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Schultz, Stephen T. "Environmental Risk Factors for Autistic Disorder". Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2006. http://wwwlib.umi.com/cr/ucsd/fullcit?p3212023.
Pełny tekst źródłaTitle from first page of PDF file (viewed July 10, 2006). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
Salzer, Jonatan. "Environmental risk factors for multiple sclerosis". Doctoral thesis, Umeå universitet, Klinisk neurovetenskap, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-64212.
Pełny tekst źródłaGartner, Coral Elizabeth. "Environmental risk factors for Parkinson's disease". Thesis, Queensland University of Technology, 2006. https://eprints.qut.edu.au/16393/1/Coral_Gartner_Thesis.pdf.
Pełny tekst źródłaGartner, Coral Elizabeth. "Environmental risk factors for Parkinson's disease". Queensland University of Technology, 2006. http://eprints.qut.edu.au/16393/.
Pełny tekst źródłaBrooks, Constance M. "Environmental risk factors and risky sexual behavior outcomes attitudes as a mediating factor /". Diss., Columbia, Mo. : University of Missouri-Columbia, 2007. http://hdl.handle.net/10355/4820.
Pełny tekst źródłaThe entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from title screen of research.pdf file (viewed on September 19, 2007) Vita. Includes bibliographical references.
Yazdy, Mahsa Mirmiran. "Environmental and infectious risk factors for gastroschisis". Thesis, Boston University, 2013. https://hdl.handle.net/2144/12946.
Pełny tekst źródłaGastroschisis is a rare congenital malformation where loops of bowel are protruding from the abdominal wall of an infant. The prevalence of gastroschisis has been increasing over the past 20 years, particularly in younger women. However, as the number of studies on gastroschisis increases each year, it continues to remain unclear why the prevalence is increasing and why it disproportionately affects younger mothers. Previous research has suggested that environmental or infectious factors may be involved in the pathogenesis of gastroschisis. This dissertation aims to explore the possibility of these two factors in relation to gastroschisis. In study 1, clusters of gastroschisis were identified in space or the combination of space and time. Cases and controls came from the National Birth Defects Study (NBDPS) or for some study centers from the birth defects surveillance systems. Generalized additive models were used to create a continuous map surface of odds ratios (OR) by smoothing over latitude and longitude. Data from the NBDPS were used for the Arkansas, California, and Utah study centers and the highest adjusted ORs detected were 2.0, 1.3, and 2.4, respectively. In Massachusetts and Texas, where surveillance data were used, the highest adjusted ORs observed were 2.4 and 1.3, respectively, with only the latter state achieving statistical significance. Texas had sufficient data to assess the combination of space and time, which identified an increased risk (OR=2.9) in the center of Texas in 2003. The results of this study suggest that clusters of gastroschisis may exist and further exploration of environmental or behavioral factors are warranted. [TRUNCATED]
Rios, Fernandez Paula. "Perinatal and environmental risk factors of childhood neuroblastoma". Thesis, Sorbonne Paris Cité, 2019. http://www.theses.fr/2019USPCB017.
Pełny tekst źródłaBackground: Neuroblastoma is the most common extra-cranial tumor in children. Little is known about the etiology of neuroblastoma. The early age at onset and the embryonic nature suggest a role for perinatal exposures. In this work, we analyzed whether childhood neuroblastoma was associated with specific perinatal characteristics and environmental exposures around pregnancy. We assessed the following birth-related characteristics: gestational age, birth-weight and fetal growth, and the presence of congenital malformations. The maternal reproductive history before the index pregnancy and maternal intake of folic acid or vitamins/minerals before or during pregnancy was also assessed. With regards to environmental exposures related to parental habits, we focused on maternal use of household pesticides during pregnancy, parental smoking and maternal alcohol consumption. Methods: We conducted a pooled analysis of two French national-based case-control studies. The mothers of 357 neuroblastoma case and 1,783 control children younger than 6 years, frequency-matched by age and gender, completed a telephone interview that focused on sociodemographic and perinatal characteristics, childhood environment and parental lifestyle. Unconditional logistic regression was used to estimate pooled odds ratios (OR) and 95% confidence intervals (CIs), including matching variables, study of origin and potential confounders. A meta-analysis of our findings with those of previous studies was also conducted with regards to maternal smoking and alcohol consumption during pregnancy. We used random effects, precision-based weighting to calculate the summary OR including our results. Results: The first part of the thesis focused on perinatal characteristics. We observed that being born either small (OR 1.4 [95% CI 1.0-2.0]) or large (OR 1.5 [95% CI 1.1–2.2]) for gestational age and, among children younger than 18 months, having congenital malformations (OR 3.6 [95% CI 1.3–8.9]), were significantly associated with neuroblastoma. Inverse associations were observed with breastfeeding (OR 0.7 [95% CI 0.5–1.0]) and maternal use of any supplements containing folic acid, vitamins or minerals (OR 0.5 [95% CI 0.3–0.9]) during the preconception period. The second part of the thesis showed that maternal use of any type of household pesticide during pregnancy was associated with neuroblastoma (OR 1.5 [95% CI 1.2–1.9]). The most commonly used type of pesticides were insecticides and there was a positive association with their use alone (OR 1.4 [95% CI 1.1–1.9]) or with other pesticides (OR 2.0 [95% CI 1.1–3.4]). In the third part, our analyses showed that maternal smoking during pregnancy was slightly more often reported for the cases (24.1%) than for the controls (19.7%) (OR 1.3 [95% CI 0.9–1.7]; Paternal smoking in the year before child’s birth was not associated with neuroblastoma as independent exposure (OR 1.1 [95%CI 0.9–1.4] but the association was stronger when both parents reported having smoked during pregnancy (OR 1.5 [95% CI 1.1–2.1]. Finally, in a meta-analysis of maternal smoking and neuroblastoma the summary OR from meta-analysis was 1.1 [95% CI 1.0–1.3]. Conclusions: Our findings support the hypothesis of a defective embryogenesis in neuroblastoma since fetal growth anomalies and congenital malformations were associated with an increased risk of neuroblastoma. This work also adds to the evidence of an association between neuroblastoma and some exposures during pregnancy, such as maternal use of household pesticides and maternal smoking, which are additional reasons why to advise pregnant women to limit these exposures in this period. Further investigations are needed to clarify the role of folic acid supplementation and breastfeeding, given their potential importance in neuroblastoma prevention
Dick, Smita. "Occupational and environmental risk factors for Parkinson's disease". Thesis, University of Aberdeen, 2005. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU201019.
