Gotowa bibliografia na temat „Lipophagie”
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Artykuły w czasopismach na temat "Lipophagie"
Zelickson, B. D., i R. K. Winkelmann. "Lipophagic panniculitis in re-excision specimens." Acta Dermato-Venereologica 71, nr 1 (1.01.1991): 59–61. http://dx.doi.org/10.2340/00015555715961.
Pełny tekst źródłaSchott, Micah B., Shaun G. Weller, Ryan J. Schulze, Eugene W. Krueger, Kristina Drizyte-Miller, Carol A. Casey i Mark A. McNiven. "Lipid droplet size directs lipolysis and lipophagy catabolism in hepatocytes". Journal of Cell Biology 218, nr 10 (7.08.2019): 3320–35. http://dx.doi.org/10.1083/jcb.201803153.
Pełny tekst źródłaJonas, Wenke, Kristin Schwerbel, Lisa Zellner, Markus Jähnert, Pascal Gottmann i Annette Schürmann. "Alterations of Lipid Profile in Livers with Impaired Lipophagy". International Journal of Molecular Sciences 23, nr 19 (6.10.2022): 11863. http://dx.doi.org/10.3390/ijms231911863.
Pełny tekst źródłaAlexandrides, C. "Lipophagic Granuloma". Acta Medica Scandinavica 154, S312 (24.04.2009): 449–60. http://dx.doi.org/10.1111/j.0954-6820.1956.tb17036.x.
Pełny tekst źródłaPeng, Peng, Wensheng Liu, Adam Utley, Colin Chavel, Louise Carlson, Scott H. Olejniczak i Kelvin P. Lee. "CD28 Induces Autophagy in Plasma Cells to Enhance Mitochondrial Respiration and Survival". Journal of Immunology 204, nr 1_Supplement (1.05.2020): 71.2. http://dx.doi.org/10.4049/jimmunol.204.supp.71.2.
Pełny tekst źródłaKumar, Ravinder, Muhammad Arifur Rahman i Taras Y. Nazarko. "Nitrogen Starvation and Stationary Phase Lipophagy Have Distinct Molecular Mechanisms". International Journal of Molecular Sciences 21, nr 23 (29.11.2020): 9094. http://dx.doi.org/10.3390/ijms21239094.
Pełny tekst źródłaLevy, Jack, Mark E. Burnett i Cynthia M. Magro. "Lipophagic Panniculitis of Childhood". American Journal of Dermatopathology 39, nr 3 (marzec 2017): 217–24. http://dx.doi.org/10.1097/dad.0000000000000721.
Pełny tekst źródłaUMBERT, I. J., i R. K. WINKELMANN. "Adult lipophagic atrophic panniculitis". British Journal of Dermatology 124, nr 3 (marzec 1991): 291–95. http://dx.doi.org/10.1111/j.1365-2133.1991.tb00578.x.
Pełny tekst źródłaWinkelmann, R. K., Marian T. McEvoy i Margot S. Peters. "Lipophagic panniculitis of childhood". Journal of the American Academy of Dermatology 21, nr 5 (listopad 1989): 971–78. http://dx.doi.org/10.1016/s0190-9622(89)70285-1.
Pełny tekst źródłaJuneja, Manish, Pankaj Raut, Milind Lohkare, Harshawardhan Ramteke, Vaishnavi Walke i Sakshi Bhatia. "Effects of Lipophagy on Atherosclerosis". Central India Journal of Medical Research 2, nr 01 (15.05.2023): 17–25. http://dx.doi.org/10.58999/cijmr.v2i01.44.
Pełny tekst źródłaRozprawy doktorskie na temat "Lipophagie"
Chang, Yu-Chin, i 張毓秦. "Activation of lipophagy protects neurons from neurodegeneration caused by sphingolipid imbalance". Thesis, 2016. http://ndltd.ncl.edu.tw/handle/59910094618794899139.
