Gotowa bibliografia na temat „Immature and mature dendritic cells”
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Artykuły w czasopismach na temat "Immature and mature dendritic cells"
Cao, Yun, Ingrid K. Bender, Athanasios K. Konstantinidis, Soon Cheon Shin, Christine M. Jewell, John A. Cidlowski, Robert P. Schleimer i Nick Z. Lu. "Glucocorticoid receptor translational isoforms underlie maturational stage-specific glucocorticoid sensitivities of dendritic cells in mice and humans". Blood 121, nr 9 (28.02.2013): 1553–62. http://dx.doi.org/10.1182/blood-2012-05-432336.
Pełny tekst źródłaXin, Hai-ming, Yi-zhi Peng, Zhi-qiang Yuan i Hao Guo. "In vitro maturation and migration of immature dendritic cells after chemokine receptor 7 transfection". Canadian Journal of Microbiology 55, nr 7 (lipiec 2009): 859–66. http://dx.doi.org/10.1139/w09-041.
Pełny tekst źródłaBell, Diana, Pascale Chomarat, Denise Broyles, George Netto, Ghada Moumneh Harb, Serge Lebecque, Jenny Valladeau, Jean Davoust, Karolina A. Palucka i Jacques Banchereau. "In Breast Carcinoma Tissue, Immature Dendritic Cells Reside within the Tumor, Whereas Mature Dendritic Cells Are Located in Peritumoral Areas". Journal of Experimental Medicine 190, nr 10 (15.11.1999): 1417–26. http://dx.doi.org/10.1084/jem.190.10.1417.
Pełny tekst źródłaYanagawa, Yoshiki, i Kazunori Onoé. "CCL19 induces rapid dendritic extension of murine dendritic cells". Blood 100, nr 6 (15.09.2002): 1948–56. http://dx.doi.org/10.1182/blood-2002-01-0260.
Pełny tekst źródłaCavrois, Marielle, Jason Neidleman, Jason F. Kreisberg, David Fenard, Christian Callebaut i Warner C. Greene. "Human Immunodeficiency Virus Fusion to Dendritic Cells Declines as Cells Mature". Journal of Virology 80, nr 4 (15.02.2006): 1992–99. http://dx.doi.org/10.1128/jvi.80.4.1992-1999.2006.
Pełny tekst źródłaHipolito, Jolly, Hagit Peretz-Soroka, Manli Zhang, Ke Yang, Soheila Karimi-Abdolrezaee, Francis Lin i Sam Kung. "A New Microfluidic Platform for Studying Natural Killer Cell and Dendritic Cell Interactions". Micromachines 10, nr 12 (5.12.2019): 851. http://dx.doi.org/10.3390/mi10120851.
Pełny tekst źródłaOgasawara, Masahiro, Junji Tanaka, Masahiro Imamura i Masaharu Kasai. "CCL19 and CCL21 Chemokines Induce Endocytosis and Augment Antigen Presentation in Human Mature Dendritic Cells." Blood 104, nr 11 (16.11.2004): 2651. http://dx.doi.org/10.1182/blood.v104.11.2651.2651.
Pełny tekst źródłaGerosa, Franca, Barbara Baldani-Guerra, Carla Nisii, Viviana Marchesini, Giuseppe Carra i Giorgio Trinchieri. "Reciprocal Activating Interaction between Natural Killer Cells and Dendritic Cells". Journal of Experimental Medicine 195, nr 3 (4.02.2002): 327–33. http://dx.doi.org/10.1084/jem.20010938.
Pełny tekst źródłaRoche, Paul A., Satoshi Ishido, Laurence C. Eisenlohr i Kyung-Jin Cho. "Activation of Dendritic Cells Alters the Mechanism of MHC Class II Antigen Presentation to CD4 T Cells". Journal of Immunology 204, nr 1_Supplement (1.05.2020): 140.14. http://dx.doi.org/10.4049/jimmunol.204.supp.140.14.
Pełny tekst źródłaKoch, F., B. Trockenbacher, E. Kämpgen, O. Grauer, H. Stössel, A. M. Livingstone, G. Schuler i N. Romani. "Antigen processing in populations of mature murine dendritic cells is caused by subsets of incompletely matured cells." Journal of Immunology 155, nr 1 (1.07.1995): 93–100. http://dx.doi.org/10.4049/jimmunol.155.1.93.
Pełny tekst źródłaRozprawy doktorskie na temat "Immature and mature dendritic cells"
Patel, Ekta. "IgM antibodies enhance the phagocytosis of apoptotic cells by immature dendritic cells". Diss., [La Jolla] : University of California, San Diego, 2009. http://wwwlib.umi.com/cr/ucsd/fullcit?p1462525.
