Gotowa bibliografia na temat „Epigentics”
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Artykuły w czasopismach na temat "Epigentics"
Ren, Jun, i Yingmei Zhang. "Emerging Therapeutic Potential Targeting Genetics and Epigentics in Heart Failure". Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease 1863, nr 8 (sierpień 2017): 1867–69. http://dx.doi.org/10.1016/j.bbadis.2017.05.023.
Pełny tekst źródłaPisarska, M. D., G. M. Barlow, N. Xu, M. O. Goodarzi, V. Funari i J. Williams. "Special research presentation: epigentic profiles in ART pregnancies". Fertility and Sterility 102, nr 3 (wrzesień 2014): e105-e106. http://dx.doi.org/10.1016/j.fertnstert.2014.07.362.
Pełny tekst źródłaKhan, Muhammad Babar, Julia R. Schneider, Kevin Kwan i John A. Boockvar. "Epigentic Regulators of Glioma Stem Cells are Potential Therapeutic Targets". Neurosurgery 82, nr 5 (16.04.2018): E104—E105. http://dx.doi.org/10.1093/neuros/nyy039.
Pełny tekst źródłaMehboob, Riffat. "Role of Epigenetic alterations in the development of cancers". Pakistan BioMedical Journal 5, nr 2 (28.02.2022): 01. http://dx.doi.org/10.54393/pbmj.v5i2.346.
Pełny tekst źródłaMu, Shengyu, Tatsuo Shimosawa, Sayoko Ogura, Hong Wang, Yuzaburo Uetake, Fumiko Mori i Toshiro Fujita. "36 SALT-SENSITIVE HYPERTENSION AND RENAL-SYMPATHETIC TONE, EPIGENTIC MODULATION OF WNK4". Journal of Hypertension 30 (wrzesień 2012): e11. http://dx.doi.org/10.1097/01.hjh.0000419862.52153.c4.
Pełny tekst źródłaMedley, T., R. Idrizi, J. Jowett, H. Sumer, P. Verma i D. Kaye. "Cell of Origin Influence Transcriptional Epigentic and Functional Profiles of iPS Cell Derived Cardiomyocytes". Heart, Lung and Circulation 21 (styczeń 2012): S75. http://dx.doi.org/10.1016/j.hlc.2012.05.189.
Pełny tekst źródłaKim, K. J., Y. D. Min, T. B. Lee i C. H. Choi. "Epigentic mechanisms involved in the differential expression of MDR1 between gastric and colon cancer cells". Journal of Clinical Oncology 23, nr 16_suppl (czerwiec 2005): 9708. http://dx.doi.org/10.1200/jco.2005.23.16_suppl.9708.
Pełny tekst źródłaTakaro, Tim K., Meaghan Jones, Michael Kobor, Jeffrey Brook, Kathleen Mclean, Yayuk Joffres, Ryan Allen, Michael Brauer i Malcolm Sears. "Epigentic Markers Of Early Life Exposures In The Canadian Healthy Infant Longitudinal Development (Child) Birth Cohort". ISEE Conference Abstracts 2015, nr 1 (20.08.2015): 2608. http://dx.doi.org/10.1289/isee.2015.2015-2608.
Pełny tekst źródłaLecamwasam, A., B. Novakovic, B. Meyer, E. Ekinci, K. Dwyer i R. Saffery. "SAT-183 DNA METHYLATION PROFILING IDENTIFIES EPIGENTIC DIFFERENCES BETWEEN EARLY VERSUS LATE STAGES OF DIABETIC CHRONIC KIDNEY DISEASE". Kidney International Reports 5, nr 3 (marzec 2020): S78. http://dx.doi.org/10.1016/j.ekir.2020.02.195.
Pełny tekst źródłaThakur, Vikram, Brian R. Davis, Irene Sarosiek, Richard W. McCallum i Munmun Chattopadhyay. "Tu1310 ALTERATIONS IN EPIGENTIC MODIFIERS IN GASTRIC ANTRAL SMOOTH MUSCLE OF PATIENTS WITH DIABETIC GASTROPARESIS COMPARED TO IDIOPATHIC ETIOLOGY". Gastroenterology 158, nr 6 (maj 2020): S—1052. http://dx.doi.org/10.1016/s0016-5085(20)33317-5.
Pełny tekst źródłaRozprawy doktorskie na temat "Epigentics"
Yong, Qian Yu. "A screen for modifiers of epigenetic reprogramming". Thesis, Queensland University of Technology, 2011. https://eprints.qut.edu.au/50955/1/Qian_Yu_Yong_Thesis.pdf.
Pełny tekst źródłaHore, Timothy Alexander, i timothy hore@anu edu au. "THE EVOLUTION OF GENOMIC IMPRINTING AND X CHROMOSOME INACTIVATION IN MAMMALS". The Australian National University. Research School of Biological Sciences, 2008. http://thesis.anu.edu.au./public/adt-ANU20081216.152553.
Pełny tekst źródłaSchön, Alexander [Verfasser], i Thomas [Akademischer Betreuer] Carell. "Synthese chemisch modifizierter, epigentisch relevanter Nukleoside und Oligonukleotide / Alexander Schön ; Betreuer: Thomas Carell". München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2020. http://d-nb.info/1225682835/34.
Pełny tekst źródłaCai, Yu. "Molecular Characterization of the mop2, a Gene Required for Epigenetic Silencing". Diss., The University of Arizona, 2006. http://hdl.handle.net/10150/195361.
Pełny tekst źródłaBelzeaux, Raoul. "Etudes de l'expression des ARN périphériques dans la dépression. : La régulation de l'expression génétique en question". Thesis, Aix-Marseille 2, 2011. http://www.theses.fr/2011AIX20727.
