Gotowe bibliografie tematyczne / Dorsolateral prefrontal cortex

Gotowa bibliografia na temat „Dorsolateral prefrontal cortex”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 11 marca 2023

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Artykuły w czasopismach na temat "Dorsolateral prefrontal cortex"

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Preuss, Todd M. "Do Rats Have Prefrontal Cortex? The Rose-Woolsey-Akert Program Reconsidered". Journal of Cognitive Neuroscience 7, nr 1 (styczeń 1995): 1–24. http://dx.doi.org/10.1162/jocn.1995.7.1.1.

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Primates are unique among mammals in possessing a region of dorsolateral prefrontal cortex with a well-developed internal granular layer. This region is commonly implicated in higher cognitive functions. Despite the histological distinctiveness of primate dorsolateral prefrontal cortex, the work of Rose, Woolsey, and Akert produced a broad consensus among neuroscientists that homologues of primate granular frontal cortex exist in nonprimates and can be recognized by their dense innervation from the mediodorsal thalamic nucleus (MD). Additional characteristics have come to be identified with dorsolateral prefrontal cortex, including rich dopaminergic innervation and involvement in spatial delayed-reaction tasks. However, recent studies reveal that these characteristics are not distinctive of the dorsolateral prefrontal region in primates: MD and dopaminergic projections are widespread in the frontal lobe, and medial and orbital frontal areas may play a role in delay tasks. A reevaluation of rat frontal cortex suggests that the medial frontal cortex, usually considered to be homologous to the dorsolateral prefrontal cortex of primates, actually consists of cortex homologous to primate premotor and anterior cin-date cortex. The lateral MD-projection cortex of rats resembles portions of primate orbital cortex. If prefrontal cortex is construed broadly enough to include orbital and cingulate cortex, rats can be said to have prefrontal cortex. However, they evidently lack homologues of the dorsolateral prefrontal areas of primates. This assessment suggests that rats probably do not provide useful models of human dorsolateral frontal lobe function and dysfunction, although they might prove valuable for understanding other regions of frontal cortex.
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Spence, Sean A., Steven R. Hirsch, David J. Brooks i Paul M. Grasby. "Prefrontal cortex activity in people with schizophrenia and control subjects". British Journal of Psychiatry 172, nr 4 (kwiecień 1998): 316–23. http://dx.doi.org/10.1192/bjp.172.4.316.

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BackgroundHypo-activation of the left dorsolateral prefrontal cortex is inconsistently found in neuroimaging studies of schizophrenia. As the left dorsolateral prefrontal cortex is involved in the generation of action, disordered function in this region may be implicated in schizophrenic symptomatology.MethodWe used H215O positron emission tomography to study dorsolateral prefrontal cortical function in men with schizophrenia (n=13) and male control subjects (n=6) performing joystick movements on two occasions, 4–6 weeks apart. The patients were initially in relapse. To clarify dorsolateral prefrontal cortical function we also scanned another group of control subjects (n=5) performing mouth movements.ResultsThe control subjects performing hand or mouth movements activated the left dorsolateral prefrontal cortex to a maximum when the movements were self-selected. The men with relapsed schizophrenia exhibited left dorsolateral prefrontal cortical hypo-activation, which remitted with symptomatic improvement.ConclusionsHypofrontality in these patients is a dynamic phenomenon across time, possibly related to current symptomatology. The most appropriate question about the presence of hypofrontality in schizophrenia may be when, rather than whether, it will occur.
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Wiyor, Hanniebey D., i Celestine A. Ntuen. "Empirical Evaluation of Visual Fatigue from Display Alignment Errors Using Cerebral Hemodynamic Responses". Journal of Medical Engineering 2013 (24.12.2013): 1–9. http://dx.doi.org/10.1155/2013/521579.

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The purpose of this study was to investigate the effect of stereoscopic display alignment errors on visual fatigue and prefrontal cortical tissue hemodynamic responses. We collected hemodynamic data and perceptual ratings of visual fatigue while participants performed visual display tasks on 8 ft × 6 ft NEC LT silver screen with NEC LT 245 DLP projectors. There was statistical significant difference between subjective measures of visual fatigue before air traffic control task (BATC) and after air traffic control task (ATC 3), (P<0.05). Statistical significance was observed between left dorsolateral prefrontal cortex oxygenated hemoglobin (l DLPFC-HbO2), left dorsolateral prefrontal cortex deoxygenated hemoglobin (l DLPFC-Hbb), and right dorsolateral prefrontal cortex deoxygenated hemoglobin (r DLPFC-Hbb) on stereoscopic alignment errors (P<0.05). Thus, cortical tissue oxygenation requirement in the left hemisphere indicates that the effect of visual fatigue is more pronounced in the left dorsolateral prefrontal cortex.
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Khundakar, Ahmad, Christopher Morris, Arthur Oakley, William McMeekin i Alan J. Thomas. "Morphometric analysis of neuronal and glial cell pathology in the dorsolateral prefrontal cortex in late-life depression". British Journal of Psychiatry 195, nr 2 (sierpień 2009): 163–69. http://dx.doi.org/10.1192/bjp.bp.108.052688.

