Gotowa bibliografia na temat „Flavivirus”
Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych
Spis treści
Zobacz listy aktualnych artykułów, książek, rozpraw, streszczeń i innych źródeł naukowych na temat „Flavivirus”.
Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.
Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.
Artykuły w czasopismach na temat "Flavivirus"
Qiu, Yang, Yan-Peng Xu, Miao Wang, Meng Miao, Hui Zhou, Jiuyue Xu, Jing Kong i in. "Flavivirus induces and antagonizes antiviral RNA interference in both mammals and mosquitoes". Science Advances 6, nr 6 (luty 2020): eaax7989. http://dx.doi.org/10.1126/sciadv.aax7989.
Pełny tekst źródłaWee, Sheena, Asfa Alli-Shaik, Relus Kek, Hannah L. F. Swa, Wei-Ping Tien, Vanessa W. Lim, Yee-Sin Leo, Lee-Ching Ng, Hapuarachchige C. Hapuarachchi i Jayantha Gunaratne. "Multiplex targeted mass spectrometry assay for one-shot flavivirus diagnosis". Proceedings of the National Academy of Sciences 116, nr 14 (18.03.2019): 6754–59. http://dx.doi.org/10.1073/pnas.1817867116.
Pełny tekst źródłavan den Elsen, Kaïn, Jun Ping Quek i Dahai Luo. "Molecular Insights into the Flavivirus Replication Complex". Viruses 13, nr 6 (21.05.2021): 956. http://dx.doi.org/10.3390/v13060956.
Pełny tekst źródłaWu, Bingan, Zhongtian Qi i Xijing Qian. "Recent Advancements in Mosquito-Borne Flavivirus Vaccine Development". Viruses 15, nr 4 (23.03.2023): 813. http://dx.doi.org/10.3390/v15040813.
Pełny tekst źródłaHeinz, Franz X., i Karin Stiasny. "Flaviviruses and flavivirus vaccines". Vaccine 30, nr 29 (czerwiec 2012): 4301–6. http://dx.doi.org/10.1016/j.vaccine.2011.09.114.
Pełny tekst źródłaLiao, Ching-Len, Yi-Ling Lin, Bi-Ching Wu, Chang-Huei Tsao, Mei-Chuan Wang, Chiu-I. Liu, Yue-Ling Huang, Jui-Hui Chen, Jia-Pey Wang i Li-Kuang Chen. "Salicylates Inhibit Flavivirus Replication Independently of Blocking Nuclear Factor Kappa B Activation". Journal of Virology 75, nr 17 (1.09.2001): 7828–39. http://dx.doi.org/10.1128/jvi.75.17.7828-7839.2001.
Pełny tekst źródłaHabarugira, Gervais, Jasmin Moran, Jessica J. Harrison, Sally R. Isberg, Jody Hobson-Peters, Roy A. Hall i Helle Bielefeldt-Ohmann. "Evidence of Infection with Zoonotic Mosquito-Borne Flaviviruses in Saltwater Crocodiles (Crocodylus porosus) in Northern Australia". Viruses 14, nr 5 (21.05.2022): 1106. http://dx.doi.org/10.3390/v14051106.
Pełny tekst źródłaFontoura, Marina Alves, Rebeca Fróes Rocha i Rafael Elias Marques. "Neutrophil Recruitment and Participation in Severe Diseases Caused by Flavivirus Infection". Life 11, nr 7 (20.07.2021): 717. http://dx.doi.org/10.3390/life11070717.
Pełny tekst źródłaBlahove, Maria Raisa, i James Richard Carter. "Flavivirus Persistence in Wildlife Populations". Viruses 13, nr 10 (18.10.2021): 2099. http://dx.doi.org/10.3390/v13102099.
Pełny tekst źródłaGibbs, Tristan, i David J. Speers. "Neurological disease caused by flavivirus infections". Microbiology Australia 39, nr 2 (2018): 99. http://dx.doi.org/10.1071/ma18029.
Pełny tekst źródłaRozprawy doktorskie na temat "Flavivirus"
Shomiad, Shueb Rafidah Hanim. "Contribution of different components of innate and adaptive immunity to severity of flavivirus-induced encephalitis in susceptible and resistant hosts". University of Western Australia. School of Biomedical, Biomolecular and Chemical Sciences, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0199.
