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Artykuły w czasopismach na temat "Mécanismes d'échappement"
Guillet, JG. "Les mécanismes d'échappement viraux." médecine/sciences 16, nr 8-9 (2000): 874. http://dx.doi.org/10.4267/10608/1751.
Pełny tekst źródłaCostello, R. T., J. A. Gastaut i D. Olive. "Mécanismes d'échappement tumoral à la réponse immunitaire". La Revue de Médecine Interne 20, nr 7 (lipiec 1999): 579–88. http://dx.doi.org/10.1016/s0248-8663(99)80107-6.
Pełny tekst źródłaMessud-Petit, F., i S. Bertagnoli. "Mécanismes d'échappement immunitaire des Orthopoxvirus Orthopoxvirus immune evasion mechanism". Médecine et Maladies Infectieuses 34 (czerwiec 2004): S38—S39. http://dx.doi.org/10.1016/s0399-077x(04)90012-1.
Pełny tekst źródłaBuffin-Bélanger, Thomas, i André G. Roy. "Vers l’intégration des structures turbulentes de l’écoulement dans la dynamique d’un cours d’eau à lit de graviers". Géographie physique et Quaternaire 54, nr 1 (2.10.2002): 105–17. http://dx.doi.org/10.7202/004776ar.
Pełny tekst źródłaBuffin-Bélanger, T., A. G. Roy i M. Levasseur. "Interactions entre les structures d'échappement et les structures à grande échelle dans l'écoulement turbulent des rivières à lit de graviers". Revue des sciences de l'eau 14, nr 3 (12.04.2005): 381–407. http://dx.doi.org/10.7202/705425ar.
Pełny tekst źródłaLi, Xia-Dong. "Nouveau mécanisme d'échappement du VHC à la réponse immune". Revue Francophone des Laboratoires 2006, nr 378 (styczeń 2006): 16. http://dx.doi.org/10.1016/s1773-035x(06)80026-2.
Pełny tekst źródłaSène, D., M. Abel, F. Levasseur, F. Charlotte, J. C. Piette, S. Caillat-Zucman i P. Cacoub. "Altérations de la voie d'activation NKG2D/MIC sur les cellules Natural Killer et CD8 au cours de l'infection chronique par le virus de l'hépatite C: nouveau mécanisme d'échappement virologique". La Revue de Médecine Interne 27 (grudzień 2006): S315—S316. http://dx.doi.org/10.1016/j.revmed.2006.10.069.
Pełny tekst źródłaRozprawy doktorskie na temat "Mécanismes d'échappement"
Bertucci, César. "Etude de l'interaction du vent solaire avec Mars : implications sur les mécanismes d'échappement atmosphérique". Toulouse 3, 2003. https://tel.archives-ouvertes.fr/tel-00010454.
Pełny tekst źródłaLesport, Emilie. "Etude des mécanismes d'échappement des tumeurs aux cellules NK et T γδ induits par la molécule HLA-G". Paris 7, 2011. http://www.theses.fr/2011PA077043.
Pełny tekst źródłaNatural Killer (NK) cells and γδ T cells are both cytotoxic effectors playing a major role in the immunosurveillance of cancers. Even though tumors can be eliminated thanks to the potent anti-tumoral fonctions of these immune cells, it is well establish that they develop various mechanisms in order to évade the immune System. In this regard, the aim of my thesis was to understand how the expression of the tolerogenic molécule HLA-G by tumor cells contributes to their escape from NK and γδ T cell recognition. We identified the molecular events leading to the inhibition of NK cell cytotoxicity induced by tumor cells expressing HLA-G. Our results demonstrate that the interaction of the ILT2 inhibitory receptor expressed by NK cells with HLA-G inhibits the reorganization of actin and tubulin cytoskeleton within the NK cells, thus preventing the polarization and the delivery of lytic granules toward the tumor target cells. In parallel, we studied the effect of HLA-G expression by tumor cells on the fonctions of γδ T cells. We showed that primary tumor cells expressing HLA-G are protected against γδ T cell-mediated cytolysis and that this inhibition was due to the interaction of HLA-G with ILT2. Moreover, our data indicate that HLA-G expression by tumor cells inhibits both the production of IFN-y and the proliferation of γδ T cells. Altogether, these results are important for a better understanding of the mechanisms linking HLA-G expression and tumor immune escape. In the future, they could contribute to the optimization of immunotherapy treatments for cancer patients expressing HLA-G
Veuillen, Caroline. "Caractérisation des mécanismes d'échappement tumoral à la lyse NK dans la LLC-B et le cancer de la prostate". Thesis, Aix-Marseille 2, 2011. http://www.theses.fr/2011AIX20708.
