Gotowa bibliografia na temat „Mitochondrial DNA”
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Artykuły w czasopismach na temat "Mitochondrial DNA"
Faria, Rúben, Eric Vivés, Prisca Boisguerin, Angela Sousa i Diana Costa. "Development of Peptide-Based Nanoparticles for Mitochondrial Plasmid DNA Delivery". Polymers 13, nr 11 (1.06.2021): 1836. http://dx.doi.org/10.3390/polym13111836.
Pełny tekst źródłaBasu, Urmimala, Alicia M. Bostwick, Kalyan Das, Kristin E. Dittenhafer-Reed i Smita S. Patel. "Structure, mechanism, and regulation of mitochondrial DNA transcription initiation". Journal of Biological Chemistry 295, nr 52 (30.10.2020): 18406–25. http://dx.doi.org/10.1074/jbc.rev120.011202.
Pełny tekst źródłaCampbell, C. L., i P. E. Thorsness. "Escape of mitochondrial DNA to the nucleus in yme1 yeast is mediated by vacuolar-dependent turnover of abnormal mitochondrial compartments". Journal of Cell Science 111, nr 16 (15.08.1998): 2455–64. http://dx.doi.org/10.1242/jcs.111.16.2455.
Pełny tekst źródłaHerrmann, J. M., R. A. Stuart, E. A. Craig i W. Neupert. "Mitochondrial heat shock protein 70, a molecular chaperone for proteins encoded by mitochondrial DNA." Journal of Cell Biology 127, nr 4 (15.11.1994): 893–902. http://dx.doi.org/10.1083/jcb.127.4.893.
Pełny tekst źródłaVarma, V. A., C. M. Cerjan, K. L. Abbott i S. B. Hunter. "Non-isotopic in situ hybridization method for mitochondria in oncocytes." Journal of Histochemistry & Cytochemistry 42, nr 2 (luty 1994): 273–76. http://dx.doi.org/10.1177/42.2.8288868.
Pełny tekst źródłaHabbane, Mouna, Julio Montoya, Taha Rhouda, Yousra Sbaoui, Driss Radallah i Sonia Emperador. "Human Mitochondrial DNA: Particularities and Diseases". Biomedicines 9, nr 10 (1.10.2021): 1364. http://dx.doi.org/10.3390/biomedicines9101364.
Pełny tekst źródłaHong, Seongho, Sanghun Kim, Kyoungmi Kim i Hyunji Lee. "Clinical Approaches for Mitochondrial Diseases". Cells 12, nr 20 (20.10.2023): 2494. http://dx.doi.org/10.3390/cells12202494.
Pełny tekst źródłaWang, Sheng-Fan, Shiuan Chen, Ling-Ming Tseng i Hsin-Chen Lee. "Role of the mitochondrial stress response in human cancer progression". Experimental Biology and Medicine 245, nr 10 (23.04.2020): 861–78. http://dx.doi.org/10.1177/1535370220920558.
Pełny tekst źródłaBradshaw, Patrick C., i David C. Samuels. "A computational model of mitochondrial deoxynucleotide metabolism and DNA replication". American Journal of Physiology-Cell Physiology 288, nr 5 (maj 2005): C989—C1002. http://dx.doi.org/10.1152/ajpcell.00530.2004.
Pełny tekst źródłaBertrand, Helmut. "Senescence is coupled to induction of an oxidative phosphorylation stress response by mitochondrial DNA mutations in Neurospora". Canadian Journal of Botany 73, S1 (31.12.1995): 198–204. http://dx.doi.org/10.1139/b95-246.
Pełny tekst źródłaRozprawy doktorskie na temat "Mitochondrial DNA"
Al, Amir Dache Zahra. "Étude de la structure de l'ADN circulant d'origine mitochondriale". Thesis, Montpellier, 2019. http://www.theses.fr/2019MONTT059.