Pełny tekst źródłaKrug, Isabel. "Environmental risk factors and therapeutical implications in eating disorders". Doctoral thesis, Universitat de Barcelona, 2008. http://hdl.handle.net/10803/2546.
Pełny tekst źródłaOur first line of investigation comprised Studies 1 to 3 and assessed the comorbidity of substance use in EDs. The results of these studies suggest that compared to healthy controls, substance use was higher in individuals with EDs (Studies 1 and 3), that the presence of a family history of alcohol dependence was associated with the comorbidity of EDs and substance use (Study 2) that substance use was particularly prevalent in patients with bulimic characteristics (Studies 1 to 3) and that EDs and substance abuse may represent expressions of a fundamental predisposition to addictive behaviour possibly related to the genetically influenced traits such as novelty seeking (Study 2). Furthermore, we revealed some significant differences for the specific forms of drugs in people with EDs (Studies 1 and 3), which emphasize the significance of assessing various drug types in EDs. Finally we also observed cross-cultural differences across various European countries in the prevalence of substance use in EDs and healthy controls (Study 3).
The second research area was labelled psychological, behavioural and environmental correlates of EDs. In our animal study (Study 4) we found that contextual conditioning of eating response was more effective when high than low density caloric food was used. This result indicates that animal models are useful for analyzing and identifying human-animal links in feeding related behaviours.
Studies 5 and 6 assessed which early individual and family eating patterns play a role in the development of EDs. Our findings agree with the growing body of research indicating that a variety of environmental and social factors are associated with dysfunctional individual and family eating patterns (e.g. food used as individualization, control and rules about food) during the first years of life and which if not detected on time could lead to a subsequent ED. Conversely, healthy eating (including eating breakfast) was negatively linked to the development of a subsequent ED. Only a few differences across ED subtypes were observed. Finally some cross-cultural differences also emerged (Study 6).
In Study 7 we compared anger expressions in individuals with EDs and healthy controls and explored the relation among ED symptoms, comorbid psychopathology, personality traits and impulsive behaviours. The results indicated that individuals with EDs obtained significantly higher scores than controls on maladaptive anger expressions. When different purging methods were assessed independently, the frequency of laxative use was associated with anger suppression. ED symptoms and specific personality traits were also positively associated to different anger expressions. At last, we observed that inappropriate anger expressions were related to self-harming behaviours.
Finally our last research line assessed the effectiveness of various treatments for EDs. In Study 8 we compared full and subthreshold BN in terms of personality, clinical characteristics and short-term response to a psychoeducational therapy. The results showed that full-BN and sub-BN share common psychopathological symptoms and personality traits. Furthermore, no differences in therapy outcome were observed in terms of general ED symptomatology and psychopathology. In Study 9 we described and assessed a non-symptom oriented CBT treatment in a congenitally blind women. A dramatic reduction in ED symptoms was observed after the maintaining and triggering factors had been reduced.
To conclude, relatively little research has been performed towards understanding the aetiology of EDs. The findings from our investigations represent a major enhancement in the state of the art of EDs, and lead to the development of a new overall etiological model of EDs. The model we propose is a general understanding of how various ED predisposing and precipitating factors might eventually lead to EDs, and maintain the course of a disorder. Future work should address the effects of genes, environment and gene-environment interaction on the development and maintenance of EDs. Of specific interest is the query of which of these factors are non-specifically related to mental disorders and which factors may be more explicit factors that predispose an individual to EDs and related states, but not to mental disorders in general. The answers to these questions should hopefully become perceptible in the next couple of years.
El objetivo principal de esta tesis ha sido el abordar los Trastornos de la alimentación (TCA) desde una aproximación multidisciplinar, que nos ayude a combatir los TCA desde una perspectiva traslacional, en la que confluyan investigaciones básicas y aplicadas. En nuestros estudios hemos analizado en profundidad los siguientes aspectos: a.) Factores clínicos y comorbilidad [especialmente abuso de sustancias en TCA] (Estudios 1 a 3); b.) Correlatos psicosociales, conductuales y ambientales en TCA (Estudios 4 a 7); y c.) Eficacia de tratamientos específicos en TCA (Estudios 8 a 9).
Nuestra primera línea de investigación (Estudios 1-3), ha estudiado la comorbilidad de abuso de sustancias en TCA. Los resultados de estos estudios sugieren que: a) abuso de sustancias es más prevalente en TCA que en sujetos de control (Estudios 1 y 3); b) la presencia abuso de sustancias en pacientes con TCA, está asociada a historia familiar previa de dependencia de alcohol (Estudio 2); c) abuso de sustancias es especialmente prevalente en pacientes con características bulímicas (Estudios 1 a 3); y d) que presencia de abuso de sustancias en TCA puede ser expresión de una predisposición a conductas adictivas, cuya susceptibilidad viene determinada por rasgos de personalidad específicos (búsqueda de sensaciones) (Estudio 2). Asimismo, encontramos aspectos diferenciales en TCA, en base al tipo de droga que consumen (Estudio 1 y 3), hecho que enfatiza la importancia de evaluar los distintos tipos de drogas utilizados por estos pacientes. Finalmente, encontramos diferencias culturales entre distintos países europeos, respecto a la prevalencia de abuso de sustancias en TCA, al ser comparados con grupos de control (estudio 3).
Como segunda área de investigación, nos centramos en aspectos psicológicos, conductuales y ambientales en TCA. En un estudio, realizado siguiendo un modelo animal (estudio 4), encontramos que el condicionamiento contextual de respuesta alimentaria es más efectivo cuando es utilizado un alimento con alta densidad calórica, que cuando se utiliza un alimento con baja densidad calórica. Estos resultados sugieren qué modelos animales son útiles a la hora de analizar e identificar asociaciones entre comportamiento en humanos y animales, respecto a su conducta alimentaria.
