Rozprawy doktorskie na temat „Metastatic carcinoma”
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Nordman, Ina IC Clinical School St Vincent's Hospital Faculty of Medicine UNSW. "Surrogate endpoints of survival in metastatic carcinoma". Publisher:University of New South Wales. Clinical School - St Vincent's Hospital, 2008. http://handle.unsw.edu.au/1959.4/42791.
Pełny tekst źródłaLee, Yee-ki Carol. "Effect of dietary fatty acids on metastatic hepatocellular carcinoma /". Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/hkuto/record/B39707349.
Pełny tekst źródłaMalek, Joël. "Genetic alterations of the metastatic lesions in ovarian carcinoma". Thesis, Paris 11, 2011. http://www.theses.fr/2011PA11T109.
Pełny tekst źródłaOvarian cancer is the most deadly gynecological cancer. The high rate of mortality is due to the large tumor burden with extensive metastatic lesion of the abdominal cavity. There are few studies on genetic alterations and their consequences in peritoneal metastatic tumors when compared to their matched ovarian primary tumors. Our hypothesis is that differences between the metastatic and primary lesions might be the cause of residual disease and, most importantly may have a role in post-chemotherapeutic recurrences. Methods: We conducted integrated genomics analysis on matched primary and metastatic tumors from 9 patients. In the papers presented here we analyze genome-wide Copy Number Variations (CNVs) using SNP Arrays targeting peritoneal metastasis differences, Gene expression differences using Microarrays also targeting peritoneal metastasis differences, and for some patients, Single Nucleotide Polymorphisms (SNPs) in genes through Exome sequencing.Results: Here we show that CNVs vary significantly between primary and metastatic tumors and include genes that have been considered potential chemotherapeutic targets based on primary tumor only data. Gene expression differences, while minor, showed highly statistically significant enrichment of genes in ovarian cancer critical pathways. In agreement with findings in other cancers, exome sequencing data revealed very few SNP differences of which most metastasis enriched SNPs were present at very low levels in the primary tumor. The results presented here should allow better design of therapies to target residual ovarian cancer disease
Bartrolí, Comellas Mariona. "Prognostic markers and therapeutic targets for metastatic renal cell carcinoma". Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/664198.
Pełny tekst źródłaRecentment, l’estudi de la metàstasi ha guanyat importància amb l’objectiu d’augmentar la supervivència dels pacients amb càncer. En el càncer renal (RCC), el descobriment de biomarcadors metastàtics i dianes terapèutiques és necessari degut a que la majoria de pacients presenten metàstasi en el moment del diagnòstic. L’objectiu d’aquesta tesi ha estat el descobriment de nous biomarcadors i dianes terapèutiques pel càncer renal metastàtic a través de dues variants d’un model animal orthoxenograft (PDOX) de RCC de cèl·lula clara (ccRCC). Els models PDOX han guanyat molta importància en l’estudi de la progressió del càncer i la metàstasi, ja que mimetitzen la histologia, la capacitat metastàtica i la resposta als tractaments. Prèviament, s’havien seqüenciat les dues variants d’aquest model PDOX tant a nivell de DNA com de RNA, juntament amb un anàlisi FISH. En primer lloc, la Carbxoxipeptidasa E (CPE), un dels gens més expressats en la variant metastàtica, ha demostrat ser important en la invasió quan és secretada al medi, tot i no ser suficient per generar metàstasi in vivo. A més, s’ha associat amb el ccRCC i anti-correlacionat amb la supervivència d’aquests pacients. En segon lloc, hem estudiat dues molècules de la cascada de coagulació, una de les més alterades en nivells de RNA. Hem demostrat que el Factor XIII (FXIII o F13) està relacionat amb CPE in vivo, malgrat que l’expressió de les dues molècules no és suficient per a que es desenvolupi la metàstasi. Tot i així, el F13 afecta la supervivència de pacients amb ccRCC, suggerint aquestes dues molècules com a possibles biomarcadors d’aquest tipus de càncer. A més, la inhibició del Receptor del Factor de Coagulació II (F2R) ha demostrat reduir les fases inicials i finals del procés metastàtic. Així doncs, l’ús d’inhibidors de F2R, juntament amb el fet que la cascada de coagulació es relaciona amb el pronòstic dels pacients, fa que aquesta tesi obri noves oportunitats per al tractament de la metàstasi i la malignització del càncer. En resum, hem descobert nous biomarcadors i dianes terapèutiques que, juntament amb futures validacions, sobretot en clínica, poden ser útils per als pacients metastàtics de ccRCC.
