Książki na temat „Gour”

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1

Khayat, S. A. Gour gah. Tehran: Hirmand, 1999.

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2

Le gour des abeilles: Récit. [Paris]: Gallimard, 1985.

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3

Gour met guide to Paris. New York: Vendome Press, 1995.

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4

1950-, Vājapeyī Santosha Kumāra, i Renner Zsuzsanna, red. Catalogue of the collection of the Harisingh Gour Archaeological Museum of Sagar University. New Delhi: Northern Book Centre, 2012.

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5

Maniatopoulos, Kōstas. Sukiko Haa: "ti apegine ho gouv-gou?". Place of publication not identified]: Ekdoseis tou Kampou, 2009.

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6

Czarnocki, Jan. Klimenie Gór Świętokrzyskich =: Klimenii Sventokshiskikh gor = Clymeniids of the Góry Świętokrzyskie Mts. Warszawa: Wydawnictwa Geologiczne, 1989.

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7

National Conference on Electrical and Optical Properties of Solids (1987 Dept. of Physics, Dr. H.S. Gour University). Electrical and optical behaviour of solids: Proceedings of National Conference on Electrical and Optical Properties of Solids, held during March 26, 27, 28, 1987, at the Department of Physics, Dr. H.S. Gour University, Sagar, India. Delhi, India: Mittal Publications, 1989.

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8

Newcombe, David S. Gout. Redaktor Dwight R. Robinson. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-4264-5.

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9

Dennis, Keesmaat, red. Goud. Amsterdam: Prometheus, 2012.

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10

Goud. [Vianen]: The House of Books, 2000.

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11

Geeraerts, Jef. Goud. Wyd. 2. Amsterdam: Meulenhoff, 1995.

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12

K, Choi Hyon, red. Gout. Philadelphia: Saunders, 2006.

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13

Tong feng ke yi ke fu. Taibei Shi: Zhu fu zhi you chu ban shi ye you xian gong si, 2000.

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14

Shengling, Yu, red. Tong feng ju tong xiao chu fa. Taibei: Da zhan chu ban she, 1996.

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15

Tong feng jiang zuo. Taibei Shi: Hao yüan wen hua shi yeh yu xian gong si, 1996.

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16

Newcombe, David S. Gout: Basic science and clinical practice. London: Springer, 2013.

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17

Kusuki, Nishioka, Su Jung-yüan i Yang Hongru, red. Tʾung feng ti chen tuan yü pao chien. Tʾai-pei shih: Shu chʾüan ch'u pan she, 1992.

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18

Pang, Chun Mei. The precaution & treatment against gout. Taiwan: Nuan Liu, 2004.

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19

Tong feng: Zheng zhuang, yü fang, zhi liao. Taibei Shi: Lian guang tu shu gu fen yu xian gong si, 1994.

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20

John, Breeschoten, red. Als jij goud zegt, is het goud. Amsterdam [etc.]: Contact, 2009.

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21

Emile Goué. [Paris]: Bleu nuit, 2012.

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22

Blauw goud. Amsterdam: Sijthoff, 2012.

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23

Turks goud. Amsterdam: Van Gennep, 2007.

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24

Goud!: Roman. Leuven: Van Halewyck, 2004.

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25

Gourd seed. Athens, Ga: Maypop Books, 1993.

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26

(Gerrit-Jan), Kramer Gert-Jan, red. Goud geld. [Laren (NH)]: Verbum Crime, 2009.

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27

translator, Yang Muxi, red. Deng dai gou gou. Taibei Shi: Huang guan wen hua chu ban you xian gong si, 2016.

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28

Gou gou bing tuan. Batu Caves, Selangor, Malaysia: Da jiang chu ban she, 2010.

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29

Gou shuo gou jiang. Kunming Shi: Yunnan ren min chu ban she, 2001.

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30

Cowley, Joy. Chong wu gou gou. Shijiazhuang Shi: He bei shao nian er tong chu ban she, 2015.

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31

er, Fei nai, i Zhang he ling. Gou gou xin shi. Bei jing: Jing hua chu ban she, 2010.

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32

Cherpillod, Gaston. Le gour noir: Recits. L'Aire bleue, 1995.

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33

Clancy, Tom. Submarine: A Guide Gour Inside a Nuclear Warship ('He zi jian zhi lu', in traditional Chinese, NOT in English). Xing Guang, 1994.

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34

Rascaroli, Laura. Framing. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780190238247.003.0008.

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Essay films performatively display the process of thinking; hence, issues of textual and contextual framing are at the center of their practice. To frame is to detach an object from its background and, thus, to carve a gap between object and world. The chapter starts from a discussion of Irina Botea’s Picturesque (2012): an argument centered on the tourist image is predicated on a dual recourse to the frame—first intended as the literal operation of mise en cadre and then as narrative, ideological, and cultural framing. It goes on to show that the specificity of the essay film is to be sought not in its production of objects, but in their arrangement and that this arrangement reflects a fundamental structure of gap. The method of framing as visible search for an object is further explored via two archival essays, Mohammadreza Farzad’s Gom o gour (Into Thin Air, 2010) and Peter Thompson’s Universal Diptych (1982).
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35

Dr. Sir Harisingh Gour commemoration volume, December 25, 1957, eighth death-anniversary: Ḍô. Sara Harisiṃha Gaura smāraka-grantha, 25 Disambara, 1957, āṭhavīṃ nidhana tithi. Sagar, M.P: University of Saugar, 1985.