Pełny tekst źródłaPlasma transports blood cells with a mixture of compounds, including nutrients, waste, antibodies, and chemical messengers...throughout the body. Non-soluble factors such as circulating DNA and extracellular vesicles have recently been added to the list of these components and have been the subject of extensive research due to their role in intercellular communication. Circulating DNA (cirDNA) is composed of cell-free and particle-associated DNA fragments, which can be released by all cell types. cirDNA is derived not only from genomic DNA but also from extrachromosomal mitochondrial DNA. Numerous studies carried out lately indicate that the quantitative and qualitative analysis of cirDNA represents a breakthrough in clinical applications as a non-invasive biomarker for diagnosis, prognosis and therapeutic follow-up. However, despite the promising future of cirDNA in clinical applications, particularly in oncology, knowledge regarding its origins, composition and functions, that could considerably optimize its diagnostic value, is still lacking.The main goal of my thesis was to identify and characterize the structural properties of extracellular DNA of mitochondrial origin. By examining the integrity of this DNA, as well as the size and density of associated structures, this work revealed the presence of dense particles larger than 0.2 µm containing whole mitochondrial genomes. We characterized these structures by electron microscopy and flow cytometry and identified intact mitochondria in the extracellular medium in vitro and ex vivo (in plasma samples from healthy individuals). Oxygen consumption by these mitochondria was detected by the Seahorse technology, suggesting that at least some of these intact extracellular mitochondria may be functional.In addition, I contributed to other studies carried out in the team, such as studies aiming at evaluating (1) the influence of pre-analytical and demographic parameters on the quantification of nuclear and mitochondrial cirDNA on a cohort of 104 healthy individuals and 118 patients with metastatic colorectal cancer, (2) the influence of hypoxia on the release of cirDNA in vitro and in vivo, and (3) the potential of cirDNA analysis in the early detection and screening of cancer.This manuscript present a recent review on cirDNA and its different mechanisms of release, which go hand in hand with the structural characterization of this DNA, its functional aspects and its clinical applications. In addition, this thesis provides new knowledge on the structure of extracellular mitochondrial DNA and opens up new avenues for reflection, particularly on the potential impact that could have those circulating mitochondria on cell-cell communication, inflammation and clinical applications
Berg, Alonso Laetitia. "Déficits de la chaîne respiratoire mitochondriale avec instabilité de l’ADN mitochondrial : identification de nouveaux gènes et mécanismes". Thesis, Université Côte d'Azur (ComUE), 2016. http://www.theses.fr/2016AZUR4101/document.
Pełny tekst źródłaNon communiqué
Rebelo, Adriana. "Probing Mitochondrial DNA Structure with Mitochondria-Targeted DNA Methyltransferases". Scholarly Repository, 2009. http://scholarlyrepository.miami.edu/oa_dissertations/344.
Pełny tekst źródłaBoyer, Hélène. "The mamalian circadian clock regulates the abundance and expression of mitochondrial DNA in the nuclear compartment". Thesis, Lyon, 2020. http://www.theses.fr/2020LYSEN015.
