Artykuły w czasopismach na temat „Kv7.2/3”
Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych
Sprawdź 47 najlepszych artykułów w czasopismach naukowych na temat „Kv7.2/3”.
Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.
Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.
Przeglądaj artykuły w czasopismach z różnych dziedzin i twórz odpowiednie bibliografie.
Hernandez, Ciria C., Björn Falkenburger i Mark S. Shapiro. "Affinity for phosphatidylinositol 4,5-bisphosphate determines muscarinic agonist sensitivity of Kv7 K+ channels". Journal of General Physiology 134, nr 5 (26.10.2009): 437–48. http://dx.doi.org/10.1085/jgp.200910313.
Pełny tekst źródłaMiceli, Francesco, Maria V. Soldovieri, Paolo Ambrosino, Laura Manocchio, Ilaria Mosca i Maurizio Taglialatela. "Pharmacological Targeting of Neuronal Kv7.2/3 Channels: A Focus on Chemotypes and Receptor Sites". Current Medicinal Chemistry 25, nr 23 (4.07.2018): 2637–60. http://dx.doi.org/10.2174/0929867324666171012122852.
Pełny tekst źródłaPeretz, Asher, Anton Sheinin, Cuiyong Yue, Nurit Degani-Katzav, Gilad Gibor, Rachel Nachman, Anna Gopin i in. "Pre- and Postsynaptic Activation of M-Channels By a Novel Opener Dampens Neuronal Firing and Transmitter Release". Journal of Neurophysiology 97, nr 1 (styczeń 2007): 283–95. http://dx.doi.org/10.1152/jn.00634.2006.
Pełny tekst źródłaWright, Andrew B., Khrystyna Yu Sukhanova i Keith S. Elmslie. "KV7 channels are potential regulators of the exercise pressor reflex". Journal of Neurophysiology 126, nr 1 (1.07.2021): 1–10. http://dx.doi.org/10.1152/jn.00700.2020.
Pełny tekst źródłaBarro-Soria, Rene. "Effects of small molecules on neurodevelopmental disorder-associated Kv7.2/3 mutations". Biophysical Journal 123, nr 3 (luty 2024): 528a. http://dx.doi.org/10.1016/j.bpj.2023.11.3192.
Pełny tekst źródłaPeretz, Asher S., Eti Patrich, Polina Kornilov, Nataly Menaker i Bernard Attali. "A Novel Compound Targeting Kv7.2/3 Channels Relieves Inflammatory and Neuropathic Pain". Biophysical Journal 106, nr 2 (styczeń 2014): 141a. http://dx.doi.org/10.1016/j.bpj.2013.11.820.
Pełny tekst źródłaLiu, Wenjing, i Jérôme J. Devaux. "Calmodulin orchestrates the heteromeric assembly and the trafficking of KCNQ2/3 (Kv7.2/3) channels in neurons". Molecular and Cellular Neuroscience 58 (styczeń 2014): 40–52. http://dx.doi.org/10.1016/j.mcn.2013.12.005.
Pełny tekst źródłaSurur, Abdrrahman S., Christian Bock, Kristin Beirow, Konrad Wurm, Lukas Schulig, Markus K. Kindermann, Werner Siegmund, Patrick J. Bednarski i Andreas Link. "Flupirtine and retigabine as templates for ligand-based drug design of KV7.2/3 activators". Organic & Biomolecular Chemistry 17, nr 18 (2019): 4512–22. http://dx.doi.org/10.1039/c9ob00511k.
Pełny tekst źródłaLi, S., V. Choi i T. Tzounopoulos. "Pathogenic plasticity of Kv7.2/3 channel activity is essential for the induction of tinnitus". Proceedings of the National Academy of Sciences 110, nr 24 (28.05.2013): 9980–85. http://dx.doi.org/10.1073/pnas.1302770110.
Pełny tekst źródłaMiranda, Pablo, Alba Cadaveira-Mosquera, Rafaela Gonzalez-Montelongo, Alvaro Villarroel, Jose Antonio Lamas, Diego Alvarez de la Rosa i Teresa Giraldez. "Regulation of the Kv7.2/3 Channels by the Neuronal Serum-and Gluococorticoids-Regulated Kinase 1.1". Biophysical Journal 104, nr 2 (styczeń 2013): 268a. http://dx.doi.org/10.1016/j.bpj.2012.11.1505.
