Gotowa bibliografia na temat „Metabolic depression”
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Artykuły w czasopismach na temat "Metabolic depression"
Vogelzangs, Nicole, Aartjan T. F. Beekman, Ingrid G. Boelhouwer, Stefania Bandinelli, Yuri Milaneschi, Luigi Ferrucci i Brenda W. J. H. Penninx. "Metabolic Depression". Journal of Clinical Psychiatry 72, nr 05 (5.04.2011): 598–604. http://dx.doi.org/10.4088/jcp.10m06559.
Pełny tekst źródłaLamers, F., Y. Milaneschi, P. de Jonge, E. J. Giltay i B. W. J. H. Penninx. "Metabolic and inflammatory markers: associations with individual depressive symptoms". Psychological Medicine 48, nr 7 (11.09.2017): 1102–10. http://dx.doi.org/10.1017/s0033291717002483.
Pełny tekst źródłaFauziyati, Ana, Agus Siswanto, Luthfan Budi Purnomo i Hemi Sinorita. "Type of psychosocial stressor as risk factor of depressive symptom in metabolic syndrome". Bangladesh Journal of Medical Science 15, nr 2 (10.08.2016): 262–68. http://dx.doi.org/10.3329/bjms.v15i2.28867.
Pełny tekst źródłaHeiskanen, Tuula H., Leo K. Niskanen, Jukka J. Hintikka, Heli T. Koivumaa-Honkanen, Kirsi M. Honkalampi, Kaisa M. Haatainen i Heimo T. Viinamäki. "Metabolic Syndrome and Depression". Journal of Clinical Psychiatry 67, nr 09 (15.09.2006): 1422–27. http://dx.doi.org/10.4088/jcp.v67n0913.
Pełny tekst źródłaHeuser, I. "Metabolic syndrome and depression". Journal of Affective Disorders 107 (marzec 2008): S24—S25. http://dx.doi.org/10.1016/j.jad.2007.12.158.
Pełny tekst źródłaGary, Tiffany L., Kesha Baptiste-Roberts, Rosa M. Crum, Lisa A. Cooper, Daniel E. Ford i Frederick L. Brancati. "Changes in Depressive Symptoms and Metabolic Control over 3 Years among African Americans with Type 2 Diabetes". International Journal of Psychiatry in Medicine 35, nr 4 (grudzień 2005): 377–82. http://dx.doi.org/10.2190/bq22-4hu0-p6ek-l7yx.
Pełny tekst źródłaFlores-Ramos, Mónica, Martín Armando Burrola-Suárez, Rodrigo Guiza-Zayas, J. Miguel Enciso-Araujo, Dannia Islas-Preciado i Erika Estrada Camarena. "Evaluation of hormonal and metabolic factors related to depression in reproductive age women". Salud mental 43, nr 1 (30.01.2020): 35–41. http://dx.doi.org/10.17711/sm.0185-3325.2020.006.
Pełny tekst źródłaMarijnissen, R. M., N. Vogelzangs, M. E. Mulder, R. H. S. van den Brink, H. C. Comijs i R. C. Oude Voshaar. "Metabolic dysregulation and late-life depression: a prospective study". Psychological Medicine 47, nr 6 (12.12.2016): 1041–52. http://dx.doi.org/10.1017/s0033291716003196.
Pełny tekst źródłaJohn, George, Mona Asghari, Vipin VP i Valsamma Eapen. "Depression and Metabolic Syndrome: Two Sides of the Same Coin". Journal of Biomedical and Clinical Research 12, nr 1 (1.07.2019): 3–9. http://dx.doi.org/10.2478/jbcr-2019-0001.
Pełny tekst źródłaOlvera, Rene L., Douglas E. Williamson, Susan P. Fisher-Hoch, Kristina P. Vatcheva i Joseph B. McCormick. "Depression, Obesity, and Metabolic Syndrome". Journal of Clinical Psychiatry 76, nr 10 (21.10.2015): e1300-e1305. http://dx.doi.org/10.4088/jcp.14m09118.
Pełny tekst źródłaRozprawy doktorskie na temat "Metabolic depression"
Muir, Timothy J. "Mechanisms and phylogenetic breadth of urea-induced hypometabolism". Oxford, Ohio : Miami University, 2009. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=miami1247688904.
Pełny tekst źródłaTweedy, Maureen P. "Metabolic Syndrome and Psychosocial Factors". Thesis, University of North Texas, 2009. https://digital.library.unt.edu/ark:/67531/metadc11005/.
