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Gotowe bibliografie tematyczne / Ischaemic heart disease / Artykuły w czasopismach

Artykuły w czasopismach na temat „Ischaemic heart disease”

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Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 20 lutego 2023

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1

Shah, Dr Aashka M. "Fibrinogen and Ischaemic Heart Disease". International Journal of Scientific Research 3, nr 6 (1.06.2012): 257–58. http://dx.doi.org/10.15373/22778179/june2014/81.

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2

Saeid, Feyzizadeh, Javadi Aniseh, Badalzadeh Reza i Vafaee S. Manouchehr. "Signaling mediators modulated by cardioprotective interventions in healthy and diabetic myocardium with ischaemia–reperfusion injury". European Journal of Preventive Cardiology 25, nr 14 (14.02.2018): 1463–81. http://dx.doi.org/10.1177/2047487318756420.

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Ischaemic heart diseases are one of the major causes of death in the world. In most patients, ischaemic heart disease is coincident with other risk factors such as diabetes. Patients with diabetes are more prone to cardiac ischaemic dysfunctions including ischaemia–reperfusion injury. Ischaemic preconditioning, postconditioning and remote conditionings are reliable interventions to protect the myocardium against ischaemia–reperfusion injuries through activating various signaling pathways and intracellular mediators. Diabetes can disrupt the intracellular signaling cascades involved in these myocardial protections, and studies have revealed that cardioprotective effects of the conditioning interventions are diminished in the diabetic condition. The complex pathophysiology and poor prognosis of ischaemic heart disease among people with diabetes necessitate the investigation of the interaction of diabetes with ischaemia–reperfusion injury and cardioprotective mechanisms. Reducing the outcomes of ischaemia–reperfusion injury using targeted strategies would be particularly helpful in this population. In this study, we review the protective interventional signaling pathways and mediators which are activated by ischaemic conditioning strategies in healthy and diabetic myocardium with ischaemia–reperfusion injury.

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3

Fox, K., i D. G. Julian. "Ischaemic heart disease". Current Opinion in Cardiology 1, nr 4 (lipiec 1986): 451–52. http://dx.doi.org/10.1097/00001573-198607000-00001.

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4

Julian, D. G., i K. Fox. "Ischaemic heart disease". Current Opinion in Cardiology 1, nr 6 (1986): 825–26. http://dx.doi.org/10.1097/00001573-198611000-00001.

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5

&NA;. "Ischaemic heart disease". Current Opinion in Cardiology 1, nr 6 (1986): 963–76. http://dx.doi.org/10.1097/00001573-198611000-00029.

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6

Julian, D. G. "Ischaemic heart disease". Current Opinion in Cardiology 2, nr 6 (listopad 1987): 947–48. http://dx.doi.org/10.1097/00001573-198711000-00001.

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7

&NA;. "Ischaemic heart disease". Current Opinion in Cardiology 3, nr 4 (lipiec 1988): 561–651. http://dx.doi.org/10.1097/00001573-198803040-00012.

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8

&NA;. "Ischaemic heart disease". Current Opinion in Cardiology 3, nr 4 (lipiec 1988): 561–651. http://dx.doi.org/10.1097/00001573-198807000-00012.

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9

Crouse, JohnR. "ISCHAEMIC HEART DISEASE". Lancet 341, nr 8850 (kwiecień 1993): 931–32. http://dx.doi.org/10.1016/0140-6736(93)91218-b.

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10

Peters, David, P. G. F. Nixon, Wayne Perry i Val Hopwood. "Ischaemic heart disease". Complementary Therapies in Medicine 2, nr 1 (styczeń 1994): 14–20. http://dx.doi.org/10.1016/0965-2299(94)90154-6.

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11

HANSEN, BIRGIT FISCHER. "ISCHAEMIC HEART DISEASE". Acta Pathologica Microbiologica Scandinavica Series A :Pathology 90A, nr 1-6 (19.08.2009): 37–49. http://dx.doi.org/10.1111/j.1699-0463.1982.tb00061_90a.x.

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12

Fryearson, John, i Dawn L. Adamson. "Heart disease in pregnancy: Ischaemic heart disease". Best Practice & Research Clinical Obstetrics & Gynaecology 28, nr 4 (maj 2014): 551–62. http://dx.doi.org/10.1016/j.bpobgyn.2014.03.011.

