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Artykuły w czasopismach na temat "Meiosis"
Zhang, Qian, Wenzhe Zhang, Xinyi Wu, Hanni Ke, Yingying Qin, Shidou Zhao i Ting Guo. "Homozygous missense variant in MEIOSIN causes premature ovarian insufficiency". Human Reproduction 38, Supplement_2 (1.11.2023): ii47—ii56. http://dx.doi.org/10.1093/humrep/dead084.
Pełny tekst źródłaHasenkampf, C. A., A. A. Taylor, N. U. Siddiqui i C. D. Riggs. "meiotin-1 gene expression in normal anthers and in anthers exhibiting prematurely condensed chromosomes". Genome 43, nr 4 (1.08.2000): 604–12. http://dx.doi.org/10.1139/g00-021.
Pełny tekst źródłaGoldway, M., A. Sherman, D. Zenvirth, T. Arbel i G. Simchen. "A short chromosomal region with major roles in yeast chromosome III meiotic disjunction, recombination and double strand breaks." Genetics 133, nr 2 (1.02.1993): 159–69. http://dx.doi.org/10.1093/genetics/133.2.159.
Pełny tekst źródłaRoss, Lyle O., Susannah Rankin, Michèle F. Shuster i Dean S. Dawson. "Effects of Homology, Size and Exchange on the Meiotic Segregation of Model Chromosomes in Saccharomyces cerevisiae". Genetics 142, nr 1 (1.01.1996): 79–89. http://dx.doi.org/10.1093/genetics/142.1.79.
Pełny tekst źródłaSun, H., D. Dawson i J. W. Szostak. "Genetic and physical analyses of sister chromatid exchange in yeast meiosis". Molecular and Cellular Biology 11, nr 12 (grudzień 1991): 6328–36. http://dx.doi.org/10.1128/mcb.11.12.6328-6336.1991.
Pełny tekst źródłaSun, H., D. Dawson i J. W. Szostak. "Genetic and physical analyses of sister chromatid exchange in yeast meiosis." Molecular and Cellular Biology 11, nr 12 (grudzień 1991): 6328–36. http://dx.doi.org/10.1128/mcb.11.12.6328.
Pełny tekst źródłaUranishi, Kousuke, Masataka Hirasaki, Yuka Kitamura, Yosuke Mizuno, Masazumi Nishimoto, Ayumu Suzuki i Akihiko Okuda. "Two DNA Binding Domains of MGA Act in Combination to Suppress Ectopic Activation of Meiosis-Related Genes in Mouse Embryonic Stem Cells". Stem Cells 39, nr 11 (14.07.2021): 1435–46. http://dx.doi.org/10.1002/stem.3433.
Pełny tekst źródłaTsuchiya, Dai, Claire Gonzalez i Soni Lacefield. "The spindle checkpoint protein Mad2 regulates APC/C activity during prometaphase and metaphase of meiosis I in Saccharomyces cerevisiae". Molecular Biology of the Cell 22, nr 16 (15.08.2011): 2848–61. http://dx.doi.org/10.1091/mbc.e11-04-0378.
Pełny tekst źródłaPage, A. W., i T. L. Orr-Weaver. "The Drosophila genes grauzone and cortex are necessary for proper female meiosis". Journal of Cell Science 109, nr 7 (1.07.1996): 1707–15. http://dx.doi.org/10.1242/jcs.109.7.1707.
Pełny tekst źródłaPaliulis, Leocadia V., i R. Bruce Nicklas. "The Reduction of Chromosome Number in Meiosis Is Determined by Properties Built into the Chromosomes". Journal of Cell Biology 150, nr 6 (18.09.2000): 1223–32. http://dx.doi.org/10.1083/jcb.150.6.1223.
Pełny tekst źródłaRozprawy doktorskie na temat "Meiosis"
Canales, C. "Characterisation of extra sporogenous cells (ESP) : an avbidopsis gene required for another development". Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365861.