Pełny tekst źródłaTweedy, Maureen P. Guarnaccia Charles Anthony. "Metabolic syndrome and psychosocial factors". [Denton, Tex.] : University of North Texas, 2009. http://digital.library.unt.edu/permalink/meta-dc-11005.
Pełny tekst źródłaBishop, T. "Living at a snail's pace : the cellular basis of metabolic depression". Thesis, University of Cambridge, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.596665.
Pełny tekst źródłaWang, Yijie, i 王怡洁. "Associations among type A and type D personalities, metabolicsyndrome, and anxiety/depression". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B47849496.
Pełny tekst źródłapublished_or_final_version
Social Work and Social Administration
Doctoral
Doctor of Philosophy
Silveira, Lilian Cristina da. "Reorganização estrutural e metabólica do tecido cardíaco associada à dormência e jejum sazonal em lagartos teiú Tupinambis merianae". Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/41/41135/tde-13052011-100148/.
Pełny tekst źródłaThe structural and metabolic flexibility of cardiac response to a variable physiological demand is notable. During seasonal dormancy, interruption of feeding together with inactivity and reduced heart beating, cause a large decrease of demand which probably brings about structural and metabolic heart tissue reorganization. These aspects were studied during the annual cycle in young tegu lizards Tupinambis merianae to investigate the hypothesis of seasonal changes of the heart capacities given by adjustments of tissue mass, structure and composition, by regulation of flux of substrates in the pathways of energy production, and by changes in the composition of fatty acids of tissue membranes. Groups of animals were killed in selected phases during the first year cycle of young tegus and after a 20 days fasting period during spring activity. Heart ventricle was removed and weighed and a tissue sample was collected and transfered to fixative solution, being used to obtain tissue slices of 10μm width for histological analysis with stereological tools. The remaining tissue was cut and split into aliquots, frozen in liquid N2 and stored at -80ºC. Later, the aliquots were used to assess the content of water, total and soluble proteins, and total lipids, by standard assays, the maximum activity of enzymes by spectrophotometry, and neutral and polar fatty acids profiles by gas chromatography. In early fall, the relative mass ventricle is 0.16%, and 31% increased in late fall, when the spongy myocardium appears dense and with few lacunar spaces which area corresponds to 8% of slice total área. During dormancy, the ventricle mass increases further 29%, decreasing to values of late fall during early arousal. After food intake, mass ventricle is again increased together with a small increase of the lacunar spaces, which appear highly expanded later in spring (29% of the total area), when tissue mass is 0,24% increased in relation to early fall. Unlike dormancy, fasting during spring caused a decrease of 19% of the ventricle mass. The cardiomyocytes density in the spongy layer is 37% decreased during dormancy while estimated cell volume is 52% increased, in relation to spring activity. There was no seasonal changes in the content of water and proteins in the groups analysed, except to a tendency to increase in the water content during dormancy, and to decrease in the soluble proteins in early arousal and in fasted animals. Myofibrillar protein is lower during fall and dormancy in relation to spring, increasing soon in early arousal after water intake. Total lipids decrease in the tissue during dormancy in relation to late fall by similar proportion than after fasting during spring. The glycolytic enzymes PK e LDH are unchanged during the year cycle, whereas the mitochondrial CS shows a tendency to increase, and HOAD, a β-oxidation enzyme, is decreased during dormancy and early arousal, in relation to fall. Unlike, PK and CS are decreased, while HOAD is unchanged after a period of fasting during spring. Fatty acids (FA) profiles of neutral lipids suggest that unsaturated FA are preferentially mobilized during dormancy and arousal, whereas all FA would be equally used during spring fasting. FA of polar lipids are remarkably constant during the year, suggesting that membrane FA in the heart tissue are not generally affected by season, and thus, results do not support a predominant role played by compositional changes of membranes in metabolic depression in the tegu. In addition, the otherwise distinct findings with hibernating mammals suggest that changes of FA composition in these animals would be an adaptation to the low body temperature of torpor, rather than mechanism of metabolic inhibition. Regression analysis indicate significant relationships of C18:1n-9, C22:5n-6, and C22:6n-3 contents as a function of body mass in young tegus, and changes in the allometric patterns are consistent with a putative relationship between these FA levels and the scaling of mass specific metabolic rates of young tegus during the year cycle.
Hoopes, Lisa Ann. "Metabolic and thermoregulatory capabilities of juvenile steller sea lions, Eumetopias jubatus". [College Station, Tex. : Texas A&M University, 2007. http://hdl.handle.net/1969.1/ETD-TAMU-1390.
