Gotowe bibliografie tematyczne / Immune response

Gotowa bibliografia na temat „Immune response”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 29 lipca 2024

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Artykuły w czasopismach na temat "Immune response"

A, Kavitha, Priavadhana Rajan Prasaad, Bheema Rao G i Hemalatha Ganapathy. "Immune Response in Tuberculosis - CD4/CD68 Epitope Mapping". Annals of Pathology and Laboratory Medicine 5, nr 9 (14.09.2018): A759–763. http://dx.doi.org/10.21276/apalm.1960.

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Pleskanovskaya, S. A. "ADАPTIVE TRANSFER OF THE IMMUNE RESPONSE – NEW APPROACHES". European Journal of Natural History, nr 6 2020 (2020): 11–16. http://dx.doi.org/10.17513/ejnh.34134.

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GC, Velasquez Serra. "Monitoring of Immune Response Following Covid-19 Vaccination". Virology & Immunology Journal 7, nr 1 (4.01.2023): 1–9. http://dx.doi.org/10.23880/vij-16000311.

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Introduction: SARS-CoV-2 is an infectious, viral, and contagious disease in which most patients present a mild respiratory condition whose recovery is autonomous and guided by the immune system itself. Objectives: The objectives of this research were to evaluate the immune system, the vaccination doses administered to the inhabitants of the parish of El Recreo, located in the Duran Canton of the Province of Guayas (Ecuador), according to age groups and gender, the vaccination coverage and the incidence of new diseases in the population under study. Material and Methods: This was a descriptive, non-experimental, prospective, cross-sectional, field study. It was carried out during the period from November 2021 to March 2022, during the pandemic of the "Omicron" variant. Results: the predominant age group corresponded to the range of 50-59 years (26.83% and 42; 29.79%) for both genders. We found (90.10%) of the population vaccinated. Another group (3.31%) did not receive the biologic and (6.59%) indicated that they were reluctant to receive the product. The second dose was administered to (83.5%) people in the community, while (14.28%) indicated having received the third dose. (3.04%) became ill after receiving the first doses; (2.63%) the second doses and none of those who received the third doses. It is necessary to alert the population of the need to benefit from the application of the vaccine since it seems to confer a certain degree of protection to the inhabitants and thus, the viral spread, hospitalization and death.

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Batchelor, J. R. "Immune response". Nature 337, nr 6204 (styczeń 1989): 220. http://dx.doi.org/10.1038/337220a0.

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Malinova, Dessi. "Immune response". Opticon1826 7, nr 12 (30.04.2012): 22. http://dx.doi.org/10.5334/opt.121214.

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Pearson, Steven D. "Immune Response". JAMA: The Journal of the American Medical Association 256, nr 22 (12.12.1986): 3088. http://dx.doi.org/10.1001/jama.1986.03380220054009.

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Jamieson, Amanda Mercedes, Meredith Crane, Yun Xu i Kayla Lee. "Immune triage: prioritization of host immune responses." Journal of Immunology 196, nr 1_Supplement (1.05.2016): 197.20. http://dx.doi.org/10.4049/jimmunol.196.supp.197.20.

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Abstract The immune response is important in many functions, including host defense against pathogens, wound healing, development, response to cancer, and maintenance of homeostatic physiological responses. We are interested in the concept of immune triage, in that a given host must be able to deal effectively with multiple insults, and at times prioritize immune responses. It is important for the overall health status of the host that the immune system responds effectively to protect essential organs. We have developed several mouse models, focusing on the lung immune response, that allow us to examine different aspects of immune triage. The lung is an essential and delicate organ and thus pulmonary immune responses must be tightly regulated. We have determined that lung infection with influenza A virus (IAV) alters the response to bacterial lung infections. Depending on the bacterial infection, previous infection with IAV can suppress or augment the immune response to bacteria. We have also determined that pulmonary infection with IAV alters many aspects of the systemic immune response. There is a global suppression to systemic bacterial infection, and a decrease in the wound healing response. Our data indicate that the immune system prioritizes lung infections over many other responses. This is most likely due to the importance of the lung in host survival. We have established several regulatory mechanisms by which this immune triage occurs. By understanding how the immune system responds to multiple insults we can improve our understanding of the immune network on a global level.

