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Artykuły w czasopismach na temat "Genome damage"
Proshkina, Ekaterina, Mikhail Shaposhnikov i Alexey Moskalev. "Genome-Protecting Compounds as Potential Geroprotectors". International Journal of Molecular Sciences 21, nr 12 (24.06.2020): 4484. http://dx.doi.org/10.3390/ijms21124484.
Pełny tekst źródłaHenkel, Ralf R., i Daniel R. Franken. "Sperm DNA Fragmentation: Origin and Impact on Human Reproduction". Journal of Reproductive and Stem Cell Biotechnology 2, nr 2 (grudzień 2011): 88–108. http://dx.doi.org/10.1177/205891581100200204.
Pełny tekst źródłaLIU, Wei-Feng, Shan-Shan YU, Guan-Jun CHEN i Yue-Zhong LI. "DNA Damage Checkpoint, Damage Repair, and Genome Stability". Acta Genetica Sinica 33, nr 5 (maj 2006): 381–90. http://dx.doi.org/10.1016/s0379-4172(06)60064-4.
Pełny tekst źródłaAlhegaili, Alaa S., Yunhee Ji, Nicolas Sylvius, Matthew J. Blades, Mahsa Karbaschi, Helen G. Tempest, George D. D. Jones i Marcus S. Cooke. "Genome-Wide Adductomics Analysis Reveals Heterogeneity in the Induction and Loss of Cyclobutane Thymine Dimers across Both the Nuclear and Mitochondrial Genomes". International Journal of Molecular Sciences 20, nr 20 (15.10.2019): 5112. http://dx.doi.org/10.3390/ijms20205112.
Pełny tekst źródłaFerrand, Juliette, Beatrice Rondinelli i Sophie E. Polo. "Histone Variants: Guardians of Genome Integrity". Cells 9, nr 11 (5.11.2020): 2424. http://dx.doi.org/10.3390/cells9112424.
Pełny tekst źródłaThomas, Mark, Gaetan Burgio, David J. Adams i Vivek Iyer. "Collateral damage and CRISPR genome editing". PLOS Genetics 15, nr 3 (14.03.2019): e1007994. http://dx.doi.org/10.1371/journal.pgen.1007994.
Pełny tekst źródłaPetruseva, I. O., A. N. Evdokimov i O. I. Lavrik. "Molecular Mechanism of Global Genome Nucleotide Excision Repair". Acta Naturae 6, nr 1 (15.03.2014): 23–34. http://dx.doi.org/10.32607/20758251-2014-6-1-23-34.
Pełny tekst źródłaTrakhtenberg, I. M., Y. I. Gubsky, E. L. Levitsky i I. F. Belenichev. "Biochemical mechanisms of free-radical damage to the nuclear genome by cadmium". Ukrainian Biochemical Journal 90, nr 3 (25.06.2018): 5–16. http://dx.doi.org/10.15407/ubj90.03.005.
Pełny tekst źródłaCollura, Ada, Joel Blaisonneau, Giuseppe Baldacci i Stefania Francesconi. "The Fission Yeast Crb2/Chk1 Pathway Coordinates the DNA Damage and Spindle Checkpoint in Response to Replication Stress Induced by Topoisomerase I Inhibitor". Molecular and Cellular Biology 25, nr 17 (1.09.2005): 7889–99. http://dx.doi.org/10.1128/mcb.25.17.7889-7899.2005.
Pełny tekst źródłaHu, Jinchuan, Jason D. Lieb, Aziz Sancar i Sheera Adar. "Cisplatin DNA damage and repair maps of the human genome at single-nucleotide resolution". Proceedings of the National Academy of Sciences 113, nr 41 (29.09.2016): 11507–12. http://dx.doi.org/10.1073/pnas.1614430113.
Pełny tekst źródłaRozprawy doktorskie na temat "Genome damage"
Banerjee, Ujjwal Kumar. "3-D Genome organization of DNA damage repair". Thesis, Strasbourg, 2017. http://www.theses.fr/2017STRAJ121.
Pełny tekst źródłaOur genome is constantly under attack by endogenous and exogenous factors which challenge its integrity and lead to different types of damages. Double strand breaks (DSBs) constitute the most deleterious type of damage since they maylead to loss of genetic information, translocations and cell death. All the repair processes happen in the context of a highly organized and compartmentalized chromatin. Chromatin can be divided into an open transcriptionally active compartment (euchromatin) and a compacted transcriptionally inactive compartment (heterochromatin). These different degrees of compaction play important roles in regulating the DNA damage response. The goal of my first project was to understand the influence of 3D genome organization on DNA repair. I used two complementary approaches to induce and map DSBs in the mouse genome. My results have shown that enrichment of the DNA damage repair factor γH2AX occurs at distinct loci in the mouse embryonic stem cell genome and that the damage persists in the heterochromatin compartment while the euchromatin compartment is protected from DNA damage. For my second project, I mapped the genomic footprint of 53BP1, a factor involved in DSBs repair, in asynchronous and G1 arrested U2OS cells to identify novel 53BP1 binding sites. My results have identified novel 53BP1 binding domains which cover broad regions of the genome and occur in mid to late replicating regions of the genome
Alrumaihi, Faris Abdulrahman I. "Assessment of UVR-induced DNA damage and repair in nuclear genome versus mitochondrial genome". Thesis, University of Leicester, 2016. http://hdl.handle.net/2381/37614.
