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Książki na temat „Endocrine resistance”

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Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 1 lutego 2022

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1

Cohen, Margo Panush, i Piero P. Foà, red. Hormone Resistance and Other Endocrine Paradoxes. New York, NY: Springer New York, 1987. http://dx.doi.org/10.1007/978-1-4612-4758-6.

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2

Galland, Leo. The fat resistance diet: Reprogram your body to stay thin forever. Emmaus, Penna: Rodale, 2008.

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3

Hormone resistance and hypersensitivity: From genetics to clinical management. Basel: Karger, 2013.

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4

The Fat resistance diet: Unlock the secret of the hormone leptin to--eliminate cravings, supercharge your metabolism, lose weight, and reprogram your body to stay thin-forever. New York: Broadway Books, 2006.

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5

Audrey, Tea, red. Can't lose weight?: Unlock the secrets that make you store fat! : this is the only book that investigates all the hidden causes of weight excess! Glendale, AZ: SCB International Inc., 2001.

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6

P, Cohen Margo, i Foà Piero P. 1911-, red. Hormone resistance and other endocrine paradoxes. New York: Springer-Verlag, 1987.

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7

Wiffen, Philip, Marc Mitchell, Melanie Snelling i Nicola Stoner. Therapy-related issues: endocrine system. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199603640.003.0021.

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Diabetes mellitus 444Monitoring and control 449Thyroid disorders 450Diabetes mellitus (DM) affects approximately 4% of the UK population. In 2009, Diabetes UK reported that 2.6 million people in the UK have diabetes.Type 2 diabetes accounts for 90% of all diabetes and is a result of insulin resistance and pancreatic β-cell dysfunction. Type 1 diabetes results from an absolute insulin deficiency secondary to autoimmune dysfunction....
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8

Beck-Peccoz, Paolo. Syndromes of Hormone Resistance on the Hypothalamic-Pituitary-Thyroid Axis (Endocrine Updates). Springer, 2004.

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9

Evanthia, Diamanti-Kandarkis, red. Insulin resistance and polycystic ovarian syndrome: Pathogenesis, evaluation, and treatment. Totowa, N.J: Humana Press, 2007.

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10

Cabot, Sandra. Can't Lose Weight?: Unlock the Secrets That Keep You Fat! Ten Speed Press, 2002.

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11

Can't Lose Weight?: You Could Have Syndrome X. Ten Speed Press, 2002.

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12

Evanthia, Diamanti-Kandarkis, red. Insulin resistance and polycystic ovarian syndrome: Pathogenesis, evaluation, and treatment. Totowa, N.J: Humana Press, 2007.

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13

(Editor), John E. Nestler, Evanthia Diamanti-Kandarakis (Editor), Renato Pasquali (Editor) i Dimitrios Panidis (Editor), red. Insulin Resistance and Polycystic Ovarian Syndrome: Pathogenesis, Evaluation, and Treatment (Contemporary Endocrinology). Humana Press, 2007.

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14

Jolly, Elaine, Andrew Fry i Afzal Chaudhry, red. Diabetes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199230457.003.0008.

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Chapter 8 covers the basic science and clinical topics relating to the endocrine system which trainees are required to learn as part of their basic training and demonstrate in the MRCP. It covers diabetes mellitus, diabetic emergencies, diabetes-long-term management, diabetic retinopathy, diabetic neuropathy, diabetic nephropathy, the diabetic foot, diabetic skin, the diabetic pregnancy, and metabolic syndrome and insulin resistance.
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15

Berns, P. M. J. J., Romijn J. C i Schröder F. H, red. Mechanisms of progression to hormone-independent growth of breast and prostatic cancer. Carnforth, Lancs, UK: Parthenon Pub. Group, 1991.

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16

Lancellotti, Patrizio, i Bernard Cosyns. Systemic Disease and Other Conditions. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713623.003.0017.

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This chapter describes the effect of various activities on the heart and associated disorders. It details the echocardiographic findings of athlete’s heart and differential diagnosis. It considers pregnancy which induces several haemodynamic changes: increase in heart rate, stroke volume, cardiac output, and decrease in systemic vascular resistance. Several echocardiographic changes may also present in normal pregnancy and these must be recognized. Echocardiography should be performed in each pregnant woman with cardiac signs or symptoms to search for new cardiac disease occurring during pregnancy and especially peripartum cardiomyopathy. Pregnancy is well tolerated by most woman with cardiac disease. Pregnancy in contraindicated in woman with pulmonary hypertension. Although the heart is not the principal affected organ in systemic disease there is some involvement. This chapter also details the echo findings of a range of systemic diseases including amyloidosis, connective tissue disease, endocrine disease, and HIV.
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17

C Diaz, Eva, Celeste C Finnerty i David N. Herndon. Severe Burn Injuries and Their Long-Term Implications. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0016.

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Burn injury is notable for the degree and duration of pathophysiological alterations that it induces. Burn triggers profound changes in metabolism, immune function, and endocrine function, leading to a host of negative effects, including catabolism of muscle and bone and insulin resistance. These changes may persist or evolve for years after the injury has occurred, delaying recovery. This chapter discusses all of these consequences of burn injury, along with other adverse outcomes, specifically growth delay in children and hypertrophic scarring. Particular attention is placed on what is known about the mechanisms underlying each of these pathological changes and, in some cases, current practice in their management. A description is also provided of some of the pharmacologic (i.e. oxandrolone and recombinant human growth hormone) and non-pharmacologic (i.e. exercise therapy) approaches that hold promise in the treatment of burn injury and its consequences.
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18

Dussaule, Jean-Claude, Martin Flamant i Christos Chatziantoniou. Function of the normal glomerulus. Redaktor Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0044_update_001.

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Glomerular filtration, the first step leading to the formation of primitive urine, is a passive phenomenon. The composition of this primitive urine is the consequence of the ultrafiltration of plasma depending on renal blood flow, on hydrostatic pressure of glomerular capillary, and on glomerular coefficient of ultrafiltration. Glomerular filtration rate (GFR) can be precisely measured by the calculation of the clearance of freely filtrated exogenous substances that are neither metabolized nor reabsorbed nor secreted by tubules: its mean value is 125 mL/min/1.73 m² in men and 110 mL/min/1.73 m² in women, which represents 20% of renal blood flow. In clinical practice, estimates of GFR are obtained by the measurement of creatininaemia followed by the application of various equations (MDRD or CKD-EPI) and more recently by the measurement of plasmatic C-cystatin. Under physiological conditions, GFR is a stable parameter that is regulated by the intrinsic vascular and tubular autoregulation, by the balance between paracrine and endocrine agents acting as vasoconstrictors and vasodilators, and by the effects of renal sympathetic nerves. The mechanisms controlling GFR regulation are complex. This is due to the variety of vasoactive agents and their targets, and multiple interactions between them. Nevertheless, the relative stability of GFR during important variations of systemic haemodynamics and volaemia is due to three major operating mechanisms: autoregulation of the afferent arteriolar resistance, local synthesis and action of angiotensin II, and the sensitivity of renal resistance vessels to respond to NO release.
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