Gotowe bibliografie tematyczne / Endocrine aspects

Gotowa bibliografia na temat „Endocrine aspects”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 30 lipca 2024

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Artykuły w czasopismach na temat "Endocrine aspects"

Akopova, R. A., i T. V. Kokoreva. "ENDOCRINE ASPECTS OF MALE INFERTILITY". Bulletin "Biomedicine and sociology" 3, nr 4 (30.12.2018): 17–19. http://dx.doi.org/10.26787/nydha-2618-8783-2018-3-4-17-19.

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Fountoulakis, Stelios, i Agathocles Tsatsoulis. "Molecular genetic aspects and pathophysiology of endocrine hypertension". HORMONES 5, nr 2 (15.04.2006): 90–106. http://dx.doi.org/10.14310/horm.2002.11173.

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KALTENBACH, JANE C. "Endocrine Aspects of Homeostasis". American Zoologist 28, nr 2 (maj 1988): 761–73. http://dx.doi.org/10.1093/icb/28.2.761.

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Shalet, SM. "Endocrine aspects of cancer". British Journal of Cancer 70, nr 3 (wrzesień 1994): 569. http://dx.doi.org/10.1038/bjc.1994.347.

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Glass, Allan R. "Endocrine Aspects of Obesity". Medical Clinics of North America 73, nr 1 (styczeń 1989): 139–60. http://dx.doi.org/10.1016/s0025-7125(16)30696-4.

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Proietto, Joseph. "Endocrine aspects of obesity". Molecular and Cellular Endocrinology 316, nr 2 (marzec 2010): 103. http://dx.doi.org/10.1016/j.mce.2009.12.004.

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Sica, G. "Pathophysiologic and Endocrine Aspects". Journal of International Medical Research 18, nr 1_suppl (styczeń 1990): 8–10. http://dx.doi.org/10.1177/03000605900180s103.

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Drent, M. "Endocrine aspects of obesity". Netherlands Journal of Medicine 47, nr 3 (wrzesień 1995): 127–36. http://dx.doi.org/10.1016/0300-2977(95)00008-b.

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Vagenakis, A. G. "Endocrine aspects of menopause". Clinical Rheumatology 8, S2 (czerwiec 1989): 48–51. http://dx.doi.org/10.1007/bf02207233.

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Rapaport, Robert. "Endocrine Aspects of Aging". Endocrinology and Metabolism Clinics of North America 52, nr 2 (czerwiec 2023): xiii. http://dx.doi.org/10.1016/j.ecl.2023.02.007.

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Rozprawy doktorskie na temat "Endocrine aspects"

Ritchie, Catherine Marian. "Aspects of hypertension in endocrine disease". Thesis, Queen's University Belfast, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.357488.

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Delgado, Verdugo Alberto. "Genetic Aspects of Endocrine Tumorigenesis : A Hunt for the Endocrine Neoplasia Gene". Doctoral thesis, Uppsala universitet, Endokrinkirurgi, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-224111.

