Gotowa bibliografia na temat „Dystrophin”
Utwórz poprawne odniesienie w stylach APA, MLA, Chicago, Harvard i wielu innych
Spis treści
Zobacz listy aktualnych artykułów, książek, rozpraw, streszczeń i innych źródeł naukowych na temat „Dystrophin”.
Przycisk „Dodaj do bibliografii” jest dostępny obok każdej pracy w bibliografii. Użyj go – a my automatycznie utworzymy odniesienie bibliograficzne do wybranej pracy w stylu cytowania, którego potrzebujesz: APA, MLA, Harvard, Chicago, Vancouver itp.
Możesz również pobrać pełny tekst publikacji naukowej w formacie „.pdf” i przeczytać adnotację do pracy online, jeśli odpowiednie parametry są dostępne w metadanych.
Artykuły w czasopismach na temat "Dystrophin"
Straub, Volker, Jill A. Rafael, Jeffrey S. Chamberlain i Kevin P. Campbell. "Animal Models for Muscular Dystrophy Show Different Patterns of Sarcolemmal Disruption". Journal of Cell Biology 139, nr 2 (20.10.1997): 375–85. http://dx.doi.org/10.1083/jcb.139.2.375.
Pełny tekst źródłaCui, Chang-Hao, Taro Uyama, Kenji Miyado, Masanori Terai, Satoru Kyo, Tohru Kiyono i Akihiro Umezawa. "Menstrual Blood-derived Cells Confer Human Dystrophin Expression in the Murine Model of Duchenne Muscular Dystrophy via Cell Fusion and Myogenic Transdifferentiation". Molecular Biology of the Cell 18, nr 5 (maj 2007): 1586–94. http://dx.doi.org/10.1091/mbc.e06-09-0872.
Pełny tekst źródłaSteen, Michelle S., Marvin E. Adams, Yan Tesch i Stanley C. Froehner. "Amelioration of Muscular Dystrophy by Transgenic Expression of Niemann-Pick C1". Molecular Biology of the Cell 20, nr 1 (styczeń 2009): 146–52. http://dx.doi.org/10.1091/mbc.e08-08-0811.
Pełny tekst źródłaPeter, Angela K., Jamie L. Marshall i Rachelle H. Crosbie. "Sarcospan reduces dystrophic pathology: stabilization of the utrophin–glycoprotein complex". Journal of Cell Biology 183, nr 3 (3.11.2008): 419–27. http://dx.doi.org/10.1083/jcb.200808027.
Pełny tekst źródłaChen, Yi-Wen, Po Zhao, Rehannah Borup i Eric P. Hoffman. "Expression Profiling in the Muscular Dystrophies". Journal of Cell Biology 151, nr 6 (11.12.2000): 1321–36. http://dx.doi.org/10.1083/jcb.151.6.1321.
Pełny tekst źródłaYeadon, J. E., H. Lin, S. M. Dyer i S. J. Burden. "Dystrophin is a component of the subsynaptic membrane." Journal of Cell Biology 115, nr 4 (15.11.1991): 1069–76. http://dx.doi.org/10.1083/jcb.115.4.1069.
Pełny tekst źródłaTeramoto, Naomi, Hidetoshi Sugihara, Keitaro Yamanouchi, Katsuyuki Nakamura, Koichi Kimura, Tomoko Okano, Takanori Shiga i in. "Pathological evaluation of rats carrying in-frame mutations in the dystrophin gene: a new model of Becker muscular dystrophy". Disease Models & Mechanisms 13, nr 9 (28.08.2020): dmm044701. http://dx.doi.org/10.1242/dmm.044701.
Pełny tekst źródłaSpaulding, HR, C. Ballmann, JC Quindry, MB Hudson i JT Selsby. "Autophagy in the heart is enhanced and independent of disease progression in mus musculus dystrophinopathy models". JRSM Cardiovascular Disease 8 (styczeń 2019): 204800401987958. http://dx.doi.org/10.1177/2048004019879581.
