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Artykuły w czasopismach na temat "HMGA2"
Ismail, A. A., S. Wagner, H. Murua Escobar, S. Willenbrock, K. A. Sterenczak, M. T. Samy, A. M. Abd El-Aal, I. Nolte i P. Wefstaedt. "Effects of High-Mobility Group A Protein Application on Canine Adipose-Derived Mesenchymal Stem CellsIn Vitro". Veterinary Medicine International 2012 (2012): 1–10. http://dx.doi.org/10.1155/2012/752083.
Pełny tekst źródłaPalumbo Júnior, Antonio, Vanessa Paiva Leite de Sousa, Francesco Esposito, Marco De Martino, Floriana Forzati, Fábio Carvalho de Barros Moreira, Tatiana de Almeida Simão i in. "Overexpression of HMGA1 Figures as a Potential Prognostic Factor in Endometrioid Endometrial Carcinoma (EEC)". Genes 10, nr 5 (15.05.2019): 372. http://dx.doi.org/10.3390/genes10050372.
Pełny tekst źródłaLi, Liping, Wenyan Lu, Alison R. Moliterno, Lingling Xian, Joseph Kim, Ophelia Rogers, Jerry L. Spivak i Linda Resar. "High Mobility Group A1 Chromatin Regulators: Key Epigenetic Switches and Therapeutic Targets Required for Leukemic Transformation in JAK2 Mutant MPN". Blood 134, Supplement_1 (13.11.2019): 1680. http://dx.doi.org/10.1182/blood-2019-130262.
Pełny tekst źródłaParisi, Silvia, Silvia Piscitelli, Fabiana Passaro i Tommaso Russo. "HMGA Proteins in Stemness and Differentiation of Embryonic and Adult Stem Cells". International Journal of Molecular Sciences 21, nr 1 (6.01.2020): 362. http://dx.doi.org/10.3390/ijms21010362.
Pełny tekst źródłaVignali, Robert, i Silvia Marracci. "HMGA Genes and Proteins in Development and Evolution". International Journal of Molecular Sciences 21, nr 2 (19.01.2020): 654. http://dx.doi.org/10.3390/ijms21020654.
Pełny tekst źródłaMeireles Da Costa, Nathalia, Luis Felipe Ribeiro Pinto, Luiz Eurico Nasciutti i Antonio Palumbo Jr. "The Prominent Role of HMGA Proteins in the Early Management of Gastrointestinal Cancers". BioMed Research International 2019 (13.10.2019): 1–7. http://dx.doi.org/10.1155/2019/2059516.
Pełny tekst źródłaTessari, Michela A., Monica Gostissa, Sandro Altamura, Riccardo Sgarra, Alessandra Rustighi, Clio Salvagno, Giuseppina Caretti i in. "Transcriptional Activation of the Cyclin A Gene by the Architectural Transcription Factor HMGA2". Molecular and Cellular Biology 23, nr 24 (15.12.2003): 9104–16. http://dx.doi.org/10.1128/mcb.23.24.9104-9116.2003.
Pełny tekst źródłaResar, Linda, Donna Marie Williams, Zhizhuang Joe Zhao, Ophelia Rogers, Lingling Xian, Jerry L. Spivak i Alison R. Moliterno. "High Mobility Group A1/2 Chromatin Remodeling Proteins Associate with Polycythemia Vera Transformation to Acute Leukemia in Humans and a JAK2 V617F Transgenic Mouse Model". Blood 128, nr 22 (2.12.2016): 1958. http://dx.doi.org/10.1182/blood.v128.22.1958.1958.
Pełny tekst źródłaMoliterno, Alison R., Donna Marie Williams, Liping Li, Lingling Xian, Li Luo, Amy S. Duffield, Ophelia Rogers, Jerry L. Spivak i Linda Resar. "The High Mobility Group A1 Chromatin Regulator Is Required for Pathologic Megakaryocyte Development and Progression to Myelofibrosis in JAK2V617F Murine Models". Blood 134, Supplement_1 (13.11.2019): 472. http://dx.doi.org/10.1182/blood-2019-131432.
Pełny tekst źródłaHuang, Shaoyue, Zhen Hong, Leguo Zhang, Jian Guo, Yanhua Li i Kuo Li. "HMGA2 Promotes Brain Injury in Rats with Cerebral Infarction by Activating TLR4/NF-κB Signaling Pathway". Mediators of Inflammation 2022 (4.08.2022): 1–8. http://dx.doi.org/10.1155/2022/1376959.
Pełny tekst źródłaRozprawy doktorskie na temat "HMGA2"
Kahli, Malik. "Implication des protéines HMGA et HMGA2 dans les changements du programme de réplication au cours de la sénescence cellulaire". Thesis, Montpellier 2, 2011. http://www.theses.fr/2011MON20059/document.