Pełny tekst źródłaManning, Francis C. R. "The persistence of carcinogen damage in specific regions of the genome". Thesis, University of Nottingham, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.277377.
Pełny tekst źródłaAlpi, Arno. "DNA damage checkpoint pathways and the maintenance of genome stability in C. elegans". Diss., lmu, 2004. http://nbn-resolving.de/urn:nbn:de:bvb:19-24487.
Pełny tekst źródłaKasparek, Torben Rudolf. "Identification and characterisation of determinants of genome stability in response to a double-strand break". Thesis, University of Oxford, 2013. http://ora.ox.ac.uk/objects/uuid:78e0a145-22c8-4abd-a746-e18c1939f5c9.
Pełny tekst źródłaDurant, Stephen Thomas. "The role of DNA mismatch repair in cellular responses to DNA damage and drug resistance". Thesis, University of Glasgow, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.312133.
Pełny tekst źródłaFrigola, Rissech Joan 1991. "Determinants of the local mutation rate variability along the genome". Doctoral thesis, Universitat Pompeu Fabra, 2020. http://hdl.handle.net/10803/669530.
Pełny tekst źródłaLa velocitat a la que les mutacions s’acumulen al llarg del genoma no és uniforme sinó que depèn de diversos factors. Alguns dels més coneguts són l’empaquetament de la cromatina, el moment de replicació o la transcripció. La majoria d’aquests factors creen variacions mutacionals que abarquen grans àrees del genoma, incloent varies megabases. En els últims anys, però, també s’ha identificat variabilitat en el ritme en que s’acumulen les mutacions a escala molt més petita, en regions de poques bases. Aquesta tesi es centra en l’estudi de dos d’aquestes variacions locals en el ritme en que les mutacions tenen lloc. Primer, hem descrit una reducció en el número de mutacions somàtiques en els exons causades per errors de la AND polimerasa, que hem atribuït a una major eficàcia del mecanisme encarregat aquest tipus d’errors en els exons. En segon lloc, hem estudiat com les lesions en el DNA causades per la llum ultraviolada es generen i són reparades als llocs d’unió dels factors de transcripció i hem determinat fins a quin punt cada un d’aquests processos permeten explicar l’inesperat número de mutacions en aquestes regions. La presència d’aquestes variacions locals la velocitat a la que les mutacions s’acumulen al llarg del genoma posen de manifest la dificultat de modelar correctament aquest procés, un procediment central en molts estudis evolutius i de genòmica del càncer.
Blance, Stephen J. "DNA repair and recombination in Streptomyces coelicolor". Thesis, University of Liverpool, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.367139.
Pełny tekst źródłaFinneran, Bryan P. "Developing and Testing an ELISA Biosensor for Measuring UV-Induced Viral Genome and Protein Damage". The Ohio State University, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1593640837647181.
Pełny tekst źródłaBhattacharjee, Sonali. "The role of Fml1 and its partner proteins Mhf1 and Mhf2 in promoting genome stability". Thesis, University of Oxford, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711640.
Pełny tekst źródłaKsiążki na temat "Genome damage"
Aging of the genome: The dual role of the DNA in life and death. Oxford ; New York: Oxford University Press, 2007.
Znajdź pełny tekst źródłaYosef, Shiloh, i SpringerLink (Online service), red. The DNA Damage Response: Implications on Cancer Formation and Treatment. Dordrecht: Springer Netherlands, 2009.
Znajdź pełny tekst źródłaGenes and the environment. London: Taylor & Francis, 1999.
Znajdź pełny tekst źródłaAchary, V. Mohan Murali, Anca Macovei, Kaoru Okamoto Yoshiyama i Ayako N. Sakamoto, red. Maintenance of Genome Integrity: DNA Damage Sensing, Signaling, Repair and Replication in Plants. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-820-7.
Pełny tekst źródłaVijg, Jan. Aging of the Genome: The Dual Role of DNA in Life and Death. Oxford University Press, USA, 2007.
