Gotowe bibliografie tematyczne / Gene family

Gotowa bibliografia na temat „Gene family”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 20 lutego 2023

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Artykuły w czasopismach na temat "Gene family"

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Alam, S. M. Khorshed, Rupasri Ain, Toshihiro Konno, Jennifer K. Ho-Chen i Michael J. Soares. "The rat prolactin gene family locus: species-specific gene family expansion". Mammalian Genome 17, nr 8 (sierpień 2006): 858–77. http://dx.doi.org/10.1007/s00335-006-0010-1.

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Ehsani, Sepehr, Hairu Huo, Ashkan Salehzadeh, Cosmin L. Pocanschi, Joel C. Watts, Holger Wille, David Westaway, Ekaterina Rogaeva, Peter H. St. George-Hyslop i Gerold Schmitt-Ulms. "Family reunion – The ZIP/prion gene family". Progress in Neurobiology 93, nr 3 (marzec 2011): 405–20. http://dx.doi.org/10.1016/j.pneurobio.2010.12.001.

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Töpel, Mats, i Paul Jarvis. "The Tic20 gene family". Plant Signaling & Behavior 6, nr 7 (lipiec 2011): 1046–48. http://dx.doi.org/10.4161/psb.6.7.15631.

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Kaelin, William G. "The p53 gene family". Oncogene 18, nr 53 (grudzień 1999): 7701–5. http://dx.doi.org/10.1038/sj.onc.1202955.

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Wong, Howard, i Michael C. Schotz. "The lipase gene family". Journal of Lipid Research 43, nr 7 (lipiec 2002): 993–99. http://dx.doi.org/10.1194/jlr.r200007-jlr200.

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Mendoza, Michael, Garni Mandani i Jamil Momand. "The MDM2 gene family". BioMolecular Concepts 5, nr 1 (1.03.2014): 9–19. http://dx.doi.org/10.1515/bmc-2013-0027.

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AbstractMDM2 is an oncoprotein that blocks p53 tumor suppressor-mediated transcriptional transactivation, escorts p53 from the cell nucleus to the cytoplasm, and polyubiquitylates p53. Polyubiquitylated p53 is rapidly degraded in the cytoplasm by the 26S proteasome. MDM2 is abnormally upregulated in several types of cancers, especially those of mesenchymal origin. MDM4 is a homolog of MDM2 that also inhibits p53 by blocking p53-mediated transactivation. MDM4 is required for MDM2-mediated polyubiquitylated of p53 and is abnormally upregulated in several cancer types. MDM2 and MDM4 genes have been detected in all vertebrates to date and only a single gene homolog, named MDM, has been detected in some invertebrates. MDM2, MDM4, and MDM have similar gene structures, suggesting that MDM2 and MDM4 arose through a duplication event more than 440 million years ago. All members of this small MDM2 gene family contain a single really interesting new gene (RING) domain (with the possible exception of lancelet MDM) which places them in the RING-domain superfamily. Similar to MDM2, the vast majority of proteins with RING domains are E3 ubiquitin ligases. Other RING domain E3 ubiquitin ligases that target p53 are COP1, Pirh2, and MSL2. In this report, we present evidence that COP1, Pirh2, and MSL2 evolved independently of MDM2 and MDM4. We also show, through structure homology models of invertebrate MDM RING domains, that MDM2 is more evolutionarily conserved than MDM4.
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Liberles, David A., i Katharina Dittmar. "Characterizing gene family evolution". Biological Procedures Online 10, nr 1 (grudzień 2008): 66–73. http://dx.doi.org/10.1251/bpo144.

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Parenti, Giancarlo, Germana Meroni i Andrea Ballabio. "The sulfatase gene family". Current Opinion in Genetics & Development 7, nr 3 (czerwiec 1997): 386–91. http://dx.doi.org/10.1016/s0959-437x(97)80153-0.

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Bahram, S., i T. Spies. "The MIC gene family". Human Immunology 47, nr 1-2 (kwiecień 1996): 64. http://dx.doi.org/10.1016/0198-8859(96)85038-5.

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Macleod, Kay, Dominique Leprince i Dominique Stehelin. "The ets gene family". Trends in Biochemical Sciences 17, nr 7 (lipiec 1992): 251–56. http://dx.doi.org/10.1016/0968-0004(92)90404-w.

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Rozprawy doktorskie na temat "Gene family"

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Newton, Craig Hunter. "A Drosophila tRNA gene family". Thesis, University of British Columbia, 1989. http://hdl.handle.net/2429/29252.

