Gotowa bibliografia na temat „Melanoma”
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Artykuły w czasopismach na temat "Melanoma"
Panizzon i Guggisberg. "Clinical aspects and histopathology of melanoma". Therapeutische Umschau 56, nr 6 (1.06.1999): 302–8. http://dx.doi.org/10.1024/0040-5930.56.6.302.
Pełny tekst źródłaBarcala, Natália da Silva, Felipe Narracci, Gabriel Martins Perillo, Júlia Cauneto Banheti Corredato, Lilian Mariano Fonsatti, Francielli Elaine de Araújo Sampaio, Lorraine da Rosa Bueno i in. "MELANOMA: UMA ANÁLISE ABRANGENTE". Brazilian Journal of Implantology and Health Sciences 5, nr 5 (8.12.2023): 4856–69. http://dx.doi.org/10.36557/2674-8169.2023v5n5p4856-4869.
Pełny tekst źródłaAvello Canisto, Francisco, i Allan Avello Peragallo. "Melanoma en la región de cabeza y cuello." Anales de la Facultad de Medicina 76, nr 2 (10.07.2015): 187. http://dx.doi.org/10.15381/anales.v76i2.11146.
Pełny tekst źródłaRader, Ryan K., Katie S. Payne, Uday Guntupalli, Harold S. Rabinovitz, Maggie C. Oliviero, Rhett J. Drugge, Joseph J. Malters i William V. Stoecker. "The Pink Rim Sign: Location of Pink as an Indicator of Melanoma in Dermoscopic Images". Journal of Skin Cancer 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/719740.
Pełny tekst źródłaHansson, Johan, Mia Bergenmar, Per-Åke Hofer, Göran Lundell, Eva Månsson-Brahme, Ulrik Ringborg, Ingrid Synnerstad, Annika Ternesten Bratel, Ann-Marie Wennberg i Inger Rosdahl. "Monitoring of Kindreds With Hereditary Predisposition for Cutaneous Melanoma and Dysplastic Nevus Syndrome: Results of a Swedish Preventive Program". Journal of Clinical Oncology 25, nr 19 (1.07.2007): 2819–24. http://dx.doi.org/10.1200/jco.2007.11.4108.
Pełny tekst źródłaPalacios-Diaz, Rodolfo David, Blanca de Unamuno-Bustos, Carlos Abril-Pérez, Mónica Pozuelo-Ruiz, Javier Sánchez-Arraez, Ignacio Torres-Navarro i Rafael Botella-Estrada. "Multiple Primary Melanomas: Retrospective Review in a Tertiary Care Hospital". Journal of Clinical Medicine 11, nr 9 (22.04.2022): 2355. http://dx.doi.org/10.3390/jcm11092355.
Pełny tekst źródłaApostu, Adina Patricia, Loredana Ungureanu, Salomea Ruth Halmagyi, Ioana Irina Trufin i Simona Corina Șenilă. "Multiple primary melanomas: A literature review". Medicine 102, nr 30 (28.07.2023): e34378. http://dx.doi.org/10.1097/md.0000000000034378.
Pełny tekst źródłaBol, Kalijn Fredrike, Marco Donia, Steffen Heegaard, Jens Folke Kiilgaard i Inge Marie Svane. "Genetic Biomarkers in Melanoma of the Ocular Region: What the Medical Oncologist Should Know". International Journal of Molecular Sciences 21, nr 15 (23.07.2020): 5231. http://dx.doi.org/10.3390/ijms21155231.
Pełny tekst źródłaPapageorgiou, Chrysoula, Demetrios Ioannides, Zoe Apalla, Efstratios Vakirlis, Elisabeth Lazaridou, Eleni Sotiriou i Aimilios Lallas. "Dermoscopy of difficult-to-diagnose Melanomas". Serbian Journal of Dermatology and Venereology 8, nr 3 (1.09.2016): 121–27. http://dx.doi.org/10.1515/sjdv-2016-0011.
Pełny tekst źródłaDumaz, Nicolas, Fanélie Jouenne, Julie Delyon, Samia Mourah, Armand Bensussan i Céleste Lebbé. "Atypical BRAF and NRAS Mutations in Mucosal Melanoma". Cancers 11, nr 8 (8.08.2019): 1133. http://dx.doi.org/10.3390/cancers11081133.
