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Gotowe bibliografie tematyczne / Hormones, Sex – Receptors / Książki

Książki na temat „Hormones, Sex – Receptors”

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Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 9 lutego 2022

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1

Gronemeyer, H., U. Fuhrmann i K. Parczyk, red. Molecular Basis of Sex Hormone Receptor Function. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-662-03689-1.

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2

Sex steroid receptors: Handbook on receptor research. Rome: Field Educational Italia, Acta Medica, 1985.

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3

O'Malley, Bert. Receptors for Reproductive Hormones. Springer, 2012.

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4

Moudgil, Virinder K. Molecular Mechanism of Steroid Hormone Action: Recent Advances. Walter De Gruyter Inc, 1985.

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1945-, Moudgil V. K., red. Molecular mechanism of steroid hormone action: Recent advances. Berlin: Walter de Gruyter, 1985.

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Moudgil, Virinder K. Molecular Mechanism of Steroid Hormone Action: Recent Advances. Walter de Gruyter, 1985.

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7

Chen, George G. Female Sex Hormones and Cancers. Nova Science Publishers, Incorporated, 2010.

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8

C, Spelsberg T., i Kumar Rajiv 1949-, red. Steroid and sterol hormone action. Boston: Nijhoff, 1987.

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9

Cao, Zhiping. Chemical signaling in neural circuits that mediate sexual behaviors. 1994.

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10

H, Gronemeyer, Fuhrmann U. 1959- i Parczyk K. 1960-, red. Molecular basis of sex hormone receptor function: New targets for intervention. Berlin: Springer-Verlag, 1998.

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11

1945-, Moudgil V. K., red. Recent advances in steroid hormone action. Berlin: De Gruyter, 1987.

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12

W, Leavitt Wendell, American Society for Cell Biology. Meeting i Symposium on Cell Biology of the Uterus (1986 : Bethesda, Md.), red. Cell and molecular biology of the uterus. New York: Plenum Press, 1987.

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13

Leavitt, W. Cell and Molecular Biology of the Uterus. Springer, 2012.

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14

Cell and Molecular Biology of the Uterus. Springer, 1988.

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15

Molecular Basis of Sex Hormone Receptor Function: New Targets for Intervention (Ernst Schering Research Foundation Workshop, 24). Springer-Verlag Telos, 1998.

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16

Novel approaches to treatment of osteoporosis. Berlin: Springer, 1998.

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17

Leavitt, Wendell W. Hormones and Cancer. Springer, 2013.

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18

Leavitt, Wendell W. Hormones and Cancer. Springer, 2012.

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19

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne i Gareth Morris-Stiff. Targeted and biological therapies. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199689842.003.0009.

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Hormone therapy describes the role of hormones in the growth of a variety of cancers, and the therapeutic effects of manipulation of hormone levels in these diseases. Sex hormones stimulate the growth of breast and prostate cancers, many of which respond to surgical removal of the hormone-secreting gonad. Pharmacological measures to deliver hormone therapy in these diseases include luteinising hormone releasing hormone (LHRH) agonists and antagonists, inhibitors of sex hormone synthesis, and inhibitors of hormone-receptor binding. These treatments have established benefits in both in the control of advanced disease and the adjuvant therapy of early-stage disease. The pros and cons of combination hormone therapy are discussed. Resistance to hormone therapy may be primary or acquired, and the likely mechanisms are described.

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20

Sex Hormone Receptor Signals in Human Malignancies. MDPI, 2019. http://dx.doi.org/10.3390/books978-3-03921-174-6.

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21

1958-, Parhar Ishwar S., red. Gonadotropin-releasing hormone: Molecules and receptors. Amsterdam: Elsevier, 2002.

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22

(Editor), Jonathan J. Li, Sara A. Li (Editor) i Janet R. Daling (Editor), red. Hormonal Carciogenesis III: Proceedings of the Third International Symposium. Springer, 2001.

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23

The effects of plant-derived compounds on sex hormone receptors: Implications for hormone-dependent cancer development and treatment. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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24

Advances In Rapid Sexsteroid Action New Challenges And New Chances In Breast And Prostate Cancers. Springer, 2011.

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25

Estrogen - Mystery Drug for the Brain?: The Neuroprotective Activities of the Female Sex Hormone. Springer, 2001.

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26

Manzon, Lori-Ann. Cloning and developmental expression of sea lamprey (Petromyzon marinus) thyroid hormone and retinoid X receptors. 2006.

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27

Elder, Grahame J. Metabolic bone disease after renal transplantation. Redaktor Jeremy R. Chapman. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0288.

