Gotowe bibliografie tematyczne / Gap junctions

Gotowa bibliografia na temat „Gap junctions”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 10 marca 2023

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Artykuły w czasopismach na temat "Gap junctions"

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Spray, D. C., R. L. White, F. Mazet i M. V. Bennett. "Regulation of gap junctional conductance". American Journal of Physiology-Heart and Circulatory Physiology 248, nr 6 (1.06.1985): H753—H764. http://dx.doi.org/10.1152/ajpheart.1985.248.6.h753.

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Gap junctional conductance is regulated by the number of channels between coupled cells (the balance between formation and loss of these channels) and by the fraction of these channels that are open (gating mechanisms). A variety of treatments are known to affect junction formation. Adenosine 3',5'-cyclic monophosphate (cAMP) is involved in some cases, and protein synthesis may be required but precursor molecules can also exist. Junction removal occurs both by dispersion of particles and by internalization of junctional membrane. Factors promoting removal are not well understood. A variety of gating mechanisms exist. Coupling may be controlled by changes in conductance of nonjunctional membranes. Several kinds of voltage dependence of junctional conductance are known, but rat ventricular junctions at least are electrically linear. Cytoplasmic acidification decreases conductance of most gap junctions. Sensitivity in rat ventricular myocytes allows modulation of coupling by moderate changes near normal internal pH. Increasing intracellular Ca also decreases junctional conductance, but in the better studied cases sensitivity is much lower to Ca than H. A few data support low sensitivity to Ca in cardiac cells, but quantitative studies are lacking. Higher alcohols such as octanol block junctional conductance in a wide range of tissues including rat ventricular myocytes. An antibody to liver gap junctions blocks junctions between rat ventricular myocytes. Cross reactivity indicates at least partial homology between many gap junctions. Although differences among gap junctions are known, a general physiology is being developed, which may have considerable relevance to normal cardiac function and also to conduction disorders of that tissue.
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Lo, W. K., i T. S. Reese. "Multiple structural types of gap junctions in mouse lens". Journal of Cell Science 106, nr 1 (1.09.1993): 227–35. http://dx.doi.org/10.1242/jcs.106.1.227.

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Gap junctions in the epithelium and superficial fiber cells from young mice were examined in lenses prepared by rapid-freezing, and processed for freeze-substitution and freeze-fracture electron microscopy. There appeared to be three structural types of gap junction: one type between epithelial cells and two types between fiber cells. Epithelial gap junctions seen by freeze-substitution were approximately 20 nm thick and consistently associated with layers of dense material lying along both cytoplasmic surfaces. Fiber gap junctions, in contrast, were 15–16 nm (type 1) or 17–18 nm thick (type 2), and had little associated cytoplasmic material. Type 1 fiber gap junctions were extensive in flat expanses of cell membrane and had a thin, discontinuous central lamina, whereas type 2 fiber gap junctions were associated with the ball-and-socket domains and exhibited a dense, continuous central lamina. Both types of fiber gap junction had a diffuse arrangement of junctional intramembrane particles, whereas particles and pits of epithelial gap junctions were in a tight, hexagonal configuration. The type 2 fiber gap junctions, however, had a larger particle size (approximately 9 nm) than the type 1 (approximately 7.5 nm). In addition, a large number of junctional particles typified the E-faces of both fiber types but not the epithelial type of gap junction. Gap junctions between fiber and epithelial cells had structural features of type 1 fiber gap junctions.(ABSTRACT TRUNCATED AT 250 WORDS)
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Naus, Christian CG. "Gap junctions and tumour progression". Canadian Journal of Physiology and Pharmacology 80, nr 2 (1.02.2002): 136–41. http://dx.doi.org/10.1139/y02-009.

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Gap junctional intercellular communication has been implicated in growth control and differentiation. The mechanisms by which connexins, the gap junction proteins, act as tumor suppressors are unclear. In this review, several different mechanisms are considered. Since transformation results in a loss of the differentiated state, one mechanism by which gap junctions may control tumour progression is to promote or enhance differentiation. Processes of differentiation and growth control are mediated at the genetic level. Thus, an alternative or complimentary mechanism of tumour suppression could involve the regulation of gene expression by connexins and gap junctional coupling. Finally, gap junction channels form a conduit between cells for the exchange of ions, second messengers, and small metabolites. It is clear that the sharing of these molecules can be rather selective and may be involved in growth control processes. In this review, examples will be discussed that provide evidence for each of these mechanisms. Taken together, these findings point to a variety of mechanims by which connexins and the gap junction channels that they form may control tumour progression.Key words: gap junctions, connexin, cancer.
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Verheule, Sander, Marjan J. A. van Kempen, Sjoerd Postma, Martin B. Rook i Habo J. Jongsma. "Gap junctions in the rabbit sinoatrial node". American Journal of Physiology-Heart and Circulatory Physiology 280, nr 5 (1.05.2001): H2103—H2115. http://dx.doi.org/10.1152/ajpheart.2001.280.5.h2103.