Pełny tekst źródłaMany experimental and clinical data have enlightened the importance of Natural Killer (NK) cells in tumor immunosurveillance. Therapeutic strategies based on NK cells could be an alternative in the treatment of certain cancers. We focused our study on two types of incurable cancers despite recent advances in treatment: B chronic lymphocytic leukemia (B-CLL) and prostate cancer. The aim of our study is a better understanding of the mechanisms set up by leukemic B cells and prostate cancer cells to escape from NK antitumor response. The knowledge of these escape mechanisms is an essential prerequisite to the use of NK cells in antitumor therapies. Regarding B-CLL, our results suggest that NK cells, although functionally competent, can not initiate an appropriate immune response against leukemic B cells due to a lack of recognition of the latter. Concerning the prostate cancer, our preliminary data show that circulating NK cells are functionally competent, whatever the stage of disease, despite the significant decrease in expression of the receptor NKp30. Thus, the degree of immunogenicity of leukemic B cells and of the prostate cancer cells must be taken into account as well as the functionality of NK cells in strategies aiming at improving NK antitumor activity
Levasseur, Franck. "Mécanismes d'échappement à la réponse immune innée de l'hôte au cours de l'infection chronique par le virus de l'hépatite C (VHC)". Paris 5, 2009. http://www.theses.fr/2009PA05T040.
Pełny tekst źródłaUnderstanding how hepatitis C virus (HCV) induces and circumvents innate and adaptive immunity of the host is of critical importance in efforts to design effective therapeutics. We report here the decreased expression of the NKG2D activating receptor as a novel strategy adopted by HCV to evade NK-cell mediated responses. We show that, upon TLR4 stimulation by the HCV NS5A protein, monocytes secrete high amounts of TGFβ that downmodulates NKG2D expression on NK cells, leading to their impaired lytic potential and IFNy production. Using the JFH1-replicating Huh7. 5. 1 cell system, we provide evidence that NS5A released from apoptotic infected cells binds to monocytes. NS5A-induced signaling through TLR4 elicits p38- and PI3 kinase-dependent IL-10 production, which in turn triggers TGFβ release while inhibiting IL-12 production. Exogenous IL-15 can rescue NKG2D expression at normal levels and induce functional reprogramming of NK cells
Leray, Alexis. "Identification des mécanismes physico-chimiques impliqués dans le post-traitement plasma des gaz d'échappement et études comparatives des différentes technologies plasma". Thesis, Orléans, 2012. http://www.theses.fr/2012ORLE2049.
