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Artykuły w czasopismach na temat "Mechanism of action"

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Dubey, Bidhyut Kumar, Mohini Chaurasia i Jyoti Yadav. "ACTIVE CHEMICAL CONSTITUENTS FROM MEDICINAL PLANTS AND MECHANISM OF ACTION AS ANTIPARKINSONIAN". Era's Journal of Medical Research 7, nr 1 (czerwiec 2020): 120–25. http://dx.doi.org/10.24041/ejmr2019.120.

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Dubey, Bidhyut Kumar, Mohini Chaurasia i Jyoti Yadav. "ACTIVE CHEMICAL CONSTITUENTS FROM MEDICINAL PLANTS AND MECHANISM OF ACTION AS ANTIPARKINSONIAN". Era's Journal of Medical Research 7, nr 1 (czerwiec 2020): 120–25. http://dx.doi.org/10.24041/ejmr2020.20.

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Edwards, David I. "Nitroimidazole drugs-action and resistance mechanisms I. Mechanism of action". Journal of Antimicrobial Chemotherapy 31, nr 1 (1993): 9–20. http://dx.doi.org/10.1093/jac/31.1.9.

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Zhong, Shan, Jeong Woo Choi, Nadia G. Hashoush, Diana Babayan, Mahsa Malekmohammadi, Nader Pouratian i Vassilios Christopoulos. "A neurocomputational theory of action regulation predicts motor behavior in neurotypical individuals and patients with Parkinson’s disease". PLOS Computational Biology 18, nr 11 (17.11.2022): e1010111. http://dx.doi.org/10.1371/journal.pcbi.1010111.

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Surviving in an uncertain environment requires not only the ability to select the best action, but also the flexibility to withhold inappropriate actions when the environmental conditions change. Although selecting and withholding actions have been extensively studied in both human and animals, there is still lack of consensus on the mechanism underlying these action regulation functions, and more importantly, how they inter-relate. A critical gap impeding progress is the lack of a computational theory that will integrate the mechanisms of action regulation into a unified framework. The current study aims to advance our understanding by developing a neurodynamical computational theory that models the mechanism of action regulation that involves suppressing responses, and predicts how disruption of this mechanism can lead to motor deficits in Parkinson’s disease (PD) patients. We tested the model predictions in neurotypical individuals and PD patients in three behavioral tasks that involve free action selection between two opposed directions, action selection in the presence of conflicting information and abandoning an ongoing action when a stop signal is presented. Our results and theory suggest an integrated mechanism of action regulation that affects both action initiation and inhibition. When this mechanism is disrupted, motor behavior is affected, leading to longer reaction times and higher error rates in action inhibition.
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Kubala Havrdová, Eva. "Cladribine - mechanism of action". Neurologie pro praxi 18, Suppl.F (1.12.2017): 5–8. http://dx.doi.org/10.36290/neu.2017.122.

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Rodan, Gideon, i Alfred Reszka. "Bisphosphonate Mechanism of Action". Current Molecular Medicine 2, nr 6 (1.09.2002): 571–77. http://dx.doi.org/10.2174/1566524023362104.

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KASUGA, Masato. "Mechanism of Insulin Action". Folia Endocrinologica Japonica 69, nr 10 (1993): 1029–34. http://dx.doi.org/10.1507/endocrine1927.69.10_1029.

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Hurtley, Stella M. "ISRIB mechanism of action". Science 359, nr 6383 (29.03.2018): 1480.8–1481. http://dx.doi.org/10.1126/science.359.6383.1480-h.

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Weiner, George J. "Rituximab: Mechanism of Action". Seminars in Hematology 47, nr 2 (kwiecień 2010): 115–23. http://dx.doi.org/10.1053/j.seminhematol.2010.01.011.

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Fuller, Ray W., Charles M. Beasley, Robert A. King, George M. Anderson, Ann Rasmusson i Mark A. Riddle. "Fluoxetine Mechanism of Action". Journal of the American Academy of Child & Adolescent Psychiatry 30, nr 5 (wrzesień 1991): 849. http://dx.doi.org/10.1097/00004583-199109000-00030.

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Rozprawy doktorskie na temat "Mechanism of action"

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Tatarski, Miloš. "Molecular mechanism of dBigH1 action". Doctoral thesis, Universitat de Barcelona, 2018. http://hdl.handle.net/10803/663021.

