Gotowa bibliografia na temat „MicroRNA”
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Artykuły w czasopismach na temat "MicroRNA"
Varga, Zoltán V., Ágnes Zvara, Nóra Faragó, Gabriella F. Kocsis, Márton Pipicz, Renáta Gáspár, Péter Bencsik i in. "MicroRNAs associated with ischemia-reperfusion injury and cardioprotection by ischemic pre- and postconditioning: protectomiRs". American Journal of Physiology-Heart and Circulatory Physiology 307, nr 2 (15.07.2014): H216—H227. http://dx.doi.org/10.1152/ajpheart.00812.2013.
Pełny tekst źródłaLiu, Zhong, Yong-Hua Tuo, Jian-Wen Chen, Qing-Yuan Wang, Songlin Li, Ming-Chang Li, Gang Dai i in. "NADPH oxidase inhibitor regulates microRNAs with improved outcome after mechanical reperfusion". Journal of NeuroInterventional Surgery 9, nr 7 (20.06.2016): 702–6. http://dx.doi.org/10.1136/neurintsurg-2016-012463.
Pełny tekst źródłaZinovyeva, Anna Y., Isana Veksler-Lublinsky, Ajay A. Vashisht, James A. Wohlschlegel i Victor R. Ambros. "Caenorhabditis elegans ALG-1 antimorphic mutations uncover functions for Argonaute in microRNA guide strand selection and passenger strand disposal". Proceedings of the National Academy of Sciences 112, nr 38 (8.09.2015): E5271—E5280. http://dx.doi.org/10.1073/pnas.1506576112.
Pełny tekst źródłaJongen-Lavrencic, Mojca, Su Ming Sun, Menno K. Dijkstra, Peter J. M. Valk i Bob Löwenberg. "MicroRNA expression profiling in relation to the genetic heterogeneity of acute myeloid leukemia". Blood 111, nr 10 (15.05.2008): 5078–85. http://dx.doi.org/10.1182/blood-2008-01-133355.
Pełny tekst źródłaMarcucci, Guido, Krzysztof Mrózek, Michael D. Radmacher, Ramiro Garzon i Clara D. Bloomfield. "The prognostic and functional role of microRNAs in acute myeloid leukemia". Blood 117, nr 4 (27.01.2011): 1121–29. http://dx.doi.org/10.1182/blood-2010-09-191312.
Pełny tekst źródłaKiseleva, Y. Y., K. G. Ptitsyn, S. P. Radko, V. G. Zgoda i A. I. Archakov. "Digital droplet PCR - a prospective technological approach to quantitative profiling of microRNA". Biomeditsinskaya Khimiya 62, nr 4 (2016): 403–10. http://dx.doi.org/10.18097/pbmc20166204403.
Pełny tekst źródłaLi, Li-Jie, Wei-Min Chang i Michael Hsiao. "Aberrant Expression of microRNA Clusters in Head and Neck Cancer Development and Progression: Current and Future Translational Impacts". Pharmaceuticals 14, nr 3 (27.02.2021): 194. http://dx.doi.org/10.3390/ph14030194.
Pełny tekst źródłaQin, Li-Xuan. "An Integrative Analysis of microRNA and mRNA Expression–-A Case Study". Cancer Informatics 6 (styczeń 2008): CIN.S633. http://dx.doi.org/10.4137/cin.s633.
Pełny tekst źródłaKarkhane, Maryam, Hamed Esmaeil Lashgarian, Maryam Hormozi, Shirzad Fallahi, Kourosh Cheraghipour i Abdolrazagh Marzban. "Oncogenesis and Tumor Inhibition by MicroRNAs and its Potential Therapeutic Applications: A Systematic Review". MicroRNA 9, nr 3 (13.04.2020): 198–215. http://dx.doi.org/10.2174/2211536608666191104103834.
Pełny tekst źródłaZhang, Xiaomin, Gohar Azhar, Emmanuel D. Williams, Steven C. Rogers i Jeanne Y. Wei. "MicroRNA Clusters in the Adult Mouse Heart: Age-Associated Changes". BioMed Research International 2015 (2015): 1–12. http://dx.doi.org/10.1155/2015/732397.
Pełny tekst źródłaRozprawy doktorskie na temat "MicroRNA"
Khoshnaw, Sarkawt Majeed Omar. "Significance of microRNAs and microRNA maturation regulators in breast cancer". Thesis, University of Nottingham, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.594865.
