Gotowe bibliografie tematyczne / Heart disease

Gotowa bibliografia na temat „Heart disease”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 11 marca 2023

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Artykuły w czasopismach na temat "Heart disease"

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Nurmamadovna, Ishankulova Nasiba. "Coronary Heart Disease". American Journal of Medical Sciences and Pharmaceutical Research 03, nr 02 (28.02.2021): 31–36. http://dx.doi.org/10.37547/tajmspr/volume03issue02-04.

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The article covers the etiology, pathogenesis, classification, diagnosis, clinical picture and treatment of coronary heart disease, provides a literature review. Cardiovascular disease (CVD) represents the leading cause of death among women as well as men. The number of deaths due to CVD in women are greater than in men. There are significant gender-related differences concerning CVD.
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Martins, Cristiane. "Congenital heart disease". Clinical Cardiology and Cardiovascular Interventions 3, nr 11 (20.11.2020): 01–02. http://dx.doi.org/10.31579/2641-0419/097.

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Sudhakar, B. G. K. "Structural heart disease interventions". Clinical Research and Clinical Trials 3, nr 5 (25.06.2021): 01–05. http://dx.doi.org/10.31579/2693-4779/042.

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Seed for invasive cardiology was sown in early part of nineties. Cardiac catheterization was actually pioneered by Werner Forssmann in 1929. However, credit for therapeutic interventional cardiology should go to US vascular radiologist, Charles Theodore Dotter for performing first peripheral arterial angioplasty [PTA] in 1964. Subsequently, a German cardiologist by name Andreas Gruentzig adapted the technique in 1974 to suit coronary artery disease and performed the first human coronary balloon angioplasty to treat blockage in coronary artery in 1977.
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Siu, S. C. "CONGENITAL HEART DISEASE: Heart disease and pregnancy". Heart 85, nr 6 (1.06.2001): 710–15. http://dx.doi.org/10.1136/heart.85.6.710.

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Shakhnoza, Iskandarova, i Amilova Asalya. "PREVENTION OF CORONARY HEART DISEASE". American Journal of Medical Sciences and Pharmaceutical Research 04, nr 04 (1.04.2022): 19–21. http://dx.doi.org/10.37547/tajmspr/volume04issue04-05.

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Primary prevention, based on healthy lifestyle habits that prevent the emergence of risk factors, is the preferred method of reducing cardiovascular risk. Reducing the prevalence of obesity is the most urgent task, and it is pleiotropic in that it affects blood pressure, lipid profile, glucose metabolism, inflammation, progression of atherothrombotic disease. Physical activity also improves several risk factors, with the added potential to lower heart rate.
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Shah, Dr Aashka M. "Fibrinogen and Ischaemic Heart Disease". International Journal of Scientific Research 3, nr 6 (1.06.2012): 257–58. http://dx.doi.org/10.15373/22778179/june2014/81.

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Allan, L. "CONGENITAL HEART DISEASE: Antenatal diagnosis of heart disease". Heart 83, nr 3 (1.03.2000): 367. http://dx.doi.org/10.1136/heart.83.3.367.

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Jarvelin, M. R. "CONGENITAL HEART DISEASE: Fetal and infant markers of adult heart diseases". Heart 84, nr 2 (1.08.2000): 219–26. http://dx.doi.org/10.1136/heart.84.2.219.

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Taksande, Amar, i Sachin Dhamke. "Critical Congenital Heart Disease in Newborns". Pediatric Education and Research 5, nr 2 (2017): 87–95. http://dx.doi.org/10.21088/per.2321.1644.5217.17.

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C Blankenship, James. "Racial Disparities in Ischemic Heart Disease". Clinical Cardiology and Cardiovascular Interventions 5, nr 7 (8.08.2022): 01–07. http://dx.doi.org/10.31579/2641-0419/269.