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In comparison to the cellular basis of pacemaking, the electrical interactions mediating synchronization and conduction in the sinoatrial node are poorly understood. Therefore, we have taken a combined immunohistochemical and electrophysiological approach to characterize gap junctions in the nodal area. We report that the pacemaker myocytes in the center of the rabbit sinoatrial node express the gap junction proteins connexin (Cx)40 and Cx46. In the periphery of the node, strands of pacemaker myocytes expressing Cx43 intermingle with strands expressing Cx40 and Cx46. Biophysical properties of gap junctions in isolated pairs of pacemaker myocytes were recorded under dual voltage clamp with the use of the perforated-patch method. Macroscopic junctional conductance ranged between 0.6 and 25 nS with a mean value of 7.5 nS. The junctional conductance did not show a pronounced sensitivity to the transjunctional potential difference. Single-channel recordings from pairs of pacemaker myocytes revealed populations of single-channel conductances at 133, 202, and 241 pS. With these single-channel conductances, the observed average macroscopic junctional conductance, 7.5 nS, would require only 30–60 open gap junction channels.
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Churchill, D., S. Coodin, R. R. Shivers i S. Caveney. "Rapid de novo formation of gap junctions between insect hemocytes in vitro: a freeze-fracture, dye- transfer and patch-clamp study". Journal of Cell Science 104, nr 3 (1.03.1993): 763–72. http://dx.doi.org/10.1242/jcs.104.3.763.

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Gap junctions form between insect hemocytes (blood cells) when they encapsulate foreign objects in the hemocoel (body cavity). In this study we show that hemocytes from cockroach (Periplaneta americana) form gap-junctions rapidly in vitro. Freeze-fracture replicas of hemocyte aggregates fixed 5 minutes after bleeding contain gap-junctional plaques. Dye passage was detected between carboxyfluorescein diacetate- labelled and unlabelled hemocytes within 3 minutes of bleeding, when the cells made contact as they flattened rapidly onto coverslips. When double whole-cell voltage-clamp was used to measure gap-junction formation between cells which were pushed together, electrical coupling was detected within one second of cell-cell contact. To prevent extensive flattening, cells were plated onto lipophorin-coated coverslips. Junctional conductance increased in staircase fashion with steps corresponding to an average single channel conductance of 345 pS. Assuming all channels to have this conductance, the maximal accretion rate of channels to the growing junction was one channel per second. Junctional currents and dye-coupling were detected in the absence of Ca2+, indicating that involvement of Ca2+-dependent adhesion molecules is not a prerequisite for gap-junction formation in hemocytes. Hemocytes from distantly related insects (cockroach and moth) form functional gap junctions with each other, suggesting sequence homology among gap- junction proteins in insects. The function of rapid gap-junction formation between hemocytes during encapsulation and wound healing in vivo are discussed.
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Rook, M. B., A. C. van Ginneken, B. de Jonge, A. el Aoumari, D. Gros i H. J. Jongsma. "Differences in gap junction channels between cardiac myocytes, fibroblasts, and heterologous pairs". American Journal of Physiology-Cell Physiology 263, nr 5 (1.11.1992): C959—C977. http://dx.doi.org/10.1152/ajpcell.1992.263.5.c959.

