Gotowa bibliografia na temat „Fibulin-3”
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Artykuły w czasopismach na temat "Fibulin-3"
Hulleman, John D., Steven J. Brown, Hugh Rosen i Jeffery W. Kelly. "A High-Throughput Cell-Based Gaussia Luciferase Reporter Assay for Identifying Modulators of Fibulin-3 Secretion". Journal of Biomolecular Screening 18, nr 6 (10.12.2012): 647–58. http://dx.doi.org/10.1177/1087057112469405.
Pełny tekst źródłaJiang, Zhaoqiang, Shibo Ying, Wei Shen, Xianglei He, Junqiang Chen, Hailing Xia, Min Yu i in. "Plasma Fibulin-3 as a Potential Biomarker for Patients with Asbestos-Related Diseases in the Han Population". Disease Markers 2017 (2017): 1–8. http://dx.doi.org/10.1155/2017/1725354.
Pełny tekst źródłaGodyna, S., M. Diaz-Ricart i WS Argraves. "Fibulin-1 mediates platelet adhesion via a bridge of fibrinogen". Blood 88, nr 7 (1.10.1996): 2569–77. http://dx.doi.org/10.1182/blood.v88.7.2569.bloodjournal8872569.
Pełny tekst źródłaLivingstone, Imogen, Vladimir N. Uversky, Dominic Furniss i Akira Wiberg. "The Pathophysiological Significance of Fibulin-3". Biomolecules 10, nr 9 (8.09.2020): 1294. http://dx.doi.org/10.3390/biom10091294.
Pełny tekst źródłaMabotuwana, N., L. Murtha, S. Hardy i A. Boyle. "Fibulin-3 in Cardiac Fibrosis". Heart, Lung and Circulation 26 (2017): S133. http://dx.doi.org/10.1016/j.hlc.2017.06.207.
Pełny tekst źródłaKovac, Viljem, Metoda Dodic-Fikfak, Niko Arneric, Vita Dolzan i Alenka Franko. "Fibulin-3 as a biomarker of response to treatment in malignant mesothelioma". Radiology and Oncology 49, nr 3 (1.09.2015): 279–85. http://dx.doi.org/10.1515/raon-2015-0019.
Pełny tekst źródłaLuong, Trang T. D., Nadeshda Schelski, Beate Boehme, Manousos Makridakis, Antonia Vlahou, Florian Lang, Burkert Pieske, Ioana Alesutan i Jakob Voelkl. "Fibulin-3 Attenuates Phosphate-Induced Vascular Smooth Muscle Cell Calcification by Inhibition of Oxidative Stress". Cellular Physiology and Biochemistry 46, nr 4 (2018): 1305–16. http://dx.doi.org/10.1159/000489144.
Pełny tekst źródłaTian, Meiling, Jing Wang, Yuqin Wei, Qingle Lu i Baohua Huang. "Serum and vitreous fibulin-1 concentrations in patients with diabetic retinopathy". Journal of Investigative Medicine 64, nr 7 (15.07.2016): 1209–12. http://dx.doi.org/10.1136/jim-2016-000130.
Pełny tekst źródłaZhang, Youwen, i Lihua Y. Marmorstein. "Focus on molecules: Fibulin-3 (EFEMP1)". Experimental Eye Research 90, nr 3 (marzec 2010): 374–75. http://dx.doi.org/10.1016/j.exer.2009.09.018.
Pełny tekst źródłaWakabayashi, Toru, Akihiko Matsumine, Shigeto Nakazora, Masahiro Hasegawa, Takahiro Iino, Hideki Ota, Hikaru Sonoda, Akihiro Sudo i Atsumasa Uchida. "Fibulin-3 negatively regulates chondrocyte differentiation". Biochemical and Biophysical Research Communications 391, nr 1 (styczeń 2010): 1116–21. http://dx.doi.org/10.1016/j.bbrc.2009.12.034.
Pełny tekst źródłaRozprawy doktorskie na temat "Fibulin-3"
Djokic, Jelena. "Molecular properties of fibulin-3, -4 and-5". Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=114359.