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BackgroundLate-life depression has been associated with cerebrovascular disease and especially with ischaemic white matter hyperintensities on magnetic resonance imaging. Neuroimaging and morphometric studies have identified abnormalities in the dorsolateral prefrontal cortex.AimsTo examine glial and neuronal density and neuronal volume in the dorsolateral prefrontal cortex in late-life major depression.MethodWe used the disector and nucleator methods to estimate neuronal density and volume and glial density of cells in the dorsolateral prefrontal cortex in a post-mortem study of 17 individuals with late-life major depression and 10 age-matched controls.ResultsWe found a reduction in the volume of pyramidal neurones in the whole cortex, which was also present in layer 3 and more markedly in layer 5. There were no comparable changes in non-pyramidal neurones and no glial differences.ConclusionsOverall, we found a decrease in pyramidal neuronal size in the dorsolateral prefrontal cortex in late-life depression.
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Macoveanu, Julian, Kirsa M. Demant, Maj Vinberg, Hartwig R. Siebner, Lars V. Kessing i Kamilla W. Miskowiak. "Towards a biomarker model for cognitive improvement: No change in memory-related prefrontal engagement following a negative cognitive remediation trial in bipolar disorder". Journal of Psychopharmacology 32, nr 10 (4.07.2018): 1075–85. http://dx.doi.org/10.1177/0269881118783334.

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Background: Cognitive deficits are prevalent in bipolar disorder during remission but effective cognition treatments are lacking due to insufficient insight into the neurobiological targets of cognitive improvement. Emerging data suggest that dorsal prefrontal cortex target engagement is a key neurocircuitry biomarker of pro-cognitive treatment effects. Aims: In this randomized controlled functional magnetic resonance imaging study, we test this hypothesis by investigating the effects of an ineffective cognitive remediation intervention on dorsal prefrontal response during strategic memory encoding and working memory engagement. Methods: Bipolar disorder patients in partial remission with subjective cognitive difficulties were randomized to receive 12-week group-based cognitive remediation ( n = 13) or to continue their standard treatment ( n = 14). The patients performed a strategic episodic picture encoding task and a spatial n-back working memory task under functional magnetic resonance imaging at baseline and following cognitive remediation or standard treatment. Results: The right dorsolateral prefrontal cortex was commonly activated by both strategic memory tasks across all patients. The task-related prefrontal engagement was not altered by cognitive remediation relative to standard treatment. The dorsolateral prefrontal cortex response was not significantly associated with recall accuracy or working memory performance. Conclusions: As hypothesized, no task-related change in prefrontal activity was observed in a negative cognitive remediation trial in remitted bipolar disorder patients. By complementing previous findings linking cognitive improvement with increased dorsolateral prefrontal cortex engagement, our negative findings provide additional validity evidence to the dorsal prefrontal target engagement biomarker model of cognitive improvement by strengthening the proposed causality between modulation of dorsolateral prefrontal cortex engagement and pro-cognitive effects.
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Zhu, Xueling, Shaohui Liu, Weihua Liao, Lingyu Kong, Canhua Jiang i Fulai Yuan. "Executive function deficit in betel-quid-dependent chewers: Mediating role of prefrontal cortical thickness". Journal of Psychopharmacology 32, nr 12 (31.10.2018): 1362–68. http://dx.doi.org/10.1177/0269881118806299.

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Background: Betel quid is the fourth most popular psychoactive agent worldwide. Neuroimaging studies have suggested betel-quid dependence is accompanied by abnormality in brain structure and function. However, the neural correlates of executive function deficit and prefrontal cortical thickness associated with betel-quid chewing still remain unclear. Objective: The present study aimed to examine the relationship between executive function deficit and prefrontal cortical thickness in chronic betel-quid chewers. Methods: Twenty-three betel-quid-dependent chewers and 26 healthy controls were recruited to participate in this study. Executive function was tested using three tasks. Cortical thickness analysis was analyzed with the FreeSurfer software package. Results: Behavioral results suggested a profound deficit of executive function in betel-quid-dependent chewers. Cortical thickness analysis revealed thinner cortex in the bilateral dorsolateral prefrontal cortex in betel-quid-dependent chewers. Further analysis suggested that cortical thickness of the bilateral dorsolateral prefrontal cortex mediated the correlation of betel-quid chewing and executive function. Conclusions: These results suggest the important role of executive function and cortical thickness of the dorsolateral prefrontal cortex with betel-quid chewing. Our findings provide evidence that executive function deficit may be mediated by the cortical thickness of the dorsolateral prefrontal cortex. These results could potentially help us develop novel ways to diagnose and prevent betel-quid dependence.
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Snyder, Janice J., i Anjan Chatterjee. "The Frontal Cortex and Exogenous Attentional Orienting". Journal of Cognitive Neuroscience 18, nr 11 (listopad 2006): 1913–23. http://dx.doi.org/10.1162/jocn.2006.18.11.1913.

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Normal functioning of the attentional orienting system is critical for effective behavior and is predicated on a balanced interaction between goal-directed (endogenous) processes and stimulus-driven (exogenous) processes. Although both systems have been subject to much investigation, little is known about the neural underpinnings of exogenous orienting. In the present study, we examined the early facilitatory effects and later inhibition of return effects of exogenous cues in patients with frontal and parietal lesions. Three novel findings emerged from this study. First, unilateral frontoparietal damage appears not to affect the early facilitation effects of exogenous cues. Second, dorsolateral prefrontal damage, especially lesions involving the inferior frontal gyrus, produces an exogenous disengage deficit (i.e., the sluggish withdrawal of attention from the ipsilesional to the contralesional field). Third, a subset of patients with dorsolateral prefrontal damage, with lesions involving the middle frontal gyrus, have a reorienting deficit that extends in duration well beyond established boundaries of the normal reflexive orienting system. These results suggest that the dorsolateral prefrontal cortex plays an important role in exogenous orienting and that component processes of this system may be differentially impaired by damage to different parts of the dorsolateral prefrontal cortex.
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Shin, Sangho, i Euitae Kim. "T137. THE RELATIONSHIP BETWEEN CORTICOSTRIATAL CONNECTIVITY AND STRIATAL DOPAMINE FUNCTION IN SCHIZOPHRENIA: AN 18F-DOPA PET AND DIFFUSION TENSOR IMAGING STUDY". Schizophrenia Bulletin 46, Supplement_1 (kwiecień 2020): S282—S283. http://dx.doi.org/10.1093/schbul/sbaa029.697.

