Gotowe bibliografie tematyczne / MiR-29c

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  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Streszczenia konferencji

Gotowa bibliografia na temat „MiR-29c”

Autor: Grafiati
Data publikacji: 6 września 2023

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Artykuły w czasopismach na temat "MiR-29c"

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Huang, Yu-Qing, Jie Li, Cheng Huang, and Ying-Qing Feng. "Plasma MicroRNA-29c Levels Are Associated with Carotid Intima-Media Thickness and is a Potential Biomarker for the Early Detection of Atherosclerosis." Cellular Physiology and Biochemistry 50, no. 2 (2018): 452–59. http://dx.doi.org/10.1159/000494158.

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Background/Aims: Atherosclerosis is a serious disease that increases the risk of myocardial infarction and ischemic stroke. Previous studies have demonstrated that microRNA (miR)-29c could play significant roles in atherosclerosis via regulating inflammatory processes. However, the relationship between miR-29c and carotid intima-media thickness (CIMT) remains unknown. This study investigated associations between miR-29c and atherosclerosis and tested whether plasma miR-29c levels could be used to detect atherosclerosis. Methods: Plasma miR-29c levels were estimated by quantitative real-time PC
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Liu, Jiazheng, Guilu Tao, Cundi Zhong, and Xiao Liu. "Upregulation of miR-29c-3p Hinders Melanoma Progression by Inhibiting CDCA4 Expression." BioMed Research International 2021 (August 28, 2021): 1–15. http://dx.doi.org/10.1155/2021/7065963.

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Objective. To investigate the expression and regulation mechanism of miR-29c-3p and cell division cycle associated 4 (CDCA4) in melanoma (MM). Data and Methods. Fifty-nine patients with MM admitted to our hospital were enrolled as the MM group. They were followed up for 3 years to analyze the prognostic factors; meanwhile, 51 healthy subjects were allocated into a normal group. MM cell lines (M21 and C8161) were transfected with miR-29c-3p-mimics, miR-29c-3p-inhibitor, miR-NC, si-CDCA4, and sh-CDCA4. The expression of miR-29c-3p, CDCA4, Bax, Caspase3, Bcl-2, N-cadherin, vimentin, and E-cadheri
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Cao, Yanqun, Xiangxiang Tan, Quzhe Lu, Kai Huang, Xiaoer Tang, and Zhiming He. "MiR-29c-3p May Promote the Progression of Alzheimer’s Disease through BACE1." Journal of Healthcare Engineering 2021 (December 15, 2021): 1–11. http://dx.doi.org/10.1155/2021/2031407.

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The aim of this study was to explore the specific role of miR-29c-3p in Alzheimer’s disease (AD). Animal models of AD were established by injecting streptozotocin (STZ) into mice through the lateral ventricle, while cell models of AD were induced by 10 μM β-amyloid (Aβ). We detected miR-29c-3p and β-site amyloid precursor protein cleaving enzyme 1 (BACE1) contents and measured AD cell proliferation and apoptosis. A low miR-29c-3p level and a high BACE1 level were detected in the brain tissue of AD animal models and AD cell models. Aβ-processed cells had markedly lower proliferation activity, h
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Wang, Shaoqiang, Pengfei Yi, Na Wang, Min Song, Wenhui Li, and Yingying Zheng. "LncRNA TUG1/miR-29c-3p/SIRT1 axis regulates endoplasmic reticulum stress-mediated renal epithelial cells injury in diabetic nephropathy model in vitro." PLOS ONE 16, no. 6 (2021): e0252761. http://dx.doi.org/10.1371/journal.pone.0252761.

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Long non-coding RNAs (lncRNAs) are important regulators in diabetic nephropathy. In this study, we investigated the potential role of lncRNA TUG1 in regulating endoplasmic reticulum stress (ERS)-mediated apoptosis in high glucose induced renal tubular epithelial cells. Human renal tubular epithelial cell line HK-2 was challenged with high glucose following transfection with lncRNA TUG1, miR-29c-3p mimics or inhibitor expression plasmid, either alone or in combination, for different experimental purposes. Potential binding effects between TUG1 and miR-29c-3p, as well as between miR-29c-3p and S
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Dai, Qijun, Jian Sun, Tianyi Dai, Qin Xu, and Yueqin Ding. "miR-29c-5p knockdown reduces inflammation and blood–brain barrier disruption by upregulating LRP6." Open Medicine 17, no. 1 (2022): 353–64. http://dx.doi.org/10.1515/med-2022-0438.

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Abstract Blood–brain barrier participates in the pathological process of ischemic stroke. MicroRNA-29c-5p was highly expressed in clinical samples from patients with ischemic stroke. In this study, oxygen-glucose deprivation (OGD) treatment of astrocytes enhanced the permeability of brain microvascular endothelial cells (BMECs), and the miR-29c-5p expression was elevated in clinical samples from patients with ischemic stroke. For the function of miR-29c-5p in ischemic stroke, the miR-29c-5p knockdown decreased the permeability and the tight junction protein (TJP) destruction of BMECs and ameli
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Wang, X., K. Xu, XY Yang, J. Liu, Q. Zeng та FS Wang. "Upregulated miR-29c suppresses silica-induced lung fibrosis through the Wnt/β-catenin pathway in mice". Human & Experimental Toxicology 37, № 9 (2017): 944–52. http://dx.doi.org/10.1177/0960327117741750.