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This thesis describes a tRNA[sup Arg] gene family in the fruit fly D. melanogaster. The study was initiated in order to better understand the gene organization of a subset of this family. Of a total of 10 tRNA[sup Arg] gene copies that comprise this family, four genes are arranged tandemly on repeated sequences 200 bp and 600 bp in length. This organization suggests these four genes have undergone recent gene duplication. To investigate the significance of such events, these tRNA[sup Arg] genes were compared to other members of this gene family in regards their structure, in vitro function, and organization between different D. melanogaster strains and sibling species. The results show that the four repeated genes differ in sequence at a single nucleotide (CI3) relative to six additional gene copies. Five of these additional genes are identical in sequence and one differs at two nucleotides (A16, A37). The gene family is organized at four different chromosomal sites. Six of the 10 gene copies occur at polytene region 12E1-2 on the X chromosome. These include the four repeated genes (R12.1-R12.4) and two additional gene copies (R12.5-R12.6). A single gene occurs at another X-linked locus located at 19F (R19.1). The three remaining gene copies occur on chromosome 3R as a gene doublet at 85C (R85.1-R85.2) and as a single gene at 83AB (R83.1). All 10 genes in this family are active as templates for in vitro transcription in. homologous Drosophila extracts. The predicted 5' initiation sites are all very similar and occur at conserved nucleotides 4-5 bp upstream from the mature 5' ends of the genes. Six of the ten gene copies are transcribed efficiently in vitro. The four repeated gene copies however, have novel transcription properties; they are much less efficient templates in Drosophila extracts (2-5 fold) and are inhibited at KCl concentrations where other gene copies transcribe optimally. These properties do not result from the single nucleotide difference in coding sequence or an inability to form stable pre-initiation complexes. Instead they appear to result from the upstream 5' flanking sequence. These novel transcription properties are not observed in heterologous transcription systems containing human cell extracts. Instead they are transcribed efficiently relative to other members of the gene family and no longer exhibit sensitivity to KCl. Comparison of the repeated tRNA[sup Arg] gene locus between several D. melanogaster strains indicates that this is the predominant form in the majority of wild and laboratory stocks. However, a fraction of populations (5/45) contain a variant locus that consists of only three gene copies. These have apparently lost, or not gained, one of the repeats found in the majority of populations. Similar comparisons between D. melanogaster sibling species (D. simualns, D. teissieri, D. erecta, and D. yakuba) show that only the D. melanogaster lines contain the repeated genes. Thus in the time since the divergence from its closest related sibling species (D. simulans ), D . melanogaster lines have acquired three new gene copies by duplication of a putative ancestral single gene. Analysis of the four, three, and single gene loci isolated from D. melanogaster (pDt27R, p27ry2) and D. simulans (p27simC), respectively, at the nucleotide level suggests a model to account for the evolution of this gene cluster. One additional sequence associated with this gene family consists of a half tRNA[sup Arg] gene composed of the 3' 37 bp. This half gene contains a 3' CCA sequence and is flanked on one side by a region that is > 90% homologous to the LTR of the Drosophila retrotransposon mdg 1. It is not clear if this half gene is itself part of a related retrotransposon or has originated by recombination or aberrant reverse transcription with mdg 1. The 3' ends tRNA[sup Arg] have been proposed to function as primers for at least two classes of retrotransposon, mdg 1 and 412 (Yuki et al., 1986).
Medicine, Faculty of
Biochemistry and Molecular Biology, Department of
Graduate
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Cadieux, Benoît. "The zebrafish progranulin gene family /". Thesis, McGill University, 2003. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=84486.

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Granulins are small cysteine-rich peptide growth factors that, in mammals, are derived from a common glycoprotein precursor (progranulin) containing one half and seven non-identical tandemly repeated granulin domains. While there is evidence for only one progranulin gene in mammalian genomes, work presented in this thesis demonstrates that granulins form an extended gene family in teleost fish. Two zebrafish genes that constitute likely co-orthologues to mammalian progranulin, progranulin-a and progranulin-b, encode precursors that encode 10 and 9 copies of the granulin motif, respectively. Two additional genes in zebrafish, designated progranulin-1 and progranulin-2, each give rise to precursors consisting of one and one-half granulin-like repeats only. The progranulin-1 and progranulin-2 genes are organized in tandem, and possess exonic complementarity to a single non-coding RNA gene transcribed in the antisense orientation from the complementary DNA strand. A cDNA encoding a chimeric precursor consisting of the amino-terminal progranulin-1 followed by the carboxyl-terminal region of progranulin-2 was characterized and is likely generated through the mechanism of splicing in trans between the two primary transcripts for progranulin-1 and progranulin-2. Chromosomal assignment of the zebrafish progranulin genes indicates that progranulin-a, but not progranulin-b, is located on a chromosome that displays syntenic correspondence to mammalian progranulin. Cumulatively, the data suggest that an ancestral progranulin gene was duplicated at the base of the vertebrate radiation to generate precursors with distinct architectures and that one was lost in the lineage leading to tetrapods after the split between sarcopterygians and actinopterygians. Like their mammalian counterpart, the expression of the zebrafish progranulins is widespread in adult tissues. During development, the maternal expression of progranulin-a and progranulin-b parallels that
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Janousek, Vaclav, Robert Karn i Christina Laukaitis. "The role of retrotransposons in gene family expansions: insights from the mouse Abp gene family". BioMed Central, 2013. http://hdl.handle.net/10150/610385.

