Gotowe bibliografie tematyczne / Estrogenization

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Gotowa bibliografia na temat „Estrogenization”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 15 lutego 2022

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Artykuły w czasopismach na temat "Estrogenization"

Santti, Risto, Sari Mäkelä, Liisa Pylkkänen, Retha R. Newbold i John A. McLachlan. "Developmental estrogenization and prostatic neoplasia". Prostate 24, nr 2 (luty 1994): 67–78. http://dx.doi.org/10.1002/pros.2990240204.

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LONGHURST, PENELOPE A. "In Vitro Rat Bladder Function After Neonatal Estrogenization". Journal of Urology 168, nr 6 (grudzień 2002): 2695–99. http://dx.doi.org/10.1016/s0022-5347(05)64246-2.

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Zurawa-Janicka, Dorota, Jaroslaw Kobiela, Tomasz Stefaniak, Agnieszka Wozniak, Joanna Narkiewicz, Michał Wozniak, Janusz Limon i Barbara Lipinska. "Changes in expression of serine proteases HtrA1 and HtrA2 during estrogen-induced oxidative stress and nephrocarcinogenesis in male Syrian hamster." Acta Biochimica Polonica 55, nr 1 (30.01.2008): 9–20. http://dx.doi.org/10.18388/abp.2008_3123.

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Serine proteases HtrA1 and HtrA2 are involved in cellular stress response and development of several diseases, including cancer. Our aim was to examine the involvement of the HtrA proteins in acute oxidative stress response induced in hamster kidney by estrogen treatment, and in nephrocarcinogenesis caused by prolonged estrogenization of male Syrian hamster. We used semi-quantitative RT-PCR to estimate the HtrA1 and HtrA2 mRNA levels in kidney tissues, and Western blotting to monitor the amount of the HtrA proteins. Within the first five hours following estrogen administration both HtrA1 mRNA and the protein levels were increased significantly. No changes in the expression of HtrA2 were observed. This indicates that HtrA1 may be involved in the response against oxidative stress induced by estrogen treatment in hamster kidney. During prolonged estrogenization, a significant reduction of the HtrA1 mRNA and protein levels was observed after 6 months of estradiol treatment, while the expression of HtrA2 was significantly elevated starting from the third month. This suggests an involvement of the HtrA proteins in estrogen-induced nephrocarcinogenesis in hamster. Using fluorescence in situ hybridization we localized the HtrA1 gene at the qb3-4 region of Syrian hamster chromosome 2, the region known to undergo a nonrandom deletion upon prolonged estrogenization. It is possible that the reduced level of HtrA1 expression is due to this chromosomal aberration. A full-length cDNA sequence of the hamster HtrA1 gene was obtained. It codes for a 50 kDa protein which has 98 and 96% identity with mouse and human counterparts, respectively.

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Saffarini, Camelia M., Elizabeth V. McDonnell-Clark, Ali Amin i Kim Boekelheide. "A Human Fetal Prostate Xenograft Model of Developmental Estrogenization". International Journal of Toxicology 34, nr 2 (29.01.2015): 119–28. http://dx.doi.org/10.1177/1091581815569364.

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Prostate cancer is a common disease in older men. Rodent models have demonstrated that an early and later-life exposure to estrogen can lead to cancerous lesions and implicated hormonal dysregulation as an avenue for developing future prostate neoplasia. This study utilizes a human fetal prostate xenograft model to study the role of estrogen in the progression of human disease. Histopathological lesions were assessed in 7-, 30-, 90-, 200-, and 400-day human prostate xenografts. Gene expression for cell cycle, tumor suppressors, and apoptosis-related genes (ie, CDKN1A, CASP9, ESR2, PTEN, and TP53) was performed for 200-day estrogen-treated xenografts. Glandular hyperplasia was observed in xenografts given both an initial and secondary exposure to estradiol in both 200- and 400-day xenografts. Persistent estrogenic effects were verified using immunohistochemical markers for cytokeratin 10, p63, and estrogen receptor α. This model provides data on the histopathological state of the human prostate following estrogenic treatment, which can be utilized in understanding the complicated pathology associated with prostatic disease and early and later-life estrogenic exposures.

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Kopylova, I. V., i M. A. Kareva. "Risk factors of impaired fertility in the patients presenting with congenital adrenal cortical hyperplasia". Problems of Endocrinology 59, nr 3 (15.06.2013): 51–56. http://dx.doi.org/10.14341/probl201359351-56.

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The analysis of publications concerning the impairment of fertility in the patients presenting with congenital adrenal cortical hyperplasia was focused on the reduction of the degree of estrogenization. It included the data on the course of sexual development and the outcomes of feminizing plastic procedures in the girls suffering this disease depending on the degree of its compensation and the date of onset of the treatment.

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Lehtimaki, Jyrki, Sari Makela, Jaakko Viljamaa, Ahmed Yagi, Jorma Paranko i Risto Santti. "Neonatal Estrogenization of the Male Mouse Results in Urethral Dysfunction". Journal of Urology 156, nr 6 (grudzień 1996): 2098–103. http://dx.doi.org/10.1016/s0022-5347(01)65443-0.

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Aceitero, J., F. Gaytan i F. B. Ranz. "Effects of neonatal estrogenization on rat bone development: A histomorphometric study". Calcified Tissue International 40, nr 4 (lipiec 1987): 189–93. http://dx.doi.org/10.1007/bf02556620.