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Kannan, Rajni, Kathleen Madden i Stephanie Andrews. "Primer on Immuno-Oncology and Immune Response". Clinical Journal of Oncology Nursing 18, nr 3 (27.05.2014): 311–17. http://dx.doi.org/10.1188/14.cjon.311-317.

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AK, Vyas. "The Muddle of Immune Response in Coronavirus Disease-19". Open Access Journal of Microbiology & Biotechnology 5, nr 3 (2020): 1–3. http://dx.doi.org/10.23880/oajmb-16000171.

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Coronavirus Disease-19 (COVID19) caused by the enormously spreadable SARS- CoV-2 virus has appeared as a global pandemic and leads to high mortalities. Approximately 31,243,339 people have been infected so far with this disease which has led to the death of more than 965,103patients as of 21st Sept 2020. There are many drug molecules under-trials are in phase I and II. The numerous possible candidates for vaccine development against this infectious virus are also in the pipeline. Although, so far no molecule as a therapeutics or vaccine for prevention has been approved. Recent reports have observed that severely ill patients have a differential immunological profile compared to mild COVID-19 infection. Current studies globally observed that the cytokine storm maybe leads to the severity of COVID19 infection. In this article, our focus is to describe the present knowledge and status of differential immune profile among patients infected with COVID19 infection and their association with disease progression mild to severe.

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Joob, Beuy, i Viroj Wiwanitkit. "Neurocysticercosis, immune status and immune response". Arquivos de Neuro-Psiquiatria 70, nr 9 (wrzesień 2012): 750. http://dx.doi.org/10.1590/s0004-282x2012000900023.

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Rozprawy doktorskie na temat "Immune response"

Nissinen, A. (Antti). "Humoral immune response to phosphatidylethanol". Doctoral thesis, Oulun yliopisto, 2011. http://urn.fi/urn:isbn:9789514295232.

