Gotowa bibliografia na temat „Medical Biochemistry: Lipids”
Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych
Zobacz listy aktualnych artykułów, książek, rozpraw, streszczeń i innych źródeł naukowych na temat „Medical Biochemistry: Lipids”.
Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.
Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.
Artykuły w czasopismach na temat "Medical Biochemistry: Lipids"
Honek, John F. "Glyoxalase biochemistry". Biomolecular Concepts 6, nr 5-6 (1.12.2015): 401–14. http://dx.doi.org/10.1515/bmc-2015-0025.
Pełny tekst źródłaOostendorp, Marlies, Udo FH Engelke, Michèl AAP Willemsen i Ron A. Wevers. "Diagnosing Inborn Errors of Lipid Metabolism with Proton Nuclear Magnetic Resonance Spectroscopy". Clinical Chemistry 52, nr 7 (1.07.2006): 1395–405. http://dx.doi.org/10.1373/clinchem.2006.069112.
Pełny tekst źródłaAHSAN, HASEEB. "Clinical Chemistry and Biochemistry: The Role of Biomarkers and Biomolecules". Asian Journal of Science Education 4, nr 1 (22.04.2022): 17–24. http://dx.doi.org/10.24815/ajse.v4i1.24431.
Pełny tekst źródłaWild, R. A., D. Applebaum-Bowden, L. M. Demers, M. Bartholomew, J. R. Landis, W. R. Hazzard i R. J. Santen. "Lipoprotein lipids in women with androgen excess: independent associations with increased insulin and androgen". Clinical Chemistry 36, nr 2 (1.02.1990): 283–89. http://dx.doi.org/10.1093/clinchem/36.2.283.
Pełny tekst źródłaJordan, P., D. Brubacher, U. Moser, H. B. Stähelin i K. F. Gey. "Vitamin E and vitamin A concentrations in plasma adjusted for cholesterol and triglycerides by multiple regression". Clinical Chemistry 41, nr 6 (1.06.1995): 924–27. http://dx.doi.org/10.1093/clinchem/41.6.924.
Pełny tekst źródłaBel’Skaya, L. V., E. A. Sarf i D. V. Solomatin. "DETERMINATION OF THE QUANTITATIVE CONTENT OF LIPIDS IN A BIOLOGICAL MATERIAL BY THE METHOD OF IR SPECTROSCOPY". Russian Clinical Laboratory Diagnostics 64, nr 4 (7.10.2019): 204–9. http://dx.doi.org/10.18821/0869-2084-2019-64-4-204-209.
Pełny tekst źródłaFranck, P., J. L. Sallerin, H. Schroeder, M. A. Gelot i P. Nabet. "Rapid determination of fecal fat by Fourier transform infrared analysis (FTIR) with partial least-squares regression and an attenuated total reflectance accessory". Clinical Chemistry 42, nr 12 (1.12.1996): 2015–20. http://dx.doi.org/10.1093/clinchem/42.12.2015.
Pełny tekst źródłaGuille, Jennifer, John K. Raison i Janusz M. Gebicki. "Radiation-induced lipid peroxidation and the fluidity of erythrocyte membrane lipids". Free Radical Biology and Medicine 3, nr 2 (styczeń 1987): 147–52. http://dx.doi.org/10.1016/s0891-5849(87)80010-2.
Pełny tekst źródłaWratten, M. L., A. A. van't Veld, U. A. van der Heide, G. van Ginkel, A. Sevanian i Y. K. Levine. "Structural and dynamic effects of oxidized lipids in unsaturated lipid membranes". Free Radical Biology and Medicine 9 (styczeń 1990): 124. http://dx.doi.org/10.1016/0891-5849(90)90618-s.
Pełny tekst źródłaFriedlander, Y., J. D. Kark i Y. Stein. "Variability of plasma lipids and lipoproteins: the Jerusalem Lipid Research Clinic Study." Clinical Chemistry 31, nr 7 (1.07.1985): 1121–26. http://dx.doi.org/10.1093/clinchem/31.7.1121.
Pełny tekst źródłaRozprawy doktorskie na temat "Medical Biochemistry: Lipids"
Jonnalagadda, Deepa. "HETEROGENEITY IN PLATELET EXOCYTOSIS". UKnowledge, 2013. http://uknowledge.uky.edu/biochem_etds/8.
