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Artykuły w czasopismach na temat "IgE"
Mastrangelo, G., C. Veller Fornasa, S. Pavanello, G. Marcer, M. Lazzaro, G. Milan, E. Fadda, U. Fedeli i E. Clonfero. "Polyaromatic Hydrocarbons Administered in Humans by Dermal Route Increase Total IgE". International Journal of Immunopathology and Pharmacology 16, nr 2 (maj 2003): 145–50. http://dx.doi.org/10.1177/039463200301600208.
Pełny tekst źródłaLedford, Dennis K. "Increased IgE in IgA Deficiency". Journal of Allergy and Clinical Immunology: In Practice 5, nr 6 (listopad 2017): 1795. http://dx.doi.org/10.1016/j.jaip.2017.07.013.
Pełny tekst źródłaKolopp-Sarda, M. N., i G. C. Faure. "Pourquoi les immunoglobulines s’appellent-elles IgG, IgA, IgM, IgD et IgE ?" Revue Francophone des Laboratoires 2016, nr 484 (lipiec 2016): 57–58. http://dx.doi.org/10.1016/s1773-035x(16)30250-7.
Pełny tekst źródłaAbbal, Michel. "IgE totales et IgE spécifiques". Bio Tribune Magazine 6, nr 1 (czerwiec 2003): 20–22. http://dx.doi.org/10.1007/bf03010285.
Pełny tekst źródłaCorrado, Alessia, Richard P. Ramonell, Matthew C. Woodruff, Christopher Tipton, Sarah Wise, Joshua Levy, John DelGaudio i in. "Extrafollicular IgD+ B cells generate IgE antibody secreting cells in the nasal mucosa". Mucosal Immunology 14, nr 5 (28.05.2021): 1144–59. http://dx.doi.org/10.1038/s41385-021-00410-w.
Pełny tekst źródłaSabra, Aderbal, Joseph A. Bellanti, Jonathan M. Rais, Henry J. Castro, Julia Mendez de Inocencio i Selma Sabra. "IgE and non-IgE food allergy". Annals of Allergy, Asthma & Immunology 90, nr 6 (czerwiec 2003): 71–76. http://dx.doi.org/10.1016/s1081-1206(10)61664-x.
Pełny tekst źródłaBoltansky, Howard, Jean Dyer, Steve Esworthy i Michael Kaliner. "IgE-immunotoxins. I. IgE-intact ricin". Immunopharmacology 14, nr 1 (wrzesień 1987): 35–45. http://dx.doi.org/10.1016/0162-3109(87)90007-5.
Pełny tekst źródłaXie, Liping, John T. Schroeder, Jacqueline M. Langdon, Rebecca S. Sora-Scott, Toshiaki Kawakami i Susan M. MacDonald. "Human IgE+ and IgE− are not equivalent to mouse highly cytokinergic IgE". Journal of Allergy and Clinical Immunology 121, nr 4 (kwiecień 2008): 1027–33. http://dx.doi.org/10.1016/j.jaci.2007.12.1157.
Pełny tekst źródłaDECLERCK, L., M. WESTEDT, A. CATS, B. VERMEER, E. WELTEVREDEN, C. BRIDTS i W. STEVENS. "391 IgE, IgE-containing circulating immune complexes (IgE-CIC) IgE-rheumatoid factor (RF) and skin IgE-deposition in rheumatoid arthritis (RA) patients (pts)". Journal of Allergy and Clinical Immunology 75, nr 1 (styczeń 1985): 202. http://dx.doi.org/10.1016/0091-6749(85)90527-5.
Pełny tekst źródłaPankovics, Gergő. "Az ige jelentésének összefüggése az igei vonzatstruktúrával". Magyar Nyelvőr 145, nr 2 (2021): 211–19. http://dx.doi.org/10.38143/nyr.2021.2.211.
Pełny tekst źródłaRozprawy doktorskie na temat "IgE"
Suzuki, Lisandra Akemi. "Resposta imune humoral na neurocisticercose". [s.n.], 2010. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308741.
