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Artykuły w czasopismach na temat "DNA model"
Frank-Kamenetskii, M. D., V. V. Anshelevich i A. V. Lukashin. "Polyelectrolyte model of DNA". Uspekhi Fizicheskih Nauk 151, nr 4 (1987): 595. http://dx.doi.org/10.3367/ufnr.0151.198704b.0595.
Pełny tekst źródłaHandelsman, Jo. "Call for Papers: Unique Model Systems". DNA and Cell Biology 27, nr 6 (czerwiec 2008): 287. http://dx.doi.org/10.1089/dna.2008.1504.
Pełny tekst źródłaFrank-Kamenetskiĭ, M. D., V. V. Anshelevich i A. V. Lukashin. "Polyelectrolyte model of DNA". Soviet Physics Uspekhi 30, nr 4 (30.04.1987): 317–30. http://dx.doi.org/10.1070/pu1987v030n04abeh002833.
Pełny tekst źródłaDavies, S. W., i D. A. Seale. "DNA Microarray Stochastic Model". IEEE Transactions on Nanobioscience 4, nr 3 (wrzesień 2005): 248–54. http://dx.doi.org/10.1109/tnb.2005.853665.
Pełny tekst źródłaMiddleton, James. "Handy DNA Nucleotide Model". American Biology Teacher 81, nr 3 (1.03.2019): 193–96. http://dx.doi.org/10.1525/abt.2019.81.3.193.
Pełny tekst źródłaAlireza, Sepehri, Shoorvazi Somayyeh i Moradi Marjaneh Aliakbar. "Calculating the Specific Heat of DNA by using Phononic Model". Greener Journal of Biological Sciences 3, nr 5 (13.07.2013): 187–91. http://dx.doi.org/10.15580/gjbs.2013.5.051613617.
Pełny tekst źródłaKelchner, Scot A. "Phylogenetic models and model selection for noncoding DNA". Plant Systematics and Evolution 282, nr 3-4 (30.07.2008): 109–26. http://dx.doi.org/10.1007/s00606-008-0071-6.
Pełny tekst źródłaXU, Jin, i Yue-Ke FAN. "Classical Ramsey Number DNA Computing Model (Ⅱ): Add-Bit-Sequence DNA computing Model". Chinese Journal of Computers 31, nr 12 (16.10.2009): 2081–89. http://dx.doi.org/10.3724/sp.j.1016.2008.02081.
Pełny tekst źródłaZHAO, YUQI, i HOWARD B. LIEBERMAN. "Schizosaccharomyces pombe:A Model for Molecular Studies of Eukaryotic Genes". DNA and Cell Biology 14, nr 5 (maj 1995): 359–71. http://dx.doi.org/10.1089/dna.1995.14.359.
Pełny tekst źródłaMohamad, Abdul Adheem, i Tsukasa Yashiro. "A TOPOLOGICAL MODEL OF DNA REPLICATION WITH DNA-LINKS". Far East Journal of Mathematical Sciences (FJMS) 107, nr 1 (27.09.2018): 241–55. http://dx.doi.org/10.17654/ms107010241.
Pełny tekst źródłaRozprawy doktorskie na temat "DNA model"
高銘謙 i Ming-him Ko. "A multi-agent model for DNA analysis". Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1999. http://hub.hku.hk/bib/B31222778.
Pełny tekst źródłaKo, Ming-him. "A multi-agent model for DNA analysis /". Hong Kong : University of Hong Kong, 1999. http://sunzi.lib.hku.hk/hkuto/record.jsp?B21949116.
Pełny tekst źródłaLuchetti, Andrea <1976>. "Evolution of repetitive DNA in model arthropods". Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2007. http://amsdottorato.unibo.it/338/.
Pełny tekst źródłaFriedrich, Tomáš. "Komprese DNA sekvencí". Master's thesis, Vysoké učení technické v Brně. Fakulta informačních technologií, 2010. http://www.nusl.cz/ntk/nusl-237222.
Pełny tekst źródłaSantos, Elmer Buluran. "Biologic response to papillomavirus DNA in COPV model". Thesis, University of Cambridge, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.621132.
Pełny tekst źródłaArredondo, Ryan. "Properties of Graphs Used to Model DNA Recombination". Scholar Commons, 2014. https://scholarcommons.usf.edu/etd/4979.