Pełny tekst źródłaKhou, Cécile. "Etude du neurotropisme des Flavivirus neuropathogènes". Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCC305/document.
Pełny tekst źródłaNeuropathogenic Flaviviruses, such as Japanese encephalitis virus (JEV), West Nile virus (WNV), yellow fever virus (YFV) and Zika virus (ZIKV), cause neurological diseases. These diseases are due to viral infection of central nervous system (CNS) cells. The CNS is a privileged organ, isolated from pathogenic agents by a barrier between the blood and the barrier, called the blood-brain barrier (BBB). Neuropathogenic Flaviviruses which can cross this BBB in order to reach their target cells in the CNS, are neuroinvasive. This study aims at understanding the cellular mechanisms by which YFV and JEV Flaviviruses cross the BBB and the effects of viral infection by WNV and ZIKV of the CNS cells during neocortex development.YFV is a hepatrotopic virus, which mostly infects the liver and the kidneys. The two live-attenuated vaccines against YFV, the FNV (for French Neurotropic Virus) vaccine and the 17D vaccine, were obtained empirically by several passages in suckling mouse brain of YFV virulent strains. These vaccines do not cause any disease targeting the liver or the kidneys, but can sometimes cause post-vaccine encephalitis. These encephalitis cases suggest that the vaccine strains have become neurovirulent and neuroinvasive. Due to high risks of post-vaccine encephalitis, the FNV vaccine use was discontinued in the 1980s.JEV is a neurotropic virus, causing acute encephalitis in South East Asia. To date, there is a live-attenuated vaccine against JEV, the JEV SA14-14-2 vaccine, which was obtained empirically by several passages in primary hamster kidney cells. This vaccine is less neurovirulent and less neuroinvasive than JEV virulent strains in mouse model, and it protects against JEV infections. However, some cases of post-vaccine encephalitis were reported. It thus seems that, in some cases, the vaccine strain JEV SA14-14-2 is able to cross the BBB and infect neuronal cells.The recent ZIKV epidemics in French Polynesia and South America were linked to an increase in the number of congenital malformations, rising questions regarding the capacity of a Flavivirus to induce CNS congenital malformations.In this study, we have identified cellular mechanisms involved in Flavivirus neuroinvasion and studied the effect of ZIKV and WNV infection of neuronal cells under development.To study CNS development, we have infected mouse embryos brain slices. We were able to show that ZIKV has a preferential tropism for neuronal progenitors, whereas WNV has a preferential tropism for neuronal cells. We also show that infection of neuronal progenitors by ZIKV impairs the cell life cycle, whereas no effect on the cell life cycle was observed for WNV-infected cells. Studies on apoptosis induction did not show any difference between both viruses at early time points of infection.To study Flavivirus neuroinvasion, we have used an in vitro model of BBB composed of human endothelial hCMEC/D3 cells that can form tight junctions. These cells were cultivated on Transwell inserts and placed above human neuronal cells. Using this system, we show that YFV FNV cross the BBB more efficiently than YFV 17D, suggesting that YFV FNV is more neuroinvasive than YFV 17D. This observation can explain the higher post-vaccine encephalitis risks associated with YFV FNV vaccine compared to YFV 17D vaccine. We also confirmed that JEV SA14-14-2 vaccine strain is less neuroinvasive than JEV RP9.We also examined how JEV crosses the BBB and the endothelial cell response following JEV treatment. We show that both JEV RP9 and SA14-14-2 are able to cross the BBB without infecting its endothelial cells and without disrupting the BBB. Preliminary results suggest that JEV RP9, but not JEV SA14-14-2, crosses the BBB by dynamin-dependant transcytosis. Transcriptomic analysis of endothelial cells treated by either virus show slight, but significant, differences in regulation of genes implicated in several pathways associated with CNS diseases
Lequime, Sébastian. "Interactions flavivirus-moustiques : diversité et transmission". Thesis, Paris 6, 2016. http://www.theses.fr/2016PA066081/document.