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36

Electrical and optical behaviour of solids: Proceedings of National Conference on Electrical and Optical Properties of Solids, held during March 26, 27, ... Dr. H.S. Gour University, Sagar, India. Mittal Publications, 1989.

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37

Gout and Rheumatic Gout. Creative Media Partners, LLC, 2022.

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38

Gout and Rheumatic Gout. HardPress, 2020.

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39

Gout and Rheumatic Gout. BiblioLife, 2008.

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40

Carioscia, Marshall. Gout Cookbook : Gout-Friendly Recipes: Gout Friendly Cookbook. Independently Published, 2021.

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41

Dalbeth, Nicola. Gout research tools. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198748311.003.0012.

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Although most tools used in gout research are generic, there are some important gout-specific instruments. New gout classification criteria were published in 2015. Outcome measure domains have been identified for both acute and chronic gout studies. A preliminary flare definition has been reported. Gout-specific, patient-reported outcome measure instruments allow assessment of gout disease activity and impact of tophi. Imaging scoring systems allow quantification of joint damage, inflammation, and urate burden in gout.
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42

Gout. Franklin Classics, 2018.

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43

Dalbeth, Nicola. Gout. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0141.

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Gout is a common and treatable disorder of purine metabolism. Gout typically presents as recurrent self-limiting episodes of severe inflammatory arthritis affecting the foot. In the presence of persistent hyperuricaemia, tophi, chronic synovitis, and joint damage may develop. Diagnosis of gout is confirmed by identification of monosodium urate (MSU) crystals using polarizing light microscopy. Hyperuricaemia is the central biochemical cause of gout. Genetic variants in certain renal tubular urate transporters including SLC2A9 and ABCG2, and dietary factors including intake of high-purine meats and seafood, beer, and fructose, contribute to development of hyperuricaemia and gout. Gout treatment includes: (1) management of the acute attack using non-steroidal anti-inflammatory drugs (NSAIDs), corticosteroids, or low-dose colchicine; (2) prophylaxis against gout attacks when commencing urate-lowering therapy (ULT), with NSAIDs or colchicine; and (3) long-term ULT to achieve a target serum urate of less than 0.36 mmol/litre. Interleukin (IL)-1β‎ is a central mediator of acute gouty inflammation and anti-IL-1β‎ therapies show promise for treatment of acute attacks and prophylaxis. The mainstay of ULT remains allopurinol. However, old ULT agents such as probenecid and benzbromarone and newer agents such as febuxostat and pegloticase are also effective, and should be considered in patients in whom allopurinol is ineffective or poorly tolerated. Management of gout should be considered in the context of medical conditions that frequently coexist with gout, including type 2 diabetes, hypertension, dyslipidaemia, and chronic kidney disease. Patient education is essential to ensure that acute gout attacks are promptly and safely managed, and long-term ULT is maintained.
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44

Dalbeth, Nicola. Gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0141_update_003.

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Gout is a common and treatable disorder of purine metabolism. Gout typically presents as recurrent self-limiting episodes of severe inflammatory arthritis affecting the foot. In the presence of persistent hyperuricaemia, tophi, chronic synovitis, and joint damage may develop. Diagnosis of gout is confirmed by identification of monosodium urate (MSU) crystals using polarizing light microscopy. Hyperuricaemia is the central biochemical cause of gout. Genetic variants in certain renal tubular urate transporters including SLC2A9 and ABCG2, and dietary factors including intake of high-purine meats and seafood, beer, and fructose, contribute to development of hyperuricaemia and gout. Gout treatment includes: (1) management of the acute attack using non-steroidal anti-inflammatory drugs (NSAIDs), corticosteroids, or low-dose colchicine; (2) prophylaxis against gout attacks when commencing urate-lowering therapy (ULT), with NSAIDs or colchicine; and (3) long-term ULT to achieve a target serum urate of less than 0.36 mmol/litre. Interleukin (IL)-1β‎ is a central mediator of acute gouty inflammation and anti-IL-1β‎ therapies show promise for treatment of acute attacks and prophylaxis. The mainstay of ULT remains allopurinol. However, old ULT agents such as probenecid and benzbromarone and newer agents such as febuxostat and pegloticase are also effective, and should be considered in patients in whom allopurinol is ineffective or poorly tolerated. Management of gout should be considered in the context of medical conditions that frequently coexist with gout, including type 2 diabetes, hypertension, dyslipidaemia, and chronic kidney disease. Patient education is essential to ensure that acute gout attacks are promptly and safely managed, and long-term ULT is maintained.
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45

Porter, Roy, Roy Porter G. S. Rousseau i G. S. Rousseau. Gout. Yale University Press, 2000.

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46

Gout. Elsevier, 2019. http://dx.doi.org/10.1016/c2016-0-05041-6.

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47

Lipsky, Peter E., i Naomi Schlesinger. Gout. Elsevier - Health Sciences Division, 2018.

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48

Mortis, Igor. Gout. Legend Press, 2013.

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49

Luff, Arthur Pearson. Gout. Creative Media Partners, LLC, 2018.

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50

Dalbeth, Nicola, Lisa Stamp i Tony Merriman. Gout. Oxford University Press, 2016.

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