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Abstract Heavy alcohol consumption places a substantial burden on health all over the world. Metabolites of alcohol evoke alterations that lead to tissue damage in many organs. Phosphatidylethanol (PEth) is a unique phospholipid formed in the cellular membranes during the metabolism of ethanol after alcohol consumption. PEth has attracted special attention as it is postulated to be a reliable marker of long term heavy alcohol consumption. The aims of present study were to investigate the immunogenicity of phosphatidylethanol in mice and to analyze the plasma antibodies binding to phosphatidylethanol in humans. In this study a clear immune response was generated in mice immunized with PEth in human low density lipoprotein (LDL) carrier. Mouse monoclonal IgM antibodies binding specifically to phosphoethyl head group of PEth were generated using hybridoma technology. Since PEth was shown to be immunogenic in mice, plasma was analyzed for the presence of antibodies also in humans. PEth-specific antibodies of IgG, IgA and IgM isotypes in plasma were detected in heavy drinkers of alcohol with or without pancreatitis as well as in the controls. The plasma levels of the antibodies binding to PEth were significantly lower in the study subjects with heavy alcohol use and in this present study sample the low IgA levels to PEth were better indicators of heavy alcohol consumption as compared to the some of the traditional markers of heavy alcohol use. The antibody levels to PEth associated significantly to plasma antibodies binding to malondialdehyde-acetaldehyde adducts that are known to be formed during alcohol metabolism but not to antibodies binding to phosphocholine which is generated by lipid oxidation in humans. In conclusion, this study demonstrates that phosphatidylethanol is immunogenic in mice when using carriers such as human LDL in the immunization process. The binding of the monoclonal antibodies specifically to the PEth head group suggests that it would be feasible to develop a diagnostic immunoassay to PEth. The presence of antibodies binding to PEth in plasma indicates that PEth may be a target of humoral immunity in humans
Tiivistelmä Runsas alkoholinkulutus aiheuttaa maailmanlaajuisesti merkittäviä terveydellisiä haittoja. Alkoholin aineenvaihduntatuotteet muuttavat kudoksien rakenteita ja aiheuttavat kudosvaurioita. Fosfatidyylietanoli on alkoholin aineenvaihdunnan tuloksena solukalvoilla syntyvä fosfolipidi, jota on tutkittu kahdenkymmenen vuoden ajan lupaavana alkoholin suurkulutuksen merkkiaineena. Tutkimuksen tavoitteena oli selvittää fosfatidyylietanolin immunisoinnin aiheuttamaa vasta-aineiden muodostumista koe-eläinmallina käytetyissä hiirissä sekä määrittää ihmisten plasmanäytteistä vasta-aineita, jotka sitoutuvat fosfatidyylietanoliin. Tutkimuksessa havaittiin immuunivasteen muodostuminen hiirissä, jotka immunisoitiin ihmisen LDL hiukkasiin liitetyllä fosfatidyylietanolilla. Hiiren monoklonaalisia fosfatidyylietanoliin sitoutuvia IgM-luokan vasta-aineita tuotettiin tutkimuksessa soluviljelyn avulla. Fosfatidyylietanolin aiheuttama vasta-aineiden muodostuminen hiirillä johdatti mittaamaan fosfatidyylietanoliin sitoutuvia vasta-aineita myös ihmisiltä. Tutkimuksessa havaittiin fosfatidyylietanoliin sitoutuvia IgG-, IgA- ja IgM-luokan vasta-aineita alkoholin suurkuluttajilla, alkoholihaimatulehdusta sairastavilla ja verrokkihenkilöillä. Vasta-aineiden pitoisuudet olivat alkoholia runsaasti käyttävillä koehenkilöillä merkitsevästi pienemmät kuin verrokkiryhmällä. Matalat IgA-vasta-ainepitoisuudet osoittautuivat aineistossa paremmaksi alkoholin suurkulutuksen osoittajiksi kuin eräät tavanomaisesti käytetyt alkoholinkäytön merkkiaineet. Plasman fosfatidyylietanoli-vasta-aineiden ja alkoholin aineenvaihdunnan seurauksena syntyvien malondialdehydi-asetaldehydi-addukteihin sitoutuvien vasta-aineiden määrän välillä havaittiin merkitsevä yhteys, jota ei havaittu rasvojen hapettumisen seurauksena syntyvien fosfokoliini-vasta-aineiden ja fosfatidyylietanoli-vasta-aineiden välillä. Tutkimus osoittaa, että hiirillä voidaan aikaansaada vasta-ainevälitteinen immuunivaste, kun ne rokotetaan ihmisen LDL-hiukkaseen liitetyllä fosfatidyylietanolilla. Fosfatidyylietanoliin spesifisesti sitoutuvien monoklonaalisten vasta-aineiden tuottaminen voi tulevaisuudessa johtaa immunologisen diagnostisen määritysmenetelmän kehittämiseen. Fosfatidyylietanoliin sitoutuvien plasman vasta-aineiden havaitseminen viittaa siihen, että fosfatidyylietanoli on vasta-ainevälitteisen immuunivasteen kohde myös ihmisillä

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De, Oliveira O. L. P. "Immune response to Thy-1". Thesis, Open University, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.484410.

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Baldwin, Lisa Michelle. "The immune response to biomaterials". Thesis, University of Liverpool, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.420298.

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Brown, Anna. "The immune response to salmomellae". Thesis, University of Cambridge, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.292915.

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Flynn, Karen Mildred Ryan 1969. "GP96 and the immune response". Thesis, Massachusetts Institute of Technology, 2001. http://hdl.handle.net/1721.1/8586.

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Yang, Jun. "Immune response to orthopaedic biomaterials". Case Western Reserve University School of Graduate Studies / OhioLINK, 1995. http://rave.ohiolink.edu/etdc/view?acc_num=case1058195837.

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Solinas, Cinzia. "Immune response in breast cancer". Doctoral thesis, Universite Libre de Bruxelles, 2019. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/282008.