Pełny tekst źródłaTitle from first page of PDF file (viewed May 8, 2009). Available via ProQuest Digital Dissertations. Includes bibliographical references (p. 45-47).
Blair, Derek. "Signalling mechanisms regulating proliferation and apoptosis in immature and mature B cells". Thesis, University of Glasgow, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.400789.
Pełny tekst źródłaGeng, Shuo. "Discovery of a New Dendritic Cell Subset Derived from Immature Granulocytes". University of Toledo Health Science Campus / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=mco1294155495.
Pełny tekst źródłaIsmail, Ida Shazrina Binti. "Identification of peptides capable of targeting immature dendritic cells using phage display". Thesis, Imperial College London, 2012. http://hdl.handle.net/10044/1/10484.
Pełny tekst źródłaKhanolkar, Rahul Chaitanya. "Molecular analysis of ABIN1 expression and immunosuppressive function in immature myeloid cells". Thesis, University of Aberdeen, 2013. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=202767.
Pełny tekst źródłaSolanes, Paola. "IP3 Receptor 3 controls migration persistency and environment patrolling by immature dendritic cells". Thesis, Paris 5, 2013. http://www.theses.fr/2013PA05T020/document.
Pełny tekst źródłaThe immune response heavily relies on the migration capacity of leukocytes. These cells must stop in precise anatomical locations to fulfill a particular task. But whether and how specific functions are coordinated with migration by cell-intrinsic mechanisms is not known. We here show that in dendritic cells, which patrol their environment for the presence of antigens by internalizing extracellular material, macropinocytosis is coupled to cell migration. Coupling relies on the diversion of the Myosin II motor from its migratory function at the cell rear to macropinosomes at the cell front by the Invariant Chain, a cell-specific regulator of antigen presentation. Transient Myosin II recruitment at the cell front promotes antigen macropinocytosis and antigen delivery to endolysosomes but antagonizes cell migration. Thus, the requirement for Myosin II for both migration and antigen capture provides a molecular mechanism to couple these two processes and allow their coordination in time and space. However, the signaling pathways involved in back/front coupling in migrating immature DCs remain unknown. Here we show that calcium released from the endoplasmic reticulum through IP3 Receptors (IP3Rs) is required to maintain Myosin regulatory light Chain (MLC) phosphorylation and Myosin II back/front polarization during DC locomotion. We found that while IP3R1, 2 and 3 are required for immature DCs to reach maximal speed in 2-Dimensional and 3-Dimensional environments, IP3R3 and to a lesser extent IP3R1 positively regulate their persistency. On the contrary, silencing of IP3R3 increases antigen uptake by immature DCs, consistent with our finding showing that antigen capture is inversely coupled to DC locomotion (Appendix, manuscript #1). We propose that by promoting myosin II activity, calcium released from the ER help DCs to transiently slow-down to uptake extracellular antigens without losing their polarity and thereby optimizes their environment sampling capacity
Ito, Takeshi. "Bone marrow endothelial cells induce immature and mature B cell egress in response to erythropoietin". Kyoto University, 2018. http://hdl.handle.net/2433/232127.
Pełny tekst źródłaGutzeit, Cindy. "Interference of Varicella-Zoster Virus (VZV) with the CD1 antigen presenting system on immature dendritic cells". Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2009. http://dx.doi.org/10.18452/16059.
Pełny tekst źródłaVaricella-zoster virus (VZV) which belongs to the family of herpesviruses is restricted to humans and distributed worldwide. Primary infection of VZV causes chickenpox characterized by a disseminated rash. Thereafter, VZV establishes a lifelong latency and can be reactivated to cause herpes zoster. Since 2004 the attenuated strain V-Oka of VZV was licensed for Germany to immunize children against VZV infection. In contrast to infection by circulating virulent VZV strains, vaccination with V-Oka remains asymptomatic. The skin is the major replication site of VZV and immunological differences between virulent VZV and the vaccine should become most apparent within this immune organ. In summary, this study discovered a new immune evasion strategy of virulent VZV strains which might explain how virulent VZV strains overcome innate antiviral responses. A strong infiltration of myeloid-derived inflammatory DCs has been detected in skin lesions of herpes zoster patients. In vitro studies with monocyte-derived dendritic cells (DCs), reflecting inflammatory DCs, showed that they were efficiently infected by both, the vaccine and a virulent VZV strain. Intriguingly, a significant upregulation of CD1c molecules on VZV-infected DCs was observed. Functional investigations using intraepithelial CD1c-restricted gamma delta T cells revealed that DCs infected with the vaccine virus were fully instructed to mature, thereby promoting IFN-gamma secretion of gamma-delta T cells. In striking contrast, DCs infected with virulent VZV strains were efficiently blocked to mature functionally. In detail, they did not secrete bioactive IL-12 which is an instrumental cytokine for generation of antiviral T helper 1 responses. Moreover, virulent VZV blocked Toll-like receptor 2 (TLR2) signaling in DCs thereby preventing production of bioactive IL-12 which in turn inhibited IFN-gamma secretion by gamma-delta T cells.