Pełny tekst źródłaThe mitochondrial genome is minimal and most of the mitochondrial proteins are encoded in the nuclear genome. Thus, although mitochondrial and nuclear genomes are physically separated in the cell, anterograde (nuclear to mitochondrial) and retrograde (mitochondrial to nuclear) signals are essential for mitochondrial biogenesis to be coordinated with the cellular energetic demands. Those demands are cyclical in nature, and the circadian clock regulates numerous aspects of mitochondrial biology, including the dynamics of fusion and fission that shape the architecture of the mitochondrial network. In murine livers, the network oscillates between fused (during the day) and fragmented structures (during the night). A fused network is associated with a more efficient ATP production whereas fragmentation is associated with elevated mitochondrial ROS levels and mitophagy. In other words, if mtDNA was to ever escape mitochondria, fission would help. Complementation experiments in yeast have shown that mitochondrial DNA (mtDNA) is able to escape from the mitochondria and enter the nucleus. In human cells (HeLa), the intact and full-length mitochondrial genome has been detected in the nucleus. Evolutionary analyses of nuclear inserted mitochondrial sequences (numts) suggest an ongoing process of integration of mitochondrial sequences into the nuclear genome. Also, abundant somatically acquired mitochondrial- nuclear genome fusion events (simts) have been shown to occur in human cancer cells - an extreme context of genomic instability and disrupted circadian rhythms. The availability of mtDNA in the cytoplasm, protected by vesicles, to be taken up by the nucleus is thought to result from mitophagy. As mitophagy and mitochondrial dynamics are regulated by the circadian clock, we investigated whether mtDNA would accumulate in the nuclear compartment as a function of circadian time. We addressed this question in the mouse liver, a differentiate mammalian tissue. This work demonstrates that the nuclear abundance of mtDNA in murine livers is regulated by the circadian clock – with a zenith at the end of the circadian night. Nuclear mtDNA is differentially hydroxymethylated relative to the total mtDNA extracted from the same tissue. Also, circadian clock disruption altered the phase and abundance of nuclear mtDNA. Additionally, we observed that concurrent accumulation of nuclear mtRNA was sensitive to nutritional challenges. Probably, these dynamics are driven by mitochondrial network remodeling dynamics. Increased nuclear presence and insertions of mtDNA in cancer cells or aging tissues, which are often associated with disrupted circadian oscillators- may thus arise from the loss of a physiological rhythm in mitochondrial-network remodeling
Korhonen, Jenny. "Functional and structural characterization of the human mitochondrial helicase /". Stockholm : Karolinska institutet, 2007. http://diss.kib.ki.se/2007/978-91-7357-102-2/.
Pełny tekst źródłaBerg, Alonso Laetitia. "Déficits de la chaîne respiratoire mitochondriale avec instabilité de l’ADN mitochondrial : identification de nouveaux gènes et mécanismes". Electronic Thesis or Diss., Université Côte d'Azur (ComUE), 2016. http://www.theses.fr/2016AZUR4101.
Pełny tekst źródłaNon communiqué
Weber, Katharina Karin. "Studies of mitochondrial DNA". Thesis, University of Newcastle Upon Tyne, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295072.
Pełny tekst źródłaMyers, K. A. "Alkylation of mitochondrial DNA". Thesis, University of Manchester, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234216.
Pełny tekst źródłaJohansson, Jennie. "Epigenetic Regulation of Mitochondrial DNA". Thesis, Linköpings universitet, Institutionen för fysik, kemi och biologi, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-166684.
Pełny tekst źródłaWertzler, Kelsey Janel. "High mobility group A1 and mitochondrial transcription factor A compete for binding to mitochondrial DNA". Pullman, Wash. : Washington State University, 2009. http://www.dissertations.wsu.edu/Thesis/Summer2009/k_wertzler_051409.pdf.
Pełny tekst źródłaTitle from PDF title page (viewed on July 21, 2009). "School of Molecular Biosciences." Includes bibliographical references.
Książki na temat "Mitochondrial DNA"
Copeland, William C. Mitochondrial DNA. New Jersey: Humana Press, 2002. http://dx.doi.org/10.1385/1592592848.
Pełny tekst źródłaStuart, Jeffrey A., red. Mitochondrial DNA. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-521-3.
Pełny tekst źródłaMcKenzie, Matthew, red. Mitochondrial DNA. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3040-1.
Pełny tekst źródłaNicholls, Thomas J., Jay P. Uhler i Maria Falkenberg, red. Mitochondrial DNA. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2922-2.
Pełny tekst źródłaJohn, Justin C. St. Mitochondrial DNA, mitochondria, disease, and stem cells. New York: Humana Press, 2013.
Znajdź pełny tekst źródłaSt. John, Justin C., red. Mitochondrial DNA, Mitochondria, Disease and Stem Cells. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-101-1.
Pełny tekst źródłaS, DiMauro, i Wallace Douglas C, red. Mitochondrial DNA in human pathology. New York: Raven Press, 1993.
Znajdź pełny tekst źródłaJames, Holt Ian, red. Genetics of mitochondrial diseases. Oxford: Oxford University Press, 2003.
Znajdź pełny tekst źródłaSun, Hongzhi, i Xiangdong Wang, red. Mitochondrial DNA and Diseases. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-6674-0.