Pełny tekst źródłaDarko, Janice. "Fluorescent Labeling of Antibiotic Resistant Bacteria Model DNA". BYU ScholarsArchive, 2018. https://scholarsarchive.byu.edu/etd/7600.
Pełny tekst źródłaAllegrini, Paolo. "Model for Long-range Correlations in DNA Sequences". Thesis, University of North Texas, 1996. https://digital.library.unt.edu/ark:/67531/metadc279189/.
Pełny tekst źródłaTello, Cajiao John James 1990. "The influence of the DNA conformation on the radiation-induced DNA damage probabilities = A influência da conformação do DNA nas probabilidades de dano induzido por radiações". [s.n.], 2016. http://repositorio.unicamp.br/jspui/handle/REPOSIP/305738.
Pełny tekst źródłaDissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Física Gleb Wataghin
Made available in DSpace on 2018-08-30T22:42:12Z (GMT). No. of bitstreams: 1 TelloCajiao_JohnJames_M.pdf: 2614936 bytes, checksum: e5bdfc91b42434b003cad0b5fa850afb (MD5) Previous issue date: 2016
Resumo: O objetivo deste trabalho é estudar a influência da conformação do DNA na probabilidade de dano direto produzido por partículas ionizantes. Além disso, os fundamentos mecanicísticos do modelo Linear-Quadrático são investigadas através de um modelo biofísico desenvolvido neste trabalho, baseado na TADR (Teoria da Ação Dual da Radiação). Para este fim, três modelos geométricos do material genético foram construídos. Os modelos têm resolução atomística e levam em conta ? 10^9 pares de base (bps) nas configurações A,B e Z do DNA. A partir de um único bp, os diferentes níveis organizacionais no interior do núcleo da célula foram criados por meio de transformações lineares. Em seguida, o código Monte Carlo (MC) GEANT4-DNA foi usado para simular o transporte de prótons de 0.5, 1, 5, 7 e 10 MeV assim como de partículas alfa de 2, 5, 7 e 10M eV . O número de partículas em cada caso é de tal modo que as doses absorvidas estão entre 0.5 ? 16Gy. Os três modelos foram consistentes com as dimensões das estruturas reais. Em particular, os modelos foram compatíveis com a exigência de que o diâmetro da cromatina seja de 30 nm, bem como com os volumes bp reportados em outros trabalhos. Os rendimentos tanto das quebras totais quanto das quebras duplas (TSBY e DSBY) foram obtidos para cada qualidade de radiação. Além disso, a probabilidade de impacto (SHP) definida como a razão entre o volume do DNA e o volume núcleo, foi calculada teoricamente e a partir das simulações. O modelo biofísico em conjunto com as simulações MC forneceu o número de lesões letais (N_LL) em função da dose, para prótons de 0,5 e 10 MeV, e para partículas alfa de 2 e 10 MeV . Os N_LL puderam ser divididos em aqueles criados por uma única trajetória e aqueles originados pela interacção de duas trajetórias. Concluiu-se que o TSBY é praticamente determinada pela SHP e depende fracamente da qualidade de radiação incidente. No entanto, o DSBY mostrou forte dependência tanto da conformação do DNA quanto da qualidade de radiação. Isto é devido à relação entre a capacidade de agrupamento das deposições de energia para uma radiação dada e o empacotamento do DNA. Por outro lado, a análise dos mecanismos de produção de dano, baseada na TADR e testada com o modelo biofísico desenvolvido, mostraram que os efeitos de uma única trajetória (de primeira ordem) dependem linearmente com a dose. Além disso, os efeitos inter-trajetórias seguem um comportamento quadrático com a dose, com um termo linear que influencia o mecanismo de primeira ordem. Isto significa que o comportamento linear-quadrático do N_LL com a dose, tem fundamentos mecanicistas, pelo menos, na primeira fase do dano