Pełny tekst źródłaRozprawy doktorskie na temat "Melanoma"
Bolander, Åsa. "Prognostic factors in malignant melanoma /". Uppsala : Acta Universitatis Upsaliensis : Universitetsbiblioteket [distributör], 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9511.
Pełny tekst źródłaShort, Candice, i Ryan Short. "Uveal Melanoma". Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/etsu-works/7353.
Pełny tekst źródłaShort, Candice, i Ryan Short. "Uveal Melanoma". Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/etsu-works/7357.
Pełny tekst źródłaAliprandini, Eduardo. "Efeito da melanina e do oxigenio singlete na morte celular e fluxo de cálcio em células Melan-A e B16-F10". reponame:Repositório Institucional da UFPR, 2010. http://hdl.handle.net/1884/22552.
Pełny tekst źródłaDissertação (mestrado) - Universidade Federal do Paraná, Setor de Ciências Biológicas, Programa de Pós-Graduação em Bioquímica. Defesa: Curitiba, 12/02/2010
Bibliografia: 65-74
Área de concentração: Bioquímica
Resumo
Resumo: O melanoma e um tipo de cancer bastante relevante ja que as opcoes de tratamento eficazes sao limitadas. A presenca da melanina protege os individuos de pele escura contra os efeitos da radiacao solar, a principal causa de formacao do melanoma pela geracao de especies reativas de oxigenio (ROS). Porem, a melanina tambem pode ter um papel duplo, que e a de gerar especies reativas durante sua sintese que podem prejudicar a celula. Portanto, o objetivo deste trabalho foi a avaliacao das caracteristicas de morte celular causadas por uma ROS, o oxigenio singlete (1O2), nas celulas de melanoma murino B16-F10 com e sem estimulo para producao de melanina e nas celulas de melanocito murino Melan-a. O estimulo para a sintese de melanina foi obtido incubando-se as celulas por 48 horas com meio RPMI 1640 enriquecido com 400 ƒÊmol/L de L-tirosina e 10 mmol/L de cloreto de amonio. A concentracao de melanina aumentou em mais de nove vezes nas celulas B16-F10 e as celulas Melan-a tiveram aumento de menos de duas vezes. Foi utilizado o endoperoxido DHPNO2 10 mmol/L por 2 horas para a geracao de 1O2. Essa condicao causou queda na viabilidade avaliada pelo metodo do MTT para 78,0% nas celulas B16-F10, 70,2% nas B16-F10 estimuladas (B16-F10 Y) e 79,3% nas Melan-a. O ensaio foi feito apos 24 horas do inicio do tratamento com DHPNO2 e a viabilidade caiu para 49,7% nas B16-F10, 53,3% nas B16- F10 Y e 72,5% nas Melan-a. A avaliacao da morte celular utilizando laranja de acridina e brometo de etidio mostrou que apos o tratamento por 2 horas, somente as celulas B16- F10 tiveram aumento significativo na quantidade de celulas em apoptose, e as B16-F10 Y tiveram leve queda na quantidade de celulas viaveis, com tendencia ao aumento de celulas em apoptose. As celulas Melan-a nao tiveram diferenca entre os tratamentos. A liberacao do citocromo c foi determinada por HPLC e mostrou-se que apos 2 horas, as celulas B16-F10 tratadas com 1O2 tiveram mais citocromo c liberado para o citoplasma comparado com o controle. Nos demais grupos, nao houve alteracao com o tratamento. Porem, as celulas controle com mais melanina tiveram maior liberacao de citocromo comparado com o controle das celulas nao estimuladas, mostrando que as celulas estavam sofrendo algum dano inerente da sintese de melanina. A analise da fragmentacao do DNA apos 2 e 24 horas mostrou que nao houve aparecimento de quebras caracteristicas de apoptose pelo tratamento com 1O2 em nenhum dos grupos testados. As celulas B16-F10 Y controle apresentaram DNA fragmentado inespecificamente, representado como um arraste no gel de agarose, que nao foi alterado pelo tratamento. A razao entre ADP e ATP foi quantificada para avaliar o estado energetico da celula, que pode refletir algumas caracteristicas de morte. Nenhuma das celulas teve diferenca estatistica apos o tratamento de 2 horas com 1O2, mas foi observado que as razoes ADP/ATP das celulas controle B16-F10 com e sem estimulo apresentaram valores acima do valor considerado para celulas viaveis/proliferativas. Os resultados da celula Melan-a foram bem proximos dos valores ditos normais. O fluxo de calcio tambem foi avaliado e o 1O2 foi capaz de liberar calcio das reservas intracelulares para o citoplasma nas celulas B16-F10, sendo que nas celulas estimuladas, o aumento do calcio citoplasmatico foi menor, indicando a possivel recaptacao do calcio pela melanina. As celulas Melan-a nao sofreram grandes alteracoes na quantidade de calcio liberado para o citoplasma. Nao houve diferenca na liberacao de AIF em nenhuma das celulas. Em conjunto, os resultados mostram que a sintese da melanina estimulada pela suplementacao do meio foi deleteria as celulas, pois causou fragmentacao no DNA, liberacao de citocromo c e aumentou a razao ADP/ATP para valores considerados de celula em apoptose. Por outro lado, a presenca da melanina parece ter protegido as celulas da acao do 1O2, pois alguns resultados indicam uma tendencia de melhora dos parametros avaliados.