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MORALES-MONTOR, J., I. ARRIETA, L. I. DEL CASTILLO, M. RODRÍGUEZ-DORANTES, M. A. CERBÓN i C. LARRALDE. "Remote sensing of intraperitoneal parasitism by the host's brain: regional changes of c-fos gene expression in the brain of feminized cysticercotic male mice". Parasitology 128, nr 3 (marzec 2004): 343–51. http://dx.doi.org/10.1017/s0031182003004529.

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Experimental intraperitoneal Taenia crassiceps cysticercosis in mice exhibits distinct genetical, immunological and endocrinological features possibly resulting from the complex interactive network of their physiological systems. Very notable is the tendency of parasites to grow faster in hosts of the female sex. It is also remarkable in the feminization process that the infection induces in chronically infected male mice, characterized by their estrogenization, deandrogenization and loss of sexual and aggressive patterns of behaviour. The proto-oncogene c-fos is a sex steroid-regulated transcription factor gene, expressed basally and upon stimulation by many organisms. In the CNS of rodents, c-fos is found expressed in association to sexual stimulation and to various immunological and stressful events. Hence, we suspected that changes in c-fos expression in the brain could be involved in the feminization of the infected male mice. Indeed, it was found that c-fos expression increased at different times during infection in the hypothalamus, hippocampus, less so in the preoptic area and cortex, and not in several other organs. The significant and distinctive regional changes of c-fos in the CNS of infected mice indicate that the brain of the host senses intraperitoneal cysticercosis and may also announce its active participation in the regulation of the host–parasite relationship. Possibly, the host's CNS activity is involved in the network that regulates the estrogenization and deandrogenization observed in the chronically infected male mice, as well as in the behavioural and immunological peculiarities observed in this parasitic infection.

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Prins, Gail S. "Developmental estrogenization: Prostate gland reprogramming leads to increased disease risk with aging". Differentiation 118 (marzec 2021): 72–81. http://dx.doi.org/10.1016/j.diff.2020.12.001.

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Soto, Pedro, Hilda María Echevarría, Cristina Esther Monteavaro i María del Carmen Catena. "Experimentally induced intravaginal Tritrichomonas foetus infection in a mouse model". Pesquisa Veterinária Brasileira 25, nr 4 (grudzień 2005): 225–30. http://dx.doi.org/10.1590/s0100-736x2005000400007.

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The interest to develop research on the host-parasite relationship in bovine tritrichomonosis has accomplished the use of experimental models alternative to cattle. The BALB/c mouse became the most appropriate species susceptible to vaginal Tritrichomonas foetus infection requiring previous estrogenization. For the need of an experimental model without persistent estrogenization and with normal estrous cycles, the establishment and persistence of vaginal infection on BALB/c mouse with different concentrations of T. foetus in two experimental groups was evaluated. Group A was treated with 5mg of b-estradiol 3-benzoate to synchronize the estrous, 48 hours before the T. foetus vaginal inoculation, and Group B was inoculated in natural estrus. At 5-7 days after treatment, estrogenic effect decreased allowing all animals to cycle regularly during the experiment. From the first week post-infection, samples of vaginal mucus were taken from all animals during 34 weeks, in order to evaluate the course of infection and the stage of the estrus cycle. Group A showed 93.6% of infected animals, and Group B showed 38%. Different doses of T. foetus were assayed to establish the vaginal infection, with a persistence of 34 weeks. Although different behavior was observed in each subgroup belonging to either Group A or Group B, there were no significant differences among the infecting doses used. The b-estradiol 3-benzoate treatment had a favorable effect on the establishment of the infection (P<0.0001), but it did not influence its persistence (P=0.1097). According to the results, an experimental mouse model is presented, appropriate for further studies on mechanisms of pathogenicity, immune response, protective evaluation of immunogen and therapeutic effect of drugs.

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Rozprawy doktorskie na temat "Estrogenization"

Ponza, Pattareeya, i pattareeya pon@biotec or th. "Molecular markers of ecotoxicological interest in the rainbowfish Melanotaenia fluviatilis". RMIT University. Applied Science, 2007. http://adt.lib.rmit.edu.au/adt/public/adt-VIT20080102.121231.

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The Crimson-spotted rainbowfish (Melanotaenia fluviatilis) from the Murray-Darling basin of Australia is a common indicator species in Australian ecotoxicology. Biochemical changes have been investigated in this species, but not molecular markers of ecotoxicological interest. In this study genes of M. fluviatilis were isolated using a cDNA library and sequences analysed. Of 345 randomly selected clones, 94 shared similarity with 26 different genes in other organisms in public databases. Amongst these, reproductive genes coding for vitellogenin, retinol binding protein, sialyltransferase and zona pellucida protein were considered of interest in ecotoxicology. The vitellogenin gene was selected for study as it has been widely used as a molecular marker of exposure to 17â-estradiol (E2) in teleosts. Gene expression was examined via northern blot, RT-PCR and Real-Time PCR relative to the housekeeping gene (18S rRNA). The expression of vitellogenin mRNA was observed a t 12 hours post-exposure, peaked at 48 hours according to northern blot analysis; and cleared within 4 days, partly consistent with RT-PCR. However, Real-time PCR yielded an inconclusive result, probably due to differences between pooled and individual samples. Vitellogenin in blood plasma was confirmed by western blot, found to be significantly increased and retained in the plasma in fish treated with E2 compared to controls. It was concluded that vitellogenin mRNA is a molecular marker of exposure to 17â-estradiol in the rainbowfish, and could potentially be used as a marker of exposure to environmental estrogenic chemicals. Further investigations of the expression of genes in the cDNA library, could establish other molecular markers of ecotoxicological interest in M. fluviatilis.

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