Gotowe bibliografie tematyczne / Interleukin 10

Spis treści

  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „Interleukin 10”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 11 marca 2023

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Artykuły w czasopismach na temat "Interleukin 10"

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&NA;. "Interleukin-10". Drugs in R & D 1, nr 3 (styczeń 1999): 262–64. http://dx.doi.org/10.2165/00126839-199901030-00017.

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Asadullah, K., R. Sabat, M. Friedrich, W. Docke, H. Volk i W. Sterry. "Interleukin-10". Anti-Inflammatory & Anti-Allergy Agents in Medicinal Chemistry 5, nr 3 (1.08.2006): 223–31. http://dx.doi.org/10.2174/187152306778017700.

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Parsons, Polly E. "Interleukin-10". Critical Care Medicine 26, nr 5 (maj 1998): 818–19. http://dx.doi.org/10.1097/00003246-199805000-00007.

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Goldman, Michel, Thierry Velu i Marina Pretolani. "Interleukin-10". BioDrugs 7, nr 1 (styczeń 1997): 6–14. http://dx.doi.org/10.2165/00063030-199707010-00002.

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Moore, K. W., A. O'Garra, R. W. Malefyt, P. Vieira i T. R. Mosmann. "Interleukin-10". Annual Review of Immunology 11, nr 1 (kwiecień 1993): 165–90. http://dx.doi.org/10.1146/annurev.iy.11.040193.001121.

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Umetsu, Dale T., i Rosemarie H. DeKruyff. "Interleukin-10". American Journal of Respiratory Cell and Molecular Biology 21, nr 5 (listopad 1999): 562–63. http://dx.doi.org/10.1165/ajrcmb.21.5.f171.

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Zlotnik, Albert, i Kevin W. Moore. "Interleukin 10". Cytokine 3, nr 5 (wrzesień 1991): 366–71. http://dx.doi.org/10.1016/1043-4666(91)90039-g.

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Klava, Andrew. "Interleukin-10". Archives of Surgery 132, nr 4 (1.04.1997): 425. http://dx.doi.org/10.1001/archsurg.1997.01430280099016.

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DEWAALMALEFYT, R., M. RONCAROLO, H. SPITS i J. DEVRIES. "Interleukin-10". Current Opinion in Immunology 4, nr 3 (czerwiec 1992): 314–20. http://dx.doi.org/10.1016/0952-7915(92)90082-p.

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Reineke, Ulrich, Robert Sabat, Hans-Dieter Volk i Jens Schneider-Mergener. "Mapping of the interleukin-10/interleukin-10 receptor combining site". Protein Science 7, nr 4 (kwiecień 1998): 951–60. http://dx.doi.org/10.1002/pro.5560070412.

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Rozprawy doktorskie na temat "Interleukin 10"

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Rees, Louisa Elizabeth Natasha. "The regulation of interleukin-10". Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322627.

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Schmid-Lossberg, Juliette. "Nachweis von Interleukin-1α, Interleukin-1ß, Interleukin-5 und Interleukin-10 im vaginalen Abstrich von gesunden Frauen". Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-114787.

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Powell, Mark Jason. "Gene regulation of murine interleukin -10". Thesis, University of Oxford, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299106.

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Crawley, Esther Madeleine. "Interleukin 10 in juvenile idiopathic arthritis". Thesis, University College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.249187.

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Forrester, Megan Amy. "Identification of interleukin-10 producing cells specific for Epstein-Barr virus latent membrane protein 1 and the involvement of interleukin-27 in their induction". Thesis, University of Aberdeen, 2011. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=167956.