Pełny tekst źródłaArseni, Varvara. "MicroRNAs and the canonical microRNA biogenesis pathway in the planarian Schmidtea mediterranea". Thesis, University of Nottingham, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.581999.
Pełny tekst źródłaSætrom, Ola. "Predicting MicroRNA targets". Thesis, Norwegian University of Science and Technology, Department of Computer and Information Science, 2005. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-9266.
Pełny tekst źródłaMicroRNAs are a large family of short non-encoding RNAs that regulated protein production by binding to mRNAs. A single miRNA can regulate an mRNA by itself, or several miRNAs can cooperate in regulating the mRNAs. This is all dependent on the degree of complementarity between the miRNA and the target mRNA. Here, we present the program TargetBoost that, using a classifier generated by a combination of hardware accelerated genetic programming and boosting, allows for screening several large dataset against several miRNAs, and computes a likelihood of that genes in the dataset is regulated by the set of miRNAs used in the screening. We also present results from comparison of several different scoring functions for measuring cooperative effects. We found that the classifier used in TargetBoost is best for finding target sites that regulate mRNAs by themselves. A demo of TargetBoost can be found on http://www.interagon.com/demo.
BERTOLAZZI, Giorgio. "MicroRNA Interaction Networks". Doctoral thesis, Università degli Studi di Palermo, 2021. http://hdl.handle.net/10447/498927.
Pełny tekst źródłaBertolazzi’s thesis focuses on developing and applying computational methods to predict microRNA binding sites located on messenger RNA molecules. MicroRNAs (miRNAs) regulate gene expression by binding target messenger RNA molecules (mRNAs). Therefore, the prediction of miRNA binding is important to investigate cellular processes. Moreover, alterations in miRNA activity have been associated with many human diseases, such as cancer. The thesis explores miRNA binding behavior and highlights fundamental information for miRNA target prediction. In particular, a machine learning approach is used to upgrade an existing target prediction algorithm named ComiR; the original version of ComiR considers miRNA binding sites located on mRNA 3’UTR region. The novel algorithm significantly improves the ComiR prediction capacity by including miRNA binding sites located on mRNA coding regions.
Ammari, Meryem. "Rôle de miR-146a dans la régulation des fonctions monocytaires dans l’arthrite". Thesis, Montpellier 1, 2014. http://www.theses.fr/2014MON1T020.
Pełny tekst źródłaIntroduction : Monocytes represent a prototypic cell type when investigating the interplay between immune and skeletal systems as they can give rise to different cell types including dendritic cells, macrophages and osteoclasts (OC), which play key roles in immunity and bone homeostasis. Circulating monocytes consist of at least two main functional subsets, Ly6Chigh and Ly6Clow monocytes. It has been suggested that OC might develop preferentially from the Ly6Chigh monocyte subset, which excessive and prolonged activation is a hallmark of many inflammatory diseases. Among key molecular rheostats of cell fate, micro(mi)RNAs are a class of regulatory RNAs that control basic biological functions and orchestrate inflammatory responses. Few miRNAs have been involved in osteoclastogenesis. The present study aimed at investigating the role of miRNAs in osteoclastogenesis in the context of monocyte subsets, under steady state and inflammatory conditions. Methods & Results : Using genome-wide miRNA expression study we have identified miRNAs and putative targeted pathways that are differentially expressed between Ly6Chigh and LyC6low FACS sorted mouse monocytes, and common to their human counter parts CD14+CD16- and CD14dimCD16+ monocytes, respectively. Among these, miR-146a showed higher expression in Ly6Clow monocytes when compared to Ly6Chigh monocytes. Under inflammatory arthritis conditions, expression of miR-146a in Ly6Chigh monocytes was down regulated as compared to healthy controls. Using mouse deficient for miR-146a, we showed that knockdown of miR-146a increased OC differentiation in vitro. While no bone phenotype was evidenced in miR-146a deficient mice, nor under steady state or ovariectomized conditions, arthritis-induced bone resorption and bone loss were increased in miR-146a knockout mice. Finally, using a liposomal formulation able to delivermiR-146a mimics to Ly6Chigh monocytes upon intravenous injection, we showed that enforced expression of miR-146a led to decreased number of TRAP positive cells within the synovium of arthritic mice, and efficiently reduced bone erosion in inflammatory arthritis. This effect was associated with decreased RelB expression in miR-146a-overexpressing Ly6Chigh osteoclast progenitors. Conclusion : Overall, our results show that specific over-expression of miR-146a in Ly6Chigh monocytes altered OC differentiation and decreased bone erosion in inflammatory arthritis. These data suggest a novel role for miR-146a in controlling osteoclast fate of Ly6Chigh monocyte progenitors and that reduced miR-146a expression in Ly6Chigh monocytes under arthritic conditions contributes to pathogenic bone loss. Finally, delivery of miR-146a mimics to Ly6Chigh monocytes may offer valuable therapeutic potential to interfere with pathological bone loss
Dogini, Danyella Barbosa. "Quantificação de diferentes microRNAs no sistema nervoso central = implicações nos mecanismos de desenvolvimento e processos fisiopatologicos". [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/309739.
