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Artykuły w czasopismach na temat "Lipotrope"
Fardet, Anthony, Jean-François Martin i Jean-Michel Chardigny. "Lipotropic capacity of raw plant-based foods: A new index that reflects their lipotrope density profile". Journal of Food Composition and Analysis 24, nr 7 (listopad 2011): 895–915. http://dx.doi.org/10.1016/j.jfca.2011.03.013.
Pełny tekst źródłaMoon, Yang S., Wanda L. Keller i Chung S. Park. "Dietary lipotrope-mediated mammary carcinogenesis in female rats". Nutrition Research 18, nr 9 (wrzesień 1998): 1605–14. http://dx.doi.org/10.1016/s0271-5317(98)00134-1.
Pełny tekst źródłaRogers, Adrianne E., Rizwan Akhtar i Steven H. Zeisel. "Procarbazine carcinogenicity in methotrexate-treated or lipotrope-deficient male rats". Carcinogenesis 11, nr 9 (1990): 1491–95. http://dx.doi.org/10.1093/carcin/11.9.1491.
Pełny tekst źródłaChoi, Chang B., Myung G. Baik, Wanda L. Keller i Chung S. Park. "Lipotrope‐modified diets enhance nitrosomethylurea‐induced mammary carcinogenesis in female rats". Nutrition and Cancer 20, nr 3 (1.01.1993): 215–21. http://dx.doi.org/10.1080/01635589309514289.
Pełny tekst źródłaWainfan, Elsie, Mark Dizik, Margaret Hluboky i M. Earl Balis. "Altered tRNA methylation in rats and mice fed lipotrope-deficient diets". Carcinogenesis 7, nr 3 (1986): 473–76. http://dx.doi.org/10.1093/carcin/7.3.473.
Pełny tekst źródłaMoon, Y. S., C. S. Park i S. H. Kim. "Lipotrope-mediatedc-jun and ornithine decarboxylase mRNA expression in mammary acinar cells". In Vitro Cellular & Developmental Biology - Animal 32, nr 6 (czerwiec 1996): 313–14. http://dx.doi.org/10.1007/bf02722954.
Pełny tekst źródłavan Helden, Paul D., Albertus D. Beyers, André J. Bester i Kasimir Jaskiewicz. "Esophageal cancer: Vitamin and lipotrope deficiencies in an at‐risk South African population". Nutrition and Cancer 10, nr 4 (styczeń 1987): 247–55. http://dx.doi.org/10.1080/01635588709513962.
Pełny tekst źródłaSaito, Reisuke, Elizabeth Jahnke-Spinnenweber, Hisashi Shinozuka i Benito Lombardi. "On the role of compensatory mitogenesis in the hepatocarcinogencity of choline and multiple-lipotrope devoid diets". Carcinogenesis 15, nr 7 (1994): 1413–19. http://dx.doi.org/10.1093/carcin/15.7.1413.
Pełny tekst źródłaTrimble, Kenneth C., Anne M. Molloy, John M. Scot i Donald G. Weir. "The effect of ethanol on one-carbon metabolism: Increased methionine catabolism and lipotrope methyl-group wastage". Hepatology 18, nr 4 (październik 1993): 984–89. http://dx.doi.org/10.1002/hep.1840180433.
Pełny tekst źródłaBoyd, Juanell N., Elizabeth S. Graham, Thomas C. Graham i Bud C. Tennant. "A Comparison of the Response of Woodchucks and Rats to Variations in Dietary Lipotrope and Lipid Content". Journal of Nutrition 115, nr 9 (1.09.1985): 1136–46. http://dx.doi.org/10.1093/jn/115.9.1136.
Pełny tekst źródłaRozprawy doktorskie na temat "Lipotrope"
Braud, Laura. "Effets lipotropes des molécules antioxydantes du thé (Camellia sinensis)". Thesis, Toulon, 2015. http://www.theses.fr/2015TOUL0014/document.
Pełny tekst źródłaNon-alcoholic fatty liver disease (NAFLD) is the most common chronic liver disease in industrializedcountries because being strongly associated with the development of metabolic syndrome and relateddyslipidemia. To date, the mechanisms of pathology remain poorly defined and available therapeutic meanshave moderate efficacy. Epidemiological studies have reported a beneficial effect of tea consumption in thefight against liver disorders and cardiovascular risk factors such as dyslipidemia. However, the mechanismsby which a blend of green tea, oolong tea and Pu-erh tea, Hao Ling tea, reduces fatty liver and dyslipidemiaremain unknown. Therefore, the objective of this thesis was to evaluate the effects and mechanisms of actionof Hao Ling tea on NAFLD and dyslipidemia, through two approaches, one on cellular model and the other onanimal model. Our results show that Hao Ling tea reduces the hepatic lipogenesis in vitro and in vivo and thusattenuates steatosis induced by a high fat-high sucrose diet in a rat model. We observed that this tea improvesthe blood lipid profile by increasing plasma HDL levels. We were also able to highlight that tea ownsantioxidant and hepato-protective properties to counteract an inducer of oxidative stress in vitro and todecrease lipid peroxidation in vivo. Finally, we have shown that the oxidative stress per se resulted in anaccumulation of intracellular lipid in isolated hepatocytes and that the tea, due to its antioxidant properties,prevented this phenomenon. The Hao Ling tea is a good nutritional approach in preventing NAFLD and tomaintain LDL/HDL ratio
Pons, Olivier. "Infusions de thés et santé : Dyslipidémie ˸ protocole d'étude du thé Hao Ling® chez des sujets atteints d'hypercholestérolémie. : Effet hypolipémiant de Camellia sinensis (Hao Ling® Tea) chez des sujets atteints d'hypercholestérolémie non traités par statine : protocole d'étude pour un essai randomisé, en double aveugle, contrôlé par placebo". Electronic Thesis or Diss., Toulon, 2023. http://www.theses.fr/2023TOUL1001.
