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Artykuły w czasopismach na temat "Genetic risk factor"
Koch, Linda. "Genetic T2DM risk factor found". Nature Reviews Endocrinology 10, nr 3 (14.01.2014): 128. http://dx.doi.org/10.1038/nrendo.2013.273.
Pełny tekst źródłaReitsma, Pieter H. "Genetic Risk Factors of Thrombosis." Blood 114, nr 22 (20.11.2009): SCI—43—SCI—43. http://dx.doi.org/10.1182/blood.v114.22.sci-43.sci-43.
Pełny tekst źródłaSokolova, I. V., D. A. Mustafina, A. G. Sadertdinova, A. N. Zagitova, D. A. Khusnullin, L. G. Sadertdinova i N. A. Mescheryakov. "GENETIC RISK FACTOR FOR IDIOPATHIC SCOLIOSIS". International Journal of Applied and Fundamental Research (Международный журнал прикладных и фундаментальных исследований), nr 1 2023 (2023): 25–29. http://dx.doi.org/10.17513/mjpfi.13501.
Pełny tekst źródłaOhishi, Mitsuru, Kenshi Fujii, Takazo Minamino, Jitsuo Higaki, Atsushi Kamitani, Hiromi Rakugi, Yi Zhao, Hiroshi Mikami, Tetsuro Miki i Toshio Ogihara. "A potent genetic risk factor for restenosis". Nature Genetics 5, nr 4 (grudzień 1993): 324–25. http://dx.doi.org/10.1038/ng1293-324.
Pełny tekst źródłaSenior, Kathryn. "New genetic risk factor for sporadic PD". Nature Reviews Neurology 5, nr 7 (lipiec 2009): 354. http://dx.doi.org/10.1038/nrneurol.2009.79.
Pełny tekst źródłaBertina, Rogier M. "Factor V Leiden and other coagulation factor mutations affecting thrombotic risk". Clinical Chemistry 43, nr 9 (1.09.1997): 1678–83. http://dx.doi.org/10.1093/clinchem/43.9.1678.
Pełny tekst źródłaCushman, Mary. "Inherited Risk Factors for Venous Thrombosis". Hematology 2005, nr 1 (1.01.2005): 452–57. http://dx.doi.org/10.1182/asheducation-2005.1.452.
Pełny tekst źródłaCheekurthy, Alice Jayapradha. "Predisposition of Obesity through Genetic and Non-Genetic Risk Factors". Journal of Endocrinology Research 2, nr 2 (6.02.2021): 27. http://dx.doi.org/10.30564/jer.v2i2.2767.
Pełny tekst źródłaSchwab, Manfred, Andreas Claas i Larissa Savelyeva. "BRCA2: a genetic risk factor for breast cancer". Cancer Letters 175, nr 1 (styczeń 2002): 1–8. http://dx.doi.org/10.1016/s0304-3835(01)00752-2.
Pełny tekst źródłaEVANS, JEFF. "Major Genetic Risk Factor Is Discovered for Parkinson's". Clinical Psychiatry News 38, nr 2 (luty 2010): 33. http://dx.doi.org/10.1016/s0270-6644(10)70097-7.
Pełny tekst źródłaRozprawy doktorskie na temat "Genetic risk factor"
Ruth, Katherine Sarah. "Identification of genetic and non-genetic factors contributing to female reproductive ageing". Thesis, University of Exeter, 2015. http://hdl.handle.net/10871/19189.
Pełny tekst źródłaChen, Hong [Verfasser]. "Plasminogen is a genetic risk factor of periodontitis / Hong Chen". Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2018. http://d-nb.info/1196803110/34.
Pełny tekst źródłaPerdigão, Catarina. "The impact of the genetic risk factor BIN1 to Alzheimer’s disease development". Doctoral thesis, Universidade Nova de Lisboa. Instituto de Tecnologia Quimica e Biológica António Xavier, 2021. http://hdl.handle.net/10362/132008.
