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Artykuły w czasopismach na temat "Functional reprogramming"
Trakala, Marianna, Sara Rodríguez-Acebes, María Maroto, Catherine E. Symonds, David Santamaría, Sagrario Ortega, Mariano Barbacid, Juan Méndez i Marcos Malumbres. "Functional Reprogramming of Polyploidization in Megakaryocytes". Developmental Cell 32, nr 2 (styczeń 2015): 155–67. http://dx.doi.org/10.1016/j.devcel.2014.12.015.
Pełny tekst źródłaKubatiev, A. A., i A. A. Pal'tsyn. "INTRACELLULAR BRAIN REGENERATION: A NEW VIEW". Annals of the Russian academy of medical sciences 67, nr 8 (11.08.2012): 21–25. http://dx.doi.org/10.15690/vramn.v67i8.345.
Pełny tekst źródłaKumar, Satish, Joanne E. Curran, David C. Glahn i John Blangero. "Utility of Lymphoblastoid Cell Lines for Induced Pluripotent Stem Cell Generation". Stem Cells International 2016 (2016): 1–20. http://dx.doi.org/10.1155/2016/2349261.
Pełny tekst źródłaPaoletti, Camilla, Carla Divieto i Valeria Chiono. "Impact of Biomaterials on Differentiation and Reprogramming Approaches for the Generation of Functional Cardiomyocytes". Cells 7, nr 9 (21.08.2018): 114. http://dx.doi.org/10.3390/cells7090114.
Pełny tekst źródłaÖzcan, Ismail, i Baris Tursun. "Identifying Molecular Roadblocks for Transcription Factor-Induced Cellular Reprogramming In Vivo by Using C. elegans as a Model Organism". Journal of Developmental Biology 11, nr 3 (31.08.2023): 37. http://dx.doi.org/10.3390/jdb11030037.
Pełny tekst źródłaKalo, Eric, Scott Read i Golo Ahlenstiel. "Reprogramming—Evolving Path to Functional Surrogate β-Cells". Cells 11, nr 18 (8.09.2022): 2813. http://dx.doi.org/10.3390/cells11182813.
Pełny tekst źródłaPeng, Bo, Hui Li i Xuan-Xian Peng. "Functional metabolomics: from biomarker discovery to metabolome reprogramming". Protein & Cell 6, nr 9 (2.07.2015): 628–37. http://dx.doi.org/10.1007/s13238-015-0185-x.
Pełny tekst źródłaTian, E., Guoqiang Sun, Guihua Sun, Jianfei Chao, Peng Ye, Charles Warden, Arthur D. Riggs i Yanhong Shi. "Small-Molecule-Based Lineage Reprogramming Creates Functional Astrocytes". Cell Reports 16, nr 3 (lipiec 2016): 781–92. http://dx.doi.org/10.1016/j.celrep.2016.06.042.
Pełny tekst źródłaZhu, Hui, Srilatha Swami, Pinglin Yang, Frederic Shapiro i Joy Y. Wu. "Direct Reprogramming of Mouse Fibroblasts into Functional Osteoblasts". Journal of Bone and Mineral Research 35, nr 4 (30.12.2019): 698–713. http://dx.doi.org/10.1002/jbmr.3929.
Pełny tekst źródłaZhou, Huanyu, Matthew E. Dickson, Min Soo Kim, Rhonda Bassel-Duby i Eric N. Olson. "Akt1/protein kinase B enhances transcriptional reprogramming of fibroblasts to functional cardiomyocytes". Proceedings of the National Academy of Sciences 112, nr 38 (9.09.2015): 11864–69. http://dx.doi.org/10.1073/pnas.1516237112.
Pełny tekst źródłaRozprawy doktorskie na temat "Functional reprogramming"
Hoffmann, Daniel [Verfasser], i Hans-Ulrich [Akademischer Betreuer] Mösch. "Functional reprogramming of Candida glabrata epithelial adhesins by exchange of variable structural motifs / Daniel Hoffmann ; Betreuer: Hans-Ulrich Mösch". Marburg : Philipps-Universität Marburg, 2021. http://d-nb.info/1227580169/34.
Pełny tekst źródłaFARIA, PEREIRA MARLENE CRISTINA. "EPIGENETIC AND FUNCTIONAL ASSESSMENT OF ENHANCEROPATHIES ACROSS HUMAN MODELS: FOCUS ON GABRIELE-DE VRIES SYNDROME". Doctoral thesis, Università degli Studi di Milano, 2022. https://hdl.handle.net/2434/945230.
Pełny tekst źródłaJing, Chenzhi. "Characterisation of the effect and functional significance of Fcγ receptor crosslinking on metabolic processes in macrophages". Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/280316.