Znajdź pełny tekst źródłaVijg, Jan. Aging of the Genome: The Dual Role of DNA in Life and Death. Oxford University Press, USA, 2007.
Znajdź pełny tekst źródłaDodds, Chris, Chandra M. Kumar i Frédérique Servin. Pathophysiological changes of ageing and their relevance to anaesthesia. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198735571.003.0002.
Pełny tekst źródłaClement, Jan, i Piet Maes. Hantaviral infections. Redaktor Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0188_update_001.
Pełny tekst źródłaCattran, Daniel C., i Heather N. Reich. Membranous glomerulonephritis. Redaktor Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0064_update_001.
Pełny tekst źródłaCatherine, Rice-Evans, i Burdon R. H, red. Free radical damage and its control. Amsterdam: Elsevier, 1994.
Znajdź pełny tekst źródłaCzęści książek na temat "Genome damage"
Rasmussen, Lene Juel, i Keshav K. Singh. "Oxidative Damage and Repair in the Mitochondrial Genome". W Oxidative Damage to Nucleic Acids, 109–22. New York, NY: Springer New York, 2007. http://dx.doi.org/10.1007/978-0-387-72974-9_9.
Pełny tekst źródłaEvans, M. D., i M. S. Cooke. "Oxidative Damage to DNA in Non-Malignant Disease: Biomarker or Biohazard?" W Genome and Disease, 53–66. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000092500.
Pełny tekst źródłaEppink, Berina, Jeroen Essers i Roland Kanaar. "Interplay and Quality Control of DNA Damage Repair Mechanisms". W Genome Organization and Function in the Cell Nucleus, 395–415. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2011. http://dx.doi.org/10.1002/9783527639991.ch16.
Pełny tekst źródłaRichard, Derek J., i Kum Kum Khanna. "Single-Stranded DNA Binding Proteins Involved in Genome Maintenance". W The DNA Damage Response: Implications on Cancer Formation and Treatment, 349–66. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-2561-6_16.
Pełny tekst źródłaGolato, T., i D. M. Wilson III. "Chapter 30. DNA Damage and the Maintenance of Nuclear Genome Integrity in Aging and Associated Phenotypes". W DNA Damage, DNA Repair and Disease, 388–425. Cambridge: Royal Society of Chemistry, 2020. http://dx.doi.org/10.1039/9781839162541-00388.
Pełny tekst źródłaMao, Peng, i John J. Wyrick. "Genome-Wide Mapping of UV-Induced DNA Damage with CPD-Seq". W The Nucleus, 79–94. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0763-3_7.
Pełny tekst źródłaMitra, J., H. Wang, M. Kodavati, S. Mitra i M. L. Hegde. "Chapter 13. Emerging Roles of Non-canonical RNA Binding Proteins in the Repair of Genome Damage Linked to Human Pathologies". W DNA Damage, DNA Repair and Disease, 301–22. Cambridge: Royal Society of Chemistry, 2020. http://dx.doi.org/10.1039/9781839160769-00301.
Pełny tekst źródłaSakai, Wataru, i Kaoru Sugasawa. "DNA Damage Recognition and Repair in Mammalian Global Genome Nucleotide Excision Repair". W DNA Replication, Recombination, and Repair, 155–74. Tokyo: Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-55873-6_7.
Pełny tekst źródłaRechkunova, N. I., i O. I. Lavrik. "Nucleotide Excision Repair in Higher Eukaryotes: Mechanism of Primary Damage Recognition in Global Genome Repair". W Subcellular Biochemistry, 251–77. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-90-481-3471-7_13.
Pełny tekst źródłaRavi, Dashnamoorthy, i Alexander James Roy Bishop. "Identification of Genes Required for Damage Survival Using a Cell-Based RNAi Screen Against the Drosophila Genome". W Methods in Molecular Biology, 9–26. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-998-3_2.
Pełny tekst źródłaStreszczenia konferencji na temat "Genome damage"
"Lesion recognition and cleavage of damage-containing G-quadruplexes by DNA glycosylases". W Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-357.
Pełny tekst źródła"DNA damage to nervous tissue due to lead intoxication combined with glucose loading". W Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-338.
Pełny tekst źródłaGreen, Abby M., Sebastien Landry, James P. Evans, Sophia Shalhout, Ashok S. Bhagwat i Matthew D. Weitzman. "Abstract 3016: APOBEC3 enzymes induce damage to the cellular genome during DNA replication". W Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3016.
Pełny tekst źródłaGilbertson, Matthew, Radhika Patel, Karin C. Nitiss i John L. Nitiss. "Abstract 3580: Topoisomerase II mediated DNA damage generates unique classes of genome rearrangements". W Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-3580.
