of force adaptation in vitro and to explore the impact of this phenomenon on respiratory function in vivo. In a first study, which was conducted at the beginning of my PhD, we assessed the effect of tone on airway responsiveness in young healthy adults. We demonstrated that tone, which was generated by repeated inhalations of low doses of methacholine during 30 min, enhances airway responsiveness to a high dose of methacholine. Moreover, with the use of the force oscillation technique, we demonstrated that this effect was predominant in the peripheral airways. Therefore, this study confirmed that airway smooth muscle tone increases airway responsiveness in young healthy adults. In a second study, conducted over the entire course of my PhD, we investigated the molecular mechanisms responsible for the gain in contractile capacity induced by tone. We observed that force adaptation does not rely on molecular mechanisms enhancing the phosphorylation of the myosin light chain but rather occurs in conjunction with an increase in actin filamentogenesis. We further demonstrated that this increase in actin filamentogenesis may stem not only from actin polymerization but also from the inhibition of actin filament depolymerization via the inhibition of the protein cofilin. Therefore, the results of this study suggested that tone increase the contractile capacity of airway smooth muscle by fostering actin filamentogenesis. Finally, in a third study started at the end of my PhD, we are trying to understand the links between the gain in contractile capacity induced by tone and airway inflammation in asthma. We are using a mouse model of allergic airway inflammation induced by repeated exposures to house dust mite. While allergic inflammation increases airway responsiveness, it seems to diminish the phenomenon of force adaptation. However, the results obtained so far will require further investigations. It is currently impossible to reach authoritative conclusions. We are still left wondering whether airway inflammation alters the gain in contractile capacity induced by tone. Overall, this thesis is demonstrated that force adaptation increases airway responsiveness in vivo in human and, at the molecular level, the phenomenon seems to rely on an active remodeling of the actin cytoskeleton. Moreover, this thesis opens new research areas, which will need to be further explored in order to determine whether the gain in contractile capacity induced by tone is implicated in airway hyperresponsiveness in asthma
Watt, Kevin. "Regualtion of myogenesis and skeletal muscle size by the myostatin-Smad and mammalian Hippo signalling transduction pathways". Available from the University of Aberdeen Library and Historic Collections Digital Resources, 2009. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=62160.
Pełny tekst źródłaLosseff, Stéphane. "Les abcès du psoas, à propos de 7 cas vus en deux ans à l'hôpital de Perpignan". Montpellier 1, 1996. http://www.theses.fr/1996MON11115.
Pełny tekst źródłaVidal, Marie-Claire. "Un cas de rupture complète du muscle demi-membraneux". Bordeaux 2, 1995. http://www.theses.fr/1995BOR2M121.
Pełny tekst źródłaVlahovich, Nicole. "The role of cytoskeletal tropomyosins in skeletal muscle and muscle disease". View thesis, 2007. http://handle.uws.edu.au:8081/1959.7/32176.
Pełny tekst źródłaA thesis presented to the University of Western Sydney, College of Health and Science, School of Natural Sciences, in fulfilment of the requirements for the degree of Doctor of Philosophy. Includes bibliographies.
Simmers, Jessica L. "nNos localization, muscle function and atrophy in skeletal muscle disorders". Thesis, The Johns Hopkins University, 2013. http://pqdtopen.proquest.com/#viewpdf?dispub=3573097.
Pełny tekst źródłaIn skeletal muscle, loss of neuronal nitric oxide synthase (nNOS) from the sarcolemma has been observed in a few muscular dystrophies and myopathies. However, the extent of this phenomenon, its mechanism, and its physiological impact are not well understood. Using immunofluorescent staining for nNOS, a survey of 161 patient biopsies found absent or reduced sarcolemmal nNOS in 43% of patients. Patient mobility and muscle functional status correlated with nNOS mislocalization from the sarcolemma. Mouse models of inherited and acquired myopathies showed similar loss of sarcolemmal nNOS and impaired mobility and muscle function. A proteomic approach, using mass spectrometry and differentially labeled control and steroid-induced myopathy (SIM) mouse samples, found novel nNOS binding proteins including alpha-actinin-3 (ACTN3), which exhibited decreased interaction with nNOS after steroid treatment. It revealed a potential explanation for impaired muscle function in SIM as nNOS interactions were lost at the sarcomere and gained at the sarcoplasmic reticulum impairing contractility. Treating nNOS-deficient mice with steroids demonstrated that loss of sarcolemmal nNOS reduces muscle contractility and strength in SIM through increased nitric oxide (NO) signaling. In SIM mice treated with a nitric oxide donor and steroids, nitric oxide partially protects the muscle from atrophy and improves muscle fatigability and recovery suggesting nNOS mislocalization also decreases NO availability. These findings show that loss of sarcolemmal nNOS is a common phenomenon that negatively impacts muscle function. Therapeutic strategies targeting nNOS or NO signaling need to allow for the complexity of local nitric oxide content and cellular context.
