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Artykuły w czasopismach na temat "Extracellular matrix"
TANZER, M. L. "Extracellular Matrix: Extracellular Matrix Biochemty." Science 227, nr 4684 (18.01.1985): 289–90. http://dx.doi.org/10.1126/science.227.4684.289-a.
Pełny tekst źródłaB, Saberi. "Glioma and Extracellular Matrix, A Review on the Integrins as the Receptors of the Extracellular Matrix". Bioequivalence & Bioavailability International Journal 7, nr 1 (4.01.2023): 1–2. http://dx.doi.org/10.23880/beba-16000190.
Pełny tekst źródłaLabat-Robert, J., M. Bihari-Varga i L. Robert. "Extracellular matrix". FEBS Letters 268, nr 2 (1.08.1990): 386–93. http://dx.doi.org/10.1016/0014-5793(90)81291-u.
Pełny tekst źródłaBernfield, M. "Extracellular matrix". Current Opinion in Cell Biology 1, nr 5 (październik 1989): 953–55. http://dx.doi.org/10.1016/0955-0674(89)90064-1.
Pełny tekst źródłaMcDonald, John A. "Extracellular Matrix Assembly". Annual Review of Cell Biology 4, nr 1 (listopad 1988): 183–207. http://dx.doi.org/10.1146/annurev.cb.04.110188.001151.
Pełny tekst źródłaFessler, J. H., i L. I. Fessler. "Drosophila Extracellular Matrix". Annual Review of Cell Biology 5, nr 1 (listopad 1989): 309–39. http://dx.doi.org/10.1146/annurev.cb.05.110189.001521.
Pełny tekst źródłaRuoslahti, Erkki. "Brain extracellular matrix". Glycobiology 6, nr 5 (1996): 489–92. http://dx.doi.org/10.1093/glycob/6.5.489.
Pełny tekst źródłaRienks, Marieke, Anna-Pia Papageorgiou, Nikolaos G. Frangogiannis i Stephane Heymans. "Myocardial Extracellular Matrix". Circulation Research 114, nr 5 (28.02.2014): 872–88. http://dx.doi.org/10.1161/circresaha.114.302533.
Pełny tekst źródłaMueller, Andrea R., Klaus-Peter Platz, Cordula Heckert, Michaela H??usler, Olaf Guckelberger, Detlef Schuppan, Hartmut Lobeck i Peter Neuhaus. "THE EXTRACELLULAR MATRIX". Transplantation 65, nr 6 (marzec 1998): 770–76. http://dx.doi.org/10.1097/00007890-199803270-00002.
Pełny tekst źródłaAplin, J. D., L. J. Foden i A. K. Charlton. "Decidual extracellular matrix". Placenta 7, nr 5 (wrzesień 1986): 458. http://dx.doi.org/10.1016/s0143-4004(86)80062-5.
Pełny tekst źródłaRozprawy doktorskie na temat "Extracellular matrix"
Šišková, Zuzana. "Extracellular matrix and (re)myelination". [S.l. : [Groningen : s.n.] ; University Library Groningen] [Host], 2006. http://irs.ub.rug.nl/ppn/297675567.
Pełny tekst źródłaVincent-Héroux, Jonathan. "Extracellular matrix receptors in astrogliosis". Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=32608.
Pełny tekst źródłaDystroglycan et les intégrines sont deux types de récepteurs de la matrice extracellulaire (RMEC) impliqués dans l'adhésion et la migration des astrocytes. Les mécanismes moléculaires qui contrôlent leur distribution aux plates-formes de signalisation de la membrane plasmique demeurent peu étudiés. Dans la présente étude, nous définissons quatre stages d'adhésion cellulaire pour les astrocytes dans lesquels DG et les intégrines sont distribués différemment. Dans la cellule en migration, les deux récepteurs sont recrutés à l'avant, possiblement dans des îlots riches en GM1. En combinant la microscopie à fluorescence et l'usage de perturbateurs du cytosquelette dans notre modèle de lésion in vitro, nous démontrons aussi que la formation des protrusions riches en microtubules n'est pas actine-indépendante. Cependant, la formation d'actine corticale est indépendante du réseau polarisé de microtubules. Nous confirmons l'activation rapide de Cdc42 et Rac1 suite à une lésion in vitro et avons développé une lignée d'astrocytes DG-nulles pour étudier le rôle de DG dans l'activation des Rho GTPases. Nous concluons que la distribution des RMEC est étroitement régulée avec l'appareil de réorganisation cytoskelettique dans la prolifération astrocytaire.