Pełny tekst źródłaWeaver, Alice N., Tiffiny S. Cooper, Hoa Q. Trummell, James A. Bonner, Eben L. Rosenthal i Eddy S. Yang. "Abstract A1-63: Characterizing the DNA damage repair defect in HPV-positive oropharyngeal squamous cell carcinoma". W Abstracts: AACR Special Conference: Translation of the Cancer Genome; February 7-9, 2015; San Francisco, CA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.transcagen-a1-63.
Pełny tekst źródłaAlhegaili, Alaa, George D. Jones i Marcus S. Cooke. "Abstract LB-163: Genome-wide analysis of DNA damage and repair reveals differential sites and rates of repair, together with differential sensitivities to damage". W Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-lb-163.
Pełny tekst źródłaLu, Wei-ting, Lykourgos-Panagiotis Zalmas, Thomas Webber, Nnennaya Kanu i Charles Swanton. "Abstract 2567: TRACERx: Intra-tumor subclonal driver mutation results in defective DNA damage response (DDR) and genome instability". W Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-2567.
Pełny tekst źródłaLu, Wei-ting, Lykourgos-Panagiotis Zalmas, Thomas Webber, Nnennaya Kanu i Charles Swanton. "Abstract 2567: TRACERx: Intra-tumor subclonal driver mutation results in defective DNA damage response (DDR) and genome instability". W Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-2567.
Pełny tekst źródłaGout, J., L. Perkhofer, M. Morawe, F. Arnold, E. Roger, M. Müller, T. Seufferlein, Frappart PO i A. Kleger. "PARP inhibitor resistance induces massive genome alterations responsible of the acquisition of multidrug resistance in DNA damage repair-deficient pancreatic cancer". W DGVS Digital: BEST OF DGVS. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1716148.
Pełny tekst źródłaDaly, G., C. M. Francis, V. M. Pastukh, D. Absher, R. J. Langley i M. N. Gillespie. "Rapid Redistribution of Oxidative Base Damage in DNA Regulatory Sequences Accompanies Transcriptional Modulation by Hypoxia in the Human Endothelial Cell Genome". W American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4191.
Pełny tekst źródłaRaporty organizacyjne na temat "Genome damage"
Dynan, William S. Links between persistent DNA damage, genome instability, and aging. Office of Scientific and Technical Information (OSTI), listopad 2016. http://dx.doi.org/10.2172/1332061.
Pełny tekst źródłaStewart, Robert D. Kinetic Modeling of Damage Repair, Genome Instability, and Neoplastic Transformation. Office of Scientific and Technical Information (OSTI), marzec 2007. http://dx.doi.org/10.2172/900981.
Pełny tekst źródłaDynan, William S. Final report- Links between persistent DNA damage, genome instability, and aging. Office of Scientific and Technical Information (OSTI), listopad 2016. http://dx.doi.org/10.2172/1333814.
Pełny tekst źródłaMordes, Daniel, Heather L. Ball, Mark Ehrhardt, Daniel Mordes i David Cortez. Maintenance of Genome Stability and Breast Cancer: Molecular Analysis of DNA Damage-Activated Kinases. Fort Belvoir, VA: Defense Technical Information Center, marzec 2008. http://dx.doi.org/10.21236/ada494969.
Pełny tekst źródłaBall, Heather L., Mark Ehrhardt, Daniel Mordes i David Cortez. Maintenance of Genome Stability and Breast Cancer: Molecular Analysis of DNA Damage-Activated Kinases. Fort Belvoir, VA: Defense Technical Information Center, marzec 2007. http://dx.doi.org/10.21236/ada470345.
Pełny tekst źródłaAbagyan, Ruben, i Jianghong An. Genome-Wide Identification and 3D Modeling of Proteins involved in DNA Damage Recognition and Repair (Final Report). Office of Scientific and Technical Information (OSTI), sierpień 2005. http://dx.doi.org/10.2172/893896.
Pełny tekst źródłaRuben A. Abagyan, PhD. Genome-Wide Identification and 3D Modeling of Proteins involved in DNA Damage Recognition and Repair (Final Report). Office of Scientific and Technical Information (OSTI), kwiecień 2004. http://dx.doi.org/10.2172/823103.
Pełny tekst źródłaGreen, Brian M. DNA Damage and Genomic Instability Induced by Inappropriate DNA Re-Replication. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2005. http://dx.doi.org/10.21236/ada436928.
Pełny tekst źródłaGreen, Brian M., i Joachim J. Li. DNA Damage and Genomic Instability Induced by Inappropriate DNA Re-replication. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2007. http://dx.doi.org/10.21236/ada467931.
Pełny tekst źródłaGreen, Brian. DNA Damage and Genomic Instability Induced by Inappropriate DNA Re-replication. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2006. http://dx.doi.org/10.21236/ada482750.
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