Pełny tekst źródłaThe new HCCI combustion mode is well adapted to improve nitrogen oxide and particulate matter reduction from Diesel engine in order to meet future emission regulations adopted in the Euro zone. However, HCCI engines emit relatively high amounts of unburned hydrocarbons and carbon monoxide due to lower engine exhaust temperature increasing the catalyst light-off time and decreasing the average efficiency of the Diesel oxidation catalyst (DOC). In this environmental and economic context, the combination of plasma with DOC has been considered especially for intermittent use during the cold start. The thesis presents the combination of nonthermal plasma upstream Diesel oxidation catalyst (Pt-Pd/Al2O3) applied to the treatment of simulating Diesel HCCI exhaust gas (O2-NO-H2O-CO-CO2-CH4-C3H6-C7H8-C10H22-N2). The studies were conducted at atmospheric pressure with a pilot-scale dielectric barrier discharge reactor (DBD) on two experimental devices. The first is a laboratory scale set-up (low flow rate : 20 Lmin−1) used to understand the physico-chemical involving the plasma and the catalyst by focusing on the by-products reactions. The second is an industrial scale (gas flow rate up to 260 Lmin−1) used to study the feasibility and the efficiency of the plasma-DOC system under conditions similar to those encountered in Diesel exhaust engine. The effects of the plasma, the DOC and the plasma-DOC systems on the exhaust gas have been investigated under various conditions. The main contribution of the plasma was to give a « thermal » and a chemical « push » to the DOC resulting in the decrease of light-off temperature for CO and HC oxidation. These improvements were shown to depend on the treatment conditions (injected energy i.e. energy density, space velocity, gas temperature and nature of the driving cycle). It is shown that for a simulated European Driving Cycle (NEDC), the combination of plasma upstream DOC reduces the cumulative mass of CO and hydrocarbons by about 68% and 42%, respectively, in accordance with the Euro 6 standard (2014). The efficiency of plasma for hydrocarbons and NO oxidation at low temperature in high flow conditions (up to 900 Lmin−1 on the NEDC) has been confirmed and the main reaction products identified and quantified
Zervas, Efthimios. "Etude des mécanismes de formation des polluants spécifiques émis par les moteurs à combustion interne". Mulhouse, 1996. https://tel.archives-ouvertes.fr/tel-02966575.
Pełny tekst źródłaMethods for the analysis on sulfur dioxide, alcohols and organic acids have been developed. The first one includes the capture of the sulfur dioxide in a solution of oxygenated water and the analysis by ionic chromatography with a conductimetric detector. The second one includes the capture in pure water and an analysis by gas chromatography/flame ionisation detector. The third one uses the capture in pure water and the analysis of the formic acid by an ionic chromatography and of the other acids by gas chromatography. These methods have been applied in the case of vehicles' non-regulated pollutants research. An experiment design, combined specified fuels and analysis of the exhaust gazes, has been applied on a spark ignition engine. These tests proved several qualitative and quantitative correlations between the composition of the fuel and the emitted pollutants. Precursors of hydrocarbons, aldehydes, ketones, alcohols and organic acids have been found. These results show that aromatics and cyclohexane contribute for the benzene's formation, 1-hexene and cyclohexane for the 1,3 butadiene's, aromatics are the precursors of the propionic acid and o xylene of the butyric acid
Aloui, Hichem. "Etude de l'oxydation catalytique des suies Diesel sur poudre métallique activée". Poitiers, 1999. http://www.theses.fr/1999POIT2330.
Pełny tekst źródłaGravelle, Pauline. "Influence de l'organisation tridimensionnelle des cellules de lymphome folliculaire sur l'expression génique : contribution à la caractérisation des mécanismes d'échappement immunitaire et de chimiorésistance". Toulouse 3, 2013. http://thesesups.ups-tlse.fr/2104/.