Abstract: Melanoma is a relevant type of cancer since the options for efficient treatment are limited. The presence of melanin protects the dark-skinned people against the effects of solar radiation, the main cause for melanoma development by the generation of reactive species of oxygen (ROS). However, melanin may also have a role on the generation of reactive species during its synthesis, which may harm the cell. So, the objective of this work was the evaluation of the characteristics of cell death caused by a ROS, the singlet oxygen (1O2) on murine melanoma cells B16-F10 with or without stimulation for synthesis of melanin and on murine melanocytes cells Melan-a. The stimulus for the synthesis of melanin was obtained treating the cells for 48 hours with medium RPMI 1640 supplemented with 400 ìmol/L of L-Tyrosine and 10 mmol/L of ammonium chloride. The concentration of melanin increased more than nine times in the B16-F10 cells and the Melan-a cells increase was almost twice the amount of the control. It was used the endoperoxide DHPNO2 10 mmol/L for 2 hours for the generation of 1O2. This condition caused the decrease in the viability determined by the method of MTT to 78% in B16-F10, 70,2% in B16-F10 that were stimulated (B16-F10 Y) and 79,3% in Melana cells. The test was performed after 24 hours from the beginning of the treatment with DHPNO2 and the viability decreased to 49,7% in B16-F10, 53,3% in B16-F10 Y and 72,5 % in Melan-a. The evaluation of cell death using acridine orange and ethidium bromide showed that after the two-hour treatment, only the B16-F10 cells had a significant increase of the number of apoptotic cells, and the B16-F10 Y cells had a slight decrease of the amount of viable cells, with the tendency of the increase of apoptotic cells. Melan-a cells did not show difference among the treatments. The release of cytochrome c was determined by HPLC and it showed that after two hours, B16-F10 cells had more cytochrome c released to the cytoplasm compared to the control. There was not any alteration for other groups of cells with the treatment. However, the control cells that had more melanin showed increased cytochrome c release compared to the control of the not-stimulated cells, demonstrating that the previous cells were suffering some kind of damage from the melanin synthesis. The analysis of DNA fragmentation revealed the absence of typical apoptosis fragmentation in any of the groups. B16-F10 Y control cells displayed unspecific DNA damage observed as a smear in the agarose gel, which was not altered by 1O2 treatment. The same result was observed after the treatment for 2 and 24 hours. The ratio between ADP and ATP was quantified to evaluate the energetic state of the cell, which may reflect some characteristics of cell death. None of the cells showed results statistically significant after the two-hour treatment 1O2, but it was shown that the values of ADP/ATP ratio of the B16-F10 control cells with and without stimulation were above of the threshold accepted for viable/proliferative cells. The results of Melan-a cells were very close to the values considered normal. The calcium flux was also evaluated and it was evidenced that 1O2 was capable of releasing calcium from the intracellular stores to the cytoplasm in B16- F10 cells, and the release of calcium was lower in B16-F10 Y, indicating the possibility of the binding of the metal to melanin. The Melan-a cells did not showed much increase in the quantities of calcium released to the cytoplasm. There was no difference in the release of AIF in any group. Over all, the results support that the synthesis of melanin that was stimulated by the supplementation of the medium was deleterious to the cells, since it caused DNA fragmentation, release of cytochrome c and increase of the ratio ADP/ATP to values of cells in apoptosis. On the other hand, the presence of melanin seemed to protect the cells against the action of 1O2, because some results indicate a tendency of improvement in the parameters evaluated.