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13

&NA;. "Ischaemic heart disease I". Current Opinion in Cardiology 1, nr 4 (lipiec 1986): 555–78. http://dx.doi.org/10.1097/00001573-198607000-00015.

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14

Fox, K., i D. G. Julian. "Ischaemic heart disease Overview". Current Opinion in Cardiology 2, nr 4 (lipiec 1987): 569–70. http://dx.doi.org/10.1097/00001573-198707000-00001.

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15

&NA;. "Ischaemic heart disease II". Current Opinion in Cardiology 2, nr 6 (listopad 1987): 1077–110. http://dx.doi.org/10.1097/00001573-198711000-00021.

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16

Fuster, V., i D. G. Julian. "Ischaemic heart disease Overview". Current Opinion in Cardiology 3, nr 4 (lipiec 1988): 459–60. http://dx.doi.org/10.1097/00001573-198803040-00001.

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17

Fuster, V., i D. G. Julian. "Ischaemic heart disease Overview". Current Opinion in Cardiology 3, nr 4 (lipiec 1988): 459–60. http://dx.doi.org/10.1097/00001573-198807000-00001.

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18

Abergel, E., Y. Bernard, E. Brochet, C. Chauvel, A. Cohen, B. Cormier, J. F. Forissier i in. "Chronic ischaemic heart disease". Archives of Cardiovascular Diseases 101, nr 4 (kwiecień 2008): 271–77. http://dx.doi.org/10.1016/s1875-2136(08)73704-5.

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19

Bell, Robert M. "Remote ischaemic conditioning and ischaemic heart disease". British Journal of Hospital Medicine 75, Sup1 (styczeń 2014): C13—C16. http://dx.doi.org/10.12968/hmed.2014.75.sup1.c13.

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20

Iung, B. "VALVE DISEASE: Interface between valve disease and ischaemic heart disease". Heart 84, nr 3 (1.09.2000): 347–52. http://dx.doi.org/10.1136/heart.84.3.347.

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21

Haines, A. "Psychological characteristics and fatal ischaemic heart disease". Heart 85, nr 4 (1.04.2001): 385–89. http://dx.doi.org/10.1136/heart.85.4.385.

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22

Rasmiena, Aliki A., Theodore W. Ng i Peter J. Meikle. "Metabolomics and ischaemic heart disease". Clinical Science 124, nr 5 (12.11.2012): 289–306. http://dx.doi.org/10.1042/cs20120268.

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Ischaemic heart disease accounts for nearly half of the global cardiovascular disease burden. Aetiologies relating to heart disease are complex, but dyslipidaemia, oxidative stress and inflammation are cardinal features. Despite preventative measures and advancements in treatment regimens with lipid-lowering agents, the high prevalence of heart disease and the residual risk of recurrent events continue to be a significant burden to the health sector and to the affected individuals and their families. The development of improved risk models for the early detection and prevention of cardiovascular events in addition to new therapeutic strategies to address this residual risk are required if we are to continue to make inroads into this most prevalent of diseases. Metabolomics and lipidomics are modern disciplines that characterize the metabolite and lipid complement respectively, of a given system. Their application to ischaemic heart disease has demonstrated utilities in population profiling, identification of multivariate biomarkers and in monitoring of therapeutic response, as well as in basic mechanistic studies. Although advances in magnetic resonance and mass spectrometry technologies have given rise to the fields of metabolomics and lipidomics, the plethora of data generated presents challenges requiring specific statistical and bioinformatics applications, together with appropriate study designs. Nonetheless, the predictive and re-classification capacity of individuals with various degrees of risk by the plasma lipidome has recently been demonstrated. In the present review, we summarize evidence derived exclusively by metabolomic and lipidomic studies in the context of ischaemic heart disease. We consider the potential role of plasma lipid profiling in assessing heart disease risk and therapeutic responses, and explore the potential mechanisms. Finally, we highlight where metabolomic studies together with complementary -omic disciplines may make further inroads into the understanding, detection and treatment of ischaemic heart disease.