En los estudios 5 y 6 se analizaron en qué medida conductas alimentarias tempranas y patrones alimentarios en la familia, influyen en la aparición posterior de un TCA. Los resultados de estos estudios sugieren, en concordancia con un creciente volumen estudios recientes, la influencia que poseen aspectos ambientales y sociales en el desarrollo posterior de una conducta alimentaria anormal (p.e., control y reglas entorno a la alimentación). Por contrario, patrones alimentarios regulares (p.e., desayunar antes de ir al colegio) se encontró negativamente asociado (factor protector) a subsecuente desarrollo de un TCA. Asimismo, fueron encontradas limitadas diferencias entre subtipos diagnósticos, aunque algunas hacían referencia al contexto cultural (estudio 6).
En el estudio 7 comparamos la expresión de ira en sujetos con un TCA y un grupo control, y analizamos su asociación con sintomatología alimentaria, psicopatología general comórbida, rasgos de personalidad y conductas impulsivas. Nuestros resultados indicaron que pacientes con TCA manifestaban sentimientos de ira de forma más inadecuada que grupos control. Al analizar los distintos procedimientos de purga, la frecuencia de abuso de laxantes iba asociada a supresión de sentimientos de ira.
La sintomatología alimentaria y algunos rasgos de personalidad se asociaban a determinadas formas inadecuadas de expresión de sentimientos. Asimismo, observamos que una inadecuada expresión de sentimientos en TCA se asociaba con una mayor frecuencia de conductas autoagresivas.
Finalmente, en nuestra última línea de investigación analizamos la eficacia de distintos tipos de tratamiento en TCA. En el estudio 8 comparamos casos totales y parciales de Bulimia nerviosa, en base rasgos de personalidad, sintomáticas alimentaria, psicopatología general y respuesta a un tratamiento de carácter psicoeducativo. Los resultados indicaron que los casos totales y parciales de BN comparten similitudes sintomáticas, psicopatológicas y de personalidad. Asimismo, no se obtuvieron diferencias respecto a la respuesta al tratamiento entre ambos grupos de pacientes. En el estudio 9, describimos y evaluamos un tratamiento cognitivo-conductual no centrado en los síntomas alimentarios, en una paciente ciega. En este caso, fue constatada una drástica reducción de los síntomas alimentarios tras este tipo de tratamiento.
En conclusión, hasta el momento, existen escasas referencias en la literatura que sirvan para esclarecer los aspectos etiopatológicos implicados en TCA. Los resultados obtenidos en nuestras investigaciones ayudan sobre el conocimiento actual de TCA, y contribuyen al desarrollo de un nuevo modelo multimodal de entendimiento de éstos. El modelo que proponemos combina la interacción de diversos factores (predisponentes, precipitantes y mantenedores). Trabajos futuros deberían prestar atención a la relevancia que tienen factores genéticos, ambientales e interacción genes-ambiente, en el desarrollo y mantenimiento de los TCA. Un especial interés presenta la pregunta de cuáles de estos factores son específicos para trastornos mentales y cuáles los son para TCA de forma específica.
RESUM:
L'objectiu principal d'aquesta tesi ha estat l'abordar els Trastorns de la conducta alimentària (TCA) des d'una aproximació multidisciplinar, que ens ajudi a combatre'ls des d'una perspectiva traslacional, en la que conflueixin investigacions bàsiques i aplicades. En els nostres estudis hem analitzat en profunditat els següents aspectes: a.) Factors clínics i comorbilitat [especialment abús de substàncies en TCA] (Estudis 1 a 3); b.) Correlats psicosocials, conductuals i ambientals en TCA (Estudis 4 a 7); i c.) Eficàcia de tractaments específics en TCA (Estudis 8 a 9).
La nostra primera línia d'investigació (Estudis 1 a 3), ha estudiat la comorbilitat d'abús de substàncies en TCA. Els resultats d'aquests estudis suggereixen que: a) l'abús de substàncies és més prevalent en TCA que en subjectes control (Estudis 1 i 3); b) la presència d'abús de substàncies en pacients amb TCA, està associada a història familiar prèvia de dependència d'alcohol (Estudi 2); c) l'abús de substàncies és especialment prevalent en pacients amb característiques bulímiques (Estudis 1 a 3); i d) que la presència d'abús de substàncies en TCA pot ser expressió d'una predisposició a conductes addictives, la susceptibilitat de les quals ve determinada per trets de personalitat específics (recerca percaça de sensacions) (Estudi 2). Així mateix, trobem aspectes diferencials en TCA, en base al tipus de droga que consumeixen (Estudis 1 i 3), fet que emfatitza la importància d'avaluar els diferents tipus de drogues utilitzats per aquests pacients. Finalment trobem diferències culturals entre distints països europeus, respecte a la prevalença d'abús de substàncies en TCA, al ser comparats amb grups control (Estudi 3).
Com a segona àrea d'investigació, ens centrem en aspectes psicològics, conductuals i ambientals en TCA. En un estudi realitzat seguint un model animal (Estudi 4), trobem que el condicionament contextual de resposta alimentària és més efectiu quan s'utilitza un aliment amb alta densitat calòrica, que quan se'n utilitza un amb baixa densitat calòrica. Aquests resultats suggereixen que els models animals són útils a l'hora d'analitzar i identificar associacions entre comportament en humans i animals, respecte la seva conducta alimentària.
En els estudis 5 i 6 es varen analitzar fins a quin punt conductes alimentàries primerenques i patrons alimentaris en la família influeixen en l'aparició posterior d'un TCA. Els resultats d'aquests estudis suggereixen, en concordància amb un volum creixent d'estudis recents, la influència que tenen aspectes ambientals i socials en el desenvolupament posterior d'una conducta alimentària anormal (per exemple, control i normes entorn a l'alimentació). Tanmateix, patrons alimentaris regulars (per exemple, esmorzar abans d'anar a l'escola) estaven negativament associats (factor protector) al subseqüent desenvolupament d'un TCA. Així mateix, es varen trobar diferències limitades entre subtipus diagnòstics. Finalment, es varen trobar algunes diferències en relació al context sociocultural. (Estudi 6).
En l'estudi 7 vàrem comparar expressió d'ira en subjectes amb un TCA i un grup control, i vàrem analitzar la seva associació amb simptomatologia alimentària, psicopatologia general comòrbida, trets de personalitat i conductes impulsives. Els nostres resultats varen indicar que pacients amb TCA manifestaven sentiments d'ira de forma més inadequada que grups control. A l'analitzar els diferents procediments de purga, la freqüència d'abús de laxants anava associada a la supressió de sentiments d'ira.