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The immunogenicity of breast cancer (BC) is quite heterogeneous among the clinical subtypes, with immune responses identified most frequently in triple negative (TNBC) and HER2-positive tumors. The extent, spatial localization, distribution patterns, organization and phenotype of the BC immune infiltrate are currently being widely investigated but require standardization before they can be used clinically. One highly relevant unmet clinical need is to understand how immune features are linked to prognosis and potential benefit from treatments, particularly immunotherapy. The present work investigated tumor-infiltrating lymphocytes (TIL), tertiary lymphoid structures (TLS), the expression of multiple targetable inhibitory immune checkpoint molecules (PD-1, PD-L1 and PD-L2, CTLA-4, LAG3 and TIM3) and their clinical relevance in primary BC. Different technical approaches were employed including: flow cytometry (FC) on fresh tissue homogenates; immunohistochemistry (IHC) and immunofluorescence (IF) on formalin-fixed paraffin embedded (FFPE) tissue blocks from untreated primary tumors; and gene expression on a large dataset of BC patients with available long-term survival data. Flow cytometric analysis of PD-1 expression and its principal ligands PD-L1 and PD-L2 together with CTLA-4, LAG3 and TIM3 on TIL in fresh untreated primary tumors revealed that PD-1 and CTLA-4 are most highly expressed on BC TIL and PD-L1 is the principal PD-1 ligand in BC. Immune checkpoint molecule expression parallels the extent of TIL infiltration and TLS presence and number, with the patterns detected similar to that observed in secondary lymphoid organs. Significantly improved disease-specific survival (DSS) has been associated with PD-1hi HER2-enriched and PD-L1hi, PD-L2hi and CTLA-4hi basal-like BC; however there is significant heterogeneity between individual tumors even within the same subtype. These observations suggest that determining expression levels of multiple targetable inhibitory immune checkpoint molecules in patients might help to successfully target them in BC patients most likely to respond.We examined the concordance between two experienced immuno-pathologists who read 800 IHC-stained slides from five independent series over a period of four years to determine the reproducibility of assessing multiple immune biomarkers. This included scoring TIL, TLS, PD-1 and PD-L1 together with detailed information on the spatial localization and cell types expressing these molecules in the tumor microenvironment (TME). The interobserver reproducibility for the assessment of TIL and TLS was consistently good to excellent overtime, while the concordance for PD-L1 evaluation ranged from fair to excellent when it was only expressed on tumor cells (TC); and the concordance for PD-1 evaluation was fair to excellent when it was expressed in TLS and evaluated in primary tumors. Neither PD-L1 expression by TC, nor PD-1 expression within a TLS was significantly associated with prognosis in our datasets.The extent of TIL, TLS and PD-1 and PD-L1 expression were studied in a cohort of TNBC patients who underwent genetic counseling for their personal/familial history of BC or ovarian cancer (OC). This study revealed a remarkable similarity in patterns of immune infiltration between the two cohorts. Interestingly, a higher prevalence of TIL intermediate cases (≥10% and <50% TIL) was detected in the BRCA-mutated cohort, suggesting that this group may be more immunogenic.We next investigated whether the extent and presence of these immune parameters were associated with prognosis in the most highly infiltrated, aggressive BC subtypes (TNBC and HER2-positive). We determined the ideal cut-off for each subtype (TNBC and HER2-positive) to use TIL as a categorical variable. This study found a consistent prognostic impact from TIL (in any tumor compartment including stromal, intratumoral and global areas) and a novel association between PD-L1 expression within TLS and better survival in TNBC. This last effect was driven by baseline stromal TIL, strengthening the importance of reliably quantifying the levels of TIL in BC. Overall, our analyses show that among the targetable inhibitory immune checkpoint molecules investigated in BC, PD-1 and CTLA-4 are most highly expressed by BC TIL and are associated with higher infiltration of TIL; PD-L1 is the principal ligand for PD-1; TIL and TLS are reproducibly scored on IHC-stained tissues; and TIL levels are associated with a better prognosis in TNBC independent of their location in the TME at optimal cut-offs. Our data also provide new insight on targetable inhibitory immune checkpoint molecule expression and location as well as showing a prognostic role for TIL assessed by IHC in primary BC, which identifies these biomarkers as ideal candidates for further investigation.
Doctorat en Sciences médicales (Médecine)
info:eu-repo/semantics/nonPublished

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Díaz, de Ståhl Teresita. "Fcγ Receptors in the Immune Response". Doctoral thesis, Uppsala University, Department of Genetics and Pathology, 2001. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1545.