Pełny tekst źródłaLa famille de protéines fibuline est composée de huit membres, se situant dans la matrice extracellulaire, associée aux fibres élastiques et aux membranes basales. Les fibres élastiques confèrent l'élasticité requise aux organes, tels que les artères majeures, les poumons et la peau. La formation des fibres élastiques requiert la coordination de nombreuses protéines. Les souris knockout ont impliqué les fibulines courtes (fibuline-3, -4, et -5) dans la formation, la stabilisation, et le fonctionnement des fibres élastiques.Les fibulines courtes recombinantes ont été exprimées par les cellules HEK293 et purifiées par chromatographie d'affinité métallique. Les trois protéines migrent en plusieurs bandes sur les gels de SDS-PAGE. Le séquençage de l'extrémité N-terminale de ces bandes a démontré que les trois protéines sont coupées dans le linker du premier domaine de facteur de croissance épidermique lié au calcium. Cet effet de plusieurs bandes est en partie causé pas la N-glycosylation et la multimérisation des fibulines courtes. Un effet similaire se produit après l'incubation des fibulines courtes avec certaines métalloprotéinases matricielles. La protéolyse de fibuline-3 est presque complètement inhibée par l'addition de doxycycline (un inhibiteur de tous les métalloprotéinases matricielles) aux cellules HEK293.Les fibroblastes dermiques humains adhèrent fortement aux fibulines courtes, ce qui a été mesuré avec la coloration des cellules au cristal violet. Les fibroblastes pulmonaires humains et les cellules musculaires lisses de veine ombilicale et d'artère ombilicale adhèrent un peu moins fort. Les fibulines courtes interagissent aussi avec l'héparine immobilisée, en la présence de calcium. Ceci suggère que les fibulines courtes peuvent adhérer au sulfate d'héparane à la surface des cellules.
Zayas-Santiago, Astrid, Samuel D. Cross, James B. Stanton, Alan D. Marmorstein i Lihua Y. Marmorstein. "Mutant Fibulin-3 Causes Proteoglycan Accumulation and Impaired Diffusion Across Bruch's Membrane". ASSOC RESEARCH VISION OPHTHALMOLOGY INC, 2017. http://hdl.handle.net/10150/624956.
Pełny tekst źródłaPapon, Marie-Amélie. "Régulation des sous-types d’hétérodimères du récepteur GABAB dans la moelle épinière en conditions de douleurs neuropathiques : rôle des protéines partenaires". Thesis, Bordeaux 2, 2009. http://www.theses.fr/2009BOR21671/document.
Pełny tekst źródłaIn the central nervous system, the inhibitory GABAB receptor is an obligate heterodimeric GPCR that requires the association between GABAB1 (B1a or B1b) and GABAB2 subunits. The heterodimeric GABAB receptor activation has a well-known antinociceptive action in acute pain but its effect appears limited in pathological states. Our hypothesis is that the GABAB activation and signaling could be altered by partner proteins, thus resulting in desinhibition processes in the spinal cord. In the present study, we investigated the role of two partner proteins overexpressed in neuropathic states which decrease GABAB activation through two different mechanisms. On the one hand, the cytosolic 14-3-3? protein induces the dissociation of the heterodimer B1b/B2. This effect occurs in post-synaptic compartments. On the other hand, fibulin-2, an extracellular matrix protein, which decreases the activation of the heterodimer B1a/B2 localized preferentially in presynaptic compartments. Anti-sens strategies (anti-14-3-3? or anti-fibulin-2 siRNA) or competing peptides specific of 14-3-3?/B1b interaction, potentiate the antinociceptive effects of GABAB agonist in an animal model of neuropathic pain. Taken together, our data suggest that GPCR oligomeric state can be modulated in vivo by endogenous partners proteins that are involved in the development and the maintenance of pain sensitization
Vukovic, Jana. "An in vitro and in vitro study on the role of the glycoprotein fibulin-3 in olfactory nerve growth and repair". University of Western Australia. School of Anatomy and Human Biology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0182.
Pełny tekst źródłaSchubert, Andrea [Verfasser], Nicolai [Akademischer Betreuer] Miosge, Anette [Akademischer Betreuer] Wiegand i Martin [Akademischer Betreuer] Oppermann. "Vergleichende histologische Untersuchungen oraler Gewebe der Wildtyp- und der DDR1-Knockout-Maus hinsichtlich ihrer Struktur und der Expression von Fibulin-3, -4 und -5 / Andrea Schubert. Gutachter: Anette Wiegand ; Martin Oppermann. Betreuer: Nicolai Miosge". Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2014. http://d-nb.info/1064404022/34.
Pełny tekst źródłaSchubert, Andrea. "Vergleichende histologische Untersuchungen oraler Gewebe der Wildtyp- und der DDR1-Knockout-Maus hinsichtlich ihrer Struktur und der Expression von Fibulin-3, -4 und -5". Doctoral thesis, 2014. http://hdl.handle.net/11858/00-1735-0000-0023-9952-4.
Pełny tekst źródłaCzęści książek na temat "Fibulin-3"
Topalovski, Mary, i Rolf A. Brekken. "The Extracellular Matrix of Tumors: A Focus on Fibronectin and Fibulin-5". W Extracellular Matrix in Tumor Biology, 1–15. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-60907-2_1.