Pełny tekst źródłaNon-Hodgkin lymphomas (NHL) are the most frequent malignant hematological diseases. Among these, follicular lymphomas (FL) are the second most common subtype of indolent NHL in frequency. FL cells arise from germinal center and are characterized by the constitutive over-expression of BCL2 proto-oncogene, due to t(14,18) chromosomal translocation. In carcinomas, spatial organization is described as modulating major cell functions such as tumor growth, intracellular signaling, but also cell survival, sensitivity to drugs, or metastatic potential. But although FL develops in 3D in patients, the contribution of spatial organization on FL cells biology was never investigated until now. To this aim, we developed the first three-dimensional culture model of LF cells (MALC for Multicellular Aggregates of Lymphoma Cell). We studied, in this system, by comparison with conventional suspension cultures, gene expression profile, proliferation, stress responses as well as sensitivity to genotoxics, targeted therapies, and immune cellular effectors. The present study shows that spatial organization profoundly affects gene expression profile (more than 600 genes are modulated). The gene signatures found in MALC cells are specific of negative regulation of cell cycle, hypoxic stress, and NF-kB pathway activation. These 3 signatures are closer to those identified in patient samples. Phenotypic analysis further supports decreased cell proliferation, and for the half of MALC cells, quiescence entry through a HIF1?-dependent mechanism. Cells cultured in 3D are more resistant to anthracyclines and alkylating agents, as to NK cells. However, these cells remain equally sensitive to targeted therapies (Rituximab(r) and mTOR kinase inhibitors). To conclude, this study shows that FL cells biology is considerably determined by 3D organization. This also suggests that the MALC model offers a unique opportunity for pre-clinical screening of new bio-active molecules, MALC being more representative than conventional 2D culture
Rouanne, Mathieu. "Mechanisms of Resistance to BCG Immunotherapy in Bladder Cancer". Thesis, Université Paris-Saclay (ComUE), 2019. http://www.theses.fr/2019SACLS342.
Pełny tekst źródłaBladder cancer is a heterogeneous disease that displays invasive and non-invasive histological features, and a wide spectrum of molecular alterations and subtypes. Treatment of non-invasive tumors with high-risk features (carcinoma in situ, high-grade Ta, T1) includes trans-urethral resection of the tumor, followed by intravesical instillations of bacillus Calmette-Guérin (BCG). Despite a multitude of evidence for anti-tumor efficacy, 50% of patients with high-risk NMIBC develop tumor recurrence and 20-30% disease progression. Ultimately, 10-15% of patients die of metastatic disease. New therapeutic strategies are currently in clinical development to treat BCG-unresponsive tumors including antagonistic antibodies directed against the T-cell immune checkpoints PD-1, PD-L1 and CTLA-4, but also recombinant adenovirus interferon α (Ad-IFNα/Syn3), oncolytic virus and STING agonists. Although recent studies have identified potential immune parameters that could impact clinical response, mechanisms of tumor resistance to BCG immunotherapy remain poorly understood. Additionally, tumor heterogeneity and plasticity of cancer cells undermine our attempts to precise dynamics of immune escape under selective pressure. How cancer cells evade to the anti-tumor immune response, and whether cancer cells acquire intrinsic undesirable characteristics upon BCG exposure remain unknown. Altogether, this highlights the crucial need to better understand the mechanisms of tumor resistance that occur during BCG immunotherapy in order to identify new targetable pathways and treatment strategies
Guillonneau, Richard. "Diversité des interactions microbiennes au sein de l'environnement marin ˸ : De biofilms multi-spécifiques à multi-organismes". Electronic Thesis or Diss., Toulon, 2018. http://www.theses.fr/2018TOUL0009.
Pełny tekst źródłaThe formation of multi-organisms biofilms is poorly studied especially with marine organisms. This work first showed that strains harvested in biofilms from the Mediterranean Sea displayed a heterogeneity in their biofilm formation abilities and a diversity of matrix compounds. The study of multi-species biofilms revealed antagonistic and beneficial effects of some strains on the biofilm development of their partners. The interactions between amoebae and marine bacteria inoculated at a low ratio showed that all the strains tested were phagocytosed by Acanthamoeba castellanii. However, different mechanisms of escapement from their predators have been unraveled, such as a bacterial localization within the cell nucleolus or within fecal pellets expelled from amoebae. However, when the amoebae were added to a preformed bacterial monospecies or multispecies biofilms, a majority of bacteria detached from the surface. The amoebae supernatant induced also a bacterial detachment on two of the bacteria in monospecies biofilms, as well as morphological changes of these bacteria, suggesting that amoeba chemical cues are secreted and detected by the bacteria. Therefore, although a simple grazing of non-pathogenic marine bacteria by amoebae could have been expected, a diversity of bacterial behaviors was unraveled giving an idea on the diversity of interaction mechanisms that may exist in the marine environment