Pełny tekst źródłaTese (doutorado) - Universidade Estadual de Campinas. Faculdade de Ciencias Medicas
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Resumo: A neurocisticercose (NC) e uma importante causa de doença neurológica em muitos paises em desenvolvimento, incluindo o Brasil. O diagnostico clinico da NC e dificultado pelo polimorfismo e pela não especificidade dos sintomas. As tecnicas de neuroimagem e pesquisa de anticorpos específicos tem contribuído para o diagnostico da NC e uma melhor compreensão dos processos fisiopatológicos dessa infecção. O presente trabalho teve como objetivo avaliar, por meio de técnicas imunoenzimaticas (ELISA), a resposta imune humoral na NC, utilizando como preparações antigênicas o liquido vesicular (LV) e uma fração glicoproteica obtida do extrato bruto de cisticercos de Taenia solium (T. solium) com afinidade por lentil-lectina (fração Gp). Cinquenta e seis amostras de liquido cefalorraquidiano (LCR), 22 de pacientes com NC e 34 de pacientes com outros problemas neurológicos, foram utilizadas para a pesquisa de IgG e suas subclasses, com os seguintes resultados: IgG-LV: 100% de sensibilidade e especificidade; IgG1 -LV: 72,73% de sensibilidade e 100% de especificidade; IgG2-LV: 81,81% de sensibilidade e 100% de especificidade; IgG3-LV: 59,09% de sensibilidade e 97,06% de especificidade; IgG4-LV: 90,91% de sensibilidade e 97,06% de especificidade; IgG-fração Gp: 90,91% de sensibilidade e 97,06% de especificidade; IgG1-fração Gp: 59,09% de sensibilidade e 91,18% de especificidade; IgG2-fração Gp: 68,18% de sensibilidade e 94,12% de especificidade; IgG3-fração Gp: 36,36% de sensibilidade e 100% de especificidade; IgG4-fração Gp: 86,36% de sensibilidade e 100% de especificidade. Quarenta e sete amostras de LCR, 16 de pacientes com NC e 31 de pacientes com outros problemas neurológicos foram utilizadas para a pesquisa de IgE, com os seguintes resultados: IgE-LV e IgE-fração Gp: 93,75% de sensibilidade e 100% de especificidade. Cinquenta e sete amostras de soros, 22 de pacientes com NC, 18 de pacientes com outras infecções e 17 de pessoas presumivelmente sadias, foram utilizadas para a pesquisa da IgG e suas subclasses, IgE, IgA e IgM, com os seguintes resultados: IgG-LV: 100% de sensibilidade e especificidade; IgG1-LV: 86,36% de sensibilidade e 94,28% de especificidade; IgG2-LV: 90,91% de sensibilidade e 97,14% de especificidade; IgG3-LV: 86,36% de sensibilidade e 97,14% de especificidade; IgG4-LV: 100% de sensibilidade e de especificidade; IgG-fração Gp: 95,45% de sensibilidade e 100% de especificidade; IgG1-fração Gp: 63,64% de sensibilidade e 94,28% de especificidade; IgG2-fração Gp: 68,18% de sensibilidade e 97,14% de especificidade; IgG3-fração Gp: 54,54% de sensibilidade e 88,57% de especificidade; IgG4-fração Gp: 90,91% de sensibilidade e 100% de especificidade; IgELV: 90,91% de sensibilidade e 97,14% de especificidade; IgE-fração Gp: 86,36% de sensibilidade e 100% de especificidade; IgA-LV: 54,54% de sensibilidade e 94,28% de especificidade; IgA-fração Gp: 13,63% de sensibilidade e 100% de especificidade. Anticorpos IgM não foram detectados com as preparações de LV e fração Gp. Nossos resultados mostraram que, com ambas as preparações antigênicas, tanto em amostras de LCR quanto em amostras de soros, a maior positividade foi obtida na detecção de anticorpos das classes IgG e IgE, seguida da positividade da IgA. Anticorpos IgM não foram detectados em amostras de soros com reações de ELISA realizadas com LV e fração Gp. Com relação as subclasses da IgG, a IgG4 apresentou, tanto em amostras de LCR como em amostras de soros, valores de positividade e concentração iguais ou superiores as outras subclasses. As reações ELISA realizadas com LV mostraram sensibilidades iguais ou superiores aquelas obtidas com a fração Gp. Considerando a complexidade e o custo final da obtenção da fração Gp, o LV pode ser considerado mais adequado para a pesquisa de anticorpos em amostras de LCR e soros de pacientes com NC.