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INTRODUCTION: For decades it was known that many species contain embryo specific linker histone H1 variants that replace the somatic H1 during early embryogenesis. This is especially important because the early embryo shows typically zero to very little activity of transcription, and the first cleavages of the embryo depend exclusively on maternally deposited factors that are important for transcription and chromatin assembly. The first species shown to contain an embryo specific histone H1 was the sea urchin. Other species like the mouse, Xenopus or the zebrafish followed. Even in humans there are embryo specific H1 variants. Drosophila seemed to be an exception to this, until in 2013 the first linker histone H1 variant was discovered that was called dBigH1. Like other embryo H1 variants, dBigH1 is expressed in the early embryo and disappears when cellularization starts and it gets replaced by the somatic H1. Likewise, to its counterparts in other species, dBigH1 is responsible for the inhibition of transcription during the early stage of fly development. OBJECTIVES: In this thesis, we addressed the questions about the mechanism of inhibition of dBigH1 as well as the factors that are responsible for its deposition into chromatin. RESULTS: To answer the first question, we used an in vitro system for chromatin reconstitution based on an extract from early Drosophila embryos (DREX) that contains dBigH1 and all other factors needed for proper chromatin assembly. We then used the reconstituted chromatin in transcription experiments using HeLa nuclear extract that contains all factors needed for transcription. We saw that transcription for chromatin reconstituted in DREX could be reduced when the extract was previously depleted from dBigH1 using specific antibodies against it. By adding back recombinant dBigH1 to the depleted extract we were able to restore the initial lever of transcription. This showed us that dBigH1 was the repressive factor, as it was already confirmed in vivo. We then used a truncated construct of dBigH1 where we depleted the N-terminal domain of the protein. This was of particular interest as the N-terminal region of dBigH1 is the one that differs most form the somatic H1. It is much longer and more importantly very enriched with acidic residues, something that is very unique amongst all embryo specific H1 variants. We saw that when using the truncated construct, transcription was inhibited to a much lesser extent than with the full length dBigH1, proposing that the N-terminal domain is indeed responsible for the inhibition of transcription. To answer the second question about the factors needed for dBigH1 deposition, we used Drosophila testis to study dBigH1 in vivo. dBigH1 shows a very similar expression pattern in testis as the chromatin remodeler ACF1. This is why we decided to investigate a possible interaction between those two proteins. Additionally, we knew that ACF1 uses NAP1 as a histone chaperone for H1 in some species, so we also asked if NAP1 could play a role in dBigH1 deposition as well. Indeed, we saw that when using flies deficient for ACF1 we see much less dBigH1 in the testis tip where the germal stem cells (GSC) reside, suggesting that ACF1 plays an important role in dBigH1 deposition. In accordance, we see more dBigH1 in the GSCs when using flies overexpressing ACF1. At the same time, we can see that when depleting NAP1 from DREX, we see more dBigH1.
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Attarzadeh, Yazdi Ghassem. "Molecular mechanism of glucocorticoid action". Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/26161.

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The aim of this thesis project was to investigate the mechanisms by which glucocorticoid hormones regulate the activity of BK channels in human embryonic kidney 293 (HEK293) cells as the model system for glucocorticoids-action. It was shown that glucocorticoids act via endogenously expressed type II receptors in a concentration- and time-dependent manner in these cells. Dexamethasone (100 nM) had no significant effect on Dexras1 mRNA but significantly increased serum- and glucocorticoid-induced protein kinase 1 (SGK-1) mRNA. Biochemical analysis showed that SGK-1 protein is increased by dexamethasone in a Triton X-100 insoluble fraction. Further work was directed toward analysing the possible association of SGK-1 and protein phosphatases with two BK channel α-subunit variants: ZERO-BK and STREX-BK, the latter contains the 59 amino-acid splice insert encoded by the stress hormone induced exon (STREX). HEK293 cells stably expressing the respective channel subunits were analysed. Immunoprecipitations with antisera directed against the BK α-subunits showed that protein phosphatase 2A (PP2A) but not SGK-1 is constitutively associated with the STREX as well as the ZERO variant BK channel. Furthermore, the cytoplasmic C-terminal segment of the STREX-BK channel was necessary for cell-surface expression of the channel and the association of the channel with PP2A. Dexamethasone, failed to change the apparent amount of immunoreactive PP2A co-immunoprecipitating with the channel. In conclusion: SGK-1 but not Dexras1 is a protein rapidly induced by dexamethasone in HEK293 cells. PP2A but not SGK-1 is in complex with both ZERO and STREX-BK channels, and dexamethasone does not alter this association. The cytoplasmic tail of the BK channels is essential for PP2A interaction.
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Conti, Lucio. "The molecular mechanism of TFL1 action". Thesis, University of East Anglia, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.423575.