Pełny tekst źródłaPhizicky, David V. (David Vincent). "Mechanisms preventing DNA replication between Meiosis I and Meiosis II". Thesis, Massachusetts Institute of Technology, 2018. http://hdl.handle.net/1721.1/117786.
Pełny tekst źródłaThis electronic version was submitted by the student author. The certified thesis is available in the Institute Archives and Special Collections.
Cataloged student-submitted from PDF version of thesis.
Includes bibliographical references.
The vast majority of multicellular organisms reproduce using sexual reproduction, which requires the production of haploid gametes. These gametes are produced by meiosis, a specialized cell division during which one round of DNA replication is followed by two rounds of chromosome segregation, Meiosis I (MI) and Meiosis II (MII). This imbalance between rounds of DNA replication and chromosome segregation causes diploid cells to produce haploid gametes. In contrast, mitotically-dividing cells maintain ploidy by alternating between rounds of replication and segregation. It is unclear how meiosis accomplishes two sequential chromosome segregation events without an intervening round of DNA replication. In mitotic cells, both DNA replication and chromosome segregation are regulated by oscillations of cyclin-dependent kinase (CDK) activity. Both events initiate during G1 due to the associated low CDK-activity state, and both events are completed later in the cell cycle due to increased CDK activity. During meiosis, uncoupling replication and segregation presents a unique problem. After completion of MI, CDK activity decreases and then increases to drive MII chromosome segregation. However, DNA replication must remain inhibited between MI and MII. Given that an oscillation of CDK activity is sufficient for genome re-duplication in mitotic cells, I sought to understand how meiotic cells prevent DNA replication while resetting the chromosome segregation program. In this thesis, I show that meiotic cells inhibit two distinct steps of DNA replication: (1) loading of the replicative helicase onto replication origins, and (2) activation of the replicative helicase. CDK and the meiosis-specific kinase Ime2 cooperatively inhibit helicase loading during the meiotic divisions, and their simultaneous inhibition causes inappropriate helicase reloading. Further studies of Ime2 revealed two mechanisms by which it inhibits this process. First, I showed that Ime2-phosphorylation of the helicase directly inhibits its loading onto origins. Second, Ime2 cooperated with CDK to transcriptionally and proteolytically repress Cdc6, an essential helicase-loading protein. In addition, I found that meiotic cells use CDK and the polo-like kinase Cdc5 to promote degradation of Sld2, an essential helicase-activation protein. Together, these data demonstrate that multiple kinases inhibit both helicase loading and activation between MI and MII, thereby ensuring a reduction in ploidy.
by David V. Phizicky.
Ph. D.
Marcet, Ortega Marina. "Surveillance mechanisms in mammalian meiosis". Doctoral thesis, Universitat Autònoma de Barcelona, 2016. http://hdl.handle.net/10803/387429.