Pełny tekst źródłaN/A
Caglayan, Safak [Verfasser]. "SORLA/SORL1 as genetic risk factor in Alzheimer disease / Safak Caglayan". Berlin : Freie Universität Berlin, 2013. http://d-nb.info/1043480935/34.
Pełny tekst źródłaNewsome, Jamie. "Resilience and Vulnerability in Adolescents at Risk for Delinquency: A Behavioral Genetic Study of Differential Response to Risk". University of Cincinnati / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1367937532.
Pełny tekst źródłaMutize, Tinashe. "DNA methylation : a risk factor for type 2 diabetes mellitus". Thesis, Cape Peninsula University of Technology, 2016. http://hdl.handle.net/20.500.11838/2388.
Pełny tekst źródłaThe early detection of individuals who are at risk of developing type 2 diabetes mellitus (T2DM) would decrease the morbidity and mortality associated with this disease. DNA methylation, the most widely studied epigenetic mechanism, offers unique opportunities in this regard. Aberrant DNA methylation is associated with disease pathogenesis and is observed during the asymptomatic stage of disease. DNA methylation has therefore attracted increasing attention as a potential biomarker for identifying individuals who have an increased risk of developing T2DM. The identification of high risk biomarkers for T2DM could facilitate risk stratification and lifestyle interventions, which could ultimately lead to better ways to prevent, manage and control the T2DM epidemic that is rampant worldwide. The aim of the study was to investigate global DNA methylation as a potential risk factor for T2DM by studying the association between the global DNA methylation levels and hyperglycaemic states. A cross-sectional, quantitative study design, involving 564 individuals of mixed ancestry descent, residing in Bellville South, South Africa was used. Participants were classified as normal, pre-diabetic (impaired fasting glucose (IFG) and/or impaired glucose tolerance (IGT)) or diabetic (screen detected diabetic and known diabetics) according to WHO criteria of 1998. DNA was extracted from whole blood using the salt extraction method. The percentage global DNA methylation was measured by an enzyme-linked immunosorbent assay (ELISA). The association between global DNA methylation and hyperglycaemia, as well as other biochemical markers of T2DM was tested in a robust linear regression analysis adjusted for age, gender and smoking.
Kirin, Mirna. "Genetic analysis of retinal traits". Thesis, University of Edinburgh, 2014. http://hdl.handle.net/1842/9619.
Pełny tekst źródłaMahlman, M. (Mari). "Genetic background and antenatal risk factors of bronchopulmonary dysplasia". Doctoral thesis, Oulun yliopisto, 2018. http://urn.fi/urn:isbn:9789526219530.
Pełny tekst źródłaTiivistelmä Ennenaikaisen syntymän ja keskoslasten hoidon kehittymisen myötä yhä useammat huomattavan epäkypsinä syntyneet lapset jäävät henkiin. Samalla erityisesti juuri näitä lapsia uhkaavien sairauksien esiintyvyys on pysynyt korkeana. Bronkopulmonaalinen dysplasia (BPD, keskosen krooninen keuhkosairaus) on yksi näistä sairauksista. BPD:n molekyylitasoinen tautimekanismi on vielä osin tuntematon, eikä BPD:tä tehokkaasti estävää tai siitä parantavaa hoitoa ole. Myös BPD riskin arvioiminen vastasyntyneen keskoslapsen kohdalla on vaikeaa. BPD on huomattavan perinnöllinen tauti. BPD:lle altistavista geeneistä on kuitenkin vasta vähän tietoa. Tämän tutkimuksen tavoitteena oli lisätä tietoa BPD:n tautimekanismista tutkimalla BPD:lle altistavia geenejä. Lisäksi tutkimuksessa tarkasteltiin BPD:n esiintyvyyttä ja syntymää edeltäviä riskitekijöitä erityisesti kaksosten osalta. Ehdokasgeenitutkimuksessa verisuonten endoteelikasvutekijää koodaava geeni ei assosioitunut toistuvasti BPD:hen. Kit ligandia koodaava geeni sen sijaan assosioitui. Koko genomin assosiaatiotutkimuksessa C-reaktiivista proteiinia (CRP) koodaavan geenin lähistöltä löydettiin BPD:hen mahdollisesti assosioituva alue. Lisäksi ensimmäisen viikon CRP-arvojen osoitettiin ennakoivan myöhemmin kehittyvää BPD:tä. BPD-riskin todettiin olevan matalampi kaksi- kuin yksisikiöisistä raskauksista syntyneillä lapsilla. Tutkimuksen tulokset lisäävät tietoa BPD:n perinnöllisyydestä ja sitä kautta BPD:n tautimekanismista. Tutkimus toi myös uutta tietoa BPD:n riskitekijöistä parantaen vastasyntyneen keskoslapsen BPD-riskin arviota
Bayoumy, Nervana M. K. "Genetic analysis of plasma von Willebrand factor antigen levels as a risk factor for arterial and venous thrombosis". Thesis, University of Aberdeen, 2006. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU223247.