Pełny tekst źródłaIbrahim Sory, P., T. Sidi, L. Guida, K. Boureima, M. Alassane Bameye, T. Mohomodine Ibrahim, K. Abdoulaye i C. Idrissa Ahmadou. "Dystrophie Musculaire de Duchenne: Aspects cliniques, biologiques et évolutifs à propos de cinq cas dans le service de Rhumatologie au CHU du Point G." Rhumatologie Africaine Francophone 6, nr 2 (19.01.2024): 18–23. http://dx.doi.org/10.62455/raf.v6i2.53.
Pełny tekst źródłaZabłocka, Barbara, Dariusz C. Górecki i Krzysztof Zabłocki. "Disrupted Calcium Homeostasis in Duchenne Muscular Dystrophy: A Common Mechanism behind Diverse Consequences". International Journal of Molecular Sciences 22, nr 20 (13.10.2021): 11040. http://dx.doi.org/10.3390/ijms222011040.
Pełny tekst źródłaRozprawy doktorskie na temat "Dystrophin"
Gaschen, Lorrie. "Cardiomyopathy in dystrophin-deficient hypertrophic feline muscular dystrophy /". [S.l.] : [s.n.], 1998. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.
Pełny tekst źródłaHoward, Judith. "Electrodiagnostic evaluation of dystrophin-deficient hypertrophic feline muscular dystrophy /". [S.l.] : [s.n.], 2000. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.
Pełny tekst źródłaThorley, Matthew. "Analysis of the dystrophin interactome". Thesis, Paris 6, 2016. http://www.theses.fr/2016PA066619/document.
Pełny tekst źródłaThe aim of this project was to systematically identify new interaction partners of the dystrophin protein within differentiated human skeletal muscle cells in order to uncover new roles in which dystrophin is involved, and to better understand how the global interactome is affected by the absence of dystrophin. hTERT/cdk4 immortalized myogenic human cell lines represent an important tool for skeletal muscle research however, disruption of the cell cycle has the potential to affect many other cellular processes to which it also linked. A transcriptome-wide analysis of healthy and diseased lines comparing immortalized lines with their parent primary populations in both differentiated and undifferentiated states testing their myogenic character by comparison with non-myogenic cells found that immortalization has no measurable effect on the myogenic cascade or on any other cellular processes, and that it was protective against the senescence. In this context the human muscle cell lines are a good in vitro model to study the dystrophin interactome. We investigated dystrophin’s interactors using the high-sensitivity proteomics ‘QUICK’ approach. We identified 18 new physical interactors of dystrophin which displayed a high proportion of vesicle transport related proteins and adhesion proteins, strengthening the link between dystrophin and these roles. The proteins determined through previously published data together with the newly identified interactors were incorporated into a web-based data exploration tool: sys-myo.rhcloud.com/dystrophin-interactome, intended to provide an easily accessible and informative view of dystrophins interactions in skeletal muscle
Acharyya, Swarnali. "Elucidating molecular mechanisms of muscle wasting in chronic diseases". Columbus, Ohio : Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1180096565.
Pełny tekst źródłaThorley, Matthew. "Analysis of the dystrophin interactome". Electronic Thesis or Diss., Paris 6, 2016. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2016PA066619.pdf.
Pełny tekst źródłaThe aim of this project was to systematically identify new interaction partners of the dystrophin protein within differentiated human skeletal muscle cells in order to uncover new roles in which dystrophin is involved, and to better understand how the global interactome is affected by the absence of dystrophin. hTERT/cdk4 immortalized myogenic human cell lines represent an important tool for skeletal muscle research however, disruption of the cell cycle has the potential to affect many other cellular processes to which it also linked. A transcriptome-wide analysis of healthy and diseased lines comparing immortalized lines with their parent primary populations in both differentiated and undifferentiated states testing their myogenic character by comparison with non-myogenic cells found that immortalization has no measurable effect on the myogenic cascade or on any other cellular processes, and that it was protective against the senescence. In this context the human muscle cell lines are a good in vitro model to study the dystrophin interactome. We investigated dystrophin’s interactors using the high-sensitivity proteomics ‘QUICK’ approach. We identified 18 new physical interactors of dystrophin which displayed a high proportion of vesicle transport related proteins and adhesion proteins, strengthening the link between dystrophin and these roles. The proteins determined through previously published data together with the newly identified interactors were incorporated into a web-based data exploration tool: sys-myo.rhcloud.com/dystrophin-interactome, intended to provide an easily accessible and informative view of dystrophins interactions in skeletal muscle
Pearce, Marcela. "Genomic structure of the human utrophin gene". Thesis, University of Oxford, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318897.