Kohli, Jaskaren Singh. "Senescence and immortalisation in melanoma progression and multiple primary melanoma". Thesis, St George's, University of London, 2016. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.706529.
Pełny tekst źródłaWilson, Kenneth Scott. "Cutaneous malignant melanoma". Thesis, University of Edinburgh, 1994. http://hdl.handle.net/1842/27062.
Pełny tekst źródłaWiguna, Arlina Permatasari. "Immunoregulation in melanoma". Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2015. http://dx.doi.org/10.18452/17107.
Pełny tekst źródłaIL-10 and TGF-beta are immunosuppressive cytokines expressed in tumors including melanoma and, therefore, deemed major cause for failing anti-tumor immune responses. To re-evaluate their role, their expression was compared by quantitative RT-PCR in melanoma and skin of healthy individuals, their induction in dendritic cells and T cells co-cultured with tumor cells, and their effects on the immune cells were tested. Both cytokines as well as their receptors were expressed in melanoma at significantly lower levels than in healthy skin. Consequently, the expressions of IL-10-responsive SOCS-3 and TGF-beta-responsive Smad-7 were low in tumors but high in healthy skin. T cells co-cultured with tumor cells developed an anergic state but without increased IL-10 or TGF-beta expression. In vitro tumor-associated iDCs produced high IL-10 levels and were less efficient in inducing T cell proliferation. Nonetheless, they could be induced to mature, and blocking IL-10 did not alter the capacity of the resulting mDCs to induce T cell proliferation. mDCs co-cultured with tumor cells produced increased IL-10 but similar or decreased TGF-beta level and were more efficient in inducing T cell proliferation. The lack of correlation of IL-10 and TGF-beta with immune deficits in situ and in vitro suggests a necessity of re-evaluating their roles in cancer.
Winklmeier, Andreas. "Rolle der Transkription und Aktivität von MIA ("Melanoma Inhibitory Activity") im malignen Melanom". kostenfrei, 2009. http://www.opus-bayern.de/uni-regensburg/volltexte/2009/1301/.
Pełny tekst źródłaHering, Kathrin, Anke Bresch, Donald Lobsien, Wolf Müller, Rolf-Dieter Kortmann i Clemens Seidel. "Primary intradural extramedullary spinal melanoma in the lower thoracic spine". Universitätsbibliothek Leipzig, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-205904.
Pełny tekst źródłaGilbert, Amy. "Humoral immune response to melanoma : discovery and evaluation of anti-melanoma antibodies". Thesis, King's College London (University of London), 2013. https://kclpure.kcl.ac.uk/portal/en/theses/humoral-immune-response-to-melanoma-discovery-and-evaluation-of-antimelanoma-antibodies(34c0be73-e94a-4d3f-92b9-8888178c4cd2).html.
Pełny tekst źródłaKsiążki na temat "Melanoma"
Hargadon, Kristian M., red. Melanoma. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1205-7.
Pełny tekst źródłaRiker, Adam I., red. Melanoma. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78310-9.
Pełny tekst źródłaFisher, David E., i Boris C. Bastian, red. Melanoma. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4614-7322-0.
Pełny tekst źródłaKaufman, Howard L., i Janice M. Mehnert, red. Melanoma. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-22539-5.
Pełny tekst źródłaBorden, Ernest C., red. Melanoma. Totowa, NJ: Humana Press, 2002. http://dx.doi.org/10.1007/978-1-59259-159-6.
Pełny tekst źródłaM, Mackie Rona, i British Council, red. Melanoma. Edinburgh: Published on behalf of the British Council by Churchill Livingstone, 1995.
Znajdź pełny tekst źródłaname, No. Melanoma. Totowa: Humana Press, Inc, 2002.
Znajdź pełny tekst źródłaNational Cancer Institute (U.S.), red. Melanoma. [Bethesda, MD]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1992.