Yu, Hyun Jae. "HIV Traffics Through a Specialized, Surface-accessible Intracellular Compartment During Trans-infection of T Cells by Mature Dendritic Cells". Case Western Reserve University School of Graduate Studies / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=case1266871870.
Pełny tekst źródłaKather, Angela. "Pro- and antiapoptotic events in Herpes simplex virus type 1 (HSV-1) infection of immature dendritic cells". Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2012. http://dx.doi.org/10.18452/16464.
Pełny tekst źródłaHerpes simplex virus type 1 (HSV-1) is a human pathogen which belongs to the family Herpesviridae. HSV-1 encodes several genes, which serve to efficiently prevent apoptosis in most infected cell types, thereby ensuring successful virus replication. In contrast, HSV-1 infection of one central cell type of the immune system, immature dendritic cells (iDCs), results in apoptosis. This could be one aspect of HSV-1 immunevasion. So far, the mechanisms underlying apoptosis of HSV-1 infected iDCs were poorly defined. However, it has been shown that the antiapoptotic cellular protein c-FLIP is reduced in HSV-1 infected iDCs. In this work, the amount of c-FLIP was for the first time successfully reduced in iDCs by RNA interference. This confirmed the importance of c-FLIP for viability of iDCs. Therefore, it is likely that c-FLIP reduction after HSV-1 infection also sensitizes iDCs to apoptosis. HSV-1 induced c-FLIP reduction occurred at late stages of infection and was dependent on proper expression of early and leaky late virus genes. Furthermore, it was not operative at the RNA level and was independent from the proteasome and binding to the death inducing signaling complex. Rather, c-FLIP was presumably degraded by a viral or cellular protease. In this work it was shown for the first time, that in addition to changes in the cellular apoptosis signaling network, the lack of one antiapoptotic viral factor contributes to apoptosis of HSV-1 infected iDCs. HSV-1 latency-associated transcripts (LATs) were significantly lower expressed in apoptotic iDCs compared to non-apoptotic epithelial cells, determined by microarray analysis of HSV-1 gene expression. It is known that in neurons and epithelial cells, LATs possess a potent antiapoptotic activity. This could compensate the lack of c-FLIP. Consistent with this hypothesis, a LAT deletion mutant of HSV-1 induced more apoptosis in iDCs compared to the respective wild type virus.
Części książek na temat "Immature and mature dendritic cells"
Rolink, A. G., F. Melchers i J. Andersson. "The Transition from Immature to Mature B Cells". W Current Topics in Microbiology and Immunology, 39–44. Berlin, Heidelberg: Springer Berlin Heidelberg, 1999. http://dx.doi.org/10.1007/978-3-642-60162-0_5.
Pełny tekst źródłaSchuler, Gerold, i Nikolaus Romani. "Generation of Mature Dendritic Cells from Human Blood". W Advances in Experimental Medicine and Biology, 7–13. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4757-9966-8_2.
Pełny tekst źródłaFanales-Belasio, Emanuele, Giovanna Zambruno, Andrea Cavani i Giampiero Girolomoni. "Activation of Immature Dendritic Cells Via Membrane Sialophorin (CD43)". W Advances in Experimental Medicine and Biology, 207–12. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4757-9966-8_35.
Pełny tekst źródłaLoh, Horace H., Andrew P. Smith i Nancy M. Lee. "Effects of Opioids on Proliferation of Mature and Immature Immune Cells". W Advances in Experimental Medicine and Biology, 29–33. Boston, MA: Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2980-4_5.
Pełny tekst źródłaHäusser, G. A., C. Hultgren, K. Akagawa, Y. Tsunetsugu-Yokota i A. Meyerhans. "Infection of Cultured Immature Dendritic Cells with Human Immunodeficiency Virus Type 1". W Advances in Experimental Medicine and Biology, 477–79. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1971-3_107.
Pełny tekst źródłaRuedl, C., S. Hubele, C. Rieser i H. Wolf. "The Role of CD11c+ Cells as Possible Candidates for Immature Dendritic Cells in the Murine Peyer’s Patches". W Advances in Experimental Medicine and Biology, 111–14. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4757-9966-8_18.
Pełny tekst źródłaYamashita, M., Y. Katakura, S. Matsumoto, T. Tamura, K. Teruya i S. Shirahata. "Monocytes Suppress Immune Responses of Peripheral Blood Lymphocytes: Possible Implication of Immature Dendritic Cells". W Animal Cell Technology: Basic & Applied Aspects, 237–42. Dordrecht: Springer Netherlands, 2003. http://dx.doi.org/10.1007/978-94-017-0726-8_41.