Pilborough, Alice Elizabeth. "Tumour-stromal crosstalk in metastatic lymph nodes of oral squamous cell carcinoma". Thesis, University of Sheffield, 2018. http://etheses.whiterose.ac.uk/22053/.
Pełny tekst źródłaHo, Chi-lai, i 何志禮. "Dual-tracer positron emission tomography in the evaluation ofprimary & metastatic hepatocellular carcinoma". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45205024.
Pełny tekst źródłaGriffin, Ryan D. "MULTISPECTRAL CO-OCCURRENCE ANALYSIS FOR AUTOMATED TUMOR DETECTION IN METASTATIC MEDULLARY THYROID CARCINOMA". The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1285101309.
Pełny tekst źródłaOkuno, Tomoko. "Loss of heterozygosity on 10q23 is involved in metastatic recurrence of hepatocellular carcinoma". Kyoto University, 2011. http://hdl.handle.net/2433/142555.
Pełny tekst źródłaKerr, Ian Balfour. "A study of metastasis in colorectal carcinoma using DNA recombinant and molecular hybridisation techniques". Thesis, University of Edinburgh, 1989. http://hdl.handle.net/1842/19010.
Pełny tekst źródłaSwofford, Brenen P., i Tomislav Dragovich. "Durable and Complete Response to Herceptin Monotherapy in Patients with Metastatic Gastroesophageal Cancer". KARGER, 2017. http://hdl.handle.net/10150/626424.
Pełny tekst źródłaForsyth, Leigh James. "Identification of DNA sequences involved in the metastatic phenotype of human prostatic carcinoma cells". Thesis, University of Liverpool, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.269601.
Pełny tekst źródłaDhanda, Jagtar. "Molecular indicators and tumour models of extracapsular spread in metastatic oral squamous cell carcinoma". Thesis, University of Liverpool, 2014. http://livrepository.liverpool.ac.uk/19295/.
Pełny tekst źródłaKelly, Stephen Richard. "The expression of matrix metalloproteinases (MMPs) and their inhibitors (TIMPs) in metastatic colorectal carcinoma (CRC)". Thesis, University of Southampton, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.412204.
Pełny tekst źródłaYau, Wing-lung, i 邱泳龍. "Identification of miR-106b over-expression in metastatic hepatocellular carcinoma by using the orthotopic animal model". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2010. http://hub.hku.hk/bib/B45204032.
Pełny tekst źródłaLand, Walker, Dan Margolis, Ronald Gottlieb, Elizabeth Krupinski i Jack Yang. "Improving CT prediction of treatment response in patients with metastatic colorectal carcinoma using statistical learning theory". BioMed Central, 2010. http://hdl.handle.net/10150/610011.
Pełny tekst źródłaMinata, Mutsuko. "Postoperative detection of α-fetoprotein mRNA in blood as a predictor for metastatic recurrence of hepatocellular carcinoma". Kyoto University, 2002. http://hdl.handle.net/2433/149325.
Pełny tekst źródłaSharma, Purva, James Kim, Devapiran Jaishankar i Sakshi Singal. "EXTENDED PROGRESSION-FREE SURVIVAL ON FIRST LINE TREATMENT WITH DOCETAXEL IN PATIENT WITH METASTATIC TRIPLE NEGATIVE BREAST CARCINOMA". Digital Commons @ East Tennessee State University, 2021. https://dc.etsu.edu/asrf/2021/presentations/43.
Pełny tekst źródłaXu, Wei. "Cytogenetic analysis of a murine mammary carcinoma in vitro and during progression from primary to metastatic growth in vivo". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp04/mq20717.pdf.
Pełny tekst źródłaShestowsky, William S. "Production and characterization of a monoclonal antibody to a highly metastatic and organ-selective variant of the Lewis lung carcinoma". Thesis, McGill University, 1988. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=61747.