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Cultures of neonatal rat heart cells contain predominantly myocytes and fibroblastic cells. Most abundant are groups of synchronously contracting myocytes, which are electrically well coupled through large gap junctions. Cardiac fibroblasts may be electrically coupled to each other and to adjacent myocytes, be it with low intercellular conductances. Nevertheless, synchronously beating myocytes interconnected via a fibroblast were present, demonstrating that nonexcitable cardiac cells are capable of passive impulse conduction. In fibroblast pairs as well as in myocyte-fibroblast cell pairs, no sensitivity to junctional voltage could be detected when transjunctional conductance was > 1-2 nS. However, in pairs coupled by a conductance of < 1 nS, complex voltage-dependent gating was evident; gap junction channel open probability decreased with increasing junctional voltage but a nongated residual conductance remained at all voltages tested. Single gap junction channel conductance between fibroblasts was approximately 21 pS, very similar to an approximately 18-pS channel conductance that was found between myocytes next to the major conductance of 43 pS. Single-channel conductance in heterologous myocyte-fibroblast gap junctions was approximately 32 pS, which matches the theoretical value of 29 pS for gap junction channels composed of a fibroblast connexon and the major myocyte connexon. A site-directed antibody against rat heart gap junction protein connexin43 recognized gap junctions between neonatal cardiomyocytes, as demonstrated by immunocytochemical labeling. In contrast, junctions between fibroblasts showed no labeling, while in myocyte-fibroblast junctions labeling occasionally was present. Our results suggest the existence of two gap junction proteins between neonatal rat cardiocytes, connexin43 and another yet unidentified connexin. An alternative explanation (cell-specific regulation of the conductance of connexin43 channels) is discussed.
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Warner, Anne E. "The role of gap junctions in amphibian development". Development 89, Supplement (1.11.1985): 365–80. http://dx.doi.org/10.1242/dev.89.supplement.365.

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The possibility that communication through gap junctions may be important during embryonic development has often been raised since gap junctions were first described between early embryonic cells. It is now known that this direct cell-to-cell communication pathway disappears between groups of embryonic cells with different developmental fates as the embryo progresses through development, suggesting that transfer through the gap junctional pathway may play some part in controlling events during development. Supportive evidence for a role for gap junctions comes from experiments demonstrating that the properties of gap junctions differ at the border separating each segment in insect epidermis. Recently it has been shown that the ability to exchange small dyes between cells in the amphibian embryo depends on the position of each cell with respect to the grey crescent. When communication through gap junctions is prevented, by injecting antibodies to gap junctions protein, pattern formation is severely disturbed in the non-communicating region. The paper describes experiments on the pattern of junctional communication at early stages of development of the amphibian embryo and illustrates how anti-gap junction antibodies are being used to determine when and where communication through gap junctions may play an important role during development.
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Boitano, Scott, Zeenat Safdar, Donald G. Welsh, Jahar Bhattacharya i Michael Koval. "Cell-cell interactions in regulating lung function". American Journal of Physiology-Lung Cellular and Molecular Physiology 287, nr 3 (wrzesień 2004): L455—L459. http://dx.doi.org/10.1152/ajplung.00172.2004.

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Tight junction barrier formation and gap junctional communication are two functions directly attributable to cell-cell contact sites. Epithelial and endothelial tight junctions are critical elements of the permeability barrier required to maintain discrete compartments in the lung. On the other hand, gap junctions enable a tissue to act as a cohesive unit by permitting metabolic coupling and enabling the direct transmission of small cytosolic signaling molecules from one cell to another. These components do not act in isolation since other junctional elements, such as adherens junctions, help regulate barrier function and gap junctional communication. Some fundamental elements related to regulation of pulmonary barrier function and gap junctional communication were presented in a Featured Topic session at the 2004 Experimental Biology Conference in Washington, DC, and are reviewed in this summary.
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FitzGerald, PG. "Gap junction Heterogeneity in Liver, Heart, and Lens". Physiology 3, nr 5 (1.10.1988): 206–11. http://dx.doi.org/10.1152/physiologyonline.1988.3.5.206.

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Gap junctions are nearly ubiquitous structures that ionically and metabolically couple adjacent cells. Molecular analysis of junctional proteins is establishing the presence of families of unique but homologous junctional proteins, opening the door to an explanation of tissue specificity in gap junction structure and function.
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Ko, Kevin, Pamela Arora, Wilson Lee i Christopher McCulloch. "Biochemical and functional characterization of intercellular adhesion and gap junctions in fibroblasts". American Journal of Physiology-Cell Physiology 279, nr 1 (1.07.2000): C147—C157. http://dx.doi.org/10.1152/ajpcell.2000.279.1.c147.