Abstract: Neurocysticercosis (NC) is an important cause of neurological disease in many developing countries, including Brazil. The clinical diagnosis of NC is hindered by the polymorphism and non-specificity of the symptoms. Neuroimaging techniques and detection of specific antibodies have contributed to the diagnosis of NC and a better understanding of the physiopathological processes of this infection. The purpose of this study was to evaluate the humoral immune response in NC by using immunoenzymatic techniques (ELISA) in which vesicular fluid (VF) and a glycoprotein fraction purified from a crude extract of Taenia solium cysticerci with affinity for lentil-lectin (fraction Gp) were used as antigenic preparations. Fifty-six cerebrospinal fluid (CSF) samples, 22 from patients with NC and 34 from patients with other neurological disorders, were assayed for IgG and IgG subclasses, with the following results: IgG-VF: 100% sensitivity and specificity, IgG1 - VF: 72.73% sensitivity and 100% specificity, IgG2 -VF: 81.81% sensitivity and 100% specificity, IgG3 -VF: 59.09% sensitivity and 97.06% specificity, IgG4 -VF: 90.91% sensitivity and 97.06% specificity, IgG-fraction Gp: 90.91% sensitivity and 97.06% specificity, IgG1- fraction Gp: 59.09% sensitivity and 91.18% specificity, IgG2-fraction Gp: 68.18% sensitivity and 94.12% specificity, IgG3 -fraction Gp: 36.36% sensitivity and 100% specificity, IgG4 - fraction Gp: 86.36% sensitivity and 100% specificity. Forty-seven CSF samples, 16 from patients with NC and 31 from patients with other neurological disorders, were assayed for IgE, with the following results: IgE-VF and IgE-fraction Gp: 93.75% sensitivity and 100% specificity. Fifty-seven serum samples, 22 from patients with NC, 18 from patients with other infections and 17 from presumably healthy individuals, were assayed for IgG, IgG subclasses, IgE, IgA and IgM, with the following results: IgG-VF: 100% sensitivity and specificity, IgG1-VF: 86.36% sensitivity and 94.28% specificity, IgG2 -VF: 90.91% sensitivity and 97.14% specificity, IgG3 -VF: 86.36% sensitivity and 97.14% specificity, IgG4 -VF:100% sensitivity and specificity, IgG-fraction Gp: 95.45% sensitivity and 100% specificity, IgG1- fraction Gp: 63.64% sensitivity and 94.28% specificity, IgG2 -fraction Gp: 68.18% sensitivity and 97.14% specificity, IgG3 -fraction Gp: 54.54% sensitivity and 88.57% specificity, IgG4 - fraction Gp: 90.91% sensitivity and 100% specificity, IgE-VF: 90.91% sensitivity and 97.14% specificity, IgE-fraction Gp: 86.36% sensitivity and 100% specificity, IgA-VF: 54.54% sensitivity and 94.28% specificity, IgA-fraction Gp: 13.63% sensitivity and 100% specificity. No specific IgM antibodies were detected with VF and fraction Gp antigenic preparations. These results show that with the two antigenic preparations the highest positivity in CSF and serum samples was obtained for IgG and IgE antibodies, followed by positivity for IgA. No IgM antibodies were detected in serum samples assayed with VF and fraction Gp. With regard to IgG subclasses, IgG4 positivity and concentration in CSF and serum samples were higher than or equal to the other subclasses. ELISA reactions done with VF showed equal or higher sensitivities than those obtained with fraction Gp. Considering the complexity and high cost of obtaining fraction Gp, VF could be more suitable for detecting specific antibodies in CSF and serum samples from patients with NC.
Doutorado
Ciencias Biomedicas
Doutor em Ciências Médicas
Hjelm, Fredrik. "Early Immunostimulatory Effects of IgE- and IgG Antibodies". Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7209.
Pełny tekst źródłaRenault, Neil. "Construction, method development and comparative testing of an 'All-Diet' protein microarray to measure IgA, IgM, IgG and IgE in human sera and milk". Thesis, University of Nottingham, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.503929.
Pełny tekst źródłaGilbert, Sophie. "Studies on feline IgE". Thesis, University of Edinburgh, 1998. http://hdl.handle.net/1842/29776.
Pełny tekst źródłaDing, Zhoujie. "Feedback Enhancement of Immune Responses by IgE, IgM, and IgG3 Antibodies". Doctoral thesis, Uppsala universitet, Institutionen för medicinsk biokemi och mikrobiologi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-237337.
Pełny tekst źródłaAshburn, David. "The relevance of IgA and IgE assays, IgG avidity and western blotting in the diagnosis of Toxoplasma infection". Thesis, University of Aberdeen, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361776.