Naamani, Randa. "The mechanical effects of muscle contractions of muscle blood flow /". Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=60010.
Pełny tekst źródłaTarabees, Reda Zakaria Ibrahim. "Endotoxin induced muscle wasting in avian and murine skeletal muscle". Thesis, University of Nottingham, 2011. http://eprints.nottingham.ac.uk/13001/.
Pełny tekst źródłaCharge, Sophie Barbara Pauline. "Skeletal muscle hypertrophy : its regulation and effect on muscle regeneration". Thesis, King's College London (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340500.
Pełny tekst źródłaMoura, Ferreira Polyana. "Reorganization of jaw muscle activity during experimental jaw muscle pain". Thesis, The University of Sydney, 2017. http://hdl.handle.net/2123/18255.
Pełny tekst źródłaPESSINA, PATRIZIA. "Necdin enhances muscle reconstitution of dystrophic muscle by mesoangioblast cells". Doctoral thesis, Università degli Studi di Milano-Bicocca, 2009. http://hdl.handle.net/10281/7594.
Pełny tekst źródłaYeung, Wai Ella, i 楊慧. "Eccentric contraction-induced injury in mammalian skeletal muscle". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B29750313.
Pełny tekst źródłaGoddard-Marshall, Ayana A. "Characterization of the activity of the involuntary calf muscle pump". Diss., Online access via UMI:, 2009.
Znajdź pełny tekst źródłaIncludes bibliographical references.
Arnold, Ludovic Chazaud Bénédicte. "Rôle des macrophages dans la régénération du muscle strié squelettique". Créteil : Université de Paris-Val-de-Marne, 2007. http://doxa.scd.univ-paris12.fr:8080/theses-npd/th0357710.pdf.
Pełny tekst źródłaLau, Wing Yin. "Pain assessment and possible mechanism of delayed onset muscle soreness". Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2014. https://ro.ecu.edu.au/theses/1275.
Pełny tekst źródłaAskling, Carl. "Hamstring muscle strain". Doctoral thesis, Swedish School of Sport and Health Sciences, GIH, Laboratory for Biomechanics and Motor Control, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:gih:diva-425.
Pełny tekst źródłaBackground: Acute hamstring strains are common injuries in different sports. They are often serious, causing long rehabilitation times and a proneness for re-injury. Preliminary observations indicate that the injuries can be of at least two types, one occurring during high-speed running and the other during motions where the hamstring muscles reach extreme lengths.
Aims: To investigate the possible existence of different types of acute hamstring strains in two specific athletic groups, namely sprinters and dancers, as well as the generality of these findings in other sports.
Methods: In the first project, 18 sprinters and 15 dancers with acute first time hamstring strains were prospectively included. All subjects were examined, clinically and with magnetic resonance imaging (MRI), on 4 occasions after injury: at day 2-4, 10, 21 and 42. The follow-up period was 2 years. In the second project, 30 subjects from 21 different sports were prospectively included. All subjects were examined clinically and with MRI. The follow-up period lasted until the subjects returned to sport or finished their sport activity due to the injury.
Results: All sprinters sustained their injuries during competitive high-speed running. In contrast, all dancers encountered their injuries during slow-speed stretching type of exercises. The initial loss of strength and flexibility was significantly greater in sprinters than in dancers. At 42 days after injury, both groups could perform more than 90% of the test values of the uninjured leg. All the sprinters’ injuries were primarily located in biceps femoris long head, whereas the dancers’ injuries were mainly (87%) involving the proximal free tendon of semimembranosus. For the sprinters, involvement of the proximal free tendon, as estimated by MRI, and proximity to the ischial tuberosity, as estimated both by palpation and MRI, were associated with significantly longer time to return to pre-injury level. In the dancers, there were no significant correlations between clinical or MRI parameters and time to return to per-injury level. The time to pre-injury level was significantly longer (median 50 weeks, range 30-76) for the dancers compared to the sprinters (16, 6-50). In the second project, all injuries occurred during movements reaching a position with combined extensive hip flexion and knee extension. They were all located close to the ischial tuberosity and 83% involved the proximal free tendon of semimembranosus. Fourteen subjects (47%) decided to end their sport activity and for the remaining 16 subjects the median time back to sport was 31 (range 9-104) weeks. There were no significant correlations between clinical and MRI parameters and time to return to sport.
