Gotowa bibliografia na temat „Maladie artérielle périphérique – chirurgie”
Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych
Spis treści
Zobacz listy aktualnych artykułów, książek, rozpraw, streszczeń i innych źródeł naukowych na temat „Maladie artérielle périphérique – chirurgie”.
Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.
Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.
Artykuły w czasopismach na temat "Maladie artérielle périphérique – chirurgie"
Qi, Arthur A., i Christina S. Korownyk. "L’exercice contre la maladie artérielle périphérique". Canadian Family Physician 68, nr 4 (kwiecień 2022): 278–79. http://dx.doi.org/10.46747/cfp.6804278.
Pełny tekst źródłaElouakili, I., M. Bouayad, Y. Tijani, L. Benjilali, B. Lekehal, Y. Sefiani, A. El Mesnaoui, F. Ammar i Y. Bensaid. "Maladie de Horton et atteinte artérielle périphérique". Journal des Maladies Vasculaires 35, nr 5 (wrzesień 2010): 313. http://dx.doi.org/10.1016/j.jmv.2010.07.061.
Pełny tekst źródłaChaparala, Ramakrishna P. C., Nicolas M. Orsi, Nigel J. Lindsey, Raman S. Girn i Shervanthi Homer-Vanniasinkam. "Profil Inflammatoire de la Maladie Artérielle Périphérique". Annales de Chirurgie Vasculaire 23, nr 2 (marzec 2009): 186–93. http://dx.doi.org/10.1016/j.acvfr.2009.05.012.
Pełny tekst źródłaKeller, Sanjiv, Luca Calanca, Enrica Porceddu, Marco Fresa, Céline Deslarzes i Lucia Mazzolai. "Maladie artérielle périphérique : du diagnostic au traitement". Revue Médicale Suisse 19, nr 853 (2023): 2304–9. http://dx.doi.org/10.53738/revmed.2023.19.853.2304.
Pełny tekst źródłaLengani, A., A. Ouédraogo, G. Sanou, J. I. Nitiéma, Y. J. Bonzi, S. H. Traoré i G. Coulibaly. "Maladie artérielle périphérique chez les patients atteints de maladie rénale chronique non dialysés". Néphrologie & Thérapeutique 16, nr 5 (wrzesień 2020): 308. http://dx.doi.org/10.1016/j.nephro.2020.07.160.
Pełny tekst źródłaBulaïd, Y., C. Klein, R. Gouron, O. Jardé i F. Deroussen. "Résultats du traitement des pieds creux médiaux d’enfants atteints de la maladie de Charcot-Marie-Tooth par des orthèses de détorsion nocturnes innovantes". Médecine et Chirurgie du Pied 37, nr 1 (marzec 2021): 1–9. http://dx.doi.org/10.3166/mcp-2021-0065.
Pełny tekst źródłaAuboire, L., M. Becquet i C. Le Hello. "Atteinte artérielle périphérique dans la maladie de Horton : comment différencier athérome et inflammation ?" Journal des Maladies Vasculaires 36, nr 4 (lipiec 2011): 280–84. http://dx.doi.org/10.1016/j.jmv.2011.06.003.
Pełny tekst źródłaKeller, Sanjiv, Rosaria Del Giorno, Giacomo Buso, Céline Deslarzes, Luca Calanca, Stefano Lanzi i Lucia Mazzolai. "Passeport vasculaire : un outil pour la prévention secondaire des patients avec maladie artérielle périphérique". Revue Médicale Suisse 17, nr 762 (2021): 2128–31. http://dx.doi.org/10.53738/revmed.2021.17.762.2128.
Pełny tekst źródłaBlomstrand, David, Tilo Kölbel, Bengt Lindblad i Anders Gottsäter. "Le complexe Protéine C activée - inhibiteur de protéine C dans la maladie artérielle périphérique". Annales de Chirurgie Vasculaire 24, nr 5 (lipiec 2010): 642–50. http://dx.doi.org/10.1016/j.acvfr.2010.12.044.
Pełny tekst źródłaBoned, A., R. Granger, M. Bousquet, L. Richard, V. Gremeaux, M. Labrunée i T. Guiraud. "Effets du TENS sur la distance de marche chez un patient atteint d’une maladie artérielle périphérique". Annals of Physical and Rehabilitation Medicine 55 (październik 2012): e317. http://dx.doi.org/10.1016/j.rehab.2012.07.799.
Pełny tekst źródłaRozprawy doktorskie na temat "Maladie artérielle périphérique – chirurgie"
Mougin, Justine. "Mise au point et optimisation d'un modèle lagomorphe hypercholestérolémique de resténose intra-stent et application pour le développement d'un stent obtenu par électrofilage". Electronic Thesis or Diss., Université de Lille (2022-....), 2023. http://www.theses.fr/2023ULILS019.