La simptomatologia alimentària i determinats trets de personalitat, s'associaven a determinades formes inadequades d'expressió de sentiments. Així mateix, vàrem observar que una expressió inadequada de sentiments en TCA anava associada a una freqüència major de conductes autoagressives.
Finalment, en la nostra darrera línia d'investigació vàrem analitzar l'eficàcia de diferents tipus de tractament en TCA. En l'estudi 8 vàrem comparar casos totals i parcials de Bulímia nerviosa (BN), en base a trets de personalitat, simptomatologia alimentària, psicopatologia general i resposta a un tractament de caràcter psicoeducatiu. Els resultats varen indicar que els casos totals i parcials de BN, comparteixen similituds simptomatològiques, psicopatològiques i de personalitat. Així mateix, no es varen obtenir diferències respecte a la resposta del tractament entre ambdós grups de pacients. En l'estudi 9, vàrem descriure i avaluar un tractament cognitiu-conductual no centrat en els símptomes alimentaris, en una pacient cega. En aquest cas es va constatar una reducció dràstica dels símptomes alimentaris després d'aquest tipus de tractament.
Fins el moment, existeixen escasses referències en la literatura que serveixin per aclarir els aspectes etiopatogènics implicats en els TCAs.
Els resultats obtinguts en les nostres investigacions ajuden en el coneixement actual dels TCA i contribueixen al desenvolupament d'un nou model multimodal per la comprensió de la seva etiologia. El model que proposem combina la interacció de diversos factors (predisponents, precipitants i mantenidors). Els treballs futurs haurien de incidir en la rellevància que tenen els factors genètics, ambientals i la interacció d'ambdós, en el desenvolupament i manteniment dels TCA. És d'especial interès la qüestió de quins d'aquests factors són específics per trastorns mentals i quins ho són per TCA.
Zumel, Marne María Ángela 1984. "Environmental factors and brain tumour risk in young people". Doctoral thesis, Universitat Pompeu Fabra, 2019. http://hdl.handle.net/10803/668182.
Pełny tekst źródłaLos factores de riesgo y el diagnóstico en los jóvenes han sido poco explorados, a pesar de que los tumores cerebrales (TC) son uno de los tipos de tumores más frecuentes en los niños y jóvenes. El propósito de esta tesis doctoral es el estudio de 1) de las características clínicas y los síntomas de los TC en los jóvenes, basados en el estudio internacional de casos y controles MOBI-Kids; 2) una revisión sistemática de la literatura sobre el riesgo de TC en jóvenes en relación con factores ambientales; 3) el riesgo de TC en relación con los productos químicos presentes en el agua potable y con los metales pesados. Los análisis de las características clínicas revelaron que la gran mayoría de los tumores eran neuroepiteliales (principalmente gliomas), seguidos de tumores embrionarios y meningiomas. En general, los síntomas más frecuentes fueron dolor de cabeza, seguido de signos y síntomas neurológicos focales, náuseas/ vómitos y problemas en la visión, siendo un 4% de los casos asintomáticos. El tiempo promedio de diagnóstico tendió a ser corto (mediana 1,42 meses), aunque esto varió según el tipo de tumor, la edad y el tipo de síntoma. Encontré muchos estudios que encontraron asociación entre los factores ambientales (incluido el humo del tabaco, los pesticidas y la dieta, entre otras exposiciones) y el riesgo de TC en la revisión sistemática. Sin embargo, debido a limitaciones metodológicas, la evidencia sobre el papel de estos factores en la etiología de esta enfermedad aún es incierta. Nuestros análisis en relación con los productos químicos del agua mostraron unos OR por debajo de 1 para exposiciones a THMs, y OR por encima de 1 para exposición a nitrato, tanto en períodos de exposición prenatales como postnatales, algunos estadísticamente significativos. Nuestros análisis de metales pesados mostraron ORs por debajo de 1 para la exposición al cromo. Sin embargo, la literatura es escasa sobre esta asociación. En general, esta tesis sirvió para llenar un vacío en el conocimiento sobre 1) las características clínicas de la TC en los jóvenes, útiles tanto para la práctica clínica como para la investigación etiológica; 2) causas de esta enfermedad; 3) el papel de los metales pesados y los químicos ubicuos en el agua. Se ha identificado la necesidad de realizar más investigaciones sobre la etiología y la prevención de las TC en los jóvenes.
Costello, Sadie Cash. "Environmental, behavioral and familial risk factors for Parkinson's disease". Diss., Restricted to subscribing institutions, 2008. http://proquest.umi.com/pqdweb?did=1679291501&sid=1&Fmt=2&clientId=1564&RQT=309&VName=PQD.
Pełny tekst źródłaWall, Tamara. "A content analysis : health and environmental risk factors of children in methamphetamine living environments /". Available to subscribers only, 2008. http://proquest.umi.com/pqdweb?did=1559858731&sid=1&Fmt=2&clientId=1509&RQT=309&VName=PQD.
Pełny tekst źródłaCarpenter, Rachel K., Jill D. Stinson, Rebecca H. Gilley i Lydia L. Eisenbrandt. "Partner Violence and Environmental Risk Factors: A Generalized Mixed Model". Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/etsu-works/7921.
Pełny tekst źródłaFilipsson, Monika. "Uncertainty, variability and environmental risk analysis". Doctoral thesis, Linnéuniversitetet, Institutionen för naturvetenskap, NV, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-11193.