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Despite their significance in wound healing, little is known about the molecular determinants of cell-to-cell adhesion and gap junctional communication in fibroblasts. We characterized intercellular adherens junctions and gap junctions in human gingival fibroblasts (HGFs) using a novel model. Calcein-labeled donor cells in suspension were added onto an established, Texas red dextran (10 kDa)-labeled acceptor cell monolayer. Cell-to-cell adhesion required Ca2+ and was >30-fold stronger than cell-to-fibronectin adhesion at 15 min. Electron micrographs showed rapid formation of adherens junction-like structures at ∼15 min that matured by ∼2–3 h; distinct gap junctional complexes were evident by ∼3 h. Immunoblotting showed that HGF expressed β-catenin and that cadherins and connexin43 were recruited to the Triton-insoluble cytoskeletal fraction in confluent cultures. Confocal microscopy localized the same molecules to intercellular contacts of acceptor and donor cells. There was extensive calcein dye transfer in a cohort of Texas red dextran-labeled cells, but this was almost completely abolished by the gap junction inhibitor β-glycyrrhetinic acid and the connexin43 mimetic peptide GAP 27. This donor-acceptor cell model allows large numbers (>105) of cells to form synchronous cell-to-cell contacts, thereby enabling the simultaneous functional and molecular studies of adherens junctions and gap junctions.
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Rozprawy doktorskie na temat "Gap junctions"

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Sheela, Thomas Vinaya. "Regulation of Connexin40 Gap Junctions". Digital Archive @ GSU, 2008. http://digitalarchive.gsu.edu/biology_diss/55.

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Gap junctions provide direct electrical and biochemical communication between cardiomyocytes in the heart. Connexin40 (Cx40) is the major connexin in the atria of the heart and little is known regarding its regulation. Thus, the goal was to investigate the regulation of Cx40 in both physiological and pathophysiological conditions. The first objective of this thesis was to determine whether Cx40 gap junctions were regulated by â-adrenergic receptor activation. Cx40 has previously been shown to be acutely activated by cAMP, this cAMP-induced increase in Cx40-mediated cell-to-cell dye transfer has been shown to be effected through the â-adrenergic receptor-adenylyl cyclase- Protein Kinase A (PKA) pathway in Cx40-transfected HeLa cells. The second objective of this thesis was to determine whether Cx40 gap junctions were regulated by intracellular Ca2+ concentration ([Ca2+]i ). [Ca2+]i was increased by addition of the ionophore ionomycin and elevating extracellular calcium [Ca2+]o from 1.8 mM to 21.8 mM. This resulted in an elevation of [Ca2+]i and effected an inhibition of Cx40-mediated cell-to-cell dye transfer (IC50 of 500 ± 0.72 nM) which was Calmodulin-dependent. The third objective of this thesis was to determine whether Cx40 gap junctions were regulated by ischemia. Inducing ischemia chemically by inhibiting the electron transport chain with sodium cyanide and glycolysis with iodoacetate and 2-deoxyglucose effected an inhibition of Cx40-mediated cell-to-cell dye transfer that was shown to be Calmodulin dependent. The main conclusions of this thesis were: (1) â-adrenergic receptor activation increases Cx40-mediated cell-to-cell dye transfer which requires the activation of PKA; (2) A sustained elevation in [Ca2+]i causes a partial inhibition of Cx40 gap junction-mediated cell-to-cell dye transfer which was Ca2+-and Calmodulin dependent; (3) Chemical ischemia causes a partial inhibition of Cx40 gap junction-mediated cell-to-cell dye transfer which was shown to be Calmodulin-dependent.
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Carolan, E. J. "Gap junctions in lymphocyte ontogeny". Thesis, University of Glasgow, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.233181.

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Nishimura, Tamiko. "Gap junctions in early human placental development". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp05/MQ63198.pdf.

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Ko, Yu-Shien. "Connexin heterogeneity in vascular cell gap junctions". Thesis, Imperial College London, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.393781.

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Pintsch, Monika. "Cx37 abhängige Calciumsignalausbreitung durch myoendotheliale Gap Junctions". Diss., Ludwig-Maximilians-Universität München, 2015. http://nbn-resolving.de/urn:nbn:de:bvb:19-182614.