Pełny tekst źródłaPolasek, Ozren. "Investigating the role of human genome-wide heterozygosity as a health risk factor". Thesis, University of Edinburgh, 2009. http://hdl.handle.net/1842/4799.
Pełny tekst źródłaKsiążki na temat "Genetic risk factor"
Petrakis, Peter L. Alcoholism, and inherited disease. Rockville, Md: U.S. Dept. of Health and Human Services, Public Health Service, Alcohol, Drug Abuse, and Mental Health Administration, National Institute on Alcohol Abuse and Alcoholism, 1985.
Znajdź pełny tekst źródłaYoung, Ian D. Introduction to risk calculation in genetic counselling. Oxford: Oxford University Press, 1991.
Znajdź pełny tekst źródłaJ, Alberts Mark, red. Genetics of cerebrovascular disease. Armonk, NY: Futura Pub. Co., 1999.
Znajdź pełny tekst źródłaUri, Goldbourt, De Faire Ulf i Berg Kåre, red. Genetic factors in coronary heart disease. Dordrecht: Kluwer Academic, 1994.
Znajdź pełny tekst źródłaOffit, Kenneth. Clinical cancer genetics: Risk counseling and management. New York: Wiley-Liss, 1998.
Znajdź pełny tekst źródłaKåre, Berg, red. Genetic approaches of coronary heart disease and hypertension. Berlin: Springer-Verlag, 1991.
Znajdź pełny tekst źródłaGarte, Seymour J. Genetic susceptibility to cancer. Boston: Kluwer Academic, 1998.
Znajdź pełny tekst źródłaSymposium on Phenotypic Variation in Populations: Relevance to Risk Assessment (1986 Brookhaven National Laboratory). Phenotypic variation in populations: Relevance to risk assessment. New York: Plenum Press, 1988.
Znajdź pełny tekst źródłaA, Levin Morris, i Strauss Harlee S, red. Risk assessment in genetic engineering. New York: McGraw-Hill, 1991.
Znajdź pełny tekst źródłaYoung, Ian D. Introduction to risk calculation in genetic counseling. Wyd. 2. Oxford [England]: Oxford University Press, 1999.
Znajdź pełny tekst źródłaCzęści książek na temat "Genetic risk factor"
Waldinger, Marcel D. "Risks Factors in Premature Ejaculation: The Genetic Risk Factor". W Premature Ejaculation, 111–23. Milano: Springer Milan, 2012. http://dx.doi.org/10.1007/978-88-470-2646-9_9.
Pełny tekst źródłaAstermark, Jan. "Genetic and Environmental Risk Factors for Factor VIII Inhibitor Development". W Textbook of Hemophilia, 48–52. Oxford, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118398258.ch6.
Pełny tekst źródłaBerg, Kåre. "Lp(a) Lipoprotein: A Monogenic Risk Factor for Cardiovascular Disease". W Genetic factors in coronary heart disease, 275–87. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1130-0_18.
Pełny tekst źródłaFriedlander, Yechiel. "Familial Clustering of Coronary Heart Disease: A Review of its Significance and Role as a Risk Factor for the Disease". W Genetic factors in coronary heart disease, 37–53. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1130-0_3.