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23

Silverdal, Jonas, Helen Sjöland, Entela Bollano, Aldina Pivodic, Ulf Dahlström i Michael Fu. "Prognostic impact over time of ischaemic heart disease vs. non‐ischaemic heart disease in heart failure". ESC Heart Failure 7, nr 1 (7.01.2020): 265–74. http://dx.doi.org/10.1002/ehf2.12568.

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24

Sheridan, J. V., P. Thomas i D. J. Sheridan. "Felodipine in Ischaemic Heart Disease". Drugs 34, Supplement 3 (1987): 71–78. http://dx.doi.org/10.2165/00003495-198700343-00016.

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25

Mahmood, Manzoor, Md Harisul Hoque, SA Mahmood, Mohammad Abdul Quayum, Jahanara Arzu i Kazi Sharmin Rahman. "Prevention of Ischaemic Heart Disease". University Heart Journal 10, nr 1 (20.08.2015): 27–30. http://dx.doi.org/10.3329/uhj.v10i1.24594.

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Prevention strategies are based on identification and modulation of cardiovascular risk factors. Targeting high risk individuals is cost-effective. More relevant to the practising clinicians is Identification of those individuals in whom intervention (life style or pharmacological) will prevent cardiovascular events (fatal and non-fatal myocardial infarction) and/or the need for interventional or surgical procedures, extending duration and quality of life (QOL). Modification of reversible risk factors (rather than interventional procedures) has been largely responsible for the decline in age-adjusted cardiovascular mortality.University Heart Journal Vol. 10, No. 1, January 2014; 27-30

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26

Prichard, B. N. C. "Carvedilol in Ischaemic Heart Disease". Cardiology 82, nr 3 (1993): 34–39. http://dx.doi.org/10.1159/000175932.

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27

WOOD, D. A. "AETIOLOGY OF ISCHAEMIC HEART DISEASE". British Journal of Anaesthesia 61, nr 1 (lipiec 1988): 3–10. http://dx.doi.org/10.1093/bja/61.1.3.

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28

Gershlick, A. H. "Platelets and ischaemic heart disease". Current Opinion in Cardiology 1, nr 4 (lipiec 1986): 453–64. http://dx.doi.org/10.1097/00001573-198607000-00002.

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29

Meade, T. W. "Fibrinogen in Ischaemic Heart Disease". European Heart Journal 16, suppl A (2.03.1995): 31–35. http://dx.doi.org/10.1093/eurheartj/16.suppl_a.31.

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30

Sleight, Peter. "Smoking and Ischaemic Heart Disease". European Journal of Cardiovascular Risk 6, nr 5 (październik 1999): 285–86. http://dx.doi.org/10.1177/204748739900600501.

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31

Cauldwell, Matthew, Lucia Baris, Jolien W. Roos-Hesselink i Mark R. Johnson. "Ischaemic heart disease and pregnancy". Heart 105, nr 3 (15.11.2018): 189–95. http://dx.doi.org/10.1136/heartjnl-2018-313454.

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Although ischaemic heart disease is currently rarely encountered in pregnancy, occurring between 2.8 and 6.2 per 100 000 deliveries, it is becoming more common as women delay becoming pregnant until later life, when medical comorbidities are more common, and because of the higher prevalence of obesity in the pregnant population. In addition, chronic inflammatory diseases, which are more common in women, may contribute to greater rates of acute myocardial infarction (AMI). Pregnancy itself seems to be a risk factor for AMI, although the exact mechanisms are not clear. AMI in pregnancy should be investigated in the same manner as in the non-pregnant population, not allowing for delays, with investigations being conducted as they would outside of pregnancy. Maternal morbidity following AMI is high as a result of increased rates of heart failure, arrhythmia and cardiogenic shock. Delivery in women with history of AMI should be typically guided by obstetric indications not cardiac ones.

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32

De Cristofaro, R. "Anticoagulation in ischaemic heart disease". Heart 92, nr 8 (1.08.2006): 1011–12. http://dx.doi.org/10.1136/hrt.2005.085555.

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33

Currie, C. J., P. Conway i C. D. Poole. "Ischaemic heart disease in men". Heart 93, nr 11 (1.11.2007): 1471. http://dx.doi.org/10.1136/hrt.2007.130567.