Pełny tekst źródłaAmano, Satoshi. "Fibulin-5 Deposition in Human Skin: Decrease with Aging and UVB Exposure and Increase in Solar Elastosis". W Textbook of Aging Skin, 641–49. Berlin, Heidelberg: Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-662-47398-6_32.
Pełny tekst źródłaAmano, Satoshi. "Fibulin-5 Deposition in Human Skin: Decrease with Aging and UVB Exposure and Increase in Solar Elastosis". W Textbook of Aging Skin, 333–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-89656-2_32.
Pełny tekst źródłaAmano, Satoshi. "Fibulin-5 Deposition in Human Skin: Decrease with Aging and UVB Exposure and Increase in Solar Elastosis". W Textbook of Aging Skin, 1–9. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27814-3_32-2.
Pełny tekst źródłaJeronimo Rafael, Rodríguez-Cid, i Flores-Mariñelarena Rodrigo Rafael. "Predictive and Prognosis Factors of Clinical Utility in Mesothelioma". W Mesothelioma. IntechOpen, 2020. http://dx.doi.org/10.5772/intechopen.91769.
Pełny tekst źródłaStreszczenia konferencji na temat "Fibulin-3"
Pinelli, Valentina, Donatella Fini, Enrico Battolla, Paola Ferro, Maria Cristiana Franceschini, Vincenzo Fontana, Maria Pia Pistillo, Silvio Roncella i Pier Aldo Canessa. "Comparison between mesothelin and fibulin-3 detection in pleural malignant mesothelioma (MPM) patients". W ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa5075.
Pełny tekst źródłaEspinosa, Gabriela, Lisa Bennett, William Gardner i Jessica Wagenseil. "The Effects of Extracellular Matrix Protein Insufficiency and Treatment on the Stiffness of Arterial Smooth Muscle Cells". W ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14131.
Pełny tekst źródłaNandhu, Mohan Sobhana, Prajna Behera, Vivek Bhaskaran, Ennio Antonio Chiocca i Mariano S. Viapiano. "Abstract 3797: Validation of a novel antibody against fibulin-3 for targeted therapy of glioblastoma". W Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-3797.
Pełny tekst źródłaKoba, T., Y. Takeda, R. Narumi, M. Ito, H. Yoshimura, Y. Nojima, J. Adachi i A. Kumanogoh. "Proteomics of Serum Extracellular Vesicles Identifies a Novel COPD Biomarker, Fibulin-3 from Elastic Fibres". W American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a4346.
Pełny tekst źródłaNandhu, Mohan Sobhana, Chandra Goparaju, Sharon L. Longo, Harvey I. Pass i Mariano S. Viapiano. "Abstract 2769: Targeting malignant mesothelioma with an antibody against the tumor extracellular matrix protein fibulin-3". W Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-2769.
Pełny tekst źródłaLongo, Sharon L., Prajna Behera, Mariano S. Viapiano i Mohan Sobhana Nandhu. "Abstract 1706: Inhibition of fibulin-3 reverses macrophage polarization in glioblastoma and increases anti-tumor inflammatory responses". W Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-1706.
Pełny tekst źródłaRoshini, Arivazhagan, Chandra Goparaju, Mohan S. Nandhu, Sharon L. Longo, John A. Longo, Joan Chou, Frank A. Middleton, Harvey I. Pass i Mariano S. Viapiano. "Abstract 3954: Validation of the extracellular matrix protein fibulin-3 as a molecular target in malignant pleural mesothelioma". W Proceedings: AACR Annual Meeting 2020; April 27-28, 2020 and June 22-24, 2020; Philadelphia, PA. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/1538-7445.am2020-3954.
Pełny tekst źródłaWan, William, i Rudolph L. Gleason. "Collagen Fiber Angle Quantification of Carotid Arteries From Fibulin-5 Null Mice". W ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53685.
Pełny tekst źródłaPesatori, AC, L. Angelici, C. Favero, L. Dioni, C. Mensi, C. Bareggi, A. Palleschi i in. "1769d Combination of mirnas, mesothelin and fibulin-3 as potential biomarkers in malignant pleural mesothelioma and asbestos-exposed subjects". W 32nd Triennial Congress of the International Commission on Occupational Health (ICOH), Dublin, Ireland, 29th April to 4th May 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/oemed-2018-icohabstracts.438.
Pełny tekst źródłaHu, Bin, Paula A. Agudelo, Joshua C. Saldivar, Hosung Sim, Claire E. Dolan, Maria E. Mora, Gerard J. Nuovo, Susan E. Cole i Mariano S. Viapiano. "Abstract 2353: Fibulin-3, an extracellular matrix protein, regulates Notch signaling and promotes brain tumor cell invasion and survival". W Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2353.
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