Pełny tekst źródłaNegativa effekter orsakade av skadliga ämnen och möjliga åtgärder bedöms och utvärderas i en miljöriskanalys, som kan delas i riskbedömning, riskkommunikation och riskhantering. Osäkerhet som beror på kunskapsbrist samt naturlig variabilitet finns alltid närvarande i denna process. Syftet med avhandlingen är att utvärdera några tillvägagångssätt samt diskutera hur osäkerhet och variabilitet hanteras då det är nödvändigt att båda hanteras trovärdigt och transparent för att riskbedömningen ska vara användbar för beslutsfattande. Metallers katalytiska effekt på bildning av klorerade aromatiska ämnen under upphettning av flygaska undersöktes (artikel I). Koppar visade en positiv katalytisk effekt medan kobolt, krom och vanadin istället katalyserade nedbrytningen. Kunskap om katalytisk potential för bildning av skadliga ämnen är viktigt vid val och design av förbränningsprocesser för att minska utsläppen, men det är också ett exempel på hur en fara kan identifieras och karaktäriseras. Information om exponeringsfaktorer som är viktiga i riskbedömning (fysiologiska parametrar, tidsanvändning och livsmedelskonsumtion) samlades in och analyserades (artikel II). Interindividuell variabilitet karaktäriserades av medel, standardavvikelse, skevhet, kurtosis (toppighet) och multipla percentiler medan osäkerhet i dessa parametrar skattades med konfidensintervall. Hur dessa statistiska parametrar kan tillämpas i exponeringsbedömningar visas i artikel III och IV. Probability bounds analysis användes som probabilistisk metod, vilket gör det möjligt att separera osäkerhet och variabilitet i bedömningen även när tillgången på data är begränsad. Exponeringsbedömningen i artikel III visade att vid nu rådande föroreningshalter i sediment i en badsjö så medför inte bad någon hälsofara. I artikel IV visades att osäkerhetsintervallet i den skattade exponeringen ökar när hänsyn tas till förändringar i klimatkänsliga modellvariabler. Riskhanterare måste ta hänsyn till försiktighetsprincipen och en ökad osäkerhet kan därmed få konsekvenser för riskhanteringsbesluten. Artikel V fokuserar på riskhantering och en enkät skickades till alla anställda som arbetar med förorenad mark på länsstyrelserna i Sverige. Det konstaterades att anställdas kön, ålder och erfarenhet har en inverkan på granskningsprocessen av riskbedömningar. Kön var den mest signifikanta variabeln, vilken också påverkade perceptionen av kunskap. Skillnader i de anställdas svar kunde också ses beroende på om riskbedömningen finansierades av statliga bidrag eller av en ansvarig verksamhetsutövare.
Stojanova, Jana. "Environmental and genetic risk factors for post-transplant lymphoproliferative disease". Limoges, 2013. http://aurore.unilim.fr/theses/nxfile/default/6c517c4c-5de7-490e-a6f5-5fc542155ba9/blobholder:0/2013LIMO310E.pdf.
Pełny tekst źródłaLes lymphomes post-transplantation (PTLD) sont les complications malignes les plus fréquentes de l’immunosuppression (IS) après les cancers cutanés, et présentent la première cause de mortalité et de perte du greffon due aux cancers chez les transplantés. De plus, les lymphomes posttransplantation peuvent se comporter de manière plus agressive avec un plus mauvais pronostic. L’EBV joue un rôle clef dans la physiopathologie de la majorité des PTLDs. Chez l’hôte immunocompétent, les lymphocytes T cytotoxiques (LTC) spécifiques de l’EBV empêchent la croissance des lymphocytes B porteurs de l’EBV, mais cette immunovigilance est abaissée lors d’une immunosuppression, qu’elle soit thérapeutique ou pathologique. Les études s’intéressant aux facteurs de risque des PTLD ont été effectuées sur des données issues de larges registres de transplantation et qui sont par nature insuffisamment détaillées pour étudier l’influence du maintien d’un traitement IS au cours de temps sur le risque de PTLD. Les études s’intéressant aux risques génétiques des PTLDs ont été focalisées sur les polymorphismes des gènes des cytokines, mais elles étaient limitées par l’effectif réduit et/ou l’hétérogénéité des populations cas et témoins. Etant donnée l’étiologie virale des PTLDs et le rôle important du système immunologique, nous avons émis l’hypothèse qu’une hypoactivité spontanée des protéines cibles et des éléments de leurs voies de signalisation, ou une hypersensibilité de ces protéines en réponse à l’effet des IS seraient à l’origine d’une immunosuppression trop intense et par conséquent favoriseraient la lymphomagénèse. De plus, ce travail tente de répondre à la question suivante: est-ce que certains IS ont une influence particulière sur la survenue des PTLD, indépendamment de leur effet sur le système immunitaire? L'effet des inhibiteurs de la calcineurine (ICN) sur la transformation des cellules sanguines mononuclées (PBMC) par l’EBV a été exploré avec deux panels de 10 marquages par cytométrie en flux : l’un pour caractériser les lymphoblastes émergents et l’autre pour caractériser la réponse T. L’effet de l’administration chronique de la ciclosporine a été exploré sur un modèle de lymphome spontanée chez la souris, exprimant le transgène CD40/LMP1. Nous montrons que les ICN ne semblent pas avoir de rôle autre que l’inhibition des LTC. Chez les souris CD40/LMP1, la ciclosporine produit une augmentation des lymphocytes B activés dans la rate. Le profil des infiltrations des lymphocytes T dans la rate ne semble pas être expliqué seulement par une inhibition de LTC par la ciclosporine. Finalement, une étude pharmacogénétique clinique de type cas-témoins a été effectuée, et montre que des polymorphismes des gènes IL10 et IL2 sont associés à la pathologie, ainsi que l’administration d’azathioprine, un médicament immunosuppresseur heureusement aujourd’hui largement abandonné en transplantation
Rumchev, Krassi. "Indoor environmental risk factors for respiratory symptoms and asthma in young children". Thesis, Curtin University, 2001. http://hdl.handle.net/20.500.11937/1494.
Pełny tekst źródłaSayols, Baixeras Sergi 1988. "DNA methylation and its relationship with lifestyle, environmental and cardiovascular risk factors". Doctoral thesis, Universitat Pompeu Fabra, 2019. http://hdl.handle.net/10803/665398.
Pełny tekst źródłaCoronary artery disease is the first cause of death worldwide, and as a complex disease implies the interplay between genetic and environmental factors. Common genetic variants explain only 15-20% of its heritability. DNA methylation, which regulates the gene expression without altering the DNA sequence, has been proposed as a heritable signature to explain this missing heritability and as a mediator effect of lifestyle and environment on health. To study the relationship between DNA methylation and smoking, air pollution, and cardiovascular risk factors (obesity, lipid profile, and HDL functionality) we used epigenome-wide association studies, Mendelian randomization, and multi-stage omics integration approaches. We included data from ten studies, but the results were mainly based on the REGICOR (REgistre GIroní del COR) cohort. We report the association between smoking and 63 methylation sites (CpGs). We did not find association between air pollution and DNA methylation. We identified 94 CpGs associated with obesity and 14 with lipid profile. We observed that methylation at CPT1A and SLC7A11 can modify or be affected by the triglycerides levels, highlighting the complexity of the lipids homeostasis. Finally, we performed the first study showing an association between DNA methylation and HDL functionality. As conclusions of this research, smoking is strongly associated with a distinctive methylation pattern and there is a relationship between DNA methylation and several cardiovascular risk factors, although its causality is complex.