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Endocrine tumors arise from endocrine glands. Most endocrine tumors are benign but malignant variants exist. Several endocrine neoplasms display loss of parts of chromosome 11 or 18, produce hormones and responds poorly to conventional chemotherapeutics. The multiple endocrine neoplasia syndromes are mainly confined to endocrine tumors. This opens the question if there exists a single or several endocrine tumor genes. The aim of the study was to describe genetic derangements in endocrine tumors. Paper I: Investigation of mutational status of SDHAF2 in parathyroid tumors. SDHAF2 is located in the proximity of 11q13, a region that frequently displays loss in parathyroid tumors. We established that mutations in SDHAF2 are infrequent in parathyroid tumors. Paper II: Study of SDHAF2 gene expression in a cohort of benign pheochromocytomas (PCC) (n=40) and malignant PCC (n=10). We discovered a subset of benign PCC (28/40) and all malignant PCC (10/10) with significantly lower SDHAF2 expression. Benign PCC with low SDHAF2 expression and malignant tumors consistently expressing low levels of SDHAF2 were methylated in the promoter region. SDHAF2 expression was restored in vitro after treatment with 5- aza-2-deoxycytidine. Paper III: HumanMethylation27 array (Illumina) covering 27578 CpG sites spanning over 14495 genes were analyzed in a discovery cohort of 10 primary small neuroendocrine tumors (SI-NETs) with matched metastases. 2697 genes showed different methylation pattern between the primary tumor and its metastasis. We identified several hypermethylated genes in key regions. Unsupervised clustering of the tumors identified three distinct clusters, one with a highly malignant behavior. Paper IV: Loss of chromosome 18 is the most frequent genetic aberration in SI-NETs. DNA from SI-NETs were subjected to whole exome capture sequencing and high resolution SNP array. Genomic profiling revealed loss of chromosome 18 in 5 out of 7 SI-NETs. No tumor-specific somatic mutation on chromosome 18 was identified which suggests involvement of other mechanisms than point mutations in SI-NET tumorigenesis. Paper V: The cost for diagnostic genetic screening of common susceptibility genes in PCC is expensive and labor intensive. Three PCC from three patients with no known family history were chosen for exome capture sequencing. We identified three variants in known candidate genes. We suggest that exome-capture sequencing is a quick and cost-effective tool.

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Merani, Salima A. "Development of a specific and sensitive assay for cholecystokinin, and applications thereof". Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=37619.

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Cholecystokinin, or "CCK" peptides, originally identified in the gastrointestinal tract, are now considered to be one of the most abundant peptide systems in the mammalian central nervous system. Prompted by recent findings that implicated the cholecystokinergic system in the pathophysiology of various illnesses, we developed a novel assay system to measure the various forms of cholecystokinin peptides in human plasma and cerebrospinal fluid. The system detects CCK-4, sulfated CCK-8 (CCK-8s) and nonsulfated CCK-8 (CCK-8ns) with equal affinity, with the lower detection limit of 2.7 fmol and an ED50 of 10.6 +/- 2.2 fmol. Using the assay system, we determined that mean CCK-like immunoreactivity (CCK-LI) in the plasma of 12 healthy subjects was 12.9 +/- 2.1 pM CCK-4 equivalents.
After developing the cholecystokinin assay system, we were able to combine our unique methodology with other established techniques to investigate the role of CCK in illnesses such as premenstrual dysphoric disorder (PMDD), anxiety, bulimia nervosa, and cardiomyopathy.
Briefly, we observed no significant differences in plasma CCK levels between women with PMDD and healthy volunteers. However, we found that, independent of diagnosis, plasma cholecystokinin concentrations were higher in women during their first visit to the clinic to participate in the study, as compared to later visits.
In addition, application of our assay system allowed us to determine that oral ingestion of caffeine increased plasma CCK-LI levels 2--4 fold in humans. Moreover, we observed substantial variation in post-caffeine cholecystokinin levels among individuals.
In another study of cholecystokinin and anxiety, we used our CCK assay to determine the effects of ondansetron, a serotonin receptor antagonist, on cholecystokinin levels in plasma. We found that multiple oral doses of ondansetron influence the pharmacokinetic parameters of exogenous CCK.
We also used the three-step assay system to measure CCK-LI in patients with the eating disorder, bulimia nervosa. Baseline fasted cholecystokinin plasma levels were lower in bulimic women as compared to control subjects. However, at "satiety", or the post-binge stage, CCK levels in bulimic women were similar to those of control women.
Finally, our investigation into the role of cholecystokinin in cardiomyopathy revealed that neuronal cholecystokinin receptor density was altered in the cardiomyopathic hamster brain, as compared to age- and sex-matched control animals. (Abstract shortened by UMI.)

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Heaney, Anthony Patrick. "Aspects of pituitary and adrenal disease". Thesis, Queen's University Belfast, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295358.

