Gotowa bibliografia na temat „Immunogenetics”
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Artykuły w czasopismach na temat "Immunogenetics"
Grennan, D. M. "Immunogenetics". Annals of the Rheumatic Diseases 44, nr 6 (1.06.1985): 429. http://dx.doi.org/10.1136/ard.44.6.429-c.
Pełny tekst źródłaSteinmuller, David. "Immunogenetics". Mayo Clinic Proceedings 60, nr 4 (kwiecień 1985): 281. http://dx.doi.org/10.1016/s0025-6196(12)60324-3.
Pełny tekst źródłaVelosa, Jorge A. "Immunogenetics". Mayo Clinic Proceedings 60, nr 10 (październik 1985): 721. http://dx.doi.org/10.1016/s0025-6196(12)60757-5.
Pełny tekst źródłaWakeland, Edward K. "Immunogenetics". Current Opinion in Immunology 14, nr 5 (październik 2002): 607–8. http://dx.doi.org/10.1016/s0952-7915(02)00391-6.
Pełny tekst źródłaConley, Mary Ellen. "Immunogenetics". Current Opinion in Immunology 15, nr 5 (październik 2003): 567–70. http://dx.doi.org/10.1016/s0952-7915(03)00106-7.
Pełny tekst źródłaCallard, R. "Immunogenetics". Journal of Immunological Methods 78, nr 1 (kwiecień 1985): 168–69. http://dx.doi.org/10.1016/0022-1759(85)90346-1.
Pełny tekst źródłaAlper, Chester A., i Charles E. Larsen. "Immunogenetics". Current Opinion in Immunology 16, nr 5 (październik 2004): 623–25. http://dx.doi.org/10.1016/j.coi.2004.08.003.
Pełny tekst źródłaWakeland, Edward K. "Immunogenetics". Current Opinion in Immunology 18, nr 5 (październik 2006): 605–7. http://dx.doi.org/10.1016/j.coi.2006.07.018.
Pełny tekst źródłaBender, K. "Immunogenetics". Experientia 42, nr 10 (październik 1986): 1138–47. http://dx.doi.org/10.1007/bf01941288.
Pełny tekst źródłaNorman, Paul J. "Immunogenetics special issue 2023: Immunogenetics of infectious disease". Immunogenetics 75, nr 3 (23.05.2023): 197–99. http://dx.doi.org/10.1007/s00251-023-01301-z.
Pełny tekst źródłaRozprawy doktorskie na temat "Immunogenetics"
Middleton, D. "Histocompatibility and immunogenetics". Thesis, Queen's University Belfast, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.368778.
Pełny tekst źródłaFanning, Gregory Charles. "Immunogenetics of systemic sclerosis". Thesis, University of Oxford, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.284535.
Pełny tekst źródłaThomson, W. "Immunogenetics of rheumatoid arthritis". Thesis, University of Manchester, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.383908.
Pełny tekst źródłaSaini, Surinder Singh. "Molecular immunogenetics of bovine antibody". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape8/PQDD_0005/NQ40388.pdf.
Pełny tekst źródłaAnderson, Amy Elizabeth. "The immunogenetics of Helicobacter infection". Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.414822.
Pełny tekst źródłaGhosh, Soumitra. "Immunogenetics of Type I diabetes". Thesis, University of Oxford, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.306514.
Pełny tekst źródłaElse, Kathryn J. "Immunogenetics of Trichuris muris infection". Thesis, University of Nottingham, 1989. http://eprints.nottingham.ac.uk/12875/.
Pełny tekst źródłaTozatto, Maio Karina. "Immunogenetics in sickle cell disease". Thesis, Sorbonne Paris Cité, 2019. http://www.theses.fr/2019USPCC093.