Znajdź pełny tekst źródłaNational Cancer Institute (U.S.), red. Melanoma. [Bethesda, MD]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1992.
Znajdź pełny tekst źródłaDickie, Lauren E. Melanoma. Bethesda, Md: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1988.
Znajdź pełny tekst źródłaCzęści książek na temat "Melanoma"
Bährle-Rapp, Marina. "Melanom, auch: Melanoma". W Springer Lexikon Kosmetik und Körperpflege, 345–46. Berlin, Heidelberg: Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-71095-0_6403.
Pełny tekst źródłaRuiz-Llorente, Lidia, María Jesús Ruiz-Rodríguez, Claudia Savini, Teresa González-Muñoz, Erica Riveiro-Falkenbach, José L. Rodríguez-Peralto, Héctor Peinado i Carmelo Bernabeu. "Correlation Between Endoglin and Malignant Phenotype in Human Melanoma Cells: Analysis of hsa-mir-214 and hsa-mir-370 in Cells and Their Extracellular Vesicles". W Advances in Experimental Medicine and Biology, 253–72. Cham: Springer Nature Switzerland, 2023. http://dx.doi.org/10.1007/978-3-031-26163-3_14.
Pełny tekst źródłaThompson, John F., Richard Kefford, Graham Stevens i Richard Scolyer. "History of Melanoma". W Melanoma, 1–13. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78310-9_1.
Pełny tekst źródłaElmslie, Paul. "International Models of Melanoma Management (Australia)". W Melanoma, 159–76. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78310-9_10.
Pełny tekst źródłaWassef, Cindy, Lea J. Bellomo i David I. Silverstein. "Pigmented Lesions: Biopsy Methods and Emerging Non-invasive Imaging Techniques". W Melanoma, 177–91. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78310-9_11.
Pełny tekst źródłaMessina, Jane L., i Rahel A. John. "The Pathology of Melanoma". W Melanoma, 193–212. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78310-9_12.
Pełny tekst źródłaKumar, Radhika Sreeraman, Jane L. Messina, Damon R. Reed i Vernon K. Sondak. "Pediatric Melanoma and Atypical Melanocytic Neoplasms". W Melanoma, 213–37. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78310-9_13.
Pełny tekst źródłaWalker, Joanna L., Annie Wang, George Kroumpouzos i Martin A. Weinstock. "Melanoma in Pregnancy". W Melanoma, 239–52. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78310-9_14.
Pełny tekst źródłaMikkelsen, Lauge Hjorth, i Steffen Heegaard. "Mucosal Melanoma". W Melanoma, 253–72. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78310-9_15.
Pełny tekst źródłaMilam, Ronald W., i Anthony B. Daniels. "Uveal Melanoma". W Melanoma, 273–312. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-78310-9_16.
Pełny tekst źródłaStreszczenia konferencji na temat "Melanoma"
Marijanović, Inga, Teo Buhovac i Gordana Berić Jović. "KEMOTERAPIJA U LIJEČENJU METASTATSKOG MELANOMA". W Okrugli sto s međunarodnim učešćem "Melanom". Akademija nauka i umjetnosti Bosne i Hercegovine, 2018. http://dx.doi.org/10.5644/pi2019.180.03.
Pełny tekst źródłaŠimić, Dubravka, Maja Vujica, Jasna Zeljko Penavić, Marina Prlić i Anita Gunarić. "POVEZANOST ČIMBENIKA RIZIKA I UČESTALOSTI OBOLIJEVANJA OD MELANOMA KOŽE". W Okrugli sto s međunarodnim učešćem "Melanom". Akademija nauka i umjetnosti Bosne i Hercegovine, 2018. http://dx.doi.org/10.5644/pi2019.180.01.
Pełny tekst źródłaFočo, Faris, Aiša Rašiti i Salih Saračević. "MELANOMI MAKSILOFACIJALNE REGIJE – EPIDEMIOLOŠKE I KLINIČKE KARAKTERISTIKE PACIJENATA S HIRURŠKI TRETIRANIM MELANOMOM". W Okrugli sto s međunarodnim učešćem "Melanom". Akademija nauka i umjetnosti Bosne i Hercegovine, 2018. http://dx.doi.org/10.5644/pi2019.180.05.