Abstract: The aim of this work is to study the influence of the DNA conformation on the probability of direct damage induction by ionizing particles. Also, the mechanistic grounds of the Linear-Quadratic radiobiological model are investigated through the eyes of a home-made biophysical model based on the DRAT (Dual Radiation Action Theory). To this end, three geometrical models of the genetic material were constructed. The models have atomistic resolution and account for ? 10^9 base pairs (bps) in the A-, B- and Z-DNA configurations. Starting from a single bp, the different organizational levels inside the cell nucleus were created by means of linear transformations. Next, the Monte Carlo (MC) code GEANT4-DNA was used to simulate the transport of protons of 0.5, 1, 5, 7 and 10 MeV , and alpha particles of 2, 5, 7 and 10 MeV. The number of particles in each case is such that the absorbed doses range between 0.5 Gy and 16 Gy. The three models proved to be consistent with the dimensions of the real structures. In particular, the models were compatible with the 30 nm chromatin fiber diameter requirement as well as with the bp volumes reported in other works. The Total and Double Strand Break Yields (TSBY and DSBY) were obtained for every radiation quality. Also, the Site-Hit Probability (SHP) defined as the total target to the nucleus volume ratio, was computed theoretically and from the simulations. The biophysical model in conjunction with the MC simulations furnished the number of lethal lesions (N_LL) as a function of dose, for protons of 0.5 and 10 MeV , and for alpha particles of 2 and 10 MeV . The N_LL could be split into those created by a single track and those originated by interaction of two tracks. It is concluded that the TSBY is practically determined by the SHP and depends weakly on the incident radiation quality. Nevertheless, the DSBY showed strong dependence on both the DNA conformation and the radiation quality. This is due to the interplay between the energy deposition clustering capacity of a given radiation and the DNA spatial packing. On the other hand, the analysis of the mechanisms of damage production based on the DRAT and tested with the biophysical model developed, showed that single-track (first order) effects depend linearly on the dose. Moreover, inter-track effects follows a quadratic behavior with the dose, having a linear term that influences the first order mechanism. This means that the Linear-Quadratic behavior of the N_LL with the dose, has mechanistic groundings at least at the first stage of the damage
Mestrado
Física
Mestre em Física
1370449/2014
CAPES
Lee, Kyeong Eun. "Bayesian models for DNA microarray data analysis". Diss., Texas A&M University, 2005. http://hdl.handle.net/1969.1/2465.
Pełny tekst źródłaKsiążki na temat "DNA model"
Sakalosky, G. P. The predictor model. Gatlinburg, TN: Grams Communications Publication, 1992.
Znajdź pełny tekst źródłaRintala, Anne C. DNA repair in a radioresistant breast cancer model system. Sudbury, Ont: Laurentian University, 2000.
Znajdź pełny tekst źródłaSnapka, Robert M. The SV40 replicon model for analysis of anticancer drugs. Austin, TX: R.G. Landes, 1996.
Znajdź pełny tekst źródłaThe SV40 replicon model for analysis of anticancer drugs. [San Diego, Calif.]: Academic Press, 1996.
Znajdź pełny tekst źródłaThe resonant recognition model of macromolecular bioactivity: Theory and applications. Basel: Birkhäuser Verlag, 1997.
Znajdź pełny tekst źródłaSŏ, Yŏng-nok. Yujŏn toksŏng chipʻyo yujŏnja rŭl iyong han yujŏn toksŏng pʻyŏngka model surip yŏnʼgu =: Study on the establishment of evaluation system for genotoxicity using genotoxic biomarker genes. [Seoul]: Sikpʻum Ŭiyakpʻum Anjŏnchʻŏng, 2007.
Znajdź pełny tekst źródłaGurgi, Mohamedkamal Ahmed. ESI-MS[n] of anticancer pt[iv] organoamido complexes and their interactions with DNA-model compounds. St. Catharines, Ont: Brock University, Department of Chemistry, 2001.
Znajdź pełny tekst źródłaDimitriadi, Konstantina. The use of three pronuclei embryos as a model to analyze the uptake of paternal mitochondrial DNA. Birmingham: University of Birmingham, 1999.
Znajdź pełny tekst źródła1959-, Kivshar Y. S., red. The Frenkel-Kontorova model: Concepts, methods, and applications. Berlin: Springer, 2003.
Znajdź pełny tekst źródłaZoya, Ignatova, Martinez-Perez Israel Marck i SpringerLink (Online service), red. DNA Computing Models. Boston, MA: Springer-Verlag US, 2008.
Znajdź pełny tekst źródłaCzęści książek na temat "DNA model"
Nelson, David O., i Terence P. Speed. "Recovering DNA Sequences from Electrophoresis Data". W Image Models (and their Speech Model Cousins), 141–52. New York, NY: Springer New York, 1996. http://dx.doi.org/10.1007/978-1-4612-4056-3_8.
