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Artykuły w czasopismach na temat "Kidney disease"
Verma, Dr Amit Kumar. "Periodontal Disease with Diabetes or Diabetes Kidney Disease". International Journal of Trend in Scientific Research and Development Volume-3, Issue-1 (31.12.2018): 1043–51. http://dx.doi.org/10.31142/ijtsrd19176.
Pełny tekst źródłaS, Memon. "Hyponatremia in Chronic Kidney Disease". Open Access Journal of Urology & Nephrology 7, nr 2 (4.04.2022): 1–7. http://dx.doi.org/10.23880/oajun-16000202.
Pełny tekst źródłaParmar, Dr Jigar A., Anant G. Joshi i Dr Manish Chakrabarti. "Dyslipidemia and Chronic Kidney Disease". International Journal of Scientific Research 3, nr 5 (1.06.2012): 396–97. http://dx.doi.org/10.15373/22778179/may2014/123.
Pełny tekst źródłaMal, Pooran, Muhammad Nadeem Ahsan, Mehwish Bukhari i Abdul Manan Junejo. "CHRONIC KIDNEY DISEASE". Professional Medical Journal 25, nr 09 (9.09.2018): 1380–85. http://dx.doi.org/10.29309/tpmj/18.4360.
Pełny tekst źródłaBollenbecker, Seth, Brian Czaya, Orlando M. Gutiérrez i Stefanie Krick. "Lung-kidney interactions and their role in chronic kidney disease-associated pulmonary diseases". American Journal of Physiology-Lung Cellular and Molecular Physiology 322, nr 5 (1.05.2022): L625—L640. http://dx.doi.org/10.1152/ajplung.00152.2021.
Pełny tekst źródłaFatima, Tanveer, Aurangzeb Afzal i Sania Ashraf. "CHRONIC KIDNEY DISEASE". Professional Medical Journal 25, nr 06 (9.06.2018): 887–91. http://dx.doi.org/10.29309/tpmj/18.4418.
Pełny tekst źródłaPaul, Binu M., i Gregory B. Vanden Heuvel. "Kidney: polycystic kidney disease". Wiley Interdisciplinary Reviews: Developmental Biology 3, nr 6 (3.09.2014): 465–87. http://dx.doi.org/10.1002/wdev.152.
Pełny tekst źródłaNishi, Shinichi. "Chronic Kidney Disease and Cardiovascular Disease: Progression of Arterial Diseases in Chronic Kidney Disease". Nihon Naika Gakkai Zasshi 105, nr 5 (2016): 791–92. http://dx.doi.org/10.2169/naika.105.791.
Pełny tekst źródłaNeyra, Javier A., i Lakhmir S. Chawla. "Acute Kidney Disease to Chronic Kidney Disease". Critical Care Clinics 37, nr 2 (kwiecień 2021): 453–74. http://dx.doi.org/10.1016/j.ccc.2020.11.013.
Pełny tekst źródłaIdan, Ahmed Fadhil. "Effect of Coronavirus among Kidney Disease Patients". Journal of Communicable Diseases 54, nr 02 (30.06.2022): 28–32. http://dx.doi.org/10.24321/0019.5138.202267.
Pełny tekst źródłaRozprawy doktorskie na temat "Kidney disease"
Herrera, Añazco Percy, Holguín Edward Mezones i Adrian V. Hernández. "Global kidney disease". Elsevier B.V, 2014. http://hdl.handle.net/10757/322401.
Pełny tekst źródłaRevisión por pares
Wei, Jin. "Acute Kidney Injury and Chronic Kidney Disease". Scholar Commons, 2017. http://scholarcommons.usf.edu/etd/6780.
Pełny tekst źródłaPhilips, L. G. "Disease management in chronic kidney disease /". abstract and full text PDF (free order & download UNR users only), 2005. http://0-wwwlib.umi.com.innopac.library.unr.edu/dissertations/fullcit/1430446.
Pełny tekst źródła"May, 2005." Includes bibliographical references (leaves 92-97). Online version available on the World Wide Web. Library also has microfilm. Ann Arbor, Mich. : ProQuest Information and Learning Company, [2005]. 1 microfilm reel ; 35 mm.
Papadopoulos, Theofilos. "MiRNAs in kidney disease". Thesis, Toulouse 3, 2016. http://www.theses.fr/2016TOU30194/document.