Pełny tekst źródłaTese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas
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Resumo: MicroRNAs são moléculas recém-descobertas de RNA não-codificadores que possuem de 21 a 24 nucleotídeos e que regulam a expressão após a transcrição dos genes alvo. Essa regulação pode ser realizada através da inibição da tradução ou da degradação do RNA mensageiro. Os miRNAs estão envolvidos em vários processo biológicos como, diferenciação celular e desenvolvimento embrionário, além de apresentarem expressão tecido e tempo-específica. Eles podem regular a expressão de pelo menos 1/3 de todos os genes humanos e estão envolvidos com a regulação do metabolismo e da apoptose. Os miRNAs são a chave como reguladores pós-transcricionais da neurogênese; estudos mostram que eles possuem a expressão associada com a transição entre proliferação e diferenciação e também tem expressão constitutiva em neurônios maduros, evidenciando o envolvimento dessas moléculas com o desenvolvimento do sistema nervoso central (SNC). Outros miRNAs estão sendo estudados e verifica-se que eles agem como reguladores de genes envolvidos em doenças como Alzheimer, Parkinson e, provavelmente, também devam possuir um papel na regulação das epilepsias. No primeiro trabalho, apresentado no segundo capítulo, investigamos o papel dos miRNAs no desenvolvimento do SNC através da quantificação de 104 miRNAs em cérebros em desenvolvimento de camundongos. No segundo trabalho, apresentado no terceiro capítulo, para analisarmos o papel dos miRNAs na epilepsia de lobo temporal, verificamos se havia presença de miRNAs com expressão diferenciada entre tecidos removidos de pacientes que se submeteram a cirurgia de hipocampectomia e tecidos normais provenientes de autópsias. Para ambos os experimentos, foram extraídos os RNAs dos tecidos e quantificados por PCR em tempo real com o kit MicroRNA Assay baseado em iniciadores com estrutura em stem loop. Nos camundongos, análises de bioinformática encontraram quatro cluster de acordo com a expressão dos miRNAs. Um cluster (C1) com 12 miRNAs (miR-9; miR-17- 5p; miR-124a; miR-125a; miR-125b;miR-130a; miR-140; miR-181a; miR-199a; miR-205; miR-214; miR-301) apresentou expressão com diferença significativa durante o desenvolvimento. Nos tecidos dos pacientes, após a análise de bioinformática, encontramos três miRNAs com expressão diferenciada entre pacientes e controle (miR-29b, miR-30d e let-7). Em ambos os experimentos analisamos os possíveis genes alvo desse miRNAs. Nos camundongos, nossos resultados sugerem a presença de um padrão específico de expressão no cluster C1, indicando que esses miRNAs possam ter um papel na regulação de genes envolvidos na neurogênese. Nos tecidos humanos, os genes alvo encontrados estão envolvidos, principalmente, em proliferação celular, neurogênese e apoptose, indicando uma provável atuação dos miRNAs na regulação de genes que estão envolvidos na epilepsia de lobo temporal
Abstract: MicroRNAs are a new class of small RNA molecules (21-24 nucleotide-long) that negatively regulate gene expression either by translational repression or target mRNA degradation. It is believed that about 30% of all human genes are targeted by these molecules. MiRNAs are involved in many important biological processes including cell differentiation, embryonic development and central nervous system formation, besides they showed specific temporal-space expression. They can regulate 1/3 of human genes and are involved in metabolism and apoptosis. miRNAs are the key as neurogenesis postranscriptional regulation; studies previous indicates miRNA expression associate with proliferation and differentiation in development of central nervous system (CNS) and housekeeping expression in mature neurons. They are involved in several diseases as Alzkeimer's and Parkinson and may have a role in epilepsy regulation. In second chapter, we analyze the miRNA expression in mouse brain during four stages of CNS development; in third chapter, we analyze hippocampal tissue of four patients who underwent selective resection of the mesial temporal