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During Arabidopsis development the shoot apical meristem (SAM) generates lateral primordia which display stage-specific traits. In long days, wild-type Arabidopsis generates leaves in an initial vegetative phase (V). Upon integration of environmental and endogenous signals, the SAM enters the reproductive phase. First it makes an 11 phase, which consists of 2-3 leaves (cauline) subtending secondary shoots (coflorescences). Next it enters the 12 phase and produces flowers on its flanks. The TFL 1 gene is a key component of the phase change machinery as mutations in TFL 1 affect the timing of phase switching. Also ffl1 mutants enter a novel phase whereby the SAM, after 12, is converted into a terminal flower, a phase normally absent in wild type. The molecular mechanism of how TFL 1 protein acts is unclear. In animal systems, TFL 1-like proteins have been shown to be components of signal transduction pathways. To understand the mechanism underlying TFL 1 function I aimed to identify proteins interacting with TFL 1 by introducing into Arabidopsis a functional TAP tag version of TFL 1 under the control of the 35S promoter. I set up conditions which allowed me to isolate and visualize by total protein staining TAPtag TFL 1. However, no obvious proteins appeared to co-purify with TFL 1. To understand how TFL 1 is modified, and to follow TFL 1 protein expression throughout development and in cell fractions, I developed polyclonal antibodies against TFL 1. These antibodies recognized TFL 1 in vivo and were used to characterize TFL 1 biochemically. TFL 1 detection by immunoblots in conjunction with mass-spectrometry analysis showed that TFL 1 was not subjected to obvious modifications unlike animal homologues. Moreover, from cellular fractionation experiments TFL 1 was located in the cytosol. To reveal essential downstream functions required for TFL 1 signaling, I characterized a suppressor mutant, called sof1, of plants ectopically expressing TFL 1. I mapped sof1 within a confined region on the bottom of chromosome 3. Physiological analysis of sof1 led to a model of SOT1 action in controlling phase change. TFL 1 mRNA is found in a unique expression domain which comprises a group of cells in the centre of the SAM and yet TFL 1 affects the identity of lateral primordia. By using affinity purified anti-TFL 1 antibodies I showed that TFL 1 protein moves and is distributed throughout the SAM. This might account for the effect of TFL 1 on controlling overall shoot identity and raises important questions on the role of the TFL 1 protein outside its mRNA expression domain.
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Beastall, J. C. "The mechanism of action of azone". Thesis, University of Nottingham, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.380112.

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Williams, Daffydd Griffin. "Mechanism of action of penetration enhancers". Thesis, Cardiff University, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.320625.

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Taylor, Catherine. "A mechanism of action of strigolactone". Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.709140.

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Taylor, M. R. G. "Mechanism of action of Rad51 paralogs". Thesis, University College London (University of London), 2015. http://discovery.ucl.ac.uk/1458671/.

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Homologous recombination (HR) is an essential DNA break repair mechanism that remains incompletely understood. HR is a complex multistep process initiated by the loading of RAD-51 recombinase as filaments onto single stranded DNA (ssDNA). This structure directly invades an intact homologous duplex, which serves as a template for repair DNA synthesis. Numerous positive regulators of HR have been described, including the Rad51 paralogs, but the mechanism of action of Rad51 paralogs in promoting HR is unknown. In this study, I have characterized the mechanism of action of a novel Rad51 paralog complex, RFS-1/RIP-1, from C. elegans. RFS-1 is a Rad51 paralog required for RAD-51 focus formation at stalled replication forks, indicating an early positive regulatory role in HR. I demonstrate that RFS-1 interacts with a nematode-specific orphan protein, RIP-1. I identify a cryptic Walker B ATPase-like motif within RIP-1, which is functionally important in establishing the RFS-1/RIP-1 interaction interface. rip-1 and rfs-1 mutant animals phenocopy for essentially all phenotypes analysed. Together these data suggest RFS-1/RIP-1 functions as a constitutive complex. I show recombinant RFS-1/RIP-1 can be purified and specifically binds ssDNA but lacks measurable ATPase activity. RFS-1/RIP-1 also strongly stimulates strand invasion activity by RAD-51, consistent with a pro-recombinogenic function in vivo. I define for the first time the mechanism of action underlying the intrinsic ability of Rad51 paralogs to stimulate HR. Using a combination of biochemical and biophysical approaches, notably electrophoretic mobility shift assays, stopped-flow reaction kinetics and nuclease protection assays, I show RFS-1/RIP-1 dramatically alters the properties of RAD-51-ssDNA filaments such that RAD-51 is more stably associated with ssDNA yet the ssDNA is more sensitive to nuclease degradation. RFS-1/RIP-1 exerts these effects primarily downstream of filament formation, ruling out a major role in RAD-51 loading. I propose RFS-1/RIP-1 remodels RAD-51-ssDNA filaments to a conformation poised for pairing with the template duplex and strand invasion.
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Phisit, Prapunwattana Yongyuth Yuthavong. "Mechanism of antimalarial action of tetracycline /". abstract, 1986. http://mulinet3.li.mahidol.ac.th/thesis/2529/29E-Phisit-P.pdf.