Pełny tekst źródłaSickle cell disease (SCD) is the most common inherited hemoglobinopathy, caused by a single nucleotide polymorphism (SNP) in the beta-globin (HBB) gene. This SNP determines the synthesis of S haemoglobin (HbS), which polymerizes under stress conditions, sickling the red blood cell (RBC). Sickle RBC are less deformable, more adherent to the endothelium, and more susceptible to haemolysis. SCD complications are explained by the interaction between haemolysis, vaso-occlusion and inflammatory activation, determined by the RBC sickling. Patients with SCD may present several complications, affecting all organs. Clinical presentation is very heterogeneous, ranging from patients who have mild symptoms to patients who die from disease complications. Because inflammation plays a major role in SCD, polymorphisms in inflammatory genes are potential targets to explain this heterogeneity. Haematopoietic stem cell transplantation (HSCT) is the only curative therapy currently available for SCD, with good results shown after human leukocyte antigen (HLA) identical sibling HSCT. However, most patients will not have a matched sibling donor. Patients with SCD are mostly from African origin, the less represented ethnic group in stem cell donor registries. To date, few studies using local registries were performed to find the probability of having a potential unrelated donor in SCD settings. This study aimed to assess the role of inflammatory genes encoding Toll-like receptors (TLR) in the occurrence of bacterial infections in patients with SCD, because infection is a leading cause of mortality in SCD, and TLR recognize a wide range of bacteria. Patients included had DNA samples and clinical data available. SNPs were genotyped by real-time polymerase chain reaction (RT-PCR). Four hundred thirty patients, mostly from Brazilian and Sub-Saharan African origin, were divided in two groups: infected (n=235, patients who presented at least one episode of bacterial infection), and non-infected (n=195, patients who never presented bacterial infections). The T/A genotype of SNP rs4696480 in TLR2 was less frequent in infected patients (50% versus 67%, OR=0.50, 95% CI 0.34-0.75, p<0.001). In addition, the T/T genotype of this SNP was more frequent among infected patients (15% versus 5%, OR=0.50, 95% CI 0.34-0.75, p<0.001). Previous reports in other settings showed that A/A carriers had higher secretion of inflammatory markers, while T allele was associated with less occurrence and severity of inflammatory diseases. Hence, T/A genotype might express the ideal inflammatory response to defeat bacteria, while the weaker inflammatory response determined by the T/T genotype increases susceptibility to bacterial infections in SCD settings
A doença falciforme (DF) é a hemoglobinopatia hereditária mais frequente, causada por um polimorfismo de nucleotídeo único (SNP) no gene da betaglobina (HBB). A ocorrência desse SNP determina a síntese de hemoglobina S, que polimeriza sob condições de stress, alterando a conformação das hemácias, que adquirem forma de drepanócitos. Os drepanócitos são menos deformáveis, mais aderentes ao endotélio e mais suscetíveis à hemolise. As complicações clínicas da DF podem ser explicadas pela interação entre a vasoclusão, hemólise e ativação inflamatória resultantes da presença dos drepanócitos na circulação. Os pacientes com DF podem apresentar numerosas complicações, que afetam todos os órgãos. A apresentação clínica da DF é muito heterogênea, variando de pacientes pouco sintomáticos a pacientes que falecem por complicações da doença. Visto que a inflamação tem um papel importante na fisiopatologia da DF, polimorfismos em genes inflamatórios poderiam explicar essa heterogeneidade.O transplante de células tronco hematopoiéticas (TCPH) é a única terapia curativa disponível atualmente para a DF, com bons resultados demonstrados em TCPH de doador aparentado antígeno leucocitário humano (HLA) idêntico. Não obstante, a maioria dos pacientes não dispõe de doador aparentado HLA idêntico. A DF ocorre em pacientes normalmente de origem africana, o grupo étnico menos representado em registro de doadores de células tronco. Nos dias de hoje, poucos estudos, utilizando registros locais, avaliaram a probabilidade de encontrar potenciais doadores não aparentados para pacientes com DF. Este estudo teve por objetivo avaliar o papel de genes inflamaórios que codificam receptores Toll-like (TLR) na ocorrência de infecções bacterianas em pacientes com DF, visto que infecção é uma das principais causas de mortalidade em DF, e os TLR reconhecem diversos tipos de bactérias. Os pacientes incluídos no estudo tinham amostras de DNA e dados clínicos disponiveis. Os SNPs foram genotipados por reação em cadeia de polimerase em tempo real (RT-PCR). Quatrocentos e trinta pacientes, a maioria de orgem brasileira ou africana subsaariana, foram divididos em dois grupos, infectados (n=235, pacientes que apresentaram ao menos um episodio de infecção bacteriana), e não infectados (n=195, pacientes que nunca tiveram tais infecções). O genótipo T/A do SNP rs4696480 foi menos frequente em pacientes infectados (50% versus 67%, OR=0.50, 95% CI 0.34-0.75, p<0.001). Além disso, o genótipo T/T do mesmo SNP foi mais frequente em pacientes infectados (15% versus 5%, OR=0.50, 95% CI 0.34-0.75, p<0.001). Estudos prévios mostraram que indivíduos com genótipo A/A apresentavam mais secreção de marcadores inflamatórios, enquanto o alelo T foi associado a menor ocorrência e menor gravidade de doenças inflamatórias
Jeffery, Katherine Joanna Mary. "The immunogenetics of HTLV-I infection". Thesis, Imperial College London, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392152.
Pełny tekst źródłaGoodman, Reyna Suzanne. "Immunogenetics of haematopoietic stem cell transplantation". Thesis, Anglia Ruskin University, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.478885.