Pełny tekst źródłaSenescence, considered as an irreversible cell cycle arrest, is characterized by dramatic changes in genes expression and chromatin organisation forming dense heterochromatic foci (SAHF). These changes are concomitant to a progressive decline of the capactity to replicate the genome. My PhD topic was to investigate whether the chromatin changes induced by SAHF formation could influence the replication program and modify the origin distribution along the genome at replicative senescence. We first compared the origin distribution of proliferative and pre-senescent primary fibroblasts by DNA molecular combing. Then, we mapped the origins positions in whole human genome by using the nascent strand purification assay coupled to deep sequencing.As HMGA1 and HMGA2 proteins are essential to induce SAHF formation, we designed inducible cell lines wich overexpress these proteins, triggering premature senescence. We made the same type of experiments in these cell lines in order to investigate the implication of these proteins on the changes of the replication program we observed during senescence
Saada-Bouzid, Esma. "Étude génomique et fonctionnelle de la dérégulation du gène HMGA2 dans les tumeurs adipocytaires". Thesis, Nice, 2015. http://www.theses.fr/2015NICE4000/document.
Pełny tekst źródłaBenign adipocytic tumors (AT) are mainly represented by lipomas whereas most malignant AT are Atypical Lipomatous Tumors/Well-differentiated liposarcomas (ALT/WDLPS) and dedifferentiated liposarcomas (DDLPS). HMGA2 gene (High Mobility Group A2) is rearranged in some lipomas and amplified in ALT/WDLPS and DDLPS. Thus, we hypothesized that HMGA2 played a fundamental role in benign and malignant AT genesis. In favor of this hypothesis, we observed a constant overexpression of HMGA2 in amplified ALT/WDLPS and DDLPS, and in rearranged lipomas. In a case of lipomatosis, that is a pathological proliferation of the adipocytic tissu without rearrrangement of HMGA2, the overexpression of HMGA2 was asssociated with an inhibition of the expression of several let-7 microRNAs. However, we did not find a leading role of let-7 microRNAs in the deregulation of HMGA2 expression in AT. We also studied partner fusion genes of HMGA2 in lipomas and have specifically identified a new fusion involving PPAP2B (Phosphatidic Acid Phosphatase type 2B) which is located in 1p32. We also confirmed the role of NFIB gene (9p22) in lipomas. Finally, we have established prognostic correlations in a series of 116 ALT/WDLPS and DDLPS: HMGA2 amplification was associated with ALT/WDLPS histotype and a longer survival whereas respective CDK4 and JUN amplification were associated with DDLPS and shorter survival. Thus, our data support the hypothesis of an early and major role of HMGA2 in the genesis well differentiated AT
Wei, Linxuan, Xiaolin Liu, Wenjing Zhang, Yuyan Wei, Yingwei Li, Qing Zhang, Ruifen Dong i in. "Overexpression and oncogenic function of HMGA2 in endometrial serous carcinogenesis". E-CENTURY PUBLISHING CORP, 2016. http://hdl.handle.net/10150/614759.
Pełny tekst źródłaHawsawi, Ohuod. "Role of High Mobility Group A2 (HMGA2) in Prostate Cancer". DigitalCommons@Robert W. Woodruff Library, Atlanta University Center, 2019. http://digitalcommons.auctr.edu/cauetds/184.
Pełny tekst źródłaAnnewanter, Franka Maria [Verfasser]. "Expression von TRAIL-Rezeptoren und HMGA2 im duktalen Pankreasadenokarzinom / Franka Maria Annewanter". Kiel : Universitätsbibliothek Kiel, 2014. http://d-nb.info/1064306101/34.
Pełny tekst źródłaNatarajan, Suchitra. "Roles of high mobility group AT-hook protein 2 (HMGA2) in human cancers". Elsevier, 2013. http://hdl.handle.net/1993/31092.
Pełny tekst źródłaFebruary 2016
Saada-Bouzid, Esma. "Étude génomique et fonctionnelle de la dérégulation du gène HMGA2 dans les tumeurs adipocytaires". Electronic Thesis or Diss., Nice, 2015. http://www.theses.fr/2015NICE4000.