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Eine effektive Regulation der Gewebedurchblutung erfordert eine Koordination der Reaktion einzelner Gefäßzellen bzw. verschiedener Gefäßabschnitte. Der zur Koordination erforderliche interzelluläre Signalaustausch kann zumindest teilweise über Gap Junction-Kanäle erfolgen, die als interzelluläre Verbindungen den Austausch von elektrischen und chemischen Signalstoffen zwischen benachbarten Zellen ermöglichen. Dieser Austausch kann über die Modulation der Permeabilität von Gap Junction-Kanälen reguliert werden. Aus Untersuchungen an Modellzellen (HeLA-Zellen) war bereits bekannt dass NO eine solche Modulatorwirkung ausübt, wenn die Gap Junctions nur Connexin 37 (Cx37) enthalten während kein Effekt von NO auf Gap Junctions zu beobachtet war, wenn Gap Junctions aus Cx43 oder Cx40 gebildet wurden. Da Endothelzellen normalerweise alle drei Connexine exprimieren, sollte in der vorliegenden Arbeit untersucht werden, inwieweit NO in diesen Zellen überhaupt eine nachweisbare Wirkung auf die Gap Junction Permeabilität und damit auf den Signalaustausch entfaltet. Als Modell des Signalaustauschs wurde die Ausbreitung von Calciumwellen jeweils zwischen Endothelzellen oder glatten Muskelzellen allein oder zwischen beiden Zelltypen untersucht. Nach Auslösung von interzellulären Calciumwellen als Folge einer mechanischen Stimulation von einzelnen Zellen konnte zunächst gezeigt werden, dass die interzelluläre Ausbreitung von Calcium unter den gewählten Versuchsbedingungen über Gap Junctions-erfolgte. Im Gegensatz zum Modellsystem der HeLa Zellen, in denen nur Cx37 exprimiert war, zeigte NO in den Endothelzellen (humane Nabelschnur, alle drei Connexine exprimiert) abgesehen von einer geringradigen Verzögerung keinen Hemmeffekt auf die Gap Junction-abhängige Ausbreitung von Calcium-Signalen. Wurde jedoch Cx43 durch Behandlung mit siRNA herunterreguliert, führte NO auch in den Endothelzellen zu einer Hemmung der interzellulären Calciumwellenausbreitung. Auch in intakten Endothelzellen, die mit glatten Muskelzellen kokultiviert wurden, ließ sich bei genauerer Analyse ein Hemmeffekt von NO nachweisen. Dieser war jedoch auf die Zellbereiche beschränkt, in denen Endothelzellen und glatte Muskelzellen unmittelbar benachbart waren (myoendotheliale Junctions). In diesen myoendo-thelialen Gap Junctions, fanden wir auf der Endothelseite immunhistochemisch überwiegend Cx37 exprimiert. Aufgrund dieser präferentiellen Lokalisation von Cx37 scheint daher NO eine besondere Rolle bei der Modulation des Calciumaustauschs (und potentiell auch anderer Signalmoleküle wie IP3 oder cyclische Nukleotide) zu spielen. Die Kontrolle des Calciumaustauschs könnte funktionell eine calciumabhängige glattmuskuläre Kontraktion bei Endothelstimulation verhindern und somit die endothelabhängige Dilatation verstärken. Diese bisher unbekannte NO-Wirkung auf Cx37-exprimierende Gap Junctions könnte einen weiteren Mechanismus der Gefäßtonusregulation darstellen.
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Rahman, Salman. "Biochemical and immunological studies on gap junctions". Thesis, University College London (University of London), 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266786.

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Kalapothakis, Evanguedes. "Expression and biomolecular assembly of gap junctions". Thesis, Open University, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.332822.

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Calkins, Travis L. "Gap Junctions in the Mosquito, Aedes aegypti". The Ohio State University, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu149217328492135.

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Gakhar, Gunjan. "Role of gap junctions in breast cancer". Diss., Manhattan, Kan. : Kansas State University, 2009. http://hdl.handle.net/2097/2228.

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Moore, Lisa Karen. "Regulation of gap junctions in vascular smooth muscle". Diss., The University of Arizona, 1993. http://hdl.handle.net/10150/186240.

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Gap junctions form low resistance pathways between neighboring cells and thereby provide for coordination of tissue function. In vascular smooth muscle these channels are believed to be important in maintenance of and coordination of changes in vessel tone. In this study we demonstrate that vascular smooth muscle cells from vessels of different origin and species differ in the connexin protein expressed as well as in the size of the channel formed by these proteins. We have shown that pig coronary and rat mesentery express mRNA for Cx43 and exhibit a single channel conductance of 60 or 80 pS respectively. We have determined the A7r5 express Cx40 in the form of a 70 pS channel and Cx43 as 108 and 141 pS channel. And finally we show that human coronary appear to express Cx40 exclusively, yet have two channel sizes with conductances of 52 and 104 pS. We further demonstrate that the effect of oleic acid (OA), the predominant fatty acid found in the cell membrane differs in its effect on the A7r5 vs. heart cells. The A7r5 cells responded with a rapid uncoupling to ∼50% at low dose, and did not further uncouple with increasing concentrations. Single channel analysis suggests the 70 pS channel was very sensitive to OA. The 140 pS channel appeared to be insensitive to OA. Lastly, we examined the effect of the monoamine, serotonin on gap junctions from vessel beds known to differ in their regulation. Junctional conductance and dye-coupling was increased on both long- and short-term exposure to 1,5, or 10 μM doses in all cell types. In the pig coronary cells and the rat mesentery, no change in unitary conductance was observed. The relative frequencies of the channel populations were shifted in both the A7r5 and the human coronary cells. These data support the conclusion that multiple strategies exist for gap junction regulation.
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Książki na temat "Gap junctions"