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Circulating immune complexes play an important role in the modulation of antibody responses and in the pathogenesis of immune diseases. This thesis deals with the in vivo regulatory properties of antibodies and their specific Fc receptors.

The immunosuppressive function of IgG is used clinically, to prevent rhesus-negative women from becoming sensitized to rhesus-positive erythrocytes from the fetus. The mechanism behind this regulation is poorly understood but involvement of a receptor for IgG, FcγRII, has been suggested. It is shown in this thesis that IgG and also IgE induce immunosuppression against sheep erythrocytes to a similar extent both in mice lacking all the known Fc receptors as in wild-type animals. These findings imply that antibody-mediated suppression of humoral responses against particulate antigens is Fc-independent and that the major operating mechanism is masking of epitopes.

Immunization with soluble antigens in complex with specific IgG leads to an augmentation of antibody production. The cellular mechanism behind this control is examined here and it is found that the capture of IgG2a immune complexes by a bone marrow-derived cell expressing FcγRI (and FcγRIII) is essential. An analysis of the ability of IgG3 to mediate this regulation indicated that, in contrast, this subclass of IgG augments antibody responses independently of FcγRI (and FcγRIII). These findings suggest that distinct mechanisms mediate the enhancing effect of different subclasses of antibodies.

Finally, the contribution of FcγRIII was studied in the development of collagen-induced arthritis (CIA), an animal model for rheumatoid arthritis in humans. It was discovered that while DBA/1 wild-type control mice frequently developed severe CIA, with high incidence, FcγRIII-deficient mice were almost completely protected, indicating a crucial role for FcγRIII in CIA.

The results presented here help to understand how immune complexes regulate immune responses in vivo and show that Fc receptors for IgG, if involved, could be new targets for the treatment of immune complex-related disorders.

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Ågren, Karin. "Immune response in human tonsil tissue /". Stockholm, 1997. http://diss.kib.ki.se/1997/91-628-2714-6.

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O'Rourke, Sara Marie. "The cytotoxic immune response to HBV". Thesis, Imperial College London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297231.

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Książki na temat "Immune response"

Helfrich, Richard. Immune response. Palm Springs, CA: Health Spectrum, 1997.

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R, Lamb Jonathan, red. Immune recognition. Oxford: IRL Press, 1988.

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Vrana, Nihal Engin. Biomaterials and Immune Response. Boca Raton : Taylor & Francis, 2018. | Series: Devices, circuits, and systems: CRC Press, 2018. http://dx.doi.org/10.1201/b22419.

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Saunders, Mary E., Ph.D. i Mak Tak W. 1945-, red. Primer to the immune response. Amsterdam: Academic Press, 2008.

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Potts, Eve. Understanding your immune system. New York: Avon Books, 1986.

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Kaufmann, Stefan H. E., Barry T. Rouse i David L. Sacks, red. The Immune Response to Infection. Washington, DC, USA: ASM Press, 2010. http://dx.doi.org/10.1128/9781555816872.

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D, Gregory Christopher, red. Apoptosis and the immune response. New York: Wiley-Liss, 1995.

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New York Academy of Sciences, red. Glycobiology of the immune response. Boston, Mass: Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2012.

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1937-, Cohen Stanley, red. Lymphokines and the immune response. Boca Raton, Fla: CRC Press, 1990.

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W, Ades Edwin, Rest Richard F i Morse Stephen A, red. Microbial pathogenesis and immune response. New York, N.Y: New York Academy of Sciences, 1994.

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Części książek na temat "Immune response"

Merritt, K. "Immune response". W Handbook of Biomaterial Properties, 513–28. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-5801-9_30.