Pełny tekst źródłaSander, Svenja E., Catherine Lambrecht i Angelika Richter. "The KV7.2/3 preferring channel opener ICA 27243 attenuates L-DOPA-induced dyskinesia in hemiparkinsonian rats". Neuroscience Letters 545 (czerwiec 2013): 59–63. http://dx.doi.org/10.1016/j.neulet.2013.04.017.
Pełny tekst źródłaPablo, Juan Lorenzo, i Geoffrey S. Pitt. "FGF14 is a regulator of KCNQ2/3 channels". Proceedings of the National Academy of Sciences 114, nr 1 (19.12.2016): 154–59. http://dx.doi.org/10.1073/pnas.1610158114.
Pełny tekst źródłaLee, Inn-Chi, Jiann-Jou Yang, Ying-Ming Liou i Swee-Hee Wong. "KCNQ2 Selectivity Filter Mutations Cause Kv7.2 M-Current Dysfunction and Configuration Changes Manifesting as Epileptic Encephalopathies and Autistic Spectrum Disorders". Cells 11, nr 5 (5.03.2022): 894. http://dx.doi.org/10.3390/cells11050894.
Pełny tekst źródłaRoeloffs, Rosemarie, Alan D. Wickenden, Christopher Crean, Stephen Werness, Grant McNaughton-Smith, James Stables, James O. McNamara, Neil Ghodadra i Greg C. Rigdon. "In Vivo Profile of ICA-27243 [N-(6-Chloro-pyridin-3-yl)-3,4-difluoro-benzamide], a Potent and Selective KCNQ2/Q3 (Kv7.2/Kv7.3) Activator in Rodent Anticonvulsant Models". Journal of Pharmacology and Experimental Therapeutics 326, nr 3 (24.06.2008): 818–28. http://dx.doi.org/10.1124/jpet.108.137794.
Pełny tekst źródłaEl-Chemali, Léa El, Suzan Boutary, Song Liu, Guo-Jun Liu, Ryan J. Middleton, Richard B. Banati, Gregor Bahrenberg, Rainer Rupprecht, Michael Schumacher i Liliane Massaad-Massade. "GRT-X Stimulates Dorsal Root Ganglia Axonal Growth in Culture via TSPO and Kv7.2/3 Potassium Channel Activation". International Journal of Molecular Sciences 25, nr 13 (3.07.2024): 7327. http://dx.doi.org/10.3390/ijms25137327.
Pełny tekst źródłaSedivy, Vojtech, Shreena Joshi, Youssef Ghaly, Roman Mizera, Marie Zaloudikova, Sean Brennan, Jana Novotna, Jan Herget i Alison M. Gurney. "Role of Kv7 channels in responses of the pulmonary circulation to hypoxia". American Journal of Physiology-Lung Cellular and Molecular Physiology 308, nr 1 (1.01.2015): L48—L57. http://dx.doi.org/10.1152/ajplung.00362.2013.
Pełny tekst źródłaWu, Yong-Jin, Charles M. Conway, Li-Qiang Sun, Frederic Machet, Jie Chen, Ping Chen, Huan He i in. "Discovery of (S,E)-3-(2-fluorophenyl)-N-(1-(3-(pyridin-3-yloxy)phenyl)ethyl)-acrylamide as a potent and efficacious KCNQ2 (Kv7.2) opener for the treatment of neuropathic pain". Bioorganic & Medicinal Chemistry Letters 23, nr 22 (listopad 2013): 6188–91. http://dx.doi.org/10.1016/j.bmcl.2013.08.092.
Pełny tekst źródłaDickson, Eamonn J., Björn H. Falkenburger i Bertil Hille. "Quantitative properties and receptor reserve of the IP3 and calcium branch of Gq-coupled receptor signaling". Journal of General Physiology 141, nr 5 (29.04.2013): 521–35. http://dx.doi.org/10.1085/jgp.201210886.