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Ischemic heart disease is a leading cause of death for most racial and ethnic minorities and is estimated to be responsible for one in seven deaths in the United States. Black adults have the highest burden of cardiovascular disease. Despite significant advances in treatment of ischemic heart disease and an increased awareness of racial disparities, racial and ethnic minority groups remained disproportionately burdened with ischemic heart disease and experience higher mortality and rehospitalization rates compared to Whites. This review details racial disparities in different syndromes, treatments, and outcomes of ischemic heart disease and suggests strategies for reducing or eliminating these disparities.
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Rozprawy doktorskie na temat "Heart disease"

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Holt, Jim. "Heart Disease". Digital Commons @ East Tennessee State University, 2003. https://dc.etsu.edu/etsu-works/6509.

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Quigley, Gillian Margaret. "Inflammation of the heart in heart disease". Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/inflammation-of-the-heart-in-heart-disease(eae19e58-aeb4-4673-924e-1dbd1c831fec).html.

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Heart failure patients have dysfunction of the cardiac conduction system that contributes to a high burden of arrhythmias including atrial fibrillation and sudden cardiac death. Heart failure has been associated with the inflammatory response, but it is unknown if inflammation is playing a role in the remodelling of the cardiac conduction system in heart failure. Inflammation has been shown to be present in the myocardium from failing hearts and it is known to have detrimental effects on cardiac function, inducing fibrosis, remodelling of ion channels and even arrhythmias. However, the effect of inflammation on the cardiac conduction system has not been investigated. The aims of this study were to determine if there is an increase of pro-inflammatory cytokines and inflammatory cells in the cardiac conduction system in heart failure. In addition, to identify if there is possible inflammation-associated fibrosis and apoptosis in the cardiac conduction system in heart failure. To test these aims, three models of heart failure were used: a rat model of pulmonary arterial hypertension, a rabbit model of congestive heart failure and a rat model of myocardial infarction. In the rat model of pulmonary arterial hypertension there was a bradycardia, a prolongation of the QT interval, and an increase in the atrioventricular and ventricular refractory periods, suggesting electrical remodelling in these animals. The rats with pulmonary arterial hypertension displayed an increase in pro-inflammatory cytokines such as interleukins 1β and TGFβ in the right side of the heart, including the sinoatrial node and right Purkinje fibres of the cardiac conduction system. In addition, in these areas, there was an increase in components of the extracellular matrix, including fibronectin, collagen I and vimentin. Histology revealed regions of non-myocyte nuclei, only in the right ventricle of the rats with pulmonary arterial hypertension. Immunohistochemistry demonstrated patches of CD68 and vimentin expression (markers for macrophages and fibroblasts, respectively) in the right side of the heart in these animals. TUNEL staining also revealed an increase in apoptosis in the right side of the heart. In the rabbit model of congestive heart failure, the region most affected by inflammation was the right atrium, while few changes were measured in the ventricles or cardiac conduction system. Although these results are surprising, it is suggested that the atria could be more sensitive to the physical stretch produced in this model. In the rat model of myocardial infarction, there were regions of non-myocyte nuclei in the border zone. This region also had increases in pro-inflammatory and fibrosis markers. In conclusion, this work has presented the novel finding that there can be inflammation in the cardiac conduction system in heart failure. This could be contributing to the arrhythmias seen in heart failure patients. This could possibly lead the way to anti-inflammatories as a possible novel therapeutic for heart failure patients.
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Rix, Louise Katharine. "Biochemistry of heart disease". Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334889.

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Misner, Scottie. "Heart Disease in Women". College of Agriculture and Life Sciences, University of Arizona (Tucson, AZ), 1999. http://hdl.handle.net/10150/146457.

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Heart disease, or cardiovascular disease (CVD), includes stroke and coronary heart disease, which are serious disorders of the heart and blood vessel system. Heart disease is the leading cause of illness and death in North American women. This publication addresses what kind of heart diseases affect women and how to live the heart-healthy way.
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Brown, Jessie Ann. "RUNX2 in Embryonic Heart Development and Heart Disease". Thesis, The University of Arizona, 2011. http://hdl.handle.net/10150/144250.

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Zapanta, Laurence (Laurence F. ). "Heart rate variability in mice with coronary heart disease". Thesis, Massachusetts Institute of Technology, 2005. http://hdl.handle.net/1721.1/34118.