Pełny tekst źródłaOlvera, Anna E. "Diabetes in Latinas : depression, metabolic control and the roles of acculturation and social support". Access to abstract only; dissertation is embargoed until after 12/20/2006, 2005. http://www4.utsouthwestern.edu/library/ETD/etdDetails.cfm?etdID=135.
Pełny tekst źródłaSharovsky, Lilian Lopes. "Análise de sintomas depressivos e ansiosos nas variáveis clínicas da síndrome metabólica". Universidade de São Paulo, 2010. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-22062010-131123/.
Pełny tekst źródłaObjective: Metabolic Syndrome (MetS) has been associated to depression sintomatology by means of the hypothalamus-pituitary-adrenal axle activation and consequent alterations of the cortisol levels. The objective of this study is to explore the association of depressive and anxiety symptoms severity with MetS clinical variables and salivary cortisol. Methods: We studied 136 consecutive ambulatory patients (aged= 55,37±7,62), n= 69 women, who presented MetS criteria according to the National Cholesterol Education Program Revised (NCEP-ATPIII-R). At the first visit they all completed a social-demographic questionnaire.Then, we assessed depressive symptoms by Beck Depression Inventory (BDI) and anxiety symptoms by Hamilton Anxiety Rating Scale (HARS). Salivary cortisol was assessed at 7:00h A.M. Results: We observed that the higher the intensity of the depressive symptoms by the BDI, the higher the glycemia value (p=0,01) in the female group (p 0,001). The same correlation was observed between HARS and glycemia (p=0,001) and HARS and genre (p=0,02). No correlation between depressive and anxiety symptoms with the other MetS clinical variable was found. The correlation was also positive between HARS and BDI. No correlation between depressive and anxiety symptoms with salivary cortisol was found. Furthermore, there was no association between cortisol and MS clinical variables. Conclusions: In the studied population, a presence of a higher intensity of depressive and anxiety symptoms was verified in the female group. We observed that, in this group, both the BDI and HARS value was explained by an increased fasting glucose. There was neither a correlation between depressive and anxiety symptoms and salivary cortisol
Kemp, David E. "Use of Insulin Sensitizers as a Novel Treatment for Major Depressive Disorder: A Pilot Study of Pioglitazone for Major Depression Accompanied by Abdominal Obesity". Case Western Reserve University School of Graduate Studies / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=case1275567223.
Pełny tekst źródłaKsiążki na temat "Metabolic depression"
Farooqui, Akhlaq A. Metabolic syndrome: An important risk factor for stroke, Alzheimer disease, and depression. New York, NY: Springer, 2013.
Znajdź pełny tekst źródłaJay, Lombard, i Monte Tom, red. Freedom from disease: The breakthrough approach to preventing cancer, heart disease, alzheimer's, and depression by controlling insulin. New York: St. Martin's Griffin, 2009.
Znajdź pełny tekst źródłaJay, Lombard, i Monte Tom, red. Freedom from disease: The breakthrough approach to preventing cancer, heart disease, Alzheimer's, and depression by controlling insulin. New York: St. Martin's, 2008.
Znajdź pełny tekst źródłaOsher, Susan. One-carbon metabolism in adults with major depression. Ottawa: National Library of Canada, 1999.
Znajdź pełny tekst źródłaJones, Shirley Anne. Tetrahydrobiopterin metabolism in depression and senile dementia of Alzheimer type. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1988.
Znajdź pełny tekst źródła1935-, Takada Akikazu, i Curzon G. 1928-, red. Serotonin in the central nervous system and periphery: Proceedings of the Symposium on Serotonin in the Central Nervous System and Periphery, April 1-2, 1995, Nagoya, Japan. Amsterdam: Elsevier, 1995.
Znajdź pełny tekst źródłaGissey, Lidia Castagneto, James R. Casella Mariolo, Geltrude Mingrone i Francesco Rubino. Metabolic surgery and depression. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198789284.003.0012.
Pełny tekst źródłaBertocci, Michele A., i Mary L. Phillips. Neuroimaging of Depression. Redaktorzy Dennis S. Charney, Eric J. Nestler, Pamela Sklar i Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0025.
Pełny tekst źródłaFarooqui, Akhlaq A. Metabolic Syndrome: An Important Risk Factor for Stroke, Alzheimer Disease, and Depression. Springer, 2013.
Znajdź pełny tekst źródłaFarooqui, Akhlaq A. A. Metabolic Syndrome: An Important Risk Factor for Stroke, Alzheimer Disease, and Depression. Springer, 2015.
Znajdź pełny tekst źródłaCzęści książek na temat "Metabolic depression"
Farooqui, Akhlaq A. "Metabolic Syndrome as a Risk Factor for Depression". W Metabolic Syndrome, 343–78. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-7318-3_9.