structures for the treatment of clinically refractory seizures. In addition we used control samples from autopsy (n=4) for comparison. In both experiments, total RNA was isolated from tissues and used in real-time PCR reactions with TaqMan¿ microRNA assays (Applied Biosystems) to quantify 104 (mouse brain) or 157 (human tissue) different miRNAs. In mouse brain analysis, we were able to identified four different clusters (C1, C2, C3 and C4) of miRNAs expression. Significant differences in expression during development were observed only in miRNAs included in C1. Our results suggest the presence of a specific expression pattern in C1, indicating that these miRNAs could have an important role in gene regulation during neurogenesis. We found a significant decrease (p<0,05) in expression of 12 miRNAs (miR-9; miR-17-5p; miR-124a; miR-125a; miR-125b;miR-130a; miR-140; miR-181a; miR-199a; miR-205; miR-214; miR- 301) belonging to cluster C1 in latter stages of development. Computational target identification showed that 10 of the 12 miRNAs present in C1 could be involved in neurogenesis. In human tissues, bioinformatics analyzes identified three miRNAs species which were differently expressed in patients as compared to controls: let7a was over expressed in patients (4 fold increased), miR-29b and miR-30d were down-regulated in patients (2.5 fold and 0.5 fold decreased, respectively). Possible target genes for let-7a are NME6 and NCAM1 (which would be down-regulated in patients); for miR-29b is MCL-1 and for miR30d are CTNND2, LGI1 and SON (which would be up-regulated in patients). We have identified three different miRNA species differently expressed in hippocampal sclerosis. Gene functions related to the possible miRNA targets are involved mainly with cell proliferation, neurogenesis, cell adhesion and apoptosis. Our results indicate new molecular targets which should be explored in additional studies addressing miRNA regulation in hippocampal sclerosis
Doutorado
Neurociencias
Doutor em Fisiopatologia Medica
Wang, Qi. "Using Imputed Microrna Regulation Based on Weighted Ranked Expression and Putative Microrna Targets and Analysis of Variance to Select Micrornas for Predicting Prostate Cancer Recurrence". Thesis, North Dakota State University, 2014. https://hdl.handle.net/10365/27341.
Pełny tekst źródłaKwan, Chun-kit Peter, i 關駿傑. "The expression of microRNA-34c and microRNA processing enzymes in preimplantation embryos". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B44659283.
Pełny tekst źródłaD'Ario, G. "IDENTIFICATION OF INTRONIC MICRORNAS ALTERED IN BREAST CANCER THROUGH MICROARRAY HOST GENE EXPRESSION ANALYSIS". Doctoral thesis, Università degli Studi di Milano, 2011. http://hdl.handle.net/2434/157939.
Pełny tekst źródłaSmith, Daniel. "Electrochemical detection of microRNA". Thesis, Cardiff University, 2017. http://orca.cf.ac.uk/107718/.
Pełny tekst źródłaKsiążki na temat "MicroRNA"
Ying, Shao-Yao, red. MicroRNA Protocols. Totowa, NJ: Humana Press, 2006. http://dx.doi.org/10.1385/1597451231.
Pełny tekst źródłaRani, Sweta, red. MicroRNA Profiling. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6524-3.
Pełny tekst źródłaSantulli, Gaetano, red. microRNA: Cancer. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-23730-5.
Pełny tekst źródłaYing, Shao-Yao, red. MicroRNA Protocols. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-083-0.
Pełny tekst źródłaYing, Shao-Yao, red. MicroRNA Protocols. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7601-0.
Pełny tekst źródłaKye, Min Jeong, red. MicroRNA Technologies. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7175-6.
Pełny tekst źródłaRani, Sweta, red. MicroRNA Profiling. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2823-2.
Pełny tekst źródłaShao-Yao, Ying, red. MicroRna protocols. Totowa, N.J: Humana Press, 2006.
Znajdź pełny tekst źródłaJ, Rossi John, i Hannon Gregory J. 1964-, red. MicroRNA methods. San Diego, Calif: Academic Press, 2007.
Znajdź pełny tekst źródłaservice), SpringerLink (Online, red. MicroRNA Interference Technologies. Berlin, Heidelberg: Springer-Verlag Berlin Heidelberg, 2009.