Pełny tekst źródłaLipotropic effects of antioxidant molecules of tea (Camellia sinensis) on the most common chronic liver disease in industrialized countries because being strongly associated with the development related dyslipidaemia. To date, the mechanisms of pathology remain poorly defined and available therapeutic means have moderate efficacy. Epidemiological studies have reported a beneficial effect of tea consumption in the fight against liver disorders and cardiovascular risk factors such as dyslipidaemia. However, the mechanisms by which a blend of green tea, oolong tea and Pu-erh tea, Hao Ling tea, reduces fatty liver and dyslipidaemia remain unknown. Therefore, the objective of this thesis was to evaluate the effects and mechanisms of action of Hao Ling tea on cholesterol and dyslipidaemia, through two approaches, one on cellular model and the other on animal model. Our results show that Hao Ling tea reduces the hepatic lipogenesis in vitro and in vivo and thus attenuates steatosis induced by a high fat-high sucrose diet in a rat model. We observed that this tea improves the blood lipid profile by increasing plasma HDL levels. We were also able to highlight that tea owns antioxidant and hepato-protective properties to counteract an inducer of oxidative stress in vitro and to decrease lipid peroxidation in vivo. Finally, we have shown that the oxidative stress per se resulted in an accumulation of intracellular lipid in isolated hepatocytes and that the tea, due to its antioxidant properties, prevented this phenomenon. The Hao Ling tea is a good nutritional approach in preventing Cholestérol and to maintain LDL/HDL ratio
JEANNIN, CHRISTINE. "Beta-lipotropine et homeostasie glucidique". Besançon, 1991. http://www.theses.fr/1991BESA3058.
Pełny tekst źródłaLindner, Ulrike. "Nachweis spezifischer Bindungsstellen für das Hypophysenhormon [beta]H-Lipotropin [Beta-H-Lipotropin] auf dem Adhäsionsmolekül Vitronektin und auf Monozyten der humanen Zelllinie THP-1". [S.l.] : [s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=972758186.
Pełny tekst źródłaFrize, Graham. "Understanding the impact of facial lipotrophy insights form evolutionary psychology". Thesis, City University London, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.537591.
Pełny tekst źródłaCho, Kwang Bog. "Role of Lipotropic (Methyl) Nutrients on Mammary Development and Carcinogenesis in Female Rat Offspring". Thesis, North Dakota State University, 2015. https://hdl.handle.net/10365/27898.
Pełny tekst źródłaFoster, Michelle Tranace. "Central nervous system regulation of fat cell lipid mobilization the role of the sympathetic nervous system /". restricted, 2005. http://etd.gsu.edu/theses/available/etd-11162005-154631/.
Pełny tekst źródłaTimothy Bartness, committee chair; Elliott Albers, Ruth Harris , Sarah Pallas, committee members. Electronic text (181 p. : ill.)) : digital, PDF file. Description based on contents viewed July 17, 2007. Includes bibliographical references (p. 148-181).
Groenhagen, Björn [Verfasser]. "Interaktion der Proopiomelanocortin-Fragmente β-Endorphin [Beta-Endorphin] und β-Lipotropin [Beta-Lipotropin] mit Monozyten einer Leukämie-Zelllinie des Menschen, THP-1 / vorgelegt von Björn Groenhagen". 2009. http://d-nb.info/999684264/34.
Pełny tekst źródłaLindner, Ulrike [Verfasser]. "Nachweis spezifischer Bindungsstellen für das Hypophysenhormon βH-Lipotropin [Beta-H-Lipotropin] auf dem Adhäsionsmolekül Vitronektin und auf Monozyten der humanen Zelllinie THP-1 / vorgelegt von Ulrike Lindner". 2004. http://d-nb.info/972758186/34.
Pełny tekst źródłaYou, Jhih-Cyuan, i 游智全. "Lipotropic effect of Spiranthes sinensis extract on the FL83B cells and alcoholic fatty liver mice". Thesis, 2017. http://ndltd.ncl.edu.tw/handle/g5g2en.