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Abstract Background Striatal dopamine dysfunction caused by cortical abnormalities is a leading hypothesis of schizophrenia pathophysiology, which underlies in majority of treatment-responsive patients. Although supported by findings that prefrontal cortical lesions lead to striatal dopamine dysregulation and that recently, prefrontal structural volume is negatively correlated with striatal dopamine synthesis, the relationship between corticostriatal connectivity and striatal dopamine synthesis has not been tested in patients with schizophrenia. We therefore investigated the relationship between corticostriatal connectivity and striatal dopamine synthesis capacity in treatment-responsive patients with schizophrenia, and compared them to treatment-resistant patients and healthy control subjects. Methods Twenty-four patients with schizophrenia and twelve matched healthy control subjects underwent 18F-DOPA PET scans to measure dopamine synthesis capacity (indexed as the influx rate constant Kicer), structural and diffusion 3T MRI. Connectivity(indexed as Fractional anisotropy, FA) were assessed in 3 major corticostriatal tracts (dorsolateral prefrontal cortex-associative striatum, ventromedial prefrontal cortex-limbic striatum, and pre/primary motor cortex-sensorimotor striatum). Furthermore, these measures were tested whether they were correlated with a measure of Wisconsin Card Sorting Test (WCST). Results Treatment responsive patients showed a negative correlation between connectivity of dorsolateral prefrontal cortex-associative striatum and striatal dopamine synthesis capacity of associative striatum, but this was not evident in treatment-resistant patients. Furthermore, WCST negatively correlated with Kicer in associative striatum and positively correlated with FA in dorsolateral prefrontal cortex-associative striatum in whole subjects and treatment responsive patients but not in treatment-resistant patients. Discussion These findings demonstrate that different mechanisms underlie the pathophysiology of treatment-responsive and treatment-resistant schizophrenia and especially, connectivity of dorsolateral prefrontal cortex-associative striatum is a core part for the different pathophysiology.
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Guo, Jiayue, Jiani Luo, Yi An i Tiansheng Xia. "tDCS Anodal Stimulation of the Right Dorsolateral Prefrontal Cortex Improves Creative Performance in Real-World Problem Solving". Brain Sciences 13, nr 3 (6.03.2023): 449. http://dx.doi.org/10.3390/brainsci13030449.

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Brain regions associated with creativity is a focal point in research related to the field of cognitive neuroscience. Previous studies have paid more attention to the role of activation of the left dorsolateral prefrontal cortex in creativity tasks, which are mostly abstract conceptual tasks, and less attention to real-world creativity tasks. The right dorsolateral prefrontal cortex is involved in functions such as visuospatial processing, which may have a positive impact on innovative solutions to real-world problems. In this study, tDCS technology was used to explore the effect of anodal stimulation of the right dorsolateral prefrontal cortex on design creativity performance in a real-word problem-solving task related to product design. The experimental task comprised three stages, of which the first two were idea generation stages based on divergent thinking using text and graphics, respectively, whereas the third was the creative evaluation stage based on convergent thinking. Thirty-six design students were recruited to partake in the experiment. They were randomly assigned into anodal stimulation and sham stimulation groups. The results showed that anodal stimulation of the right dorsolateral prefrontal cortex produced a significant positive effect during the creative evaluation stage, promoting the usefulness of ideas (p = 0.009); thus, improving product creativity scores. However, there was no significant impact on the idea generation stage (p > 0.05), which is dominated by divergent thinking. The results suggest that activating the right dorsolateral prefrontal cortex with tDCS can improve people’s performance in creative activities by promoting convergent thinking rather than divergent thinking. It also provides further evidence that the right hemisphere of the brain has an advantage in solving complex problems that require the participation of visuospatial information.
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Bodner, Mark, James Kroger i Joaquín M. Fuster. "Auditory memory cells in dorsolateral prefrontal cortex". NeuroReport 7, nr 12 (sierpień 1996): 1905–8. http://dx.doi.org/10.1097/00001756-199608120-00006.

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Rozprawy doktorskie na temat "Dorsolateral prefrontal cortex"

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Greenberg, Paul Arthur. "Functional Stability and Learning in the Dorsolateral Prefrontal Cortex". Diss., Tucson, Arizona : University of Arizona, 2005. http://etd.library.arizona.edu/etd/GetFileServlet?file=file:///data1/pdf/etd/azu%5Fetd%5F1030%5F1%5Fm.pdf&type=application/pdf.

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Lanoue, Amelie Cecile. "Neuropathology in the dorsolateral prefrontal cortex in Parkinson's disease". Thesis, Boston University, 2013. https://hdl.handle.net/2144/11112.

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Thesis (Ph.D.)--Boston University
Degeneration of dopaminergic neurons in the substantia nigra pars compacta is the hallmark neuropathological feature of Parkinson's disease (PD). Multiple lines of evidence from anatomical and imaging studies indicate that cell loss or cell dysfunction also occur in other brain regions. The dorsolateral prefrontal cortex (DLPFC) is a region of interest because it could be implicated in both cognitive and motor symptoms of PD. However, studies in this brain region are limited and the extent of pathology is unclear. Work in this thesis was aimed at identifying possible neuropathology in post-mortem PD tissue from Brodmann area 9 (BA9), a region of the DLPFC. In the first study, using design-based stereology and radioisotopic in situ hybridization histochemistry (ISHH), we found that expression of two mitochondrial genes, NDUFS1 and COX1, was not altered and that no global loss of neurons occurs in BA9 in PD. In a second study, using ISHH and gene expression microarray analysis (One-Color Agilent 60-mer Whole Human Genome Microarray), we found decreased gene expression of the GABA-synthesizing enzyme glutamic acid decarboxylase (GAD67) in BA9 in PD, an effect that was not paralleled by a decrease in the numbers of GAD67 mRNA-expressing neurons. In a third study, using ISHH, we found that gene expression of the calcium-binding protein parvalbumin, which is selectively expressed in a subset of cortical GABAergic interneurons, is decreased in BA9 in PD. However, we found no loss of immunolabeled parvalbumin-positive neurons in BA9 in PD. In summary, the results indicate that expression of two key markers of GABAergic activity, GAD67 and parvalbumin, is depressed in BA9 in PD and that these effects are not due to a loss of neurons. This suggests that GABAergic neurotransmission is deficient in the DLPFC in PD and we propose that treatments aimed at restoring GABAergic inhibition in BA9 would have therapeutic efficacy in the symptomatic treatment of PD.
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Fernandes, Ninette M. "The Detection of Prefrontal Cortex Development into Early Adulthood". Marietta College / OhioLINK, 2006. http://rave.ohiolink.edu/etdc/view?acc_num=marietta1164924291.