Pełny tekst źródłaA, Dudareva N., i Salganik, R. I. (Rudolʹf Iosifovich), red. Mitokhondrialʹnyĭ genom. Novosibirsk: "Nauka," Sibirskoe otd-nie, 1990.
Znajdź pełny tekst źródłaCzęści książek na temat "Mitochondrial DNA"
Mainieri, Avantika. "Mitochondrial DNA". W Encyclopedia of Evolutionary Psychological Science, 1–4. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-16999-6_2229-1.
Pełny tekst źródłaVerma, Mukesh, i Deepak Kumar. "Mitochondrial DNA". W Encyclopedia of Cancer, 1–4. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_3765-2.
Pełny tekst źródłaVerma, Mukesh, i Deepak Kumar. "Mitochondrial DNA". W Encyclopedia of Cancer, 2867–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46875-3_3765.
Pełny tekst źródłaMishra, Alaknanda. "Mitochondrial DNA". W Encyclopedia of Animal Cognition and Behavior, 4329–32. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-319-55065-7_162.
Pełny tekst źródłaVerma, Mukesh, i Deepak Kumar. "Mitochondrial DNA". W Encyclopedia of Cancer, 2331–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_3765.
Pełny tekst źródłaMishra, Alaknanda. "Mitochondrial DNA". W Encyclopedia of Animal Cognition and Behavior, 1–4. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-47829-6_162-1.
Pełny tekst źródłaMainieri, Avantika. "Mitochondrial DNA". W Encyclopedia of Evolutionary Psychological Science, 5150–52. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-319-19650-3_2229.
Pełny tekst źródłaGojobori, Jun. "Mitochondrial DNA". W Evolution of the Human Genome II, 103–20. Tokyo: Springer Japan, 2021. http://dx.doi.org/10.1007/978-4-431-56904-6_4.
Pełny tekst źródłaReynier, P., Y. Malthièry i P. Lestienne. "Mitochondrial DNA Analysis". W Mitochondrial Diseases, 379–87. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59884-5_28.
Pełny tekst źródłaCasane, D., i M. Guéride. "Mitochondrial DNA Inheritance in Mammals". W Mitochondrial Diseases, 17–31. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-59884-5_3.
Pełny tekst źródłaStreszczenia konferencji na temat "Mitochondrial DNA"
Joseph Mathuram, T. L., Y. Su, M. Hatzoglou, Y. Perry, Y. Wu i A. Blumental-Perry. "Mitochondria-to-Nucleus Retrograde Signaling Via Mitochondrial DNA Encoded Non-coding RNA Regulates Mitochondrial Bioenergetics". W American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a4400.
Pełny tekst źródłaCristea, Paul Dan, i Rodica Tuduce. "Mitochondrial DNA Analysis Using Genomic Signals". W 2009 16th International Conference on Systems, Signals and Image Processing. IEEE, 2009. http://dx.doi.org/10.1109/iwssip.2009.5367711.
Pełny tekst źródłaNesbitt, V., i R. McFarland. "G259 Mitochondrial DNA disease in children". W Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference and exhibition, 13–15 May 2019, ICC, Birmingham, Paediatrics: pathways to a brighter future. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-rcpch.251.
Pełny tekst źródłaXu, W., R. Chen, B. Hu, J. G. Zein, C. Liu, S. A. A. Comhair, M. A. Aldred i in. "Mitochondrial DNA Variation and Severe Asthma". W American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2961.
Pełny tekst źródłaMelamud, M. M., E. A. Ermakov, P. I. Brit, E. S. Zhuravlev, E. A. Balakhonova, G. A. Stepanov, D. A. Kamaeva, S. A. Ivanova, G. A. Nevinsky i V. N. Buneva. "ASSOCIATION BETWEEN HIGH CONCENTRATIONS OF CIRCULATING CELL-FREE DNA AND SUICIDE ATTEMPTS IN SCHIZOPHRENIA". W X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-347.