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Bennett, Michael V. L. 1931-, Spray David C i Cold Spring Harbor Laboratory, red. Gap junctions. Cold Spring Harbor, N.Y: Cold Spring Harbor Laboratory, 1985.

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E, Hall J., Zampighi G i Davis R. M, red. Gap junctions. Amsterdam: Elsevier, 1993.

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L, Hertzberg Elliot, red. Gap junctions. Stamford, Conn: Jai Press, 2000.

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International Gap Junction Conference (8th 1997 Key Largo, Fla.). Gap junctions: Proceedings of the 8th International Gap Junction Conference, Key Largo, Florida. Amsterdam: IOS Press, 1998.

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Y, Kanno, i International Meeting on Gap Junctions (1993 : Hiroshima-shi, Japan), red. Intercellular communication through gap junctions. Amsterdam: Elsevier, 1995.

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1930-, Sperelakis Nick, i Cole William C. 1955-, red. Cell interactions and gap junctions. Boca Raton, Fla: CRC Press, 1989.

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Winterhager, Elke, red. Gap Junctions in Development and Disease. Berlin, Heidelberg: Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/3-540-28621-7.

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Spray, David C., i Rolf Dermietzel. Gap Junctions in the Nervous System. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-662-21935-5.

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C, Spray David, i Dermietzel Rolf, red. Gap junctions in the nervous system. New York: Chapman & Hall, 1996.

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Nishimura, Tamiko. Gap junctions in early human placental development. Ottawa: National Library of Canada, 2001.

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Części książek na temat "Gap junctions"

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Börgers, Christoph. "Gap Junctions". W An Introduction to Modeling Neuronal Dynamics, 165–73. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-51171-9_21.

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Alonso, Angel. "Gap Junctions". W Encyclopedia of Cancer, 1–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27841-9_2325-2.

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Churko, Jared M., i Dale W. Laird. "Gap Junctions". W Cellular Domains, 339–47. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2011. http://dx.doi.org/10.1002/9781118015759.ch20.

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Wagner, Peter, Frank C. Mooren, Hidde J. Haisma, Stephen H. Day, Alun G. Williams, Julius Bogomolovas, Henk Granzier i in. "Gap Junctions". W Encyclopedia of Exercise Medicine in Health and Disease, 351. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2424.

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Alonso, Angel. "Gap Junctions". W Encyclopedia of Cancer, 1843–44. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-46875-3_2325.

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Alonso, Angel. "Gap Junctions". W Encyclopedia of Cancer, 1502–3. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_2325.

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Pavelka, Margit, i Jürgen Roth. "Tight Junctions and Gap Junctions". W Functional Ultrastructure, 168–69. Vienna: Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_88.

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Duffy, Heather S., Alfredo G. Fort i David C. Spray. "Cardiac Connexins: Genes to Nexus". W Cardiovascular Gap Junctions, 1–17. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000092550.

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van Veen,, Toon A. B., Harold V. M. van Rijen i Habo J. Jongsma. "Physiology of Cardiovascular Gap Junctions". W Cardiovascular Gap Junctions, 18–40. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000092560.

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Giepmans, Ben N. G. "Role of Connexin43-Interacting Proteins at Gap Junctions". W Cardiovascular Gap Junctions, 41–56. Basel: KARGER, 2006. http://dx.doi.org/10.1159/000092561.

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Streszczenia konferencji na temat "Gap junctions"

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Bathany, Ce´dric, Thomas Suchyna i Susan Z. Hua. "A Microfluidic Chip for Studying Intercellular Communication via Gap Junction Channels". W ASME 2010 First Global Congress on NanoEngineering for Medicine and Biology. ASMEDC, 2010. http://dx.doi.org/10.1115/nemb2010-13135.