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Keifer, Ekaterina. "Performance of patients with ventromedial prefrontal, dorsolateral prefrontal, and non-frontal lesions on the Delis-Kaplan Executive Function System". Diss., University of Iowa, 2010. https://ir.uiowa.edu/etd/830.

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Executive functioning is a multidimensional concept encompassing higher-order adaptive abilities, such as judgment, decision-making, self-monitoring, planning, and emotional regulation. Disruption in executive functioning often results in devastating impairments in vitally-important areas of life, such as one's ability to hold employment and maintain social relationships. Executive functions have been associated primarily with the prefrontal cortex. However, the nature and degree of the association between frontal lobe damage and performance on executive functioning tests remains controversial. Research suggests that the association may vary based on the specific location of damage within the prefrontal cortex, as well as the used measure of executive functioning. Few investigations have systematically addressed these variables. The current study employed the lesion method to investigate the relationship between performance on a battery of executive functioning tests and damage to specific regions of the prefrontal cortex. Three groups of participants with lesions in one of the locations of interest [ventromedial prefrontal (VMPC, n = 14), dorsolateral prefrontal (DLPC, n = 14), and non-frontal (n = 18)] were administered the Delis-Kaplan Executive Function System (D-KEFS, 2001), a comprehensive battery of executive functioning tests. Results revealed no statistically-significant differences between group performances on the D-KEFS primary measures. However, a qualitative analysis of the results revealed several meaningful group differences. It appears that some relationship exists between frontal lobe damage, particularly in the DLPC, and decreased performance on several executive functioning tests but further research overcoming the methodological limitations of most existing literature on this topic is needed to clearly resolve this issue.
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Nord, Camilla Laxmi. "The role of dorsolateral prefrontal cortex dysfunction in depression and its treatment with non-invasive brain stimulation". Thesis, University College London (University of London), 2017. http://discovery.ucl.ac.uk/10038161/.

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Major depression is a common and debilitating condition. However, initial treatment is ineffective for almost half of all patients. This thesis aims to clarify the mechanisms of a novel putative treatment for depression, transcranial direct current stimulation (tDCS), which targets the dorsolateral prefrontal cortex (DLPFC). The first experimental chapter tests whether DLPFC tDCS alters emotional face perception, akin to the acute effects of antidepressant drugs. Our analyses revealed that tDCS does not exert an antidepressant-like effect on emotion perception, but may affect non-emotional cognition. The second experimental chapter examines neural activation in depressed patients, unaffected first-degree relatives of depressed patients, and healthy controls during the n-back working memory task and a facial emotion processing task. During the n-back, depressed patients showed pronounced DLPFC hypoactivation, while at-risk participants were indistinguishable from healthy controls, consistent with the hypothesis that DLPFC dysfunction might be a useful target for depression treatment. In the final two chapters, I report results from a double-blind randomized controlled trial that for the first time tested DLPFC tDCS as an augmentation strategy to psychotherapy in depression, measuring its neural, cognitive, and clinical effects. On the primary outcome measure (observer-rated depressive symptoms) active tDCS did not show a significant improvement over sham stimulation, although the difference was in the hypothesised direction. However, baseline DLPFC activation during the n-back strongly predicted clinical outcome, with this association specific to the active tDCS condition. Thus, baseline DLPFC activation might serve as a putative ‘biomarker’ for clinical response to tDCS. In the general discussion, these experimental findings are discussed in the context of contemporary theories of depression. This thesis adds new insights into the possible mechanisms of tDCS as a treatment for depression. It also demonstrates the added value of neuroimaging to psychiatry clinical trials, highlighting a potential role for predicting treatment outcome.
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Plakke, Anderson Bethany Joy. "Auditory working memory: contributions of lateral prefrontal cortex and acetylcholine in non-human primates". Diss., University of Iowa, 2010. https://ir.uiowa.edu/etd/1060.

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Traditionally, working memory and its neural underpinnings have been studied in the visual domain. A rich and diverse amount of research has investigated the lateral prefrontal cortex (lPFC) as a primary area for visual working memory, while another line of research has found the neurotransmitter acetylcholine (ACh) to be involved. This dissertation used auditory cues and found similar patterns of activity for processing auditory working memory information within a task compared to visual working memory processes. The first two experimental chapters demonstrated that the cholinergic system is involved in auditory working memory in a comparable fashion to its role in visual working memory. In chapter 2, blocking ACh impaired performance on an auditory working memory task in a dose dependent manner. Chapter 3 investigated the specificity of the effect of blocking ACh by administering an ACh agonist (physostigmine) at the same time as an ACh antagonist (scopolamine). When both drugs were administered together performance on the delayed matching-to-sample task (DMTS) task improved compared to performance on scopolamine alone. These results support the hypothesis that ACh is involved in auditory working memory. Chapter 4 investigated the neural correlates of auditory working memory in area 46 and found that this region of the lPFC contains neurons that are responsive to auditory working memory components in a very similar way to how it this region encodes information during visual working memory tasks. Neurons in the lPFC are responsive to visual or auditory cues, the delay portion of tasks, the wait time (i.e. decision making period), response, and reward times. This type of coding provides support for the theories that position the lPFC as a key player in recognition and working memory regardless of modality.
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Miléa, Dan. "Le lobe frontal dans le contrôle de la motricité oculaire". Paris 6, 2003. http://www.theses.fr/2003PA066225.