Pełny tekst źródłaZimmer, Anja. "Futtermittel-spezifisches IgG und IgE vor und nach Eliminationsdiäten bei allergischen Hunden". Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-148673.
Pełny tekst źródłaGuerra, Fernanda Garcia. "Anticorpos igg e ige para auto-antígenos nucleares no lúpus eritematoso sistêmico". Instituto de Ciências da Saúde, 2005. http://repositorio.ufba.br/ri/handle/ri/21734.
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CNPq; PPGIM – UFBA
O Lúpus Eritematoso Sistêmico (LES) é uma doença reumática autoimune, classificada como uma reação de hipersensibilidade tipo III, que cursa com exuberante produção de auto-anticorpos de diferentes especificidades, e reação inflamatória crônica. O LES acomete predominantemente pessoas do sexo feminino, com prevalência de 5-50 casos/100.000. Contudo, estudos epidemiológicos têm mostrado diferenças raciais na prevalência e aspectos clínicos e laboratoriais do LES. Diferenças têm sido demonstradas principalmente na freqüência dos auto-anticorpos contra antígenos nucleares extraíveis (ENA, extractable nuclear antigens) e de anticorpos IgG anti-DNA fita dupla. Existem poucos dados sobre a prevalência destes auto-anticorpos em pacientes brasileiros, principalmente usando imunoensaios sensíveis. Assim, neste estudo foram investigadas as freqüências de anticorpos antinúcleo dos isotipos IgG e IgE com especificidade antigênica para as proteínas nucleares SSA, SSB, Sm e U1-RNP, além de anticorpos IgG anti-DNA fita dupla. Soros de 21 pacientes do sexo feminino e de 26 doadoras sadias, idade entre 15-65 anos foram inicialmente triados para a presença de anticorpos antinucleares IgG e IgE através de reação de imunofluorescência indireta (IFI, FAN-IgG e FAN-IgE) com células HEp-2. Anticorpos IgG anti-DNA fita dupla, e IgG e IgE anti-ENA foram investigados por técnica de ELISA (Enzyme-Linked Immunosorbent Assay) indireto, usando fase sólida coberta com os autoantígenos purificados. A concentração sérica de IgE foi determinada por ELISA de captura. Todos os soros dos pacientes com LES (100%) foram reativos no teste de FAN-IgG (mediana do título = 640), enquanto 15/21 (71%) amostras reagiram no teste de FAN-IgE. Anticorpos IgG anti-DNA fita dupla foram detectados em 12/21 (52%) soros (mediana do título = 777 UI/ml, sensibilidade de 35,5%). Quatorze soros reagiram em ELISA-IgG para anticorpos anti-ENA, apresentando as seguintes sensibilidades: anti-RNP = 40%; anti-SSA e anti-Sm = 20%, e anti SSB = 10,5%. Anticorpos IgE anti-ENA foram detectados em sete soros, apresentando o teste de ELISA-IgE uma sensibilidade de 2,5% para 13 anticorpos anti-SSA e SSB, e de 5,3% e 17,6% para anti-Sm e anti-RNP, respectivamente. Os testes de ELISA-IgE para anticorpos anti-Sm e anti-SSA mostraram 100% de especificidade, enquanto uma especificidade de 95% foi encontrada para os testes com anticorpos anti-SSB e anti-RNP. Um aumento na concentração de IgE sérica for observado em 6 (29%) das amostras (mediana = 426 UI/ml), existindo uma correlação positiva entre os títulos de FAN-IgG e IgG anti-RNP (r = 0.796, P = 0.002), entre os títulos de IgG e IgE anti-RNP (r = 0.594, P = 0.0045) e também entre IgE anti-RNP e IgE total (r = 0.680, P = 0.0007). Concluindo, a freqüência de FAN-IgG e anticorpos IgG anti- SSA, SSB e anti-Sm nos pacientes brasileiros com LES concordou com os resultados de outros estudos internacionais, existindo contudo uma forte predominância de anticorpos anti-RNP nestes indivíduos.
Bracher, Marguerite. "IgE in immunotherapy of cancer". Thesis, King's College London (University of London), 2006. https://kclpure.kcl.ac.uk/portal/en/theses/ige-in-immunotherapy-of-cancer(08abceea-54a8-436c-9504-24742d57538d).html.
Pełny tekst źródłaKarnowski, Alexander. "Post-transcriptional regulation of IgE". [S.l. : s.n.], 2004. http://www.bsz-bw.de/cgi-bin/xvms.cgi?SWB10990069.