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Díaz, i. Cirac Anna. "Mechanism of action of cyclic antimicrobial peptides". Doctoral thesis, Universitat de Girona, 2011. http://hdl.handle.net/10803/38252.

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This PhD thesis is the result of the combination of experimental and computational techniques with the aim of understanding the mechanism of action of de novo cyclic decapeptides with high antimicrobial activity. By experimental techniques the influence of the replacement of the phenylalanine for tryptophan residue in their antimicrobial activity was tested and the stability in human serum was also analyzed, in order to evaluate their potential therapeutic application as antitumor agents. On the other hand, the interaction amongst the peptide BPC194 c(KKLKKFKKLQ), the best candidate from the whole library of cyclic peptides, and a model anionic membrane was simulated. The results showed a structure-function relationship derived from the stable conformation of the peptides involved in the membrane permeabilization. As a result, a rational design was performed being BPC490 the peptide with best antimicrobial activity compared with the best active peptide from the original library.
Aquesta tesi doctoral resulta de la combinació d’estudis mitjançant tècniques experimentals i computacionals amb l’objectiu d’entendre el mecanisme d’acció de "de novo" decapèptids cíclics amb elevada activitat antimicrobiana. Experimentalment, es va avaluar la influència de la substitució dels residus de fenilalanina per triptòfan en la seva activitat antimicrobiana i també la seva estabilitat sèrum humà, per tal de valorar la seva possible aplicació terapèutica envers el càncer. Per altra banda, es va simular la interacció del pèptid BPC194 c(KKLKKFKKLQ), millor candidat de la biblioteca de pèptids cíclics, amb models aniònics de bicapa lipídica. Els resultats van posar en manifest una relació estructura-funció derivada de la conformació estable dels pèptids que participen directament en la permeabilització de la membrana. Es va procedir doncs al disseny racional de nous pèptids cíclics sent el pèptid BPC490 el que va presentar una millor activitat bacteriana en comparació amb el pèptid més actiu de la llibreria original.
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Reißmann, Stefanie. "Mechanism of Action of Group II Chaperonins:". Diss., lmu, 2007. http://nbn-resolving.de/urn:nbn:de:bvb:19-73195.

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Książki na temat "Mechanism of action"

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1925-, Kuby Stephen Allen, red. Mechanism of enzyme action. Boca Raton: CRC Press, 1991.

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Govil, J. N., Amani S. Awaad i Geetanjali Kaushik. Mechanism and action of phytoconstituents. Houston, TX: Studium Press, 2011.

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Golan, Adam E. Cellulase: Types and action, mechanism, and uses. New York, N.Y: Nova Science Publishers, 2011.

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1945-, Moudgil V. K., red. Molecular mechanism of steroid hormone action: Recent advances. Berlin: Walter de Gruyter, 1985.

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Woo, Junghoon. Interrogating Drug Mechanism of Action Using Network Dysregulation Analysis. [New York, N.Y.?]: [publisher not identified], 2015.

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Deans, Bryan. Studies on the mechanism of action on antitumour imidazotetrazinones. Birmingham: Aston University. Department ofPharmaceutical and Biological Sciences, 1994.

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Hickman, John Angus. Studies of the mechanism of action of antitumour compounds. Birmingham: Aston University. Department of Pharmaceutical Sciences, 1989.

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1946-, Barnes Peter J., red. The Mechanism of action of Xanthines in respiratory disease. London: Royal Society of Medicine Services, 1988.

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Lewis, John. Mechanism of action of overbased additives in hydrocarbon media. Norwich: University of East Anglia, 1991.

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1950-, Aggarwal Bharat B., i Vilček J. 1933-, red. Tumor necrosis factors: Structure, function, and mechanism of action. New York: M. Dekker, 1992.