Pełny tekst źródłaIn order to protect germinal cells from genomic instability, surveillance mechanisms ensure that meiosis occurs properly. In mammals, spermatocytes that display recombination or sex body defects experience an arrest at pachytene stage. Previous studies from our lab described that the MRE11 complex-ATM-CHK2 pathway activates the recombination-dependent arrest in the presence of unrepaired double strand breaks (DSBs). In this work we aimed to identify if p53 family members, which are putative targets of ATM and CHK2, participate in the activation of the recombination-dependent arrest. As a genetic approach, we bred double mutant mice carrying a mutation of a member of the p53 family (p53, TAp63, p73) in a Trip13 defective background. Trip13 mutation causes recombination defects, which activate the recombination-dependent arrest in pachytene-stage spermatocytes. Thus, we studied how the absence of p53 family members affected the arrest phenotype of Trip13mod/mod spermatocytes. Our data showed that p53 and TAp63 deficiency, but not p73, allowed spermatocytes to progress further into late pachynema, despite accumulating numerous unrepaired DBSs. In addition, lack of p53 or TAp63 resulted in a decrease of apoptotic spermatocytes at early pachytene stage. Therefore, our results indicate that p53 and TAp63 are responsible to activate the recombination-dependent arrest in mouse spermatocytes. Even though, double mutant spermatocytes still arrested at pachytene stage. To study if double mutant spermatocytes were arresting due to the activation of the sex body deficient arrest we analyzed MSCI functionality in Trip13 mutants. Thus, by bypassing the recombination-dependent arrest has allowed us to elucidate a role for TRIP13 protein in meiotic silencing, which consequently triggers apoptosis in double mutants at late pachytene stage due to sex body impairment. These results infer that the recombination-dependent and the sex-body deficient arrest are activated by two genetically separated mechanisms. From the observation that TRIP13 is required to implement MSCI silencing, we performed an exhaustive analysis of transcription in Trip13 mutants. Our results suggested that RNA expression in Trip13 mutants was increased in early meiotic stage spermatocytes, assessed by EU-labeling RNA and phosphorylated(S2)-RNA polymerase II. Moreover, RNA sequencing data highlighted the observation that sex chromosome genes and pre-meiotic genes are overexpressed in Trip13 mutants, suggesting that TRIP13 is required to maintain the expression of these genes at low levels. Overall, the data presented in this work contributes to the understanding on how surveillance mechanisms control several crucial steps of meiotic prophase progression in mammalian spermatocytes.
Fabig, Gunar. "Dynamic and ultrastructural characterization of chromosome segregation in C. elegans male meiosis". Technische Universität Dresden, 2018. https://tud.qucosa.de/id/qucosa%3A32727.
Pełny tekst źródłaConnor, Colette. "Investigating the role of Cdc14 in the regulation of the meiosis I to meiosis II transition". Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/21086.
Pełny tekst źródłaIgea, Fernández Ana. "CPEB4 replaces CPEB1 to complete meiosis". Doctoral thesis, Universitat Pompeu Fabra, 2009. http://hdl.handle.net/10803/22687.
Pełny tekst źródłaÇetin, Bülent. "Chromosome segregation in mitosis and meiosis". Thesis, University of Oxford, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.669990.
Pełny tekst źródłaRattani, Ahmed Anwer Ali. "Regulation of anaphase in mammalian meiosis". Thesis, University of Oxford, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.639733.
Pełny tekst źródłaWinters, Tristan. "The role of STAG3 in mammalian meiosis". Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2018. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-233399.
Pełny tekst źródłaFernandes, Joiselle Blanche. "Identification et caractérisation fonctionnelle de gènes contrôlant la fréquence de crossovers méiotiques". Thesis, Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLS303/document.