Pełny tekst źródłaHaraszti, Reka A. "Engineered Exosomes for Delivery of Therapeutic siRNAs to Neurons". eScholarship@UMMS, 2018. https://escholarship.umassmed.edu/gsbs_diss/971.
Pełny tekst źródłaKénanian, Gérald. "Staphylococcus aureus se met transitoirement en dormance pour utiliser les acides gras de l'hôte et échapper à une inhibition par un anti-FASII : quel signal active son réveil ?" Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLS242.
Pełny tekst źródłaTreatment of infections caused by multidrug-resistant bacteria is a major medical challenge of the 21st century, which has stimulated the search for essential bacterial functions as potential antimicrobial drug targets. The fatty acid synthesis (FASII) pathway enzymes are considered essential, and numerous antibiotics called (anti-FASII), have been developed to eliminate pathogens of the Firmicutes phylum. However, our laboratory has shown that several pathogens bypass FASII inhibitors by incorporating exogenous fatty acids (FAs), which are abundant in the host (in blood, organs and foods). FASII bypass thus compromises the use of FASII-based antibiotics. The status of the major pathogen, Staphylococcus aureus, has remained in debate. S. aureus synthesizes an FA not produced by the host and according to the literature, is required and would not be available in the host. However, work in my lab showed that indeed bypasses FASII. The goal of my research project is to understand the mechanisms used by S. aureus to bypass FASII antibiotics. Two mechanisms are highlighted: I- High frequency mutations of the fabD gene allow S. aureus to use exogenous FAs; our study indicates that higher availability of ACP in these mutants facilitates FA utilization. II- A strategy without detectable mutation occurs in the presence of host fluids such as serum. It comprises a first "dormancy" step of about 8 to 10 hours, followed by outgrowth; FAs are incorporated throughout these steps. The latency phase appears to be due to a division block, during which cells undergo morphological changes. During "normal" growth recovery, S. aureus freely uses exogenous FAs and remains insensitive to anti-FASII. Using « time-lapse » microscopic study, we showed in our test conditions that about 3% of the bacterial population adapted to FASII antibiotics. Ours results point to an adaptation mechanism in which serum decreases bacterial stress, leading to increased availability of ACP and exogenous FAs. These substrates can then be used for phospholipid synthesis. These results resolve the debate by showing that S. aureus can replace endogenous FAs with exogenous FAs when FASII is blocked. Contrary to current dogma, FASII is not essential in S. aureus. To identify the loci involved in adaptation to anti-FASII, we performed proteomic analyses and also screened a S. aureus mutant library. Functions of stress response, cell division, and lipid metabolism appear to be involved in this adaptation. To conclude, this study clarified the steps leading to S. aureus adaptation to FASII antibiotics. Although our results show that S. aureus bypasses anti-FASII, a combinatorial approach could be considered in which FASII antibiotics could be coupled to a second inhibitor that would prevent exit from the dormancy
(7041221), Shayak Samaddar. "Delivery Strategies for Nucleic Acids". Thesis, 2019.
Znajdź pełny tekst źródła(8933363), Ahmad Abdurahman M. Alhulail. "FAT AND SODIUM QUANTIFICATION AND CORRELATION BY MRSI". Thesis, 2020.
Znajdź pełny tekst źródłaLipids and sodium (23Na) are two essential components of the human body. They play a role in almost all biological systems. However, an increase in their levels is associated with metabolic diseases. The elevation of their contents can cause similar health disorders. Examples of prevalent disorders that share an increase of musculoskeletal lipids and 23Na are hypertension and diabetes. However, the relationship between in vivo lipid and sodium levels in pathophysiology has not been studied enough and therefore is still unclear. Additionally, the available quantification methods to facilitate such a study may not be practical. They are either invasive, not sensitive enough, or require an impractical measurement time.
Therefore, in this work, our aims were to develop practical in vivo methods to quantify the absolute sodium concentration as well as the concentration of each lipid component individually, and to study the correlation between them within the skeletal muscles.
Since lipids and 23Na have different nuclear magnetic resonance properties, their quantification by magnetic resonance (MR) techniques face different challenges. Thus, we optimized different MR spectroscopic imaging (MRSI) techniques for lipids and 23Na.