Pełny tekst źródłaVinayan, Anup. "Targeted anti-angiogenic therapy in metastatic renal cell carcinoma and methodological improvements in assessment of therapeutic response with imaging biomarkers". Thesis, University of Hertfordshire, 2018. http://hdl.handle.net/2299/20960.
Pełny tekst źródłaKale, Hrishikesh P. "Economic Burden of Renal Cell Carcinoma (RCC) and Treatment Patterns, Overall Survival and Healthcare Costs among Older Metastatic RCC Patients". VCU Scholars Compass, 2018. https://scholarscompass.vcu.edu/etd/5555.
Pełny tekst źródłaWu, Rita Shiu-fung. "Effects of B7.1, IFN-gamma, and antisense TGF-beta gene transfer on the tumorigenicity of murine 4T1 metastatic mammary carcinoma cells". Diss., The University of Arizona, 2001. http://hdl.handle.net/10150/280475.
Pełny tekst źródłaHall, Charles. "Ex vivo reprogramming of tumor-reactive immune cells from FVBN202 mice bearing lung metastatic mammary carcinoma: an immunotherapeutic opportunity revealed against recurrence". VCU Scholars Compass, 2013. http://scholarscompass.vcu.edu/etd/3176.
Pełny tekst źródłaDuffie, Gordon Patrick. "Tumoricidal activity of pulmonary alveolar macrophages isolated from C57BL/6 mice bearing either a cloned metastatic or nonmetastatic variant of Lewis lung carcinoma". Virtual Press, 1988. http://liblink.bsu.edu/uhtbin/catkey/558376.
Pełny tekst źródłaDepartment of Biology
Raposo-Ferreira, Talita Mariana Morata [UNESP]. "Estudo em larga escala da expressão gênica de carcinomas mamários em cadelas". Universidade Estadual Paulista (UNESP), 2016. http://hdl.handle.net/11449/143484.
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Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
Os tumores mamários são os principais tumores que acometem as cadelas, sendo também os tumores mais frequentes em mulheres e, entre ambas as espécies, são observadas semelhanças relacionadas ao comportamento biológico e molecular dessa neoplasia. Assim, as cadelas podem ser um excelente modelo comparativo para o entendimento do processo carcinogênico desta neoplasia. A metástase é uma consequência comum e a principal causa de mortalidade em decorrência dessa enfermidade, em ambas as espécies. O perfil de expressão gênica global permite o melhor entendimento do processo de carcinogênese e de metástases, por permitir a identificação de inúmeros genes que possam estar envolvidos com esses processos. Assim, o objetivo desse estudo foi a realização do estudo em larga escala, através da técnica de microarray, em amostras de tecidos mamários caninos, considerando amostras de glândulas mamárias normais, tumores mamários benignos e tumores mamários malignos (carcinomas simples e mistos), além da avaliação de carcinomas mamários metastáticos e não metastáticos, para identificação de genes diferencialmente expressos entre esses grupos e relacionados com a tumorigênese e o desenvolvimento de metástases dos tumores mamários caninos. Observou-se, aproximadamente 1000 genes diferencialmente expressos entre as amostras tumorais e as glândulas mamárias normais e 465 genes diferencialmente expressos entre os tumores mamários benignos e malignos, sendo observados genes relacionados com o ciclo celular, desenvolvimento da glândula mamária e supressores tumorais. Em relação aos carcinomas mamários metastáticos e não metastáticos, verificou-se 633 genes diferencialmente expressos. Os genes supressores tumorais, como pertencentes a via de sinalização do ATM e do BRCA1, sugeriram importante papel tanto na progressão tumoral quanto no desenvolvimento de metástases. Outras vias observadas foram as relacionadas com angiogênese e de organização da matriz extracelular. Portanto, a análise do perfil gênico em larga escala permitiu a identificação de inúmeros genes e vias envolvidas tanto no processo de progressão tumoral como envolvidas com o desenvolvimento de metástases, permitindo a realização de estudos futuros em busca de marcadores prognósticos específicos.
