Gotowa bibliografia na temat „Kallikrein-Related peptidase 5”
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Artykuły w czasopismach na temat "Kallikrein-Related peptidase 5"
Ismail, Maha Imam Ahmed, *. Manal Basyouni Ahmed * Manal Basyouni Ahmed i Muneera Al-Sheeha. "Evaluation of kallikrein-related peptidase 5 [KLK5] and Survivin as Prognostic Markers in Breast Cancer". International Journal of Scientific Research 3, nr 5 (1.06.2012): 398–401. http://dx.doi.org/10.15373/22778179/may2014/124.
Pełny tekst źródłaZingkou, Eleni, Georgios Pampalakis i Georgia Sotiropoulou. "Exacerbated dandruff in the absence of kallikrein‐related peptidase 5 protease". Journal of Dermatology 47, nr 3 (7.01.2020): 311–13. http://dx.doi.org/10.1111/1346-8138.15174.
Pełny tekst źródłaSidiropoulos, Konstantinos G., Nicole M. A. White, Anna Bui, Qiang Ding, Peter Boulos, Georgios Pampalakis, Heba Khella, Joseph N. Samuel, Georgia Sotiropoulou i George M. Yousef. "Kallikrein-related peptidase 5 induces miRNA-mediated anti-oncogenic pathways in breast cancer". Oncoscience 1, nr 11 (24.10.2014): 709–24. http://dx.doi.org/10.18632/oncoscience.91.
Pełny tekst źródłaMalachias, Apostolos, Georgia Papachristopoulou, Dimitrios Kypraios, Stefanos P. Bassioukas, Maria Nikaki, Dimitrios Xinopoulos i Maroulio Talieri. "Sa1973 Analysis and Clinical Evaluation of Kallikrein-Related Peptidase 5 (KLK5) in Colon Cancer". Gastroenterology 148, nr 4 (kwiecień 2015): S—372. http://dx.doi.org/10.1016/s0016-5085(15)31246-4.
Pełny tekst źródłaOrtloff, A., F. A. Bustamante, L. Molina, J. Ojeda, C. D. Figueroa i P. Ehrenfeld. "Kallikrein-related Peptidase 5 (KLK5) Expression and Distribution in Canine Cutaneous Squamous Cell Carcinoma". Journal of Comparative Pathology 174 (styczeń 2020): 113–19. http://dx.doi.org/10.1016/j.jcpa.2019.11.009.
Pełny tekst źródłaSakabe, Jun-ichi, Mami Yamamoto, Satoshi Hirakawa, Akira Motoyama, Isao Ohta, Kazuki Tatsuno, Taisuke Ito, Kenji Kabashima, Toshihiko Hibino i Yoshiki Tokura. "Kallikrein-related Peptidase 5 Functions in Proteolytic Processing of Profilaggrin in Cultured Human Keratinocytes". Journal of Biological Chemistry 288, nr 24 (29.04.2013): 17179–89. http://dx.doi.org/10.1074/jbc.m113.476820.
Pełny tekst źródłaLin, Yongbo, Li Ma, Hanliang Dan, Gang Chen, Jian Dai, Liang Xu i Yuqi Liu. "MiR-107-3p Knockdown Alleviates Endothelial Injury in Sepsis via Kallikrein-Related Peptidase 5". Journal of Surgical Research 292 (grudzień 2023): 264–74. http://dx.doi.org/10.1016/j.jss.2023.07.013.
Pełny tekst źródłaYoon, Hyesook, i Isobel A. Scarisbrick. "Kallikrein-related peptidase 6 exacerbates disease in an autoimmune model of multiple sclerosis". Biological Chemistry 397, nr 12 (1.12.2016): 1277–86. http://dx.doi.org/10.1515/hsz-2016-0239.
Pełny tekst źródłaWu, Yanhua, Yingjian Chen, Qing Li, Yanwen Gong, Xiaohong Liu, Liquan Bi i Chengjin Hu. "Upregulation of kallikrein-related peptidase 5 is associated with the malignant behavior of colorectal cancer". Molecular Medicine Reports 14, nr 3 (13.07.2016): 2164–70. http://dx.doi.org/10.3892/mmr.2016.5516.
Pełny tekst źródłaPetraki, Constantina, Youssef M. Youssef, William Dubinski, Zsuzsanna Lichner, Andreas Scorilas, Maria D. Pasic, Vassilios Komborozos i in. "Evaluation and prognostic significance of human tissue kallikrein-related peptidase 10 (KLK10) in colorectal cancer". Tumor Biology 33, nr 4 (22.03.2012): 1209–14. http://dx.doi.org/10.1007/s13277-012-0368-5.