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Tiwari, Jawahar L., i Paul I. Terasaki. "Immune Response". W HLA and Disease Associations, 431–44. New York, NY: Springer New York, 1985. http://dx.doi.org/10.1007/978-1-4613-8545-5_23.

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Mårdh, Pers-Anders, Jorma Paavonen i Mirja Puolakkainen. "Immune Response". W Chlamydia, 47–55. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0719-8_4.

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Neumann, Detlef, i Klaus Resch. "Immune Response". W Encyclopedia of Immunotoxicology, 442–46. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-54596-2_749.

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Neumann, Detlef, i Klaus Resch. "Immune Response". W Encyclopedia of Immunotoxicology, 1–5. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27786-3_749-2.

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Parija, Subhash Chandra. "Immune Response". W Textbook of Microbiology and Immunology, 211–26. Singapore: Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-3315-8_16.

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Sedlacek, H. Harald, i Tarik Möröy. "Generation of the IgA response". W Immune Reactions, 151–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79558-9_9.

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Harr, Jeffrey N., Philip F. Stahel, Phillip D. Levy, Antoine Vieillard-Baron, Yang Xue, Muhammad N. Iqbal, Jeffrey Chan i in. "Humoral Immune Response". W Encyclopedia of Intensive Care Medicine, 1157. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_3149.

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Citerio, G., C. Giussani, Hugo Sax, Didier Pittet, Xiaoyan Wen, John A. Kellum, Angela M. Mills i in. "Innate Immune Response". W Encyclopedia of Intensive Care Medicine, 1251. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_3172.

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Landreth, Kenneth S., i Sarah V. M. Dodson. "Neonatal Immune Response". W Encyclopedia of Immunotoxicology, 657–59. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-54596-2_1064.

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Streszczenia konferencji na temat "Immune response"

Shen, Yi, Fuqiang Liang, Yanhui Niu, Huiyun Lin, Ying Gu, Brian C. Wilson i Buhong Li. "Monitoring vascular targeted PDT response with multimode optical imaging". W Biophotonics and Immune Responses XIV, redaktor Wei R. Chen. SPIE, 2019. http://dx.doi.org/10.1117/12.2513578.

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Hamblin, Michael R. "Strategies to potentiate immune response after photodynamic therapy (Conference Presentation)". W Biophotonics and Immune Responses XII, redaktor Wei R. Chen. SPIE, 2017. http://dx.doi.org/10.1117/12.2255860.

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Zhang, Zhihong. "Intravital molecular imaging of fluorescent model antigen-elicited specific immune response against tumor". W Biophotonics and Immune Responses XVIII, redaktor Wei R. Chen. SPIE, 2023. http://dx.doi.org/10.1117/12.2651344.

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Abdoli, Neman, Patrik Gilley, Ke Zhang, Xuxin Chen, Theresa C. Thai, Kathleen Moore, Robert S. Mannel i Yuchen Qiu. "Comparing the effectiveness of 2D and 3D features on predicting the response to chemotherapy for ovarian cancer patients". W Biophotonics and Immune Responses XVIII, redaktor Wei R. Chen. SPIE, 2023. http://dx.doi.org/10.1117/12.2655153.

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Sourvanos, Dennis, Timothy Zhu, Andreea Dimofte, Theresa Busch, Weibing Yang, Justin Burrell, Rodrigo Neiva i in. "Optimizing biophotonics and immune response research: a proposal for in vivo dose escalation and light dosimetry analysis in porcine models". W Biophotonics and Immune Responses XIX, redaktorzy Wei R. Chen i Feifan Zhou. SPIE, 2024. http://dx.doi.org/10.1117/12.3005183.

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Verhoeven, Victoria I. "Immune response regulation inDermacentor variabilis". W 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.108512.

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Burns, John, i Heather J. Ruskin. "In-Silico Visualization of Immune Response". W 2007 1st International Conference on Bioinformatics and Biomedical Engineering. IEEE, 2007. http://dx.doi.org/10.1109/icbbe.2007.312.

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Rogge, Lars. "SP0054 DECONVOLUTION OF THE IMMUNE RESPONSE". W Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.8574.

