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Książki na temat „Gram Negative Baterial Infections”

Autor: Grafiati

Data publikacji: 6 września 2023

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1

Burd, Randall S. Immunotherapy of gram-negative bacterial sepsis. Austin, Tex: R.G. Landes, 1992.

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2

Bill, McWilliams, i Boyce Dean, red. Gram-negative burn wound infection: An evidence based approach. Saarbrücken, Germany: Lambert Academic Publishing, 2011.

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3

Ming, Tan, i Patrik M. Bavoil. Intracellular pathogens I: Chlamydiales. Washington, DC: ASM Press, 2012.

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4

Chlamydia atherosclerosis lesion: Discovery, diagnosis and treatment. London: Springer, 2007.

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5

Hawley, Louise B. High-yield microbiology and infectious diseases. Philadelphia: Lippincott Williams & Wilkins, 2000.

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6

V, Tetz V., i Totolian A. A, red. Molecular biology of bacteria. Commack, N.Y: Nova Science Publishers, 1997.

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7

Wiwanitkit, Viroj. Focus on emerging food borne infections. New York: Nova Science Publishers, 2008.

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8

Chlamydial infection: A clinical and public health perspective. Basel: Karger, 2013.

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9

J, Klastersky, i Merck Sharp & Dohme., red. Prophylaxis of gram negative infections in neutropenic patients: Based on a symposium in Zurich, Switzerland on 6 June 1987. Oxford: Pergamon Press, 1988.

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10

1941-, Prior Richard B., red. Clinical applications of the Limulus amoebocyte lysate test. Boca Raton, Fla: CRC Press, 1990.

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11

1962-, Schmidt Axel, red. Bartonella and Afipia species emphasizing Bartonella henselae. Basel: Karger, 1998.

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12

E, Bergogne-Bérézin, Friedman Herman 1931-2007 i Bendinelli Mauro, red. Acinetobacter biology and pathogenesis. New York: Springer, 2008.

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13

F, Baquero, red. Evolutionary biology of bacterial and fungal pathogens. Washington, D.C: ASM Press, 2007.

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14

E, Faist, i Merck Sharp & Dohme., red. Differential release and impact of antibiotic-induced endotoxin. New York: Raven Press, 1995.

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15

Jean, Klastersky, red. Prophylaxis of gram negative infections in neutropenic patients: Based on a symposium in Zurich, Switzerland on 6 June 1987 sponsored by Merck, Sharp and Dohme. Oxford: Pergamon Press, 1988.

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16

Bell, C. Campylobacter: A practical approach to the organism and its control in foods. Oxford, U.K: Blackwell, 2010.

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17

Stephanie, Payne, i Sutton Alison RGN, red. Genito-urinary medicine for nurses. London: Whurr Publishers, 1996.

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18

Organization, World Health, i Food and Agriculture Organization of the United Nations., red. Risk assessments of Salmonella in eggs and broiler chickens. Geneva, Switzerland: World Health Organization, 2002.

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19

Matthews, Philippa C. Infections caused by Gram-negative bacteria. Redaktor Philippa C. Matthews. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198737773.003.0004.

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This chapter consists of short notes, diagrams, and tables to summarize Gram-negative organisms that are significant causes of disease in the tropics and subtropics. This includes Escherichia coli, Shigella, and Salmonella species (including typhoid and paratyphoid), Brucella, melioid, Campylobacter, and meningococci. For ease of reference, each topic is broken down into sections, including classification, epidemiology, microbiology, pathophysiology, clinical syndromes, diagnosis, treatment, and prevention.

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20

Martinov, Svetoslav P. Chlamydiae and Chlamydial Infections. River Publishers, 2022.

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21

Martinov, Svetoslav P. Chlamydiae and Chlamydial Infections. River Publishers, 2018.

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22

Martinov, Svetoslav P. Chlamydiae and Chlamydial Infections. River Publishers, 2018.

