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Książki na temat „E.coli”

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Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 31 lipca 2024

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1

Burgess-Brown, Nicola A., red. Heterologous Gene Expression in E.coli. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6887-9.

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2

Evans,, Thomas C., i Ming-Qun Xu, red. Heterologous Gene Expression in E.coli. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-61737-967-3.

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3

undifferentiated, Christopher Yates. Copper resistance proteins from E.coli. Birmingham: University of Birmingham, 1998.

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4

Ownis, Ali A. Verocytotoxin expression by E.coli O157:H7. [s.l.]: typescript, 1999.

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5

R, Palmer Stephen, red. E.coli: Environmental health issues of VTEC O157. New York: Spon Press, 2002.

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6

R, Palmer Stephen, red. E.coli: Environmental health issues of VTEC O157. New York: Spon Press, 2002.

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7

Stocks, Stuart Michael. The flocculation of high concentration cell debris from E.coli. Birmingham: University of Birmingham, 1997.

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8

Cox, Graham L. Determination: Into the E.Coli 0 157 fatal accident inquiry. [Airdrie}: s.n., 1999.

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9

McNeish, Iain Alexander. Virus directed enzyme prodrug therapy using E.COLI nitroreductase and CB1954. Birmingham: University of Birmingham, 1998.

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10

Schofield, Richard. E.coli 0157: Private wealth V public wealth? : a public interest report. Bolton: Bolton Business School, 1998.

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11

Evans, Paul David. The interactions of the E.coli trp repressor with its operator DNA sequences. Birmingham: University of Birmingham, 1995.

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12

Heersink, Mary. E.coli O157: The true story of a mother's battle with a killer microbe. Far Hills, NJ: New Horizon Press, 1996.

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13

Boeckh, Clemens. Expression der frühen Funktionen des mutagenen E.coli-Bakteriophagen Mu und ihre Wirkung auf die Wirtszellen. Konstanz: Hartung-Gorre, 1986.

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14

Department, Scotland Health. Scottish Executive and Food Standards Agency response to the report of the Task Force E.coli 0157. Edinburgh: Scottish Executive, 2002.

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15

Zürich, Universität, red. Expression of the recombinant sea-urchin metallothionein gene in E.coli and determination of the structure of the recombinant protein. [s.l.]: [s.n.], 1995.

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16

Parker, Louise Michelle. Assessing the relationship between animal cleanliness and carcse faecal contamination as indicated by E.coli levels in beef cattle: A dissertation submitted in partial fulfilment of the Bachelor of Technology (honours) degree in Food production and Land. Ayr: SAC Auchincruive, 1998.

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17

Gutstein, Donald. E.con: How the Internet undermines democracy. Toronto: Stoddart, 1999.

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18

Eric, Olijdam, Spoor Richard H i During Caspers, Elisabeth C. L., red. Intercultural relations between south and southwest Asia: Studies in commemoration of E.C.L. During Caspers (1934-1996). Oxford: Archaeopress, 2008.

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19

Palmer, Stephen, i Sharon Parry. E.coli. Routledge, 2002. http://dx.doi.org/10.4324/9780203302712.

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20

Bagel, Simone. Adhärenzeigenschaften chinolonresistenter E.coli-Isolate. 1998.

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21

Palmer, Stephen. E.coli: Environmental Health Issues of VTEC 0157. Taylor & Francis, 2002.

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22

Derichs, Ute. Untersuchungen zum antibakteriellen Einfluss von Mukosaepithelzellen und Modulatoren der E.coli-Epithelzellinteraktion: Etablierung einer standardisierten Methode zur Messung der E.coli-Proliferation. 2000.

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23

Giles, Suzanne L. The natural history of E.coli in Samish Bay, Washington. 1996.

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24

Hofmeister, Susanne. In Vivo-Aktivität von Apalcillin und Piperacillin bei E.Coli. 1987.

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25

Gutenberg-Universität, Johannes, red. Hämolytische E.coli wirken nach Phagozytose zytotoxisch auf polymorphkernige neutrophile Granulozyten. 1998.

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26

Heterologous Gene Expression in E.coli: Methods and Protocols (Methods in Molecular Biology). Humana Press , 2017.

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27

Münster, Universität, red. Die Wirkung des Gyrase-Hemmers Norfloxacin auf die akute experimentelle E.Coli Pyelonephritis. 1987.

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28

Jehser, Thomas. Einfluss der frühen Infektion durch E.coli auf den Verlauf einer tierexperimentellen nekrotisierenden Pankreatitis. 1995.

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29

Lamden, Ken, Sam Rowell i Andrew Fox. Vero cytotoxigenic Escherichia coli (VTEC) O157. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198745471.003.0005.

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This chapter describes a cluster of diarrhoeal disease caused by E.coli O157 in a case scenario involving infection following a farm visit, and possible subsequent spread within a nursery. Background information on the epidemiology of E.coli O157 indicates the role of farm contact in infection, and the risk factors for transmission. In addition to providing clear case definitions, the risk groups of contacts are described in detail. ‘Top tips’ are given, to assist the health protection practitioner take timely and appropriate action, and ‘tools of the trade’ list the laboratory, epidemiological, and veterinary components of the investigation. The statutory safety legislation required at the source farm is explained, and the importance of communication with the affected families is emphasized. The final part of the chapter considers other possible scenarios, including the possibility of food from the farm café being the source.
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30

Götzelmann, Astrid. Einfluss des CAPD-Dialysats auf das Spontanwachstum von E.coli und auf die Wirksamkeit von Antibiotika. 2002.