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Henderson, Keith Meade. "Aspects of the endocrine control of ovarian function". Thesis, University of Edinburgh, 1991. http://hdl.handle.net/1842/14052.

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This thesis consists of papers, published by the candidate between 1975 and 1991, describing the results of investigations into the endocrine control of ovarian function. The papers are presented in chronological order in each of five sections. Each section deals with a different apsect of the research undertaken by the candidate during the sixteen year period. Section 2 is concerned with investigations of follicle development and follicle steroidogenesis. Papers concerned with inhibin are presented in section 3. The papers in section 4 describe studies to investigate the mechanism(s) responsible for the high ovulation rates observed in Booroola ewes carrying a fecundity (F) gene. Section 5 contains papers related to the manipulation of ovulation rate.

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Meloni, T. "SOME PERINATAL ENDOCRINE AND MORPHOLOGICAL ASPECTS OF CANINE SPECIES". Doctoral thesis, Università degli Studi di Milano, 2015. http://hdl.handle.net/2434/265723.

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Despite the huge literature about dog medicine, the perinatal period of growth and development and possible perturbations of the health of fetuses and newborns are still scarcely investigated, but with a recent, increasing scientific interest. The present thesis was therefore aimed to investigate some perinatal aspects of canine perinatology. Firstly, the study focused on the presence of some growth and metabolic factors and protective substances in dog fetal fluids at term of normal pregnancy. About growth and metabolic factors, the first study showed that insulin-like growth factors I (IGF-I) were higher in amniotic (AM) than allantoic (AL) fluid and the effect of breed size, even if opposite, on both IGF-I and non-esterified fatty acids (NEFA) was evidenced. The findings suggested that AM IGF-I could be used as an indicator of growth potential in canine species, whereas AM NEFA could work as a marker of fat mobilization for an energy request. The second study on fetal fluids, aimed to assess AM and AL IgG and lysozyme concentrations, documented higher IgG levels in AM than AL fluid, as a consequence of a supposed direct fetal IgG production, whereas no significant differences were found in lysozyme values between the two fluids. Maternal parity was demonstrated to affect IgG concentrations. A second area of study was the assessment of newborns hair and nails usefulness as the newest non invasive matrices for a long-term retrospective investigation of the Hypothalamic-Pituitary-Adrenal axis activation, by cortisol (C) concentrations analysis. The study evidenced that C was detectable in both matrices, with significant higher values in premature puppies compared to term-born dead puppies or puppies dead within the first 30 days of age. Furthermore, because of the newborn puppy high susceptibility to bacterial infection, often followed by death, the fourth study investigated the bacterial involvement in canine neonatal mortality, demonstrating that bacterial infections, above all by E. coli, alone or in association with other bacteria, represent an important cause of neonatal losses. In addition, the study highlighted the importance of the antimicrobial susceptibility test in case of suspected neonatal bacterial infection for a more targeted therapy of surviving litter-mates and for a better management of further gestations in bitch with previous neonatal mortality. The last study was designed to investigate some aspects of skeletal development during the neonatal period in the attempt to provide further knowledge about the first month of skeletal growth in puppies, but also aimed to assess the possible use of some radiographic, morphometric, and anatomic parameters for the age estimation in newborn dogs. The study proved that the neonatal growth occurs gradually as the age progresses, and simultaneously in the body, limbs, and skull. The radiographic evaluation of the ossification centers appearance resulted a useful tool to estimate the neonatal age in puppies, even if during the first 14 days of age significant skeletal changes were not observed. In this respect, the radiographic and anatomic measures of the hindlimb long bones and skull length seem to provide better guarantees; specifically, the neurocranium width as well as tibial and femoral lengths resulted the most correlated measurements with the age. Thus, in the future radiographic and/or anatomical morphometry of limbs and skull could become the best tool for neonatal age estimation.