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BACKGROUND:Retrotransposons have been suggested to provide a substrate for non-allelic homologous recombination (NAHR) and thereby promote gene family expansion. Their precise role, however, is controversial. Here we ask whether retrotransposons contributed to the recent expansions of the Androgen-binding protein (Abp) gene families that occurred independently in the mouse and rat genomes.RESULTS:Using dot plot analysis, we found that the most recent duplication in the Abp region of the mouse genome is flanked by L1Md_T elements. Analysis of the sequence of these elements revealed breakpoints that are the relicts of the recombination that caused the duplication, confirming that the duplication arose as a result of NAHR using L1 elements as substrates. L1 and ERVII retrotransposons are considerably denser in the Abp regions than in one Mb flanking regions, while other repeat types are depleted in the Abp regions compared to flanking regions. L1 retrotransposons preferentially accumulated in the Abp gene regions after lineage separation and roughly followed the pattern of Abp gene expansion. By contrast, the proportion of shared vs. lineage-specific ERVII repeats in the Abp region resembles the rest of the genome.CONCLUSIONS:We confirmed the role of L1 repeats in Abp gene duplication with the identification of recombinant L1Md_T elements at the edges of the most recent mouse Abp gene duplication. High densities of L1 and ERVII repeats were found in the Abp gene region with abrupt transitions at the region boundaries, suggesting that their higher densities are tightly associated with Abp gene duplication. We observed that the major accumulation of L1 elements occurred after the split of the mouse and rat lineages and that there is a striking overlap between the timing of L1 accumulation and expansion of the Abp gene family in the mouse genome. Establishing a link between the accumulation of L1 elements and the expansion of the Abp gene family and identification of an NAHR-related breakpoint in the most recent duplication are the main contributions of our study.
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Thangavelu, Madan. "The actin gene family of tobacco". Thesis, University of Cambridge, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335212.

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Cotton, James A. "Vertebrate phylogenomics and gene family evolution". Thesis, University of Glasgow, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.274764.

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Chattaway, John Antony. "Characterising the polymorphic BG gene family". Thesis, University of Cambridge, 2014. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.707953.

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Dubb, Lindsey. "A likelihood model of gene family evolution /". Thesis, Connect to this title online; UW restricted, 2005. http://hdl.handle.net/1773/10264.

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Dörr, Daniel [Verfasser]. "Gene family-free genome comparison / Daniel Dörr". Bielefeld : Universitätsbibliothek Bielefeld, 2016. http://d-nb.info/1096457229/34.

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Rawson, Stephen J. "The Chinese hamster phosphoglycerate kinase gene family". Thesis, University of Leicester, 1986. http://hdl.handle.net/2381/35136.

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I. A phosphoglycerate kinase (PGK) deficient variant cell line, R1.1.7, derived from the Chinese hamster ovary cell line, CHO-K1, is used as a recipient in the development of a DNA-mediated transfection system designed for the study of in vivo expression of exogenous PGK gene sequences. II. Several PGK DNA sequences are detected in the Chinese hamster genome by hybridisation of genomic DNA digests with a human PGK cDNA probe. A number of these sequences are shown to be X-linked. Four of the PGK sequences observed in the blot hybridisations are isolated from a CHO-K1 DNA genomic library and analysed by probing with different regions of the PGK cDNA, heteroduplex mapping and DNA sequencing. One of these sequences is a 572 bp exon from the functional X-linked PGK gene (PGK-1) which is expressed in all somatic cells. The three remaining PGK sequences are intronless pseudogenes which were apparently derived independently, from an ancestral Chinese hamster PGK gene, within the last 50 million years. They exhibit features typical of 'processed' pseudogenes which are generated via an mRNA intermediate and integrated into breaks in the chromosomal DNA.
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Drummond, Revel Scott MacGregor. "The AtMRS2 gene family from Arabidopsis thaliana". Thesis, University of Auckland, 2004. http://hdl.handle.net/2292/15.