Pełny tekst źródłaFlaviviruses are RNA virus among which some are arboviruses transmitted between vertebrate hosts and arthropod vectors, like mosquitoes. The interaction with mosquitoes is key in the biology of flaviviruses because it influences their genetic diversity and transmission. However, some aspects however are still poorly understood. At the heart of the work presented in this dissertation, strategies based on ‘big data’, both by taking advantage of modern technologies and by compiling older literature, highlighted new aspects of the complex relationships between flaviviruses and mosquitoes. While exploring Anopheles mosquito genomes, we identified and characterized endogenous viral elements of flaviviral origin in Anopheles sinensis and An. minimus, which supports the existence of flaviviruses infecting Anopheles mosquitoes and highlights new aspected of their diversity. Besides, we explored, by deep sequencing, the fine-tuned interaction between genotypes of the mosquito Aedes aegypti and the intra-host diversity of dengue virus 1. Our results showed a strong effect of genetic drift during initial infection, reducing the relative importance of natural selection, and a modulation of the intra-host viral genetic diversity by the mosquito genotype. Finally, we assembled the litterature on arbovirus vertical transmission in the mosquito vector, i.e. from an infected female to her offspring, in order to identify underlying technical and biological predictors. Our results increase our understanding of this transmission mode and the strategies employed by arboviruses to persist in their environment
Lequime, Sébastian. "Interactions flavivirus-moustiques : diversité et transmission". Electronic Thesis or Diss., Paris 6, 2016. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2016PA066081.pdf.
Pełny tekst źródłaFlaviviruses are RNA virus among which some are arboviruses transmitted between vertebrate hosts and arthropod vectors, like mosquitoes. The interaction with mosquitoes is key in the biology of flaviviruses because it influences their genetic diversity and transmission. However, some aspects however are still poorly understood. At the heart of the work presented in this dissertation, strategies based on ‘big data’, both by taking advantage of modern technologies and by compiling older literature, highlighted new aspects of the complex relationships between flaviviruses and mosquitoes. While exploring Anopheles mosquito genomes, we identified and characterized endogenous viral elements of flaviviral origin in Anopheles sinensis and An. minimus, which supports the existence of flaviviruses infecting Anopheles mosquitoes and highlights new aspected of their diversity. Besides, we explored, by deep sequencing, the fine-tuned interaction between genotypes of the mosquito Aedes aegypti and the intra-host diversity of dengue virus 1. Our results showed a strong effect of genetic drift during initial infection, reducing the relative importance of natural selection, and a modulation of the intra-host viral genetic diversity by the mosquito genotype. Finally, we assembled the litterature on arbovirus vertical transmission in the mosquito vector, i.e. from an infected female to her offspring, in order to identify underlying technical and biological predictors. Our results increase our understanding of this transmission mode and the strategies employed by arboviruses to persist in their environment
Grard, Gilda. "Génomique et évolution des flavivirus transmis par les tiques et découverte d'un nouveau lignage du genre flavivirus". Aix-Marseille 2, 2006. http://www.theses.fr/2006AIX20679.
Pełny tekst źródłaGollins, S. W. "Mechanisms of flavivirus neutralization and cellular infection". Thesis, University of Oxford, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.355752.
Pełny tekst źródłaDayaraj, Cecilia. "Molecular and immunological studies on flavivirus virulence". Thesis, University of Oxford, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.279888.
Pełny tekst źródłaCarney, Jennifer. "Viral Determinants of Flavivirus Neurotropism in Humans". Thesis, University of Liverpool, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.526956.
Pełny tekst źródłaPacca, Carolina Colombelli. "Screening de novos antivirais inibidores de flavivirus". Faculdade de Medicina de São José do Rio Preto, 2013. http://bdtd.famerp.br/handle/tede/201.