Znajdź pełny tekst źródłaCzęści książek na temat "MicroRNA"
Patne, Ketki, i Rohini Muthuswami. "Controlling the Biogenesis of the Smallest Regulators". W MicroRNA, 1–20. Boca Raton : Taylor & Francis, 2018. | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2018. http://dx.doi.org/10.1201/b22195-1.
Pełny tekst źródłaMuiwo, Pamchui, Priyatama Pandey i Alok Bhattacharya. "Computational Analysis of miRNAs, Their Target Sequences and Their Role in Gene Regulatory Networks". W MicroRNA, 21–38. Boca Raton : Taylor & Francis, 2018. | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2018. http://dx.doi.org/10.1201/b22195-2.
Pełny tekst źródłaAnand, Sneha, i Rentala Madhubala. "miRNAs: Small RNAs with Big Regulatory Functions in Parasitic Diseases". W MicroRNA, 39–56. Boca Raton : Taylor & Francis, 2018. | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2018. http://dx.doi.org/10.1201/b22195-3.
Pełny tekst źródłaThirugnanasambantham, Krishnaraj, Villianur Ibrahim Hairul Islam, Subramanian Saravanan, Venugopal Senthil Kumar, Ganapathy Ashok i Muthiah Chellappandian. "Role of miRNA in Multiple Sclerosis". W MicroRNA, 57–76. Boca Raton : Taylor & Francis, 2018. | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2018. http://dx.doi.org/10.1201/b22195-4.
Pełny tekst źródłaPaul, Jaishree, i Swati Valmiki. "miRNA Dysregulation in Inflammatory Bowel Disease and Its Consequences". W MicroRNA, 77–96. Boca Raton : Taylor & Francis, 2018. | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2018. http://dx.doi.org/10.1201/b22195-5.
Pełny tekst źródłaBhattacharyya, Malay, i Sanghamitra Bandyopadhyay. "Involvement of MicroRNAs in Alzheimer’s Disease". W MicroRNA, 97–112. Boca Raton : Taylor & Francis, 2018. | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2018. http://dx.doi.org/10.1201/b22195-6.
Pełny tekst źródłaSarangdhar, Mayuresh Anant, i Beena Pillai. "MicroRNAs in Neurogenesis and Neurodegeneration". W MicroRNA, 113–42. Boca Raton : Taylor & Francis, 2018. | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2018. http://dx.doi.org/10.1201/b22195-7.
Pełny tekst źródłaPant, Kishor, Amit Kumar Mishra i Senthil Kumar Venugopal. "MicroRNAs in the Progression of Hepatocellular Carcinoma". W MicroRNA, 143–72. Boca Raton : Taylor & Francis, 2018. | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2018. http://dx.doi.org/10.1201/b22195-8.
Pełny tekst źródłaVinchure, Omkar, i Ritu Kulshreshtha. "MicroRNA Regulation of Invasive Phenotype of Glioblastoma". W MicroRNA, 173–200. Boca Raton : Taylor & Francis, 2018. | “A CRC title, part of the Taylor & Francis imprint, a member of the Taylor & Francis Group, the academic division of T&F Informa plc.”: CRC Press, 2018. http://dx.doi.org/10.1201/b22195-9.
Pełny tekst źródłaLangenberger, David, Sebastian Bartschat, Jana Hertel, Steve Hoffmann, Hakim Tafer i Peter F. Stadler. "MicroRNA or Not MicroRNA?" W Advances in Bioinformatics and Computational Biology, 1–9. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-22825-4_1.
Pełny tekst źródłaStreszczenia konferencji na temat "MicroRNA"
Zhao, Xing-Hua, Jia-Feng Yu, Yan-ke Tang i Ji-Hua Wang. "G-MicroRNA: A New Tool for MicroRNA Genomics". W 2009 1st International Conference on Information Science and Engineering (ICISE 2009). IEEE, 2009. http://dx.doi.org/10.1109/icise.2009.614.
Pełny tekst źródłaSpivack, Simon D. "MicroRNA Affinity Assay". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2298.
Pełny tekst źródłaKim, Jungeun, Ying Zhang, Fadila Guessous i Roger Abounader. "Abstract 3165: microRNA-148a: A novel oncogenic microRNA in glioblastoma". W Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3165.