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During latent Epstein-Barr virus (EBV) infection, the T helper cell response to the EBV latent membrane protein (LMP)1 is dominated by the production of IL-10, but by IFN- γ during acute EBV infection. The purpose of this thesis was to develop methods for the enumeration and characterisation of the IL-10 producing CD4+ T cells that respond to peptides of the EBV protein LMP1, and to investigate the possible involvement of IL-27 in the development of these cells. It was found that some human donors have very high concentrations of IL-27 within serum, which is not dependent on EBV infection status but demonstrates relatively low heritability. The addition of IL-27 to cultures of human T cells did not induce IL-10 but the production of IL-17 was inhibited. To identify and characterise LMP1 responsive cells I used CD154 as a marker of activated T cells. Having optimised the methodology, 14 donors of known EBV serostatus were tested for activated IL-10 producing cells after culturing peripheral blood mononuclear cells with LMP1 peptides. However in most cases the frequency of CD4+ lymphocytes upregulating CD154 and IL-10 in response to LMP1 peptides was below the assay’s sensitivity. When the CD154+IL-10+ CD4+ cells were stained for T helper cell subset markers they were positive for every marker and isotype control, suggesting that the cells were non-specifically binding labelled antibody. Both single positive CD154+ and single positive IL-10+CD4+ cells were also present in response to LMP1, but again typically at frequencies below the level of sensitivity for this assay. The conclusions of the work are that IL-27 responses are heterogeneous, but unlikely to play an important role in the induction of IL-10+ T cells in EBV infection. The frequency of LMP1 responsive T cells is very low (<0.08%) in most donors.
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Dohmen, Klaus Malte. "Die kritische Rolle von Interleukin-10 bei der Induktion nasaler Toleranz in der experimentellen Autoimmun-Myokarditis (EAM)". [S.l. : s.n.], 2007. http://nbn-resolving.de/urn:nbn:de:bsz:289-vts-58242.

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Walter, Claudia. "Bestimmung der Zytokine Interleukin-1ra, Interleukin-6, Interleukin-10 und Interleukin-12 im Vaginalsekret bei Frauen mit Bakterieller Vaginose". Diss., lmu, 2004. http://nbn-resolving.de/urn:nbn:de:bvb:19-28461.

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Capper, Emma Rachel. "The human interleukin-10 and interleukin-10 receptor system : molecular & genetic analysis of SLE and recurrent miscarriage". Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.424695.

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Clarke, Christopher Jeremy Paul. "Molecular mechanisms of the anti-inflammatory cytokines interleukin-4 and interleukin-10". Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285172.

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Rempel, Julia D. "Interleukin-12 and interleukin-10 regulation of antibody responses upon protein antigen immunization". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ41623.pdf.

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Książki na temat "Interleukin 10"

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de Vries, Jan E., i René de Waal Malefyt. Interleukin-10. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2.

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M, Marincola Francesco, red. Interleukin-10. Georgetown, Tex: Landes Bioscience, 2005.

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Fillatreau, Simon, i Anne O'Garra, red. Interleukin-10 in Health and Disease. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-43492-5.

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Stuart, Amanda Denise. Structure-function analysis of the interleukin-10 homologue encoded by Epstein-Barr virus. Manchester: University of Manchester, 1995.

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Klein, Lazar Victor. Discordant effects of interleukin-10 upon organ injury in a model of ruptured abdominal aortic aneurysm. Ottawa: National Library of Canada, 2002.

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Marincola, Francesco M. Interleukin-10. Taylor & Francis Group, 2006.

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Interleukin-10. Austin: R.G. Landes, 1995.

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Vries, Jan E. de, i Rene de Waal Malefyt. Interleukin-10. Springer, 2013.

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Interleukin-10. Georgetown, TX: Landes Bioscience/Eurekah.com, 2005.

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Jan E. de Vries (Editor) i Rene de Waal Malefyt (Editor), red. Interleukin-10. Springer, 1996.

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Części książek na temat "Interleukin 10"

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Moore, Kevin W., Alice S. Y. Ho i Jiangchun Xu-Amano. "Molecular Biology of Interleukin-10 and its Receptor". W Interleukin-10, 1–9. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_1.

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Carvalho, Edgar M. "IL-10 In Human Leishmaniasis". W Interleukin-10, 91–100. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_10.

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Schuitemaker, Hanneke, Neeltje A. Kootstra i Frank Miedema. "IL-10 and HIV-1 Replication". W Interleukin-10, 101–12. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_11.

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Roncarolo, Maria-Grazia. "Interleukin-10 and Transplantation Tolerance". W Interleukin-10, 113–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_12.

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Zlotnik, Albert, i Rachel A. Freiberg. "Effects of IL-10 in Lipopolysaccharide- and Superantigen-Induced Lethal Shock In Vivo". W Interleukin-10, 121–26. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_13.

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Powrie, Fiona, i Robert L. Coffman. "Inhibition of Cell-Mediated Immune Responses In Vivo by Recombinant IL-10". W Interleukin-10, 127–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_14.

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Lee, Myung-Shik, i Nora Sarvetnick. "IL-10 Transgenic Mice". W Interleukin-10, 135–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_15.