Pełny tekst źródłaCoovert, Daniel David. "Analysis of dystrophin in duchenne muscular dystrophy and SMN in spinal muscular atrophy /". The Ohio State University, 1998. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487951595500021.
Pełny tekst źródłaReza, Mojgan. "Engineering and optimisation of mini-dystrophin constructs for Duchenne muscular dystrophy gene therapy". Thesis, University of Newcastle upon Tyne, 2015. http://hdl.handle.net/10443/2827.
Pełny tekst źródłaJohnson, Eric K. "A new model for the dystrophin associated protein complex in striated muscles". The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1354554580.
Pełny tekst źródłaSteen, Michelle Sabrina. "Analyses of alpha-dystrobrevin-null mice implicate Niemann-Pick C1 in muscular dystrophy /". Thesis, Connect to this title online; UW restricted, 2008. http://hdl.handle.net/1773/10537.
Pełny tekst źródłaKsiążki na temat "Dystrophin"
J, Winder Steve, red. Molecular mechanisms of muscular dystrophies. Georgetown, Tex: Landes Bioscience : Eurekah.com, 2006.
Znajdź pełny tekst źródła1958-, Brown Susan C., i Lucy Jack A. 1929-, red. Dystrophin: Gene, protein, and cell biology. Cambridge, U.K: Cambridge University Press, 1997.
Znajdź pełny tekst źródłaD'Souza, Vinita N. Dystrophin expression in the retina. Ottawa: National Library of Canada, 1995.
Znajdź pełny tekst źródłaBestard, Jennifer. Dystrophin gene regulation in muscle. Ottawa: National Library of Canada, 2000.
Znajdź pełny tekst źródłaThanh, Le Thiet. Exon-specific monoclonal antibodies against dystrophin. Salford: University of Salford, 1995.
Znajdź pełny tekst źródłaDally, Ghassan Y. Characterization of nommuscle isoforms of dystrophin. Ottawa: National Library of Canada, 1996.
Znajdź pełny tekst źródłaCisternas, Felipe A. The function of alternatively spliced isoforms of dystrophin. Ottawa: National Library of Canada, 2000.
Znajdź pełny tekst źródłaEmery, Alan E. H. Muscular dystrophy, the facts. Wyd. 2. Oxford: Oxford University Press, 2000.
Znajdź pełny tekst źródłaMuscular dystrophies. Amsterdam: Elsevier, 2011.
Znajdź pełny tekst źródłaH, Emery Alan E., red. The muscular dystrophies. Oxford, England: Oxford University Press, 2001.
Znajdź pełny tekst źródłaCzęści książek na temat "Dystrophin"
Lu-Nguyen, Ngoc, Alberto Malerba i Linda Popplewell. "Use of Small Animal Models for Duchenne and Parameters to Assess Efficiency upon Antisense Treatment". W Methods in Molecular Biology, 301–13. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2010-6_20.
Pełny tekst źródłaAgarwal, Aishwarya, Kunal Verma, Shivani Tyagi, Khushi Gupta, Satish Kumar Gupta, Shrestha Sharma i Shobhit Kumar. "Muscular Dystrophy: Mutations in the Dystrophin Gene". W Mechanism and Genetic Susceptibility of Neurological Disorders, 341–57. Singapore: Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-9404-5_15.
Pełny tekst źródłaDickson, George, i Matthew Dunckley. "Human dystrophin gene transfer: genetic correction of dystrophin deficiency". W Molecular and Cell Biology of Muscular Dystrophy, 283–302. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1528-5_11.
