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Artykuły w czasopismach na temat "Familial aggregation"
Houwing-Duistermaat, Jeanine J., Bert H. F. Derkx, Frits R. Rosendaal i Hans C. van Houwelingen. "Testing Familial Aggregation". Biometrics 51, nr 4 (grudzień 1995): 1292. http://dx.doi.org/10.2307/2533260.
Pełny tekst źródłaSUSSER, EZRA, i MERVYN SUSSER. "FAMILIAL AGGREGATION STUDIES". American Journal of Epidemiology 129, nr 1 (styczeń 1989): 23–30. http://dx.doi.org/10.1093/oxfordjournals.aje.a115119.
Pełny tekst źródłaGuo, S. W. "Familial Aggregation of Environmental Risk Factors and Familial Aggregation of Disease". American Journal of Epidemiology 151, nr 11 (1.06.2000): 1121–31. http://dx.doi.org/10.1093/oxfordjournals.aje.a010156.
Pełny tekst źródłaRYBICKI, BENJAMIN A, MICHAEL C IANNUZZI, MARGARET M FREDERICK, BRUCE W THOMPSON, MILTON D ROSSMAN, EDDY A BRESNITZ, MICHAEL L TERRIN i in. "Familial Aggregation of Sarcoidosis". American Journal of Respiratory and Critical Care Medicine 164, nr 11 (grudzień 2001): 2085–91. http://dx.doi.org/10.1164/ajrccm.164.11.2106001.
Pełny tekst źródłaWing, Yun-Kwok, Lei Chen, Siu-Ping Lam, Albert M. Li, Nelson L. S. Tang, Margaret H. L. Ng, Suk-Hang Cheng i in. "Familial aggregation of narcolepsy". Sleep Medicine 12, nr 10 (grudzień 2011): 947–51. http://dx.doi.org/10.1016/j.sleep.2011.05.007.
Pełny tekst źródłaHeun, Reinhard, i Sandra Hein. "Familial aggregation of depression, but no familial aggregation of individual depressive symptoms". European Psychiatry 22, nr 1 (styczeń 2007): 16–21. http://dx.doi.org/10.1016/j.eurpsy.2006.09.001.
Pełny tekst źródłaBOLTON, DEREK, FRÜHLING RIJSDIJK, THOMAS G. O'CONNOR, SEAN PERRIN i THALIA C. ELEY. "Obsessive–compulsive disorder, tics and anxiety in 6-year-old twins". Psychological Medicine 37, nr 1 (26.09.2006): 39–48. http://dx.doi.org/10.1017/s0033291706008816.
Pełny tekst źródłaKendler, Kenneth S. "Familial risk factors and the familial aggregation of psychiatric disorders". Psychological Medicine 20, nr 2 (maj 1990): 311–19. http://dx.doi.org/10.1017/s0033291700017621.
Pełny tekst źródłaShakhtarina, Svetlana Vasilevna, A. A. Danilenko i N. A. Falaleeva. "Familial Aggregation in Hodgkin’s Lymphoma". Clinical oncohematology 14, nr 2 (2021): 193–97. http://dx.doi.org/10.21320/2500-2139-2021-14-2-193-197.
Pełny tekst źródłaPage, Jessica M., Tsegaselassie Workalemahu, Nathan R. Blue, Alison Fraser, Michael W. Varner, Ware D. Branch i Robert M. Silver. "1036 Familial aggregation of stillbirth". American Journal of Obstetrics and Gynecology 224, nr 2 (luty 2021): S642—S643. http://dx.doi.org/10.1016/j.ajog.2020.12.1061.
Pełny tekst źródłaRozprawy doktorskie na temat "Familial aggregation"
Tahir, Hassaan. "Familial Aggregation of Severe Preeclampsia". Thesis, Linköpings universitet, Statistik, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-73266.
Pełny tekst źródłaPappas, Sylvie Rachelle. "The familial aggregation of agoraphobia". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape10/PQDD_0017/MQ47079.pdf.
Pełny tekst źródłaBeaujeux, Timothy Paul. "Protein aggregation in a mouse model of familial motor neurone disease". Thesis, University of Sheffield, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.427349.
Pełny tekst źródłaSeeley, John Robert. "Comorbidity between conduct disorder and major depression : phenomenology, correlates, course, and familial aggregation /". view abstract or download file of text, 2001. http://wwwlib.umi.com/cr/uoregon/fullcit?p3035576.
Pełny tekst źródła"Based on data collected from the Oregon Adolescent Depresssion Project (OADP)."--Abstract. Typescript. Includes vita and abstract. Includes bibliographical references (leaves 73-84). Also available for download via the World Wide Web; free to University of Oregon users.