Pełny tekst źródłaMicroRNAs are now recognized as key players in the regulation of proteins and any abnormality in their function is a cause for pathway instability, leading to pathological conditions. Numerous reports from a variety of pathologies provide new data about microRNAs function, their targets and their potential as biomarkers and possible ways to control microRNAs' expression for potential therapeutic purpose. A number of reports also connect microRNAs with pathological conditions in the kidney and point to the use of microRNAs as biomarkers for diagnosis and prognosis of kidney disease in blood, serum, tissue and urine samples. In this thesis, we researched:1) A possible role of the microRNAs in the progression of adult chronic kidney disease (CKD), a disease representing a global burden with the tendency to rise worldwide. Progression of CKD is still very hard to detect non-invasively with the currently used clinical tools (eGFR and albuminuria). In our work we studied alterations of the level of the microRNAs in human urine samples of patients with fast or slow progression of CKD, in order to identify new potential biomarkers for non-invasive progression of CKD. Using Next Generation Sequencing, we analyzed urinary microRNA modifications in urine samples of 70 patients with established CKD and correlated their expression profiles to disease progression. This lead to the identification of 25 urinary microRNAs significantly associated to CKD progression (adjusted pvalue<0.05). Among those, four microRNAs (hsa-miR-34c-5p, hsa-miR-410-3p, hsa-miR-301b-3p, and hsa-miR-145-5p) were selected for validation in an independent cohort of 52 patients with CKD. Increased urinary abundance of hsa-miR-145-5p was confirmed to be associated to progression of CKD. In vitro exploration of the effects of hsa-miR-145-5p inhibition in human kidney cells showed that the microRNA seemed to be involved in necrotic processes. In conclusion we have identified hsa-miR-145-5p as potential urinary microRNA marker of CKD progression. 2) The identification of microRNAs associated to obstructive nephropathy, a frequently encountered disease in children that can lead, in severe cases, to end stage renal disease (ESRD). In this study we used a comprehensive system biology analysis in which we combined micro- and mRNA data from human and animal obstructive nephropathy to obtain information on possible mechanisms involved in this disease. In particular, we have studied in parallel the urinary miRNome of infants with ureteropelvic junction (UPJ) obstruction and the kidney tissue miRNome and transcriptome of the corresponding neonatal partial unilateral ureteral obstruction (UUO) mouse model. Several hundreds of microRNAs and mRNAs displayed changed abundance during disease. Combination of microRNAs in both species and associated mRNAs let to the prioritization of 5 microRNAs and 35 mRNAs associated to disease. In vitro and in vivo validation identified consistent dysregulation of let-7a-5p and miR-29-3p and new potential targets, E3 ubiquitin-protein ligase (DTX4) and neuron navigator 1 (NAV1). Our study is the first to correlate a mouse model of neonatal partial UUO with human UPJ obstruction in a comprehensive systems biology analysis. Our data revealed let-7a and miR-29b as molecules potentially involved in the development of fibrosis in UPJ obstruction via the control of DTX4 in both man and mice that would not be identified otherwise
Ramzan, Naveen, Shimin Zheng, Hemang Panchal, Edward Leinaar, Christian Nwabueze i Timir K. Paul. "Investigating The Association Between Chronic Kidney Disease and Clinical Outcomes". Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/asrf/2019/schedule/21.
Pełny tekst źródłaGale, D. "Genetic investigation of kidney disease". Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/763753/.
Pełny tekst źródłaWong, Germaine. "Cancer and chronic kidney disease". Thesis, The University of Sydney, 2008. https://hdl.handle.net/2123/28229.
Pełny tekst źródłaBrunmark, Charlott. "Type IV collagen and renal disease". Lund : Dept. of Nephrology, University of Lund, 1994. http://books.google.com/books?id=owdrAAAAMAAJ.
Pełny tekst źródłaAntoniv, A. A. "Kidneys functional status in patients with chronic kidney disease and nonalcoholic steatohepatitis". Thesis, БДМУ, 2020. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/18082.
Pełny tekst źródłaPatenaude, Anne-Marie. "Wnt signaling in kidney development and implication in polycystic kidney disease". Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84066.
Pełny tekst źródłaThere is also evidence that non-canonical Writ signaling may be involved in the development of renal cysts but this pathway is uncharacterized. We therefore studied the ontogeny of a downstream marker of this pathway (NFAT) and its localization in the developing kidney. Here we report that NFAT activity is high in early stages of kidney development and is rapidly downregulated at birth. The NFAT signal is diffuse and is expressed in both mesenchymal and epithelial cells of the developing kidney.
Książki na temat "Kidney disease"
L, Watson Michael, i Torres Vicente E, red. Polycystic kidney disease. Oxford: Oxford University Press, 1996.
Znajdź pełny tekst źródłaNational Institutes of Health (U.S.), red. Kidney disease. [Bethesda, Md.?]: National Institutes of Health, 1985.