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Brooks, Samantha J., Jonathan Cedernaes i Helgi B. Schiöth. "Increased prefrontal and parahippocampal activation with reduced dorsolateral prefrontal and insular cortex activation to food images in obesity : a meta-analysis of fMRI studies". Uppsala universitet, Funktionell farmakologi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-199757.

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BACKGROUND AND OBJECTIVES: Obesity is emerging as the most significant health concern of the twenty-first century. A wealth of neuroimaging data suggest that weight gain might be related to aberrant brain function, particularly in prefrontal cortical regions modulating mesolimbic addictive responses to food. Nevertheless, food addiction is currently a model hotly debated. Here, we conduct a meta-analysis of neuroimaging data, examining the most common functional differences between normal-weight and obese participants in response to food stimuli. DATA SOURCE: We conducted a search using several journal databases and adhered to the 'Preferred Reporting Items for Systematic Reviews and Meta-analyses' (PRISMA) method. To this aim, 10 studies were found with a total of 126 obese participants, 129 healthy controls, equaling 184 foci (146 increased, 38 decreased activation) using the Activation Likelihood Estimation (ALE) technique. Out of the 10 studies, 7 investigated neural responses to food versus non-food images. RESULTS: In response to food images, obese in comparison to healthy weight subjects had increased activation in the left dorsomedial prefrontal cortex, right parahippocampal gyrus, right precentral gyrus and right anterior cingulate cortex, and reduced activation in the left dorsolateral prefrontal cortex and left insular cortex. CONCLUSIONS: Prefrontal cortex areas linked to cognitive evaluation processes, such as evaluation of rewarding stimuli, as well as explicit memory regions, appear most consistently activated in response to images of food in those who are obese. Conversely, a reduced activation in brain regions associated with cognitive control and interoceptive awareness of sensations in the body might indicate a weakened control system, combined with hypo-sensitivity to satiety and discomfort signals after eating in those who are prone to overeat.
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Hawco, Colin Shaun. "The role of the dorsolateral prefrontal cortex in self-initiating elaborative episodic encoding: evidence from fMRI and TMS". Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=114200.