Pełny tekst źródłaLo, Presti Caroline. "Reprogrammation métabolique dans les leucémies aigues myéloblastiques (LAM) : Impact clinique et mécanismes oncogéniques De novo adult acute myeloid leukemia patients display at diagnosis functional deregulation of redox balance correlated with molecular subtypes and overall survival". Thesis, Université Grenoble Alpes, 2020. http://www.theses.fr/2020GRALV017.
Pełny tekst źródłaCells metabolism is strongly disturbed and deregulated in cancers. Several examples reflect this phenomenon, including metabolic reprogramming described in the Warburg effect, functional deregulations of particular metabolic pathways such as the increase of the ROS production in cancer cells, or the identification of oncometabolites linked to acquired mutations such as IDH1/2 mutations, which lead to the production of a metabolite directly linked to the leukemic process in AML. In order to characterize the metabolic reprogramming associated with the leukemic process, we analyzed by an HRMAS approach the metabolites produced by different leukemic cell lines representing different subtypes of AML (different genotype and phenotype). In this model, we have shown that each type of cell line exhibited a particular metabolism in the basal state, witnessing a different metabolic signature depending on the nature of the cell line. In condition of metabolic stress (culture in a serum-free environment), all these cell lines develop mechanisms to adapt their metabolism to nutrient deficiency. Particularly, there is a common signature characterized by the overexpression of metabolites of the phospholipid pathway and of regulation of oxidative stress after 24 hours of culture in a medium without serum. Thanks to these adaptation mechanisms, the leukemic cells find after 48 hours a viability higher than 95% and a metabolic profile almost identical to normal conditions. These results show that leukemic cells develop common survival mechanisms, notably involving deregulations of lipid metabolism, which allow them to continue to proliferate in condition of metabolic stress. Other experimental conditions have been tested, in particular in glucose deficiency conditions in order to explore the path of deregulation of some amino acids such as alanine in these cell lines. Moreover, the quantitative and qualitative study of fatty acids in AMLs through a lipidomic approach reveals a similar adaptation of the lipidomic profiles of the cell lines in the same serum-free conditions previously tested. In parallel, in a study on 54 patients diagnosed with AML, we confirmed by the HRMAS approach that there were differences in metabolic profile in AML patients according to the AML subtype. We also showed that these metabolic signatures were significantly correlated with cytogenetic prognostic subgroups, response to chemotherapy treatment and patient survival. We show in particular that the metabolites overexpressed in patients with poor prognosis are found overexpressed also in patients refractory to treatment. The analysis of these metabolites shows the particular role of several metabolic pathways in the prognosis of AML: i) deregulation of the synthesis of 2-hydroxyglutarate associated with mutations in the IDH1/2 enzyme, ii) deregulation of the metabolism of phospholipids, showing an overexpression of phospholipids in adverse prognosis patients plasmas, and iii) overexpression of the synthesis of some amino acids in chemoresistant patients, suggesting an involvement of the LKB1/AMPK signaling pathway
Deva, Nathan Aurélia. "Caractérisation des bases moléculaires et cellulaires de la reprogrammation fonctionnelle radio-induite des macrophages dans le cadre du traitement du cancer". Electronic Thesis or Diss., université Paris-Saclay, 2023. http://www.theses.fr/2023UPASL079.
Pełny tekst źródłaTumor-associated macrophages (TAMs) are key components of the tumor microenvironment that display immunosuppressive functions and are associated with poor prognosis in most cancers. The functional reprogramming of these macrophages with pro-tumor properties towards a proinflammatory phenotype with anti-tumor properties promotes the development of an anti-tumor response. Our team recently studied how ionizing radiation modulates macrophage reprogramming towards a proinflammatory phenotype. Increasing the ability of ionizing radiation to reprogram TAMs into proinflammatory macrophages is a key objective to improve the effectiveness of cancer treatments.In this context, my thesis work enabled (i) to further characterize the molecular mechanisms involved in the radiation-induced macrophage reprogramming, (ii) to identify the role of the purinergic receptor P2Y2 as a negative modulator of the proinflammatory reprogramming of macrophages; (ii) to characterize the molecular bases of this biological process, and (iii) to propose the inhibition of the biological activity of P2Y2 receptor, to increase the ability of ionizing radiation, triggering the pro-inflammatory activation of macrophages
Ozmadenci, Duygu. "Netrin-1 function in somatic cell reprogramming and pluripotency". Thesis, Lyon, 2017. http://www.theses.fr/2017LYSE1254/document.