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Silicosis is an irreversible lung disease resulting from long-term inhalation of occupational dust containing silicon dioxide. However, the pathogenesis of silicosis has not been clearly understood yet. Accumulating evidence suggests that miR-29 may have a significant anti-fibrotic capacity, meanwhile it may relate to Wnt/β-catenin pathway. The purpose of this study was to discuss the role of miR-29 in the progression of silicosis. A lentiviral vector was constructed, named Lv-miR-29c, which was overexpressing miR-29c. In vivo, intratracheal treatment with Lv-miR-29c significantly increased ex
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Fang, Yi, Xiaofang Yu, Yong Liu та ін. "miR-29c is downregulated in renal interstitial fibrosis in humans and rats and restored by HIF-α activation". American Journal of Physiology-Renal Physiology 304, № 10 (2013): F1274—F1282. http://dx.doi.org/10.1152/ajprenal.00287.2012.

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Treatment with l-mimosine, which activates hypoxia-inducible factor-α (HIF-α), attenuates renal tubulointerstitial injury and improves renal function in a rat remnant kidney model. The miR-29 family of microRNAs directly targets a large number of extracellular matrix genes and reduces renal interstitial fibrosis. We analyzed microRNA expression profiles in rat remnant kidneys with or without treatment with l-mimosine. The expression of miR-29c was downregulated in rat remnant kidneys compared with sham control and significantly restored by the l-mimosine treatment. In cultured human kidney epi
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Chuang, Tsai-Der, William J. Pearce, and Omid Khorram. "miR-29c induction contributes to downregulation of vascular extracellular matrix proteins by glucocorticoids." American Journal of Physiology-Cell Physiology 309, no. 2 (2015): C117—C125. http://dx.doi.org/10.1152/ajpcell.00254.2014.

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Maternal undernutrition increases maternal glucocorticoids (GCs) and alters microRNA expression in offspring. Given that the mechanisms of GC action on vascular development are not clear, this study examined the influence of GCs on microRNA 29c (miR-29c) and its predicted targets in primary rat aorta smooth muscle cells (RAOSMCs). Dexamethasone (Dex) and corticosterone (Cor) time-dependently increased miR-29c expression and reduced collagen type III (Col3A1), collagen type IV (Col4A5), elastin (ELN), and matrix metalloproteinase-2 (MMP2) protein in RAOSMCs. These effects were blocked by mifepr
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Batliner, Jasmin, Mathias Jenal, Martin F. Fey, and Mario P. Tschan. "Mir-29c and Mir-424 Are Novel Myeloid Differentiation-Associated MicroRNAs in Acute Promyelocytic Leukemia." Blood 112, no. 11 (2008): 3346. http://dx.doi.org/10.1182/blood.v112.11.3346.3346.

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Abstract MicroRNAs (miRNAs) are a class of small non-coding RNAs that regulate gene expression at the post-transcriptional level. Recent studies showed that they are critically involved in hematopoietic differentiation and function by a coordinating multi-target repression of hematopoiesis-related genes. To identify miRNAs involved in the pathogenesis of acute promyelocytic leukemia (APL), characterized by the t(15;17) translocation, we performed TaqMan Low Density Array-based miRNA expression profiling on blast cells from an APL patient under all-trans retinoic acid (ATRA) treatment. Although
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Huang, Limin, Chaoquan Hu, Hui Cao, et al. "MicroRNA-29c Increases the Chemosensitivity of Pancreatic Cancer Cells by Inhibiting USP22 Mediated Autophagy." Cellular Physiology and Biochemistry 47, no. 2 (2018): 747–58. http://dx.doi.org/10.1159/000490027.

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Background/Aims: Pancreatic cancer (PC) is an aggressive malignancy with a poor survival rate. Despite advances in the treatment of PC, the efficacy of therapy is limited by the development of chemoresistance. Here, we examined the role of microRNA-29c (miR-29c) and the involvement of autophagy and apoptosis in the chemoresistance of PC cells in vivo and in vitro. Methods: We employed qRT-PCR, western blot and immunofluorescence to examine the expression level of miR-29c, USP22 and autophagy relative protein. In addition, we used MTT assay to detect cell proliferation and transwell assay to me
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Rozprawy doktorskie na temat "MiR-29c"

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Gersmann, Ann-Kathrin [Verfasser], and de Fend Leticia [Akademischer Betreuer] Quintanilla-Martinez. "Untersuchung der direkten Regulation der miR-29c- und miR-146a-abhängig exprimierten Zielgene mittels Reportergensassay im ALK-positiven ALCL / Ann-Kathrin Gersmann ; Betreuer: Leticia Quintanilla-Martinez de Fend." Tübingen : Universitätsbibliothek Tübingen, 2017. http://d-nb.info/1199465267/34.

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Streszczenia konferencji na temat "MiR-29c"

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Poli, Elizabeth Carla, Rachel Tolbert, Jing Zhang, Yoo-Jeong Han, and Olufunmilayo I. Olopade. "Abstract 3559A: Molecular subtype-specific methylation of the miR-29c promoter in breast cancer correlates with basal-like pattern of gene expression." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3559a.

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Lien, Ming-Yu, and Chih-Hsin Tang. "Abstract 200: The MCP-1/CCR2 axis enhances VEGF-A-dependent angiogenesis by directly downregulating miR-29c expression in oral squamous cell carcinoma." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-200.

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Lien, Ming-Yu, and Chih-Hsin Tang. "Abstract 200: The MCP-1/CCR2 axis enhances VEGF-A-dependent angiogenesis by directly downregulating miR-29c expression in oral squamous cell carcinoma." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-200.

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