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Części książek na temat "Mechanism of action"

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Safe, Stephen H., Thomas Gasiewicz i James P. Whitlock. "Mechanism of Action". W Environmental Toxin Series, 61–91. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-70556-4_3.

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Mostafa, Manal, Amal-Asran, Hassan Almoammar i Kamel A. Abd-Elsalam. "Nanoantimicrobials Mechanism of Action". W Nanotechnology in the Life Sciences, 281–322. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91161-8_11.

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Soultanas, Panos, i Edward Bolt. "Helicase Mechanism of Action". W Molecular Life Sciences, 1–12. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-6436-5_291-1.

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Rasmussen, Howard, i Irving L. Schwartz. "Mechanism of Hormone Action". W Endocrinology, 335–68. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4614-7436-4_12.

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Jain, Akanksha, Sonia Bajaj, Swati Tanti i Suchitra Panigrahy. "Biosorbents Mechanism of Action". W Biosorbents, 96–122. Boca Raton: CRC Press, 2024. http://dx.doi.org/10.1201/9781003366058-6.

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White, Morris F. "Mechanism of Insulin Action". W Textbook of Diabetes, 114–32. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118924853.ch8.

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Soultanas, Panos, i Edward Bolt. "Helicase Mechanism of Action". W Molecular Life Sciences, 516–26. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4614-1531-2_291.

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Timell, T. E. "Mechanism of Compression Wood Action". W Compression Wood in Gymnosperms, 1745–97. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-61616-7_17.

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Waley, S. G. "ß-Lactamase: mechanism of action". W The Chemistry of β-Lactams, 198–228. Dordrecht: Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2928-2_6.

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Frank, J., M. Dijkstra, C. Balny, P. E. J. Verwiel i J. A. Duine. "Methanol Dehydrogenase: Mechanism of Action". W PQQ and Quinoproteins, 13–22. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0957-1_2.

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Streszczenia konferencji na temat "Mechanism of action"

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Cai, Yiheng, Xinran Kong i Xueyan Wang. "Temporal Action Detection with Long Action Seam Mechanism". W the 2nd International Conference. New York, New York, USA: ACM Press, 2018. http://dx.doi.org/10.1145/3278198.3278224.

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Nelson, Jack, Nick Klein, Andrew Swift, Pete Matthews i Emily Drapal. "Doptelet Mechanism of Action Animation". W SIGGRAPH '24: ACM SIGGRAPH 2024 Electronic Theater. New York, NY, USA: ACM, 2024. http://dx.doi.org/10.1145/3641230.3653313.

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Masoudi, Ramin, Stephen Birkett i John McPhee. "Dynamic Model of a Vertical Piano Action Mechanism". W ASME 2009 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2009. http://dx.doi.org/10.1115/detc2009-87680.

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The dynamic behavior of a vertical piano action mechanism is studied using a simulation model and compared qualitatively to observations obtained by high-speed imaging of a real action. The simulated response of all components is obtained for two different prescribed input force profiles applied at the key front. These inputs represent in simplified form the general shape of a typical force input by a pianist measured at the key surface for a strong (forte) strike, or two key strikes in rapid succession. The graph-theoretic multibody model constructed represents the components and their interactions. Explicit contact edges provide forces generated between two bodies as a function of their kinematic states, using a special contact model to represent the compression of felt lined interfaces that can separate during the key stroke. Masses and geometrical parameters of the action were measured by importing scanned images from a real action into CAD software. The highly nonlinear system of five ordinary differential equations of motion was derived symbolically and solved by a numerical stiff solver in Maple. The effects of two components not present in the horizontal grand piano action, the bridle strap and hammer butt spring, were examined using simulations. The butt spring is seen to serve an important function in assisting the return of the hammer to its rest position on key release. The model will be useful in future studies to compare vertical actions to horizontal grand piano actions, as these are known to exhibit quite different playing characteristics.
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Wang, Yunlong, Xiang Liu, Chuang Yu i Zhaowei Guo. "Mechanism Action Detection Based on YOLO". W 2023 International Conference on Image Processing, Computer Vision and Machine Learning (ICICML). IEEE, 2023. http://dx.doi.org/10.1109/icicml60161.2023.10424896.

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Hirschkorn, Martin C., John McPhee i Stephen Birkett. "Dynamic Modelling and Experimental Testing of a Piano Action Mechanism". W ASME 2005 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2005. http://dx.doi.org/10.1115/detc2005-84511.