Pełny tekst źródłaSachett, Mariana Linhares, GIANCARLLO DANEZI FELIN, GIULLIANO DANEZI FELIN, CAROLLINA DANEZI FELIN i FELLIPE DANEZI FELIN. "GENÉTICA MOLECULAR DO MELANOMA MALÍGNO: UM ALVO TERAPÊUTICO". W I Congresso Nacional de Pesquisas e Estudos Genéticos On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/geneticon/9012.
Pełny tekst źródłaM, Janani, i Gershom King Rose J. k. "Highly Accurate Deep Learning PNN Classifier Based Skin Cancer and Depth Analysis". W The International Conference on scientific innovations in Science, Technology, and Management. International Journal of Advanced Trends in Engineering and Management, 2023. http://dx.doi.org/10.59544/iykv7105/ngcesi23p39.
Pełny tekst źródłaPessanha, Gabriel R. G., i Eleanderson Campos. "Um Modelo de Inteligência Artificial Para Detecção de Melanoma via Redes Neurais Convolucionais". W Escola Regional de Computação Aplicada à Saúde. Sociedade Brasileira de Computação - SBC, 2021. http://dx.doi.org/10.5753/ercas.2021.17436.
Pełny tekst źródłaAlamdari, Nasim, Nicholas MacKinnon, Fartash Vasefi, Reza Fazel-Rezai, Minhal Alhashim, Alireza Akhbardeh, Daniel L. Farkas i Kouhyar Tavakolian. "Effect of Lesion Segmentation in Melanoma Diagnosis for a Mobile Health Application". W 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3522.
Pełny tekst źródłaHuang, Xiyong, Michael D. Protheroe, Ahmed M. Al-Jumaily i Sharad P. Paul. "The Significance of Hair Thermal Diffusivity on Melanoma Incidence". W ASME 2017 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/imece2017-71693.
Pełny tekst źródłaRoss, Maria Laura da Rosa Dal, Vinícius Rosa Dos Santos, Luísa Sant Anna Blaskoski Cardoso i Julia Da Costa Cunha. "MELANOMA AMELANÓTICO EM FELINO: RELATO DE CASO". W I Congresso On-line Nacional de Clínica Veterinária de Pequenos Animais. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1933.
Pełny tekst źródłaOliveira, Viviane Marques de, Thays M. Brandão, Samantha Rocha i Moara Rocha. "MELANOMA PALPEBRAL EM CÃES: UMA REVISÃO BIBLIOGRÁFICA". W I Congresso On-line Nacional de Clínica Veterinária de Pequenos Animais. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/1907.
Pełny tekst źródłaRaporty organizacyjne na temat "Melanoma"
Quinn, Thomas, i Herbert Moore. Targeted Therapy for Melanoma. Office of Scientific and Technical Information (OSTI), grudzień 2016. http://dx.doi.org/10.2172/1334319.
Pełny tekst źródłaMarchetti, Dario. Heparanase Mechanisms in Melanoma Brain Metastasis. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2014. http://dx.doi.org/10.21236/ada613639.
Pełny tekst źródłaShy, C. M., H. Checkoway i E. G. Marshall. Malignant melanoma at a scientific laboratory. Office of Scientific and Technical Information (OSTI), listopad 1985. http://dx.doi.org/10.2172/6041000.
Pełny tekst źródłaMarchetti, Dario. Heparanase Mechanisms in Melanoma Brain Metastasis. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2013. http://dx.doi.org/10.21236/ada598581.
Pełny tekst źródłaLibermann, Towia. Testing New Drugs for Treatment of Melanoma Patients Applying Connectivity Map Database Analysis with Melanoma Gene Signatures. Fort Belvoir, VA: Defense Technical Information Center, październik 2012. http://dx.doi.org/10.21236/ada592898.
Pełny tekst źródłaWu, Xu. Targeting Palmitoyl Acyltransferases in Mutant NRAS-Driven Melanoma. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2014. http://dx.doi.org/10.21236/ada613837.
Pełny tekst źródłaBiggs, M. W., i J. E. Eiselein. Immunotherapy of metastatic melanoma by reversal of immune suppression. Office of Scientific and Technical Information (OSTI), styczeń 1997. http://dx.doi.org/10.2172/454026.
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