Pełny tekst źródłaKsiążki na temat "IgE"
Penichet, Manuel L., i Erika Jensen-Jarolim, red. Cancer and IgE. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-451-7.
Pełny tekst źródłaKim, Tong-gil. MB ... ige mwŏmnikka. [Seoul]: Chʻŏng Midiŏ, 2009.
Znajdź pełny tekst źródłaL, Spiegelberg Hans, red. IgE immunopathology I. New York: Springer Verlag, 1990.
Znajdź pełny tekst źródłaTong-gil, Kim. MB ... ige mwŏmnikka. [Seoul]: Chʻŏng Midiŏ, 2009.
Znajdź pełny tekst źródłaBruna, Dick. Ŏmma, ige mwŏya? Sŏul Tʻŭkpyŏlsi: Chigyŏngsa, 1993.
Znajdź pełny tekst źródłaNam, Mun-hŭi. Ǒmma~ ige mwŏya? Sŏul: Pukk'iang, 2005.
Znajdź pełny tekst źródłaChang, Kang-myŏng. Ch'aek, ige mwŏrago. Kyŏnggi-do P'aju-si: Arŭt'e, 2020.
Znajdź pełny tekst źródłaTong-gil, Kim. MB ... ige mwŏmnikka. [Seoul]: Chʻŏng Midiŏ, 2009.
Znajdź pełny tekst źródłaM, MacDonald Susan, red. IgE immunopathology II. Berlin: Springer International, 1993.
Znajdź pełny tekst źródłaEndre, Gyökössy. Élet és ige. Budapest: Budapest Szabadságtéri Református Egyház, 1990.
Znajdź pełny tekst źródłaCzęści książek na temat "IgE"
Mehlhorn, Heinz. "IgE". W Encyclopedia of Parasitology, 1324. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_4701.
Pełny tekst źródłaMehlhorn, Heinz. "IgE". W Encyclopedia of Parasitology, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_4701-1.
Pełny tekst źródłaGooch, Jan W. "IgE". W Encyclopedic Dictionary of Polymers, 900. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_13976.
Pełny tekst źródłavan Balen, J. A. M., A. A. Demeulemeester, M. Frölich, K. Mohrmann, L. M. Harms, W. C. H. van Helden, L. J. Mostert i J. H. M. Souverijn. "IgE". W Memoboek, 138. Houten: Bohn Stafleu van Loghum, 2012. http://dx.doi.org/10.1007/978-90-313-9129-5_75.
Pełny tekst źródłaGartler, Stanley M., R. Scott Hansen, Vinzenz Oji, Heiko Traupe, Julia Horn, Bodo Grimbacher, Srijita Sen-Chowdhry i in. "IGE". W Encyclopedia of Molecular Mechanisms of Disease, 1031. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8932.
Pełny tekst źródłaPoslussny, P., K. Vinzenz i F. Zekert. "Serumimmunglobuline (IgA, IgE, IgG, IgM) bei Patienten mit Kopf-Halskarzinomen". W Chirurgische Therapie von Kopf-Hals-Karzinomen, 335–42. Vienna: Springer Vienna, 1992. http://dx.doi.org/10.1007/978-3-7091-9087-6_39.
Pełny tekst źródłavan de Veen, Willem. "IgE Receptor". W Encyclopedia of Medical Immunology, 386–89. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9194-1_75.
Pełny tekst źródłavan de Veen, Willem. "Specific IgE". W Encyclopedia of Medical Immunology, 622–24. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9194-1_77.
Pełny tekst źródłaEiwegger, Thomas, i Zsolt Szépfalusi. "IgE Testing". W Encyclopedia of Medical Immunology, 389–92. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9194-1_78.
Pełny tekst źródłaSicherer, Scott H. "IgE- and Non-IgE-Mediated Food Allergy". W Eosinophilic Esophagitis, 219–38. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-515-6_16.
Pełny tekst źródłaStreszczenia konferencji na temat "IgE"
Jones, Thomas, Victoria Jeffery, Scott Elliott, Daniel Neville, Ellie Lanning, Thomas Brown i Anoop Chauhan. "Total IgE thresholds for detection of a positive specific IgE". W ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa1087.
Pełny tekst źródłaHoffmann, Hans Jürgen, Pernille M. Frandsen, Peter Oluf Schiøtz i Ronald Dahl. "Modulation By IgE And Anti-IgE Of FceRI Expression On Cultured Mast Cells". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1431.