Our proposed proton MRSI was able to provide eight lipid fat fraction (FF) maps representing each musculoskeletal lipid component (fatty acid) detected by our MRSI technique, and demonstrated a superior sensitivity compared to the conventional MR imaging methods.
For 23Na, our developed 23Na-MRSI was able to measure and map the absolute 23Na concentration with values agreeing with those reported previously in biopsy studies, and with a high repeatability (CV < 6 %) within significantly shorter acquisition time compared to other available techniques.
Finally, the 23Na concentration and the fat fractions of each lipid component within healthy skeletal muscles were measured and correlated using our developed MRSI methods. Our findings suggest a positive regional relationship between 23Na and lipids and negative correlation between 23Na and BMI under healthy conditions.
Sinn, Natalie. "Omega-3 fatty acids, micronutrients and cognitive and behaviour problems associated with child attention deficit hyperactivity disorder". 2006. http://arrow.unisa.edu.au:8081/1959.8/46377.
Pełny tekst źródła(6597242), Clint M. Alfaro. "DEVELOPMENT OF AMBIENT IONIZATION MASS SPECTROMETRY FOR INTRAOPERATIVE CANCER DIAGNOSTICS AND SURGICAL MARGIN ASSESSMENT". Thesis, 2019.
Znajdź pełny tekst źródłaGulati, Sonia. "Characterizing the Interaction of the ATP Binding Cassette Transporters (G subfamily) with the Intracellular Protein Lipid Environment". Thesis, 2011. https://doi.org/10.7916/D8ZW1SW7.
Pełny tekst źródła(10725291), Priya Prakash. "Characterizing Microglial Response to Amyloid: From New Tools to New Molecules". Thesis, 2021.
Znajdź pełny tekst źródłaMicroglia are a population of specialized, tissue-resident immune cells that make up around 10% of total cells in our brain. They actively prune neuronal synapses, engulf cellular debris, and misfolded protein aggregates such as the Alzheimer’s Disease (AD)-associated amyloid-beta (Aβ) by the process of phagocytosis. During AD, microglia are unable to phagocytose Aβ, perhaps due to the several disease-associated changes affecting their normal function. Functional molecules such as lipids and metabolites also influence microglial behavior but have primarily remained uncharacterized to date. The overarching question of this work is, How do microglia become dysfunctional in chronic inflammation? To this end, we developed new chemical tools to better understand and investigate the microglial response to Aβ in vitro and in vivo. Specifically, we introduce three new tools. (1) Recombinant human Aβ was developed via a rapid, refined, and robust method for expressing, purifying, and characterizing the protein. (2) A pH-sensitive fluorophore conjugate of Aβ (called AβpH) was developed to identify and separate Aβ-specific phagocytic and non-phagocytic glial cells ex vivo and in vivo. (3) New lysosomal, mitochondrial, and nuclei-targeting pH-activable fluorescent probes (called LysoShine, MitoShine, and NucShine, respectively) to visualize subcellular organelles in live microglia. Next, we asked, What changes occur to the global lipid and metabolite profiles of microglia in the presence of Aβ in vitro and in vivo? We screened 1500 lipids comprising 10 lipid classes and 700 metabolites in microglia exposed to Aβ. We found significant changes in specific lipid classes with acute and prolonged Aβ exposure. We also identified a lipid-related protein that was differentially regulated due to Aβ in vivo. This new lipid reprogramming mechanism “turned on” in the presence of cellular stress was also present in microglia in the brains of the 5xFAD mouse model, suggesting a generic response to inflammation and toxicity. It is well known that activated microglia induce reactive astrocytes during inflammation. Therefore, we asked, What changes in proteins, lipids, and metabolites occur in astrocytes due to their reactive state? We provide a comprehensive characterization of reactive astrocytes comprising 3660 proteins, 1500 lipids, and 700 metabolites. These microglia and astrocytes datasets will be available to the scientific community as a web application. We propose a final model wherein the molecules secreted by reactive astrocytes may also induce lipid-related changes to the microglial cell state in inflammation. In conclusion, this thesis highlights chemical neuroimmunology as the new frontier of neuroscience propelled by the development of new chemical tools and techniques to characterize glial cell states and function in neurodegeneration.
He, Ke. "Studies of amphiphilic helical peptides interacting with lipid bilayer membranes by x-ray and neutron scattering". Thesis, 1996. http://hdl.handle.net/1911/16991.