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Cells in tissues and organs coordinate their activities with each other, and this communication is mainly mediated by specialized channels, called Gap Junctions. To date, our understanding of specificity of reagents and extracellular stimuli to gap junctions is very limited. Existing techniques for gap junctions assay are tedious and not convenient. We have developed a microfluidic chip that is capable of detecting chemical diffusion across gap junctions, as well as screening reagents for specific gap junction channels.
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Calkins, Travis Lee. "Gap junctions and the mosquito blood meal". W 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.113636.

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Hejri, Farrokh, Mladen Veletić i Ilangko Balasingham. "On the Cardiac Gap Junctions Channel Modeling". W NANOCOM '19: The Sixth Annual ACM International Conference on Nanoscale Computing and Communication. New York, NY, USA: ACM, 2019. http://dx.doi.org/10.1145/3345312.3345475.

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Wildie, Mark, Wayne Luk, Simon R. Schultz, Philip H. W. Leong i Andreas K. Fidjeland. "Reconfigurable acceleration of neural models with gap junctions". W 2009 International Conference on Field-Programmable Technology (FPT). IEEE, 2009. http://dx.doi.org/10.1109/fpt.2009.5377639.

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Ienna, Antonio, Michele Migliore i Cesare Valenti. "Visualization of Simulated Arrhythmias due to Gap Junctions". W CompSysTech'18: 19th International Conference on Computer Systems and Technologies. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3274005.3274011.

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Appukuttan, Shailesh, Rohan Sathe i Rohit Manchanda. "Modular approach to modeling homotypic and heterotypic gap junctions". W 2015 IEEE 5th International Conference on Computational Advances in Bio and Medical Sciences (ICCABS). IEEE, 2015. http://dx.doi.org/10.1109/iccabs.2015.7344707.

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Nguyen, M. Chung, V. Hung Nguyen, P. Dollfus i H. Viet Nguyen. "Conduction gap of strained/unstrained graphene junctions: Direction dependence". W 2014 International Workshop on Computational Electronics (IWCE). IEEE, 2014. http://dx.doi.org/10.1109/iwce.2014.6865868.

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Loginova, Nadezhda, Nikola Panov, Nikolai Kositsyn i Mikhail Svinov. "THE ROLE OF GAP JUNCTIONS IN THE CEREBRAL ISCHEMIA DEVELOPMENT". W XIV International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2018. http://dx.doi.org/10.29003/m188.sudak.ns2018-14/305.

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Krishnan, J., V. S. Chakravarthy i S. Radhakrishnan. "On the role of gap junctions in cardiac memory effect". W Computers in Cardiology, 2005. IEEE, 2005. http://dx.doi.org/10.1109/cic.2005.1588020.

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Li, L., G. A. Gusarova, M. N. Islam i J. Bhattacharya. "Siglec-F Determines Lung Immunity Through Regulation of Macrophage Gap Junctions". W American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a5832.

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Raporty organizacyjne na temat "Gap junctions"

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Chen, J., J. F. Zasadzinski, K. E. Gray, J. L. Wagner, D. G. Hinks, K. Kouznetsov i L. Coffey. BCS-like gap structure of HgBa{sub 2}CuO{sub 4+{delta}} tunnel junctions. Office of Scientific and Technical Information (OSTI), grudzień 1994. http://dx.doi.org/10.2172/10104581.

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Myers, Janette. Application of Single Particle Electron Microscopy to Native Lens Gap Junctions and Intrinsically Disordered Signaling Complexes. Portland State University Library, styczeń 2000. http://dx.doi.org/10.15760/etd.6884.

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Donahue, Henry J. Gap Junctional Intercellular Communication and Breast Cancer Metastasis to Bone. Fort Belvoir, VA: Defense Technical Information Center, październik 2001. http://dx.doi.org/10.21236/ada405244.

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Trosko, James E. The Role of Chemical Inhibition of Gap-Junctional Intercellular Communication in Toxicology. Fort Belvoir, VA: Defense Technical Information Center, marzec 1989. http://dx.doi.org/10.21236/ada207130.

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Trosko, James E. The Role of Chemical Inhibition of Gap-Junctional Intercellular Communication in Toxicology. Fort Belvoir, VA: Defense Technical Information Center, marzec 1991. http://dx.doi.org/10.21236/ada241465.

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Trosko, James E. The Role of Chemical Inhibition of Gap-Junctional Intercellular Communication in Toxicology. Fort Belvoir, VA: Defense Technical Information Center, marzec 1990. http://dx.doi.org/10.21236/ada221480.

