Gotowa bibliografia na temat „Metastatic cancer”
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Artykuły w czasopismach na temat "Metastatic cancer"
Yuk, Hyeong-Dong, Kyoung-Hwa Lee, Hye-Sun Lee, Seung-Hwan Jeong, Yongseok Kho, Chang-Wook Jeong, Hyeon-Hoe Kim, Ja-Hyeon Ku i Cheol Kwak. "PDLIM2 Suppression Inhibit Proliferation and Metastasis in Kidney Cancer". Cancers 13, nr 12 (15.06.2021): 2991. http://dx.doi.org/10.3390/cancers13122991.
Pełny tekst źródłaSingh Randhawa, Amritjot. "Metastatic Breast Cancer to the Uterine Cervix Mimicking Cervical Cancer". Indian Journal of Cancer Education and Research 8, nr 1 (1.06.2020): 49–52. http://dx.doi.org/10.21088/ijcer.2321.9815.8120.8.
Pełny tekst źródłaChen, Chao, Haozhen Liu, Qumiao Xu, Xiuqing Zhang, Feng Mu i Jixian Liu. "Association of PTPRT Mutations with Cancer Metastasis in Multiple Cancer Types". BioMed Research International 2022 (25.06.2022): 1–9. http://dx.doi.org/10.1155/2022/9386477.
Pełny tekst źródłaGanguly, Debolina, Marcel Schmidt, Morgan Coleman, Tuong-Vi Ngo, Noah Sorrelle, Adrian Dominguez, Jason Toombs i in. "Abstract B016: Pleiotrophin drives a pro-metastatic immune niche within the breast tumor microenvironment". Cancer Research 83, nr 2_Supplement_2 (15.01.2023): B016. http://dx.doi.org/10.1158/1538-7445.metastasis22-b016.
Pełny tekst źródłaPark, Jinkyung, Dahee Jeong, Meeryoung Song i Bonglee Kim. "Recent Advances in Anti-Metastatic Approaches of Herbal Medicines in 5 Major Cancers: From Traditional Medicine to Modern Drug Discovery". Antioxidants 10, nr 4 (27.03.2021): 527. http://dx.doi.org/10.3390/antiox10040527.
Pełny tekst źródłaCheng, Liang, David G. Bostwick, Guang Li, Shaobo Zhang, Alexander O. Vortmeyer i Zhengping Zhuang. "Conserved Genetic Findings in Metastatic Bladder Cancer". Archives of Pathology & Laboratory Medicine 125, nr 9 (1.09.2001): 1197–99. http://dx.doi.org/10.5858/2001-125-1197-cgfimb.
Pełny tekst źródłaWan, Xiaochun, i Junxin Li. "DcR3 acts as a tumor marker of metastatic cancers". Journal of Immunology 200, nr 1_Supplement (1.05.2018): 178.45. http://dx.doi.org/10.4049/jimmunol.200.supp.178.45.
Pełny tekst źródłaTarragó-Celada, Josep, i Marta Cascante. "Targeting the Metabolic Adaptation of Metastatic Cancer". Cancers 13, nr 7 (1.04.2021): 1641. http://dx.doi.org/10.3390/cancers13071641.
Pełny tekst źródłaPark, Misung, Dohee Kim, Sunghyub Ko, Ayoung Kim, Kyumin Mo i Hyunho Yoon. "Breast Cancer Metastasis: Mechanisms and Therapeutic Implications". International Journal of Molecular Sciences 23, nr 12 (18.06.2022): 6806. http://dx.doi.org/10.3390/ijms23126806.
Pełny tekst źródłaPein, Maren, i Thordur Oskarsson. "Microenvironment in metastasis: roadblocks and supportive niches". American Journal of Physiology-Cell Physiology 309, nr 10 (15.11.2015): C627—C638. http://dx.doi.org/10.1152/ajpcell.00145.2015.
Pełny tekst źródłaRozprawy doktorskie na temat "Metastatic cancer"
Wander, Seth A. "p27 and Metastatic Progression: Molecular Mechanisms Underlying Bone Metastasis". Scholarly Repository, 2011. http://scholarlyrepository.miami.edu/oa_dissertations/690.
Pełny tekst źródłaNordman, Ina IC Clinical School St Vincent's Hospital Faculty of Medicine UNSW. "Surrogate endpoints of survival in metastatic carcinoma". Publisher:University of New South Wales. Clinical School - St Vincent's Hospital, 2008. http://handle.unsw.edu.au/1959.4/42791.