Znajdź pełny tekst źródła1959-, Goldsmith David, Jayawardene Satish i Ackland Penny, red. ABC of kidney disease. Malden, Mass: Blackwell Pub., 2007.
Znajdź pełny tekst źródłaABC of kidney disease. Wyd. 2. Chichester, West Sussex: John Wiley & Sons, 2013.
Znajdź pełny tekst źródłaYang, Junwei, i Weichun He, red. Chronic Kidney Disease. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-32-9131-7.
Pełny tekst źródłaWada, Takashi, Kengo Furuichi i Naoki Kashihara, red. Diabetic Kidney Disease. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-9301-7.
Pełny tekst źródłaSchulsinger, David A., red. Kidney Stone Disease. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-12105-5.
Pełny tekst źródłaGeary, Denis F., i Franz Schaefer, red. Pediatric Kidney Disease. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-52972-0.
Pełny tekst źródłaCowley,, Benjamin D., i John J. Bissler, red. Polycystic Kidney Disease. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7784-0.
Pełny tekst źródła1930-, Edelmann Chester M., i Meadow S. R, red. Pediatric kidney disease. Wyd. 2. Boston: Little, Brown, 1992.
Znajdź pełny tekst źródłaCzęści książek na temat "Kidney disease"
Bowling, C. Barrett, i Rasheeda K. Hall. "Kidney Disease". W Geriatrics for Specialists, 305–16. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-31831-8_25.
Pełny tekst źródłaCarpenter, William M., i Darren P. Cox. "Kidney Disease". W The ADA Practical Guide to Patients with Medical Conditions, 101–19. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2015. http://dx.doi.org/10.1002/9781119121039.ch5.
Pełny tekst źródłaAxelsson, Thiane G., Michal Chmielewski i Bengt Lindholm. "Kidney Disease". W Present Knowledge in Nutrition, 874–88. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781119946045.ch53.
Pełny tekst źródłaBowling, C. Barrett, i Rasheeda K. Hall. "Kidney Disease". W Geriatrics for Specialists, 301–13. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-76271-1_23.
Pełny tekst źródłaWolfe, Charles J. "Kidney Disease". W Over 55, 133–44. New York: Psychology Press, 2021. http://dx.doi.org/10.4324/9781315792651-8.
Pełny tekst źródłaRosa, Margherita, i Salvatore Di Giulio. "Kidney Disease". W Crohn’s Disease, 249–57. Milano: Springer Milan, 2010. http://dx.doi.org/10.1007/978-88-470-1472-5_23.
Pełny tekst źródłaGould, Edward R., i Anna Marie Burgner. "Glomerular Disease". W The Kidney, 175–97. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3286-3_12.
Pełny tekst źródłaJoshi, Shreyas S., Gladell P. Paner i Sam S. Chang. "Polycystic Kidney Disease". W The Kidney, 19–35. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3286-3_2.
Pełny tekst źródłaSzczech, Lynda. "Disease State: Kidney Disease". W Management of Anemia, 1–10. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7360-6_1.
Pełny tekst źródłaZhang, Rubin, i Anil Paramesh. "Transplantation: Kidney, Kidney–Pancreas Transplant". W Diabetes and Kidney Disease, 175–201. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0793-9_15.
Pełny tekst źródłaStreszczenia konferencji na temat "Kidney disease"
Periyasamy, Kasi, i Venkateswaran Iyer. "Chronic Kidney Disease Helper". W 2020 IEEE International Conference on Healthcare Informatics (ICHI). IEEE, 2020. http://dx.doi.org/10.1109/ichi48887.2020.9374299.
Pełny tekst źródłade Koning, Constance. "EMPA-KIDNEY: empagliflozin slashes kidney disease progression or CV death". W ASN Kidney Week 2022, redaktor Rachel Giles. Baarn, the Netherlands: Medicom Medical Publishers, 2022. http://dx.doi.org/10.55788/ce33490e.
Pełny tekst źródłaYildirim, Pinar. "Chronic Kidney Disease Prediction on Imbalanced Data by Multilayer Perceptron: Chronic Kidney Disease Prediction". W 2017 IEEE 41st Annual Computer Software and Applications Conference (COMPSAC). IEEE, 2017. http://dx.doi.org/10.1109/compsac.2017.84.
Pełny tekst źródłaGao, Yaozong, Yiyi Ma, Guangrui Mu, Miaofei Han, Yiqiang Zhan i Xiang Zhou. "Automatic MR kidney segmentation for autosomal dominant polycystic kidney disease". W Computer-Aided Diagnosis, redaktorzy Horst K. Hahn i Kensaku Mori. SPIE, 2019. http://dx.doi.org/10.1117/12.2512372.