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Thesis (S.M.)--Massachusetts Institute of Technology, Dept. of Electrical Engineering and Computer Science, 2005.
Includes bibliographical references (leaves 69-71).
Heart rate variability (HRV), the beat-to-beat fluctuation of the heart rate, is a non-invasive test that measures the autonomic regulation of the heart. Assessment of HRV has been shown to predict the risk of mortality in patients after an acute myocardial infarction. Recently, the Krieger lab at MIT developed genetically engineered double knockout (dKO) mice that develop coronary artery disease accompanied by spontaneous myocardial infarctions and die at a very young age. This thesis investigated whether HRV could function as a prognostic indicator in the dKO mouse. A novel method for estimating physiological state of the mouse from the electrocardiogram using an innovative activity index was developed in order to compare HRV variables at different times while controlling for physiologic state. Traditional time and frequency domain variables were used to assess the prognostic power of HRV. Results have shown that none of the HRV variables were helpful in predicting mortality in the dKO mice. Mean heart rate showed some prognostic power, but it was not consistent in all the dKO mice. Finally, the activity index developed in this thesis provided a reliable metric for activity in mice as validated by a camera with motion detection.
by Laurence Zapanta.
S.M.
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Pratt, Rebekah Janet. "Broken hearts and the heart broken : living with, and dying from, heart failure in Scotland". Thesis, University of Edinburgh, 2012. http://hdl.handle.net/1842/8273.

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Heart failure is a common and serious chronic condition, which can be as ‘malignant’ as most forms of cancer (Stewart, MacIntyre, Hole, Capewell, & McMurray, 2001). Recent estimates are that around 40,000 men and 45,000 women are living with heart failure in Scotland (Stewart, MacIntyre, Capewell, & McMurray, 2003). Heart failure is significantly influenced by socioeconomic factors, with people on lower incomes being more likely to develop, and die faster from, heart failure (McAlister et al, 2004). There is a growing body of research on the experience of living with heart failure, however, none provides serious consideration of the role of socioeconomic factors in impacting the experience of heart failure, and some qualitative research may actually obscure such factors. There were two main aims in this thesis. One was to explore how qualitative research methods can better consider the relationship between experience and broader context, such as the influence of socio-economic factors on health. The other aim was to examine the experiences of people as they live with and die from heart failure in ways that situate their accounts in the broader context of their lives. An initial research study, on which I was the main researcher, focused on the experiences of 30 people living with advanced heart failure. These people, their carers and key health professionals were interviewed, where possible, three times over a six months period. This thesis re-examines that study, focusing on 20 of those participants, for which a total of 122 interviews were conducted. I used a dialogical approach to see whether the socioeconomic context of heart failure for these respondents, could be captured through exploring experiences, performance, relationships, discourses and institutional practices, the social processes that mediate the relationship between socioeconomic disadvantage and chronic diseases were explored. This offers important learning in relation to the experience of living with heart failure, along with the experience of providing care. The findings highlight the need to broaden our view of chronic illness beyond biomedical approaches, and grow our methodological approaches along with that, in order to develop knowledge and practice that has relevance for people who live with and die from heart failure.
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Field, Mark Leonard. "Biochemical basis of heart disease". Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.386788.

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Rowlinson, Giselle Victoria. "Connexins in congenital heart disease". Thesis, Imperial College London, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.550483.