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Several clinical populations (e.g. Schizophrenia, Alzheimer's disease, frontal lobe damage, and healthy aging with memory decline) display memory deficits which may be related to a failure to engage efficient memory encoding strategies. However, these groups often show improved memory performance when cued towards the use of efficient encoding strategies, suggesting the deficits are related to self-initiating elaborative encoding processes. At present, little is know about the neural correlates of self-initiating elaborative encoding strategies in episodic memory. The purpose of this thesis was to better understand the process of initiating elaborative encoding strategies. We hypothesized that the left dorsolateral prefrontal cortex (DLPFC) was involved in self-initiating elaborative encoding strategies. Experiment 1 was an fMRI study in which we presented conditions in which participants were either cued to use an efficient encoding strategy (semantic analysis) or were not cued to do so (a self-initiated condition), while presenting stimuli with variable semantic relatedness. We observed activity in the left DLPFC and bilateral supramarginal gyrus in response to semantic relatedness in the non-semantic (self-initiated) encoding condition. In experiment 2, we attempted to confirm the role of the left DLPFC in self-initiating elaborative encoding using transcranial magnetic stimulation (TMS), a method in which we can transiently disrupt neural activity in a limited cortical area. We performed stimulation of the left DLPFC and a control site (the vertex) during a memory encoding task. We observed a significant correlation in a subsequent cued recall task (a measure of encoding success) between the effects of TMS during encoding and participant's use of memory strategies during encoding only in the condition in which self-initiated elaborative encoding was beneficial to memory performance. This suggests a causative role for the DLPFC in self-initiating elaborative encoding. Experiment 3 was a concurrent TMS-fMRI study. Participants performed an encoding task (similar to the self-initiated condition in experiment 1) while we measured brain activity using fMRI. TMS stimulation was presented for 300ms on ¾ of trials. The onset of stimulation was varied, starting at 200ms, 600ms, or 1000ms after stimulus onset. We observed time-specific changes in neural activity in response to TMS stimulation, suggesting that concurrent TMS-fMRI can be used to measure time-varying interactions between the DLPFC and distal brain regions These three experiment provide evidence o f the role of the left DLPFC in self-initiating elaborative encoding strategies, and the utility of TMS and fMRI (separately or combined) as research techniques to address these techniques. These studies also demonstrate the utility of our selected paradigms to directly address the issue of self-initiating elaborative encoding (rather than correlating activity to specific encoding strategies).
Plusieurs populations cliniques (ex. schizophrénie, maladie d'Alzheimer, lésions du lobe frontal, vieillissement normal avec déclin de mémoire) démontrent des déficits de mémoire qui peuvent être reliés à une incapacité d'initier des stratégies efficaces d'encodage de mémoire. Cependant, ces groupes démontrent souvent une amélioration de leur performance lorsqu'on les aide à choisir une stratégie d'encodage efficace, suggérant que les déficits seraient reliés à l'utilisation spontanée de stratégies d'encodage élaborées. A ce jour, nous savons très peu de choses à propos des corrélats neuronaux de l'utilisation spontanée de stratégies d'encodage élaborées. Le but de cette thèse est de mieux comprendre les processus de l'initiation de stratégies d'encodage élaborées. Nous émettons l'hypothèse que le cortex préfrontal dorsolatéral (DLPFC) est impliqué dans l'utilisation spontanée de stratégies d'encodage élaborées. L'expérience 1 consiste en une étude d'IRMf dans laquelle nous avons présenté des conditions dans lesquelles les participants étaient guidés à utiliser une stratégie d'encodage efficace (analyse sémantique) ou non guidés d'utiliser cette stratégie (condition auto-initiée), en présentant des stimuli de relations sémantiques variées. Nous avons observé une activité dans le DLPFC gauche et le gyrus supramarginal bilatéral en réponse à la relation sémantique dans la condition d'encodage non-sémantique (auto-initiée). Dans l'expérience 2, nous avons tenté de confirmer le rôle du DLPFC gauche dans l'utilisation spontanée de stratégies d'encodage élaborées en utilisant la stimulation magnétique transcrânienne (SMT), une méthode avec laquelle nous pouvons perturber l'activité neuronale de façon transitoire dans une aire corticale limitée. Nous avons performé une stimulation du DLPFC gauche et d'un site contrôle (le vertex) durant une tâche d'encodage de mémoire. Nous avons observé une corrélation significative dans la tâche de reconnaissance subséquente (une mesure de la réussite de l'encodage) entre les effets de la SMT durant l'encodage et l'utilisation de stratégies de mémoire du participant pendant l'encodage seulement dans la condition où l'utilisation spontanée de stratégies d'encodage élaborées était bénéfique pour la performance de mémoire. Ceci suggère un rôle causal du DLPFC dans l'utilisation spontanée de stratégies d'encodage élaborées. L'expérience 3 était une étude simultanée de SMT-IRMf. Les participants devaient faire une tâche d'encodage (similaire à la condition auto-initiée de l'expérience 1) pendant que l'on mesurait l'activité du cerveau avec l'IRMf. Une SMT était faite pendant 300ms sur les trois-quarts des essais. Le début de la stimulation était varié, commençant à 200ms, 600ms ou 1000ms après le début du stimulus. Nous avons observé des changements spécifiques au temps dans l'activité neuronale en réponse à la stimulation SMT, indiquant que l'utilisation simultanée de SMT-IRMf peut être utilisée pour mesurer l'interaction en fonction du temps entre le DLPFC et les régions distales du cerveau. Ces trois expériences apportent des évidences du rôle du DLPFC gauche dans l'utilisation spontanée de stratégies d'encodage élaborées et l'utilité de la SMT et de l'IRMf (séparément ou combinées) comme techniques de recherche pour étudier ces processus. Ces études démontrent aussi l'utilité de nos paradigmes pour étudier directement l'utilisation spontanée de stratégies d'encodage élaborées (au lieu de corréler l'activité à des stratégies d'encodage spécifique).
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Bunch, Katie, i n/a. "A Relational Complexity Approach to the Development of Hot/Cool Executive Functions". Griffith University. School of Psychology, 2006. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20070713.121052.

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Previous research indicates that many important changes in executive functions, or higher cognitive capacities, occur between the ages of three and five years. Additionally, a distinction can be made between the cognitive functions associated with two different cortical regions. The functions of the dorsolateral prefrontal cortex (DL-PFC) are assessed using 'cool' tasks that are abstract and decontextualised. In contrast, the functions of the orbitofrontal cortex (OFC) are assessed using 'hot' tasks that require flexible appraisal of the affective significance of stimuli (Zelazo & Müller, 2002). Different clinical populations have been hypothesized to differ in terms of their impairment on tasks associated with each area of functioning. Current research conclusions regarding the primacy of hot versus cool executive function impairments are limited, however, as they have not taken complexity into account. That is, tasks currently used in investigations of hot and cool executive functions might differ in terms of the complexity of the cognitive processes that the tasks require. Therefore, comparisons across tasks may be misleading because these tasks vary in terms of the demands they place on participants as well as their hot versus cool status. While complexity theories have been applied to a number of cool tasks, only one hot task, those measuring theory-of-mind abilities, have been analysed in terms of complexity. One aim of the current research was to modify several tasks presumed to measure OFC performance to include a complexity manipulation. Tasks from three hot domains (conditional discrimination, the Children's Gambling Task, and future-oriented decision-making) were analysed in terms of their relational complexity, that is, the number of related entities or arguments inherent in a task or concept (Halford, 1993). Based on these complexity analyses, binary-relational and ternary-relational items of each of these tasks were developed or existing tasks were selected and/or modified. The binary-relational items were closely matched to the ternary-relational items in terms of stimuli and procedure, however, they were lower in complexity. After pilot testing, the three new measures of hot executive functioning were included in a larger test battery that was administered to a sample of 120 normally developing 3-, 4-, 5- and 6-year-old children. Existing binary- and ternary-relational items assessing theory-of-mind (a hot task) and three cool measures (transitivity, class inclusion and the Dimensional Change Card Sort test) were also included. The inclusion of measures of both hot and cool executive functions, each with complexity manipulated, allowed for the examination of a possible differential age of emergence of executive abilities associated with the DL-PFC versus the OFC. In support of the relational complexity approach, significant complexity effects were found across all seven tasks. Items at a higher level of complexity were experienced as relatively more difficult by children of all ages. Significant effects of age were also observed, with performance across all tasks increasing with age. The age effects were strongest on the ternary-relational items. The pass-fail data indicated that the majority of children in all age groups succeeded on the binary-relational items. However, it was not until a median of five years of age that children were able to process ternary relations. Consequently, the ternary-relational items produce the greatest differences in performance between the four age groups. The overall pattern of the results also suggested that a distinction can be made between the ages of emergence of abilities associated with the OFC versus the DL-PFC. The results of the pass-fail percentages, patterns of age-related change and age effects on domain factor scores all suggested that while hot executive functions may begin to develop around four years of age, similar levels of improvement are not seen in cool executive functions until five years of age. Thus, the ability to succeed on ternary-relational items of hot executive function tasks appeared to emerge slightly earlier than the cool executive function tasks. Complexity appears to be a critical factor underlying children's performance on executive function tasks, and future assessment regarding the development of executive abilities will benefit from keeping this in mind. While some refinement of new task items may be beneficial, the current test battery may have utility in further examinations of the executive profiles underlying clinical groups, such as children with autism and ADHD.
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Książki na temat "Dorsolateral prefrontal cortex"