Pełny tekst źródłaOuldridge, Thomas E. "A Novel DNA Model". W Coarse-Grained Modelling of DNA and DNA Self-Assembly, 21–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-30517-7_2.
Pełny tekst źródłaPérez-Jiménez, Mario J., i Fernando Sancho-Caparrini. "Solving Knapsack Problems in a Sticker Based Model". W DNA Computing, 161–71. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/3-540-48017-x_15.
Pełny tekst źródłaKhodor, Julia, i David K. Gifford. "Programmed Mutagenesis Is a Universal Model of Computation". W DNA Computing, 300–307. Berlin, Heidelberg: Springer Berlin Heidelberg, 2002. http://dx.doi.org/10.1007/3-540-48017-x_28.
Pełny tekst źródłaShiozaki, Masashi, Hirotaka Ono, Kunihiko Sadakane i Masafumi Yamashita. "A Probabilistic Model of the DNA Conformational Change". W DNA Computing, 274–85. Berlin, Heidelberg: Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/11925903_21.
Pełny tekst źródłaSahu, Sudheer, Peng Yin i John H. Reif. "A Self-assembly Model of Time-Dependent Glue Strength". W DNA Computing, 290–304. Berlin, Heidelberg: Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/11753681_23.
Pełny tekst źródłaOuldridge, Thomas E. "Thermodynamic Properties of Model DNA". W Coarse-Grained Modelling of DNA and DNA Self-Assembly, 71–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-30517-7_6.
Pełny tekst źródłaHarvey, Stephen C., i Robert K. Z. Tan. "Development of a Model for DNA Supercoiling". W Unusual DNA Structures, 91–101. New York, NY: Springer New York, 1988. http://dx.doi.org/10.1007/978-1-4612-3800-3_6.
Pełny tekst źródłaBaskonus, Haci Mehmet, i Carlo Cattani. "Nonlinear Dynamical Model for DNA". W Trends in Mathematics, 115–41. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-3013-1_7.
Pełny tekst źródłaSakakibara, Yasubumi, i Hiroshi Imai. "A DNA-based Computational Model Using a Specific Type of Restriction Enzyme". W DNA Computing, 315–25. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/3-540-36440-4_28.
Pełny tekst źródłaStreszczenia konferencji na temat "DNA model"
Nishioka, Yuki, Kentaro Doi i Satoyuki Kawano. "Development of an Electron Scattering Model to Detect Differences in DNA Base Molecules". W ASME-JSME-KSME 2011 Joint Fluids Engineering Conference. ASMEDC, 2011. http://dx.doi.org/10.1115/ajk2011-36031.
Pełny tekst źródłaErlander, Stig R. "DNA STRUCTURE: EXPERIMENTAL EVIDENCE AGAINST THE WATSON-CRICK DNA MODEL AND FOR THE ERLANDER DNA MODEL". W XXIst International Carbohydrate Symposium 2002. TheScientificWorld Ltd, 2002. http://dx.doi.org/10.1100/tsw.2002.719.
Pełny tekst źródłaBoyda, Denis Leonidovich. "Mathematical model of DNA lesions". W XXI International Baldin Seminar on High Energy Physics Problems. Trieste, Italy: Sissa Medialab, 2013. http://dx.doi.org/10.22323/1.173.0042.
Pełny tekst źródłaSajfert, V., Lj Mašković i D. Popov. "Model explanation of DNA Transcription". W SIXTH INTERNATIONAL CONFERENCE OF THE BALKAN PHYSICAL UNION. AIP, 2007. http://dx.doi.org/10.1063/1.2733564.
Pełny tekst źródłaLillian, Todd D., N. C. Perkins i S. Goyal. "Computational Elastic Rod Model Applied to DNA Looping". W ASME 2007 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2007. http://dx.doi.org/10.1115/detc2007-34956.
Pełny tekst źródłaHirsh, Andrew D., Todd D. Lillian i N. C. Perkins. "A Model for Highly Strained DNA in a Cavity". W ASME 2011 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2011. http://dx.doi.org/10.1115/detc2011-48711.