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Behera, Malabika. "Long term endocrine sequelae of childhood cancer survivors". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/206611.

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Background: Newer multimodality therapeutic interventions have resulted in dramatic survival rates in childhood cancers. However diverse treatment related morbidities affect the long term survivors. An Endocrine complication comprises 20-40% of these morbidities and affects the hypothalamic pituitary axis, growth, pubertal progression, fertility, bone health and glucose homeostasis. Objectives: The aim of our study was to enumerate and evaluate the frequency of endocrine complications arising as a late effect of treatment in childhood cancer survivors. Risk factors likely to be associated with these complications were also evaluated. Methodology: Retrospective analysis of medical records from the Long Term Endocrine Follow up clinic in the Department of Paediatrics and Adolescent Medicine of Queen Mary Hospital was done. Patients with a primary diagnosis of Cancer and Langerhans cell histiocytosis with endocrine sequelae arising from various treatment modalities who have survived 5 years after diagnosis were included in the study. Those who had endocrine complications arising from various treatment modalities for Thalassemia’s, Immunodeficiency’s were excluded from the study Results: 135 cases were included in the study and 27 were excluded. Leukemia and Brain tumor survivors were the majority accounting for 40% and 26.67% respectively. ALL formed majority of leukemia survivors, Medulloblastoma survivors accounted for 50% of brain tumor survivors. Most common endocrine problem was Hypogonadism in 51.1% of cases, followed by growth disturbances in 40%, Thyroid dysfunction in 23% and Hyperlipidemias in 18.5%. Pubertal problems, Central Diabetes Insipidus, Adrenal insufficiency, Obesity, Altered glucose homeostasis were rest of the problems in small frequencies. PHGN (Primary Hypogonadism) was present in 91.3% and mostly in prepubertal males. PHGN was statistically associated with Leukemia survivors with OR-2.06 (1.02- 4.15), p value 0.04. The risk factors associated were exposure to alkylating agents, radiotherapy, TBI prior to transplant. SHGN (Secondary HGN) was statistically associated with Brain tumor survivor OR - 15.8 (1.7-140.5), p value 0.013. Cranial irradiation was the major risk for SHGN. PGV (Poor growth velocity) was the major growth problem.GHD (Growth Hormone Deficiency) had a highly significant association with Brain tumors (p value ˂ 0.0001), and significantly associated when all 3 modalities of treatment given together (p value 0.01). Risk factors for GHD were cranial radiotherapy, exposure to cyclophosphamide and TBI. PH (Primary Hypothyroidism) had highly significant association with craniospinal radiotherapy (p value ˂ 0.0001), and significantly associated with brain tumors. Similar results were observed in patients of CH (Central Hypothyroidism). Hyperlipidemias were present in 18% with no statistical correlation with the type of cancer. Brain tumor survivors had a significant association of GHD, PH, CH, SHGN and CDI. Leukemia survivors had significant association with GHD and PHGN. Conclusions: Endocrine problems are frequent manifestations of late effects of cancer related treatments. Early detection and intervention of these potentially treatable problems could be done through structured long term surveillance. Increasing awareness among health care professionals to anticipate problems in suspected patients and education of patients would optimize health care and quality of life.
published_or_final_version
Paediatrics and Adolescent Medicine
Master
Master of Medical Sciences

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Fjällskog, Marie-Louise. "Current Medical Treatment of Endocrine Pancreatic Tumors and Future Aspects". Doctoral thesis, Uppsala University, Department of Medical Sciences, 2002. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-2709.

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We treated 16 patients with somatostatin analogs combined with α-interferon and achieved a biochemical and/or radiological response in 56% (median duration 22 months). We consider this treatment a good alternative for patients who fail during chemotherapy or who do not want to/cannot receive cytotoxic drugs.