Pełny tekst źródłaCoordenação de Aperfeiçoamento de Pessoal de Nível Superior
Introduction. Arboviruses, arthropod-borne viruses, are frequently associated with human outbreaks and represent a serious health problem. The genus Flavivirus, which includes both the Yellow Fever Virus (YFV) and Saint Louis Encephalitis Virus (SLEV), are important pathogens that result in high morbidity and mortality rates worldwide. In Brazil, YFV has a sylvatic cycle and occurs annually, despite the efficiency of the vaccine. Saint Louis Encephalitis is an infectious illness that can cause acute fever caused by SLEV, which is widely distributed in the Americas. The emergence of SLEV became a serious concern after the first related outbreak in Brazil in 2006, in the city of Sao Jose do Rio Preto. There is no specific antiviral drug for these viruses, only supporting treatment that can alleviate the symptoms and prevent complications. The need to develop effective and safe antiviral drugs is indispensable for the treatment of these infections. Objective. The aim of this work was to identify new possible antiviral drugs against the arboviruses that can cause acute fever and encephalitis (YFV and SLEV) and to evaluate the capacity of inhibition of these compounds in ABR mice. Material and Methods. Plaque reduction assay, flow citometry, immunofluorescence and cellular viability were used to test the compounds in vitro. ABR mice were inoculated with YFV, and the biological samples were tested for the presence of the virus through the use of plaque reduction assay and qPCR. Neutralization assay was also performed. Results. Treated cells showed efficient inhibition of viral replication at concentrations that presented minimal toxicity to the cells. The assays showed that ftalyl-tiazole and fenoxytiosemicarbazone were more effective, and that they reduced viral replication by 60% and 75% for YFV and SLEV, respectively. The analysis also revealed that the ABR mice inoculated with YFV had histopathological alterations in the liver; however, the samples did not present viral title. Neutralization assay showed a high concentration of antibodies in the serum. Conclusion. The inhibitions of viral replication were confirmed through the use of some assays in vitro, and the effectiveness of the selected compounds show that they are an option in the treatment of these viruses. More detailed studies are needed to determine the mechanism of action of these molecules. The mice were found to have histopathological alterations, which indicates viral infection; however, they also presented with high concentrations of antibodies. More studies about animal models are necessary to make in vivo experiments.
Introdução: Os arbovírus, vírus transmitidos por artrópodes, são freqüentemente associadas a surtos em seres humanos e representam um problema sério de saúde pública. Os vírus pertencentes ao gênero Flavivirus, tais como vírus da Febre Amarela (YFV) e vírus da Encefalite de Saint Louis (SLEV), são importantes patógenos que podem causar alta taxa de morbidade e mortalidade no mundo. No Brasil, YFV é mantido em ciclo silvestre notificados anualmente, a despeito da segurança e eficiência da vacina. A encefalite de Saint Louis é uma doença infecciosa febril aguda causada pelo SLEV amplamente distribuída nas Américas. A emergência do SLEV passou a ser um fato preocupante no Brasil a partir da constatação do primeiro surto no país em 2006, na cidade de São Jose do Rio Preto. Não existe tratamento específico para estas viroses, somente tratamento de suporte para ajudar a aliviar os sintomas e prevenir complicações. Desta forma, há uma grande necessidade de que sejam desenvolvidos antivirais efetivos e seguros para o tratamento destas infecções. Objetivos: O objetivo deste trabalho foi identificar potenciais compostos antivirais contra os arbovírus causadores de doença febril aguda e encefalites (YFV e SLEV) in vitro e avaliar a capacidade de inibição da replicação viral dos compostos in vivo em camundongos ABR. Materiais e Métodos: Para tanto, foram realizados ensaios de redução de placas, citometria de fluxo, imunofluorescencia, bem como testes de viabilidade celular para as analises in vitro. Além disto, camundongos ABR foram inoculados com YFV e seus materiais biológicos testados para a presença de partículas virais por ensaio de redução de placas e qPCR. Adicionalmente, foi realizado ensaio de neutralização do soro dos animais. Resultados: Celulas tratadas com os compostos mostraram eficiente inibição da replicação viral em concentrações que apresentam baixa citotoxicidade. Os ensaios mostraram que derivados de ftalyl-tiazole e fenoxytiosemicarbazone foram os mais eficazes na ação antiviral, apresentando redução de 60% e 75% para YFV e SLEV, respectivamente. Camundongos ABR inoculados com YFV apresentaram alterações histológicas no fígado, entretanto, não foi constatado título viral nas amostras testadas. O ensaio de neutralização mostra altas concentrações de anticorpos no soro dos animais. Conclusões: A inibição da replicação foi comprovada por vários ensaios in vitro evidenciando as moléculas como potentes alternativas para o tratamento dos vírus. Mais estudos são necessários para a determinação do mecanismo de ação destas moléculas. Os camundongos apresentaram alterações histopatológicas sendo um indicativo de infecção, entretanto, apresentam altas taxas de anticorpos. Mais estudos sobre modelo animal são necessários para a realização de ensaios in vivo.
Singethan, Katrin. "Untersuchungen zur Inhibition Paramyxo- und Flavivirus-induzierter Membranfusion". kostenfrei, 2009. http://www.opus-bayern.de/uni-wuerzburg/volltexte/2009/3634/.