Conclusions: There seems to be a link between the injury situation and the two types of acute hamstring strain in sprinters and dancers with respect to clinical findings, injury location, muscles and tissues involved, and time to return to pre-injury level. Proximity of the injury to the ischial tuberosity, as estimated both by palpation and MRI, is associated with longer recovery time. Also in other sports, an injury situation where the hamstrings reach extensive lengths caused a specific injury to the proximal posterior thigh similar to that described in dancers. Due to the prolonged recovery time associated with this type of injury, correct diagnosis based on history, clinical and MRI investigation, and adequate information to the athletes are essential.
För att beställa tryckta exemplar av avhandlingen kontakta Carl Askling, carl.askling@gih.se
Lewis, Colin A. "Glasbury muscle men". The Ringing World, 2005. http://hdl.handle.net/10962/d1012368.
Pełny tekst źródłaWhitlock, T. L. "Muscle physiology instrumentation". Thesis, University of Bath, 1990. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.236467.
Pełny tekst źródłaWeerakkody, Nivan Sargara. "Measurements of muscle pain, force matching ability and muscle adaptation after eccentric exercise". Monash University, Dept. of Physiology, 2003. http://arrow.monash.edu.au/hdl/1959.1/9561.
Pełny tekst źródłaWu, Cheng Kam-chee Polina. "The effect of mental training in delaying muscle fatigue /". Hong Kong : University of Hong Kong, 2000. http://sunzi.lib.hku.hk:8888/cgi-bin/hkuto%5Ftoc%5Fpdf?B23294991.
Pełny tekst źródłaDias, Wilson Vinicius. "Adaptations du métabolisme musculaire en réponse à l’exercice et à une supplémentation en antioxydants chez des patients atteints de Dystrophie Fascioscapulohumérale". Thesis, Montpellier, 2015. http://www.theses.fr/2015MONTT030.
Pełny tekst źródłaFacioscapulohumeral muscular dystrophy (FSHD), first described in 1885 by Landouzy Dejerine, is the most common inherited skeletal muscle disease of adult life affecting 4000 to 5000 persons in France. Progressive evolution of the disease leads to progressive weakness and atrophy of muscle fibers associated to a wide variability. The pattern of muscle weakness is often asymmetrical and the rate and extent of progression may vary considerably with sudden periods of unexplained rapid disease progression. This muscle disorder is characterized by progressive muscle weakness, beginning with facial muscles and the shoulder girdle, followed by the pelvic girdle and the muscles of the lower extremities. In 10 to 15% of cases, patients need to use a wheelchair. Despite major progress in the understanding of the genetic basis of FSHD, the exact mechanisms that lead to FSHD defects are not completely understood and no curative treatment is available. However, there is growing evidence that oxidative stress may contribute to FSHD pathology. The hypothesis that oxidative stress responses might be specifically altered in FSHD is supported by the deregulation of enzymes involved in oxidative stress.A prospective study realized with FSHD patients and healthy subjects unrevealed the correlation between systemic and muscular oxidative stress and functional muscle defects. Based on these data, we organized a randomized, double-blind, placebo-controlled pilot clinical trial in order to evaluate the effects of 17 weeks antioxidant supplementation in 54 FSHD patients. This clinical trial demonstrates a significant increase in muscle force and quadriceps endurance correlated to a decrease in oxidative stress and an increase in antioxidant defense in FSHD patientsFurthermore, many FSHD features may be caused or exacerbated by perturbations in the production of free radicals or inappropriate response to such stressors. Therefore the first objective was planned to investigate muscle oxygenation patterns during and after a MVCQ by near-infrared diffuse optical spectroscopy (NIRS). The second objective is to evaluate the effect of antioxidant supplementation on quadriceps volumes by IRM and determine the muscle quality using Strength/ Volume ratio of quadriceps muscles and correlate this variables with force and oxidative stress parameters.The major findings of this study show a significant decrease in oxidative capacity during voluntary isometric contraction in quadriceps and demonstrate the effect of supplementation on muscle volume and quality. Indeed, vitamin E, vitamin C, zinc and selenium supplementation improves muscle volume and quality of both quadriceps by enhancing the antioxidant defences and reducing oxidative stress.This increase are associated to increase in strength and decrease in oxidative stress and increase in antioxidant defences. Taken together, we show that oxidative stress plays an important role in FSHD and that an anti-oxidant strategy adapted to the FSHD-specific “oxidative stress” may be a relevant therapeutic approach for these patients. Further analyses of ROS production and sources could contribute to a better understanding of the pathophysiological mechanisms implicated in FSHD
Pasquet, Benjamin. "Etude de la spécificité de la commande motrice et de sa régulation pendant différents types de contractions musculaires". Doctoral thesis, Universite Libre de Bruxelles, 2009. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210280.