Thirty-six patients with neuroendocrine tumors were treated with cisplatin combined with etoposide. Of 14 patients with evaluable EPTs, 50% responded radiologically and/or biochemically (median duration 9 months). We consider this treatment useful as first-line medical treatment in aggressive EPTs or in patients failing prior chemotherapy.

Twenty-eight tumor tissues from EPTs were examined with immunohistochemistry regarding expression of somatostatin receptors (ssts) 1 to 5 on tumor cells and in intratumoral vessels. We found that sst₂ and sst₄ were highly expressed on tumor cells and in vessels. However, sst₃ and sst₅ were lacking in half of the tumor tissues and in most of the vessels. Because of the variability in sst expression, we recommend analysis of each individual’s receptor expression before starting treatment.

Endocrine pancreatic tumors (EPTs) are rare with an incidence of 4 per million inhabitants. In the majority of cases they grow slowly, but there are exceptions with very rapidly progressing malignant carcinomas. First-line medical treatment is streptozotocin combined with 5-fluorouracil.

We examined 38 tumor samples regarding expression of tyrosine kinase receptors platelet-derived growth factor receptors (PDGFRs), c-kit and epidermal growth factor receptor (EGFR). We found that the receptors were expressed in more than half of the tumor tissues. Further studies will reveal if tyrosin kinase antagonists can be part of the future treatment arsenal.

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Chu, Xia. "Aspects of MEN1 Tumorigenesis in Endocrine Pancreas and Adrenal Glands". Doctoral thesis, Uppsala universitet, Endokrin Onkologi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-254817.

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Multiple endocrine neoplasia syndrome type 1 (MEN1) is an autosomal dominantly inherited disease, which is described as an association of tumors mainly in endocrine organs, including pancreas and adrenal glands. Pancreatic neuroendocrine tumors (PNETs) are the most common cause of death in MEN1 patients. More than one third of the MEN1 patients also develop enlargement of the adrenals. MEN1 is caused by a germline mutation of MEN1 gene, a tumor suppressor gene that is located on the human chromosome 11. As noticed, the MEN1 related tumors often develop prior to inactivation of both wild type alleles, indicating MEN1 haploinsufficiency. In this thesis, I utilized a conventional Men1 mouse model that has the phenotype mimicking the human MEN 1 traits, in order to investigate MEN1 tumorigenesis in endocrine pancreas and adrenal glands. The microvascular aberrations contributing to development and maintenance of PNETs were characterized. The increased vascular density of PNETs developed in the Men1 mice was paralleled by an early and extensive redistribution of pericytes within endocrine tissue. These morphological alterations were supported by fine-tuned variations in expression of several angiogenic regulators (VEGF, FGF and PDGF) and were further potentiated by hypoxia. Vascular reactivity and blood perfusion of tumor arterioles were significantly altered in response to glucose and L-nitro-arginine methyl ester. Investigation of adrenals from10-month-old Men1 mice showed 681 proteins in mass spectrometry data sets, in which 52 proteins were commonly found in the Men1+/+ and Men1+/- adrenals, and the differential expression between the genotypes reached significant levels. Prdx3, catalyzing the reduction of oxidative stress to cell survival, is one of the overexpressed proteins. Some proteins belonging to the PPARα pathway, e.g. ACLY were also overexpressed. Subsequent microRNA (miRNA) profiling analysis of adrenals from the same age group revealed 31 miRNAs whose expression was significantly altered in comparison between the genotypes. The tumor suppressor miRNAs, miR-486, miR-330 and miR-214, were significantly downregulated in Men1+/- adrenals. The latter, miR-214, is known to inhibit ACLY expression. This finding was in concordance with the proteomic analysis. The oncogene miRNAs, miR-132 and miR-494, were significantly enhanced in the Men1+/- adrenals. Gene ontology analysis demonstrated overrepresentation of the miRNA-targeted genes that are involved in nucleic acid metabolism, vasculature development, angiogenesis, and transcription. Together, these finding after validation in humans may be exploited to improve MEN1 cancer treatment.