Brauer, Paula Mae. "Familial aggregation of diabetes, hypertension and cardiovascular conditions in a case-control study of colorectal neoplasia". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0023/NQ49836.pdf.
Pełny tekst źródłaTisler, Andras. "Analysis of the familial aggregation of hypertension among patients with different types of kidney stone disease". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0022/MQ40818.pdf.
Pełny tekst źródłaRäsänen, Maija. "Familial aggregation and risk factors for asthma and hay fever among Finnish adolescent twins : a twin family study". Helsinki : University of Helsinki, 2000. http://ethesis.helsinki.fi/julkaisut/laa/kansa/vk/rasanen/.
Pełny tekst źródłaAndersson, Karin, M. Pokrzywa, Ingrid Dacklin i Erik Lundgren. "Inhibition of TTR aggregation-induced cell death : a new role for serum amyloid P component". Umeå universitet, Institutionen för molekylärbiologi (Medicinska fakulteten), 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-65622.
Pełny tekst źródłaEpub 2013 Feb 4.
de, Zwaan Sally Elizabeth. "The Genetics of Basal Cell Carcinoma of the Skin". Thesis, The University of Sydney, 2008. http://hdl.handle.net/2123/3878.
Pełny tekst źródłade, Zwaan Sally Elizabeth. "The Genetics of Basal Cell Carcinoma of the Skin". University of Sydney, 2008. http://hdl.handle.net/2123/3878.
Pełny tekst źródłaBCC is the commonest cancer in European-derived populations and Australia has the highest recorded incidence in the world, creating enormous individual and societal cost in management of this disease. The incidence of this cancer has been increasing internationally, with evidence of a 1 to 2% rise in incidence in Australia per year over the last two decades. The main four epidemiological risk factors for the development of BCC are ultraviolet radiation (UVR) exposure, increasing age, male sex, and inability to tan. The pattern and timing of UVR exposure is important to BCC risk, with childhood and intermittent UVR exposure both associated with an increased risk. The complex of inherited characteristics making up an individual’s ‘sun sensitivity’ is also important in determining BCC risk. Very little is known about population genetic susceptibility to BCC outside of the rare genodermatosis Gorlin syndrome. Mutations in the tumour suppressor gene patched (PTCH) are responsible for this BCC predisposition syndrome and the molecular pathway and target genes of this highly conserved pathway are well described. Derangments in this pathway occur in sporadic BCC development, and the PTCH gene is an obvious candidate to contribute to non-syndromic susceptibility to BCC. The melanocortin 1 receptor (MC1R) locus is known to be involved in pigmentary traits and the cutaneous response to UVR, and variants have been associated with skin cancer risk. Many other genes have been considered with respect to population BCC risk and include p53, HPV, GSTs, and HLAs. There is preliminary evidence for specific familial aggregation of BCC, but very little known about the causes. 56 individuals who developed BCC under the age of 40 in the year 2000 were recruited from the Skin and Cancer Foundation of Australia’s database. This represents the youngest 7 – 8% of Australians with BCC from a database that captures approximately 10% of Sydney’s BCCs. 212 of their first degree relatives were also recruited, including 89 parents and 123 siblings of these 56 probands. All subjects were interviewed with respect to their cancer history and all reports of cancer verified with histopathological reports where possible. The oldest unaffected sibling for each proband (where available) was designated as an intra-family control. All cases and control siblings filled out a questionnaire regarding their pigmentary and sun sensitivity factors and underwent a skin examination by a trained examiner. Peripheral blood was collected from these cases and controls for genotyping of PTCH. All the exons of PTCH for which mutations have been documented in Gorlin patients were amplified using PCR. PCR products were screened for mutations using dHPLC, and all detectable variants sequenced. Prevalence of BCC and SCC for the Australian population was estimated from incidence data using a novel statistical approach. Familial aggregation of BCC, SCC and MM occurred within the 56 families studied here. The majority of families with aggregation of skin cancer had a combination of SCC and BCC, however nearly one fifth of families in this study had aggregation of BCC to the exclusion of SCC or MM, suggesting that BCCspecific risk factors are also likely to be at work. Skin cancer risks for first-degree relatives of people with early onset BCC were calculated: sisters and mothers of people with early-onset BCC had a 2-fold increased risk of BCC; brothers had a 5-fold increased risk of BCC; and sisters and fathers of people with early-onset BCC had over four times the prevalence of SCC than that expected. For melanoma, the increased risk was significant for male relatives only, with a 10-fold