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Patients who undergo kidney transplantation have laboratory, bone, and soft tissue abnormalities that characterize chronic kidney disease mineral and bone disorder (CKD-MBD). After successful transplantation, abnormal values of parathyroid hormone, fibroblast growth factor 23, calcium, phosphate, vitamin D sterols, and sex hormones generally improve, but abnormalities often persist. Cardiovascular risk remains high and is influenced by prevalent vascular calcification, and fracture risk increases due to a combination of abnormal bone ‘quality’, compounded by immunosuppressive drugs and reductions in bone mineral density. Patients with well managed CKD-MBD before transplantation generally have a smoother post-transplant course, and it is useful to assess patients soon after transplantation for risk factors relevant to the general population and to patients with CKD. Targeted laboratory assessment, bone densitometry, and X-ray of the spine are useful for guiding therapy to minimize post-transplant effects of CKD-MBD. To reduce fracture risk, general measures include glucocorticoid dose minimization, attaining adequate 25(OH)D levels, and maintaining calcium and phosphate values in the normal range. Calcitriol or its analogues and antiresorptive agents such as bisphosphonates may protect bone from glucocorticoid effects and ongoing hyperparathyroidism, but the efficacy of these therapies to reduce fractures is unproven. Alternate therapies with fewer data include denosumab, strontium ranelate, teriparatide, oestrogen or testosterone hormone replacement therapy, tibolone, selective oestrogen receptor modulators, and cinacalcet. Parathyroidectomy may be necessary, but is generally avoided within the first post-transplant year. A schema is presented in this chapter that aims to minimize harm when allocating therapy.

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28

Estrogens, Progestins and their Antagonists: Two-Volume SET (Hormones in Health and Disease). Birkhäuser Boston, 1997.

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29

Carter, C. Sue, Inbal Ben-Ami Bartal i Eric C. Porges. The Roots of Compassion. Redaktorzy Emma M. Seppälä, Emiliana Simon-Thomas, Stephanie L. Brown, Monica C. Worline, C. Daryl Cameron i James R. Doty. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190464684.013.14.

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Compassion for others and social support have survival value and health benefits. Although compassion is sometimes considered uniquely human, critical components of compassion have been described in nonhuman mammals. Studies originally conducted in social mammals and now in humans have implicated neuropeptide hormones, especially oxytocin, in social cognition, a sense of safety, and the capacity of sociality to permit compassionate responses. In contrast, the related peptide vasopressin and its receptor may be necessary for forming selective relationships and for the apparently paradoxical effects of oxytocin, which can include increases in fear and avoidance. Oxytocin and vasopressin may contribute to sex differences in compassion. Furthermore, among the processes through which oxytocin and vasopressin influence behavior and health are complex effects on the autonomic nervous system. Knowledge of the mechanisms underlying the benefits of compassion offers new insights into the healing power of positive social behaviors and social support.

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30

Burton, Derek, i Margaret Burton. Essential Fish Biology. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198785552.001.0001.

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This book summarizes the basic features of living fish. It is introduced by a chapter on the diversity of a group which has over 30,000 species, the largest within the vertebrates, describing the classification systems used for them and the variety of their habitats and morphology. Thereafter the physiology of fish is described and discussed initially by categories such as the outer boundary (the skin), the circulatory system, food processing, reproduction, hormones as integrators and controllers, the nervous system and the very complex set of sensory receptors including the eyes, ears, lateral line and electro-receptors. Unusual structures, adaptations and behaviours reveal the breadth of fish lifestyles from deep-ocean to shallow reef habitats, with both fresh water and marine margins favouring some near-terrestrial forms even emerging to spawn. With enormous ranges of size, shape and lifecycles, fish are capable of extreme longevity and amazing adjustments to their environment, including colour change, light emission by photophores and sporadic hermaphroditism (both sexes in one individual). The use of fish types by scientists is discussed. Referenced throughout, the scope of the book includes reviews of historically important and recent discoveries and some speculation on the future for fish and fish conservation. Appendices are provided to give in-depth information on some topics, including material briefly describing practical procedures, relevant to experimentation and aquaculture, which may prompt further investigation. The glossary with explanations of terms, and the copious illustrations help understanding of this complex subject area.

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31

Takao, Kumazawa, Kruger Lawrence i Mizumura Kazue, red. The polymodal receptor: A gateway to pathological pain. Amsterdam: Elsevier, 1996.

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32

(Editor), T. Kumazawa, L. Kruger (Editor) i K. Mizumura (Editor), red. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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