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Gap junctions are clusters of transmembrane channels, composed of connexins (Cx), that facilitate electrical and chemical communication between the cytoplasmic compartments of contiguous cells. Three connexins are expressed in cardiac myocytes, Cx40, Cx43 and Cx45. Targeted deletion of these connexin genes in mice results in cardiac malformations and conduction abnormalities. From this background, the question arises as to whether connexins play a role in human congenital heart disease. Atrial and ventricular tissue samples were studied from patients undergoing cardiac surgery. Immunoconfocal microscopy and western blot analysis of atrial tissue revealed that expression of CX40 and CX43 in children and adults with congenital heart disease is the same as that in the normal adult atrium, irrespective of the underlying malformation. Normal adult ventricular working myocytes express only CX43. Study of control ventricular samples in children confirmed that, as in adults, CX43 only is expressed. However, immunoconfocal microscopy of samples from patients with right ventricular outflow obstruction (tetralogy of Fallot and double chambered right ventricle) revealed that in addition to CX43, CX40 is also highly expressed. Expression is heterogeneous and CX40 is eo-localised with CX43. Quantitative western blot analysis showed that up to 10% of the total connexin expressed in these samples is CX40. As patients re-operated following previous repair (with markedly different underlying haemodynamics) still demonstrated high CX40 expression in the working myocardium, these results suggest that a lack of normal CX40 repression during development leads to heart malformations . . Gap junction channels formed from each connexin isofonn have distinctive biophysical properties. Connexin eo-expression further alters these properties. To investigate the functional consequences of the connexin eo-expression patterns observed in the ventricular samples, in vitro cell models were used. Intercellular communication was assessed using cell-to-cell Lucifer Yellow dye transfer in an inducible RLE cell line and conducjion properties were studied in an atrial myocyte (HL-l) cell line. The findings are consistent with the idea that altered function arising from abnormal embryonic connexin expression is a contributor to some types of human cardiac malformation.
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Grech, Victor. "Congenital heart disease in Malta". Thesis, University College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.286359.

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Książki na temat "Heart disease"

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Heart disease. Wyd. 2. New York: Macmillan Pub. Co., 1987.

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Dittmer, Lori. Heart disease. Mankato, Minn: Creative Education, 2012.

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Silverstein, Alvin. Heart disease. Minneapolis, Minn: Twenty-First Century Books, 2006.

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Arnold, Caroline. Heart disease. New York: Franklin Watts, 1990.

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Heart disease. Mankato, Minn: LifeMatters, 2001.

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Farris, Naff Clay, red. Heart disease. Detroit: Greenhaven Press, 2008.

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Gold, John Coopersmith. Heart disease. Parsippany, N.J: Crestwood House, 1996.

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Heart disease. Oxford: Oxford University Press, 2010.

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Howard, Judd. Heart disease. Springhouse, Pa: Springhouse Corp., 1986.

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Wetherill, Douglas. Heart disease. Cincinnati, OH: Betterway Books, 2000.

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Części książek na temat "Heart disease"

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Whang, William. "Heart Disease". W Encyclopedia of Behavioral Medicine, 1034. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39903-0_1266.

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Swift, Simon. "Heart Disease". W Chronic Disease Management for Small Animals, 97–134. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781119201076.ch5.

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Orbell, Sheina, Havah Schneider, Sabrina Esbitt, Jeffrey S. Gonzalez, Jeffrey S. Gonzalez, Erica Shreck, Abigail Batchelder i in. "Heart Disease". W Encyclopedia of Behavioral Medicine, 939. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4419-1005-9_1266.

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Wallen, Håkan, i Rickard Linder. "Heart disease". W Essential Guide to Blood Coagulation, 97–104. Oxford, UK: John Wiley & Sons Ltd, 2013. http://dx.doi.org/10.1002/9781118327517.ch9.

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Harr, Jeffrey N., Philip F. Stahel, Phillip D. Levy, Antoine Vieillard-Baron, Yang Xue, Muhammad N. Iqbal, Jeffrey Chan i in. "Heart Disease". W Encyclopedia of Intensive Care Medicine, 1013. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1669.

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Suri, Vanita, i Pooja Sikka. "Heart Disease". W Labour Room Emergencies, 77–83. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-10-4953-8_8.

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Whang, William. "Heart Disease". W Encyclopedia of Behavioral Medicine, 1. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4614-6439-6_1266-2.

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Wallace, Rodrick, i Deborah Wallace. "Heart Disease". W Gene Expression and Its Discontents, 87–105. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-48078-7_8.

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Theisler, Charles. "Heart Disease". W Adjuvant Medical Care, 155–56. New York: CRC Press, 2022. http://dx.doi.org/10.1201/b22898-169.

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de Melo Barbosa, Marcia, Maria do Carmo Pereira Nunes i Regina Müller. "Rheumatic Heart Disease: A Neglected Heart Disease". W Prevention of Cardiovascular Diseases, 143–57. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-22357-5_15.