Pełny tekst źródłaNikitin, A. O., A. M. Timofeeva, S. E. Sedykh i G. A. Nevinsky. "ANTIBODY ACTIVITY IN MICRORNA HYDROLYSIS: ROLE IN GENE REGULATION IN VIRAL INFECTIONS". W X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-351.
Pełny tekst źródłaZhurko, P. T., I. V. Koktysh i R. M. Smolyakova. "MicroRNA let-7e AND miR-140 AS BIOMARKERS OF DEFORMING JOINT DISEASES". W SAKHAROV READINGS 2021: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute of Belarusian State University, 2021. http://dx.doi.org/10.46646/sakh-2021-2-55-58.
Pełny tekst źródłaNarcı, Kübra, Hasan Oğul i Mahinur Akkaya. "Sequence-based MicroRNA Clustering". W 7th International Conference on Bioinformatics Models, Methods and Algorithms. SCITEPRESS - Science and and Technology Publications, 2016. http://dx.doi.org/10.5220/0005552901070116.
Pełny tekst źródłaMalmhäll, Carina, Sahar Alawieh, Jan Lötvall i Madeleine Rådinger. "MicroRNA-146a and microRNA-155 expression in induced sputum of allergic asthmatics". W Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa2552.
Pełny tekst źródłaZaman, Mohd Saif, Guoren Deng, Varahram Shahryari, Sharanjot Saini, Shahana Majid, Kamaldeep Singh, Herman Sandhu, Inik Chang, Yuichiro Tanaka i Rajvir Dahiya. "Abstract 128: MicroRNA-23b acts as an oncogenic microRNA in renal cancer". W Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-128.
Pełny tekst źródłaDracheva, K. V., I. A. Pobozheva, K. A. Anisimova, Z. M. Hamid, S. G. Balandov, D. I. Vasilevsky, S. N. Pchelina i V. V. Miroshnikova. "MICRORNA OF SERUM EXTRACELLULAR VESICLES AS MARKERS OF TYPE 2 DIABETES MELLITUS DEVELOPMENT IN OBESITY". W X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-317.
Pełny tekst źródłaTran, Nhat, Vinay Abhyankar, KyTai Nguyen, Ishfaq Ahmad, Jon Weidanz i Jean Gao. "MicroRNA dysregulational synergistic network: Learning context-specific MicroRNA dysregulations in lung cancer subtypes". W 2017 IEEE International Conference on Bioinformatics and Biomedicine (BIBM). IEEE, 2017. http://dx.doi.org/10.1109/bibm.2017.8217640.
Pełny tekst źródłaRaporty organizacyjne na temat "MicroRNA"
Hammond, Scott M. MicroRNA Inhibitors as Anticancer Therapies. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2007. http://dx.doi.org/10.21236/ada475785.
Pełny tekst źródłaGalaktionov, Konstantin. MicroRNA and Breast Cancer Progression. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2007. http://dx.doi.org/10.21236/ada480199.
Pełny tekst źródłaShukla, Girish C. MicroRNA Targets of Human Androgen Receptor. Fort Belvoir, VA: Defense Technical Information Center, maj 2013. http://dx.doi.org/10.21236/ada589690.
Pełny tekst źródłaHsieh, Jer-Tsong, i Betty Diamond. Role of MicroRNA in Aggressive Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2014. http://dx.doi.org/10.21236/ada611002.
Pełny tekst źródłaHsieh, Jer-Tsong. Role of MicroRNA in Aggressive Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2013. http://dx.doi.org/10.21236/ada591960.
Pełny tekst źródłaWatabe, Kounosuke. DCIS-Specific MicroRNA in Cancer Stem Cell. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2011. http://dx.doi.org/10.21236/ada554452.
Pełny tekst źródłaNovina, Carl. Dysregulated microRNA Activity in Shwachman-Diamond Syndrome. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2015. http://dx.doi.org/10.21236/ada624270.
Pełny tekst źródłaBock, Cathryn. MicroRNA in Prostate Cancer Racial Disparities and Aggressiveness. Fort Belvoir, VA: Defense Technical Information Center, październik 2014. http://dx.doi.org/10.21236/ada613715.
Pełny tekst źródłaPadgett, Richard W. Role of MicroRNA Genes in Breast Cancer Progression. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2006. http://dx.doi.org/10.21236/ada462585.
Pełny tekst źródłaPadgett, Richard W. Role of MicroRNA Genes in Breast Cancer Progression. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2007. http://dx.doi.org/10.21236/ada482281.
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