Hampel, Regina. "Environmental effects and gene-environment interactions: air pollution and temperature effects on cardiovascular risk factors". Diss., lmu, 2011. http://nbn-resolving.de/urn:nbn:de:bvb:19-138583.
Pełny tekst źródłaMaioli, Silvia <1982>. "Interaction between APOE4 genotype and environmental risk factors in Alzheimer's disease". Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2011. http://amsdottorato.unibo.it/3463/.
Pełny tekst źródłaMiskelly, Francis G. "Environmental factors and symptoms in infants at high risk of allergy". Thesis, Queen's University Belfast, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334472.
Pełny tekst źródłaRumchev, Krassi. "Indoor environmental risk factors for respiratory symptoms and asthma in young children". Curtin University of Technology, School of Public Health, 2001. http://espace.library.curtin.edu.au:80/R/?func=dbin-jump-full&object_id=12389.
Pełny tekst źródławho had never been diagnosed with asthma, identified from birth records accessed through the Health Department of Western Australia. Information, regarding the respiratory conditions experienced by the study children and characteristics of the home, was collected using a standardised questionnaire. The questionnaire consists of questions about potential risk factors for asthma and these factors were grouped in three categories. The first category included information on personal and social factors such as age and gender of the child, and mother's and father's educational level. The second category was related to personal susceptibility factors such as child's allergy, parental and sibling's asthma, eczema and hay fever. The last category included environmental exposure in the house such as parental and visitors smoking inside the house, exposure to gas heating and cooking, kerosene space heaters, open fireplaces, and pets. Other questions related to environmental exposure were the presence of air conditioning, humidifiers, and type of floor covering in the child's bedroom and the living room. Measurements of indoor nitrogen dioxide (NO[subscript]2), formaldehyde (HCHO), volatile organic compounds (VOCs), particulate matter (PM[subscript]10), and house dust mite exposure were made on two occasions over one year, winter (middle June through September 1998) and summer (December 1998 through March 1999), Indoor temperature and relative humidity were also measured. The atopic status of the children was assessed by skin prick tests to common allergens.The study results indicated that age, gender, family history of asthma, atopy and domestic exposure to indoor environmental factors were significant predictors of asthma early in life. The study found that indoor exposure to formaldehyde, volatile organic compounds and house dust mite significantly increased the risk of ++
having asthma. Presence of air conditioning appeared to be a protective factor for asthma.In conclusion, the study results confirmed the role of susceptibility factors in asthma and show that indoor environmental factors contribute as risk factors for asthma in early stage of fife. The observation that exposure to indoor air pollutants in early childhood is associated with asthma suggests the possibility that irritants in indoor air might be involved in the initiation phase of asthma. Since the quality of the indoor environment is potentially modifiable there might be opportunities for intervention to reduce asthma symptoms. In order to counteract the increasing prevalence in asthma, the significance of the indoor environment where children grow and spend most of their time need to be given greater attention.
Assaggaf, Hamza M. "Molecular Risk Factors of Pulmonary Arterial Hypertension". FIU Digital Commons, 2017. https://digitalcommons.fiu.edu/etd/3554.
Pełny tekst źródłaLandman-Peeters, Karlien Maria Catharina. "At risk of depression and anxiety studies into the interplay of personal and environmental risk factors /". [S.l. : [Groningen : s.n.] ; University Library Groningen] [Host], 2007. http://irs.ub.rug.nl/ppn/304261017.
Pełny tekst źródłaMillburg, Stacy N. "The effects of environmental risk factors on at-risk urban high school students' academic self-efficacy". Cincinnati, Ohio : University of Cincinnati, 2009. http://rave.ohiolink.edu/etdc/view.cgi?acc_num=ucin1243353903.
Pełny tekst źródłaAdvisor: Nancy A. Evers. Title from electronic thesis title page (viewed Aug. 12, 2009). Includes abstract. Keywords: at-risk; urban; academic self-efficacy. Includes bibliographical references.
Gunnbjörnsdóttir, María Ingibjörg. "Asthma and respiratory symptoms in Nordic countries, environmental and personal risk factors /". Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7076.
Pełny tekst źródłaGunnbjörnsdóttir, María Ingibjörg. "Asthma and Respiratory Symptoms in Nordic Countries, Environmental and Personal Risk Factors". Doctoral thesis, Uppsala University, Department of Medical Sciences, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7076.
Pełny tekst źródłaThe overall aims of our studies were to identify risk factors for respiratory symptoms and asthma in indoor environment but even to look at some personal risk factors such as body mass index and gastroesophageal reflux. The study population is based on participants of the European Community Respiratory Health Survey I and II.
In the first study, water damage and visible moulds were reported in 7.4% and 17% of the homes respectively. The combination of water damage and visible moulds was independently associated with attacks of breathlessness when resting and after activity and also to long term cough. In the second study, the prevalence of nocturnal GER increased with higher BMI and the same pattern could be seen for habitual snoring. Reported onset of asthma, wheeze and night-time symptoms increased in prevalence along with the BMI gradient. In the multivariable analysis, obesity and nocturnal GER were independent risk factors for onset of asthma, wheeze and night-time symptoms. Habitual snoring was an independent risk factor for onset of wheeze and night-time symptoms, but not for onset of asthma. In the third study, a total of 18% of the subjects reported indoor dampness in the last 12 months and 27% of the subjects reported indoor dampness since the previous survey. Respiratory symptoms and asthma were significantly more prevalent in individuals exposed to indoor dampness and indoor dampness was a risk factor for respiratory symptoms and asthma after adjusting for possible confounders. Indoor dampness was an independent risk factor for onset of respiratory symptoms but not for asthma onset. Remission of respiratory symptoms was less likely to occur if subjects reported indoor dampness. In the fourth study, the lowest prevalence of atopy and the lowest levels of all indoor allergens, bacteria and moulds were found in Iceland. A positive association was found, between cat allergen exposure and asthma symptoms and between bronchial hyperresponsiveness and the amount of viable mould in indoor air.