Pełny tekst źródłaBenign adipocytic tumors (AT) are mainly represented by lipomas whereas most malignant AT are Atypical Lipomatous Tumors/Well-differentiated liposarcomas (ALT/WDLPS) and dedifferentiated liposarcomas (DDLPS). HMGA2 gene (High Mobility Group A2) is rearranged in some lipomas and amplified in ALT/WDLPS and DDLPS. Thus, we hypothesized that HMGA2 played a fundamental role in benign and malignant AT genesis. In favor of this hypothesis, we observed a constant overexpression of HMGA2 in amplified ALT/WDLPS and DDLPS, and in rearranged lipomas. In a case of lipomatosis, that is a pathological proliferation of the adipocytic tissu without rearrrangement of HMGA2, the overexpression of HMGA2 was asssociated with an inhibition of the expression of several let-7 microRNAs. However, we did not find a leading role of let-7 microRNAs in the deregulation of HMGA2 expression in AT. We also studied partner fusion genes of HMGA2 in lipomas and have specifically identified a new fusion involving PPAP2B (Phosphatidic Acid Phosphatase type 2B) which is located in 1p32. We also confirmed the role of NFIB gene (9p22) in lipomas. Finally, we have established prognostic correlations in a series of 116 ALT/WDLPS and DDLPS: HMGA2 amplification was associated with ALT/WDLPS histotype and a longer survival whereas respective CDK4 and JUN amplification were associated with DDLPS and shorter survival. Thus, our data support the hypothesis of an early and major role of HMGA2 in the genesis well differentiated AT
INGRAHAM, SUSAN ELIZABETH. "THE BALANCED, RECIPROCAL TRANSLOCATION OF CHROMOSOMAL SUBBANDS 12q15 AND 14q24 AND ALTERED GENE EXPRESSION IN UTERINE LEIOMYOMA". University of Cincinnati / OhioLINK, 2002. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1029433658.
Pełny tekst źródłaAndrieux, Joris. "Anomalies cytogénétiques et moléculaires des myélofibroses avec métaplasie myéloi͏̈de : dérégulation et hyperexpression du gène HMGA2". Lille 2, 2003. http://www.theses.fr/2003LIL2MT21.
Pełny tekst źródłaTan, E.-Jean. "Transcriptional and Epigenetic Regulation of Epithelial-Mesenchymal Transition". Doctoral thesis, Uppsala universitet, Ludwiginstitutet för cancerforskning, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-206120.
Pełny tekst źródłaKsiążki na temat "HMGA2"
Gelder, Wiebke. Identifizierung von mit HMGA2 interagierenden Proteinen. GRIN Verlag GmbH, 2011.
Znajdź pełny tekst źródłaCzęści książek na temat "HMGA2"
Fedele, Monica, i Alfredo Fusco. "Pituitary Adenoma: Role of HMGA Proteins". W Tumors of the Central Nervous System, Volume 10, 161–68. Dordrecht: Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-5681-6_18.
Pełny tekst źródłaTravers, Andrew A. "Gene Regulation by HMGA and HMGB Chromosomal Proteins and Related Architectural DNA-Binding Proteins". W DNA Conformation and Transcription, 147–58. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/0-387-29148-2_11.
Pełny tekst źródła"HMGA2". W Encyclopedia of Cancer, 1710–11. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_2773.
Pełny tekst źródłaChauvet, Norbert, Evelyne Galibert, Anne-Cecile Meunier, Valerie Rigau, Guillaume Osterstock, Eric Baccino, Monica Fedele i in. "Cadherin Changes in Human Pituitary Adenomas Can Be Reproduced in cKO-Men1 and HMGA2 Mouse Models". W TRANSLATIONAL - Pituitary Neoplasia, P1–423—P1–423. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p4.p1-423.
Pełny tekst źródła"HMGA1". W Encyclopedia of Cancer, 1710. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_2772.
Pełny tekst źródłaChiefari, Eusebio, Francesco Paonessa, Stefania Iiritano, Ilaria Le Pera, Dario Palmieri, Giuseppe Brunetti, Angelo Lupo i in. "The Camp-HMGA1-RBP4 System". W Recent Advances in Biochemistry, 175–97. Apple Academic Press, 2011. http://dx.doi.org/10.1201/b13131-11.
Pełny tekst źródłaReeves, Raymond, i Dale Edberg. "HMGA proteins: multifaceted players in nuclear function". W Chromatin Structure and Dynamics: State-of-the-Art, 155–80. Elsevier, 2004. http://dx.doi.org/10.1016/s0167-7306(03)39007-6.
Pełny tekst źródłaReeves, Raymond. "HMGA Proteins: Isolation, Biochemical Modifications, and Nucleosome Interactions". W Chromatin and Chromatin Remodeling Enzymes, Part A, 297–322. Elsevier, 2003. http://dx.doi.org/10.1016/s0076-6879(03)75020-4.
Pełny tekst źródła"Mechanism of Heavy Metal ATPase (HMA2, HMA3 and HMA4) Genes". W Nano-Phytoremediation Technologies for Groundwater Contaminates, 104–17. IGI Global, 2020. http://dx.doi.org/10.4018/978-1-5225-9016-3.ch008.