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31

Bergens, Kai-Uwe. Die Wirkung von RO 23-6240 (AM 833) auf die experimentelle E.coli-Pyelonephritis nach subkutaner Applikation. 1988.

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32

Holliger, Philipp. Multivalent and bispecific antibody fragments from E.coli: New strategies for antibody-based diagnostics and therapeutics from bacteria. 1994.

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33

Main, Universität Frankfurt am, red. Über die Verwendung des Brillantgrün-Indol-Mannit-Nährbodens zum Nachweis von E.coli aus Oberflächen-, Trink- und Badewasserproben. 1989.

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34

Lösche, Christian. Die keimreduzierende Wirkung von Ro15-8075/006 (Cefetamet pivoxyl) und Cefaclor in hoher Dosierung auf die experimentelle E.coli-Pyelonephritis. 1989.

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35

Dept.of Environment. An Evaluation of Presence/Absence Tests for Coliform Ores and E.Coli (Methods for the Examination of Waters & Associated Minerals). Stationery Office Books, 1996.

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36

Pugh, Mark Timothy. Peripheral blood and synovial fluid immune responses to infectious agents and the E.coli Chaperone GroEL in children with juvenile chronic arthritis. 1996.

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37

Mah, Thien-Fah. A structure/function analysis of the interaction of the E.coli NusA protein with RNA polymerase, the phage [lambda] N protein, and nut site RNA. 1999.

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38

Palmer, Stephen. Deliberate release of zoonotic agents. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0002.

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Since 9/11 2001, international attention has once again focused on the risks to human and animal health from the deliberate release of infectious or toxic chemical agents. In theory any agent could be used by terrorists and disaffected people, but the most serious risk for infectious agents are mainly zoonotic (Franz et al. 1997). Three modes of exposure may be anticipated, inhalation of powder or spray or dust from explosives, direct contact or inoculation from an explosion, and ingestion. Centers for Disease Control (CDC) list 19 bioterrorism agents or groups of agents of which 14 are zoonotic. In Category A are 6 agents which can be easily disseminated or transmitted from person to person, that result in high mortality rates and have the potential for major public health impact, which might cause public panic and social disruption and which require special action from public health preparedness. Of these 6, four are zoonoses — Anthrax, Plague, Tularaemia and Viral Haemorrhagic Fevers. In Category B, are 12 groups of agents, which are moderately easy to disseminate and cause moderate morbidity. Of these 12 groups, 8 contain zoonoses: Brucellosis, Food Safety threats (e.g. Salmonella, E.coli 0157, Campylobacter), Meliodiosis, Psittacoccosis, Q Fever, Typhus, Viral encephalitis, Water safety threats (e.g. Cryptosporidium).
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39

A genetic switch: Gene control and phage [lamda]. Palo Alta., Calif: Cell Press & Blackwell Scientific Publications, 1986.

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40

Noris, Marina, i Tim Goodship. The patient with haemolytic uraemic syndrome/thrombotic thrombocytopenic purpura. Redaktor Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0174.

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The patient who presents with microangiopathic haemolytic anaemia, thrombocytopenia, and evidence of acute kidney injury presents a diagnostic and management challenge. Haemolytic uraemic syndrome (HUS) and thrombotic thrombocytopenic purpura (TTP) are two of the conditions that frequently present with this triad. They are characterized by low platelet count with normal or near-normal coagulation tests, anaemia, and signs of intravascular red cell fragmentation on blood films, and high LDH levels.HUS associated with shiga-like toxins produced usually by E.coli (typically O157 strains) may occur in outbreaks or sporadically, with geographical variations in incidence. It is predominantly a disease of young children in which painful blood diarrhoea in a minority of infected patients is succeeded by microangiopathy and acute kidney injury. Management is supportive and recovery is usual, although permanent renal damage may lead to later deterioration. Older patients may be affected and tend to have worse outcomes. Neuraminidase-producing Streptococcus pneumoniae infections (usually pneumonia) very rarely cause a similar HUS.Atypical HUS occurs sporadically and is increasingly associated with defects in the regulation of the complement pathway, either genetic or autoimmune-caused. It may respond to plasma exchange for fresh frozen plasma. Recurrences are common, including after transplantation.TTP is associated with more neurological disease and less renal involvement, but HUS and TTP overlap substantially in their manifestations. The underlying problem is in von Willebrand factor (vWF) cleavage. The plasma metalloprotease ADAMTS13 is responsible for cleaving vWF multimers, a process that is important to prevent thrombosis in the microvasculature. Autoantibodies or rarely genetic deficiency may impair this process. Plasma exchange may remove antibodies and replenish the protease.
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41

Gutstein, Donald. E.Con: How the Internet Undermines Democracy. Stoddart, 1999.

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42

Connor, Eddie. E.CON the ICON: From Pop Culture to President Barack Obama. Norbrook Publishing, 2015.

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