Pełny tekst źródłaRozprawy doktorskie na temat "Kallikrein-Related peptidase 5"
Lenga, Ma Bonda Woodys. "Rôle de la kallicréine 5 dans le remodelage de l’épithélium bronchique associé à la bronchopneumopathie chronique obstructive (BPCO)". Electronic Thesis or Diss., Tours, 2021. http://www.theses.fr/2021TOUR5001.
Pełny tekst źródłaChronic Obstructive Pulmonary Disease (COPD) is a chronic inflammatory lung disease mainly caused by cigarette smoking and characterized by a progressive decrease in the bronchus caliber and a mucus hypersecretion. The alteration of the pulmonary epithelium by cigarette smoke leads to the production of inflammation mediators, oxidative stress and imbalance of proteases/antiproteases well known to play a key role in the genesis and persistence of COPD. Among the protease involved, our study focused on a serine protease, kallikrein 5 (KLK5), which could play a role in tissue remodeling and repair mechanisms associated with the epithelium aggression. Our results showed, on reconstituted epithelium obtained from primary cells isolated from patients following a lung cancer surgical excision and grown in Air-Liquide Interface, that KLK5 cleaved E-cadherin and β-catenin epithelial junction proteins and altered the ultrastructure of these epitheliums. Using the 16HBE14o-bronchial cell line, strongly expressing KLK5, we have demonstrated that the invalidation of the gene for this protease increases the expression of E-cadherin and β-catenin as well as that of integrins thereby improving the integrity of the epithelial barrier and the repair of the cell monolayer. In order to study the mechanisms of action of KLK5, we also showed on BEAS-2B bronchial cells, with expressing low levels of KLK5, that this protease could be induced by pro-inflammatory cytokines (TNF, IL-8 and IL-6) and by hydrogen peroxide (H202), one of the components generated while smoking a cigarette and inducing the oxidative stress. In addition, exogenous KLK5 induces a loss of cellular adhesion to fibronectin by cleaving this matrix protein, as well as E-cadherin and integrins that allow cell to bind to the extracellular matrix. This adhesion loss, limited by the TFPI-2 that inhibits KLK5, is associated with Epithelial-Mesenchymal Transition, to increase cell migration by activating the MAPKinase signaling pathway. All of this data suggests that the activity of KLK5 produced during the inflammatory process associated with COPD would be involved in mechanisms related to the alteration and repair of the integrity of the bronchial epithelium
Wang, Ping [Verfasser], Viktor [Akademischer Betreuer] [Gutachter] Magdolen i Marion B. [Gutachter] Kiechle. "OV-MZ-6 ovarian cancer cells overexpressing kallikrein-related peptidases KLK4, 5, 6 and 7: effects on expression of cancer-related genes / Ping Wang ; Gutachter: Marion B. Kiechle, Viktor Magdolen ; Betreuer: Viktor Magdolen". München : Universitätsbibliothek der TU München, 2016. http://d-nb.info/1117796574/34.
Pełny tekst źródłaGong, Weiwei [Verfasser], Viktor [Akademischer Betreuer] Magdolen, Karl-Friedrich [Gutachter] Becker i Viktor [Gutachter] Magdolen. "Assessment of kallikrein-related peptidases 4, 5, 7 and 12 as prognostic biomarkers in advanced high-grade serous ovarian cancer and triple-negative breast cancer / Weiwei Gong ; Gutachter: Karl-Friedrich Becker, Viktor Magdolen ; Betreuer: Viktor Magdolen". München : Universitätsbibliothek der TU München, 2020. http://d-nb.info/1213898986/34.
Pełny tekst źródłaCzęści książek na temat "Kallikrein-Related peptidase 5"
Goettig, Peter, i Viktor Magdolen. "Kallikrein-Related Peptidase 5". W Handbook of Proteolytic Enzymes, 2772–78. Elsevier, 2013. http://dx.doi.org/10.1016/b978-0-12-382219-2.00611-6.
Pełny tekst źródłaStreszczenia konferencji na temat "Kallikrein-Related peptidase 5"
Koistinen, Hannu, Mykola Domanskyy i Ulf-Håkan Stenman. "Abstract 5304: Inhibition of kallikrein-related peptidase-4 by SPINK1". W Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-5304.
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