Pełny tekst źródłaFalkenburger, Björn H., Eamonn J. Dickson i Bertil Hille. "Quantitative properties and receptor reserve of the DAG and PKC branch of Gq-coupled receptor signaling". Journal of General Physiology 141, nr 5 (29.04.2013): 537–55. http://dx.doi.org/10.1085/jgp.201210887.
Pełny tekst źródłaAnta, Begoña, Carlos Martín-Rodríguez, Carolina Gomis-Perez, Laura Calvo, Saray López-Benito, Andrés A. Calderón-García, Cristina Vicente-García, Álvaro Villarroel i Juan C. Arévalo. "Ubiquitin-specific Protease 36 (USP36) Controls Neuronal Precursor Cell-expressed Developmentally Down-regulated 4-2 (Nedd4-2) Actions over the Neurotrophin Receptor TrkA and Potassium Voltage-gated Channels 7.2/3 (Kv7.2/3)". Journal of Biological Chemistry 291, nr 36 (21.07.2016): 19132–45. http://dx.doi.org/10.1074/jbc.m116.722637.
Pełny tekst źródłaErdem, Fatma A. "Phosphorylation of KV7.2 regulates its PIP2 sensitivity". Intrinsic Activity 3, Suppl. 2 (9.09.2015): A2.21. http://dx.doi.org/10.25006/ia.3.s2-a2.21.
Pełny tekst źródłaAndersen, Martin N., Søren-Peter Olesen i Hanne B. Rasmussen. "Kv7.1 surface expression is regulated by epithelial cell polarization". American Journal of Physiology-Cell Physiology 300, nr 4 (kwiecień 2011): C814—C824. http://dx.doi.org/10.1152/ajpcell.00390.2010.
Pełny tekst źródłaTelezhkin, Vsevolod, Alison M. Thomas, Stephen C. Harmer, Andrew Tinker i David A. Brown. "A basic residue in the proximal C-terminus is necessary for efficient activation of the M-channel subunit Kv7.2 by PI(4,5)P2". Pflügers Archiv - European Journal of Physiology 465, nr 7 (6.01.2013): 945–53. http://dx.doi.org/10.1007/s00424-012-1199-3.
Pełny tekst źródłaAndersen, Martin Nybo, Katarzyna Krzystanek, Frederic Petersen, Sofia Hammami Bomholtz, Søren-Peter Olesen, Hugues Abriel, Thomas Jespersen i Hanne Borger Rasmussen. "A Phosphoinositide 3-Kinase (PI3K)-serum- and glucocorticoid-inducible Kinase 1 (SGK1) Pathway Promotes Kv7.1 Channel Surface Expression by Inhibiting Nedd4-2 Protein". Journal of Biological Chemistry 288, nr 52 (8.11.2013): 36841–54. http://dx.doi.org/10.1074/jbc.m113.525931.
Pełny tekst źródłaDai, Shuiping, Duane D. Hall i Johannes W. Hell. "Supramolecular Assemblies and Localized Regulation of Voltage-Gated Ion Channels". Physiological Reviews 89, nr 2 (kwiecień 2009): 411–52. http://dx.doi.org/10.1152/physrev.00029.2007.
Pełny tekst źródłaAmin, Ahmad S., John R. Giudicessi, Anke J. Tijsen, Anne M. Spanjaart, Yolan J. Reckman, Christine A. Klemens, Michael W. Tanck i in. "Variants in the 3′ untranslated region of the KCNQ1-encoded Kv7.1 potassium channel modify disease severity in patients with type 1 long QT syndrome in an allele-specific manner". European Heart Journal 33, nr 6 (23.12.2011): 714–23. http://dx.doi.org/10.1093/eurheartj/ehr473.
Pełny tekst źródłaGiudicessi, J. R., A. S. Amin, A. J. Tjisen, C. A. Klemens, J. D. Kapplinger, N. Hofman, Y. Pinto, A. A. Wilde i M. J. Ackerman. "Modification of Disease Severity by Functional Variants in the 3′ Untranslated Region of the KCNQ1-Encoded Kv7.1 Channel is Most Pronounced in Patients Harboring Dominant-Negative LQT1-Causative Mutations". Heart Rhythm 8, nr 11 (listopad 2011): 1827. http://dx.doi.org/10.1016/j.hrthm.2011.09.050.