Pełny tekst źródłaMeiotic crossovers (CO) are formed by reciprocal exchange of genetic material between the homologous chromosomes. CO generate genetic diversity and are essential for the proper segregation of chromosomes during meiosis in most eukaryotes. Despite their significance and a large excess of CO precursors, CO number is very low in vast majority of species (typically one to three per chromosome pair). This indicates that COs are tightly regulated but the underlying mechanisms of this limit remain elusive. In order to identify genes that limit COs, a genetic screen was performed in Arabidopsis thaliana. This led to the identification and characterization of several anti-CO factors belonging to three different pathways: (i) The FANCM helicase and its cofactors (ii) The AAA-ATPase FIDGETIN-LIKE-1 (FIGL1) (iii) The RECQ4 -Topoisomerase 3α-RMI1 complex. The first objective was to understand the functional relationship between these three pathways and to address following questions: (1) how far can we increase recombination when combining mutations in FANCM, FIGL1 and RECQ4? We show that the highest increase in recombination was obtained in figl1 recq4, reaching to 7.5 fold the wild type level, on average genome wide. (2) How is the distribution of recombination events genome wide in mutants? The increased CO frequency in the mutants was not uniform throughout the genome. CO frequency rises from the centromere to telomeres, with distal intervals having highest COs (3) is the recombination frequency increase same in both male and female? In Arabidopsis wild type, male has higher recombination than female meiosis. In contrast, in recq4 and recq4 figl1, female recombination was higher than male. This suggests that certain constraints that apply to CO formation in wild type females are relieved in the mutant. By continuing the same genetic screen, a novel anti-CO mutant was identified. The second objective was to identify and functionally characterize the corresponding gene. Genetic mapping and protein interaction studies led to the identification of a factor that directly interacts with FIGL1 and appears to form a conserved complex both in Arabidopsis and humans. Hence, the factor was named FLIP (Fidgetin-like-1 interacting protein). Recombination frequency is increased in flip, confirming that FLIP limit COs. Epistasis studies showed that FLIP and FIGL1 act in same pathway. Further, FIGL1/FLIP proteins of Arabidopsis and humans directly interact with the recombinases RAD51 and DMC1 which catalyze a crucial step of homologous recombination, the inter homolog strand invasion. In addition flip like figl1 modifies dynamics of DMC1. We thus propose a model wherein the FLIP-FIGL1 complex negatively regulates RAD51/DMC1 to limit CO formation. Studying the conserved FIGL1-FLIP complex led to the identification of a novel mode of regulation of recombination, that likely acts at the key step of homologous strand invasion. Further the unprecedented level of CO increase in recq4figl1 in hybrids could be of great interest for crop improvement, allowing the production of novel allele combinations
Książki na temat "Meiosis"
B, Moens Peter, red. Meiosis. Orlando: Academic Press, 1987.
Znajdź pełny tekst źródłaJohn, Bernard. Meiosis. Cambridge [England]: Cambridge University Press, 1990.
Znajdź pełny tekst źródłaKeeney, Scott, red. Meiosis. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-527-5.
Pełny tekst źródłaStuart, David T., red. Meiosis. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6340-9.
Pełny tekst źródłaKeeney, Scott, red. Meiosis. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60761-103-5.
Pełny tekst źródłaRicardo, Benavente, i Volff Jean-Nicolas, red. Meiosis. Basel: Karger, 2009.
Znajdź pełny tekst źródłaMeiosis. Dordrecht: Humana Press, 2009.
Znajdź pełny tekst źródłaJohn, B. Meiosis. Cambridge: Cambridge University Press, 1990.
Znajdź pełny tekst źródłaPawlowski, Wojciech P., Mathilde Grelon i Susan Armstrong, red. Plant Meiosis. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-333-6.
Pełny tekst źródłaPradillo, Mónica, i Stefan Heckmann, red. Plant Meiosis. New York, NY: Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4939-9818-0.
Pełny tekst źródłaCzęści książek na temat "Meiosis"
Pryce, D. W., i R. J. McFarlane. "The Meiotic Recombination Hotspots of Schizosaccharomyces pombe". W Meiosis, 1–13. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166614.
Pełny tekst źródłaMuyt, A. De, R. Mercier, C. Mézard i M. Grelon. "Meiotic Recombination and Crossovers in Plants". W Meiosis, 14–25. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166616.
Pełny tekst źródłaMartinez-Perez, E. "Meiosis in Cereal Crops: the Grasses are Back". W Meiosis, 26–42. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166617.
Pełny tekst źródłaZetka, M. "Homologue Pairing, Recombination and Segregation in Caenorhabditis elegans". W Meiosis, 43–55. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166618.
Pełny tekst źródłaMcKee, B. D. "Homolog Pairing and Segregation in Drosophila Meiosis". W Meiosis, 56–68. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166619.
Pełny tekst źródłaYang, F., i P. J. Wang. "The Mammalian Synaptonemal Complex: A Scaffold and Beyond". W Meiosis, 69–80. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166620.