Pełny tekst źródłaPluripotency is the ability of embryonic epiblast cells to self-renew and to give rise to all somatic cells as well as germ cells. Somatic cells can also be reprogrammed toward pluripotency, opening new avenues for stem cell based therapies in the treatment of degenerative diseases. Deciphering the molecular mechanisms, and in particular signaling pathways that control pluripotency is crucial to improve our understanding of early embryogenesis and the use of iPSC (inducible Pluripotent Stem Cell) in regenerative medicine.Herein, I provide the first description of Netrin-1 as a regulator of reprogramming and pluripotency. Netrin-1 and its receptors are present in many cell types and are engaged in a variety of cellular processes beyond its initial characterization in the neuronal system. In the first part, I contributed to explore how Netrin-1 prevents apoptosis mediated by its dependence receptor DCC (Deleted in Colon Carcinoma) during reprogramming. In the second part, I dissected the functions and regulation of this pathway in pluripotency maintenance and in lineage commitment
Cao, Lu. "A genome wide approach to stress response and chronological ageing in yeast". Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/285995.
Pełny tekst źródłaKaemena, Daniel Fraser. "CRISPR/Cas9 genome-wide loss of function screening identifies novel regulators of reprogramming to pluripotency". Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31184.
Pełny tekst źródłaChidiac, Mounia. "A study of apolipoprotein L1 patho-physiological functions". Doctoral thesis, Universite Libre de Bruxelles, 2015. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/217789.
Pełny tekst źródłaOption Biologie moléculaire du Doctorat en Sciences
info:eu-repo/semantics/nonPublished
Vasiliauskaite, Lina [Verfasser], i Ramesh [Akademischer Betreuer] Pillai. "EMBRYONIC FUNCTIONS OF REPROGRAMMING MUTANTS MIWI2, MILI AND DNMT3L INFLUENCE ADULT MALE GERMLINE MAINTENANCE / Lina Vasiliauskaite ; Betreuer: Ramesh Pillai". Heidelberg : Universitätsbibliothek Heidelberg, 2017. http://d-nb.info/1180985400/34.
Pełny tekst źródłaKsiążki na temat "Functional reprogramming"
Venet, Fabienne, i Alain Lepape. Immunoparesis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0313.
Pełny tekst źródłaWang, Xiao-Dong, i Mathias V. Schmidt, red. Molecular Mechanisms for Reprogramming Hippocampal Development and Function by Early-Life Stress. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-806-1.
Pełny tekst źródłaCzęści książek na temat "Functional reprogramming"
Salts, Nuphar, i Eran Meshorer. "Epigenetics in Development, Differentiation and Reprogramming". W The Functional Nucleus, 421–48. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-38882-3_18.
Pełny tekst źródłaPerry, John M., i Linheng Li. "Functional Assays for Hematopoietic Stem Cell Self-Renewal". W Cellular Programming and Reprogramming, 45–54. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-691-7_3.
Pełny tekst źródłaShi, Yan. "Generation of Functional Insulin-Producing Cells from Human Embryonic Stem Cells In Vitro". W Cellular Programming and Reprogramming, 79–85. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-691-7_5.
Pełny tekst źródłaHeinrich, Christophe, Magdalena Götz i Benedikt Berninger. "Reprogramming of Postnatal Astroglia of the Mouse Neocortex into Functional, Synapse-Forming Neurons". W Methods in Molecular Biology, 485–98. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-452-0_32.
Pełny tekst źródłaZhang, Zhonghui, i Wen-Shu Wu. "Application of TALE-Based Approach for Dissecting Functional MicroRNA-302/367 in Cellular Reprogramming". W MicroRNA Protocols, 255–63. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7601-0_21.
Pełny tekst źródłaRobinson, Meghan, Oliver McKee-Reed, Keiran Letwin i Stephanie Michelle Willerth. "Direct Reprogramming Somatic Cells into Functional Neurons: A New Approach to Engineering Neural Tissue In Vitro and In Vivo". W Regenerative Medicine and Plastic Surgery, 447–62. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-19962-3_31.
Pełny tekst źródłaQiu, Boning, Ruben J. de Vries i Massimiliano Caiazzo. "Direct Cell Reprogramming of Mouse Fibroblasts into Functional Astrocytes Using Lentiviral Overexpression of the Transcription Factors NFIA, NFIB, and SOX9". W Methods in Molecular Biology, 31–43. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1601-7_3.
Pełny tekst źródłaMullin, Nicholas, i Ian Chambers. "The Function of Nanog in Pluripotency". W Nuclear Reprogramming and Stem Cells, 99–112. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-225-0_9.
Pełny tekst źródłaMasui, Shinji. "Function of Oct3/4 and Sox2 in Pluripotency". W Nuclear Reprogramming and Stem Cells, 113–25. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-225-0_10.
Pełny tekst źródłaOsakada, Fumitaka, i Masayo Takahashi. "Toward Regeneration of Retinal Function Using Pluripotent Stem Cells". W Nuclear Reprogramming and Stem Cells, 155–75. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-225-0_13.
Pełny tekst źródłaStreszczenia konferencji na temat "Functional reprogramming"
Möbus, S., J. Markovic, MP Manns, M. Ott, T. Cantz i AD Sharma. "Functional microRNA screening to improve hepatocyte formation via direct reprogramming". W 35. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0038-1677090.