Pełny tekst źródła國立東華大學
生命科學系
105
Alcoholic fatty liver disease (AFLD), mainly caused by long-term alcohol consumption and led to the accumulation of fat in liver tissues. Fat can cause oxidative stress and triggers the signal pathway and subsequently accelerates lipid droplet accumulation via the activation of adipogenesis in liver. Furthermore, it will get worse to cirrhosis or hepatoma when AFLD is not impeded shortly. Therefore, the exploration of potential candidate from natural sources for alleviation of AFLD renders interesting issue. Spiranthes sinensis, a folk herb, is still unexplored in phytochemical properties for medicinal value. This study was conducted to investigate the biological activity of S. sinensis extract for applying in FL83B cells in vitro by oil red o staining to measure lipid droplet and the results showed that S. sinesis extract could reduce lipid droplets in alcohol plus oleic acid-induced FL83B cells. S. sinensis extract also significantly reduced lipogensis SREBP-1C and ACC1 mRNA levels by qPCR. Moreover, the alcohol-induced fatty liver C75BL/6 mice was treated with S. sinensis extract to determine the efficacy of alcoholic-fatty liver via lipotropic factors using qPCR and micrographic analysis by H & E staining of liver tissues. The results revealed that the reduction of ACC1 and FASN with the increase of CPT1B, PPARα, and AMPKα with S. sinensis extract treatment resulted in alleviation of fatty liver via Western blot and qPCR. Histopathological review, SS extract slightly reduced lipid droplets in liver of alcohol-induced mice. Taken together, S. sinesis extract possesses lipotropic effect on the alcohol plus oleic-induced FL83B cells and the alcohol-induced mice as well, suggesting S. sinensis extract is evident as a candidate of liver-protection source particularly alcoholic fatty liver.
Książki na temat "Lipotrope"
Hüsgen, Hans. Über eine Lipotrope Quecksilberverbindung. Springer London, Limited, 2014.
Znajdź pełny tekst źródłaLi, Choh Hao. Lipotropin and Related Peptides. Elsevier Science & Technology Books, 2012.
Znajdź pełny tekst źródłaCzęści książek na temat "Lipotrope"
Christman, Judith K. "Lipotrope Deficiency and Persistent Changes in DNA Methylation". W Advances in Experimental Medicine and Biology, 97–106. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-0949-7_9.
Pełny tekst źródłaChristman, Judith K. "Dietary Effects on DNA Methylation: Do They Account for the Hepatocarcinogenic Properties of Lipotrope Deficient Diets?" W Nutrition and Biotechnology in Heart Disease and Cancer, 141–54. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1957-7_13.
Pełny tekst źródłaLi, Choh Hao. "β-Lipotropin". W Neural and Endocrine Peptides and Receptors, 15–22. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5152-8_2.
Pełny tekst źródłaGráf, László. "β-Lipotropin". W Neurochemical Systems, 137–58. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-7018-5_6.
Pełny tekst źródłaNewberne, Paul M. "Lipotropic Factors and Oncogenesis". W Essential Nutrients in Carcinogenesis, 223–51. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-1835-4_18.
Pełny tekst źródłaNewberne, Paul M., i Adrianne E. Rogers. "Lipotropic Factors and Carcinogenesis". W Cancer and Nutrition, 159–85. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4757-9561-5_8.
Pełny tekst źródłaCaderni, G. "The Effect of Phospholipids and Lipotropic Factors on Xenobiotic Toxicity". W Phospholipids, 167–76. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-1364-0_12.
Pełny tekst źródłaNappi, G., F. Facchinetti, G. Bono, F. Petraglia, G. Micieli, A. Volpe i A. R. Genazzani. "Lack of β-Endorphin and β-Lipotropin Circadian Rhythmicity in Episodic Cluster Headache: A Model for Chronopathology". W Updating in Headache, 269–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-88581-5_43.
Pełny tekst źródła"Lipotropin". W Encyclopedia of Genetics, Genomics, Proteomics and Informatics, 1114. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_9491.
Pełny tekst źródła"lipotropic, adj." W Oxford English Dictionary. Wyd. 3. Oxford University Press, 2023. http://dx.doi.org/10.1093/oed/6559047609.
Pełny tekst źródłaStreszczenia konferencji na temat "Lipotrope"
Fardet, Anthony, Jean-François Martin i Jean-Michel Chardigny. "Characterization of the lipotropic potential of plant-based foods". W Foods: Bioactives, Processing, Quality and Nutrition. Basel, Switzerland: MDPI, 2013. http://dx.doi.org/10.3390/bpqn2013-01164.
Pełny tekst źródłaRaporty organizacyjne na temat "Lipotrope"
Park, Chung S. Role of Lipotropes in Mammary Carcinogenesis. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 1996. http://dx.doi.org/10.21236/adb225308.
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