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Rennick, Donna, Dan Berg, Ralf Kühn i Werner Müller. "Interleukin-10 Deficient Mice". W Interleukin-10, 141–48. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_16.

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Chernoff, Amy E., Eric V. Granowitz, Leland Shapiro, Edouard Vannier, Gerhard Lonnemann, Jonathan B. Angel, Scott F. Orencole i in. "A Phase I Study of Interleukin-10 in Healthy Humans: Safety and Effects on Cytokine Production". W Interleukin-10, 149–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_17.

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Trotta, Paul P., i William T. Windsor. "Physicochemical and Structural Properties of Interleukin-10". W Interleukin-10, 11–18. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_2.

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Streszczenia konferencji na temat "Interleukin 10"

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Giannakopoulou, Charoula Eleni, Adamantia Sotiriou, Maria Dettoraki, Michael Yang, Fotis Perlikos, Dimitrios Toumpanakis, Georgios Prezerakos i in. "Interleukin-10 affects the control of breathing." W ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2530.

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Giannakopoulou, Charoula Eleni, Maria Dettoraki, Adamantia Sotiriou, Charis Roussos i Theodoros Vassilakopoulos. "Interleukin 10 Affects The Control Of Breathing". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4195.

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"Interleukin-10 levels in relation to COVID-19 vaccine". W 4th International Conference on Biological & Health Sciences (CIC-BIOHS’2022). Cihan University, 2022. http://dx.doi.org/10.24086/biohs2022/paper.714.

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The study was conducted in Cihan University-Erbil campus in November 2021. All subjects were asked questions regarding their history, using a questionnaire sheet. Blood was investigated for the Interleukin-10 level, Lactate Dehydrogenase (LDH) and hematological parameters. The mean serum level of IL-10 was significantly higher in Covid-19, non-vaccinated than Covid-19, vaccinated individuals. Moreover, the level was also equally significant and higher in non-Covid-19, vaccinated than non-Covid-19, non-vaccinated. Non-significant differences were found between Covid-19, vaccinated and non-Covid-19, non-vaccinated individuals. And between Covid-19, non-vaccinated and non-Covid-19, vaccinated individuals. The mean serum levels of LDH were found to be significantly lower in non-Covid-19, non-vaccinated, indicating a significant rise of LDH in all other groups. Despite the rise in all three groups a significant difference was not found between them. The total WBC count shows a significant increase in both non-vaccinated groups. While the mean level of lymphocytes counts reveals non-significant decrease in non-Covid-19, vaccinated group in comparison to the other groups. However, the mean monocyte counts of Covid-19, vaccinated group revealed a significant rise in comparison to other groups. In addition, the mean counts of granulocyte showed a significant increase in both non-vaccinated groups in comparison with vaccinated groups. Platelet mean count presented a non-significant difference when comparing all groups. Our current study may indicate the potency of the vaccines.
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Terai, Mizue, Masumi Eto, Garbo D. Young, Michael J. Mastrangelo, Yutaka Tamura, Kenichi Harigaya i Takami Sato. "Abstract 1913: Proinflammatory cytokine, Interleukin-6 (IL-6), promotes Interleukin-10 (IL-10) production from melanoma cell via JAK/STAT3 and Raf/ERK signal pathways". W Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1913.

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Masso-Welch, Patricia, Patricia M. Merhige, Omkara Lakshmi M. Veeranki i Shiu-Ming Kuo. "Abstract 3274: Loss of interleukin 10 (IL-10) decreases mouse mammary gland development and lactogenic differentiation". W Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3274.

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Puspodewi, Dini, Agung Dwi Wahyu W i Jusak Nugraha. "The Effect of Macrophage Addition to Interleukin 10 (IL-10) in Tuberculosis Granuloma Model In Vitro". W 2nd International Conference Postgraduate School. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0007543503860391.

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Darmaputra, I. Gusti Nyoman, Luh Mas Rusyati, Wibi Riawan, Anang Endaryanto i Cita Rosita Sigit Prakoeswa. "The Correlation between Neutrophil CD64, Interleukin-17 (Il-17), Interleukin-10 (Il-10) in Skin Tissue and Neutrophil CD64, Il-17 and Il-10 in Blood Circulation in Erythema Nodosum Leprosum (ENL) Patients". W The 23rd Regional Conference of Dermatology 2018. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0008155802860290.

