Gotowa bibliografia na temat „Lipid anabolism”
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Artykuły w czasopismach na temat "Lipid anabolism"
Krycer, James R., Lake-Ee Quek, Deanne Francis, Armella Zadoorian, Fiona C. Weiss, Kristen C. Cooke, Marin E. Nelson i in. "Insulin signaling requires glucose to promote lipid anabolism in adipocytes". Journal of Biological Chemistry 295, nr 38 (28.07.2020): 13250–66. http://dx.doi.org/10.1074/jbc.ra120.014907.
Pełny tekst źródłaSanchez-Alvarez, Miguel, Qifeng Zhang, Fabian Finger, Michael J. O. Wakelam i Chris Bakal. "Cell cycle progression is an essential regulatory component of phospholipid metabolism and membrane homeostasis". Open Biology 5, nr 9 (wrzesień 2015): 150093. http://dx.doi.org/10.1098/rsob.150093.
Pełny tekst źródłaBensinger, Steven, Yoko Kidani, M. Benjamin Hock, Joseph Argus, Evangelia Komisopoulou i Thomas Graeber. "An essential role for SREBP signaling in T cell blastogenesis (47.17)". Journal of Immunology 188, nr 1_Supplement (1.05.2012): 47.17. http://dx.doi.org/10.4049/jimmunol.188.supp.47.17.
Pełny tekst źródłaWang, Shuang, Rasool Kamal, Yue Zhang, Renhui Zhou, Liting Lv, Qitian Huang, Siriguleng Qian, Sufang Zhang i Zongbao Kent Zhao. "Expression of VHb Improved Lipid Production in Rhodosporidium toruloides". Energies 13, nr 17 (27.08.2020): 4446. http://dx.doi.org/10.3390/en13174446.
Pełny tekst źródłaDe Oliveira, Matheus Pinto, i Marc Liesa. "The Role of Mitochondrial Fat Oxidation in Cancer Cell Proliferation and Survival". Cells 9, nr 12 (4.12.2020): 2600. http://dx.doi.org/10.3390/cells9122600.
Pełny tekst źródłaSong, Rui, Mengxiao Hu, Xiyu Qin, Lili Qiu, Pengjie Wang, Xiaoxu Zhang, Rong Liu i Xiaoyu Wang. "The Roles of Lipid Metabolism in the Pathogenesis of Chronic Diseases in the Elderly". Nutrients 15, nr 15 (3.08.2023): 3433. http://dx.doi.org/10.3390/nu15153433.
Pełny tekst źródłaStuani, Lucille, Fabien Riols, Pierre Millard, Marie Sabatier, Aurélie Batut, Estelle Saland, Fanny Viars i in. "Stable Isotope Labeling Highlights Enhanced Fatty Acid and Lipid Metabolism in Human Acute Myeloid Leukemia". International Journal of Molecular Sciences 19, nr 11 (25.10.2018): 3325. http://dx.doi.org/10.3390/ijms19113325.
Pełny tekst źródłaMacKinnon, Barbara M., i D. L. Lee. "Age-related changes in Heligmosomoides polygyrus (Nematoda): neutral lipid content in developing oocytes". Canadian Journal of Zoology 66, nr 12 (1.12.1988): 2791–96. http://dx.doi.org/10.1139/z88-407.
Pełny tekst źródłaLiu, L., Y. Yang, F. Yang, Y. Lin, K. Liu, X. Wang i Y. Zhang. "A mechanistic investigation about hepatoxic effects of borneol using zebrafish". Human & Experimental Toxicology 42 (2.01.2023): 096032712211490. http://dx.doi.org/10.1177/09603271221149011.
Pełny tekst źródłaAltumairah, Mohammed A. H., i Ravindra P. Choudhary. "Overview on Diabetes Mellitus". Journal of Medical and Health Studies 2, nr 2 (21.09.2021): 63–69. http://dx.doi.org/10.32996/jmhs.2021.2.2.7.
Pełny tekst źródłaRozprawy doktorskie na temat "Lipid anabolism"
Liang, Chao. "Aptamer-functionalized lipid nanoparticles targeting osteoblasts as a novel RNA Interference-based bone anabolic strategy". HKBU Institutional Repository, 2016. https://repository.hkbu.edu.hk/etd_oa/325.
Pełny tekst źródłaSander, Simone [Verfasser], i Britta [Akademischer Betreuer] Brügger. "Relevance of ACSL3-mediated ACS activity and ACSL3 localization in anabolic and catabolic lipid droplet metabolism / Simone Sander ; Betreuer: Britta Brügger". Heidelberg : Universitätsbibliothek Heidelberg, 2019. http://d-nb.info/1177695626/34.