Chan, Daniel Kam Yin School of Physiology & Pharmacology UNSW. "Genetic and environmental risk factors for Parkinson's disease in Chinese and Australians". Awarded by:University of New South Wales. School of Physiology & Pharmacology, 2000. http://handle.unsw.edu.au/1959.4/17795.
Pełny tekst źródłaHeslop, Claire Louise. "Emerging environmental, molecular, and genetic risk factors in stable coronary artery disease". Thesis, University of British Columbia, 2009. http://hdl.handle.net/2429/11244.
Pełny tekst źródłaBasil, Paul. "Epigenetic modifications associated with prenatal environmental risk factors for neurodevelopmental psychiatric disorders". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/208545.
Pełny tekst źródłaPatel, Bipen Dahyabhai. "Environmental and genetic risk factors for the development of obstructive airways disease". Thesis, University of Cambridge, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.613675.
Pełny tekst źródłaAyuk, Adaeze Chikaodinaka. "Environmental risk factors for asthma in 13-14 year old African children". Master's thesis, University of Cape Town, 2018. http://hdl.handle.net/11427/29369.
Pełny tekst źródłaThrasher, Lakeasha. "Impact of Environmental and Individual Risk Factors on Pregnant and Parenting Teenagers". ScholarWorks, 2015. https://scholarworks.waldenu.edu/dissertations/1371.
Pełny tekst źródłaMitchell, Hailey. "Considering Environmental Toxicants as Risk Factors for Postpartum Depression: A Systematic Review". Thesis, Université d'Ottawa / University of Ottawa, 2020. http://hdl.handle.net/10393/40071.
Pełny tekst źródłaGeorge, Lena. "Spontaneous abortion : risk factors and measurement of exposures /". Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-921-1/.
Pełny tekst źródłaTurner, Michelle C. "Environmental Risk Factors for Lung Cancer Mortality in the Cancer Prevention Study-II". Thèse, Université d'Ottawa / University of Ottawa, 2012. http://hdl.handle.net/10393/20528.
Pełny tekst źródłaCole, Maxine A. "Perception of risk to health from environmental factors amongst the Mohawks of Akwesasne". Thesis, University of Ottawa (Canada), 2004. http://hdl.handle.net/10393/26612.
Pełny tekst źródłaPorcellini, Elisa <1978>. "Genetic and environmental factors associated with the risk of cognitive decline and dementia". Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2011. http://amsdottorato.unibo.it/3336/.
Pełny tekst źródłaGuthmann, Jean-Paul. "Environmental risk factors for malaria : a matched case-control study in Piura, Peru". Thesis, London School of Hygiene and Tropical Medicine (University of London), 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322496.
Pełny tekst źródłaCampos, Carlos Jose Alexandre De. "Contamination of shellfish waters with human noroviruses : environmental risk factors and management options". Thesis, Aberystwyth University, 2016. http://hdl.handle.net/2160/4ccf2977-b58c-456c-8a95-a0f8857b21a7.
Pełny tekst źródłaReider, Carson R. "Environmental risk factors for Parkinson's Disease : a matched sibling pair case-control study /". The Ohio State University, 2001. http://rave.ohiolink.edu/etdc/view?acc_num=osu1488205318509.
Pełny tekst źródłaMoreno, Mendoza Daniel. "Tumor testicular de células germinales: identificación de nuevos factores de riesgo". Doctoral thesis, Universitat Autònoma de Barcelona, 2020. http://hdl.handle.net/10803/671273.
Pełny tekst źródłaLa presente tesis es una aportación al conocimiento de nuevos factores de riesgo para el tumor testicular de células germinales (TTCG). El TTCG presenta una etiología multifactorial, atribuible a un retraso en la diferenciación de los gonocitos fetales. El TTCG es más frecuente en varones con una espermatogénesis alterada, sugiriendo una posible etiopatogenia común. El cromosoma Y contiene genes esenciales para una correcta espermatogénesis, las regiones del factor de azoospermia (AZF). La región AZF más dinámica es la región AZFc que presenta puntos frágiles que predispone a reordenamientos. El reordenamiento parcial más relevante desde el punto de vista clínico de la región AZFc es la deleción gr/gr. Se ha relacionado la delación gr/gr con un mayor riesgo de desarrollar un TTCG, pero la falta de información sobre los parámetros seminales de los pacientes no ha permitido de clarificar si la asociación observada está relacionada con la espermatogénesis alterada o si es un factor de riesgo independiente. Además, aún queda por establecer si otros tipos de deleciones y duplicaciones de la región AZFc presentan relación con el TTCG. La primera parte de esta tesis se enfoca en el estudio de los reordenamientos parciales de la región AZFc en el TTCG. Se han analizado 497 pacientes con TTCG y 2030 controles sin TTCG. Un 3.8% de los pacientes con TTCG presentaban algún tipo de deleción parcial de la región AZFc respecto al 2.5% del grupo control (p= 0.078). La deleción parcial más frecuente fue la deleción gr/gr, mientras que los otros tipos de deleciones parciales de la región AZFc resultaron ser muy raras. Según el fenotipo seminal, se observó un mayor riesgo de TTCG en pacientes normozoospérmicos portadores de deleciones parciales de la región AZFc respecto a los controles normozoospérmicos. No hubo diferencias significativas entre pacientes y controles según las duplicaciones parciales de la región AZFc. Se mostró que las alteraciones en la dosis del gen DAZ confieren un mayor riesgo de TTCG. Estos resultados confirman que un déficit del contenido génico de la región AZFc juega un papel importante en la etiopatogénesis del TTCG. En particular, la deleción gr/gr confiere un riesgo significativo para el desarrollo del TTCG independientemente de los parámetros seminales. Los factores ambientales también están involucrados en la etiopatogénesis del TTCG, especialmente si interfieren en un periodo específico del desarrollo testicular, en el denominado "masculinization programming window" (MPW). Un desequilibrio hormonal en este periodo compromete la correcta función de las células fetales de Sertoli y Leydig, originando el síndrome de disgenesia testicular (SDT). La distancia anogenital (DAG) es considerada un biomarcador de la acción de los andrógenos durante el MPW. La DAG más corta ha sido relacionada con todos los componentes del SDT, excepto con el TTCG. La segunda parte de esta tesis valora la asociación entre la DAG y el TTCG. Además evalúa el papel del polimorfismo CAG del gen AR en el desarrollo del TTCG y la DAG. Se analizaron a 156 pacientes con TTCG y 110 controles sanos normozoospérmicos. Se observó una distancia anopeneana (DAGap) y una distancia anoescrotal (DAGas) significativamente más corta en los TTCG respecto a los controles. Se definieron unos punto de corte (DAGap: 130mm ;DAGas: 53mm) que indican un mayor riesgo de TTCG en aquellos individuos que se encuentren por debajo de estos valores. No se encontró relación entre el polimorfismo CAG y el TTCG o la longitud de la DAG. En conclusión, los datos revelan que los pacientes con una DAG más corta presentan un mayor riesgo de TTCG, apoyando la teoría sobre la influencia del desequilibrio androgénico durante el desarrollo fetal en la etiopatogenia del TTCG.