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Magnesium (Mg2+) is an essential mineral nutrient for plants and is the most abundant free divalent cation in plant cells. However, our knowledge of the role of this ion in the plant cell is limited, and the mechanisms of homeostasis and transport of the ion are almost completely unknown. A. Tutone (this laboratory) identified an Arabidopsis thaliana gene by the complementation of a Mg2+-uptake yeast mutant (CM66). This gene, referred to as AtMRS2-11, was expressed as cDNA from a strong yeast promoter and allowed the growth of the CM66 yeast strain on standard media. The conceptually translated AtMRS2-11 protein sequence was used in this study to identify nine additional proteins by sequence homology searches using the BLAST algorithm. The corresponding genes have been cloned from cDNA (A. thaliana ecotype Landsberg erecta) and sequenced. Protein sequence similarity suggests that the family forms a sub-section of the CorA super-family of Mg2+ transport proteins. The mutant yeast used to identify the family initially was also used to show that two family members in addition to AtMRS2-11 were able to complement the Mg2+-dependent growth phenotype. When fused to eGFP, these proteins showed a localisation consistent with some of the protein reaching the yeast cell membrane. The other members of the family were also fused to eGFP and showed a range of localisation patterns within the yeast cell. None of the three AtMRS2 proteins previously able to complement the yeast mutant phenotype did so when fused to eGFP. RNA transcripts from the AtMRS2 family were detected by RT-PCR in organ-scale preparations of total RNA from A. thaliana. Most family members were detected in all of the organs tested. Northern analysis of AtMRS2-11 RNA transcript level showed that the gene was more highly expressed in leaf tissue, but was not affected by decreased levels of Mg2+ in the growth media. The levels of steady state AtMRS2-11 mRNA transcript were elevated two-fold in the light during the diurnal cycle, but no change was detected during light-induced greening of etiolated seedlings. A stable transgenic A. thaliana line expressing the gusA gene from the promoter region of AtMRS2-11 was used to localise the promoter's activity to cells containing chloroplasts. The expression appeared highest in younger cells. VI The AtMRS2-11 protein was predicted to contain a chloroplast targeting peptide. Western analysis demonstrated that AtMRS2-11 was enriched in the total proteins of isolated chloroplasts as compared to extracts from whole plants. The AtMRS2-11:eGFP fusion protein was also detected in chloroplasts by fluorescence microscopy. Flame atomic absorption spectroscopy was used in conjunction with isolated chloroplasts to try to determine the effects of the overaccumulation of the AtMRS2-11 protein in a transgenic A. thaliana plant line (constructed by A. Tutone). A rapid uptake or binding of Mg2+ was seen in chloroplasts isolated from both wild type and transgenic lines, but no differences were observed in either the rate of Mg2+ uptake/binding or the final Mg2+ content.
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Książki na temat "Gene family"

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Paul, Tucker Richard, i Lawler Jack, red. Thrombospondin gene family. New York: Springer, 1995.

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Adams, Josephine C. The thrombospondin gene family. Austin, Tex., U.S.A: R.G. Landes Co., 1995.

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Lackey, Karen E., red. Gene Family Targeted Molecular Design. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2008. http://dx.doi.org/10.1002/9780470423936.

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Hay, Deborah L., i Ian M. Dickerson, red. The calcitonin gene-related peptide family. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-2909-6.

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Lefebvre, Lise. Généalogie de Gene Custer (Kosar). Saint-Jean-sur-Richelieu, QC: Édition Lise Lefebvre, 2012.

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Karchaske, S. Janelle. Genealogical record of Gene Edward Vaughan. Charlotte, NC (2805 Shopton Rd., Charlotte 28217): Family History Researchers & Publishers, 1996.

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Waters, Gene Taylor. Gene Taylor & Patsy S. (Robb) Waters family. Kansas City, Mo. (5329 N. Walrond Ave, Kansas City 64119): G. and P. Waters, 2004.

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Tregear, James W. The lectin gene family of "Ricinus communis". [s.l.]: typescript, 1989.

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Potts, D. M. Queen Victoria's gene: Haemophilia and the royal family. Stroud: Sutton Pub., 1999.

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International Conference on the CEA Gene Family (1988 Sapporo-shi, Japan). The carcinoembryonic antigen gene family: Proceedings of the International Conference on the CEA Gene Family, Sapporo, 15 October, 1988. Amsterdam: Elsevier, 1989.

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Części książek na temat "Gene family"

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Beauchemin, Nicole. "CEA Gene Family". W Encyclopedia of Cancer, 1–5. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27841-9_969-3.