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譚銓株 i Chuen-chu Tam. "Hormonal effects of the lateral prostate and seminal vesicle of the guinea pig: an ultrastructural, morphometricand cytochemical study". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1989. http://hub.hku.hk/bib/B3123169X.

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Książki na temat "Endocrine aspects"

Károly, Lapis, Eckhardt S i International Union against Cancer, red. Endocrine aspects of malignancies. Budapest: Akadémiai Kiadó, 1987.

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1942-, Geelhoed Glenn W., i Chernow Bart, red. Endocrine aspects of acute illness. New York: Churchill Livingstone, 1985.

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Gosney, John R. Pulmonary endocrine pathology: Endocrine cells and endocrine tumors of the lung. Oxford: Butterworth-Heinemann, 1992.

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George, Kahaly, red. Endocrine ophthalmopathy: Molecular, immunological and clinical aspects. Basel: Karger, 1993.

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1948-, Andò S., i Brancati C. 1924-, red. Endocrine disorders in thalassemia: Physiopathological and therapeutic aspects. Berlin: Springer-Verlag, 1995.

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G, Biglieri Edward, i Melby James C, red. Endocrine hypertension. New York: Raven Press, 1990.

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Balabolkin, M. I. Emergency states in endocrinology: Clinical aspects and treatment. Moscow: Mir Publishers, 1987.

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R, Thompson Lester D., red. Endocrine pathology. Philadelphia, PA: Churchill Livingstone Elsevier, 2006.

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Masaru, Wada, Ishii Susumu 1931- i Scanes C. G, red. Endocrinology of birds: Molecular to bahavioral. Tokyo: Japan Scientific Societies Press, 1990.

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International Congress of Neuroendocrinology (1st 1986 San Francisco, Calif.). Integrative neuroendocrinology: Molecular, cellular, and clinical aspects. Redaktorzy McCann S. M. 1925- i Weiner Richard I. Basel: Karger, 1987.

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Części książek na temat "Endocrine aspects"

Vujović, Svetlana, M. Ivović, M. Tančić Gajić, L. J. Marina, Z. Arizanović, M. Barać, S. Popovic i in. "Transsexualism: Endocrine Aspects". W ISGE Series, 47–52. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-09662-9_6.

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Lloyd, Ricardo V. "Endocrine Pathology". W Pathology: Historical and Contemporary Aspects, 127–44. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-39554-3_13.

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Pozzilli, Paolo, Rocky Strollo i Nicola Napoli. "Drug-Induced Endocrine Autoimmunity". W Immunoendocrinology: Scientific and Clinical Aspects, 157–79. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-478-4_10.

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Misra, Madhusmita. "Endocrine Aspects of Anorexia Nervosa". W Handbook of Famine, Starvation, and Nutrient Deprivation, 1–19. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-40007-5_115-1.

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Misra, Madhusmita. "Endocrine Aspects of Anorexia Nervosa". W Handbook of Famine, Starvation, and Nutrient Deprivation, 603–21. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-55387-0_115.

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Kam, Peter, Ian Power, Michael J. Cousins i Philip J. Siddal. "General Aspects of Endocrine Physiology". W Principles of Physiology for the Anaesthetist, 361–65. Fourth edition. | Boca Raton : CRC Press, Taylor & Francis Group, 2020.: CRC Press, 2020. http://dx.doi.org/10.1201/9780429288210-59.

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Griffiths, K., M. E. Harper, C. L. Eaton, A. Turkes i W. B. Peeling. "Endocrine Aspects of Prostate Cancer". W Prostate Cancer 2000, 21–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-79178-9_5.

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van den Beld, Annewieke W., i Steven W. J. Lamberts. "Endocrine Aspects of Healthy Ageing in Men". W Endocrine Facets of Ageing, 3–25. Chichester, UK: John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/0470846542.ch2.