Pełny tekst źródłaGonchar, Alexander, Maxim Ameliyanovich, Kristina Zhur, Irma Mosse i Konstantin Mosse. "Molecular-Genetic Analysis of Genetic Predisposition to Myocardial Infarction and Comparison of Risk Factor Population Rates in Different Countries". W Radiobiology and Environmental Security, 111–25. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-1939-2_11.
Pełny tekst źródłaPoirier, Judes. "Apolipoprotein E4: From synaptic remodeling to genetic risk factor in both familial and sporadic Alzheimer’s disease". W Alzheimer: 100 Years and Beyond, 289–94. Berlin, Heidelberg: Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/978-3-540-37652-1_37.
Pełny tekst źródłaZheng, Wei. "Genetic Polymorphisms in the Transforming Growth Factor-β Signaling Pathways and Breast Cancer Risk and Survival". W Methods in Molecular Biology, 265–77. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-492-0_11.
Pełny tekst źródłaThiriet, Marc. "Genetic Risk Factors". W Biomathematical and Biomechanical Modeling of the Circulatory and Ventilatory Systems, 595–676. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-89315-0_7.
Pełny tekst źródłaKronenberg, Florian. "Lipoprotein(a)". W Prevention and Treatment of Atherosclerosis, 201–32. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/164_2021_504.
Pełny tekst źródłaThimm, F., i G. Fleckenstein-Grün. "Potentiation of the Age-Dependent Ca Uptake into Coronary Arteries of Rats by the Risk Factor Genetic Hypertension". W Advances in Experimental Medicine and Biology, 489–93. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4613-0333-6_62.
Pełny tekst źródłaStreszczenia konferencji na temat "Genetic risk factor"
Kulkarni, S., X. Zhou, M. Nesline, C. Murekeyisoni, N. Watroba, M. Berry, W. Davis, C. Ambrosone i Y. Zheng. "Genetic Susceptibility as a Possible Risk Factor for Breast Cancer." W Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-6067.
Pełny tekst źródłaMOTSINGER, ALISON A., BRIAN S. DONAHUE, NANCY J. BROWN, DAN M. RODEN i MARYLYN D. RITCHIE. "RISK FACTOR INTERACTIONS AND GENETIC EFFECTS ASSOCIATED WITH POST-OPERATIVE ATRIAL FIBRILLATION". W Proceedings of the Pacific Symposium. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812701626_0054.
Pełny tekst źródłaDooper, Marten. "Nitric oxide synthase genetic variant is a risk factor for suicidal behaviour". W 35th ECNP Congress, redaktor Christina Dalla. Baarn, the Netherlands: Medicom Medical Publishers, 2022. http://dx.doi.org/10.55788/c63a9364.
Pełny tekst źródłaFerreira, Nancy, Darley Ferreira i Thais Ferreira. "GENETIC EVALUATION OF MICROCALCIFICATIONS AS A PROGNOSTIC FACTOR". W Abstracts from the Brazilian Breast Cancer Symposium - BBCS 2021. Mastology, 2021. http://dx.doi.org/10.29289/259453942021v31s2101.
Pełny tekst źródłaSurniyantoro, H. N. E., i N. R. Hidayati. "Preliminary study in genetic polymorphism of hOGG1 and risk factor for thyroid cancer in Indonesia". W PROCEEDINGS OF THE INTERNATIONAL CONFERENCE AND SCHOOL ON PHYSICS IN MEDICINE AND BIOSYSTEM (ICSPMB): Physics Contribution in Medicine and Biomedical Applications. AIP Publishing, 2021. http://dx.doi.org/10.1063/5.0047941.
Pełny tekst źródłaBatai, Ken, Ebony Shah i Rick A. Kittles. "Abstract B14: Population genetics analysis of prostate cancer GWAS SNPs to evaluate West African genetic ancestry as a risk factor". W Abstracts: Sixth AACR Conference: The Science of Cancer Health Disparities; December 6–9, 2013; Atlanta, GA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7755.disp13-b14.
