Gotowa bibliografia na temat „Mitochondrial pathology”
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Artykuły w czasopismach na temat "Mitochondrial pathology"
Sarnat, Harvey B., i José Marín-García. "Pathology of Mitochondrial Encephalomyopathies". Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 32, nr 2 (maj 2005): 152–66. http://dx.doi.org/10.1017/s0317167100003929.
Pełny tekst źródłaFeng, Baoyi, Chenxi Jin, Zhenzhe Cheng, Xingle Zhao, Zhuoer Sun, Xiaofei Zheng, Xiang Li, Tingting Dong, Yong Tao i Hao Wu. "Mitochondrial Dysfunction and Therapeutic Targets in Auditory Neuropathy". Neural Plasticity 2020 (28.08.2020): 1–10. http://dx.doi.org/10.1155/2020/8843485.
Pełny tekst źródłaPicone, Pasquale, Domenico Nuzzo, Luca Caruana, Valeria Scafidi i Marta Di Carlo. "Mitochondrial Dysfunction: Different Routes to Alzheimer’s Disease Therapy". Oxidative Medicine and Cellular Longevity 2014 (2014): 1–11. http://dx.doi.org/10.1155/2014/780179.
Pełny tekst źródłaNevzorova, V. A., V. M. Chertok, T. A. Brodskaya, P. A. Selyukova i N. V. Zakharchuk. "Mitochondrial dysfunction and vascular aging in comorbid pathology". Pacific Medical Journal, nr 1 (25.03.2022): 10–16. http://dx.doi.org/10.34215/1609-1175-2022-1-10-16.
Pełny tekst źródłaAbramov, Andrey Y., i Plamena R. Angelova. "Cellular mechanisms of complex I-associated pathology". Biochemical Society Transactions 47, nr 6 (26.11.2019): 1963–69. http://dx.doi.org/10.1042/bst20191042.
Pełny tekst źródłaSchumacker, Paul T., Mark N. Gillespie, Kiichi Nakahira, Augustine M. K. Choi, Elliott D. Crouser, Claude A. Piantadosi i Jahar Bhattacharya. "Mitochondria in lung biology and pathology: more than just a powerhouse". American Journal of Physiology-Lung Cellular and Molecular Physiology 306, nr 11 (1.06.2014): L962—L974. http://dx.doi.org/10.1152/ajplung.00073.2014.
Pełny tekst źródłaPatterson, Kathleen. "Mitochondrial Muscle Pathology". Pediatric and Developmental Pathology 7, nr 6 (listopad 2004): 629–32. http://dx.doi.org/10.1007/s10024-004-5051-4.
Pełny tekst źródłaSengers, R. C. A., i A. M. Stadhouders. "Secondary mitochondrial pathology". Journal of Inherited Metabolic Disease 10, S1 (marzec 1987): 98–104. http://dx.doi.org/10.1007/bf01812850.
Pełny tekst źródłaJhun, Bong, Jin O-Uchi, Stephanie Adaniya, Michael Cypress i Yisang Yoon. "Adrenergic Regulation of Drp1-Driven Mitochondrial Fission in Cardiac Physio-Pathology". Antioxidants 7, nr 12 (18.12.2018): 195. http://dx.doi.org/10.3390/antiox7120195.
Pełny tekst źródłaLuna-Sánchez, Marta, Patrizia Bianchi i Albert Quintana. "Mitochondria-Induced Immune Response as a Trigger for Neurodegeneration: A Pathogen from Within". International Journal of Molecular Sciences 22, nr 16 (7.08.2021): 8523. http://dx.doi.org/10.3390/ijms22168523.
Pełny tekst źródłaRozprawy doktorskie na temat "Mitochondrial pathology"
Blaikie, Frances H., i n/a. "Synthesis and characterisation of probes that influence mitochondrial function". University of Otago. Department of Chemistry, 2008. http://adt.otago.ac.nz./public/adt-NZDU20080212.091116.
Pełny tekst źródłaRenken, Christian Wolfgang. "The structure of mitochondria /". Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2004. http://wwwlib.umi.com/cr/ucsd/fullcit?p3141929.
Pełny tekst źródłaJiang, Sirui. "Mitochondrial Dynamic Abnormalities in Alzheimer's Diease". Case Western Reserve University School of Graduate Studies / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1536608714970424.
Pełny tekst źródłaShum, Laura C. "Mitochondrial Metabolism in Bone Physiology and Pathology". Thesis, University of Rochester, 2018. http://pqdtopen.proquest.com/#viewpdf?dispub=10792056.