Pełny tekst źródłaKsiążki na temat "Medical Biochemistry: Lipids"
J, Quinn Peter, Wang Xiaoyuan i SpringerLink (Online service), red. Lipids in Health and Disease. Dordrecht: Springer Science+Business Media B.V., 2008.
Znajdź pełny tekst źródłaIntroduction to lipidomics: From bacteria to man. Boca Raton: CRC Press, 2013.
Znajdź pełny tekst źródłaJ, McIlhinney R. A., i Hooper N. M, red. Lipids, rafts and traffic: Biochemical Society symposium no. 72, held at BioScience2004, Glasgow, July 2004. London: Portland Press, 2005.
Znajdź pełny tekst źródłaD, Gunstone F., Harwood John L i Padley F. B. 1936-, red. The Lipid handbook. Wyd. 2. London: Chapman and Hall, 1994.
Znajdź pełny tekst źródłaTomohito, Hamazaki, i Okuyama Harumi, red. Fatty acids and lipids: New findings. Basel: Karger, 2001.
Znajdź pełny tekst źródłaH, Ong Augustine S., i Packer Lester, red. Lipid-soluble antioxidants: Biochemistry and clinical applications. Basel: Birkhäuser Verlag, 1992.
Znajdź pełny tekst źródłaONG i PACKER. Lipid-Soluble Antioxidants: Biochemistry and Clinical Applications. Birkhäuser, 2012.
Znajdź pełny tekst źródłaONG i PACKER. Lipid-Soluble Antioxidants: Biochemistry and Clinical Applications. Birkhauser Verlag, 2013.
Znajdź pełny tekst źródłaLeray, Claude. Introduction to Lipidomics: From Bacteria to Man. Taylor & Francis Group, 2012.
Znajdź pełny tekst źródłaLeray, Claude. Introduction to Lipidomics: From Bacteria to Man. Taylor & Francis Group, 2012.
Znajdź pełny tekst źródłaCzęści książek na temat "Medical Biochemistry: Lipids"
Blanco, Antonio, i Gustavo Blanco. "Lipids". W Medical Biochemistry, 99–119. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-12-803550-4.00005-7.
Pełny tekst źródłaBlanco, Antonio, i Gustavo Blanco. "Lipids". W Medical Biochemistry, 105–29. Elsevier, 2022. http://dx.doi.org/10.1016/b978-0-323-91599-1.00003-1.
Pełny tekst źródłaElbein, A. D. "Complex Lipids". W Medical Biochemistry, 367–76. Elsevier, 2009. http://dx.doi.org/10.1016/b978-0-323-05371-6.00027-9.
Pełny tekst źródłaBaynes, J. W. "Carbohydrates and Lipids". W Medical Biochemistry, 23–32. Elsevier, 2009. http://dx.doi.org/10.1016/b978-0-323-05371-6.00003-6.
Pełny tekst źródłaBHAGAVAN, N. V. "Lipids III: Plasma Lipoproteins". W Medical Biochemistry, 429–51. Elsevier, 2002. http://dx.doi.org/10.1016/b978-012095440-7/50022-6.
Pełny tekst źródłaAroor, AR. "Chapter-03 Chemistry of Lipids". W Medical Biochemistry, 70–100. Jaypee Brothers Medical Publishers (P) Ltd., 2011. http://dx.doi.org/10.5005/jp/books/11450_3.
Pełny tekst źródłaRaju, SM, i Bindu Madala. "Chemistry of Lipids". W Illustrated Medical Biochemistry, 62. Jaypee Brothers Medical Publishers (P) Ltd., 2005. http://dx.doi.org/10.5005/jp/books/10375_6.
Pełny tekst źródłaBhagavan, N. V., i Chung-Eun Ha. "Lipids I". W Essentials of Medical Biochemistry, 191–207. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-095461-2.00016-3.
Pełny tekst źródłaBhagavan, N. V., i Chung-Eun Ha. "Lipids II". W Essentials of Medical Biochemistry, 209–23. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-095461-2.00017-5.
Pełny tekst źródłaBhagavan, N. V., i Chung-Eun Ha. "Lipids III". W Essentials of Medical Biochemistry, 225–39. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-095461-2.00018-7.
Pełny tekst źródła