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Mereiles, Leonardo, Marcella Li, Danielle Loo i Edwin Liu. "Celiac Disease and Intestinal Endocrine Autoimmunity". W Immunoendocrinology: Scientific and Clinical Aspects, 535–45. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-478-4_32.

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Holt, Richard I. G., i Peter H. Sönksen. "Endocrine Aspects of the Metabolic Syndrome". W The Metabolic Syndrome, 120–38. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444347319.ch8.

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Streszczenia konferencji na temat "Endocrine aspects"

Yaglova, N. V., S. S. Obernikhin i V. V. Yaglov. "DEUTERIUM: REGULATOR OR DISRUPTOR OF THYROID FUNCTION?" W NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. LLC Institute Information Technologies, 2023. http://dx.doi.org/10.47501/978-5-6044060-3-8.35-39.

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The article discusses novel data on impact of altered deuterium body content on endocrine function. The paper represents the results of investigation of thyroid hormones and pituitary thyroid stimulating hormone production during gradual increase of deuterium body content. The research revealed sensitivity of the endocrine glands to changes in deuterium content. The revealed findings are considered from the point of view of the regulatory and dysregula-tory aspects of action of the hydrogen isotope on hormone genesis.

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VLAS, Irina. "The study of stress at young people". W Ştiință și educație: noi abordări și perspective. "Ion Creanga" State Pedagogical University, 2023. http://dx.doi.org/10.46727/c.v1.24-25-03-2023.p43-46.

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Stress and stressful events are part of our life. Until recently, it was considered that stress is not characteristic for young people, they have a normal life without special pressures. Nowadays, the conditions of modern life, the speed with which events are happening, consumer society, problems at work, in love relationship, failures and unsuccess determine the manifestation of stress among young people. We are witnessing an increase number of young people experiencing an intense level of stress. Stress is the response to certain external and / or internal factors that may have a physical, biological or psychological nature. Stress can have negative consequences on physical and mental health. A high level of stress changes the body’s function and influence on immune system, nervous system, cardiac system and endocrine system, etc. On psychological level stress causes changes in thinking, emotions and behaviour and can produce psychological discomfort, emotional instability, neuroticism. When stress becomes chronic it affects important aspects of young people life: work, rest time and social relationships.

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Sharma, Katyayani, Angelo Lanzilotto, Søren Therkelsen, Therina du Toit, Flemming S. Jørgensen i Amit Pandey. "Endocrine Disrupting Chemicals as "structural cues" for the development of drugs for the treatment of Prostate Cancer (Castration Resistant Prostate Cancer) and PCOS". W ASPET 2023 Annual Meeting Abstracts. American Society for Pharmacology and Experimental Therapeutics, 2023. http://dx.doi.org/10.1124/jpet.122.163500.

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Raporty organizacyjne na temat "Endocrine aspects"

Cnaani, Avner, Gordon Grau, Darren Lerner i Sheenan Harpaz. Gastrointestinal osmoregulatory activity in Tilapia and its effects on growth, an opportunity for fish diet developments. United States Department of Agriculture, lipiec 2014. http://dx.doi.org/10.32747/2014.7594393.bard.

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Fish living in freshwater and seawater environments experience constant osmotic pressure between their internal body and the surrounding water. Regulation of ion and water balance under these conditions is highly energetic demanding, and eventually, affects the fish growth. While the role of the gills in osmoregulation was extensively studied, the osmoregulatory activity of the gastrointestinal tract is less known. In this study we characterized the tilapia intestine as a multifunctional organ, having a role in both nutrition and in ion regulation. We studied the pituitary endocrine regulation of intestinal salinity adaptation, the salinity-dependent physiological activity along different intestinal sections, and specific genes that are linking nutrient absorption with ion and acid-base regulation. The results of this study indicate that different intestinal sections developed various specific activities. Their endocrine regulation is now better understood, a large data-set of salinity dependent gene transcript was developed, as well as new tools and methods to study new aspects of intestinal physiology.

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