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Floreth, Timothy, Randi Stern, Bharathi Laxman i Steven R. White. "Interleukin-10 Inhibits HLA-G Expression In Differentiated Human Airway Epithelial Cells". W American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2897.

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Karstens, KF, J. Kempski, A. Giannou, E. Freiwald, M. Reeh, M. Tachezy, J. Izbicki i in. "Die Rolle von systemischem Interleukin 10 und 17A bei Adenokarzinomen des Ösophagus". W DGVS Digital: BEST OF DGVS. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1716255.

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Elia, Zaid, i Saeed Hussain. "ASSESSMENT OF INERFERON GAMMA (IFN-γ )& INTERLEUKIN-10 (IL-10)IN PATIENTS AFFLECTED WITH CELIAC DISEASE". W 4th International Scientific Conference of Cihan University-Erbil on Biological Sciences. Cihan University-Erbil, 2017. http://dx.doi.org/10.24086/bios17.06.

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Raporty organizacyjne na temat "Interleukin 10"

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Grigorov, Boncho G., Anastasiya G. Trenova, Georgi S. Slavov, Lyuba D. Miteva i Spaska A. Stanilova. Interleukin‑10 (IL‑10) Promoter Polymorphism at Position –1082 in Bulgarian Patients with Multiple Sclerosis. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, luty 2019. http://dx.doi.org/10.7546/crabs.2019.01.11.

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Jafrin, Sarah, Md Abdul Aziz i Mohammad Safiqul Islam. Elevated levels of pleiotropic interleukin-6 (IL-6) and interleukin-10 (IL-10) are critically involved with the severity and mortality of COVID-19: An updated longitudinal meta-analysis and systematic review on 147 studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, kwiecień 2022. http://dx.doi.org/10.37766/inplasy2022.4.0046.

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Review question / Objective: How were serum IL-6 and IL-10 linked with the severity and mortality of COVID-19 patients? To evaluate the role of IL-6 and IL-10 in the development of the severity or morality of COVID-19 patients. The outcomes (mean difference) were calculated between the severe vs. non-severe COVID-19 patients and non-survival vs. survival patients to evaluate the risk of severity or mortality. Condition being studied: Severity and mortality among the COVID-19 patients. Information sources: The international scientific authorized databases such as Google Scholar, PubMed, Embase, CNKI, Cochrane Library, and Web of science were used as primary sources to identify and collect the eligible literature. Additional secondary databases were also comprehensively searched to extract more related studies.
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Li, Lijun, Memg Ling Cao, Dong Hua Li, Wei Xiong i Jian Gang Cao. Association of Interleukin-10 Polymorphism (rs1800896, rs1800871, and rs1800872) with Breast Cancer risk: an Updated Meta-Analysis based on different ethnic groups. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, marzec 2021. http://dx.doi.org/10.37766/inplasy2021.3.0061.

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Cao, Xianling, Xuanyou Zhou, Naixin Xu, Songchang Chang i Chenming Xu. Association of IL-4 and IL-10 Polymorphisms with Preterm Birth Susceptibility: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, kwiecień 2022. http://dx.doi.org/10.37766/inplasy2022.4.0044.

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Review question / Objective: The aim of our systematic review and meta-analysis was to summarize the effects of IL-4 and IL-10 gene polymorphism and clarify their possible association with PTB. Condition being studied: World Health Organization (WHO) defines preterm birth (PTB) as babies born alive before 37 weeks of pregnancy are completed. The new estimates show that the prevalence of PTB during 2014 ranged from 8.7% to13.4% of all live births, about 15 million preterm babies born each year. Besides, PTB is the leading cause of death worldwide for children below 5 years of age. Babies born preterm are at an increased risk of short-term and long-term complications attributed to immaturity of multiple organ systems, such as cerebral palsy, intellectual disabilities, vision and hearing impairments, and impaired cognitive development. PTB has become a worldwide public health problem, but its etiology remains unclear. Accumulating evidence shows that PTB is a syndrome that can be attributed to a variety of pathological processes(5). Inflammatory diseases and genetic background are known risk factors for PTB, many studies had shown that genetic variations in proinflammatory cytokines such as tumor necrosis factor-α (TNF-α) and interleukin-1 α (IL-1 α) are associated with increased risk of PTB, but the relationship between genetic polymorphism in anti-inflammatory cytokines and risk of PTB remains controversial.
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