Pełny tekst źródłaMaraskovsky, Eugene, Bali Pulendran, Ken Brasel, Mark Teepe, Eileen R. Roux, Ken Shortman, Stewart D. Lyman i Hilary J. Mckenna. "Dramatic Numerical Increase of Functionally Mature Dendritic Cells in FLT3 Ligand-Treated Mice". W Advances in Experimental Medicine and Biology, 33–40. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4757-9966-8_6.
Pełny tekst źródłaKaiserlian, D., K. Vidal i J. P. Revillard. "Mature la+ murine intestinal epithelial cells with APC activity share common antigens with gut interdigitating dendritic cells". W Advances in Mucosal Immunology, 34–37. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_5.
Pełny tekst źródłaZhou, Haixia, i Stanley Perlman. "Preferential Infection of Mature Dendritic Cells by the JHM Strain of Mouse Hepatitis Virus". W Advances in Experimental Medicine and Biology, 411–14. Boston, MA: Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-33012-9_74.
Pełny tekst źródłaStreszczenia konferencji na temat "Immature and mature dendritic cells"
Jackson, C. W., N. K. Hutson i S. A. Steward. "CHANGES IN PROTEIN SYNTHESIS PROFILES OF MEGAKARYOCYTES (MK) DURING MATURATION". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643545.
Pełny tekst źródłaCagnoni, Erika F., Ana Laura N. Carvalho, Marisa Dolhnikoff, Luiz F. F. Silva, Angela B. G. Santos, Maria C. R. Medeiros, Klaus Rabe i Thais Mauad. "Mature CD83+ Dendritic Cells In Mediastinal Lymph Nodes Of Fatal Asthma". W American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4401.
Pełny tekst źródłaGannon, A. R., i D. J. Kelly. "The Changing Depth Dependant Properties of Articular Cartilage During Postnatal Development". W ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14514.
Pełny tekst źródłaFredriksson, K., X. Yao, JK Lam, HB Bhakta i SJ Levine. "Exosomes Derived from Antigen-Pulsed Immature Dendritic Cells Attenuate Airway Inflammation and Hyperresponsiveness in a Murine Model of Allergic Asthma." W American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3725.
Pełny tekst źródłaNansai, Ryosuke, Mamoru Ogata, Junichi Takeda, Wataru Ando, Norimasa Nakamura i Hiromichi Fujie. "Surface and Bulk Stiffness of the Mature Porcine Cartilage-Like Tissue Repaired With a Scaffold-Free, Stem Cell-Based Tissue Engineered Construct (TEC)". W ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204404.
Pełny tekst źródłaTruxova, Iva, Lenka Kasikova, Michal Hensler, Petr Skapa, Jan Laco, Ladislav Pecen, Lucie Belicova i in. "Abstract A24: Mature dendritic cells correlate with favorable immune infiltrate and improved prognosis in ovarian carcinoma patients". W Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; November 17-20, 2019; Boston, MA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2326-6074.tumimm19-a24.
Pełny tekst źródłaTruxova, Iva, Lenka Kasikova, Michal Hensler, Petr Skapa, Jan Laco, Ladislav Pecen, Lucie Belicova i in. "Abstract B76: Mature dendritic cells correlate with favorable immune infiltrate and improved prognosis in ovarian carcinoma patients". W Abstracts: AACR Special Conference on Tumor Immunology and Immunotherapy; November 27-30, 2018; Miami Beach, FL. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2326-6074.tumimm18-b76.
Pełny tekst źródłaSusa, Tomoya, Ryosuke Nansai, Norimasa Nakamura i Hiromichi Fujie. "Influence of Permeability on the Compressive Property of Articular Cartilage: A Scaffold-Free, Stem Cell-Based Therapy for Cartilage Repair". W ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53365.
Pełny tekst źródłaNishimura, Junya, Hiroaki Tanaka, Yuichiro Miki, Tatsuro Tamura, Tatsunari Fukuoka, Go Ohira, Masatsune Shibutani i in. "Abstract 2945: Impact of intratumoral mature dendritic cells on prognosis of the patients with esophageal squamous cell carcinoma". W Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-2945.
Pełny tekst źródłaBlank, Fabian, Peter Gerber, Alke Fink, Barbara Rothen-Rutishauser, Karin de Peyer, Thomas Geiser, Laurent Nicod i Christophe von Garnier. "Dendritic Cells Exposed To Biomedical Nanoparticles Retain An Immature Like Functional State And Show Reduced Antigen Specific CD4+ T Cell Stimulation In Vitro". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3096.
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