Pełny tekst źródłaFricks, Rafael B., Andrea Bobbio i Kishor S. Trivedi. "Reliability models of chronic kidney disease". W 2016 Annual Reliability and Maintainability Symposium (RAMS). IEEE, 2016. http://dx.doi.org/10.1109/rams.2016.7448058.
Pełny tekst źródłaKumari, Sandhya, i Bhagwate Dhiraj. "Kidney Disease detection through Iris Image". W 2018 3rd IEEE International Conference on Recent Trends in Electronics, Information & Communication Technology (RTEICT). IEEE, 2018. http://dx.doi.org/10.1109/rteict42901.2018.9012423.
Pełny tekst źródłaPushpalatha, S., i A. Stella. "Kidney Disease Diagnosis using Classification Algorithm". W 2021 Fifth International Conference on I-SMAC (IoT in Social, Mobile, Analytics and Cloud) (I-SMAC). IEEE, 2021. http://dx.doi.org/10.1109/i-smac52330.2021.9640879.
Pełny tekst źródłaElsayed, Basant Moustafa, Lina Altarawneh, Suhail Doi i Tawanda Chivese. "Association between pre-existing conditions and hospitalization, intensive care services and mortality from COVID-19 – a cross sectional analysis of an international global health data repository". W Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0151.
Pełny tekst źródłaHuang, Zhongping, Jie Ren i Anilchandra Attaluri. "Experimental Study of a Hybrid Renal Replacement System". W ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14326.
Pełny tekst źródłaSnegha, J., V. Tharani, S. Dhivya Preetha, R. Charanya i S. Bhavani. "Chronic Kidney Disease Prediction Using Data Mining". W 2020 International Conference on Emerging Trends in Information Technology and Engineering (ic-ETITE). IEEE, 2020. http://dx.doi.org/10.1109/ic-etite47903.2020.482.
Pełny tekst źródłaRaporty organizacyjne na temat "Kidney disease"
Zhang, Mingzhu, Wujisiguleng Bao, Luying Sun, Zhi Yao i Xiyao Li. Efficacy and safety of finerenone in chronic kidney disease associated with type 2 diabetes: meta-analysis of randomized clinical trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, marzec 2022. http://dx.doi.org/10.37766/inplasy2022.3.0020.
Pełny tekst źródłaCorey Goldman, Corey Goldman. What's Calcium's role in heart and kidney disease? Experiment, grudzień 2013. http://dx.doi.org/10.18258/1719.
Pełny tekst źródłaGusella, Gabriele L. Role of Integrin-Beta1 in Polycystic Kidney Disease. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2012. http://dx.doi.org/10.21236/ada562319.
Pełny tekst źródłaDominguez, Jesus, K. J. Kelly i Jizhong Zhang. Intravenous Renal Cell Transplantation for Polycystic Kidney Disease. Fort Belvoir, VA: Defense Technical Information Center, październik 2013. http://dx.doi.org/10.21236/ada597871.
Pełny tekst źródłaGusella, Gabriele L. Role of Integrin-Beta 1 in Polycystic Kidney Disease. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2011. http://dx.doi.org/10.21236/ada555405.
Pełny tekst źródłaMurphy-Ullrich, Joanne E. The Endoplasmic Reticulum Stress Protein Calreticulin in Diabetic Chronic Kidney Disease. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2015. http://dx.doi.org/10.21236/ada624022.
Pełny tekst źródłaLin, Chun-Long. Prevalence and prognosis of pulmonary hypertension in patients with chronic kidney disease. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, luty 2023. http://dx.doi.org/10.37766/inplasy2023.2.0051.
Pełny tekst źródłaHua, Zi Bo, i Lv Yuan Chen. Human UCB MSC versus placebo for effect on kidney fibrosis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, październik 2022. http://dx.doi.org/10.37766/inplasy2022.10.0104.
Pełny tekst źródłaHynes, Denise, Jose Arruda, Michael Berbaum, Ifeanyi Chukwudozie, Michael Fischer, Marian Fitzgibbon, Anna Porter i Linda Schiffer. Evaluating a Patient-Centered Medical Home for Patients Receiving Dialysis for Kidney Disease. Patient-Centered Outcomes Research Institute® (PCORI), sierpień 2019. http://dx.doi.org/10.25302/5.2019.ih.12115420.
Pełny tekst źródłaKaatari, S., P. Turaga i G. Wiens. Development of a vaccine for bacterial kidney disease in salmon. [und Renibacterium salmoninarum]. Test accounts, sierpień 1989. http://dx.doi.org/10.2172/5301998.
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