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Streszczenia konferencji na temat "Heart disease"

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Mesihović-Dinarević, Senka, Mirza Halimić i Almira Kadić. "ACQUIRED AND GENETICALLY PREDISPOSED HEART DISEASE IN CHILDREN". W Acquired Heart Diseases. Academy of Sciences and Arts of Bosnia and Herzegovina, 2015. http://dx.doi.org/10.5644/pi2015-158-01.

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Begić, Fatima. "ACQUIRED VALVULAR HEART DISEASE IN CHILDREN: OUR EXAMPLES". W Acquired Heart Diseases. Academy of Sciences and Arts of Bosnia and Herzegovina, 2015. http://dx.doi.org/10.5644/pi2015-158-03.

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Haxhibeqiri-Karabdić, Ilirijana, Emir Kabil i Haris Vranić. "ISCHAEMIC HEART DISEASE – SURGICAL TREATMENT AND POST-OPERATIVE COMPLICATIONS". W Acquired Heart Diseases. Academy of Sciences and Arts of Bosnia and Herzegovina, 2015. http://dx.doi.org/10.5644/pi2015-158-07.

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Omerčahić-Dizdarević, Aida, Velma Selmanović i Adisa Čengić. "RHEUMATIC FEVER: A DISEASE THAT SHOULD NOT YET BE FORGOTTEN". W Acquired Heart Diseases. Academy of Sciences and Arts of Bosnia and Herzegovina, 2015. http://dx.doi.org/10.5644/pi2015-158-02.

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Veena, N., S. Mahalakshmi, G. Anisha Diyya, Alekhya Allada i Malavika S. Anand. "Heart Disease Prediction System". W 2021 International Conference on Forensics, Analytics, Big Data, Security (FABS). IEEE, 2021. http://dx.doi.org/10.1109/fabs52071.2021.9702552.

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Rahman, Mafizur, Maryam Mehzabin Zahin i Linta Islam. "Effective Prediction On Heart Disease: Anticipating Heart Disease Using Data Mining Techniques". W 2019 International Conference on Smart Systems and Inventive Technology (ICSSIT). IEEE, 2019. http://dx.doi.org/10.1109/icssit46314.2019.8987776.

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Dube, Harshali, Shweta Madge, Prajakta Jagtap, Pooja Potdar i Nilesh Bhandare. "Review on Heart Disease Classification". W 2020 5th International Conference on Communication and Electronics Systems (ICCES). IEEE, 2020. http://dx.doi.org/10.1109/icces48766.2020.9137880.

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González S., A. "Heart Disease Detection Using Wavelets". W MEDICAL PHYSICS: Eighth Mexican Symposium on Medical Physics. AIP, 2004. http://dx.doi.org/10.1063/1.1811827.

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Doki, Srichand, Siddhartha Devella, Sumanth Tallam, Sai Sujeeth Reddy Gangannagari, P. Sampathkrishna Reddy i G. Pradeep Reddy. "Heart Disease Prediction Using XGBoost". W 2022 Third International Conference on Intelligent Computing Instrumentation and Control Technologies (ICICICT). IEEE, 2022. http://dx.doi.org/10.1109/icicict54557.2022.9917678.

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Karthikeyan, Ganesan. "13 Heart failure and rheumatic heart disease in india". W 1st Asia Pacific Advanced Heart Failure Forum (APAHFF), 15th December 2017, Hong Kong. BMJ Publishing Group Ltd, British Cardiovascular Society and Asia Pacific Heart Association, 2018. http://dx.doi.org/10.1136/heartasia-2018-apahff.13.

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Raporty organizacyjne na temat "Heart disease"

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De Jong, Marla J., i Debra K. Moser. Anxiety and Heart Disease. Fort Belvoir, VA: Defense Technical Information Center, styczeń 2003. http://dx.doi.org/10.21236/ada420275.

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Corey Goldman, Corey Goldman. What's Calcium's role in heart and kidney disease? Experiment, grudzień 2013. http://dx.doi.org/10.18258/1719.