Yi, Ming. "Extracellular matrix proteins and angiogenesis /". Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2003. http://wwwlib.umi.com/cr/ucsd/fullcit?p3091204.
Pełny tekst źródłaLee, Myeongwoo Cheung H. Tak. "Characterization of Caenorhabditis elegans extracellular matrix". Normal, Ill. Illinois State University, 1997. http://wwwlib.umi.com/cr/ilstu/fullcit?p9803726.
Pełny tekst źródłaTitle from title page screen, viewed June 5, 2006. Dissertation Committee: H. Tak Cheung (chair), Sean Arkins, Herman E. Brockman, Paul A. Garris, Brian J. Wilkinson. Includes bibliographical references (leaves 113-121) and abstract. Also available in print.
McCann, Maureen C. "Architecture of the plant extracellular matrix". Thesis, University of East Anglia, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.279709.
Pełny tekst źródłaHan, Lin Ph D. Massachusetts Institute of Technology. "Nanomechanics of cartilage extracellular matrix macromolecules". Thesis, Massachusetts Institute of Technology, 2007. http://hdl.handle.net/1721.1/42134.
Pełny tekst źródłaIncludes bibliographical references (p. 187-201).
In this thesis, the shear and self-adhesion nanomechanical properties between opposing cartilage aggrecan macromolecules were probed. In addition, nanoscale dynamic oscillatory mechanical properties of cartilage and its type II collagen network was measured. Aggrecan shear nanomechanics was assessed via microcontact printing and lateral force microscopy. Lateral force between aggrecan and the probe tip, and compression of aggrecan was simultaneously measured in 0.001 - 1.0 M NaCl aqueous solutions. Using the microsized tip (Rtip ~ 2.5 [mu]m) enabled a large assembly of ~ 103 aggrecan molecules to interact simultaneously, closely mimicking the in vivo conditions.Both electrostatic and nonelectrostatic components were identified to importantly contribute to aggrecan shear. At near physiological IS (0.1 M), significant rate dependence was observed, suggestive of visco/poroelastic interactions within the aggrecan layer. By using an aggrecan end-functionalized colloidal tip, shear of two opposing aggrecan layers was assessed in a similar fashion. Lower lateral force and a more marked rate dependence were measured compared to the shear of a single layer, due to the aggrecan inter-layer molecular interpenetration and the different local z-dependent charge density distribution. The addition of Ca2+, at physiological-like 2 mM concentration, significantly affects cartilage shear by its electrostatic screening and binding effects. Marked aggrecan self-adhesion upon separation was discovered after static compression in the presence of electrostatic repulsion in physiological-like conditions.
(cont.) Aggrecan self-adhesion increases as increasing equilibration time and bath IS. Molecular origins of the adhesion, also present in vivo, include van der Waals, hydrogen bonding, Ca2+-mediated bridging, and molecular entanglements between the glycosaminogly-can branches of aggrecan. This self-adhesion could be an important factor in protecting cartilage matrix structural integrity and function via these energy-dissipative mechanisms. The nanoscale oscillatory dynamic deformation properties of both nontreated and proteoglycan(PG)-depleted (left mostly type II collagen) calf knee cartilage disks (- 0.5 mm thick) was measured by connecting an external electronic wave generator to the AFM. A significant increase in effective stiffness E and phase lag A (deformation with respect to force) as increasing frequency for both disks suggests poro/viscoelasticity are more critical at higher frequency. The PG-depleted disk shows a more marked dependence of E and A on deformation amplitude - 2 - 100 nm, as the nanostructure and nanomechanical properties of porous collagen network are more heterogeneous without the entrapment of aggrecan motif. A unique - 23 nm banding pattern along the type II collagen fibrils was observed, which may be relative to the cartilage swelling properties and the molecular interaction between aggrecan and the collagen network. Taken together, this study provides insights into molecular-level deformation of cartilage extracellular matrix (ECM) macromolecules (e.g., aggrecan, type II collagen) that are important to the understanding of cartilage biomechanical function. Ongoing studies are probing the age, disease (osteoarthritis), source and species related variations of cartilage ECM properties at the molecular level.
by Lin Han.