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A new model for a grand piano action is proposed in this paper. The multibody dynamic model treats each of the five main action components (key, whippen, jack, repetition lever, and hammer) as a rigid body, and incorporates a contact model to determine the normal and friction forces at 13 locations between each of the contacting bodies. All parameters in the model are directly measured from experiments on individual action components, allowing the model to be used as a prototyping tool for actions that have not yet been designed or built. The behaviour of the model was compared to the behaviour of an experimental grand piano action and found to be very accurate for high force blows, and reasonably accurate for low force blows.
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Nelson, Carl A. "Design and Dynamic Simulation of a Novel Piano Action Mechanism". W ASME 2008 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2008. http://dx.doi.org/10.1115/detc2008-49167.

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A novel piano action mechanism is presented, which is based on a multi-lobed lifting cam. The cam replaces several bodies in the standard action mechanism and allows the upright piano to achieve fast repetition of a single note, which is a quality more commonly attributed to grand piano actions. The use of simulation software to aid in tuning of the design is described, and the final simulation results are presented. These results indicate the ability of the new mechanism to achieve good performance over a range of playing intensities and to play repeated notes in rapid succession.
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Xiaoping, Liu, i Qi Liangqun. "Research on Enterprise's Dynamic Capabilities Action Mechanism". W 2011 International Conference on Information Management, Innovation Management and Industrial Engineering (ICIII). IEEE, 2011. http://dx.doi.org/10.1109/iciii.2011.415.

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Shih, Hsiao-Yi, i Arnost Reiser. "Mechanism of inhibitor action in novolak film". W SPIE's 1995 Symposium on Microlithography, redaktor Robert D. Allen. SPIE, 1995. http://dx.doi.org/10.1117/12.210351.

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Wang, Wenhao, Xiaobo Lu, Pengguo Zhang, Huibin Xie i Wenbin Zeng. "Driver Action Recognition Based on Attention Mechanism". W 2019 6th International Conference on Systems and Informatics (ICSAI). IEEE, 2019. http://dx.doi.org/10.1109/icsai48974.2019.9010589.

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Chai Lihong, Jiang Ling, Shi Yanfei i Wang Hongyuan. "Toxicity effects and action mechanism of fluoride". W 2011 International Symposium on Water Resource and Environmental Protection (ISWREP). IEEE, 2011. http://dx.doi.org/10.1109/iswrep.2011.5893495.

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Raporty organizacyjne na temat "Mechanism of action"

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Roberts, Jr, i Charles T. A Novel Mechanism of Androgen Receptor Action. Fort Belvoir, VA: Defense Technical Information Center, styczeń 2008. http://dx.doi.org/10.21236/ada489364.

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Roberts, Jr, i Charles T. A Novel Mechanism of Androgen Receptor Action. Fort Belvoir, VA: Defense Technical Information Center, styczeń 2009. http://dx.doi.org/10.21236/ada503252.

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Bates, Paula J. Mechanism of Action of Novel Antiproliferative Oligonucleotides. Fort Belvoir, VA: Defense Technical Information Center, maj 2002. http://dx.doi.org/10.21236/ada406133.

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Roberts, Jr, i Charles T. A Novel Mechanism of Androgen Receptor Action. Fort Belvoir, VA: Defense Technical Information Center, styczeń 2007. http://dx.doi.org/10.21236/ada466168.

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Kamasani, Uma R., i George Prendergast. Mechanism of RhoB/FTI Action in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, maj 2004. http://dx.doi.org/10.21236/ada446332.

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Rane, Neena S., i George C. Prendergast. Mechanism of RhoB/FTI Action in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, maj 2002. http://dx.doi.org/10.21236/ada412302.

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Rogers, Terry B. Mechanism of Action of the Presynaptic Neurotoxin, Tetanus Toxin. Fort Belvoir, VA: Defense Technical Information Center, lipiec 1991. http://dx.doi.org/10.21236/ada246780.

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Rogers, Terry B. Mechanism of Action of the Presynaptic Neurotoxins Tetanus Toxin. Fort Belvoir, VA: Defense Technical Information Center, styczeń 1992. http://dx.doi.org/10.21236/ada246495.

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Kaiser, Ivan I. Rattlesnake Neurotoxin Structure, Mechanism of Action, Immunology and Molecular Biology. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 1990. http://dx.doi.org/10.21236/ada228003.

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Leoni, Lorenzo M. Mechanism of Action of Substituted Indanones in Multidrug Resistant Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2002. http://dx.doi.org/10.21236/ada424665.

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