Pełny tekst źródłaKsiążki na temat "Immunogenetics"
Christiansen, Frank T., i Brian D. Tait, red. Immunogenetics. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-842-9.
Pełny tekst źródłaO, McDevitt Hugh, red. Immunogenetics. New York: Springer International, 1992.
Znajdź pełny tekst źródłaWilliamson, Alan R. Essential immunogenetics. Oxford: Blackwell Scientific, 1987.
Znajdź pełny tekst źródłaWilliamson, Alan R. Essential immunogenetics. Oxford: Blackwell Scientific Publications, 1987.
Znajdź pełny tekst źródłaLesage, Sylvie. Immunogenetics: Tolerance and autoimmunity. Hauppauge, N.Y: Nova Science Publishers, 2010.
Znajdź pełny tekst źródłaVillanueva, Christian J. Immunogenicity. Hauppauge, N.Y: Nova Science, 2011.
Znajdź pełny tekst źródłaBernal, J. E. Human immunogenetics: Principles and clinical applications. London: Taylor & Francis, 1986.
Znajdź pełny tekst źródłaMadrigal, Alejandro J., Margita Bencová, Derek Middleton, Dominique Charron i Tibor Nánási, red. Immunogenetics: Advances and Education. Dordrecht: Springer Netherlands, 1997. http://dx.doi.org/10.1007/978-94-011-5486-4.
Pełny tekst źródłaCarvalho, Agostinho, red. Immunogenetics of Fungal Diseases. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-50842-9.
Pełny tekst źródłaR, Farid Nadir, red. Immunogenetics of endocrine disorders. New York: Liss, 1988.
Znajdź pełny tekst źródłaCzęści książek na temat "Immunogenetics"
Welsh, K. I. "Immunogenetics". W Immunotoxicity of Metals and Immunotoxicology, 37–41. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-8443-4_4.
Pełny tekst źródłaLefranc, Marie-Paule. "Immunogenetics". W Encyclopedia of Systems Biology, 998. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-9863-7_259.
Pełny tekst źródłaFleischhauer, Katharina, Peter A. Horn i Andrea Harmer. "Immunogenetics Laboratory". W Establishing a Hematopoietic Stem Cell Transplantation Unit, 111–28. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-59358-6_8.
Pełny tekst źródłaFuggle, Susan V., i Craig J. Taylor. "Histocompatibility and Immunogenetics". W Handbook of Renal and Pancreatic Transplantation, 55–75. Chichester, UK: John Wiley & Sons, Ltd, 2012. http://dx.doi.org/10.1002/9781118305294.ch4.
Pełny tekst źródłaBarnett, A. H. "Immunogenetics of Diabetes". W Immunology of Endocrine Diseases, 103–21. Dordrecht: Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4171-7_6.
Pełny tekst źródłaHirbod-Mobarakeh, Armin, Mahsima Shabani, Mahsa Keshavarz-Fathi, Farnaz Delavari, Ali Akbar Amirzargar, Behrouz Nikbin, Anton Kutikhin i Nima Rezaei. "Immunogenetics of Cancer". W Cancer Immunology, 417–78. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-30845-2_20.
Pełny tekst źródłaAmirzargar, Ali Akbar. "Immunogenetics of Aging". W Immunology of Aging, 219–29. Berlin, Heidelberg: Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-39495-9_16.
Pełny tekst źródłaHirbod-Mobarakeh, Armin, Ali Akbar Amirzargar, Behrouz Nikbin, Mohammad Hossein Nicknam, Anton Kutikhin i Nima Rezaei. "Immunogenetics of Cancer". W Cancer Immunology, 295–341. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-44006-3_17.
Pełny tekst źródłaBach, Fritz H. "Immunogenetics of HLA". W Chronic Renal Disease, 529–35. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4826-9_54.
Pełny tekst źródłaBurns, A., P. Li i A. Rees. "Immunogenetics of Nephritis". W Immunology of Renal Disease, 1–28. Dordrecht: Springer Netherlands, 1991. http://dx.doi.org/10.1007/978-94-011-3902-1_1.
Pełny tekst źródłaStreszczenia konferencji na temat "Immunogenetics"
Dehais i Mougenot. "An interactive system for database in immunogenetics". W Proceedings of the Twenty-Seventh Annual Hawaii International Conference on System Sciences. IEEE Comput. Soc. Press, 1994. http://dx.doi.org/10.1109/hicss.1994.323594.
Pełny tekst źródłaWilliams, RMichael, i Edmond J. Yunis. "Abstract 2343: Immunogenetics of cancer and aging". W Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-2343.