Ph.D.
Baig, Shabnam Mobeen. "The extracellular matrix in Alzheimer's disease". Thesis, University of Bristol, 2006. http://hdl.handle.net/1983/f7eb48ef-9368-4f28-ab12-738fb9e517b5.
Pełny tekst źródłaThurstan, Sarah Ashley. "Acellular mechanisms of extracellular matrix degradation". Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/acellular-mechanisms-of-extracellular-matrix-degradation(7fae308d-8e54-4da6-9c27-4da89ec55ab1).html.
Pełny tekst źródłaLink, Patrick. "ELECTROSPRAYING EXTRACELLULAR MATRIX TO FORM NANOPARTICLES". VCU Scholars Compass, 2017. https://scholarscompass.vcu.edu/etd/5166.
Pełny tekst źródłaTissot, Floriane. "Rôle de CD98hc dans les fibroblastes dermiques au cours de l’homéostasie et de la tumorigenèse cutanées". Thesis, Université Côte d'Azur (ComUE), 2017. http://www.theses.fr/2017AZUR4129.
Pełny tekst źródłaThe epithelial/mesenchymal interaction is crucial for many physiopathological processes. During my PhD, I focused on mesenchymal signals that regulate epithelial cells behavior using the skin as model. The skin is composed of 2 main compartments: the epidermis (epithelium) and the dermis (mesenchyme). While this crosstalk involves integrins, its regulations are poorly understood. The transmembrane protein CD98hc interacts with amino acid transporter and regulates integrin signaling. CD98hc which is expressed at the cell membrane of proliferative cells, specifically epithelial cells, is required for tissue homeostasis. We found that besides its expression in keratinocytes, CD98hc is also expressed in post-mitotic dermal fibroblast. Hence, I hypothesized that CD98hc is involved in epidermis/dermis crosstalk. Using a conditional KO mouse model that harbor a CD98hc deletion in dermal fibroblast, I have shown that dermal CD98hc is required to maintain mechanical and biochemical properties of the dermis. Moreover, I have shown that those CD98hc-dependent dermal properties are implicated in the regulation of the epidermal cell behavior during homeostasis, cutaneous barrier disruption and tumorigenesis. Moreover, the role of CD98hc in those processes seems to be age-related. To conclude, during my PhD, I have revealed a major role of CD98hc in the maintenance of skin homeostasis during aging and tumorigenesis
Książki na temat "Extracellular matrix"
Leach, Jennie B., i Elizabeth M. Powell, red. Extracellular Matrix. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2083-9.
Pełny tekst źródłaD, Comper Wayne, red. Extracellular matrix. Amsterdam: Harwood Academic Publishers, 1996.
Znajdź pełny tekst źródłaA, Haralson M., i Hassell John R, red. Extracellular matrix: A practical approach. Oxford: IRL Press, 1995.
Znajdź pełny tekst źródłaStreuli, Charles, i Michael Grant. Extracellular Matrix Protocols. New Jersey: Humana Press, 2000. http://dx.doi.org/10.1385/1592590632.
Pełny tekst źródłaEven-Ram, Sharona, i Vira Artym, red. Extracellular Matrix Protocols. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-413-1.
Pełny tekst źródłaYamaoka, Tetsuji, i Takashi Hoshiba, red. Decellularized Extracellular Matrix. Cambridge: Royal Society of Chemistry, 2019. http://dx.doi.org/10.1039/9781788015998.
Pełny tekst źródłaVigetti, Davide, i Achilleas D. Theocharis, red. The Extracellular Matrix. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9133-4.
Pełny tekst źródłaSchmuck, Eric G., Peiman Hematti i Amish N. Raval, red. Cardiac Extracellular Matrix. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-97421-7.
Pełny tekst źródłaParks, William C., i Robert P. Mecham, red. Extracellular Matrix Degradation. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16861-1.
Pełny tekst źródłaRicard-Blum, Sylvie, red. Extracellular Matrix Omics. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-58330-9.