Pełny tekst źródłaHaick, Jennifer M., Lioubov I. Brueggemann, Leanne L. Cribbs, Mitchell F. Denning, Jeffrey Schwartz i Kenneth L. Byron. "PKC-dependent regulation of Kv7.5 channels by the bronchoconstrictor histamine in human airway smooth muscle cells". American Journal of Physiology-Lung Cellular and Molecular Physiology 312, nr 6 (1.06.2017): L822—L834. http://dx.doi.org/10.1152/ajplung.00567.2016.
Pełny tekst źródłaSheng, Zhao-Fu, Hua Zhang, PeiRu Zheng, Shanyan Chen, Zezong Gu, Jing-Jing Zhou, Jeffery G. Phaup i in. "Impaired Kv7 channel activity in the central amygdala contributes to elevated sympathetic outflow in hypertension". Cardiovascular Research, 14.02.2021. http://dx.doi.org/10.1093/cvr/cvab031.
Pełny tekst źródłaCelentano, Camilla, Lidia Carotenuto, Francesco Miceli, Giusy Carleo, Brunella Corrado, Giulia Baroli, Stefania Iervolino, Raffaele Vecchione, Maurizio Taglialatela i Vincenzo Barrese. "Kv7 CHANNELS ACTIVATION REDUCES BRAIN ENDOTHELIAL CELLS PERMEABILITY AND PREVENTS KAINIC ACID INDUCED BLOOD BRAIN BARRIER DAMAGE". American Journal of Physiology-Cell Physiology, 29.01.2024. http://dx.doi.org/10.1152/ajpcell.00709.2023.
Pełny tekst źródłaVarghese, Nissi, Bruno Moscoso, Ana Chavez, Kristen Springer, Erika Ortiz, Heun Soh, Sabato Santaniello, Atul Maheshwari i Anastasios V. Tzingounis. "KCNQ2/3 Gain-of-Function Variants and Cell Excitability: Differential Effects in CA1 vs. L2/3 pyramidal neurons". Journal of Neuroscience, 22.08.2023, JN—RM—0980–23. http://dx.doi.org/10.1523/jneurosci.0980-23.2023.
Pełny tekst źródłaLi, Shuang, Bopanna I. Kalappa i Thanos Tzounopoulos. "Noise-induced plasticity of KCNQ2/3 and HCN channels underlies vulnerability and resilience to tinnitus". eLife 4 (27.08.2015). http://dx.doi.org/10.7554/elife.07242.
Pełny tekst źródłaPerez-Vizcaino, Francisco, Angel Cogolludo i Gema Mondejar-Parreño. "Transcriptomic profile of cationic channels in human pulmonary arterial hypertension". Scientific Reports 11, nr 1 (4.08.2021). http://dx.doi.org/10.1038/s41598-021-95196-z.
Pełny tekst źródłaTracy, Gregory C., Angelina R. Wilton, Justin S. Rhodes i Hee Jung Chung. "Heterozygous Deletion of Epilepsy Gene KCNQ2 Has Negligible Effects on Learning and Memory". Frontiers in Behavioral Neuroscience 16 (19.07.2022). http://dx.doi.org/10.3389/fnbeh.2022.930216.
Pełny tekst źródłaLi, De-Pei, Zhao-Fu Sheng, Hua Zhang i Peiru Zheng. "Dysfunction of M channels in the insular cortex is involved in pathogenesis of primary hypertension". Physiology 39, S1 (maj 2024). http://dx.doi.org/10.1152/physiol.2024.39.s1.1993.
Pełny tekst źródłaFrench, Jacqueline A., Roger J. Porter, Emilio Perucca, Martin J. Brodie, Michael A. Rogawski, Simon Pimstone, Ernesto Aycardi i in. "Efficacy and Safety of XEN1101, a Novel Potassium Channel Opener, in Adults With Focal Epilepsy". JAMA Neurology, 9.10.2023. http://dx.doi.org/10.1001/jamaneurol.2023.3542.
Pełny tekst źródłaPant, Shashank, Jiaren Zhang, Eung Chang Kim, Kin Lam, Hee Jung Chung i Emad Tajkhorshid. "PIP2-dependent coupling of voltage sensor and pore domains in Kv7.2 channel". Communications Biology 4, nr 1 (14.10.2021). http://dx.doi.org/10.1038/s42003-021-02729-3.