Pełny tekst źródłaMartins, Danieli Brolo. "SISTEMA COLINÉRGICO E PEROXIDAÇÃO LIPÍDICA DE RATOS TRATADOS COM SULFATO DE VINCRISTINA E DECANOATO DE NANDROLONA". Universidade Federal de Santa Maria, 2008. http://repositorio.ufsm.br/handle/1/10011.
Pełny tekst źródłaVincristine sulphate is antitumor agent widely used in small animal clinical oncology; therefore it can cause a number of adverse effects including marrow and neuronal cytotoxicity. Nandrolone decanoate, an anabolic-androgenic steroid (AAS), has been used in association with vincristine in order to ease effects such as moderate myelosuppression. The present dissertation presents data related to the isolated or associated employment of vincristine sulphate and nandrolone decanoate, as well as their effects on the cholinergic system and oxidative profile of Wistar rats. The animals were submitted to four different doses of AAS for three weeks and its action on acetylcholinesterase (AChE) activity in four different structures of brain tissue (cerebellum, hippocampus, striatum and cerebral cortex) was studied. The second experiment shows the effects of vincristine and/or nandrolone decanoate in the brain (same parts studied previously) and blood through measurement of brain and red blood cell AChE (RBC-AChE) enzyme activity as well as brain and blood serum lipid peroxidation of rats treated for two weeks. Results show that the two highest doses used in the first experiment increased enzyme activity, suggesting interference in the cholinergical system of the striatum and cerebellum. The results obtained in the latter experiment demonstrate that the isolated use of this AAS and its association with vincristine sulphate altered brain and RBC-AChE action, both in a stimulatory and inhibitory fashion. Lipid peroxidation, in the brain and blood, increased due to the isolated use of both vincristine and nandrolone decanoate, as well as to their associated use at the highest dose of ester used. Furthermore, the data show that the association between the therapeutic dose of nandrolone decanoate and vincristine is capable of neutralizing the free radical production induced by their isolated use in brain and blood serum. Serum RBC-AChE activity and the oxidative profile presented in this study are similar to those exhibited for brain tissue. Based on these data, it can be concluded that nandrolone decanoate is capable of interfering in AChE activity, affecting the cholinergic system, which could cause an alteration of its neurotransmitter, as well as a low or high stimulation of post-synaptic receptors. Therefore, the use of the therapeutic dose of AAS studied here in association with vincristine has been shown to be beneficial, as it could protect the organism from damaging processes caused by the production of free radicals.
O sulfato de vincristina é um agente anti-tumoral bastante usado na oncologia clínica de pequenos animais, porém seus efeitos colaterais incluem citotoxicidade medular e neuronal. O decanoato de nandrolona, um esteróide anabólico androgênico (EAA), tem sido usado em associação a este medicamento para amenizar alguns de seus efeitos, como a mielossupressão moderada. Esta dissertação apresenta dados referentes ao uso isolado ou associado do sulfato de vincristina e do decanoato de nandrolona, seus efeitos no sistema colinérgico e no perfil oxidativo de ratos Wistar. Primeiramente, verificou-se quatro diferentes doses do EAA, por três semanas, e sua ação sobre a atividade da acetilcolinesterase (AChE) em quatro partes do tecido cerebral (cerebelo, estriado, hipocampo e córtex cerebral). O segundo experimento apresenta grupos tratados durante duas semanas com sulfato de vincristina e/ou decanoato de nandrolona e as ações destes no cérebro (mesmas partes pesquisadas anteriormente) e no sangue, através da mensuração da atividade enzimática da AChE cerebral e eritrocitária (RBC-AChE) e peroxidação lipídica cerebral e sérica. As duas doses mais altas utilizadas no primeiro trabalho aumentam a atividade da enzima, sugerindo que haja interferência no sistema colinérgico, no estriado e no cerebelo. Os resultados obtidos, no estudo posterior, demonstram que o uso isolado deste EAA e suas associações com o sulfato de vincristina alteram a ação da AChE cerebral e RBC-AChE, tanto de forma estimulatória quanto inibitória. A peroxidação lipídica, cerebral e sangüínea, aumenta devido ao uso isolado da vincristina e do decanoato de nandrolona, e na associação do quimioterápico com a dose mais alta usada do éster. A dose terapêutica do decanoato de nandrolona e a vincristina utilizadas são capazes de neutralizar a produção de radicais livres tanto no cérebro quanto no soro sangüíneo. A atividade da RBC-AChE e o valor do perfil oxidativo do soro apresentados nesta pesquisa são similares àqueles exibidos pelo tecido cerebral. Diante destes dados, pode-se concluir que o decanoato de nandrolona é capaz de influenciar a atividade da AChE, afetando o sistema colinérgico, o que poderia ocasionar em uma ação alterada do seu neurotransmissor, além de uma baixa ou alta estimulação dos receptores póssinápticos. Entretanto, o uso da dose terapêutica estudada deste EAA associada à vincristina mostra-se benéfico, pois poderia proteger o organismo de processos prejudiciais relacionados a produção de radicais livres.