Pełny tekst źródłaKsiążki na temat "Flavivirus"
Gregory, Bock, Goode Jamie, Novartis Foundation i Novartis Institute for Tropical Diseases., red. New treatment strategies for dengue and other flaviviral diseases. Chichester: John Wiley & Sons, 2006.
Znajdź pełny tekst źródłaShi, Pei-Yong. Molecular virology and control of flaviviruses. Norfolk, UK: Caister Academic Press, 2012.
Znajdź pełny tekst źródłaM, Chambers Thomas, red. The flaviviruses. Oxford: Academic, 2004.
Znajdź pełny tekst źródłaM, Chambers Thomas, red. The flaviviruses. Oxford: Academic, 2004.
Znajdź pełny tekst źródłaChambers, Thomas J. The Flaviviruses: Detection, Diagnosis and Vaccine Development. Burlington: Elsevier, 2003.
Znajdź pełny tekst źródłaM, Chambers Thomas, Monath Aaron J, Maramorosch Karl, Murphy Frederick A i Shatkin Aaron J, red. Advances in virus research. Amsterdam: Elsevier, 2004.
Znajdź pełny tekst źródłaM, Chambers Thomas, Monath Aaron J, Maramorosch Karl, Murphy Frederick A i Shatkin Aaron J, red. Advances in virus research. Amsterdam: Oxford, 2003.
Znajdź pełny tekst źródłaBock, Gregory, i Jamie Goode, red. New Treatment Strategies for Dengue and Other Flaviviral Diseases. Chichester, UK: John Wiley & Sons, Ltd, 2006. http://dx.doi.org/10.1002/0470058005.
Pełny tekst źródłaRuzek, Daniel, red. Flavivirus Encephalitis. InTech, 2011. http://dx.doi.org/10.5772/847.
Pełny tekst źródłaSchlesinger, Milton J., i Sondra Schlesinger. Togaviridae and Flaviviridae. Springer, 2012.
Znajdź pełny tekst źródłaCzęści książek na temat "Flavivirus"
Contigiani, Marta S., Luis A. Diaz i Lorena Spinsanti. "Flavivirus". W Arthropod Borne Diseases, 73–88. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-13884-8_6.
Pełny tekst źródłaHarnett, Gerald B., i Julia A. Cattell. "Flavivirus". W PCR for Clinical Microbiology, 241–44. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-9039-3_34.
Pełny tekst źródłaGooch, Jan W. "Flavivirus". W Encyclopedic Dictionary of Polymers, 893. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-6247-8_13767.
Pełny tekst źródłaWestaway, Edwin G. "Flavivirus‡". W The Springer Index of Viruses, 461–71. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-95919-1_67.
Pełny tekst źródłaReid, Hugh W., Herbert Weissenböck i Károly Erdélyi. "Flavivirus Infections". W Infectious Diseases of Wild Mammals and Birds in Europe, 128–45. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118342442.ch9.
Pełny tekst źródłaYu, Yufeng, Lulu Si i Yu Meng. "Flavivirus Entry Inhibitors". W Advances in Experimental Medicine and Biology, 171–97. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-8702-0_11.
Pełny tekst źródłaRice, Charles M., Ellen G. Strauss i James H. Strauss. "Structure of the Flavivirus Genome". W The Togaviridae and Flaviviridae, 279–326. Boston, MA: Springer New York, 1986. http://dx.doi.org/10.1007/978-1-4757-0785-4_10.
Pełny tekst źródłaColombarolli, Stella Garcia, Izabella Cristina Andrade Batista, Naiara Clemente Tavares, Eneida Santos de Oliveira, Camila Sales Nascimento, Philip Louis Felgner, Rafael Ramiro de Assis i Carlos Eduardo Calzavara-Silva. "Peptide Microarrays for Flavivirus Diagnosis". W Methods in Molecular Biology, 199–208. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2732-7_14.
Pełny tekst źródłaPadmanabhan, R., N. Mueller, E. Reichert, C. Yon, T. Teramoto, Y. Kono, R. Takhampunya i in. "Multiple Enzyme Activities of Flavivirus Proteins". W Novartis Foundation Symposia, 74–86. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470058005.ch6.
Pełny tekst źródłaKümmerer, Beate M. "Establishment and Application of Flavivirus Replicons". W Advances in Experimental Medicine and Biology, 165–73. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8727-1_12.