Pełny tekst źródłaMirza, Zeenat, i Sajjad Karim. "Decoding Dystrophin Gene Mutations: Unraveling the Mysteries of Muscular Dystrophy". W Mechanism and Genetic Susceptibility of Neurological Disorders, 75–90. Singapore: Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-9404-5_4.
Pełny tekst źródłaGoossens, Remko, i Annemieke Aartsma-Rus. "In Vitro Delivery of PMOs in Myoblasts by Electroporation". W Methods in Molecular Biology, 191–205. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2010-6_12.
Pełny tekst źródłaBarresi, Rita, i Susan C. Brown. "Dystrophin and Its Associated Glycoprotein Complex". W Muscle Disease, 95–101. Oxford, UK: John Wiley & Sons, Ltd, 2013. http://dx.doi.org/10.1002/9781118635469.ch8.
Pełny tekst źródłaShah, Md Nur Ahad, i Toshifumi Yokota. "Restoring Dystrophin Expression by Skipping Exons 6 and 8 in Neonatal Dystrophic Dogs". W Methods in Molecular Biology, 107–24. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2772-3_6.
Pełny tekst źródłaMurphy, Sandra, i Kay Ohlendieck. "Proteomic Profiling of the Dystrophin-Deficient Brain". W Methods in Molecular Biology, 91–105. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7374-3_7.
Pełny tekst źródłaLópez-Martínez, Andrea, Patricia Soblechero-Martín i Virginia Arechavala-Gomeza. "Evaluation of Exon Skipping and Dystrophin Restoration in In Vitro Models of Duchenne Muscular Dystrophy". W Methods in Molecular Biology, 217–33. New York, NY: Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2010-6_14.
Pełny tekst źródłaErvasti, James M., i Kevin P. Campbell. "Dystrophin-associated glycoproteins: their possible roles in the pathogenesis of Duchenne muscular dystrophy". W Molecular and Cell Biology of Muscular Dystrophy, 139–66. Dordrecht: Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1528-5_6.
Pełny tekst źródłaStreszczenia konferencji na temat "Dystrophin"
Cassino, Theresa R., Masaho Okada, Lauren Drowley, Johnny Huard i Philip R. LeDuc. "Mechanical Stimulation Improves Muscle-Derived Stem Cell Transplantation for Cardiac Repair". W ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192941.
Pełny tekst źródłaCassino, Theresa R., Masaho Okada, Lauren M. Drowley, Joseph Feduska, Johnny Huard i Philip R. LeDuc. "Using Mechanical Environment to Enhance Stem Cell Transplantation in Muscle Regeneration". W ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176545.
Pełny tekst źródłaOliveira, Marco Antônio Rodrigues Gomes de, i Isaura Maria Mesquita Prado. "Evidence and affects in Duchenne muscular dystrophy in children and Golden Retriever dogs". W XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.302.
Pełny tekst źródłade Feraudy, Yvan, Rabah Yaou, Karim Wahbi, France Leturcq i Helge Amthor. "Residual Very Low Dystrophin Levels Mitigate Dystrophinopathy towards Becker’s Muscular Dystrophy". W Abstracts of the 47th Annual Meeting of the SENP (Société Européenne De Neurologie Pédiatrique). Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1685441.
Pełny tekst źródłaHilton, S., M. Christen, T. Bilzer, K. Matiasek, V. Jagannathan, T. Leeb i U. Giger. "Dystrophin (DMD) missense variant in cats with Becker type muscular dystrophy". W 31. Jahrestagung der FG „Innere Medizin und klinische Labordiagnostik“ der DVG (InnLab) – Teil 1: Vorträge. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1760811.
Pełny tekst źródłaLima, Karlla Danielle Ferreira, Pedro Henrique Marte Arruda Sampaio, Marco Antonio Veloso Albuquerque i Edmar Zanoteli. "Evaluation of lung function and respiratory muscles in Duchenne muscular dystrophy". W XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.695.
Pełny tekst źródłaMarin, Marija. "Immunogold localization of dystrophin in the erythrocytes of patients with Duchenne-Becker muscular dystrophy". W European Microscopy Congress 2020. Royal Microscopical Society, 2021. http://dx.doi.org/10.22443/rms.emc2020.373.