Pełny tekst źródłaArnold, F., Mahaddalkar PU, W. Bergmann, J. Gout, Kraus JM, E. Roger, L. Perkhofer, T. Seufferlein, Hermann PC i A. Kleger. "Functional genomic screening during somatic cell reprogramming identifies Dkk3 as a roadblock of organ regeneration". W DGVS Digital: BEST OF DGVS. © Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1716151.
Pełny tekst źródłaImianowski, Charlotte, Paula Kuo, Edmund Poon, Matthew Lakins, Michelle Morrow i Rahul Roychoudhuri. "1069 OX40/CD137 dual agonism potentiates anti-tumour immunity by driving functional reprogramming and instability of regulatory T (Treg) cells". W SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.1069.
Pełny tekst źródłaMandula, J., Rosa Sierra-Mondragon, Darwin Chang, Rachel Jimenez, Eslam Mohamed, Jimena Trillo, Alyssa Obermayer i in. "944 Targeting of notch ligand Jagged2 in lung cancer cells drives anti-tumor immunity via notch-induced functional reprogramming of tumor-associated macrophages". W SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.0944.
Pełny tekst źródłaJensen, Helle, Rachel Fukuda, Megan Murt, Xiao Wang, Lora Zhao, Sheila Lou, Purnima Sundar i in. "232 Increased potency and functional persistencein vitroof a next-generation NY-ESO-1-specific TCR therapy incorporating Gen-R™ genetic reprogramming technology". W SITC 37th Annual Meeting (SITC 2022) Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jitc-2022-sitc2022.0232.
Pełny tekst źródłaWu, Sihan, Kai Zheng i Xin Huang. "Model Checking PV Energy System with Remote Reprogramming Function". W 2016 8th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2016. http://dx.doi.org/10.1109/itme.2016.0143.
Pełny tekst źródłaKim, Seung-Ku, Jae-Ho Lee, Kyeong Hur i Doo-Seop Eom. "Tiny Function-Linking for Energy-Efficient Reprogramming in Wireless Sensor Networks". W 2009 Third International Conference on Mobile Ubiquitous Computing, Systems, Services and Technologies (UBICOMM). IEEE, 2009. http://dx.doi.org/10.1109/ubicomm.2009.22.
Pełny tekst źródłaScherz-Shouval, Ruth, Marc L. Mendillo, Giorgio Gaglia, Irit Ben-Aharon, Andrew H. Beck, Luke Whitesell i Susan Lindquist. "Abstract PR07: Mechanisms of stromal reprogramming mediated by heat shock factor 1". W Abstracts: AACR Special Conference: The Function of Tumor Microenvironment in Cancer Progression; January 7-10, 2016; San Diego, CA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.tme16-pr07.
Pełny tekst źródłaJohnson, Kelly E., Kellie R. Machlus, Jodi A. Forward, Mason D. Tippy, Saleh A. El-Husayni, Joseph E. Italiano i Elisabeth M. Battinelli. "Abstract C12: Platelets promote breast cancer metastasis by reprogramming tumor cells to produce IL-8". W Abstracts: AACR Special Conference: The Function of Tumor Microenvironment in Cancer Progression; January 7-10, 2016; San Diego, CA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.tme16-c12.
Pełny tekst źródłaJacq, Xavier, Anamarija Jurisic, Julien Daubriac, Ian T. Lobb, Mark Wappett, Aaron Cranston, Peggy Sung, Gerald Gavory, Colin O'Dowd i Tim Harisson. "Abstract 6057: Discovery of a novel function for USP7 inhibitors: Reprogramming the tumor microenvironment". W Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-6057.
Pełny tekst źródłaRaporty organizacyjne na temat "Functional reprogramming"
Or, Etti, David Galbraith i Anne Fennell. Exploring mechanisms involved in grape bud dormancy: Large-scale analysis of expression reprogramming following controlled dormancy induction and dormancy release. United States Department of Agriculture, grudzień 2002. http://dx.doi.org/10.32747/2002.7587232.bard.
Pełny tekst źródłaMiller, Gad, i Jeffrey F. Harper. Pollen fertility and the role of ROS and Ca signaling in heat stress tolerance. United States Department of Agriculture, styczeń 2013. http://dx.doi.org/10.32747/2013.7598150.bard.
Pełny tekst źródłaBrown Horowitz, Sigal, Eric L. Davis i Axel Elling. Dissecting interactions between root-knot nematode effectors and lipid signaling involved in plant defense. United States Department of Agriculture, styczeń 2014. http://dx.doi.org/10.32747/2014.7598167.bard.
Pełny tekst źródłaSessa, Guido, i Gregory Martin. Role of GRAS Transcription Factors in Tomato Disease Resistance and Basal Defense. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7696520.bard.
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