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Mehlhorn, Heinz. "Var Gene Family". W Encyclopedia of Parasitology, 3007. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-43978-4_4364.

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Mehlhorn, Heinz. "Var Gene Family". W Encyclopedia of Parasitology, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27769-6_4364-1.

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Beauchemin, Nicole. "CEA Gene Family". W Encyclopedia of Cancer, 870–74. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-46875-3_969.

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Rodríguez, Irma. "Combs, Gene". W Encyclopedia of Couple and Family Therapy, 524–25. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-49425-8_887.

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Rodríguez, Irma. "Combs, Gene". W Encyclopedia of Couple and Family Therapy, 1–2. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-15877-8_887-1.

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Schweickart, Vicki L., Carol J. Raport, David Chantry i Patrick W. Gray. "The Chemokine Gene Family". W Chemokines in Disease, 3–18. Totowa, NJ: Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-706-2_1.

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Gupta, G. S. "Regenerating (Reg) Gene Family". W Animal Lectins: Form, Function and Clinical Applications, 847–80. Vienna: Springer Vienna, 2012. http://dx.doi.org/10.1007/978-3-7091-1065-2_39.

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Marshall, Christopher J. "The ras Gene Family". W Oncogenes and Growth Control, 192–99. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-73325-3_26.

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Wilson, J. E. "The Hexokinase Gene Family". W Frontiers in Diabetes, 18–30. Basel: KARGER, 2004. http://dx.doi.org/10.1159/000079004.

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Streszczenia konferencji na temat "Gene family"

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Lim, H. A., i I. N. Shindyalov. "HOMOLOGOUS GENE FAMILY DATABASE COMPILATION". W Proceedings of the 2nd International Conference. WORLD SCIENTIFIC, 1993. http://dx.doi.org/10.1142/9789814503655_0032.

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"CYP75 gene family in barley". W SYSTEMS BIOLOGY AND BIOINFORMATICS. Institute of Cytology and Genetics, Siberian Branch of the Russian Academy of Sciences, 2019. http://dx.doi.org/10.18699/sbb-2019-42.

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TieFang Wang, BaoSheng Ye, YunWen Li i Yi Yang. "Family gene based Cloud Trust model". W 2010 International Conference on Educational and Network Technology (ICENT 2010). IEEE, 2010. http://dx.doi.org/10.1109/icent.2010.5532096.

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Cool, D. E., i R. T. A. MacGillivray. "CHARACTERIZATION OF THe HUMAN FACTOR XII GENE". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642800.

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Surface activation of the plasma systems involved with coagulation, fibrinolysis, renin formation and kinin generation involves factor XII (Hageman factor). This protein is a 76,000 dalton glycoprotein which circulates in plasma as an inactive form of a serine protease. A human liver cDNA coding for factor XII was used to screen a human genomic phage library. Two overlapping clones were isolated, XHXII27 and XHXII76, and contain the entire gene for human factor XII. The gene is 13.5 Kbp in length and consists of 14 exons and 13 introns. The transcriptional start site of the mRNA was determined using S1 mapping and primer extension analysis. The results indicate that the 5′ untranslated end of the mRNA has a leader sequence of 47 bp and is not interrupted by an intron in the gene. DNA sequence analysis of the region upstream of the transcriptional start site does not contain TATA or CAAT sequences, which are often found in other genes transcribed by RNA polymerase II. The positions of the introns in the coding sequence separate the protein into domains which are homologous to similar regions found in fibronectin and tissue-type plasminogen activator. Furthermore, wherever protein homologies are found, the positions of the introns in the triplet codon occur in the same reading frame as in the tissue-type plasminogen activator, urokinase plasminogen activator and factor XII genes. The intron/exon organization of the factor XII gene is different to the organization of other coagulation genes such as prothrombin, factor IX and factor X. Therefore, factor XII appears to have evolved as a member of the plasminogen activator family of genes rather than as a member of the clotting factor gene family.
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Souto, Emília Correia, Carolina Maria Marin, Gustavo Carvalho Costa, Igor Braga Farias, Bruno de Mattos Lombardi Badia, Icaro França Navarro Pinto, Roberta Ismael Lacerda Machado, Paulo Victor Sgobbi de Souza, Wladimir Bocca Vieira de Rezende Pinto i Acary Souza Bulle Oliveira. "Family with atypical Parkinsonism due to CHCHD10 gene mutation". W XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.502.