Pełny tekst źródłaKrause, C., S. Kranig, J. Pöschl i H. Hudalla. "Frühe T-Zell Immundysregulation im Dystrophin defizienten Tiermodell". W 30. Kongress der Deutschen Gesellschaft für Perinatale Medizin – „Wandel als Herausforderung“. Georg Thieme Verlag, 2021. http://dx.doi.org/10.1055/s-0041-1739718.
Pełny tekst źródłaArotaritei, Dragos, Catalin Ionite i Calin Corciova. "EDUCATIONAL TOOL FOR MATHEMATICAL MODEL OF MUSCULAR DYSTROPHY". W eLSE 2019. Carol I National Defence University Publishing House, 2019. http://dx.doi.org/10.12753/2066-026x-19-174.
Pełny tekst źródłaSchaefer, Inga-Marie, Armelle Dufresne, Nacef Bahri, Marije A. J. de Rooij, Stacy M. Yanofsky, Yuexiang Wang, Chandrajit P. Raut i in. "Abstract B16: Dystrophin is a tumor suppressor in peripheral nerve sheath tumors". W Abstracts: Advances in Sarcomas: From Basic Science to Clinical Translation; May 16-19, 2017; Philadelphia, PA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1557-3265.sarcomas17-b16.
Pełny tekst źródłaRaporty organizacyjne na temat "Dystrophin"
Cox, Gregory A. Translational Research for Muscular Dystrophy. Fort Belvoir, VA: Defense Technical Information Center, maj 2014. http://dx.doi.org/10.21236/ada609750.
Pełny tekst źródłaCox, Gregory A. Translational Research for Muscular Dystrophy. Fort Belvoir, VA: Defense Technical Information Center, maj 2012. http://dx.doi.org/10.21236/ada564543.
Pełny tekst źródłaHuard, Johnny, Eric Hoffman, John Day, Kevin Campbell, Xiao Xiao i Paula Clemens. New Advanced Technology for Muscular Dystrophy. Fort Belvoir, VA: Defense Technical Information Center, listopad 2009. http://dx.doi.org/10.21236/ada536121.
Pełny tekst źródłaMahoney, My G., Ulrich Rodeck i Jouni Uitto. Molecular Characterization of Squamous Cell Carcinomas From Recessive Dystrophic Epidermolysis Bullosa. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2006. http://dx.doi.org/10.21236/ada463709.
Pełny tekst źródłaCnaan, Avital. CINRG: Infrastructure for Clinical Trials in Duchenne Dystrophy. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2012. http://dx.doi.org/10.21236/ada567633.
Pełny tekst źródłaCnaan, Avital. CINRG: Infrastructure for Clinical Trials in Duchenne Dystrophy. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 2013. http://dx.doi.org/10.21236/ada599521.
Pełny tekst źródłaMahoney, My G., Ulrich Rodeck i Jouni Uitto. Molecular Characterization of Squamous Cell Carcinomas Derived from Recessive Dystrophic Epidermolysis Bullosa. Fort Belvoir, VA: Defense Technical Information Center, czerwiec 2005. http://dx.doi.org/10.21236/ada446877.
Pełny tekst źródłaMahoney, My G., Ulrich Rodeck i Jouni Uitto. Molecular Characterization of Squamous Cell Carcinomas Derived From Recessive Dystrophic Epidermolysis Bullosa. Fort Belvoir, VA: Defense Technical Information Center, czerwiec 2003. http://dx.doi.org/10.21236/ada419358.
Pełny tekst źródłaMartin, Paul T. Translational Studies of GALGT2 Gene Therapy for Duchenne Muscular Dystrophy. Fort Belvoir, VA: Defense Technical Information Center, październik 2014. http://dx.doi.org/10.21236/ada613577.
Pełny tekst źródłaMartin, Paul T. Translational Studies of GALGT2 Gene Therapy for Duchenne Muscular Dystrophy. Fort Belvoir, VA: Defense Technical Information Center, październik 2013. http://dx.doi.org/10.21236/ada598203.
Pełny tekst źródła