Shee, Somnath. "Manipulating Bacterial and Host Reactive Oxygen Species (ROS)- based mechanisms to potentiate killing of Mycobacterium tuberculosis (Mtb)". Thesis, 2021. https://etd.iisc.ac.in/handle/2005/5680.
Pełny tekst źródłaWelcome-DBT
Smith, Kerry Ruth. "The effect of anabolic implant and dietary lipid source on intramusuclar lipid deposition in finished beef cattle". 2004. http://purl.galileo.usg.edu/uga%5Fetd/smith%5Fkerry%5Fr%5F200405%5Fphd.
Pełny tekst źródłaLong, Jared Woodrow. "Effects of supplemental lipid and anabolic growth implants on carcass characteristics, meat quality, and fatty acid composition of finishing beef cattle". 2005. http://purl.galileo.usg.edu/uga%5Fetd/long%5Fjared%5Fw%5F200512%5Fms.
Pełny tekst źródłaKsiążki na temat "Lipid anabolism"
Casaer, Michael P., i Greet Van den Berghe. Nutrition support in acute cardiac care. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0032.
Pełny tekst źródłaCasaer, Michael P., i Greet Van den Berghe. Nutrition support in acute cardiac care. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0032_update_001.
Pełny tekst źródłaCasaer, Michael P., i Greet Van den Berghe. Nutrition support in acute cardiac care. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0032_update_002.
Pełny tekst źródłaCzęści książek na temat "Lipid anabolism"
Saito, Mariko, i Abraham Rosenberg. "Regulation of Glycosphingolipid Anabolism in Fibroblasts by Ionophores, Plasma Membrane ATP-ase Inhibition, and Growth Factors". W Enzymes of Lipid Metabolism II, 591–95. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5212-9_73.
Pełny tekst źródłaLackner, K. J., W. März, G. Wolter, H. Sartor i W. Gross. "Anabolic Steroids do not Change mRNA Levels and Protein Secretion of Apolipoprotein A-I and B-100 in HepG2 Cells". W Recent Developments in Lipid and Lipoprotein Research, 151–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-84855-1_18.
Pełny tekst źródłaSasako, Takayoshi, i Kohjiro Ueki. "ER Stress Response Failure and Steatohepatitis Comorbid with Diabetes". W Psychology and Patho-physiological Outcomes of Eating [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.100054.
Pełny tekst źródłaClark, Gregory O., i William J. Kovacs. "Glucose, Lipid, and Protein Metabolism". W Textbook of Endocrine Physiology. Oxford University Press, 2011. http://dx.doi.org/10.1093/oso/9780199744121.003.0018.
Pełny tekst źródłaInfante, Marco. "The Insulin Journey in the Human Body". W Evolving Concepts in Insulin Resistance [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.107906.
Pełny tekst źródła"Metabolic Logic through the Lens of Coenzyme Forms of Human Vitamins". W The Chemical Biology of Human Vitamins, 40–70. The Royal Society of Chemistry, 2018. http://dx.doi.org/10.1039/bk9781788014649-00040.
Pełny tekst źródłaCasaer, Michael P., i Greet Van den Berghe. "Nutrition support in acute cardiac care". W The ESC Textbook of Intensive and Acute Cardiovascular Care, redaktorzy Marco Tubaro, Pascal Vranckx, Eric Bonnefoy-Cudraz, Susanna Price i Christiaan Vrints, 360–72. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198849346.003.0030.
Pełny tekst źródła"Cellular metabolism". W Oxford Assess and Progress: Medical Sciences, redaktorzy Jade Chow, John Patterson, Kathy Boursicot i David Sales. Oxford University Press, 2012. http://dx.doi.org/10.1093/oso/9780199605071.003.0014.
Pełny tekst źródłaRaporty organizacyjne na temat "Lipid anabolism"
Corscadden, Louise, i Anjali Singh. Metabolism And Measurable Metabolic Parameters. ConductScience, grudzień 2022. http://dx.doi.org/10.55157/me20221213.
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