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Introduction: Parkinson’s disease - PD is the second most common agerelated neurodegenerative disorder. Characterized by a variety of motor and non-motor symptoms that relate to the loss of dopaminergic neurons in the midbrain black substance. Although most cases of PD are sporadic, 5–10% of patients have monogenetic mutations with a description of more than 20 genes for the familial form. Mitochondrial mutation in CHCHD10 has also been reported to be associated with a wide spectrum of neurodegenerative disorders, including PD. Objectives: Description of a rare recently described genetic cause of autosomal dominant parkinsonism. Methodology: Describe the case of a Brazilian woman with atypical parkinsonism due to CHCHD10 pathogenic variant that was followed up in our service. Result: Female, 64 years old. “. He started episodes of imbalance about 5 years ago, with falls, in addition to limb stiffness, worse on the left. 4 years ago, he started myalgia to great efforts with low subsequent tolerance to light effort. 1 year ago with urinary incontinence and choking past of poor performance in physical activities without pre-motor symptoms FAMILY: mother with clinical picture of possible dementia syndrome at age 60, history in the maternal family of myalgia, intolerance to physical exercise and hearing loss in adulthood. EXOMA: presence of variant c.146C > T (p.Ala49Val) in simple heterozygosity without CHCHD10 gene. MRI with thigh muscle hypotrophy in anterior and posterior thigh compartments; slight muscle edema in the legs. Conclusion: Pathogenic variants in the CHCHD10 gene should be considered in cases of atypical parkinsonism, especially in cases of positive familial history of mitochondrial myopathy or dementia.
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Meger, Y. "Peroxidase gene family in Prunus persica (L.)". W Bioinformatics of Genome Regulation and Structure/Systems Biology (BGRS/SB-2022) :. Institute of Cytology and Genetics, the Siberian Branch of the Russian Academy of Sciences, 2022. http://dx.doi.org/10.18699/bgrs/sb-2022-020.

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"Peroxidase gene family in Prunus persica (L.)". W Bioinformatics of Genome Regulation and Structure/Systems Biology (BGRS/SB-2022) :. Institute of Cytology and Genetics, the Siberian Branch of the Russian Academy of Sciences, 2022. http://dx.doi.org/10.18699/sbb-2022-020.

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Volinia, S., P. Patracchini, F. Vannini, L. Felloni, F. Panicucci i F. Beranardi. "HAGEMAN TRAIT INVESTIGATED BY FACTOR XII cDNA PROBES". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643299.

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The presence of gene lesions and of restriction fragment length polymorphisms (RFLPs) has been investigated by means of cDNA probes for the coagulation factor XII (FXII).A TaqI additional fragment (2.1Kb) has been found in two brothers with Hageman trait and in 11 members of their paternal lineage. Digestions with different enzymes exclude that FXII gene deletion is responsible for Hageman trait in this family. A point mutation originating an additional TaqI site is likely.The abnormal pattern (not present in 40 normal subjects) is correlated with a reduced FXII activity and identifies the heterozygous subjects in the paternal lineage. The presence of two different gene lesions causing Hageman trait in this family can be inferred.The TaqI additional site has been mapped within the 5 portion of the gene.Data suggest the presence of one FXII gene per aploid genome and disagree with previous localization of FXII gene on chromosome 6.Work supported by P.F. Ing. Gen. e Basi Mol. Mai. Ered. contratto CNR N.8400877
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Ananth, Alaka, i K. Chandra Sekaran. "Service optimization in cloud using family gene technology". W 2014 International Conference on Advances in Computing, Communications and Informatics (ICACCI). IEEE, 2014. http://dx.doi.org/10.1109/icacci.2014.6968491.

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Knibbe, Carole. "What Happened to My Genes? Insights on Gene Family Dynamics from Digital Genetics Experiments". W Artificial Life 14: International Conference on the Synthesis and Simulation of Living Systems. The MIT Press, 2014. http://dx.doi.org/10.7551/978-0-262-32621-6-ch006.

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Raporty organizacyjne na temat "Gene family"

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Li, Luyuan. Targeting Human Rhomboid Family-1 Gene RHBDF 1 in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, październik 2010. http://dx.doi.org/10.21236/ada562127.

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Nordeen, Steven K. HOXC Family Gene Expression in Prostate Cancer: A Mechanism Contribution to Androgen Independence. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2005. http://dx.doi.org/10.21236/ada447559.

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Latchman, David S. Regulation of BRCA-1 Gene Expression and Mammary Tumorigenesis by the Brn-3B POU Family Transcription Factor. Fort Belvoir, VA: Defense Technical Information Center, grudzień 2001. http://dx.doi.org/10.21236/ada403379.

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Latchman, David S. Regulation of BRCA-1 Gene Expression and Mammary Tumorigenesis by the Brn-3b POU Family Transcription Factor. Fort Belvoir, VA: Defense Technical Information Center, grudzień 2002. http://dx.doi.org/10.21236/ada413037.