Mammary tumors are the main tumors that affect female dogs, as well as in women and in both species, it is observed similarity related to the biological and molecular behavior of this neoplasia. So, female dogs are considered an excellent model for the understanding of carcinogenesis process from mammary tumors. Metastasis occurs frequently and it is the main responsible for the mortality by this neoplasia in both species. Global gene expression profile allows the better understanding of carcinogenesis and metastasis process by the identification of several genes that may be involved with these processes. Therefore, the aim of study was to perform a large-scale study by microarray technique using samples from canine mammary tissues, as normal mammary gland, benign tumors and malignant tumors (simple and mixed carcinomas), beyond metastatic and non-metastatic mammary carcinomas for the identification of differentially expression genes among these groups and related to canine mammary tumors tumorigenesis and metastasis. It was observed next to 1000 differentially expression genes between tumors and normal mammary glands samples and 465 differentially expression genes between benign and malignant mammary tumors mostly related to cell cycle, mammary gland development and tumor suppressors. Related to metastatic and non-metastatic mammary carcinomas was identified 633 differentially expression genes. ATM and BRCA1, associated with tumor suppressor gene pathway, showed to play an important role in both tumor progression and metastasis development. Other networks observed were related to angiogenesis and extracellular matrix organization. Thus, large-scale gene profile analysis allowed the identification of numerous genes and networks involved with both tumor progression and metastasis, allowing new studies in search of specific prognostic markers.
FAPESP: 2013/03940-4
FAPESP: 2013/25220-3
Miller, Kurt, Rudolf Morant, Arnulf Stenzl, Manfred P. Wirth i Ivan Zuna. "A Phase II Study of the Central European Society of Anticancer-Drug Research (CESAR) Group: Results of an Open-Label Study of Gemcitabine plus Cisplatin with or without Concomitant or Sequential Gefitinib in Patients with Advanced or Metastatic Transitional Cell Carcinoma of the Urothelium". Karger, 2016. https://tud.qucosa.de/id/qucosa%3A70589.
Pełny tekst źródłaPretscher, Dominik. "Distribution of immune cells in head and neck cancer : CD8+ T-cells and CD20+ B-cells in metastatic lymph nodes are associated with favourable outcome in patients with oro- and hypopharyngeal carcinoma". kostenfrei, 2010. http://d-nb.info/1000329054/34.
Pełny tekst źródłaOstheimer, Christian Emil Arthur Verfasser], Dirk [Akademischer Betreuer] Vordermark, Bernd [Akademischer Betreuer] Schmidt i Daniel [Akademischer Betreuer] [Zips. "Prognostic and predictive significance of osteopontin and other hypoxia-related plasma proteins in the radiotherapy of locally advanced and metastatic bronchial carcinoma / Christian Emil Arthur Ostheimer. Betreuer: Dirk Vordermark ; Bernd Schmidt ; Daniel Zips". Halle, Saale : Universitäts- und Landesbibliothek Sachsen-Anhalt, 2015. http://d-nb.info/1089085419/34.
Pełny tekst źródłaKwok, Mon-sze. "Study of metastatic suppressing genes on ovarian carcinomas /". View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36586328.
Pełny tekst źródłaKwok, Mon-sze, i 郭夢思. "Study of metastatic suppressing genes on ovarian carcinomas". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B45010729.
Pełny tekst źródłaLeBedis, Christina. "Lymph node involvement in breast carcinoma metastasis". Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31255.
Pełny tekst źródła吳曉靑 i Xiaoqing Wu. "Post-radiotherapy cervical metastasis in nasopharyngeal carcinoma". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B31222018.
Pełny tekst źródłaWu, Xiaoqing. "Post-radiotherapy cervical metastasis in nasopharyngeal carcinoma /". Hong Kong : University of Hong Kong, 1999. http://sunzi.lib.hku.hk/hkuto/record.jsp?B20843252.
Pełny tekst źródłaSilva, Maria João da Costa Soares da. "Clinical and molecular characterization of feline mammary carcinomas overexpressing HER2 proto-oncogene (FMC-HER2+) : new strategies for effective diagnostic and cancer therapy". Doctoral thesis, Universidade de Lisboa, Faculdade de Medicina Veterinária, 2016. http://hdl.handle.net/10400.5/12008.