Pełny tekst źródłaWorldwide, 1 in 3 women and 1 in 5 men over age 50 will experience fractures due to a decline in bone quality. Elucidating the mechanisms for declining bone quality can lead to better therapeutics. A vital, yet overlooked aspect of bone health is the role of mitochondrial metabolism in both bone physiology and pathology. We have found that the ability of stem cells to differentiate into bone forming osteoblasts is sensitive to mitochondrial dysfunction, and therefore preserving mitochondrial function is essential to maintaining bone quality. In human patient samples, we found that osteogenesis following a spinal fusion is correlated with mitochondrial function of bone marrow stem cells. While the decline of bone with aging has been well studied, we were the first to find a concomitant decline in mitochondrial function in bone tissue. The most common mechanism of mitochondrial dysfunction is opening of the mitochondrial permeability transition pore (MPTP), a non-selective proteinaceous pore on the inner mitochondrial membrane, positively regulated by the protein cyclophilin D (CypD). Our CypD knockout mouse model has protected mitochondrial function in bone tissue and no decline in bone quality during aging. While we did show that protecting mitochondrial function is beneficial to age-associated bone loss, our ovariectomy model in the CypD knockout mouse did not show any protection. Thus, age-related and estrogen-related bone loss are likely controlled through different mechanisms. Overall, this work has shown the importance of mitochondrial metabolism in bone health and should be further explored as a new avenue for therapeutic interventions.
Hanson, Bonnie Jean. "Protein based methods for the identification and classification of mitochondrial disorders /". view abstract or download file of text, 2001. http://wwwlib.umi.com/cr/uoregon/fullcit?p3018367.
Pełny tekst źródłaTypescript. Includes vita and abstract. Includes bibliographical references (leaves 96-103). Also available for download via the World Wide Web; free to University of Oregon users.
Oglesbee, Devin. "Improving the diagnosis of mitochondrial diseases : application of monoclonal antibody technologies to NADH:ubiquinone oxidoreductase and cytochrome c oxidase defects /". view abstract or download file of text, 2004. http://wwwlib.umi.com/cr/uoregon/fullcit?p3136436.
Pełny tekst źródłaTypescript. Includes vita and abstract. Includes bibliographical references (leaves 113-119). Also available for download via the World Wide Web; free to University of Oregon users.
Slipetz, Deborah M. "Characterization of mutations in pediatric mitochondrial myopathies". Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=60101.
Pełny tekst źródłaCells from two patients with ETC complex I deficiency, showed reduced oxidation of alanine with normal oxidation of succinate. Analysis of complex I subunits indicated deficient synthesis of the 20 kDa subunit in the severely affected patient. In the milder patient, subunit abnormalities were not detected.
Fibroblasts from a patient with facioscapulohumeral disease (FSHD), showed reduced oxidation of alanine and succinate through the ETC.
A fourth patient, with decreased activity in several complexes in muscle and liver, was found to have a heteroplasmic mtDNA population in fibroblasts.
These studies exemplify the heterogeneity of mitochondrial myopathies and demonstrate the utility of fibroblasts in the investigation of these disorders.
Malik, Safarina Golfiani 1963. "Human disorder of energy transduction : molecular pathology". Monash University, Dept. of Biochemistry and Molecular Biology, 2001. http://arrow.monash.edu.au/hdl/1959.1/8335.
Pełny tekst źródłaTaylor, Robert William. "Mitochondrial respiratory chain dysfunction in human pathology : investigation, pathogenicity and treatment". Thesis, University of Newcastle Upon Tyne, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.577189.
Pełny tekst źródłavan, der Watt George Frederick. "Whole Blood Mitochondrial DNA Depletion in Human Immunodeficiency Virus-Infected Children". Master's thesis, University of Cape Town, 2010. http://hdl.handle.net/11427/2705.
Pełny tekst źródłaKsiążki na temat "Mitochondrial pathology"
Mitochondria. Wyd. 2. Hoboken, N.J: John Wiley & Sons, 2008.
Znajdź pełny tekst źródłaGary, Fiskum, red. Mitochondrial physiology and pathology. New York: Van Nostrand Reinhold, 1986.
Znajdź pełny tekst źródłaN, Gellerich Frank, Zierz S i Colloquium on Mitochondria and Myopathies (1st : 1995 : Halle an der Saale, Germany), red. Detection of mitochondrial diseases. Dordrecht: Kluwer Academic, 1997.
Znajdź pełny tekst źródłaF, Palmieri, red. Thirty years of progress in mitochondrial bioenergetics and molecular biology: Proceedings of the 23rd Bari meeting on bioenergetics, International Symposium on Thirty Years of Progress in Mitochondrial Bioenergetics and Molecular Biology : in honour of Professor E. Quagliariello's 70th birthday, Bari, Italy, 7-10 October 1994. Amsterdam: Elsevier, 1995.