Pełny tekst źródłaCzęści książek na temat "Extracellular matrix"
Huang, Zhen. "Immunohistochemical Characterization of Brain Neural and Vascular Basement Membranes". W Extracellular Matrix, 3–11. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2083-9_1.
Pełny tekst źródłaBalasubramanian, Swarnalatha, Elizabeth M. Powell i Jennie B. Leach. "Investigating Cell-ECM Interactions and ECM Synthesis in Three-Dimensional Hydrogels". W Extracellular Matrix, 101–9. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2083-9_10.
Pełny tekst źródłaSinyuk, Maksim, Justin D. Lathia i Mariano S. Viapiano. "Characterization and Analysis of Extracellular Matrix in Malignant Brain Tumors and Their Cellular Derivatives". W Extracellular Matrix, 113–38. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2083-9_11.
Pełny tekst źródłaHu, Jianli, Gabrielle M. Curinga i George M. Smith. "Chondroitinase Gene Therapy for Spinal Cord Injury". W Extracellular Matrix, 139–49. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2083-9_12.
Pełny tekst źródłaHennen, Eva, i Andreas Faissner. "Modulation of Neural Stem Cell Expressed Extracellular Matrix (ECM) by Targeting Glycosyltransferases". W Extracellular Matrix, 151–60. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2083-9_13.
Pełny tekst źródłaAddington, Caroline P., Christine Pauken i Sarah E. Stabenfeldt. "Evaluating the Spatial and Temporal Protein Production in Neural Tissue Engineering Constructs In Vitro". W Extracellular Matrix, 163–79. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2083-9_14.
Pełny tekst źródłaZuidema, Jonathan M., María C. Hyzinski-García, Alexander A. Mongin i Ryan J. Gilbert. "Cultivation and Imaging of Astrocytes on Protein-Coated Fluorescent Topographies Constructed from Aligned PLLA Electrospun Fibers". W Extracellular Matrix, 181–95. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2083-9_15.
Pełny tekst źródłaXu, Hui, i Sarah C. Heilshorn. "Engineered Microdevices to Study and Manipulate Neural Stem Cell Chemotaxis". W Extracellular Matrix, 197–209. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2083-9_16.
Pełny tekst źródłaZustiak, Silviya Petrova. "Hydrolytically Degradable Polyethylene Glycol (PEG) Hydrogel: Synthesis, Gel Formation, and Characterization". W Extracellular Matrix, 211–26. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2083-9_17.
Pełny tekst źródłaLee, Young il, i Yue Li. "The Use of Synaptic Basal Lamina and Its Components to Identify Sites of Recent Morphological Alterations at Mammalian Neuromuscular Junctions". W Extracellular Matrix, 13–22. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2083-9_2.
Pełny tekst źródłaStreszczenia konferencji na temat "Extracellular matrix"
"Extracellular matrix in experimental hepatocarcinoma-29". W Bioinformatics of Genome Regulation and Structure/Systems Biology (BGRS/SB-2022) :. Institute of Cytology and Genetics, the Siberian Branch of the Russian Academy of Sciences, 2022. http://dx.doi.org/10.18699/sbb-2022-467.
Pełny tekst źródłaFreytes, Donald O., Samuel Kolman, Sachin S. Velankar i Stephen F. Badylak. "Rheological Properties of Extracellular Matrix Derived Gels". W ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176537.
Pełny tekst źródłaJoglekar, M. M., M. L. Koloko Ngassie, N. J. Bekker, M. A. Reinders-Luinge, T. Borghuis, J. M. Vonk, S. D. Pouwels i in. "COPD-associated changes in lung extracellular matrix". W ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.2202.
Pełny tekst źródłaJanuskevicius, Andrius, Ieva Janulaityte, Reinoud Gosens, Virginija Kalinauskaite-Zukauske, Rokas Stonkus i Kestutis Malakauskas. "Eosinophils contribute to extracellular matrix remodeling by enhancing pulmonary fibroblasts proliferation, differentiation and extracellular matrix proteins production in asthma". W ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa968.
Pełny tekst źródłaShreim, Samir, Maxwell Kotlarchyk i Elliot Botvinick. "Microrheology of the Endothelial Glycocalyx and Extracellular Matrix". W Optical Trapping Applications. Washington, D.C.: OSA, 2009. http://dx.doi.org/10.1364/ota.2009.oma1.