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Doustmehraban, Maryam, Hengameh Ferdosian, Seyed Ali Emami, Elahe Sedighi, Mina Moridi i Hadi Zamanian. Efficacy of online mindfulness in heart disease: A systematic review protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, październik 2021. http://dx.doi.org/10.37766/inplasy2021.10.0055.

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Review question / Objective: The aim of this systematic review is to investigate the efficacy of mindfulness interventions on physiologic and psychological outcomes in heart disease. Condition being studied: Heart disease is one of the most important life-threatening conditions and various psychological and physical treatments are considered for them. one of the psychological interventions which is used widely in a chronic disease context, is mindfulness-based interventions that could be delivered online. Information sources: Web OF Science Core Collection; EMBASE; Scopus; Psych Info.
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Sawyer, Anthony, i Lee Anne Flagg. State Declines in Heart Disease Mortality in the United States, 2000–2019. National Center for Health Statistics ( U.S.), grudzień 2021. http://dx.doi.org/10.15620/cdc:112339.

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Krauss, Ronald. CRADA Final Report: Genetic Testing for Evaluation of Heart Disease Risk. Office of Scientific and Technical Information (OSTI), styczeń 2002. http://dx.doi.org/10.2172/1157021.

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Pleszkoch, Mark G., Hilda B. Klasky, Aneel Advani, Edmon Begoli, Aileen Boone i Stephan D. Fihn. Game Theoretic Approach for Understanding and Modeling Clinical Pathways (Stable Ischemic Heart Disease). Office of Scientific and Technical Information (OSTI), październik 2018. http://dx.doi.org/10.2172/1513403.

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Weng, JIeqiong, Jingfang Zhang, Ke Xu, Mengfei Yuan, Tingting Yao, Xinyu Wang i Xiaoxu Shen. Efficacy of Shexiang Baoxin Pills Combined with Statins on Blood Lipid Profile in Patients with Coronary Heart Disease: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, wrzesień 2022. http://dx.doi.org/10.37766/inplasy2022.9.0100.

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Review question / Objective: P（Population) ：Patients with coronary heart disease; I（Intervention) : Statins treatment in combination with Shexiang Baoxin pill; C（Comparison): Statins alone; O（Outcome）: Improvement of symptoms and blood lipids; S（Study design）：Clinical randomized trials. Eligibility criteria: To be included, trials were required to meet the following criteria: (1) patients were included in the studies according to diagnostic criteria of coronary heart disease established by the WHO, InternationalSociety of Cardiology and Association (ISCA), Internal Medicine, 7th edition ( IM-7th), Practice of InternalMedicine, 14th edition ( PIM-14th), Guidelines for the Diagnosis of Cardiovascular Diseases in InternalMedicine, 3rd edition (GIM-3rd) or conventional diagnostic criteria (CDC) including assessment of anginapectoris and electrocardiogram (ECG) results; (2) the study was conducted as a randomized controlled trial.
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Dong, Guoqi, Mengye Lu, Xiaoliang Wu, Hao Chen, Hongru Zhang i Yihuang Gu. Network meta-analysis of Traditional Chinese medicines for depression in coronary heart disease patients. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, maj 2020. http://dx.doi.org/10.37766/inplasy2020.5.0036.

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Wienke, Andreas, Anne M. Herskind, Kaare Christensen, Axel Skytthe i Anatoli I. Yashin. The influence of smoking and BMI on heritability in susceptibility to coronary heart disease. Rostock: Max Planck Institute for Demographic Research, styczeń 2002. http://dx.doi.org/10.4054/mpidr-wp-2002-003.

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Chen, Yujia, i Wen Li. Periodontitis Increase the Risk of Heart Failure. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, listopad 2022. http://dx.doi.org/10.37766/inplasy2022.11.0084.

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Review question / Objective: Can periodontitis increase the risk of heart failure? Condition being studied: Previous studies reported that periodontal disease is associated with higher risk of HF. However, their conclusions conflicted a little bit with each other. In this case, a systematic review may help drawing a more comprehensive conclusion. Therefore, the purpose of the present analysis is to tell if periodontitis is associated with HF as well as the subtypes of HF.
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