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Raymond, Kara, Laura Palacios i Evan Gwilliam. Status of climate and water resources at Big Bend National Park: Water year 2019. Redaktor Tani Hubbard. National Park Service, wrzesień 2022. http://dx.doi.org/10.36967/2294267.

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Climate and hydrology are major drivers of ecosystem structure and function, particularly in arid and semi-arid ecosystems. Understanding changes in climate, groundwater, streamflow, and water quality is central to assessing the condition of park resources. This report combines data collected on climate, groundwater, and springs at Big Bend National Park (NP) to provide an integrated look at climate and water conditions during water year (WY) 2019 (October 2018–September 2019). However, this report does not address the Rio Grande or its tributaries. Annual precipitation was higher than normal (1981–2010) for Big Bend NP at four of the five National Oceanic and Atmospheric Administration Cooperative Observer Program weather stations: 111% of normal for Chisos Basin, 122% of normal for Panther Junction, 155% of normal for Persimmon Gap, and 124% of normal for Rio Grande Village. Castolon had 88% of normal annual precipitation. All five stations had higher than normal rainfall in October and December, while rainfall totals were substantially below normal at all stations in November, February, and March. Monthly precipitation totals for April through September were more variable from station to station. Mean monthly maximum air temperatures were below normal in the fall months, with Panther Junction as much as 7.5°F below normal in October. Monthly temperatures from January through July were more variable. Temperatures in August and September were warmer than normal at every station, up to +9.4°F at Rio Grande Village and +8.7°F at Chisos Basin in July. The reconnaissance drought index values indicate generally wetter conditions (based on precipitation and evaporative demand) at Chisos Basin since WY2016 and at Panther Junction and Persimmon Gap since WY2015, except for WY2017. This report presents the manual and automatic groundwater monitoring results at nine wells. Five wells had their highest water level in or just before WY2019: Panther Junction #10 peaked at 99.94 ft below ground surface (bgs) in September 2018, Contractor’s Well peaked at 31.43 ft bgs in November 2018, T-3 peaked at 65.39 ft bgs in December 2018, K-Bar #6 Observation Well peaked at 77.78 ft bgs in February 2019, and K-Bar #7 Observation Well peaked at 43.18 ft bgs in February 2019. This was likely in response to above normal rainfall in the later summer and fall 2018. The other monitoring wells did not directly track within-season precipitation. The last measurement at Gallery Well in WY2019 was 18.60 ft bgs. Gallery Well is located 120 feet from the river and closely tracked the Rio Grande stage, generally increasing in late summer or early fall following higher flow events. Water levels in Gambusia Well were consistently very shallow, though the manual well measurement collected in April was 4.25 ft bgs—relatively high for the monitoring record—and occurred outside the normal peak period of later summer and early fall. The last manual measurement taken at TH-10 in WY2019 was 34.80 ft bgs, only 0.45 ft higher than the earliest measurement in 1967, consistent with the lack of directional change in groundwater at this location, and apparently decoupled from within-season precipitation patterns. The last water level reading in WY2019 at Oak Springs #1 was 59.91 ft bgs, indicating an overall decrease of 26.08 ft since the well was dug in 1989. The Southwest Network Collaboration (SWNC) collects data on sentinel springs annually in the late winter and early spring following the network springs monitoring protocol. In WY2019, 18 sentinel site springs were visited at Big Bend NP (February 21, 2019–March 09, 2019). Most springs had relatively few indications of natural and anthropogenic disturbances. Natural disturbances included recent flooding, drying, and wildlife use. Anthropogenic disturbances included flow modifications (e.g., springboxes), hiking trails, and contemporary human use. Crews observed one to seven facultative/obligate wetland plant...
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Guha, S. Research on high-efficiency, multi-gap, multi-junction amorphous silicon-based alloy thin-film solar cells. Office of Scientific and Technical Information (OSTI), październik 1989. http://dx.doi.org/10.2172/5164953.

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Madan, A. High Efficiency Narrow Gap and Tandem Junction Devices: Final Technical Report, 1 May 2002--31 October 2004. Office of Scientific and Technical Information (OSTI), marzec 2005. http://dx.doi.org/10.2172/15011482.

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Cameron, C. E., E. E. Thoms i C. A. Gallo. Engineering-geologic database of the proposed Alaska Natural Gas Transportation System (ANGTS) corridor from Prudhoe Bay to Delta Junction, Alaska. Alaska Division of Geological & Geophysical Surveys, lipiec 2002. http://dx.doi.org/10.14509/2862.

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