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23

Martinov, Svetoslav P. Chlamydiae and Chlamydial Infections. River Publishers, 2018.

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24

Martinov, Svetoslav P. Chlamydiae and Chlamydial Infections. River Publishers, 2022.

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25

Martinov, Svetoslav P. Chlamydiae and Chlamydial Infections. River Publishers, 2022.

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26

Aztreonam: The antibiotic discovery for gram-negative infections. London: Royal Society of Medicine, 1985.

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27

Häcker, Georg. Biology of Chlamydia. Springer, 2019.

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28

Häcker, Georg. Biology of Chlamydia. Springer, 2018.

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29

Barbara Van Der Pol (Foreword), Timothy R. Moss (Editor) i Alison J. Woodland (Editor), red. Chlamydia, The Silent Disease. Merit Publishing International, 2007.

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30

Friedman, Herman, Mauro Bendinelli i Eugénie Bergogne-Bérézin. Acinetobacter: Biology and Pathogenesis. Springer, 2010.

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31

Immunotherapy of Gram-Negative Bacterial Sepsis (Medical Intelligence Unit). Landes Bioscience, 1992.

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32

Food and Agriculture Organization of the United Nations. Selection and Application of Methods for the Detection and Enumeration of Human-Pathogenic Halophilic Vibrio Spp. in Seafood. Food & Agriculture Organization of the United Nations, 2017.

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33

Culture Negative Orthopedic Biofilm Infections Springer Series on Biofilms. Springer, 2012.

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34

Shor, Allan. Chlamydia Atherosclerosis Lesion: Discovery, Diagnosis and Treatment. Springer, 2016.

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35

Urbaschek, Bernhard, Renate Urbaschek i Erwin Neter. Gram-Negative Bacterial Infections and Mode of Endotoxin Actions: Pathophysiological, Immunological, and Clinical Aspects. Springer London, Limited, 2012.

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36

Urbaschek, Bernhard, Renate Urbaschek i Erwin Neter. Gram-Negative Bacterial Infections and Mode of Endotoxin Actions: Pathophysiological, Immunological, and Clinical Aspects. Urbaschek Bernhard, 2012.

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37

Buhner, Stephen Harrod. Healing lyme disease coinfections: Complementary and holistic treatments for Bartonella and Mycoplasma. 2013.

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38

Buhner, Stephen Harrod. Healing Lyme Disease Coinfections: Complementary and Holistic Treatments for Bartonella and Mycoplasma. Inner Traditions International, Limited, 2013.

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39

Malfertheiner, Peter, i Hans Ditschuneit. Helicobacter pylori, Gastritis and Peptic Ulcer. Springer, 2011.

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40

Malfertheiner, Peter, i Hans Ditschuneit. Helicobacter Pylori, Gastritis and Peptic Ulcer. Springer London, Limited, 2012.

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41

World Health Organization (WHO). Salmonella and Campylobacter in Chicken Meat: Meeting Report. World Health Organization, 2010.

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42

Wilson, John W., i Lynn L. Estes. Acute Native Joint Infections. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199797783.003.0109.

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•Clinical: Acute monoarticular swelling, typically of a large joint, with fever and pain•Radiology: Normal osseus structures (early) with soft-tissue swelling•Laboratory: Elevated leukocytes, erythrocyte sedimentation rate, and C-reactive protein•Arthrocentesis: >50,000–100,000 leukocytes (predominantly neutrophils), absence of crystals, Gram stain often negative...

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43

(Editor), Fernando Baquero, Cesar Nombela (Editor), Gail H. Cassell (Editor) i Jose A. Gutierrez-fuentes (Editor), red. Evolutionary Biology of Bacterial and Fungal Pathogens. ASM Press, 2007.

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44

Salmonella Infections, Networks of Knowledge, and Public Health in Britain, 1880-1975. Oxford University Press, 2015.

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45

Goffin, Eric, Laura Labriola i Michel Jadoul. Bacterial and fungal infections in patients on peritoneal dialysis. Redaktor Jonathan Himmelfarb. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0270.