Pełny tekst źródłaSolberg, Arne. "Outcome Assessments in Non- Metastatic Prostate Cancer". Doctoral thesis, Norges teknisk-naturvitenskapelige universitet, Institutt for kreftforskning og molekylær medisin, 2011. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-13881.
Pełny tekst źródłaPaon, Lenaic Marie Pierre. "Gene expression profiling of metastatic gastric cancer". Thesis, University of Liverpool, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.490659.
Pełny tekst źródłaPelon, Floriane. "Fibroblastic heterogeneity and metastatic spread in breast cancers Fibroblast heterogeneity drives metastatic spread in breast cancer through distinct mechanisms". Thesis, Sorbonne Paris Cité, 2018. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=2392&f=17330.
Pełny tekst źródłaBreast cancers are the most common cancers in women and despite great improvements in treatments, they are still responsible for many deaths worldwide. They are classified into 3 main molecular subtypes: Luminal cancers are the most frequent ones, while HER2 and TN are the most aggressive. At diagnostic, lymph node involvement is also assessed as it constitutes, in addition to molecular classification, a strong prognostic marker. Indeed, it informs on the risk to develop further distant metastases, which is the main cause of death by cancer. Solid tumors, including breast cancers, are complex ecologies comprising numerous different cell types that interact with cancer cells. Among them, cancer-associated-fibroblasts (CAF) are the most abundant and actively participate in many tumor hallmarks such as tumor growth, invasion, immunosuppression and angiogenesis. However, they do not constitute a homogeneous population but so far, only few studies have characterized this heterogeneity and linked it to CAF previously described functions. In this project, we focused on the potential involvement of CAF heterogeneity in breast cancer metastatic spread. Combining the analysis of several CAF markers, we showed that invaded LN comprise 4 CAF subsets (CAF-S1, S2, S3 and S4), similar to those found in primary tumors. Interestingly, the two myofibroblastic subsets (αSMA+) CAF-S1 and especially CAF-S4 preferentially accumulate in metastatic LN and present the same transcriptomic profiles in both tumors and LN. Importantly, both CAF-S1 and CAF-S4 display pro-invasive properties, by acting at different levels on tumor cells. On the one hand, highly motile CAF-S1 stimulate breast cancer cell proliferation, migration and EMT initiation. On the other hand, CAF-S4 exhibit an important contractility and by remodeling the matrix they are able to promote tumor cell invasion in 3D. Functional studies highlight a CXCL12/TGFβ involvement in CAF-S1 functions while CAF-S4 pro-invasive phenotype appears to be Notch-dependent. In agreement with these data, we found that CAF accumulation and subset enrichment in involved LN were two new prognostic factors, independent of breast cancer molecular subtypes and LN status at diagnosis. Indeed, stromal rich LN with a predominance of CAF-S4 are associated with long distance metastases development and poor overall survival. Thus, we propose that analyzing LN fibroblastic content at diagnosis could constitute new and useful information to breast cancer patients’ care
Chiang, Yan Ting. "Identification of metastasis-driving genes as potential therapeutic targets/ biomarkers for metastatic prostate cancer". Thesis, University of British Columbia, 2015. http://hdl.handle.net/2429/52901.
Pełny tekst źródłaMedicine, Faculty of
Graduate
Nielsen, S. R. "Metastasis-associated macrophages orchestrate the formation of a hospitable metastatic niche in pancreatic cancer". Thesis, University of Liverpool, 2017. http://livrepository.liverpool.ac.uk/3007527/.
Pełny tekst źródłaReed, Elizabeth. "The experience of living with metastatic breast cancer". Thesis, University of Southampton, 2012. https://eprints.soton.ac.uk/344746/.
Pełny tekst źródłaHe, Felicia Jane. "Targeting Metastatic Breast Cancer Using Dual-Ligand Nanoparticles". Case Western Reserve University School of Graduate Studies / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=case1499699087340348.
Pełny tekst źródłaDonald, Carlton Dewitt. "Metastatic characteristics of tumor progression in Prostate Cancer". DigitalCommons@Robert W. Woodruff Library, Atlanta University Center, 1995. http://digitalcommons.auctr.edu/dissertations/3299.
Pełny tekst źródłaKsiążki na temat "Metastatic cancer"
Swanton, Charles, i Stephen R. D. Johnston. Handbook of metastatic breast cancer. Wyd. 2. New York: Informa Healthcare, 2011.