Pełny tekst źródłaPant, Shashank, Jiaren Zhang, Eung Chang Kim, Kin Lam, Hee Jung Chung i Emad Tajkhorshid. "PIP2-dependent coupling of voltage sensor and pore domains in Kv7.2 channel". Communications Biology 4, nr 1 (14.10.2021). http://dx.doi.org/10.1038/s42003-021-02729-3.
Pełny tekst źródłaKlemz, Alexander, Florian Wildner, Ecem Tütüncü i Zoltan Gerevich. "Regulation of Hippocampal Gamma Oscillations by Modulation of Intrinsic Neuronal Excitability". Frontiers in Neural Circuits 15 (26.01.2022). http://dx.doi.org/10.3389/fncir.2021.778022.
Pełny tekst źródłaYang, Gui-mei, Fu-yun Tian, Yan-wen Shen, Chuan-yan Yang, Hui Yuan, Ping Li i Zhao-bing Gao. "Functional characterization and in vitro pharmacological rescue of KCNQ2 pore mutations associated with epileptic encephalopathy". Acta Pharmacologica Sinica, 17.03.2023. http://dx.doi.org/10.1038/s41401-023-01073-y.
Pełny tekst źródłaJULIANA, CHRISTINE, i DIVA DE LEON. "1745-P: Voltage-Gated K+ Channel Kv7.1 Is an Important Regulator of Insulin Secretion in Normal and Hyperinsulinemic Islets". Diabetes 72, Supplement_1 (20.06.2023). http://dx.doi.org/10.2337/db23-1745-p.
Pełny tekst źródłaTrancuccio, A., A. Mazzanti, D. Kukavica, M. Marino, N. Monteforte, R. Bloise, L. Braghieri i in. "Mutation site-specific risk profile in patients with Type 1 Long QT Syndrome". European Heart Journal 41, Supplement_2 (1.11.2020). http://dx.doi.org/10.1093/ehjci/ehaa946.0743.
Pełny tekst źródłaLiao, Qian-Qian, Qing-Qing Dong, Hui Zhang, Hua-Pan Shu, Yu-Chi Tu i Li-Jun Yao. "Contributions of SGK3 to transporter-related diseases". Frontiers in Cell and Developmental Biology 10 (1.12.2022). http://dx.doi.org/10.3389/fcell.2022.1007924.
Pełny tekst źródłaSvecova, O., R. Kula, L. Chmelikova, J. Hosek, I. Synkova, T. Novotny i M. Bebarova. "Clinical, genetic and functional analysis of R562S-Kv7.1 mutation associated with long QT syndrome type 1". EP Europace 23, Supplement_3 (1.05.2021). http://dx.doi.org/10.1093/europace/euab116.560.
Pełny tekst źródłaBomholtz, S. H., R. Simo-Vicens, L. Abildgaard, N. G. Edvardsson, U. S. Soerensen, M. Grunnet, J. G. Diness i B. H. Bentzen. "P699Mechanisms of action of the small conductance Ca2+-activated K+-channel modulator AP30663, a novel compound being developed for treatment of atrial fibrillation in man". European Heart Journal 40, Supplement_1 (1.10.2019). http://dx.doi.org/10.1093/eurheartj/ehz747.0304.
Pełny tekst źródłaKato, Koichi, Holly M. Isbell, Véronique Fressart, Isabelle Denjoy, Amal Debbiche, Hideki Itoh, Jacques Poinsot i in. "Novel CALM3 Variant Causing Calmodulinopathy With Variable Expressivity in a 4-Generation Family". Circulation: Arrhythmia and Electrophysiology 15, nr 3 (marzec 2022). http://dx.doi.org/10.1161/circep.121.010572.
Pełny tekst źródłaGiammarino, L., S. Nimani, S. Bains, N. Alerni, D. J. Tester, N. Christoforou, J. Louradour i in. "In vivo KCNQ1-suppression-replacement gene therapy in transgenic rabbits with type 1 long QT syndrome". Europace 25, Supplement_1 (24.05.2023). http://dx.doi.org/10.1093/europace/euad122.594.
Pełny tekst źródła