Pełny tekst źródłaWithers, Philip C., i Christine E. Cooper. "Metabolic Depression: A Historical Perspective". W Aestivation, 1–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-02421-4_1.
Pełny tekst źródłaHarvey, Brian H. "Major Depression and Metabolic Encephalopathy: Syndromes More Alike Than Not?" W Metabolic Encephalopathy, 349–69. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-79112-8_18.
Pełny tekst źródłaStorey, Kenneth B., i Janet M. Storey. "Oxygen Limitation and Metabolic Rate Depression". W Functional Metabolism, 415–42. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2005. http://dx.doi.org/10.1002/047167558x.ch15.
Pełny tekst źródłaBrand, Martin D., Tammie Bishop, Robert G. Boutilier i Julie St-Pierre. "Mitochondrial Proton Conductance, Standard Metabolic Rate and Metabolic Depression". W Life in the Cold, 413–30. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-662-04162-8_44.
Pełny tekst źródłaRosenblat, Joshua D., Ron Kakar i Roger S. McIntyre. "Metabolic-Inflammation Aspects of Depression and Cardiovascular Disease". W Cardiovascular Diseases and Depression, 211–33. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32480-7_14.
Pełny tekst źródłaLeonard, Brian E. "Depression, the Metabolic Syndrome and Neurodegeneration". W Current Topics in Neurotoxicity, 229–41. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-13602-8_11.
Pełny tekst źródłaN. Das, Undurti. "Neurochemical Linkage Among Metabolic Syndrome, Alzheimer Disease, and Depression". W Metabolic Syndrome and Neurological Disorders, 197–217. Chichester, UK: John Wiley & Sons Ltd, 2013. http://dx.doi.org/10.1002/9781118395318.ch11.
Pełny tekst źródłaOuthred, Tim, Claire McAulay, Danielle Gessler i Gin S. Malhi. "Monitoring for Metabolic Dysfunction and Cardiovascular Disease in Bipolar Disorder: A Shared Illness Process Approach". W Cardiovascular Diseases and Depression, 333–50. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32480-7_19.
Pełny tekst źródłaGoethe, John W., Bonnie L. Szarek i Charles F. Caley. "Metabolic Syndrome in Psychiatric Inpatients Treated for Depression". W Modern Trends in Pharmacopsychiatry, 90–104. Basel: KARGER, 2009. http://dx.doi.org/10.1159/000189777.
Pełny tekst źródłaStreszczenia konferencji na temat "Metabolic depression"
NIE, ZHI, TAO YANG, YASHU LIU, QINGYANG LI, VAIBHAV A. NARAYAN, GAYLE WITTENBERG i JIEPING YE. "MELANCHOLIC DEPRESSION PREDICTION BY IDENTIFYING REPRESENTATIVE FEATURES IN METABOLIC AND MICROARRAY PROFILES WITH MISSING VALUES". W Proceedings of the Pacific Symposium. WORLD SCIENTIFIC, 2014. http://dx.doi.org/10.1142/9789814644730_0043.
Pełny tekst źródłaZöllner, R., M. Bopp, P. Dietsche, H. Rekate, B. Dietsche, A. Krug, B. Hanewald, O. Steinsträter, J. Sommer i M. Zavorotnyy. "Structural and metabolic changes in the anterior cingulate cortex (ACC) after treatment with repetitive transcranial magnetic stimulation (rTMS) in patients with treatment-resistant unipolar depression (TRD)". W Abstracts of the 30th Symposium of the AGNP. Georg Thieme Verlag KG, 2017. http://dx.doi.org/10.1055/s-0037-1606408.
Pełny tekst źródłaKhoshnevis, Sepideh, Daniel W. Hensley i Kenneth R. Diller. "Measurement and Analysis of Cutaneous Perfusion Depression During Cryotherapy". W ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53853.
Pełny tekst źródłaChovancova, Olga, Jan Rabcan, Jozef Kostolny i Denisa Macekova. "Human Reliability Evaluation through Analysis of Depression Prediction based on Metabolomic Data". W 2019 10th International Conference on Dependable Systems, Services and Technologies (DESSERT). IEEE, 2019. http://dx.doi.org/10.1109/dessert.2019.8770036.
Pełny tekst źródłaXu, Jiale, Yingying Tang, C. Cecilio Baro, Xiaoliu Zhang, Ziyu Meng i Yao Li. "Left fimbria atrophy is associated with hippocampal metabolism in female major depressive disorder patients". W 2018 40th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2018. http://dx.doi.org/10.1109/embc.2018.8512472.
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