Pełny tekst źródłaStreszczenia konferencji na temat "Flavivirus"
Silva, Stephanie, Barbara Santos, Mariana Gomes, Ygara Mendes, Renata Pereira, Tiago Santos, Samir Campos, Vanessa Santos, Noemi Gardinali i Sheila Lima. "Interference of EDTA on Flavivirus infectivity". W International Symposium on Immunobiologicals. Instituto de Tecnologia em Imunobiológicos, 2023. http://dx.doi.org/10.35259/isi.2023_58027.
Pełny tekst źródłaChaley, M. B., Zh S. Tyulko i V. A. Kutyrkin. "Specifics of Coding Sequences in the Flavivirus Genomes". W Mathematical Biology and Bioinformatics. Pushchino: IMPB RAS - Branch of KIAM RAS, 2018. http://dx.doi.org/10.17537/icmbb18.10.
Pełny tekst źródła"Recognition of flavivirus species on the base of coding genome sequences". W Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-091.
Pełny tekst źródłaChaley, M. B., Zh S. Tyulko i V. A. Kutyrkin. "Fast Method to Recognize Flavivirus Species after Sequencing the Viral Genome". W Mathematical Biology and Bioinformatics. Pushchino: IMPB RAS - Branch of KIAM RAS, 2020. http://dx.doi.org/10.17537/icmbb20.12.
Pełny tekst źródławoo Kim, Chan, Se Hwan Ahn i Taeseon Yoon. "Comparison of flavivirus using datamining-Apriori, K-means, and decision tree algorithm". W 2017 19th International Conference on Advanced Communication Technology (ICACT). IEEE, 2017. http://dx.doi.org/10.23919/icact.2017.7890130.
Pełny tekst źródłaSantos, Franciellen Machado dos, VIVIANE HORN DE MELO, AMANDA PELLENZ RUIVO, FERNANDA M. S. GODINHO i RICHARD STEINER SALVATO. "DETECÇÃO MOLECULAR DE ALPHAVIRUS E FLAVIVIRUS EM PACIENTES DO RIO GRANDE DO SUL". W IV Congresso Nacional de Microbiologia Clínica On-line. Revista Multidisciplinar em Saúde, 2024. http://dx.doi.org/10.51161/conamic2024/30268.
Pełny tekst źródłaMartins, Raquel, Carolina Cajaraville, Fernando Conte i Márcia Arissawa. "Optimized production of monoclonal antibody used in Flavivirus immunoassays for different projects of vaccine development". W II Seminário Anual Científico e Tecnológico em Imunobiológicos. Instituto de Tecnologia em Imunobiológicos, 2014. http://dx.doi.org/10.35259/isi.sact.2014_28652.
Pełny tekst źródłaLima, Michelle, Paloma Pêgo, Gabriel Silva i Salvatore De Simone. "Identification of targeted epitopes of yellow fever virus based on homology with other species of flavivirus". W V Seminário Anual Científico e Tecnológico. Instituto de Tecnologia em Imunobiológicos, 2017. http://dx.doi.org/10.35259/isi.sact.2017_26145.
Pełny tekst źródłaRadzol, A. R. M., Khuan Y. Lee, W. Mansor i I. S. Omar. "PCA criterion for SVM (MLP) classifier for flavivirus biomarker from salivary SERS spectra at febrile stage". W 2016 38th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2016. http://dx.doi.org/10.1109/embc.2016.7592146.
Pełny tekst źródłaNandy, Ashesh, Sumanta Dey, Proyasha Roy, Subhas Basak i Sukhen Das. "Comparison of Base Distributions in Dengue, Zika and Other Flavivirus Envelope and NS5 Genes". W MOL2NET 2017, International Conference on Multidisciplinary Sciences, 3rd edition. Basel, Switzerland: MDPI, 2017. http://dx.doi.org/10.3390/mol2net-03-04966.
Pełny tekst źródłaRaporty organizacyjne na temat "Flavivirus"
Paul, Satashree. Flavivirus and its Threat. Science Repository, marzec 2021. http://dx.doi.org/10.31487/sr.blog.30.
Pełny tekst źródłaFournier, Maurille J., i Thomas L. Mason. Structure and Expression of Genes for Flavivirus Immunogens. Fort Belvoir, VA: Defense Technical Information Center, styczeń 1992. http://dx.doi.org/10.21236/ada252662.
Pełny tekst źródła