Pełny tekst źródłaObjective: This study aimed to evaluate the use of a double injured atherosclerotic iliac rabbit model for myointimal hyperplasia evaluation. Secondarily, this animal model was use for pre clincial evaluation of a new anti-in stent restenosis simvastatin electrospun covered stent. Methods: Twenty four New Zealand White (NZW) rabbits were included in this study. In order to enhance and accelerate atherogenesis, atherogenic diet (0.3% cholesterol and 4.9% coconut oil) and mechanical endothelial injury of iliac artery were used. Twelve rabbits (24 iliac arteries) were used to evaluate the benefit of intimal balloon injury compared to the diet alone on myointimal hyperplasia. On day 7, rabbits beneficiated balloon iliac injury on the left side only. Eight weeks they were scarified and iliac arteries were harvested to histologic examination and comparaison. Twelve rabbits were included in the second evaluation to compare anti instent restenosis effect of a new drug eluting chrome-cobalt stent (DES) coated with polycyclodextrin-chitosan-simvastatin polymer after electrospinning technique. Balloon injury were performed on both iliacs on day 7, then stenting of BMS on right iliac and DES on left iliac and animals were sacrified 4 weeks later after angiography for histologic examination. Results: Balloon iliac injuries (BI) revealed a significant higher Schwartz injury score (0.599±0.368 control vs 1.150±0.306 BI, p=0.013), percentage of stenosis (25.7±19.1 control vs 49.9±21.9 BI, p=0.012) and ratio collagen (0.252 ± 0.017 control vs 0.365 ± 0.011 BI, p=0.0001) than atherosclerotic diet only. Five rabbits died before the end of the protocol. After appaired comparison of BMS and DES, results were significantly better for BMS and pre-euthanasia angiography revealed that 3/7 DES were thrombosed versus 0/7 for BMS. Conclusions : Association of atherogenic diet and balloon injuries allow to obtain important neointimal hyperplasia and wall remodeling in rabbit iliac arteries that confirm its interest as in stent restenosis model for new DES
Gignac, Catherine. "Prévalence de la maladie artérielle périphérique dépistée lors d'une épreuve d'effort dans un centre de cardiologie tertiaire". Thesis, Université Laval, 2007. http://www.theses.ulaval.ca/2007/24201/24201.pdf.
Pełny tekst źródłaLétourneau-Montminy, Myriam. "Facteurs influençant la prescription de la médication cardioprotectrice recommandée chez les patients avec une maladie artérielle périphérique". Master's thesis, Université Laval, 2015. http://hdl.handle.net/20.500.11794/27130.
Pełny tekst źródłaIntroduction: Guidelines recommend that patients with peripheral arterial disease (PAD) should be medically treated to reduce the occurrence of serious cardiovascular events. Objective: To identified factors associated with the prescription of preventive therapies in patients with PAD. Methods: Consecutive patients with PAD (n=362) who underwent peripheral percutaneous transluminal angioplasty between 2008 and 2010 in one tertiary care center (CHU de Quebec, Canada) were considered for the study. Data were collected from the medical charts. The main outcome was the combined prescription of three therapies. Results: A total of 52% of the patients received the three combined therapies. Having at least three cardiovascular risk factors (Odds Ratio (OR)=4.51; 95% CI: 2.76-7.37) was the factor most strongly associated with the prescription of the combined therapies. Conclusion: Canadian patients with PAD are still not optimally managed. We still need to better understand the barriers and facilitators to the application of the guidelines in PAD patients.
Droupy, Stéphane. "Approches anatomique et fonctionnelle, expérimentale et clinique de la vascularisation artérielle du pénis". Paris 11, 2000. http://www.theses.fr/2000PA11T044.
Pełny tekst źródłaMarlinge, Marion. "Profil du récepteur de l’adénosine A2A dans les pathologies cardio-vasculaires". Thesis, Aix-Marseille, 2019. http://www.theses.fr/2019AIXM0670.
Pełny tekst źródłaAtherosclerosis is responsible for the decrease in the diameter of the vessels by formation of a "plaque" consisting in particular of lipids limiting the blood circulation (ischemia) and tissue oxygenation. Adenosine is able to regulate cardiovascular function, particularly through its A2A receptor, which induces vasodilation to increase blood intake. A low presence of A2AR seems to mark ischemia (without specificity of territory) while the presence of reserve receptors (maximum biological response despite a small number of occupied sites) sign a severe disturbance in the blood flow coronary (inducible ischemia). These analyzes can be done on a classic blood sample. Blood adenosine could predict the risk of death from cardiogenic shock complicating the initial disease, where peripheral organs are hypoperfused (cardiac pump dysfunction) that the body attempts to compensate for by vasoconstriction (less A2AR). This work suggests the possibility of using the adenosinergic system both at the level of diagnosis (absence of biological marker of ischemia and coronary artery disease reliable to date), prognosis, than at the therapeutic level
Pottecher, Julien. "Muscle squelettique et ischémie-reperfusion expérimentale des membres : mécanismes impliqués dans la protection ou les effets délétères de la cyclosporine et facteurs limitant les conditionnements pharmacologique et ischémique". Phd thesis, Université de Strasbourg, 2012. http://tel.archives-ouvertes.fr/tel-00871846.
Pełny tekst źródła"Prévalence de la maladie artérielle périphérique dépistée lors d'une épreuve d'effort dans un centre de cardiologie tertiaire". Thesis, Université Laval, 2007. http://www.theses.ulaval.ca/2007/24201/24201.pdf.
Pełny tekst źródłaCzęści książek na temat "Maladie artérielle périphérique – chirurgie"
Stouffer, George A., Leslie P. Wong i Marschall S. Runge. "Maladie artérielle périphérique". W Médecine interne de Netter, 234–42. Elsevier, 2011. http://dx.doi.org/10.1016/b978-2-294-70951-7.00031-1.
Pełny tekst źródła