Pełny tekst źródłaMartin, Dan, Mohamad Eid i Abdulmotaleb El Saddik. "A Haptic Enabled DNA Model Sensing". W 2008 9th International Symposium on Parallel Architectures, Algorithms and Networks (ISPAN '08). IEEE, 2008. http://dx.doi.org/10.1109/i-span.2008.47.
Pełny tekst źródłaLiu, Wei, Shouxia Sun i Ying Guo. "A DNA Computing Model of Perceptron". W 2009 Pacific-Asia Conference on Circuits, Communications and Systems (PACCS). IEEE, 2009. http://dx.doi.org/10.1109/paccs.2009.182.
Pełny tekst źródłaLiu, Yaling, i Samir M. Iqbal. "A Mesoscale Model for Molecular Interaction in Functionalized Nanopores". W ASME 2008 International Mechanical Engineering Congress and Exposition. ASMEDC, 2008. http://dx.doi.org/10.1115/imece2008-68542.
Pełny tekst źródłaLi, Wentian. "DNA segmentation as a model selection process". W the fifth annual international conference. New York, New York, USA: ACM Press, 2001. http://dx.doi.org/10.1145/369133.369202.
Pełny tekst źródłaRaporty organizacyjne na temat "DNA model"
Jackson, Peter K. DNA Replication Initiator Proteins and Genetic Instability: Creating a Mouse Model for Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, wrzesień 1999. http://dx.doi.org/10.21236/ada392190.
Pełny tekst źródłaMincheff, Milcho S. Naked DNA Immunization for Prevention of Prostate Cancer in a Dunning Rat Prostate Tumor Model. Fort Belvoir, VA: Defense Technical Information Center, czerwiec 2003. http://dx.doi.org/10.21236/ada417656.
Pełny tekst źródłaKinney, Shannon R. Examination of the Role of DNA Methylation Changes in Prostate Cancer using the Transgenic Adenocarcinoma of Mouse Prostate (TRAMP) Model. Fort Belvoir, VA: Defense Technical Information Center, marzec 2009. http://dx.doi.org/10.21236/ada502739.
Pełny tekst źródłaMorey Kinney, Shannon R. Examination of the Role of DNA Methylation Changes in Prostate Cancer using the Transgenic Adenocarcinoma of Mouse Prostate (TRAMP) Model. Fort Belvoir, VA: Defense Technical Information Center, marzec 2010. http://dx.doi.org/10.21236/ada525616.
Pełny tekst źródłaMorey, Shannon R. Examination of the Role of DNA Methylation Changes in Prostate Cancer Using the Transgenic Adenocarcinoma of Mouse Prostate (TRAMP) Model. Fort Belvoir, VA: Defense Technical Information Center, marzec 2008. http://dx.doi.org/10.21236/ada483443.
Pełny tekst źródłaChristman, Judith K. Role of DNA Methylation in Altering Gene Expression During the Early Stages of Human Breast Cancer Progression in the MCF10AT Xenograft Model. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2003. http://dx.doi.org/10.21236/ada418564.
Pełny tekst źródłaChristman, Judith K. Role of DNA Methylation in Altering Gene Expression During the Early Stages of Human Breast Cancer Progression in the MCF10AT Xenograft Model. Fort Belvoir, VA: Defense Technical Information Center, kwiecień 2004. http://dx.doi.org/10.21236/ada426221.
Pełny tekst źródłaMacedo, Luciana, i Linda Malkas. The Human Breast Cancer DNA Synthesome Can Serve as a Novel In Vitro Model System for Studying the Mechanism of Action of Anticancer Drugs. Fort Belvoir, VA: Defense Technical Information Center, lipiec 2000. http://dx.doi.org/10.21236/ada393926.
Pełny tekst źródłaCucinotta, Francis A. Systems Biology Model of Interactions between Tissue Growth Factors and DNA Damage Pathways: Low Dose Response and Cross-Talk in TGFβ and ATM Signaling. Office of Scientific and Technical Information (OSTI), wrzesień 2016. http://dx.doi.org/10.2172/1335567.
Pełny tekst źródłaO'Neill, Peter, i Jennifer Anderson. Systems Biology Model of Interactions Between Tissue Growth Factors and DNA Damage Pathways: Low Dose Response and Cross-Talk in TGFbeta and ATM Signaling. Office of Scientific and Technical Information (OSTI), październik 2014. http://dx.doi.org/10.2172/1158919.
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