Pełny tekst źródłamariajosoares@gmail.com
Considering the scarce data available in feline mammary carcinoma (FMC) and despite its importance in veterinary clinical practice, this thesis emerges in order to increase the knowledge of this tumor type, especially the FMC-HER2 positive. In the first two studies, the protocols for detection and quantification of the fHER2 and Ki-67 biomarkers were optimized and validated. These studies demonstrated that, in cats, the incidence of fHER2 overexpression were similar to women (about 30%), although no gene amplification was detected. It was also demonstrated that high levels of Ki-67 index were associated with a worse prognosis. Using a panel of protein biomarkers, the FMC were divided into six different groups that demonstrated prognostic value, similarly to what is described in women. In fact, cats with triple negative basal-like or HER2-positive subtypes were associated with shorter overall survival, contrasting with cats presenting luminal A tumors. Moreover, these studies also indicated that luminal B and triple negative basal-like subtypes are the most common in cats. When the metastatic lesions were evaluated, a marked loss of receptor expression was found, which was associated with an increase of the triple negative basal-like subtype, highlighting the importance of immunophenotyping all lesions (primary and metastatic) in cats. Considering these results, the development of diagnostic methodologies that allows the continuous follow-up of the patients would be very useful. Therefore, the last study presented in this thesis evaluates the fHER2 serum levels in cats with FMC using two different immunoassays (ELISA and dot blot). The serum levels of fHER2 were significantly associated with the fHER2 in tissue samples of FMC (assessed by IHC). This is consistent to what is described for humans and suggests that serum quantification could be an important tool for monitoring cats after the surgery. In sum, the results presented herein provide new diagnostic and prognostic tools for veterinary oncology. Considering the high prevalence and similarities with the human counterpart, cat can also represent a potential animal model for the study of luminal B and triple negative subtypes. Considering fHER2-positive FMC more studies are required in order to determine the aetiology of the protein overexpression.
RESUMO - Os tumores mamários felinos (TMF) são umas das neoplasias mais comuns em Oncologia Felina, com uma incidência que pode atingir os 40%, pelo que assumem um papel relevante na prática clínica veterinária. Estes tumores apresentam habitualmente uma etiologia maligna (carcinomas) e um comportamento agressivo, estando associados a um prognóstico reservado. Atualmente, existem poucas opções terapêuticas que permitam aumentar a qualidade e a esperança média de vida dos animais afectados por esta neoplasia. Assim, estudos que permitam uma melhor caracterização dos tumores, identificando potenciais biomarcadores que possam ser utilizados como factores de prognóstico ou preditivos, são fundamentais para o desenvolvimento da Medicina Felina. Por outro lado, os carcinomas mamários que ocorrem espontaneamente nos animais de companhia têm sido sugeridos como potenciais modelos biológicos para o estudo do Cancro da Mama, com vantagens, comparativamente aos animais de laboratório que são atualmente utilizados. De facto, nesses animais, os tumores são quimicamente induzidos ou xenotransplantados, pelo que são considerados modelos mais artificiais. Deste modo, esta tese de doutoramento surge com o objetivo de aumentar o conhecimento sobre os tumores mamários felinos, com especial interesse no recetor transmembranar para o fator de crescimento epidérmico de tipo 2 (HER2), quer numa perspetiva clínica, de forma a melhorar a qualidade de vida destes animais de companhia, abrindo portas a novos meios de diagnóstico e potenciais novos alvos terapêuticos, quer no sentido de investigar a viabilidade de a Gata ser um bom modelo animal para o estudo do Cancro da Mama na Mulher. Relativamente à Mulher, a investigação oncológica desenvolveu grandes esforços para encontrar biomarcadores que permitam otimizar o diagnóstico e o tratamento do cancro da mama. Entre estes, encontra-se a proteína HER2, uma oncoproteína que pode estar sobreexpressa em vários tumores da espécie humana (mama, pâncreas, cólon, próstata, bexiga, entre outros) e que lhes confere elevada agressividade e prognóstico reservado. No Cancro da Mama, estima-se que entre 10 a 40% dos tumores apresentem amplificação do gene HER2, o que se traduz na sobreexpressão da proteína. Estas alterações são rotineiramente detetadas através de duas técnicas moleculares, a hibridação in situ (ISH) e a imunohistoquímica (IHC), respetivamente. A avaliação do status da proteína HER2 é importante, não só pelo seu valor de prognóstico, mas também como fator preditivo, já que pacientes com sobreexpressão desta proteína são elegíveis para tratamento com terapêuticas específicas dirigidas contra o HER2, tais como os anticorpos anti-HER2 (sendo o trastuzumab o mais conhecido), o que veio aumentar consideravelmente a sobrevida destas doentes. Contrastando com a medicina humana, a literatura disponível em medicina veterinária apresenta ainda escassos estudos sobre a importância da proteína HER2 nos TMF, ou sobre os novos sistemas de classificação que subdividem os tumores mamários conforme o seu perfil imunofenotípico.(...)