Pełny tekst źródłaWest, Natalie, Kaspar Matiasek i Clare Rusbridge. "Suspected 3-hydroxy-3-methylglutaric aciduria (HMGA) in domestic shorthaired cats". W BSAVA Congress Proceedings 2020, 442. British Small Animal Veterinary Association, 2020. http://dx.doi.org/10.22233/9781910443774.61.5.
Pełny tekst źródłaStreszczenia konferencji na temat "HMGA2"
Piscuoglio, Salvatore, Pierlorenzo Pallante, Federico Cappuzzo, Inti Zlobec, Alessandro Lugli, Alfredo Fusco i Luigi M. Terracciano. "Abstract 3195: HMGA1 and HMGA2 over-expression in human lung carcinoma". W Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-3195.
Pełny tekst źródłaKaur, Harpreet, Marianne Hütt, Xing-gang Mao, Brent A. Orr, Charles G. Eberhart i Eric H. Raabe. "Abstract 3031: HMGA2 promotes invasion and stemness in glioblastoma". W Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3031.
Pełny tekst źródłaDobersch, S., K. Rubio i G. Barreto. "HMGA2 mediated histone deposition is required for TGFB1 induced transcription". W 60. Kongress der Deutschen Gesellschaft für Pneumologie und Beatmungsmedizin e. V. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1678058.
Pełny tekst źródłaKaur, Harpreet, Marianne Hütt-Cabezas, Isabella Taylor, Laura Asnaghi, Fausto Rodriguez, Brent A. Orr, Charles G. Eberhart i Eric H. Raabe. "Abstract 4222: Targeting HMGA2 suppresses GBM stemness, invasion and tumorigenicity". W Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4222.
Pełny tekst źródłaBurnett, Riesa, Hitesh Appaiah, Poornima Bhat-Nakshatri, Jim Wikel, Peter Crooks, William Mathews i Harikrishna Nakshatri. "Abstract 1344: HMGA2-targeted drug discovery for breast cancer brain metastasis". W Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-1344.
Pełny tekst źródłaLi, Zhizhong. "Abstract A58: HMGA2 controls muscle stem cell activation and rhabdomyosarcoma progression". W Abstracts: AACR Special Conference: Pediatric Cancer at the Crossroads: Translating Discovery into Improved Outcomes; November 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.pedcan-a58.
Pełny tekst źródłaHawsawi, Ohuod A., Liza Burton, Jodi Dougan, Ana Cecillia Millena, Peri Nagappan, Shafiq Khan i Valerie Odero-Marah. "Abstract 1089: Role of high mobility group A2 (HMGA2) in prostate cancer". W Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-1089.
Pełny tekst źródłaВолеводз, Н. Н., З. С. Зюзикова, Е. А. Писарева, В. В. Шестерикова, К. Д. Кокорева i К. Г. Забудская. "АТИПИЧНАЯ ФОРМА СИНДРОМА СИЛЬВЕРА-РАССЕЛА, АССОЦИИРОВАННАЯ С НОВОЙ МУТАЦИЕЙ В ГЕНЕ HMGA2". W IV Конференция по орфанным и детским эндокринным заболеваниям "Эндокринная орфанетика: достижения и перспективы". ФГБУ «НМИЦ эндокринологии» Минздрава России, 2024. http://dx.doi.org/10.14341/eoap2024-025.
Pełny tekst źródłaKaur, Harpreet, Marianne Hutt, Xing-gang Mao, Charles G. Eberhart i Eric H. Raabe. "Abstract 3742: Targeting the stem cell factor HMGA2 to improve outcomes in glioblastoma." W Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-3742.
Pełny tekst źródłaSingh, Indrabahadur, Aditi Mehta, Nihan Öztürk, Julio Cordero, Adriana Contreras, Diya Hasan, Claudia Cosentino i in. "HMGA2 mediated epigenetic regulation of Gata6 controls epithelial WNT signaling during lung development". W ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.oa3228.
Pełny tekst źródłaRaporty organizacyjne na temat "HMGA2"
Aly, Radi, James H. Westwood i Carole L. Cramer. Novel Approach to Parasitic Weed Control Based on Inducible Expression of Cecropin in Transgenic Plants. United States Department of Agriculture, maj 2003. http://dx.doi.org/10.32747/2003.7586467.bard.
Pełny tekst źródłaGinzberg, Idit, Richard E. Veilleux i James G. Tokuhisa. Identification and Allelic Variation of Genes Involved in the Potato Glycoalkaloid Biosynthetic Pathway. United States Department of Agriculture, sierpień 2012. http://dx.doi.org/10.32747/2012.7593386.bard.
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