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Infections specifically related to peritoneal dialysis include peritonitis on the one hand, and exit-site and tunnel infections on the other hand.The diagnosis of peritonitis rests on the classical triad of cloudy dialysate, abdominal pain, and presence of < 100 white-cells (including < 50 % polymorphonuclear cells) within the dialysate effluent. Because peritonitis is associated with high mortality and morbidity rates, empiric antibiotics should be initiated without delay, covering both Gram-positive and Gram-negative organisms. Most regimens include vancomycin or a first-generation cephalosporin for the former, and a third-generation cephalosporin or an aminoglycoside for the latter. Antibiotics are usually administered via the intraperitoneal route. Prophylaxis with an anti-fungal agent has to be considered in diabetic patients and in those who just received prolonged antibiotic administration. Cure is obtained in up to 80 % of the cases ; treatment failure however may occur with refractory or relapsing peritonitis episodes. This is especially common in fungal or fecal associated peritonitis, and will require catheter withdrawal. The incidence of peritonitis has dramatically decreased in recent years with the advent of new connectology systems, and both adequate preventive measures and improved patients’ education. Still it is not clearly documented that new biocompatible dialysate fluids have a favorable effect on peritonitis incidence.Exit-site and tunnel infections are defined by the presence of a purulent discharge around the catheter and by erythema, oedema and tenderness of the subcutaneous pathway of the catheter, respectively. Antibiotics are recommended in case of documented infection. Cuff shaving may sometimes be required, as well as catheter removal in case of unfavourable evolution.

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46

Sutton, Alison. Genito-Urinary Medicine for Nurses. Whurr Publishers, 1996.

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47

(Editor), C. Wray, i A. Wray (Editor), red. Salmonella in Domestic Animals (Cabi Publishing). CABI, 2000.

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48

Oostdijk, Evelien, i Marc Bonten. Oral, nasopharyngeal, and gut decontamination in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0287.

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Many infections are caused by enteric bacilli, presumably from endogenous origin. Selective decontamination of the digestive tract (SDD) was developed to selectively eliminate the aerobic Gram-negative bacilli from the digestive tract, leaving the anaerobic flora unaffected. As an alternative to SDD, investigators have evaluated the effects of selective oropharyngeal decontamination (SOpD) alone. Most detailed data on the effects of SDD and SOpD in ICU-patients come from two studies performed in Dutch ICUs. The Dutch studies provide strong evidence that SDD and SOpD reduce ICUmortality, ICU-acquired bacteraemia with Gram-negative bacteria, and systemic antibiotic use. Although successful application has been reported from several solitary ICUs across Europe, it is currently unknown to what extent these effects can be achieved in settings with different bacterial ecology. More studies are needed on the use of SDD or SOpD as a measure to control outbreaks with multidrug resistant bacteria.

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49

Risk Assessments of Salmonella in Eggs And Broiler Chickens: Interpretative Summary (Microbiological Risk Assessment Series). Bernan Assoc, 2003.

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50

Fletcher, Tom, i Nick Beeching. Rickettsial infection. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0314.

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Rickettsial infections are caused by a variety of obligate intracellular, Gram-negative bacteria from the genera Rickettsia, Orientia, Ehrlichia, and Anaplasma. Rickettsia is further subdivided into the spotted fever group and the typhus group. Bartonella and Coxiella burnetii bacteria are similar to rickettsiae and cause similar diseases. The range of recognized spotted fever group infections is rapidly expanding, complementing long-recognized examples such as Rocky Mountain spotted fever (Rickettsia rickettsii) in the US, and Australian tick typhus (Rickettsia australis), as well as those in southern Europe and Africa. Animals are the predominant reservoir of infection, and transmission to people is usually through ticks, mites, fleas, or lice, during blood-feeding or from scarification of faeces deposited on the skin. This chapter focuses on the two of the most relevant infections encountered in UK practice: African tick typhus, and Q fever.

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