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Lenhardt, Maurício Machado, Dauana Schwartz i Bruna K. de F. Silva. "Can depression be associated with the immune response". W XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.443.

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Introduction: Depression is a disease of uncertain installation and etiology, the imbalance of neurotransmitters is involved in this process, and stress can be an activator of pro-inflammatory cytokines and trigger depressive symptoms. The organism undergoes modulations due to biochemical changes and these are linked to molecular and biochemical components and by the survival instinct, the human body is stimulated to release substances as a form of protection. The objective of this study is to describe the possible association between a loss of homeostasis of the central nervous system (CNS), changes in the modulation of the immune system, and the development of depressive symptoms. Methods: This is an integrative literature review, available in the virtual health databases: PubMed, MEDLINE, SciELO, and Google Scholar published between the years 2010 to 2020. Results: Studies indicate that cytokines can interfere with the homeostasis of the CNS and that the imbalance of catecholamines and indoleamine is involved in the process of depression. In this sense, studies have focused on neuromodulation by blocking neurotransmitters and neuroreceptors to regulate the immune system. Conclusion: It’s already established that the imbalance in the release and reuptake of neurotransmitters is associated with the onset of the depression, however, current studies show that there may also be an association with the homeostasis of the immune system. Therapeutic protocols aren’t based on the correlation between the immune system and the onset of the disease, so further studies are needed to strengthen this relationship.

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Schneider, David S. "Defining the shape of an immune response". W 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.94138.

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Raporty organizacyjne na temat "Immune response"

Zelikoff, Judith T. Fish Immune Response as Biomarkers. Fort Belvoir, VA: Defense Technical Information Center, marzec 2001. http://dx.doi.org/10.21236/ada399403.

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Herrmann, Melissa S., Michael G. Kaiser i Susan J. Lamont. Rooster’s Genetic Response to Immune Stimulation. Ames (Iowa): Iowa State University, styczeń 2015. http://dx.doi.org/10.31274/ans_air-180814-1314.

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Ennis, Francis A. Human Immune Response to Dengue Infections. Fort Belvoir, VA: Defense Technical Information Center, lipiec 1990. http://dx.doi.org/10.21236/ada234922.

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Ennis, Francis A. Human Immune Response to Dengue Infections. Fort Belvoir, VA: Defense Technical Information Center, czerwiec 1991. http://dx.doi.org/10.21236/ada240717.

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Benson, J. M., D. E. Bice i K. J. Nikula. Beryllium-induced immune response in C3H mice. Office of Scientific and Technical Information (OSTI), grudzień 1995. http://dx.doi.org/10.2172/381393.

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Rubinstein, Francis, Girish Ghatikar, Jessica Granderson, Paul Haugen, Carlos Romero i David Watson. Barrier Immune Radio Communications for Demand Response. Office of Scientific and Technical Information (OSTI), luty 2009. http://dx.doi.org/10.2172/971266.

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Bunnell, B. N., i W. B. Iturrian. Social Behavior, Prolactin and the Immune Response. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 1989. http://dx.doi.org/10.21236/ada224055.

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Redmond, Sarah Beth, Rachel Tell, Derrick Coble, Carrie Mueller, Dušan Palić, Claire B. Andreasen i Susan J. Lamont. Genetic Differences in Chicken Splenic Immune Gene Expression in Response to Dietary Immune Modulation. Ames (Iowa): Iowa State University, styczeń 2010. http://dx.doi.org/10.31274/ans_air-180814-166.

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Nelson, Brad H. Deciphering the Adaptive Immune Response to Ovarian Cancer. Fort Belvoir, VA: Defense Technical Information Center, październik 2014. http://dx.doi.org/10.21236/ada613174.

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Herrmann, Melissa S., Rodrigo A. Gallardo, David A. Bunn, David A. Bunn, Terra R. Kelly i Jack C. M. Dekkers. Does Gener Impact the Immune Response of Chicks? Ames (Iowa): Iowa State University, styczeń 2017. http://dx.doi.org/10.31274/ans_air-180814-343.

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