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Funahashi, Shintaro. Dorsolateral Prefrontal Cortex. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-7268-3.

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Weinberger, Daniel R. Physiologic dysfunction of dorsolateral prefrontal cortex in schizophrenia. [Washington, D.C.?: National Institute of Mental Health, 1986.

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Funahashi, Shintaro. Dorsolateral Prefrontal Cortex: Working Memory and Executive Functions. Springer, 2022.

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Arnsten, Amy F. T., Min J. Wang i Constantinos D. Paspalas. The Neuroscience of Cognition and Cognitive Enhancing Compounds. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190214401.003.0002.

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Higher cognitive disorders involve insults to the neural circuits of the newly evolved association cortices. Although these cortices comprise the majority of the human cortex, little is understood about their molecular modulation. Research on the primate dorsolateral prefrontal cortex (dlPFC) indicates that the newly evolved layer III circuits underlying mental representation are regulated at the molecular level in a manner that is fundamentally different from classic synapses. These mechanisms must be respected to create effective treatments for human disorders, where a major goal is to optimize the network connectivity needed for persistent and precise neural representations. Understanding the needs of dlPFC circuits has led to the successful translation of guanfacine (Intuniv) for treating cognitive disorders, supporting this research strategy.
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Clark, Caroline, Jeffrey Cole, Christine Winter i Geoffrey Grammer. Transcranial Magnetic Stimulation Treatment of Posttraumatic Stress Disorder. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190205959.003.0005.

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Symptoms of post-traumatic stress disorder (PTSD) often fail to resolve with psychotherapy, pharmacotherapy, or integrative medicine treatments. Given these limitations, there is a continued push to discover treatment methods utilizing novel mechanisms of action. Transcranial magnetic stimulation (TMS) offers a non-invasive and safe method of brain stimulation that modulates neuronal activity in a focal area to achieve excitation or inhibition, and may have utility for patients suffering from PTSD, although, to date, evidence of efficacy is limited. The TMS treatment can be varied to suit the needs of the patient by altering the selection of the specific treatment parameters, such as pulse frequency or stimulation intensity. The weight of evidence to date supports treatment of either the right dorsolateral prefrontal cortex or the medical prefrontal cortex. Coupling treatment with script based exposure therapies may also assist with potentiation of the extinction response. Ultimately, stimulation parameters may be related to secondary downstream effects, and thus current targets may indirectly reverse the underlying neuronal pathophysiology. Given that PTSD is a complex illness with a poorly understood pathophysiology, it often exists with other psychiatric comorbidities or TBI. As such, TMS could be an effective part of a comprehensive treatment program.
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Części książek na temat "Dorsolateral prefrontal cortex"

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McLaughlin, Nicole C. R., i Paul Malloy. "Dorsolateral Prefrontal Cortex". W Encyclopedia of Clinical Neuropsychology, 1216–20. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_1887.

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McLaughlin, Nicole C. R., i Paul Malloy. "Dorsolateral Prefrontal Cortex". W Encyclopedia of Clinical Neuropsychology, 1–5. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_1887-2.

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Funahashi, Shintaro. "Dorsolateral Prefrontal Cortex". W Brain Science, 1–51. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-7268-3_1.

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Friston, K. J. "The dorsolateral prefrontal cortex, schizophrenia and PET". W Studies of Brain Metabolism in Psychiatric Patients: Can Standards Be Drawn?, 79–93. Vienna: Springer Vienna, 1992. http://dx.doi.org/10.1007/978-3-7091-9209-2_7.

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Levy, Richard, i Patricia S. Goldman-Rakic. "Segregation of working memory functions within the dorsolateral prefrontal cortex". W Executive Control and the Frontal Lobe: Current Issues, 23–32. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-59794-7_4.

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Petrides, Michael. "The role of the mid-dorsolateral prefrontal cortex in working memory". W Executive Control and the Frontal Lobe: Current Issues, 44–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-642-59794-7_6.

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Zaidi, Tayeb, i Kyoko Fujimoto. "Evaluation and Comparison of Simulated Electric Field Differences Using Three Image Segmentation Methods for TMS". W Brain and Human Body Modelling 2021, 75–87. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-15451-5_5.

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AbstractComputational electromagnetic modeling is a powerful technique to evaluate the effects of electrical stimulation of the human brain. The results of these simulations can vary based on the segmentation of the head and brain generated from the patient images. Using an existing boundary element fast multipole method (BEM-FMM) electromagnetic solver, this work compares the simulated electric field differences resulted by the three segmentation methods. A transcranial magnetic stimulation (TMS) coil targeting both the primary motor cortex and the dorsolateral prefrontal cortex (DLPFC) was simulated. Average field differences were small among the three methods (2% for motor cortex, 3% for DLPFC) and the average field differences in the regions directly surrounding the target stimulation point were 5% for the motor cortex and 2% for DLPFC. More studies evaluating different coils and other segmentation options may further improve the computational modeling for robust TMS treatment.
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Fish, Kenneth N., Guillermo Gonzalez-Burgos, Aleksey V. Zaitsev i David A. Lewis. "Histological Characterization of Physiologically Determined Fast-Spiking Interneurons in Slices of Primate Dorsolateral Prefrontal Cortex". W Isolated Central Nervous System Circuits, 159–81. Totowa, NJ: Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-020-5_4.