Pełny tekst źródłaAlsheimer, M. "The Dance Floor of Meiosis: Evolutionary Conservation of Nuclear Envelope Attachment and Dynamics of Meiotic Telomeres". W Meiosis, 81–93. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166621.
Pełny tekst źródłaSuja, J. A., i J. L. Barbero. "Cohesin Complexes and Sister Chromatid Cohesion in Mammalian Meiosis". W Meiosis, 94–116. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166622.
Pełny tekst źródłaKhil, P. P., i R. D. Camerini-Otero. "Variation in Patterns of Human Meiotic Recombination". W Meiosis, 117–27. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166623.
Pełny tekst źródłaGarcia-Cruz, R., I. Roig i M. Garcia Caldés. "Maternal Origin of the Human Aneuploidies. Are Homolog Synapsis and Recombination to Blame? Notes (Learned) from the Underbelly". W Meiosis, 128–36. Basel: KARGER, 2008. http://dx.doi.org/10.1159/000166638.
Pełny tekst źródłaStreszczenia konferencji na temat "Meiosis"
Wiriyasermkul, Nattavut, Veera Boobjing i Pisit Chanvarasuth. "A Meiosis Genetic Algorithm". W 2010 Seventh International Conference on Information Technology: New Generations. IEEE, 2010. http://dx.doi.org/10.1109/itng.2010.152.
Pełny tekst źródła"Chromatin and cytoskeleton reorganization in meiosis of wheat-rye substitution line (3R3B)". W Plant Genetics, Genomics, Bioinformatics, and Biotechnology. Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, 2019. http://dx.doi.org/10.18699/plantgen2019-215.
Pełny tekst źródła"3D-microscopy of prophase nucleus in the meiosis I of wheat-rye amphihaploids". W Plant Genetics, Genomics, Bioinformatics, and Biotechnology. Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, 2019. http://dx.doi.org/10.18699/plantgen2019-106.
Pełny tekst źródłaManaka, Thapelo, i Lydia Mavuru. "TEACHERS’ PREPAREDNESS AND EXPERIENCES IN TEACHING MEIOSIS TO GRADE 12 LEARNERS IN LIMPOPO PROVINCE". W International Conference on Education and New Developments 2020. inScience Press, 2020. http://dx.doi.org/10.36315/2020end032.
Pełny tekst źródłaXiaoli Yang, Rong Ge, Yufei Yang, Hao Shen, Yingjie Li i C. C. Tseng. "Interactive computer program for learning genetic principles of segregation and independent assortment through meiosis". W 2009 Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2009. http://dx.doi.org/10.1109/iembs.2009.5334401.
Pełny tekst źródłaGabitova, Linara, Andrey Gorin, Diana Restifo, Dong-Hua Yang, David Cunningham, Gail E. Herman i Igor A. Astsaturov. "Abstract 2448: Meiosis activating sterols counteract KRas-driven epithelial carcinogenesis via an LXR-dependent mechanism". W Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-2448.
Pełny tekst źródła"Retention and evolution of centromeric histone paralogs in rye: essential for the organization of chromosomes in rye meiosis". W Plant Genetics, Genomics, Bioinformatics, and Biotechnology (PlantGen2023). FRC Kazan Scientific Center RAS, Kazan, Russia;Institute of Cytology and Genetics SB RAS, Novosibirsk, Russia, 2023. http://dx.doi.org/10.18699/plantgen2023-21.
Pełny tekst źródłaStuart, Maritza Regina, Patricia Maria Raulilno de Campos, Luíz Rafaelli Coutinho, Vania Ana Silveira Muniz, Daniela Soldera i Ana Paula Madalena da Silva. "The role of the nurse in the care of children with down syndrome". W III SEVEN INTERNATIONAL MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/seveniiimulti2023-228.