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Sessa, Guido, i Gregory Martin. Role of GRAS Transcription Factors in Tomato Disease Resistance and Basal Defense. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7696520.bard.

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The research problem: Bacterial spot and bacterial speck diseases of tomato are causedby strains of Xanthomonas campestris pv. vesicatoria (Xcv) and Pseudomonas syringae pv.tomato (Pst), respectively. These bacteria colonize aerial parts of the plant and causesignificant losses in tomato production worldwide. Protection against Xcv and Pst bycultural practices or chemical control has been unsuccessful and there are only limitedsources of genetic resistance to these pathogens. In previous research supported in part byBARD IS-3237-01, we extensively characterized changes in tomato gene expression uponthe onset of spot and speck disease resistance. A remarkable finding of these studies wasthe inducibility in tomato leaves by both Xcv and Pst strains of genes encodingtranscriptional activator of the GRAS family, which has not been previously linked todisease resistance. Goals: Central goals of this research were to investigate the role of GRAS genes in tomatoinnate immunity and to assess their potential use for disease control.Specific objectives were to: 1. Identify GRAS genes that are induced in tomato during thedefense response and analyze their role in disease resistance by loss-of-function experiments.2. Overexpress GRAS genes in tomato and characterize plants for possible broad-spectrumresistance. 3. Identify genes whose transcription is regulated by GRAS family. Our main achievements during this research program are in three major areas:1. Identification of tomato GRAS family members induced in defense responses andanalysis of their role in disease resistance. Genes encoding tomato GRAS family memberswere retrieved from databases and analyzed for their inducibility by Pst avirulent bacteria.Real-time RT-PCR analysis revealed that six SlGRAS transcripts are induced during theonset of disease resistance to Pst. Further expression analysis of two selected GRAS genesshowed that they accumulate in tomato plants in response to different avirulent bacteria orto the fungal elicitor EIX. In addition, eight SlGRAS genes, including the Pst-induciblefamily members, were induced by mechanical stress in part in a jasmonic acid-dependentmanner. Remarkably, SlGRAS6 gene was found to be required for tomato resistance to Pstin virus-induced gene silencing (VIGS) experiments.2. Molecular analysis of pathogen-induced GRAS transcriptional activators. In aheterologous yeast system, Pst-inducible GRAS genes were shown to have the ability toactivate transcription in agreement with their putative function of transcription factors. Inaddition, deletion analysis demonstrated that short sequences at the amino-terminus ofSlGRAS2, SlGRAS4 and SlGRAS6 are sufficient for transcriptional activation. Finally,defense-related SlGRAS proteins were found to localize to the cell nucleus. 3. Disease resistance and expression profiles of transgenic plants overexpressing SlGRASgenes. Transgenic plants overexpressing SlGRAS3 or SlGRAS6 were generated. Diseasesusceptibility tests revealed that these plants are not more resistant to Pst than wild-typeplants. Gene expression profiles of the overexpressing plants identified putative direct orindirect target genes regulated by SlGRAS3 and SlGRAS6. Scientific and agricultural significance: Our research activities established a novel linkbetween the GRAS family of transcription factors, plant disease resistance and mechanicalstress response. SlGRAS6 was found to be required for disease resistance to Pstsuggesting that this and possibly other GRAS family members are involved in thetranscriptional reprogramming that takes place during the onset of disease resistance.Their nuclear localization and transcriptional activation ability support their proposed roleas transcription factors or co-activators. However, the potential of utilizing GRAS familymembers for the improvement of plant disease resistance in agriculture has yet to bedemonstrated.
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Raghothama, Kashchandra G., Avner Silber i Avraham Levy. Biotechnology approaches to enhance phosphorus acquisition of tomato plants. United States Department of Agriculture, styczeń 2006. http://dx.doi.org/10.32747/2006.7586546.bard.