Pełny tekst źródłaWilliams, RMichael, i Edmond J. Yunis. "Abstract 2343: Immunogenetics of cancer and aging". W Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-2343.
Pełny tekst źródłaRefae, Sadal, Nathalie Ebran, Jocelyn Gal, Josiane Otto, Damien Giacchero, Delphine Borchiellini, Joel Guigay, Frederique Peyrade, Gerard Milano i Esma Saada. "Abstract 4548: Host immunogenetics and hyperprogression under PD1/PD-L1 checkpoint inhibitors". W Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-4548.
Pełny tekst źródłaREFAE, Sadal, Jocelyn GAL, Nathalie EBRAN, Josiane OTTO, Delphine BORCHIELLINI, Frederic Peyrade, Emmanuel CHAMOREY, Patrick Brest, Gerard Alain Milano i Esma SAADA-BOUZID. "Abstract 1370: Germinal immunogenetics predicts treatment outcome for PD1 PD-L1 checkpoint inhibitors". W Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1370.
Pełny tekst źródłaREFAE, Sadal, Jocelyn GAL, Nathalie EBRAN, Josiane OTTO, Delphine BORCHIELLINI, Frederic Peyrade, Emmanuel CHAMOREY, Patrick Brest, Gerard Alain Milano i Esma SAADA-BOUZID. "Abstract 1370: Germinal immunogenetics predicts treatment outcome for PD1 PD-L1 checkpoint inhibitors". W Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1370.
Pełny tekst źródłaLevina, Julia, Leyla Namazova-Baranova, Kirill Savostyanov, Alexander Pushkov, Alexey Burdennyy, Anna Alekseeva, Kamilla Efendieva i Elena Vishneva. "GP5 The immunogenetics and risk factors of pollinosis among russian children. case-control study". W Faculty of Paediatrics of the Royal College of Physicians of Ireland, 9th Europaediatrics Congress, 13–15 June, Dublin, Ireland 2019. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2019. http://dx.doi.org/10.1136/archdischild-2019-epa.72.
Pełny tekst źródłaGu, Rong, i Hongyun Zhang. "The Application of Improved Immunogenetic Algorithm in Signal Timing". W 2009 International Joint Conference on Bioinformatics, Systems Biology and Intelligent Computing. IEEE, 2009. http://dx.doi.org/10.1109/ijcbs.2009.35.
Pełny tekst źródłaGridina, S. L., V. F. Gridin i O. I. Leshonok. "Characterization of High-Producing Cows by their Immunogenetic Status". W International scientific and practical conference "AgroSMART - Smart solutions for agriculture" (AgroSMART 2018). Paris, France: Atlantis Press, 2018. http://dx.doi.org/10.2991/agrosmart-18.2018.49.
Pełny tekst źródłaRamonda, R., L. Punzi, A. Businaro, E. Musacchio, F. Schiavon, M. Podswiadek, G. Cardinale i S. Todesco. "OP0050 Immunogenetic aspects of erosive osteoarthritis of the hand". W Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.417.
Pełny tekst źródłaRaporty organizacyjne na temat "Immunogenetics"
Hutchinson, Mark, Janet Coller, Jillian Clark, Ruth Marshall, James Middleton, Vicky Staikopoulos, Melanie Gentgall, Francesca Alvaro i Kathy Heyman. Chronic Pain Following Spinal Cord Injury: The Role of Immunogenetics and Time of Injury Pain Treatment. Fort Belvoir, VA: Defense Technical Information Center, październik 2014. http://dx.doi.org/10.21236/ada613751.
Pełny tekst źródłaHutchinson, Mark, Janet Coller, Jillian Clark, Ruth Marshall, James Middleton, Vicky Staikopoulos, Francesca Alvaro i Kathy Heyman. Chronic Pain Following Spinal Cord Injury: The Role of Immunogenetics and Time of Injury Pain Treatment. Fort Belvoir, VA: Defense Technical Information Center, październik 2012. http://dx.doi.org/10.21236/ada569291.
Pełny tekst źródłaRodriguez, Jose E., Abebe T. Hassen i James M. Reecy. Immunogenetic Factors Affecting Infectious Bovine Keratoconjuntivitis (IBK). Ames (Iowa): Iowa State University, styczeń 2006. http://dx.doi.org/10.31274/ans_air-180814-476.
Pełny tekst źródłaDavid, Lior, Yaniv Palti, Moshe Kotler, Gideon Hulata i Eric M. Hallerman. Genetic Basis of Cyprinid Herpes Virus-3 Resistance in Common Carp. United States Department of Agriculture, styczeń 2011. http://dx.doi.org/10.32747/2011.7592645.bard.
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