Clark, Richard R. "Lymph node metastasis in auricular squamous cell carcinoma". Thesis, University of Glasgow, 2009. http://theses.gla.ac.uk/547/.
Pełny tekst źródłaYuen, Po-wing. "The study of nodal metastasis of oral tongue carcinoma". Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B39793837.
Pełny tekst źródłaHaq, Mahmudul. "Host-tumor interactions in skeletal metastasis of prostate carcinoma". Thesis, McGill University, 1992. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=56996.
Pełny tekst źródłaIp, Ying-chi. "MT1-MMP in relation to metastasis of hepatocellular carcinoma". Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31490189.
Pełny tekst źródła韋霖 i William I. Wei. "Surgery for post-radiotherapy cervical metastasis in nasopharyngeal carcinoma". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1991. http://hub.hku.hk/bib/B31979543.
Pełny tekst źródłaIp, Ying-chi, i 葉瑩芝. "MT1-MMP in relation to metastasis of hepatocellular carcinoma". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B31490189.
Pełny tekst źródłaYuen, Po-wing, i 袁寶榮. "The study of nodal metastasis of oral tongue carcinoma". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B39793837.
Pełny tekst źródłaVolk, Andreas, Stephan Kersting, Ralf Konopke, Frank Dobrowolski, Stefan Franzen, Detlef Ockert, Robert Grützmann, Hans Detlev Saeger i Hendrik Bergert. "Surgical Therapy of Intrapancreatic Metastasis from Renal Cell Carcinoma". Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-136489.
Pełny tekst źródłaDieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich
Volk, Andreas, Stephan Kersting, Ralf Konopke, Frank Dobrowolski, Stefan Franzen, Detlef Ockert, Robert Grützmann, Hans Detlev Saeger i Hendrik Bergert. "Surgical Therapy of Intrapancreatic Metastasis from Renal Cell Carcinoma". Karger, 2009. https://tud.qucosa.de/id/qucosa%3A27708.
Pełny tekst źródłaDieser Beitrag ist mit Zustimmung des Rechteinhabers aufgrund einer (DFG-geförderten) Allianz- bzw. Nationallizenz frei zugänglich.
Khan, Humma. "The influence of tumour angiogenesis on the metastatic potential of colorectal carcinomas". Thesis, Kingston University, 2007. http://eprints.kingston.ac.uk/20386/.
Pełny tekst źródłaEveritt, Gemma Louise Ann. "The inflammatory infiltrate of high-grade serous carcinoma omental metastasis". Thesis, Queen Mary, University of London, 2014. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8038.
Pełny tekst źródłaNwogu, Nnenna Onyinyechi Uchechi. "The role of MCPyV ST in Merkel cell carcinoma metastasis". Thesis, University of Leeds, 2017. http://etheses.whiterose.ac.uk/19356/.
Pełny tekst źródłaChan, Pui-man Poemen. "Micrometastases of esophageal cancer /". View the Table of Contents & Abstract, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36404652.
Pełny tekst źródłaDabare, Abeysinghe Arachchige Nandike Prashanth M. "Development of new monoclonal antibodies and colorimetric assays for improved detection of testicular germ cell tumours (TGCTs) and determining the relevance of over-expressed P53 in TGCT sensitivity to treatment". Thesis, Queen Mary, University of London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312172.
Pełny tekst źródłaHomer, Jarrod James. "Studies on angiogenesis in head and neck squamous cell carcinoma". Thesis, University of Hull, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.342866.
Pełny tekst źródłaXu, Haitao. "Overexpression of PAK4 and its relevance in hepatocellular carcinoma". Click to view the E-thesis via HKUTO, 2006. http://sunzi.lib.hku.hk/hkuto/record/B38703944.
Pełny tekst źródła