Pełny tekst źródłaDoctorat en Sciences de la motricité
info:eu-repo/semantics/nonPublished
Serrancolí, Masferrer Gil. "Optimization and muscle synergy approaches for studying muscle redundancy during walking". Doctoral thesis, Universitat Politècnica de Catalunya, 2015. http://hdl.handle.net/10803/299371.
Pełny tekst źródłaEl cos humà és un sistema multisòlid sobreactuat, ja que cada grau de llibertat pot estar controlat per més d’un múscul. Per resoldre el problema d’indeterminació en el càlcul de les forces musculars, es sol utilitzar un mètode d’optimització. Consisteix en distribuir els moments articulars resultants entre els diferents músculs que actuen a l’articulació, i per tant, estimar la força que aquests realitzen. La reducció de la indeterminació en el càlcul de les forces musculars es pot aconseguir de diferents maneres. Malgrat que l’estratègia que fa servir el sistema nerviós central (SNC) per activar els músculs no es coneix amb exactitud, una de les hipòtesis més utilitzades per solucionar la indeterminació és el fet de considerar que el SNC minimitza una variable fisiològica. El primer estudi presentat en aquesta tesi tractava de resoldre el problema del repartiment muscular minimitzant la suma de les forces musculars normalitzades al quadrat. Per a tal fi, es va utilitzar una funció de cost ponderada per avaluar quins músculs es penalitzen més en la marxa d’un subjecte amb el lligament creuat anterior trencat. Els resultats mostren que la funció de cost que millor aproximava les activacions musculars amb el senyal d’EMG mesurat no tractava tots els músculs per igual. Una altra manera de reduir la indeterminació en el càlcul de les forces muscular és utilitzar la idea que els músculs s’activen sinèrgicament quan l’ésser humà realitza un moviment. En el segon estudi, es presenta una anàlisi de les sinergies musculars que compara la informació de les activacions a dos nivells: en els patrons d’activació-desactivació i en els components de les sinergies musculars d’una mostra de 18 subjectes amb ruptura del lligament creuat i una mostra de 10 subjectes sans. Es van observar diferències als dos nivells, el qual suggereix que els subjectes amb ruptura al lligament creuat alteren les activacions musculars de la seva cama lesionada per tal d’estabilitzar l’articulació lesionada, en aquest cas el genoll. Per últim, en el tercer estudi, es van utilitzar les sinergies musculars junt amb un problema d’optimització de dues etapes per tal de predir les forces musculars i de contacte al genoll de manera fisiològicament consistent, alhora que es calibren els paràmetres musculars. En el nivell exterior de l’optimització, es calibren els paràmetres musculars, mentre que en el nivell interior, es calculen les activacions musculars amb els corresponents paràmetres musculars. Els resultats indiquen que un conjunt de paràmetres musculars pot predir les forces de contacte al genoll amb alta precisió quan es disposa de les forces experimentals de contacte al genoll durant el procés de calibratge. Aquest estudi presenta les diferències entre el cas en què s’utilitzen les forces experimentals de contacte al genoll per calibrar els paràmetres i quan no s’utilitzen. A més, suggereix que si s’utilitzessin captures biomecàniques de moviments on els músculs laterals tinguessin un rol més important que en la marxa, el calibratge dels paràmetres seria més acurat. Per tant, es podrien predir les forces de contacte al genoll amb més precisió quan no es disposa d’aquestes.