Pełny tekst źródłaAllam, Ines, Aldjia Lamri, Sihem Oulacrouz, Mohamed Saidani i Reda Djidjik. "P85 The IRF5 (rs729302) polymorphism is a genetic risk factor for systemic lupus erythematosus in Algerian patients". W 12th European Lupus Meeting. Lupus Foundation of America, 2020. http://dx.doi.org/10.1136/lupus-2020-eurolupus.130.
Pełny tekst źródłaAnghel, Lucretia, Dumitru Ursu, Simona Mitincu Caramfil, Cristina Stefanescu, Stefana Maria Moisa, Anamaria Ciubara i Liliana Baroiu. "THE LINK BETWEEN LIPIDIC PROFILE, DEPRESSION AND CARDIOVASCULAR DISEASE". W The European Conference of Psychiatry and Mental Health "Galatia". Archiv Euromedica, 2023. http://dx.doi.org/10.35630/2022/12/psy.ro.17.
Pełny tekst źródłaSmyk, W., R. Hall, S. Weber, F. Grünhage, F. Lammert i M. Krawczyk. "Common genetic variant c.711A>T in the hepatobiliary phospholipid translocator ABCB4 as risk factor for liver fibrosis". W 36. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0039-3402121.
Pełny tekst źródłaFinlen Copeland, Catherine, Francine L. Kelly, Laurie D. Snyder i Scott M. Palmer. "Clinical And Genetic Predictors Of Pulmonary Aspergillus Infection, A Major Risk Factor For Bronchiolitis Obliterans Syndrome After Lung Transplantation". W American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2570.
Pełny tekst źródłaRaporty organizacyjne na temat "Genetic risk factor"
Gelmann, Edward P. Genetic Risk Factor for Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, styczeń 2005. http://dx.doi.org/10.21236/ada434784.
Pełny tekst źródłaGelmann, Edward P. Genetic Risk Factor for Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, styczeń 2003. http://dx.doi.org/10.21236/ada414867.
Pełny tekst źródłaCao, Xianling, Xuanyou Zhou, Naixin Xu, Songchang Chang i Chenming Xu. Association of IL-4 and IL-10 Polymorphisms with Preterm Birth Susceptibility: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, kwiecień 2022. http://dx.doi.org/10.37766/inplasy2022.4.0044.
Pełny tekst źródłaGelmann, Edward P. Genetics Risk Factor for Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, styczeń 2004. http://dx.doi.org/10.21236/ada422932.
Pełny tekst źródłaSarma, Aruna. Genetic and Hormonal Risk Factors for Cancer in African American Men. Fort Belvoir, VA: Defense Technical Information Center, maj 2006. http://dx.doi.org/10.21236/ada455088.
Pełny tekst źródłaMack, Thomas M. Genetic Abnormalities in Breast Cancer Tumors and Relationships to Environmental and Genetic Risk Factors Using Twins. Fort Belvoir, VA: Defense Technical Information Center, październik 1995. http://dx.doi.org/10.21236/ada303152.
Pełny tekst źródłaMack, Thomas M. Genetic Abnormalities in Breast Cancer Tumors and Relationships to environmental and Genetic Risk Factors Using Twins. Fort Belvoir, VA: Defense Technical Information Center, październik 1999. http://dx.doi.org/10.21236/ada393066.
Pełny tekst źródłaSarma, Aruna V. Genetic and Hormonal Risk Factors for Prostate Cancer in African American Men. Fort Belvoir, VA: Defense Technical Information Center, maj 2005. http://dx.doi.org/10.21236/ada442683.
Pełny tekst źródłaEshed-Williams, Leor, i Daniel Zilberman. Genetic and cellular networks regulating cell fate at the shoot apical meristem. United States Department of Agriculture, styczeń 2014. http://dx.doi.org/10.32747/2014.7699862.bard.
Pełny tekst źródłaChejanovsky, Nor, Diana Cox-Foster, Victoria Soroker i Ron Ophir. Honeybee modulation of infection with the Israeli acute paralysis virus, in asymptomatic, acutely infected and CCD colonies. United States Department of Agriculture, grudzień 2013. http://dx.doi.org/10.32747/2013.7594392.bard.
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