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Abstract: Phosphorus is one of the least available macronutrient in the soil. The high affinity phosphate transporters are known to be associated with phosphate acquisition under natural conditions. Due to unique interactions of phosphate with soil particles, up to 80% of the applied phosphates may be fixed forcing the farmers to apply 4 to 5 times the fertilizers necessary for crop production. Efficient uptake and utilization of this essential nutrient is essential for sustainability and profitability of agriculture. Many predictions point to utilization/exhaustion of high quality phosphate rocks within this century. This calls for efforts to improve the ability of plants to acquire and utilize limiting sources of phosphate in the rhizosphere. Two important molecular and biochemical components associated with phosphate efficiency are phosphate transporters and phosphatases. This research project is aimed at defining molecular determinants of phosphate acquisition and utilization in addition to generating phosphate uptake efficient plants. The main objectives of the project were; Creation and analysis of transgenic tomato plants over-expressing phosphatases and transporters Characterization of the recently identified members (LePT3 and LePT4) of the Pi transporter family Generate molecular tools to study genetic responses of plants to Pi deficiency During the project period we have successfully identified and characterized a novel phosphate transporter associated with mycorrhizal symbiosis. The expression of this transporter increases with mycorrhizal symbiosis. A thorough characterization of mutant tomato lacking the expression of this gene revealed the biological significance of LePT3 and another novel gene LePT4. In addition we have isolated and characterized several phosphate starvation induced genes from tomato using a combination of differential and subtractive mRNA hybridization techniques. One of the genes, LePS2 belongs to the family of phospho-protein phosphatase. The functionality of the recombinant protein was determined using synthetic phosphor-peptides. Over expression of this gene in tomato resulted in significant changes in growth, delay in flowering and senescence. It is anticipated that phospho-protein phosphatase may have regulatory role in phosphate deficiency responses of plants. In addition a novel phosphate starvation induced glycerol 3-phosphate permease gene family was also characterized. Two doctoral research students are continuing the characterization and functional analysis of these genes. Over expression of high affinity phosphate transporters in tobacco showed increased phosphate content under hydroponic conditions. There is growing evidence suggesting that high affinity phosphate transporters are crucial for phosphate acquisition even under phosphate sufficiency conditions. This project has helped train several postdoctoral fellows and graduate students. Further analysis of transgenic plants expressing phosphatases and transporters will not only reveal the biological function of the targeted genes but also result in phosphate uptake and utilization efficient plants.
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McCarville, Joseph, i Yue Xiong. Regulation of Ubiquitin Mediated Proteolysis of G1 Cyclins and the CDK Inhibitor p27 by the Cullin Gene Family in Normal Tumorigenic Human Breast Cells. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2001. http://dx.doi.org/10.21236/ada398199.

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Michel, Jennifer. Regulation of Ubiquitin Mediated Proteolysis of G1 Cyclins and the CDK Inhibitor p27 by the Cullin Gene Family in Normal and Tumorigenic Human Breast Cells. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2000. http://dx.doi.org/10.21236/ada393108.

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Michel, Jennifer J. Regulation of Ubiquitin Mediated Proteolysis of G1 Cyclins and the CDK Inhibitor p27 by the Cullin Gene Family in Normal and Tumorigenic Human Breast Cells. Fort Belvoir, VA: Defense Technical Information Center, lipiec 1999. http://dx.doi.org/10.21236/ada384004.

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Ori, Naomi, i Mark Estelle. Specific mediators of auxin activity during tomato leaf and fruit development. United States Department of Agriculture, styczeń 2012. http://dx.doi.org/10.32747/2012.7597921.bard.

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The plant hormone auxin is involved in numerous developmental processes, including leaf and fruit development. The tomato (Solanumlycopersicum) gene ENTIRE (E) encodes an auxin-response inhibitor from the Aux/IAA family. While most loss-offunction mutations in Aux/IAA genes are similar to the wild type due to genetic redundancy, entire (e) mutants show specific effects on leaf and fruit development. e mutants have simple leaves, in contrast to the compound leaves of wild type tomatoes. In addition, e plants produce parthenocarpic fruits, in which fruit set occurs independently of fertilization. The aim of this research program was to utilize the e mutation to identify and characterize genes that mediate the specific effect of auxin in leaf and fruit development. The specific objectives of the project were to: 1. Characterize and map modifiers of the e leaf phenotype. 2. Characterize and map suppressors of the e fruit phenotype. 3. Dissect the developmental specificity of the E gene. 4. Examine the effect of fruit-overexpression of identified genes on fruit set and seed production. To identify mediators of auxin in leaf development, we mainly focused on one mutant, crawling elephant (crel, previously called t282), which showed substantial suppression of the e phenotype and other auxin-relatedphenotypes. We have identified the CREL gene as a homolog of the Arabidopsis VRN5 gene, involved in recruiting polycomb silencing complexes to specific targets. We showed that CREL affects auxin sensitivity in tomato. Suppressors of the e fruit phenotype have been further characterized and selected for more profound effects. Expression profiling by RNAseq was used to analyze the effect of e as well as crel on gene expression in leaves and fruits. This analysis has identified putative E and CREL targets. We have initiated studies to assess the role of some of these targets in flower and fruit development. The research has identified potential mediators of auxin response in leaf, flower and fruit development.
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