Znajdź pełny tekst źródłaSherbet, G. V. The Metastatic Spread of Cancer. London: Palgrave Macmillan UK, 1987. http://dx.doi.org/10.1007/978-1-349-09577-3.
Pełny tekst źródłaKaufmann, M. Therapeutic management of metastatic breast cancer. Berlin: Walter de Gruyter & Co, 1989.
Znajdź pełny tekst źródłaLeong, Stanley P. L., red. From Local Invasion to Metastatic Cancer. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-087-8.
Pełny tekst źródłaMetastatic cancer: Clinical and biological perspectives. Austin, Texas, USA: Landes Bioscience, 2013.
Znajdź pełny tekst źródła1952-, Wick Mark R., red. Metastatic carcinomas of unknown origin. New York: DEMOS, 2008.
Znajdź pełny tekst źródłaChu, Edward. New treatment strategies for metastatic colorectal cancer. [Manhasset, NY]: CMP Medica, 2008.
Znajdź pełny tekst źródłaNew treatment paradigms in metastatic breast cancer. [Manhasset, NY]: CMPMedica, 2008.
Znajdź pełny tekst źródłaChu, Edward. New treatment strategies for metastatic colorectal cancer. [Manhasset, NY]: CMP Medica, 2008.
Znajdź pełny tekst źródłaEvans, Clive W. The metastatic cell: Behaviour and biochemistry. London: Chapman and Hall, 1991.
Znajdź pełny tekst źródłaCzęści książek na temat "Metastatic cancer"
Szmulewitz, Russell, Jennifer Taylor i Carrie Rinker-Schaffer. "Metastatic Colonization". W Encyclopedia of Cancer, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_3676-2.
Pełny tekst źródłaSzmulewitz, Russell, Jennifer Taylor i Carrie Rinker-Schaffer. "Metastatic Colonization". W Encyclopedia of Cancer, 2797–99. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46875-3_3676.
Pełny tekst źródłaSzmulewitz, Russell, Jennifer Taylor i Carrie Rinker-Schaffer. "Metastatic Colonization". W Encyclopedia of Cancer, 2271–73. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_3676.
Pełny tekst źródłaField, Kathryn M., i John R. Zalcberg. "Chemotherapy: Metastatic Disease". W Rectal Cancer, 189–222. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-567-5_12.
Pełny tekst źródłaSumpter, K., i D. Cunningham. "Metastatic Rectal Cancer". W Modern Management of Cancer of the Rectum, 157–77. London: Springer London, 2001. http://dx.doi.org/10.1007/978-1-4471-0331-8_14.
Pełny tekst źródłaHalfdanarson, Thorvardur R., i Joleen M. Hubbard. "Metastatic Rectal Cancer". W Modern Management of Cancer of the Rectum, 287–310. London: Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6609-2_20.
Pełny tekst źródłaPatel, Tejal A., i Edith A. Perez. "Metastatic Breast Cancer". W Management of Breast Diseases, 425–49. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-69743-5_23.
Pełny tekst źródłaStephens, Frederick O., i Karl Reinhard Aigner. "Metastatic (Secondary) Cancer". W Basics of Oncology, 299–303. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-92925-3_22.
Pełny tekst źródłaStephens, Frederick O., i Karl Reinhard Aigner. "Metastatic (Secondary) Cancer". W Basics of Oncology, 331–34. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-23368-0_22.
Pełny tekst źródłaHarris, Louise, Caitlin Bowen, Satish Kumar, Bernadette Coles i Malcolm Mason. "Metastatic bladder cancer". W Evidence-Based Urology, 383–89. Chichester, UK: John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119129875.ch32.
Pełny tekst źródłaStreszczenia konferencji na temat "Metastatic cancer"
Yoshimoto, T., T. Ishikawa, N. Matsuki, H. Fujiwara, Y. Imai, H. Ueno, M. Takeda i T. Yamaguchi. "Rheology of Cancer Cells With Different Metastatic Properties". W ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206593.
Pełny tekst źródłaCosta, Rafael Everton Assunção Ribeiro da, Fergus Tomás Rocha de Oliveira, Eduarda Norberto Siqueira, Ana Lúcia Nascimento Araújo i Sabas Carlos Vieira. "CUTANEOUS AND BONE METASTASIS OF OCCULT BREAST CANCER: CASE REPORT". W Abstracts from the Brazilian Breast Cancer Symposium - BBCS 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s2078.