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Flores-Leal, M., E. Sacristán-Rock, L. Jiménez-Angeles i J. Azpiroz-Leehan. "Low Frequency Repetitive Transcranial Magnetic Stimulation Effects over Dorsolateral Prefrontal Cortex in Moderate Nicotine Dependent Subjects". W VI Latin American Congress on Biomedical Engineering CLAIB 2014, Paraná, Argentina 29, 30 & 31 October 2014, 317–20. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-13117-7_82.

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Torii, T., K. Nojima, A. Matsunaga, M. Iwahashi i K. Iramina. "Comparison of Influences on P300 Latency in the Case of Stimulating Supramarginal Gyrus and Dorsolateral Prefrontal Cortex by rTMS". W IFMBE Proceedings, 492–95. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-21729-6_124.

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Streszczenia konferencji na temat "Dorsolateral prefrontal cortex"

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de Bruin, H., G. Hasey i J. Hemily. "Dorsolateral prefrontal cortex sensitivity to rTMS". W 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6090562.

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Al-Hakim, Ramsey, James Fallon, Delphine Nain, John Melonakos i Allen Tannenbaum. "A dorsolateral prefrontal cortex semi-automatic segmenter". W Medical Imaging, redaktorzy Joseph M. Reinhardt i Josien P. W. Pluim. SPIE, 2006. http://dx.doi.org/10.1117/12.653643.

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Anderson, N. R., T. Blakely, P. Brunner, D. J. Krusienski, D. W. Moran i E. C. Leuthardt. "High-frequency spectral changes in Dorsolateral Prefrontal Cortex for potential neuoroprosthetics". W 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2013. http://dx.doi.org/10.1109/embc.2013.6609984.

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DALL’AGNOL, Letizzia, Alice Medeiros de SOUZA, Lilian Campos AMADEU, Eleni VOSNIADOU i Fernanda Ishida CORRÊA. "TRANSCRANIAL DIRECT STIMULATION IN THE NEUROMODULATION OF CONTROLLING MAIN SYMPTOMS OF PARKINSON’S DISEASE: A CASE STUDY". W SOUTHERN BRAZILIAN JOURNAL OF CHEMISTRY 2021 INTERNATIONAL VIRTUAL CONFERENCE. DR. D. SCIENTIFIC CONSULTING, 2022. http://dx.doi.org/10.48141/sbjchem.21scon.06_abstract_ishida.pdf.

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Parkinson’s disease (PD) is a central nervous system neurodegenerative disorder that primarily affects the motor system, decreasing motor coordination, balance and generating tremors, and a progressive loss of everyday mobility, including walking. This study was conducted to verify the effects of Transcranial Direct Current Stimulation (tDCS) on balance, motor control, and the quality of life in Parkinson’s disease patients. The patient received three treatments consisting of 10 sessions of 20 minutes each and a one-week interval between treatments. Active stimulation was applied on the primary motor cortex (M1), the dorsolateral prefrontal cortex (DLPFC), and the dorsolateral prefrontal cortex (D Sham-tDCS. DLPFC stimulation produced the best improvements in terms of motor control, balance, gait, and overall PD symptoms, as evaluated by different scales and questionnaires. As a result, active stimulation of the DLPFC produced superior outcomes and may contribute to treating Parkinson’s disease.
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Sato, A., T. Torii, Y. Nakahara, M. Iwahashi, Y. Itoh i K. Iramina. "The impact of rTMS over the dorsolateral prefrontal cortex on cognitive processing". W 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2013. http://dx.doi.org/10.1109/embc.2013.6609919.

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Jawad Khan, M., Keum-Shik Hong, Noman Naseer i M. Raheel Bhutta. "Drowsiness detection in dorsolateral-prefrontal cortex using fNIRS for a passive-BCI". W 2015 15th International Conference on Control, Automation and Systems (ICCAS). IEEE, 2015. http://dx.doi.org/10.1109/iccas.2015.7364653.

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Ngetich, Ronald, Wenjuan Li, Donggang Jin i Ling Li. "Investigating Hemispheric Specialization of the Dorsolateral Prefrontal Cortex in Visuospatial Working Memory". W ICBBS 2021: 2021 10th International Conference on Bioinformatics and Biomedical Science. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3498731.3498760.

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De Geeter, Nele, Guillaume Crevecoeur i Luc Dupre. "The effect of inaccurate targeting of the left dorsolateral prefrontal cortex on TMS response". W 2013 6th International IEEE/EMBS Conference on Neural Engineering (NER). IEEE, 2013. http://dx.doi.org/10.1109/ner.2013.6696060.

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Nguyen Ngoc Phuong Trinh, Truong Quang Dang Khoa i Vo Van Toi. "Investigating the Deceptive Task in Dorsolateral Prefrontal Cortex by Functional Near-infrared Spectroscopy (fNIRS)". W 2013 29th Southern Biomedical Engineering Conference (SBEC 2013). IEEE, 2013. http://dx.doi.org/10.1109/sbec.2013.56.

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Dashtestani, Hadis, Rachel Zaragoza, Riley Kermanian, Kristine Knutson, Milton Halem, Afrouz Anderson i Amir Gandjbakhche. "Importance of Left Dorsolateral Prefrontal Cortex in Moral Judgment Using Functional Near-infrared Spectroscopy". W Clinical and Translational Biophotonics. Washington, D.C.: OSA, 2018. http://dx.doi.org/10.1364/translational.2018.jw3a.52.

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