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34

Mensah, G. A. "Ischaemic heart disease in Africa". Heart 94, nr 7 (1.07.2008): 836–43. http://dx.doi.org/10.1136/hrt.2007.136523.

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35

Pocock, Mamatha O., Lucy Dorrell i Paola Cicconi. "Pathophysiology of ischaemic heart disease". Current Opinion in HIV and AIDS 12, nr 6 (listopad 2017): 548–53. http://dx.doi.org/10.1097/coh.0000000000000411.

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36

Lindop, G. "Non-atherosclerotic Ischaemic Heart Disease". Histopathology 16, nr 3 (marzec 1990): 313. http://dx.doi.org/10.1111/j.1365-2559.1990.tb01125.x.

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37

Burr, Michael L. "Alcohol and Ischaemic Heart Disease". Journal of the Royal Society of Health 114, nr 4 (sierpień 1994): 216–18. http://dx.doi.org/10.1177/146642409411400411.

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38

Meade, T. W. "Thrombosis and ischaemic heart disease." Heart 53, nr 5 (1.05.1985): 473–76. http://dx.doi.org/10.1136/hrt.53.5.473.

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39

Beulens, Joline WJ, i Henk FJ Hendriks. "Alcohol and ischaemic heart disease". Lancet 367, nr 9514 (marzec 2006): 902. http://dx.doi.org/10.1016/s0140-6736(06)68377-3.

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40

Fleck, A. "LATITUDE AND ISCHAEMIC HEART DISEASE". Lancet 333, nr 8638 (marzec 1989): 613. http://dx.doi.org/10.1016/s0140-6736(89)91634-6.

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41

Jokhi, Percy, i Nicholas Curzen. "Ischaemic heart disease: stable angina". Medicine 38, nr 8 (sierpień 2010): 414–20. http://dx.doi.org/10.1016/j.mpmed.2010.05.007.

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42

Whittaker, Andrew, Percy Jokhi i Nicholas Curzen. "Ischaemic heart disease: stable angina". Medicine 42, nr 9 (wrzesień 2014): 495–501. http://dx.doi.org/10.1016/j.mpmed.2014.06.016.

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43

Gabara, Lavinia, Percy Jokhi i Nick Curzen. "Ischaemic heart disease: stable angina". Medicine 46, nr 9 (wrzesień 2018): 520–27. http://dx.doi.org/10.1016/j.mpmed.2018.06.001.

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44

Bondagji, Nabeel S. "Ischaemic heart disease in pregnancy". Journal of the Saudi Heart Association 24, nr 2 (kwiecień 2012): 89–97. http://dx.doi.org/10.1016/j.jsha.2011.12.002.

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45

Segall, JeffreyJ. "LATITUDE AND ISCHAEMIC HEART DISEASE". Lancet 333, nr 8647 (maj 1989): 1146. http://dx.doi.org/10.1016/s0140-6736(89)92430-6.

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46

Sinclair, Hugh. "LATITUDE AND ISCHAEMIC HEART DISEASE". Lancet 333, nr 8643 (kwiecień 1989): 895. http://dx.doi.org/10.1016/s0140-6736(89)92880-8.

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47

Shahar, Eyal. "Antioxidants and ischaemic heart disease". Lancet 350, nr 9078 (sierpień 1997): 667. http://dx.doi.org/10.1016/s0140-6736(05)63362-4.

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48

Leo, Maria A., i Charles S. Lieber. "Antioxidants and ischaemic heart disease". Lancet 350, nr 9078 (sierpień 1997): 667. http://dx.doi.org/10.1016/s0140-6736(05)63363-6.

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49

Violi, Francesco, i Luigi Iullano. "Antioxidants and ischaemic heart disease". Lancet 350, nr 9078 (sierpień 1997): 667–68. http://dx.doi.org/10.1016/s0140-6736(05)63364-8.

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50

Rapola, Janne M., Jarmo Virtamo i Jussi K. Huttunen. "Antioxidants and ischaemic heart disease". Lancet 350, nr 9078 (sierpień 1997): 668. http://dx.doi.org/10.1016/s0140-6736(05)63365-x.

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