Pełny tekst źródłaWan, Emily S., Michael H. Cho, Nadia Boutaoui, Barbara J. Klanderman, Jody S. Sylvia, John P. Ziniti, Augusto A. Litonjua i in. "Mitochondrial DNA Polymorphisms Are Associated With COPD". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2921.
Pełny tekst źródłaAggarwal, S., I. Ahmad, S. Gu, H. Paiste, M. N. Gillespie i S. Matalon. "Mitochondrial DNA Repair Ameliorates Inhalation Lung Injury". W American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1020.
Pełny tekst źródłavan den Heuvel, Robert. "Mitochondrial DNA levels predict COVID-19 severity". W ATS 2023 International Conference, redaktor Rachel Giles. Baarn, the Netherlands: Medicom Medical Publishers, 2023. http://dx.doi.org/10.55788/e3a1fb1a.
Pełny tekst źródłaCristea, Paul Dan, i Rodica Tuduce. "Nucleotide Genomic Signal analysis of hominidae mitochondrial DNA". W 2009 16th International Conference on Digital Signal Processing (DSP). IEEE, 2009. http://dx.doi.org/10.1109/icdsp.2009.5201251.
Pełny tekst źródłaKulvinder Singh Mann i Navjot Kaur. "Mitochondrial DNA for Bio-molecular Archaeology of mummies". W 2015 IEEE International Conference on Electrical, Computer and Communication Technologies (ICECCT). IEEE, 2015. http://dx.doi.org/10.1109/icecct.2015.7226105.
Pełny tekst źródłaRaporty organizacyjne na temat "Mitochondrial DNA"
Friddle, R. W., J. E. Klare, A. Noy, M. Corzett, R. Balhorn, R. J. Baskin, S. S. Martin i E. P. Baldwin. DNA Compaction by Yeast Mitochondrial Protein ABF2p. Office of Scientific and Technical Information (OSTI), maj 2003. http://dx.doi.org/10.2172/15007313.
Pełny tekst źródłaMathews, Christopher K. DNA Precursor Metabolism and Mitochondrial Genome Stability. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2003. http://dx.doi.org/10.21236/ada460347.
Pełny tekst źródłaSAlly A. Mackenzie. Proteomic Dissection of the Mitochondrial DNA Metabolism Apparatus in Arabidopsis. Office of Scientific and Technical Information (OSTI), styczeń 2004. http://dx.doi.org/10.2172/835670.
Pełny tekst źródłaHsieh, Jer-Tsong. Suppression of BRCA2 by Mutant Mitochondrial DNA in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, luty 2012. http://dx.doi.org/10.21236/ada564267.
Pełny tekst źródłaHsieh, Jer-Tsong. Suppression of BRCA2 by Mutant Mitochondrial DNA in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, maj 2013. http://dx.doi.org/10.21236/ada585765.
Pełny tekst źródłaHsieh, Jer-Tsong. Suppression of BRCA2 by Mutant Mitochondrial DNA in Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, maj 2011. http://dx.doi.org/10.21236/ada549344.
Pełny tekst źródłaIzhar, Shamay, i Maureen Hanson. Expression of Mitochondrial DNA Associated with Cytoplasmic Male Sterility in Petunia. United States Department of Agriculture, lipiec 1987. http://dx.doi.org/10.32747/1987.7566866.bard.
Pełny tekst źródłaIzhar, Shamay, Maureen Hanson i Nurit Firon. Expression of the Mitochondrial Locus Associated with Cytoplasmic Male Sterility in Petunia. United States Department of Agriculture, luty 1996. http://dx.doi.org/10.32747/1996.7604933.bard.
Pełny tekst źródłaStevens, Tracy. Analysis of mitochondrial DNA restriction fragment patterns in killer whales, Orcinus orca. Portland State University Library, styczeń 2000. http://dx.doi.org/10.15760/etd.5812.
Pełny tekst źródłaHaddad, Bassem R. Detection of Mitochondrial DNA Mutations in Mammary Epithelial Cells in Nipple Aspirate Fluid. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2004. http://dx.doi.org/10.21236/ada434094.
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