Pełny tekst źródła國立臺灣大學
生理學研究所
104
Sphingolipids are essential membrane components of the neuron; hence their levels need to be tightly regulated. Infertile crescent (Ifc) is an evolutionarily conserved dihydroceramide (DHC) desaturase which converts DHC to Ceramide (Cer) for the de novo synthesis of Cer in Drosophila. While the imbalance of Cer, a bioactive sphingolipid, has been associated with several neurodegenerative diseases, the neuronal function of its precursor DHC remains unknown. To investigate the role of ifc, we generated ifc knockout flies (ifc-KO). Sphingolipidomic analysis showed that loss of ifc resulted in increased DHC. Prolonged light stimuli to the ifc-KO eye led to activity-dependent degeneration of photoreceptors. Clonal analysis of ifc-KO photoreceptors revealed the accumulation of lipophagic structure and the increased H2DCF signals upon light stimuli, suggesting that DHC accumulation may activate lipophagy and induce the production of reactive oxygen species (ROS). However, it remains to be determined whether the degeneration is attributed to lipophagic cell death or the ROS insults. Reduction of ifc led to the increase of Atg8/LC3 puncta in the acidified compartment and elevation of lysosomal proteases, indicating the activated lipophagy can promote subsequent lysosomal function. ifc-dependent neurodegeneration can be partially rescued by an antioxidant AD4, indicating that ROS is at least partially responsible for the degeneration. In addition, both ROS elevation and lipid accumulation in ifc-KO was suppressed by treating with the autophagy inducer Rapamycin, suggesting that enhanced lipophagy plays a protective role in ifc-dependent neurodegeneration. Conversely, lipophagy can be downregulated by AD4, indicating ROS insults lead to the feedback upregulation of protective lipophagy. In summary, loss of ifc results in DHC accumulation and ROS generation, the latter of which subsequently activates lipophagy to protect against neurodegeneration. These findings support our hypothesis that DHC is bio-active and lipophagy can be protective, highlighting their potential as therapeutic targets for regulating sphingolipid homeostasis.
Christian, Patricia. "Investigating the Role of Autophagy in Intracellular Apolipoprotein B Traffic and Very-low-density-lipoprotein Assembly and Secretion". Thesis, 2013. http://hdl.handle.net/1807/42743.
Pełny tekst źródłaCzęści książek na temat "Lipophagie"
Martinez-Lopez, Nuria. "Regulation of Lipophagy". W Autophagy and Signaling, 147–72. Boca Raton, FL : CRC Press, 2018. | Series: Methods in signal transduction series: CRC Press, 2017. http://dx.doi.org/10.1201/9781315120638-10.
Pełny tekst źródłaSteinberg, Christian E. W. "Lipid Homeostasis and Lipophagy—‘The Greasy Stuff Balanced’". W Aquatic Animal Nutrition, 583–97. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-030-87227-4_24.
Pełny tekst źródłaMagro, Cynthia M., i Josh H. Mo. "Lipophagic/Lipoatrophic Panniculitis: A TH1-Mediated Autoimmune Disorder of the Subcutaneous Fat". W New and Emerging Entities in Dermatology and Dermatopathology, 277–86. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-80027-7_21.
Pełny tekst źródłaSathyanarayan, Aishwarya. "A Coupled Approach Utilizing Immunohistochemistry and Immunocytochemistry to Visualize Cellular Lipophagy". W Methods in Molecular Biology, 185–91. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6759-9_11.
Pełny tekst źródłaSathyanarayan, Aishwarya. "Erratum to: A Coupled Approach Utilizing Immunohistochemistry and Immunocytochemistry to Visualize Cellular Lipophagy". W Methods in Molecular Biology, E1. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6759-9_21.
Pełny tekst źródła"Lipophagic panniculitis of childhood". W Dermatology Therapy, 358. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/3-540-29668-9_1645.
Pełny tekst źródłaCristobal-Sarramian, A., M. Radulovic i S. D. Kohlwein. "Methods to Measure Lipophagy in Yeast". W Methods in Enzymology, 395–412. Elsevier, 2017. http://dx.doi.org/10.1016/bs.mie.2016.09.087.
Pełny tekst źródłaDutta, Shweta, Saraswati Prasad Mishra, Anil Kumar Sahu, Koushlesh Mishra, Pankaj Kashyap i Bhavna Sahu. "Hepatocytes and Its Role in Metabolism". W Drug Metabolism [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99083.
Pełny tekst źródłaRoccio, Federica, Aurore Claude-Taupin, Joëlle Botti, Etienne Morel, Patrice Codogno i Nicolas Dupont. "Monitoring lipophagy in kidney epithelial cells in response to shear stress". W Methods in Cell Biology. Elsevier, 2021. http://dx.doi.org/10.1016/bs.mcb.2020.12.003.
Pełny tekst źródłaEsmaeilian, Yashar, Sevgi Yusufoglu, Ece Iltumur, Gamze Bildik i Ozgur Oktem. "Visualizing Lipophagy as a New Mechanism of the Synthesis of Sex Steroids in Human Ovary and Testis Using Immunofluorescence Staining Method". W Methods in Molecular Biology. New York, NY: Springer US, 2024. http://dx.doi.org/10.1007/7651_2024_520.
Pełny tekst źródłaStreszczenia konferencji na temat "Lipophagie"
Irungbam, K., Y. Churin, M. Ocker, M. Roderfeld i E. Roeb. "CB1 knockout alleviates hepatic steatosis via lipophagy and lipolysis in HBs transgenic mice". W 35. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0038-1677164.
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