Pełny tekst źródłaMajor depression is a frequent and severe disease whose treatment is often inconsistent and patients care remains insufficient. Despite some hypothesis which implicate mono-amine and genetic factors, the pathophysiology of major depression remains unclear. Moreover, no biological marker is available in current clinical practice.Our work aims to propose methodological tools and offers preliminary results to develop such biological markers by studying gene expression in peripheral blood mononuclear cells from severe depressive patients and sex and age-matched controls in different comparative prospective studies. Candidate gene and pangenomic approaches were combined. Moreover, we explored for the first time human microRNA transcription variation in major depression by multiplex RT-qPCR.Among our main findings, we demonstrate that some well-known candidate gene such as serotonin transporter mRNA could be interesting biomarkers of major depression evolution or prognosis. In addition, pangenomic study highlights the implication of genes related to chromatin structure and gene expression regulation like histone family. We also identified variations in the expression of a set of microRNAs during a major depressive episode and, with in silico approaches, we propose putative functional interactions between candidate miRNAs and mRNAs.Overall, our work underlines the feasibility and the relevance of studying the level of expression of RNAs in a psychiatric disorder using peripheral tissues. We obtained both convergent and novel results in regard to previous investigations opening the way to better knowledge of major depression pathophysiology as well as biomarkers development
Freyer, Jennifer Sandra Silvia. "Regulation und funktionelle Rolle des murinen Transkriptionsfaktors Foxp3 in T-Zellen". Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2008. http://dx.doi.org/10.18452/15841.
Pełny tekst źródłaThe aim of the study was to analyze the function and regulation of the transcription factor Foxp3. In a first step we designed a BAC-transgenic mouse with eYFP under the control of the Foxp3 promoter. For creating these mice we use the ET- cloning method. The step of homologous recombination of the target vector into the BAC failed. Because of that, we decided to work in cooperation with the group of Tim Sparwasser from Munich and their BAC- transgenic mouse called DEREG- mouse. This mouse expresses the coding region of eGFP fused to the diphtheria- toxin- receptor under the control of the Foxp3 promoter. Therefore Foxp3+ T cells can be easily detected by eGFP expression and can even be depleted by diphtheria- toxin- application. We confirmed the co- expression of Foxp3 and eGFP and furthermore tested the functionality of the depletion- process of Foxp3+ T cells by treatment with diphtheria- toxin. In a second study, we analyzed the stability of Foxp3 expressing cells in vivo. Therefore we transferred Foxp3+ T cells in syngenic mice and analyzed these cells after 14 days for their Foxp3- expression. Furthermore, we tested the induction of Foxp3 expression through TGF-beta and the suppressive activity of these cells. We also analyzed those cells for their methylation pattern, comparing cells, which showed an induction of Foxp3- expression after one week of culture with TGF-beta to cells, which received TGF-beta for one week and were then restimulated in the absence of TGF-beta. The stability of Foxp3 expression seems to correlate with the demethylated state of the TSDR (Treg Specific Demethylated Region). To get a closer look on the region called TSDR in the murine foxp3 locus, we decided to analyze this region under different aspects. First, we checked for putative binding sites of transcription factors by database analysis of the TSDR. We also analysed histon modifications, such as acetylation of histon H3 and H4 and tri- methylation of lysine 4 at histon3, in this region. Presence of these modifications hinted an epigenetic regulation of Foxp3 involving the TSDR. In a last step, the transcriptional activity of TSDR was tested to delineate whether the TSDR serves as an alternative promoter or acts as a regulative element like an enhancer. Luciferase assays showed that TSDR is a regulative enhancer element, which loses transcriptional activity when methylated. Deletion mutants determined the most important fragment of the TSDR.
Dyer, Ethan. "The Effects of Topical Dose Delivery of Corticosterone on the Development and Hatching Success of the Zebra Finch". 2013. http://scholarworks.gsu.edu/biology_hontheses/5.
Pełny tekst źródłaHore, Tim. "The Evolution of Genomic Imprinting and X Chromosome Inactivation in Mammals". Phd thesis, 2008. http://hdl.handle.net/1885/49309.
Pełny tekst źródłaKhromov, Tatjana. "Pluripotency of multipotent adult germ-line stem cells: analysis of apoptotic and epigenetic features". Doctoral thesis, 2011. http://hdl.handle.net/11858/00-1735-0000-000D-EF56-E.
Pełny tekst źródłaHouston, Kerryn. "Epigentic silencing of the glucocorticoid receptor in small cell lung cancer cells". Thesis, 2013. http://hdl.handle.net/10413/9855.
Pełny tekst źródłaThesis (M.Sc.)-University of KwaZulu-Natal, Westville, 2013.
Części książek na temat "Epigentics"
Zawia, Nasser H., i Riyaz Basha. "Developmental Lead Exposure, Epigentics and Late Onset Alzheimer's Disease". W Developmental Neurotoxicology Research, 143–62. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2010. http://dx.doi.org/10.1002/9780470917060.ch9.
Pełny tekst źródłaStreszczenia konferencji na temat "Epigentics"
Li, Yingqin, Na Liu i Jun Ma. "Abstract 485: Epigentic silencing of miR-142-3p promotes metastasis by targeting ZEB2 in NPC". W Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-485.
Pełny tekst źródłaRaporty organizacyjne na temat "Epigentics"
Gur, Amit, Edward Buckler, Joseph Burger, Yaakov Tadmor i Iftach Klapp. Characterization of genetic variation and yield heterosis in Cucumis melo. United States Department of Agriculture, styczeń 2016. http://dx.doi.org/10.32747/2016.7600047.bard.
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