increased risk for brothers of people with early-onset BCC and 3-fold for fathers. On skin examination of cases and controls, several phenotypic factors were significantly associated with BCC risk. These included increasing risk of BCC with having fair, easyburning skin (ie decreasing skin phototype), and with having signs of cumulative sun damage to the skin in the form of actinic keratoses. Signs reflecting the combination of pigmentary characteristics and sun exposure - in the form of arm freckling and solar lentigines - also gave subjects a significantly increased risk BCC. Constitutive red-green reflectance of the skin was associated with decreased risk of BCC, as measured by spectrophotometery. Other non-significant trends were seen that may become significant in larger studies including associations of BCC with propensity to burn, moderate tanning ability and an inability to tan. No convincing trend for risk of BCC was seen with the pigmentary variables of hair or eye colour, and a non-significant reduced risk of BCC was associated with increasing numbers of seborrhoeic keratoses. Twenty PTCH exons (exons 2, 3, 5 to 18, and 20 to 23) were screened, accounting for 97% of the coding regions with published mutations in PTCH. Nine of these 20 exons were found to harbour single nucleotide polymorphisms (SNPs), seen on dHPLC as variant melting curves and confirmed on direct sequencing. SNPs frequencies were not significantly different to published population frequencies, or to Australian general population frequencies where SNP database population data was unavailable. Assuming a Poisson distribution, and having observed no mutations in a sample of 56, we can be 97.5% confident that if there are any PTCH mutations contributing to early-onset BCC in the Australian population, then their prevalence is less than 5.1%. Overall, this study provides evidence that familial aggregation of BCC is occurring, that first-degree relatives are at increased risk of all three types of skin cancer, and that a combination of environmental and genetic risk factors are likely to be responsible. The PTCH gene is excluded as a major cause of this increased susceptibility to BCC in particular and skin cancer in general. The weaknesses of the study design are explored, the possible clinical relevance of the data is examined, and future directions for research into the genetics of basal cell carcinoma are discussed.
Książki na temat "Familial aggregation"
Tisler, Andras. Analysis of the familial aggregation of hypertension among patients with different types of kidney stone disease. Ottawa: National Library of Canada, 1998.
Znajdź pełny tekst źródłaBrauer, Paula Mae. Familial aggregation of diabetes, hypertension and cardiovascular conditions in a case-control study of colorectal neoplasia. 2000.
Znajdź pełny tekst źródłaMittal, Sajjan. Amyloidosis. Redaktorzy Patrick Davey i David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0181.
Pełny tekst źródłaOtowa, Takeshi, Roxann Roberson-Nay, Mandakh Bekhbat, Gretchen N. Neigh i John M. Hettema. Genetics of Anxiety Disorders. Redaktorzy Dennis S. Charney, Eric J. Nestler, Pamela Sklar i Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0033.
Pełny tekst źródłaSamuels, Jack, Marco A. Grados, Elizabeth Planalp i O. Joseph Bienvenu. Genetic Understanding of OCD and Spectrum Disorders. Redaktor Gail Steketee. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195376210.013.0025.
Pełny tekst źródłaUgarte-Gil, Manuel F., i Graciela S. Alarcón. History of systemic lupus erythematosus. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198739180.003.0001.
Pełny tekst źródłaFeinberg, Melanie. Everyday Adventures with Unruly Data. The MIT Press, 2022. http://dx.doi.org/10.7551/mitpress/14198.001.0001.
Pełny tekst źródłaRandhawa, Gurvaneet S., i Edwin A. Lomotan. Harnessing Big Data-Based Technologies to Improve Cancer Care. Redaktorzy David A. Chambers, Wynne E. Norton i Cynthia A. Vinson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190647421.003.0034.
Pełny tekst źródłaCzęści książek na temat "Familial aggregation"
Naj, Adam C., Yo Son Park i Terri H. Beaty. "Detecting Familial Aggregation". W Methods in Molecular Biology, 119–50. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61779-555-8_8.
Pełny tekst źródłaNaj, Adam C., i Terri H. Beaty. "Detecting Familial Aggregation". W Methods in Molecular Biology, 133–69. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7274-6_8.
Pełny tekst źródłaWang, Xiaoling, i Harold Snieder. "Familial Aggregation of Blood Pressure". W Pediatric Hypertension, 241–58. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-824-9_14.
Pełny tekst źródłaSnieder, Harold. "Familial Aggregation of Blood Pressure". W Pediatric Hypertension, 265–77. Totowa, NJ: Humana Press, 2004. http://dx.doi.org/10.1007/978-1-59259-797-0_15.
Pełny tekst źródłaWang, Xiaoling, i Harold Snieder. "Familial Aggregation of Blood Pressure". W Pediatric Hypertension, 195–209. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-490-6_14.