Pełny tekst źródłaInufusa, H., N. Sagara, K. Nakano i M. Yasutomi. "CHARACTERISATION OF PLATELET AGGREGATING MATERIAL EXTRACTED FROM HUMAN LUNG ADENOCARCINOMA CELL LINE WHICH METASTASIZED IN NUDE MICE S,C, IMPLANTATION". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643199.
Pełny tekst źródłaHoskins, Meghan H., Shile Liang, Robert F. Kunz i Cheng Dong. "Cellular Mechanics and Biology of Tumor Cell-Leukocyte Interactions in the Near Wall Region Under Shear Flow Conditions". W ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176376.
Pełny tekst źródłaShareef, Sarah J., Mihai Nita-Lazar i Maria A. Kukuruzinska. "E-cadherin N-glycosylation Modulates the Strength of Adherens Junctions". W ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13013.
Pełny tekst źródłaSayyad, Megan, Madhavi Puchalapalli, Chris Canal, Theresa Swift-Scanlan, Andrew Ottens i Jennifer E. Koblinski. "Abstract 1988: Metastatic v non-metastatic cancer exosomes in brain metastasis". W Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1988.
Pełny tekst źródłaSayyad, Megan, Madhavi Puchalapalli, Chris Canal, Theresa Swift-Scanlan, Andrew Ottens i Jennifer E. Koblinski. "Abstract 1988: Metastatic v non-metastatic cancer exosomes in brain metastasis". W Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1988.
Pełny tekst źródłaMcClain, Jacob, Sara L. Tuell i Susan N. Thomas. "Tumors Change the Elastic and Viscoelastic Properties of Draining Lymph Node Tissues". W ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14419.
Pełny tekst źródłaCruz, Marcelo Ribeiro da Luz, Valeria Fernandes Roppa Cruz, Alfredo Almeida Cunha i Renato Souza Bravo. "CAN AXILLARY ULTRASONOGRAPHY WITH CORE NEEDLE BIOPSY BE A USEFUL TOOL IN THE APPROACH OF BREAST CANCER PATIENTS?" W Scientifc papers of XXIII Brazilian Breast Congress - 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s1004.
Pełny tekst źródłaFreitas, Paola Ferreira de, Renata Borges de Aquino, Andre Mattar, Jorge Yoshinori Shida i Luiz Henrique Gebrim. "CASE REPORT: PATHOLOGICAL COMPLETE RESPONSE IN BREAST IN A PATIENT WITH METASTATIC BREAST CANCER TREATED WITH ANASTROZOLE". W Abstracts from the Brazilian Breast Cancer Symposium - BBCS 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s2088.
Pełny tekst źródłaRaporty organizacyjne na temat "Metastatic cancer"
Tinker, Anna V., Neesha C. Dhani, Prafull Ghatage, Deanna McLeod, Vanessa Samouëlian, Stephen A. Welch i Alon D. Altman. Immune Checkpoint Inhibitors in Pretreated Metastatic Endometrial Cancer. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, styczeń 2023. http://dx.doi.org/10.37766/inplasy2023.1.0038.
Pełny tekst źródłaKhan, Shafiq. Biomarkers for Metastatic Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, maj 2004. http://dx.doi.org/10.21236/ada427158.
Pełny tekst źródłaWilliams, Myron N. Biomarkers for Metastatic Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, maj 2003. http://dx.doi.org/10.21236/ada416624.
Pełny tekst źródłaDeisseroth, Albert B. Vectors for Treatment of Metastatic Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2005. http://dx.doi.org/10.21236/ada448903.
Pełny tekst źródłaDeisseroth, Albert B. Vectors for Treatment of Metastatic Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2004. http://dx.doi.org/10.21236/ada429189.
Pełny tekst źródłaTakeshita, Kenichi. Retinoic Acid Receptors in Metastatic Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2001. http://dx.doi.org/10.21236/ada413284.
Pełny tekst źródłaMarchetti, Dario. Heparanase Mechanisms in Brain-metastatic Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2013. http://dx.doi.org/10.21236/ada585985.
Pełny tekst źródłaPang, Shen. Gene Delivery for Metastatic Prostate Cancer Cells. Fort Belvoir, VA: Defense Technical Information Center, styczeń 2000. http://dx.doi.org/10.21236/ada390852.
Pełny tekst źródłaTakeshita, Kenichi. Retinoic Acid Receptors in Metastatic Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, październik 1999. http://dx.doi.org/10.21236/ada392639.
Pełny tekst źródłaMarchetti, Dario. Heparanase Mechanisms in Brain-Metastatic Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, listopad 2013. http://dx.doi.org/10.21236/ada596541.
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