Pełny tekst źródłaQi, Yun, Kang-De Yao i Yuan-Lu Cui. "Biomimetic Fibrous Extracellular Matrix for Cartilage Tissue Engineering". W 2009 3rd International Conference on Bioinformatics and Biomedical Engineering (iCBBE). IEEE, 2009. http://dx.doi.org/10.1109/icbbe.2009.5162455.
Pełny tekst źródłaManni, ML, JM Tobolewski, TW Gilbert i TD Oury. "Extracellular Matrix Powder Attenuates Bleomycin-Induced Pulmonary Fibrosis." W American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a2015.
Pełny tekst źródłaLiu, A., H. Sun, A. Ledesma-Mendoza, J. Winkler, B. Reeves, M. Gulati, W. Wang, T. Lam i E. Herzog. "Sarcoidosis Extracellular Matrix Regulation of Immune Cell Phenotype". W American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5530.
Pełny tekst źródłaNarciso, M., A. Ulldemolins, D. Navajas, R. Farre, N. Gavara i I. Almendros Lopez. "Aging Induces Stiffening of the Lung Extracellular Matrix". W ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.3463.
Pełny tekst źródłaTsiklauri, L., J. Werner, K. Frommer, S. Rehart, S. Wenisch, U. Müller-Ladner i E. Neumann. "P123 Extracellular matrix attenuated matrix-degrading effects of visfatin during adipogenic MSC differentiation". W 39th European Workshop for Rheumatology Research, 28 February–2 March 2019, Lyon, France. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2019.111.
Pełny tekst źródłaRaporty organizacyjne na temat "Extracellular matrix"
Quaranta, Vito. Extracellular Matrix in Breast Cancer Invasion. Fort Belvoir, VA: Defense Technical Information Center, czerwiec 2001. http://dx.doi.org/10.21236/ada398487.
Pełny tekst źródłaHan, Xiaoxing. Quantitative In Vivo Imaging of Breast Tumor Extracellular Matrix. Fort Belvoir, VA: Defense Technical Information Center, maj 2010. http://dx.doi.org/10.21236/ada541944.
Pełny tekst źródłaHan, Xiaoxing. Quantitative In Vivo Imaging of Breast Tumor Extracellular Matrix. Fort Belvoir, VA: Defense Technical Information Center, maj 2009. http://dx.doi.org/10.21236/ada549531.
Pełny tekst źródłaHan, Xiaoxing. Quantitative In Vivo Imaging of Breast Tumor Extracellular Matrix. Fort Belvoir, VA: Defense Technical Information Center, maj 2011. http://dx.doi.org/10.21236/ada552848.
Pełny tekst źródłaPines, Mark, Arieh Bar, David A. Carrino, Arnold I. Caplan i James A. Dennis. Extracellular Matrix Molecules of the Eggshell as Related to Eggshell Quality. United States Department of Agriculture, 1997. http://dx.doi.org/10.32747/1997.7575270.bard.
Pełny tekst źródłaNovaro, Virginia. Extracellular Matrix Regulation of Estrogen Receptors in Mouse Mammary Cells. Fort Belvoir, VA: Defense Technical Information Center, październik 2002. http://dx.doi.org/10.21236/ada411451.
Pełny tekst źródłaShan, Bin. Deregulated miRNA in Mammary Epithelium by Tumor Promoting Extracellular Matrix. Fort Belvoir, VA: Defense Technical Information Center, sierpień 2010. http://dx.doi.org/10.21236/ada587722.
Pełny tekst źródłaChan, Huei-Mei. Mechanism of Abnormal Cell-Extracellular Matrix Interactions in Human Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 1998. http://dx.doi.org/10.21236/ada367382.
Pełny tekst źródłaChen, Huei-Mei. Mechanism of Abnormal Cell-Extracellular Matrix Interactions in Human Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, październik 1997. http://dx.doi.org/10.21236/ada340571.
Pełny tekst źródłaChen, Huei-Mei. Mechanisms of Abnormal Cell-Extracellular Matrix Interactions in Human Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 1999. http://dx.doi.org/10.21236/ada381192.
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