This thesis is a contribution to the knowledge of new risk factors for testicular germ cell tumor (TTCG). TTCG has a multifactorial etiology, attributable to a delay in the differentiation of fetal gonocytes. TTCG is more frequent in men with altered spermatogenesis, suggesting a possible common etiopathogenesis. The Y chromosome contains essential genes for correct spermatogenesis, the azoospermia factor (AZF) regions. The most dynamic AZF region is the AZFc region that presents fragile points that predispose to rearrangements. The most clinically relevant partial rearrangement of the AZFc region is the gr / gr deletion. gr / gr cheating has been associated with an increased risk of developing TTCG, but the lack of information on the seminal parameters of the patients has not made it possible to clarify whether the observed association is related to altered spermatogenesis or if it is a factor of independent risk. Furthermore, it remains to be established whether other types of deletions and duplications of the AZFc region are related to TTCG. The first part of this thesis focuses on the study of partial rearrangements of the AZFc region in the TTCG. 497 patients with TTCG and 2030 controls without TTCG have been analyzed. 3.8% of the patients with TTCG presented some type of partial deletion of the AZFc region compared to 2.5% of the control group (p = 0.078). The most frequent partial deletion was the gr / gr deletion, while the other types of partial deletions of the AZFc region were found to be very rare. According to the seminal phenotype, a higher risk of TTCG was observed in normozoospermic patients carrying partial deletions of the AZFc region compared to normozoospermic controls. There were no significant differences between patients and controls according to the partial duplications of the AZFc region. Alterations in the dose of the DAZ gene were shown to confer an increased risk These results confirm that a deficit in the gene content of the AZFc region plays an important role in the etiopathogenesis of TTCG. In particular, the gr / gr deletion confers a significant risk for the development of TTCG regardless of seminal parameters. Environmental factors are also involved in the aetiopathogenesis of TTCG, especially if they interfere in a specific period of testicular development, in the so-called "masculinization programming window" (MPW). A hormonal imbalance in this period compromises the correct function of the fetal Sertoli and Leydig cells, causing the testicular dysgenesis syndrome (TDS). The anogenital distance (DAG) is considered a biomarker of the action of androgens during MPW. The shorter DAG has been related to all components of the SDT, except the TTCG. The second part of this thesis assesses the association between the DAG and the TTCG. It also evaluates the role of the CAG polymorphism of the AR gene in the development of TTCG and DAG. 156 patients with TTCG and 110 healthy normozoospermic controls were analyzed. A significantly shorter anopeneal distance (DAGap) and anoscrotal distance (DAGas) were observed in TTCG compared to controls. Cut-off points were defined (DAGap: 130mm; DAGas: 53mm) that indicate a greater risk of TTCG in those individuals who are below these values. No relationship was found between the CAG polymorphism and the TTCG or the length of the DAG. In conclusion, the data reveal that patients with a shorter DAG have a higher risk of TTCG, supporting the theory about the influence of androgen imbalance during fetal development on the etiopathogenesis of TTCG.
Choi, Young-Seon. "The physical environment and patient safety: an investigation of physical environmental factors associated with patient falls". Diss., Georgia Institute of Technology, 2011. http://hdl.handle.net/1853/45974.
Pełny tekst źródłaO'Connell, Bethesda J., Megan Quinn i Phillip R. Scheuerman. "Risk Factors of Diarrheal Disease Among Children in the East African Countries of Burundi, Rwanda and Tanzania". Digital Commons @ East Tennessee State University, 2017. https://dc.etsu.edu/etsu-works/2879.
Pełny tekst źródłaJoyce, Sarah Julia. "Demographic, clinical and environmental risk factors for prelabour rupture of membranes in Western Australia". University of Western Australia. School of Population Health, 2009. http://theses.library.uwa.edu.au/adt-WU2009.0126.
Pełny tekst źródłaTarricone, Ilaria. "Incidence and environmental risk factors for psychosis in first generation migrants in northern Italy". Thesis, King's College London (University of London), 2016. https://kclpure.kcl.ac.uk/portal/en/theses/incidence-and-environmental-risk-factors-for-psychosis-in-first-generation-migrants-in-northern-italy(303617e5-8cc8-47b4-89e5-cdf53e245f95).html.
Pełny tekst źródłaPeters, Sean Michael. "Youth gang involvement in early adolescence an examination of environmental and individual risk factors /". Access restricted to users with UT Austin EID, 2001. http://wwwlib.umi.com/cr/utexas/fullcit?p3035164.
Pełny tekst źródłaJoseph, Cesar Margolite. "Poverty Rate and Occurrence of Foodborne Illness Risk Factors in Retail Facilities". ScholarWorks, 2018. https://scholarworks.waldenu.edu/dissertations/4926.
Pełny tekst źródłaZahid, Sardar Muhammad. "Green Supply Chain Management Practices and Determinant Factors: A Quantitative Study on Small and Medium Enterprises Using Structural Equation Modeling". Diss., North Dakota State University, 2017. http://hdl.handle.net/10365/25929.
Pełny tekst źródłaLanglois, Elizabeth. "Factors that Shape Environmental Perceptions: the Role of Health and Place". ScholarWorks@UNO, 2012. http://scholarworks.uno.edu/td/1595.
Pełny tekst źródłaHampel, Regina [Verfasser], i Annette [Akademischer Betreuer] Peters. "Environmental effects and gene-environment interactions : air pollution and temperature effects on cardiovascular risk factors / Regina Hampel. Betreuer: Annette Peters". München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2011. http://d-nb.info/1019291494/34.
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