Znajdź pełny tekst źródłaW, Schaffer S., i Suleiman M. -Saadeh, red. Mitochondria: The dynamic organelle. New York: Springer, 2007.
Znajdź pełny tekst źródłaAnna, Gvozdjáková, red. Mitochondrial medicine: Mitochondrial metabolism, diseases, diagnosis and therapy. Dordrecht: Springer, 2008.
Znajdź pełny tekst źródłaMitochondrial signaling in health and disease. Boca Raton: Taylor & Francis/CRC Press, 2012.
Znajdź pełny tekst źródłaBalcells, Cristy. Living well with mitochondrial disease: A handbook for patients, parents, and families. Bethesda, MD: Woodbine House, 2012.
Znajdź pełny tekst źródłaMitochondrial bioenergetics: Methods and protocols. New York: Humana Press, 2012.
Znajdź pełny tekst źródłaMitochondrial dysfunction and oxidativedamage in neurodegenerative diseases. New York: Springer, 1995.
Znajdź pełny tekst źródłaCzęści książek na temat "Mitochondrial pathology"
Buchet, K., i C. Godinot. "ATPase-ATP Synthase and Mitochondrial Pathology". W Mitochondrial Diseases, 129–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59884-5_10.
Pełny tekst źródłaPoyau, A., i C. Godinot. "Cytochrome c Oxidase and Mitochondrial Pathology". W Mitochondrial Diseases, 115–27. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59884-5_9.
Pełny tekst źródłaDuborjal, H., R. Beugnot, V. Procaccio, J. P. Issartel i J. Lunardi. "Structure, Function and Pathology of Complex I". W Mitochondrial Diseases, 73–86. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59884-5_6.
Pełny tekst źródłaLestienne, P., i C. Desnuelle. "Complex II or Succinate: Quinone Oxidoreductase and Pathology". W Mitochondrial Diseases, 87–95. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59884-5_7.
Pełny tekst źródłaBai, Renkui, i Jaimie D. Higgs. "Mitochondrial Disorders". W Molecular Pathology in Clinical Practice, 139–59. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-19674-9_10.
Pełny tekst źródłaLitvak, S., M. Hernould, E. Zabaleta, V. Blanc, D. Begu, I. Kurek, A. Breiman, X. Jordana, A. Mouras i A. Araya. "Plant Cytoplasmic Male Sterility: A Mitochondrial Pathology and Its Biotechnological Application". W Mitochondrial Diseases, 327–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59884-5_25.
Pełny tekst źródłaGenova, Maria Luisa, Milena Merlo Pich, Andrea Bernacchia, Cristina Bianchi, Annalisa Biondi, Carla Bovina, Anna Ida Falasca, Gabriella Formiggini, Giovanna Parenti Castelli i Giorgio Lenaz. "The Mitochondrial Production of Reactive Oxygen Species in Relation to Aging and Pathology". W Mitochondrial Pathogenesis, 86–100. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-662-41088-2_10.
Pełny tekst źródłaJacobs, Howard T. "Mitochondrial ATP Synthase: Structure, Biogenesis and Pathology". W Organellar Proton-ATPases, 103–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22265-2_5.
Pełny tekst źródłaBárcena, Clea, Pablo Mayoral, Pedro M. Quirós i Carlos López-Otín. "Physiological and Pathological Functions of Mitochondrial Proteases". W Proteases in Physiology and Pathology, 3–25. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-2513-6_1.
Pełny tekst źródłaNeginskaya, Maria A., i Evgeny V. Pavlov. "Inorganic Polyphosphate in Mitochondrial Energy Metabolism and Pathology". W Inorganic Polyphosphates, 15–26. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-01237-2_2.
Pełny tekst źródłaStreszczenia konferencji na temat "Mitochondrial pathology"
Hill, Marcus, Mojtaba Fazli, Rachel Mattson, Meekail Zain, Andrew Durden, Allyson Loy, Barbara Reaves i in. "Spectral Analysis of Mitochondrial Dynamics: A Graph-Theoretic Approach to Understanding Subcellular Pathology". W Python in Science Conference. SciPy, 2020. http://dx.doi.org/10.25080/majora-342d178e-00d.
Pełny tekst źródła"Mitochondrial dysfunction and redox balance alterations in the development of AD-like pathology in OXYS rats". W Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-334.
Pełny tekst źródłaSoares, Carolina, Débora G. Souza, Andreia Silva da Rocha, Luiza Machado, Bruna Bellaver i Eduardo R. Zimmer. "BRAIN ENERGETICS EVALUATION IN EARLY STAGES OF AMYLOID PATHOLOGY IN A RAT MODEL OF ALZHEIMER’S DISEASE". W XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda086.
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