Pełny tekst źródłaWoolnough, Kerry, Catherine Pashley i Andrew Wardlaw. "LATE-BREAKING ABSTRACT:Aspergillus fumigatusspecific IgE, but not total IgE orA. fumigatusspecific IgG is associated with evidence of lung damage in asthma". W Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa4357.
Pełny tekst źródłaSchneider, F., i T. Breuer. "Kalter Orbitalabszess bei Hyper-IgE-Syndrom". W Abstract- und Posterband – 90. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Digitalisierung in der HNO-Heilkunde. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1686687.
Pełny tekst źródłaMouthuy, Jonathan, Bruno Detry, Françoise Pirson i Charles Pilette. "Local IgE In Asthma: Presence Of D. Pteronyssinus-specific IgE In Induced Sputum From Intrinsic Asthma Patients". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5623.
Pełny tekst źródłaTora, I. "Idiopathic Pulmonary Fibrosis with Extremely Elevated IgE". W American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6349.
Pełny tekst źródłaRohit, Chirag V., Pranav B. Darji i Hitesh R. Jariwala. "Mitigation of IGE using STATCOM with DFIGs". W 2018 IEEE International Conference on Power Electronics, Drives and Energy Systems (PEDES). IEEE, 2018. http://dx.doi.org/10.1109/pedes.2018.8707856.
Pełny tekst źródłaSzczeklik, A., K. Sladek i J. Dropinski. "IgE-MEDIATED IMMUNOLOGICAL RESPONSE IN MYOCARDIAL INFARCTION". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643022.
Pełny tekst źródłaClerc, Sebastien, Denis Caillaud, Cara Maesano, Joana Vitte i Isabella Annesi-Maesano. "Specific IgE sensitization and asthma in French farmers". W ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa369.
Pełny tekst źródłaChu, Seung Y., Holly M. Horton, Hsing Chen, Sher Karki, Irene Leung, David E. Szymkowski i John R. Desjarlais. "Suppression Of IgE Production By XmAb7195, An Fc-Engineered Antibody That Specifically Coengages Inhibitory Receptor Fc?RIIb With IgE-BCR". W American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4068.
Pełny tekst źródłaRaporty organizacyjne na temat "IgE"
He, Dan, Hongmei Wu, Yujie Han, Min Liu i Mao Lu. A meta-analysis of topical antifungal drugs to treat atopic dermatitis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, grudzień 2021. http://dx.doi.org/10.37766/inplasy2021.12.0062.
Pełny tekst źródłaDallimore, S. R. Ice Bonding and Excess Ice. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1991. http://dx.doi.org/10.4095/132231.
Pełny tekst źródłaCoon, Max D. Sea Ice Model for Marginal Ice Zone. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2003. http://dx.doi.org/10.21236/ada615524.
Pełny tekst źródłaCoon, Max D. Sea Ice Model for Marginal Ice Zone. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2001. http://dx.doi.org/10.21236/ada626073.
Pełny tekst źródłada Rosa, Maria Inês. Accuracy of rapid IgM and IgG Antibody Test for SARS-CoV-2 Infection Diagnosis: a systematic review and meta analysis. INPLASY - International Platform of Registered Systematic Review Protocols, kwiecień 2020. http://dx.doi.org/10.37766/inplasy2020.4.0099.
Pełny tekst źródłaKarig, Fred. Ice Camp. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 1999. http://dx.doi.org/10.21236/ada629717.
Pełny tekst źródłaWadhams, Peter. Wave-Ice Interaction and the Marginal Ice Zone. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2013. http://dx.doi.org/10.21236/ada601220.
Pełny tekst źródłaWadhams, Peter, i Martin Doble. Wave-Ice Interaction and the Marginal Ice Zone. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2014. http://dx.doi.org/10.21236/ada617951.
Pełny tekst źródłaAckley, S. F., T. Maksym i S. Stammerjohn. Wave-Ice and Air-Ice-Ocean Interaction During the Chukchi Sea Ice Edge Advance. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2013. http://dx.doi.org/10.21236/ada601218.
Pełny tekst źródłaPerovich, Don, i Bonnie Light. Sunlight, Sea Ice, and the Ice Albedo Feedback in a Changing Arctic Sea Ice Cover. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2013. http://dx.doi.org/10.21236/ada601068.
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