Pełny tekst źródłaGuthrie, Megan, Moli Williams, Ajay Valji, Robert Jones, Dale Vimalachandran, Daniel Palmer, Tim Cross i Urszula McClurg. "P46 Meiotic proteins – liver cancer specific drug targets". W Abstracts of the British Association for the Study of the Liver Annual Meeting, 19–22 September 2023. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2023. http://dx.doi.org/10.1136/gutjnl-2023-basl.62.
Pełny tekst źródłaStauffer, Weston T., Liangyu Zhang i Abby Dernburg. "Diffusion through a liquid crystalline compartment regulates meiotic recombination". W Biophysics, Biology and Biophotonics IV: the Crossroads, redaktorzy Adam Wax i Vadim Backman. SPIE, 2019. http://dx.doi.org/10.1117/12.2513378.
Pełny tekst źródłaRaporty organizacyjne na temat "Meiosis"
Durham, Mary. Demonstrating Meiosis Using Manipulatable Chromosomes and Cells. Genetics Society of America Peer-Reviewed Education Portal (GSA PREP), maj 2015. http://dx.doi.org/10.1534/gsaprep.2015.002.
Pełny tekst źródłaSingh, Anjali. Estimating the Chiasma Frequency in Diplotene-Diakinesis Stage. ConductScience, wrzesień 2020. http://dx.doi.org/10.55157/cs20200925.
Pełny tekst źródłaLevy, Avraham, Clifford Weil i Wojtek Pawlowski. Enhancing the Rate of Meiotic Crossing-Over for Plant Breeding. United States Department of Agriculture, styczeń 2009. http://dx.doi.org/10.32747/2009.7696532.bard.
Pełny tekst źródłaPawlowski, Wojtek P., i Avraham A. Levy. What shapes the crossover landscape in maize and wheat and how can we modify it. United States Department of Agriculture, styczeń 2015. http://dx.doi.org/10.32747/2015.7600025.bard.
Pełny tekst źródłaHood-DeGrenier, Jennifer. Active Learning Workshops for Teaching Key Topics in Introductory Cell and Molecular Biology: Structure of DNA/RNA, Structure of Proteins, and Cell Division via Mitosis and Meiosis. Genetics Society of America Peer-Reviewed Education Portal (GSA PREP), grudzień 2015. http://dx.doi.org/10.1534/gsaprep.2015.004.
Pełny tekst źródłaGregory p. Copenhaver. Regulation of Meiotic Recombination. Office of Scientific and Technical Information (OSTI), listopad 2011. http://dx.doi.org/10.2172/1028811.
Pełny tekst źródłaDray, Eloise, Myun Hwa Dunlop, Liisa Kauppi, Joseph San San Filippo, Claudia Wiese, Miaw-Sheue Tsai, Sead Begovic i in. Molecular Basis for Enhancement of the Meiotic DMCI Recombinase by RAD51AP1. Office of Scientific and Technical Information (OSTI), listopad 2010. http://dx.doi.org/10.2172/1011037.
Pełny tekst źródłaIzhar, Shamay, Maureen Hanson i Nurit Firon. Expression of the Mitochondrial Locus Associated with Cytoplasmic Male Sterility in Petunia. United States Department of Agriculture, luty 1996. http://dx.doi.org/10.32747/1996.7604933.bard.
Pełny tekst źródłaArnaoudova, Yanina, Boyan Arnaoudov i Nasya Tomlekova. Meiotic Behaviour of Pollen Mother Cells in Eight Genotypes of Pepper ( Capsicum annuum L.) under Water Deficit. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, listopad 2021. http://dx.doi.org/10.7546/crabs.2021.11.18.
Pełny tekst źródłaHirota, Marina, Carlos A. Nobre, Ane Alencar, Julia Areiera, Francisco de Assis Costa, Bernardo Flores, Clarissa Gandour i in. Policy Brief: Um Chamado de Ação Global para Evitar os ‘Pontos de Não-Retorno da Floresta Amazônica. Sustainable Development Solutions Network (SDSN), listopad 2022. http://dx.doi.org/10.55161/wmsa6060.
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