Pełny tekst źródłaWang, Xiaoling, i Harold Snieder. "Heritability and Familial Aggregation of Blood Pressure". W Pediatric Hypertension, 159–76. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-31107-4_14.
Pełny tekst źródłaWang, Xiaoling, i Harold Snieder. "Heritability and Familial Aggregation of Blood Pressure". W Pediatric Hypertension, 1–18. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-31420-4_14-1.
Pełny tekst źródłaNagasaka, Takamura. "Familial Amyloidotic Polyneuropathy and Transthyretin". W Protein Aggregation and Fibrillogenesis in Cerebral and Systemic Amyloid Disease, 565–607. Dordrecht: Springer Netherlands, 2012. http://dx.doi.org/10.1007/978-94-007-5416-4_21.
Pełny tekst źródłaAoki, Kunio, i Hiroshi Ogawa. "Familial Cancer Among Cancer Patients Registered at the Aichi Cancer Registry - Heterogeneity of Aggregation of Familial Cancer". W Familial Cancer Control, 119–22. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77582-6_26.
Pełny tekst źródłaMalone, Kathleen E., i Kerryn W. Reding. "Inherited Predisposition: Familial Aggregation and High Risk Genes". W Breast Cancer Epidemiology, 277–99. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-0685-4_13.
Pełny tekst źródłaStreszczenia konferencji na temat "Familial aggregation"
Taytard, Jessica, Laurence Jordan, Pascal Garrec, Camille Aupiais i Nicole Beydon. "Familial aggregation of obstructive sleep apnea syndrome based on a pediatric index case". W ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa2344.
Pełny tekst źródłaChandra, D., EJ Campbell, SI Rennard, AF Barker, ML Brantly, E. Eden, NG McElvaney i in. "Familial Aggregation of Augmentation Therapy Use in Severe Alpha-1-Antitrypsin (AAT) Deficiency." W American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3526.
Pełny tekst źródłaSchwartz, Ann G. "Abstract IA03: Lung cancer risk in African Americans: Familial aggregation and genetic susceptibility". W Abstracts: Eighth AACR Conference on The Science of Health Disparities in Racial/Ethnic Minorities and the Medically Underserved; November 13-16, 2015; Atlanta, Georgia. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7755.disp15-ia03.
Pełny tekst źródłaFang, Yixin. "An Application of a Theorem of Johnstone and Forrester to Testing for Familial Aggregation". W 2008 International Conference on Biomedical Engineering And Informatics (BMEI). IEEE, 2008. http://dx.doi.org/10.1109/bmei.2008.349.
Pełny tekst źródłaGanapati, A., GU Arunachal, S. Arya, D. Shanmugasundaram, L. Jeyaseelan, ST Kumar, S. Danda i D. Danda. "403 Study of familial aggregation of autoimmune diseases in asian indian lupus patients (PROBANDS)". W LUPUS 2017 & ACA 2017, (12th International Congress on SLE &, 7th Asian Congress on Autoimmunity). Lupus Foundation of America, 2017. http://dx.doi.org/10.1136/lupus-2017-000215.403.
Pełny tekst źródłaSamama, M., J. Conard, M. H. Horellou, G. Nguyen, Van Dreden i J. H. Soria. "ABNORMALITIES OF FIBRINOGEN AND FIBRINOLYSIS IN FAMILIAL THROMBOSIS". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643716.
Pełny tekst źródłaNagayama, R., A. Hattori, I. Fuse, T. Takeshige, S. Takizawa i A. Shibata. "PLATELET IONIZED CALCIUM MOBILIZATION (AEQUORIN METHOD) IN PATIENTS WITH PRIMARY PLATELET DYSFUNCTION". W XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644571.
Pełny tekst źródłaPal, Partha, i Rupa Banerjee. "IDDF2018-ABS-0268 Familial aggregation of inflammatory bowel disease in india: prevalence, risks and impact on disease behaviour". W International Digestive Disease Forum (IDDF) 2018, Hong Kong, 9–10 June 2018. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2018. http://dx.doi.org/10.1136/gutjnl-2018-iddfabstracts.186.
Pełny tekst źródłaSmew, Awad, Cecilia Lundholm, Lars Sävendahl, Paul Lichtenstein i Catarina Almqvist. "Familial co-aggregation of asthma and type 1 diabetes mellitus in children– a Swedish population-based cohort study". W ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.1214.
Pełny tekst źródłaMorin, Matilda, Karin Hellgren i Thomas Frisell. "FRI0369 TOWARDS MORE PRECISE ESTIMATES OF THE FAMILIAL AGGREGATION AND HERITABILITY OF ANKYLOSING SPONDYLITIS – A SWEDISH NESTED CASE-CONTROL STUDY". W Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.1949.
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